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Articoli di riviste sul tema "Ly"

Autore: Grafiati

Pubblicato: 4 giugno 2021

Ultima modifica: 26 luglio 2025

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1

Andrianarijaona, V. M., D. Wulf, D. McCammon, D. G. Seely та C. C. Havener. "Radiance line ratios Ly-β/Ly-α, Ly-γ/Ly-α, Ly-δ/Ly-α, and Ly-ε/Ly-α for soft X-ray emissions following charge exchange between C6+ and Kr". Nuclear Instruments and Methods in Physics Research Section B: Beam Interactions with Materials and Atoms 350 (травень 2015): 122–26. http://dx.doi.org/10.1016/j.nimb.2015.01.040.

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2

Hori, Masatsugu. "RE-LY Trial." Nihon Naika Gakkai Zasshi 101, no. 5 (2012): 1432–39. http://dx.doi.org/10.2169/naika.101.1432.

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3

Ying, Liu. "Developing China's Regulation of Health Care Data." Biotechnology Law Report 39, no. 1 (2020): 19–24. http://dx.doi.org/10.1089/blr.2019.29153.ly.

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4

Gorman, S. D., Y. H. Sun, R. Zamoyska, and J. R. Parnes. "Molecular linkage of the Ly-3 and Ly-2 genes. Requirement of Ly-2 for Ly-3 surface expression." Journal of Immunology 140, no. 10 (1988): 3646–53. http://dx.doi.org/10.4049/jimmunol.140.10.3646.

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Abstract We have isolated the Ly-3 gene by chromosomal walking from the previously cloned Ly-2 gene. These genes are shown to be 36 kb apart and in the same transcriptional orientation. Transfection of the Ly-3 gene into mouse L cells results in cell surface expression of Ly-3 protein only in the presence of Ly-2 (or its human homolog, CD8), although Ly-2 surface expression is not similarly dependent on Ly-3. cDNA clones encoding Ly-3 have been isolated and sequenced and show little sequence similarity to Ly-2, whereas both Ly-2 and Ly-3 are homologous to Ig variable regions. One cDNA clone en

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5

&NA;. "LY 303366." Drugs in R & D 1, no. 2 (1999): 176–78. http://dx.doi.org/10.2165/00126839-199901020-00016.

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6

Roberston, D. W., and J. S. Hayes. "LY-175326." Drugs of the Future 10, no. 4 (1985): 295. http://dx.doi.org/10.1358/dof.1985.010.04.65136.

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7

Chu, S. S. "LY-171883." Drugs of the Future 10, no. 8 (1985): 632. http://dx.doi.org/10.1358/dof.1985.010.08.70125.

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8

Robertson, D. W., and J. S. Hayes. "LY-195115." Drugs of the Future 11, no. 5 (1986): 377. http://dx.doi.org/10.1358/dof.1986.011.05.53867.

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9

Mannhold, R. "LY-127210." Drugs of the Future 11, no. 9 (1986): 748. http://dx.doi.org/10.1358/dof.1986.011.09.49805.

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10

Prous, J., and J. Castañer. "LY-195448." Drugs of the Future 13, no. 12 (1988): 1045. http://dx.doi.org/10.1358/dof.1988.013.12.77457.

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11

Fromtling, R. A. "LY-303366." Drugs of the Future 19, no. 4 (1994): 338. http://dx.doi.org/10.1358/dof.1994.019.04.244501.

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12

Fromtling, R. A., and J. Castañer. "LY-333328." Drugs of the Future 23, no. 1 (1998): 17. http://dx.doi.org/10.1358/dof.1998.023.01.439603.

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13

Pflugh, David L., Stephen E. Maher, and Alfred L. M. Bothwell. "Ly-6 Superfamily Members Ly-6A/E, Ly-6C, and Ly-6I Recognize Two Potential Ligands Expressed by B Lymphocytes." Journal of Immunology 169, no. 9 (2002): 5130–36. http://dx.doi.org/10.4049/jimmunol.169.9.5130.

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14

Schlueter, A. J., T. R. Malek, C. N. Hostetler, P. A. Smith, P. deVries, and T. J. Waldschmidt. "Distribution of Ly-6C on lymphocyte subsets: I. Influence of allotype on T lymphocyte expression." Journal of Immunology 158, no. 9 (1997): 4211–22. http://dx.doi.org/10.4049/jimmunol.158.9.4211.

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Abstract The expression patterns of the Ly-6C Ag were examined on splenic and thymic lymphocyte subsets of Ly-6.1 and Ly-6.2 strains of mice using the rat mAb 15.1. Ly-6C is expressed on subsets of CD4+ and CD8+ splenocytes, and a portion of NK cells. Within the splenic and lymph node CD4+ T cell compartment, Ly-6C expression is restricted to Ly-6.2 strains of mice, and is present on a subset of naive cells. Ly-6C is expressed on the majority of peripheral CD8+ T cells in both Ly-6.1 and Ly-6.2 strains, and is found primarily on the Ag-experienced subset. In the thymus, Ly-6C is present on sub

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15

Fleming, T. J., C. O'hUigin, and T. R. Malek. "Characterization of two novel Ly-6 genes. Protein sequence and potential structural similarity to alpha-bungarotoxin and other neurotoxins." Journal of Immunology 150, no. 12 (1993): 5379–90. http://dx.doi.org/10.4049/jimmunol.150.12.5379.

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Abstract Genomic clones cross-hybridizing with Ly-6A.2 cDNA were isolated and characterized for functional Ly-6-related genes. Two new Ly-6 genes, designated Ly-6F.1 and Ly-6G.1, were found to have high nucleotide homology (&gt; or = 70%) and the characteristic four exon gene organization of Ly-6A/E and Ly-6C. By a PCR-based assay, Ly-6G.1 mRNA was readily found in bone marrow, whereas Ly-6F.1 mRNA was not detected in lymphoid tissues. Thus, Ly-6G.1 represents an additional Ly-6 gene with apparent selective expression in hematopoietic cells distinct from Ly-6A/E and Ly-6C. Using the availa

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16

Hurt, G. W. "Soil-ly Haiku." Soil Horizons 41, no. 3 (2000): 93. http://dx.doi.org/10.2136/sh2000.3.0093.

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17

Thariat, Juliette. "EXTREME-ly Hot." International Journal of Radiation Oncology*Biology*Physics 111, no. 2 (2021): 311. http://dx.doi.org/10.1016/j.ijrobp.2019.07.053.

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18

Green, Andrew. "Ly Penh Sun." Lancet 399, no. 10324 (2022): 516. http://dx.doi.org/10.1016/s0140-6736(22)00199-4.

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19

Lloyd-Hazlett, Jessica, Eleni Maria Honderich, and Karena J. Heyward. "Fa-MI-ly." Family Journal 24, no. 1 (2015): 31–37. http://dx.doi.org/10.1177/1066480715615666.

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20

Martin-Loeches, Ignacio, Marcio Soares, and Antoni Torres. "Neces-SARI-ly?" Intensive Care Medicine 42, no. 5 (2016): 928–30. http://dx.doi.org/10.1007/s00134-016-4243-5.

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21

Palfree, Roger. "Ly-6 nomenclature." Immunology Today 12, no. 1 (1991): 45–46. http://dx.doi.org/10.1016/0167-5699(91)90112-7.

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22

Paulsson, J., and G. Petrochilos. "Neer-ly Misled?" ICSID Review 22, no. 2 (2007): 242–57. http://dx.doi.org/10.1093/icsidreview/22.2.242.

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23

Sawyer, J. S. "LY-293111 Sodium." Drugs of the Future 21, no. 6 (1996): 610. http://dx.doi.org/10.1358/dof.1996.021.06.360855.

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24

Walker, Ian D., Brendan J. Murray, Louis Kirszbaum, Geoffrey W. Chambers, Nicholas J. Deacon, and Ian F. C. McKenzie. "The amino-terminal sequences of Ly-2 and Ly-3." Immunogenetics 23, no. 1 (1986): 60–63. http://dx.doi.org/10.1007/bf00376523.

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25

Bouzyk, E., I. Gradzka, T. Iwaneńko, M. Kruszewski, B. Sochanowicz, and I. Szumiel. "The response of L5178Y lymphoma sublines to oxidative stress: antioxidant defence, iron content and nuclear translocation of the p65 subunit of NF-kappaB." Acta Biochimica Polonica 47, no. 4 (2000): 881–88. http://dx.doi.org/10.18388/abp.2000_3943.

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We examined the response to hydrogen peroxide of two L5178Y (LY) sublines which are inversely cross-sensitive to hydrogen peroxide and X-rays: LY-R cells are radio-resistant and hydrogen peroxide-sensitive, whereas LY-S cells are radiosensitive and hydrogen peroxide-resistant. Higher initial DNA breaks and higher iron content (potentially active in the Fenton reaction) were found in the hydrogen peroxide sensitive LY-R cells than in the hydrogen peroxide resistant LY-S cells, whereas the antioxidant defence of LY-R cells was weaker. In particular, catalase activity is twofold higher in LY-S th

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26

Smith, Kathleen M., Jun Wu, Alexander B. H. Bakker, Joseph H. Phillips, and Lewis L. Lanier. "Cutting Edge: Ly-49D and Ly-49H Associate with Mouse DAP12 and Form Activating Receptors." Journal of Immunology 161, no. 1 (1998): 7–10. http://dx.doi.org/10.4049/jimmunol.161.1.7.

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Abstract Several members of the Ly-49 receptor family inhibit NK cell-mediated lysis of targets expressing appropriate MHC class I molecules. Ly-49D and Ly-49H, two Ly-49 molecules that lack immunoreceptor tyrosine-based inhibitory motifs (ITIM) in their cytoplasmic domains, associate with mouse DAP12, a molecule that possesses an immunoreceptor tyrosine-based activation motif (ITAM). Cotransfection of either Ly-49D or Ly-49H with DAP12 induces surface expression of both Ly-49 and DAP12. The Ly-49/DAP12 complex was coimmunoprecipitated from the transfected cells, demonstrating a physical assoc

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27

Izotov, Y. I., D. Schaerer, G. Worseck та ін. "Diverse properties of Ly α emission in low-redshift compact star-forming galaxies with extremely high [O iii]/[O ii] ratios". Monthly Notices of the Royal Astronomical Society 491, № 1 (2019): 468–82. http://dx.doi.org/10.1093/mnras/stz3041.

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ABSTRACT We present observations with the Cosmic Origins Spectrograph onboard the Hubble Space Telescope of eight compact star-forming galaxies at redshifts z = 0.02811–0.06540, with low oxygen abundances 12 + log(O/H) = 7.43–7.82 and extremely high emission-line flux ratios O32 = [O iii] λ5007/[O ii] λ3727 ∼ 22–39, aiming to study the properties of Ly α emission in such conditions. We find a diversity in Ly α properties. In five galaxies Ly α emission line is strong, with equivalent width (EW) in the range 45–190 Å. In the remaining galaxies, weak Ly α emission with EW(Ly α) ∼ 2–7 Å is superp

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28

Ortaldo, John R., Robin Winkler-Pickett, Jami Willette-Brown, et al. "Structure/Function Relationship of Activating Ly-49D and Inhibitory Ly-49G2 NK Receptors." Journal of Immunology 163, no. 10 (1999): 5269–77. http://dx.doi.org/10.4049/jimmunol.163.10.5269.

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Abstract Murine NK cells express Ly-49 family receptors capable of either inhibiting or activating lytic function. The overlapping patterns of expression of the various receptors have complicated their precise biochemical characterization. Here we describe the use of the Jurkat T cell line as the model for the study of Ly-49s. We demonstrate that Ly-49D is capable of delivering activation signals to Jurkat T cells even in the absence of the recently described Ly-49D-associated chain, DAP-12. Ly-49D signaling in Jurkat leads to tyrosine phosphorylation of TCRζ and requires Syk/Zap70 family kina

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29

Smith, Hamish R. C., Hubert H. Chuang, Lawrence L. Wang, Margarita Salcedo, Jonathan W. Heusel, and Wayne M. Yokoyama. "Nonstochastic Coexpression of Activation Receptors on Murine Natural Killer Cells." Journal of Experimental Medicine 191, no. 8 (2000): 1341–54. http://dx.doi.org/10.1084/jem.191.8.1341.

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Murine natural killer cells (NK) express lectin-like activation and inhibitory receptors, including the CD94/NKG2 family of receptors that bind Qa-1, and the Ly-49 family that recognizes major histocompatibility complex class I molecules. Here, we demonstrate that cross-linking of NK cells with a new specific anti–Ly-49H mAb induced NK cell cytotoxicity and cytokine production. Ly-49H is expressed on a subset of NK cells and can be coexpressed with Ly-49 inhibitory receptors. However, unlike Ly-49 inhibitory receptors, Ly-49H is not detectable on naive splenic CD3+ T cells, indicating that Ly-

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30

Fleming, T. J., M. L. Fleming, and T. R. Malek. "Selective expression of Ly-6G on myeloid lineage cells in mouse bone marrow. RB6-8C5 mAb to granulocyte-differentiation antigen (Gr-1) detects members of the Ly-6 family." Journal of Immunology 151, no. 5 (1993): 2399–408. http://dx.doi.org/10.4049/jimmunol.151.5.2399.

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Abstract Mouse Ly-6 proteins are characterized by lineage-restricted patterns of expression on lymphoid cells. A mAb (1A8) was produced to Ly-6G, a newly described member of the Ly-6 locus. Based on selective reactivity to cloned Ly-6 gene products expressed in EL4J cells, 1A8 was determined to be specific for Ly-6G. Furthermore, mAb to other Ly-6 specificities did not bind to Ly-6G-transfected EL4J cells, indicating that Ly-6G is distinct from other serologically defined Ly-6 specificities. FACS analysis using 1A8 demonstrated that Ly-6G was expressed in bone marrow but not substantially on o

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31

Spangrude, GJ, and DM Brooks. "Mouse strain variability in the expression of the hematopoietic stem cell antigen Ly-6A/E by bone marrow cells." Blood 82, no. 11 (1993): 3327–32. http://dx.doi.org/10.1182/blood.v82.11.3327.3327.

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Abstract The cell surface molecule Ly-6A/E provides a convenient marker for primitive stem cells in the hematopoietic tissues of both fetal and adult mice. However, previous studies have shown that Ly-6A/E expression by lymphocytes is variable depending on the haplotype of the Ly-6 locus. Therefore, strain-specific variation in Ly-6A/E expression by bone marrow (BM) cells was investigated. The results show that Ly-6a mice have, on average, 50% of the number of BM cells expressing Ly-6A/E relative to that for Ly-6b mice. Furthermore, among the 5% of BM cells that do not express antigens charact

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32

Spangrude, GJ, and DM Brooks. "Mouse strain variability in the expression of the hematopoietic stem cell antigen Ly-6A/E by bone marrow cells." Blood 82, no. 11 (1993): 3327–32. http://dx.doi.org/10.1182/blood.v82.11.3327.bloodjournal82113327.

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The cell surface molecule Ly-6A/E provides a convenient marker for primitive stem cells in the hematopoietic tissues of both fetal and adult mice. However, previous studies have shown that Ly-6A/E expression by lymphocytes is variable depending on the haplotype of the Ly-6 locus. Therefore, strain-specific variation in Ly-6A/E expression by bone marrow (BM) cells was investigated. The results show that Ly-6a mice have, on average, 50% of the number of BM cells expressing Ly-6A/E relative to that for Ly-6b mice. Furthermore, among the 5% of BM cells that do not express antigens characteristic o

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33

Nakamura, Mary C., Shigenari Hayashi, Eréne C. Niemi, James C. Ryan, and William E. Seaman. "Activating Ly-49d and Inhibitory Ly-49a Natural Killer Cell Receptors Demonstrate Distinct Requirements for Interaction with H2-Dd." Journal of Experimental Medicine 192, no. 3 (2000): 447–54. http://dx.doi.org/10.1084/jem.192.3.447.

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The activating Ly-49D receptor and the inhibitory Ly-49A receptor mediate opposing effects on natural killer (NK) cell cytotoxicity after interaction with the same major histocompatibility complex ligand, H2-Dd. To compare Ly-49D and Ly-49A interactions with H2-Dd, we created mutations in H2-Dd and examined the functional ability of these mutants to activate lysis through Ly-49D or to inhibit lysis through Ly-49A. Specific single amino acid changes in either the H2-Dd α1 helix or the α2 helix abrogated Ly-49D–mediated cytotoxicity, but these changes had no significant effect on Ly-49A–dependen

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34

Brennan, Jack, Suzanne Lemieux, J. Douglas Freeman, Dixie L. Mager, and Fumio Takei. "Heterogeneity Among Ly-49C Natural Killer (NK) Cells: Characterization of Highly Related Receptors with Differing Functions and Expression Patterns." Journal of Experimental Medicine 184, no. 6 (1996): 2085–90. http://dx.doi.org/10.1084/jem.184.6.2085.

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Ly-49C is a member of the polymorphic family of murine NK cell inhibitory receptors. The 5E6 antibody that defines a subset of NK cells responsible for the rejection of parental H-2d bone marrow by F1 mice has been shown previously to react with Ly-49C. Here, the 5E6 antibody was found to detect two Ly-49C-related molecules in B6 mice. Two cDNA clones were isolated from B6 NK cells, one identical to previously reported Ly-49CB6 and the other a novel cDNA. The deduced amino acid sequence of the latter differs from that of Ly-49CBALB at only 4 residues, whereas the previously reported Ly-49CB6 d

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35

Fleming, T. J., and T. R. Malek. "Multiple glycosylphosphatidylinositol-anchored Ly-6 molecules and transmembrane Ly-6E mediate inhibition of IL-2 production." Journal of Immunology 153, no. 5 (1994): 1955–62. http://dx.doi.org/10.4049/jimmunol.153.5.1955.

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Abstract Cross-linking of Ly-6 molecules on T lymphocytes leads to IL-2 production, whereas costimulation of T cells via Ly-6A/E and the TCR inhibits IL-2 secretion. This study was initiated to determine whether there are unique structural requirements at the level of the Ly-6 molecule for its capacity to activate or block IL-2 production. Functional studies in which transfected EL-4J cells that expressed various Ly-6 proteins or chimeric Ly-6 molecules were used have demonstrated that direct activation of IL-2 secretion or inhibition of anti-CD3-induced IL-2 production occurred after mAb bind

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36

Malek, T. R., K. M. Danis, and E. K. Codias. "Tumor necrosis factor synergistically acts with IFN-gamma to regulate Ly-6A/E expression in T lymphocytes, thymocytes and bone marrow cells." Journal of Immunology 142, no. 6 (1989): 1929–36. http://dx.doi.org/10.4049/jimmunol.142.6.1929.

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Abstract The Ly-6 alloantigens represent a family of phosphatidylinositol anchored proteins that function in the process of T lymphocyte activation and whose expression are often induced on T and B lymphocytes after activation by mitogens or Ag. Previous studies have shown that the induction of Ly-6 alloantigens in T cells is at least in part due to the action of IFN-alpha/beta or IFN-gamma. In the present report, we have demonstrated that IFN-gamma also induced Ly-6 molecules on B lymphocytes, several B cell tumors, and bone marrow cells. Furthermore, we now show that TNF also participates in

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37

Smailagić, M., M. Micic та N. Martinović. "Modelling the evolution of Ly α blobs and Ly α emitters". Monthly Notices of the Royal Astronomical Society 459, № 1 (2016): 84–98. http://dx.doi.org/10.1093/mnras/stw462.

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38

Nagendra, Sanjai, and Annette J. Schlueter. "Absence of cross-reactivity between murine Ly-6C and Ly-6G." Cytometry 58A, no. 2 (2004): 195–200. http://dx.doi.org/10.1002/cyto.a.20007.

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39

Robinson, W. H., M. M. Landolfi, H. Schäfer, and J. R. Parnes. "Biochemical identity of the mouse Ly-19.2 and Ly-32.2 alloantigens with the B cell differentiation antigen Lyb-2/CD72." Journal of Immunology 151, no. 9 (1993): 4764–72. http://dx.doi.org/10.4049/jimmunol.151.9.4764.

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Abstract Lyb-2/CD72 is a 45-kDa mouse B cell surface protein that binds CD5 (Ly-1) and has been shown to induce B cell proliferation upon mAb binding. The serologically defined Ly-19.2 and Ly-32.2 lymphocyte alloantigens have mouse strain distribution patterns similar to that of the Lyb-2/CD72 alleles and map to the same region on chromosome 4 as Lyb-2/CD72. Our recent isolation of the Lyb-2a, -2b, and -2c cDNA has enabled us in this report to examine the relationship between Ly-19, Ly-32, and Lyb-2/CD72. A rat T cell line transfected with a mouse Lyb-2a cDNA is recognized by Ly-19.2-specific

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40

ا.م. ندى عزيز يوسف. "THE SYNTACTIC BEHAVIOR OF –LYADVERBS AND –LY ADJECTIVES." Journal of the College of Basic Education 29, no. 119 (2023): 15–1. http://dx.doi.org/10.35950/cbej.v29i119.10568.

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This paper is an attempt to demonstrate the syntactic behavior of -ly adverbs and -ly adjectives. It mainly deals with -ly as an inflectional suffix that forms adverbs and adjectives It is hypothesized that there are differences between adjective-forming –ly and adverb-forming –ly.The researcher first made general and specific observations about the morphological processes of -ly adverbs and -ly adjectives. Since the study focuses on a linguistic phenomenon, its data is a set of -ly adverbs and -ly adjectives used as examples to support the hypothesis. The importance of studying the syntactic

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41

Codias, E. K., T. J. Fleming, C. M. Zacharchuk, J. D. Ashwell, and T. R. Malek. "Role of Ly-6A/E and T cell receptor-zeta for IL-2 production. Phosphatidylinositol-anchored Ly-6A/E antagonizes T cell receptor-mediated IL-2 production by a zeta-independent pathway." Journal of Immunology 149, no. 6 (1992): 1825–52. http://dx.doi.org/10.4049/jimmunol.149.6.1825.

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Abstract Ly-6A/E molecules were originally implicated in regulation of T cell activation because anti-Ly-6A/E mAb induce IL-2 production. More recently we have shown that anti-Ly-6A/E also inhibits IL-2 production induced by anti-CD3. In the present study we used mutant and transfected cell lines that varied in expression of Ly-6A/E or TCR-zeta to test whether the positive and negative modulations of IL-2 production by anti-Ly-6A/E occur by distinct mechanisms. Anti-Ly-6A/E inhibited anti-CD3-induced IL-2 production for Ly-6E.1-transfected EL4J cells, but did not affect IL-2 production of the

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42

Codias, E. K., and T. R. Malek. "Subsets of activated CD4 T lymphocytes refractory for secretion of IL-2 are distinguished by expression of Ly-6A/E in BALB/c mice." Journal of Immunology 151, no. 11 (1993): 5918–29. http://dx.doi.org/10.4049/jimmunol.151.11.5918.

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Abstract (sommario):

Abstract Ly-6A/E, a cell surface glycosylphosphatidylinositol-anchored protein that modulates T cell activation, is expressed on developing and mature T cells and is tightly regulated in a haplotype- and subset-specific manner. We examined whether Ly-6A/E(low)CD4+ and Ly-6A/E(high)CD4+ T cells comprised functional subsets. Peripheral CD4+ T cells were primed in vitro with Con A in the presence or absence of IL-4 or IFN-gamma, sorted for Ly-6A/E expression, and restimulated to induce lymphokine production. Regardless of priming conditions, IL-2 production by Ly-6A/E(high)CD4+ effector T cells w

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43

Brennan, J., D. Mager, W. Jefferies, and F. Takei. "Expression of different members of the Ly-49 gene family defines distinct natural killer cell subsets and cell adhesion properties." Journal of Experimental Medicine 180, no. 6 (1994): 2287–95. http://dx.doi.org/10.1084/jem.180.6.2287.

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The murine Ly-49 antigen belongs to a family of type II transmembrane molecules containing lectin-like domains. The original member of this family, Ly-49A, has been demonstrated to be expressed by a subpopulation of natural killer (NK) cells, bind certain class I major histocompatibility complexes (MHC), and act as a negative regulator of lytic activity. The expression patterns and functional activities of the other Ly-49s, however, is unknown. We extended the study of this family by isolating cDNAs encoding two new Ly-49 molecules. The reactivity of these and previously identified Ly-49 molec

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44

Desmara, Nadhea Lisa, Fitri Palupi Kusumawati, and Aulia Hanifah Qomar. "AN ANALYSIS OF DERIVATIONAL SUFFIXES IN JUSTIN BIEBER’S CHANGES ALBUM." Journal of English Education and Enterpreneurship (JEEP) 2, no. 1 (2022): 42–54. http://dx.doi.org/10.24127/jeep.v2i1.1746.

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Derivational suffix is a part of linguistics learn about word whichadded by affixes may change the meaning and the part of speech. This researchdeals with the derivational suffix of song lyrics. The researcher focuses onderivational suffix types and function. The objectives of the research were todescribe types and function about change class changing and class maintaining inderivational suffix in song lyrics. Descriptive qualitative method was used toanalyze the data. The techniques used in collecting the data is documentationThis research applied the theory of Plag (2002) to indentify the ty

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McGrew, J. T., and K. L. Rock. "Isolation, expression, and sequence of the TAP/Ly-6A.2 chromosomal gene." Journal of Immunology 146, no. 10 (1991): 3633–38. http://dx.doi.org/10.4049/jimmunol.146.10.3633.

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Abstract The murine Ly-6 locus controls the expression of a number of genes. One of the products of the Ly-6 locus, Ly-6A.2, has been implicated in the process of T cell activation. We have identified the chromosomal sequences encoding the Ly-6A.2 molecule using very stringent hybridization and washing conditions. We confirmed that this gene encoded the Ly-6A.2 molecule by transfection studies using a cell line genetically negative for the Ly-6A.2 gene as a DNA recipient. Sequence analysis showed that the Ly-6A.2 gene is made up of four exons. The start site of transcription was determined by

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Ribes, Sandra, Tommy Regen, Tanja Meister, et al. "Resistance of the Brain to Escherichia coli K1 Infection Depends on MyD88 Signaling and the Contribution of Neutrophils and Monocytes." Infection and Immunity 81, no. 5 (2013): 1810–19. http://dx.doi.org/10.1128/iai.01349-12.

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ABSTRACTEscherichia coliis the leading cause of Gram-negative neonatal bacterial meningitis and also causes meningitis and meningoencephalitis in older and immunocompromised patients. Here, we determined the contribution of granulocytes, monocytes, and TLR signaling cascades in the resistance of adult mice toEscherichia coliK1 brain infection. Deficiency in MyD88 (myd88−/−) but not in TRIF (triflps2) adaptor proteins dramatically reduced the survival of animals. Depletion of CD11b+Ly-6G+Ly-6Cintneutrophils by application of the anti-Ly-6G (1A8) monoclonal antibody (MAb) led to higher bacterial

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Lian, Rebecca H., Yan Li, Satoko Kubota, Dixie L. Mager, and Fumio Takei. "Recognition of Class I MHC by NK Receptor Ly-49C: Identification of Critical Residues." Journal of Immunology 162, no. 12 (1999): 7271–76. http://dx.doi.org/10.4049/jimmunol.162.12.7271.

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Abstract The Ly-49 family of inhibitory receptors plays a major role in regulating mouse NK cell cytotoxicity. Two of its members, Ly-49C and I, are recognized by the mAb 5E6, which also defines a subset of NK cells involved in the hybrid resistance phenomenon. Previous studies have shown that Ly-49C binds to a broad spectrum of class I MHC molecules, while Ly-49I apparently does not bind to any class I MHC molecules tested. In the present investigation we have defined the amino acid residues of Ly-49C that are critical for determining its ligand specificities. First, using quantitative COS ce

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Ortega, G., P. E. Korty, E. M. Shevach, and T. R. Malek. "Role of Ly-6 in lymphocyte activation. I. Characterization of a monoclonal antibody to a nonpolymorphic Ly-6 specificity." Journal of Immunology 137, no. 10 (1986): 3240–46. http://dx.doi.org/10.4049/jimmunol.137.10.3240.

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Abstract In studies with alloantisera and monoclonal antibodies (mAb) a number of antigenic determinants have been defined that are the products of the Ly-6 locus on murine chromosome 2 and that are expressed primarily on B and T lymphoid cells. It remains controversial whether these antigenic determinants are encoded by a single gene or a multigene complex. We have characterized a new rat mAb, D7, which recognizes a cell surface antigen whose expression on nonactivated peripheral lymphocytes varies from strain to strain. The phenotype of the staining profile, i.e., high or low percentage of D

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Goldman, Jonathan Wade, Lee S. Rosen, Alain Patrick Algazi, et al. "First-in-human dose escalation study of LY2875358 (LY), a bivalent MET antibody, as monotherapy and in combination with erlotinib (E) in patients with advanced cancer." Journal of Clinical Oncology 31, no. 15_suppl (2013): 8093. http://dx.doi.org/10.1200/jco.2013.31.15_suppl.8093.

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8093 Background: Activation of the hepatocyte growth factor (HGF)/MET receptor pathway promotes tumor growth, invasion and dissemination. LY is a humanized IgG4 monoclonal bivalent antibody against MET which inhibits ligand dependent- and ligand independent activation of MET. Based on preclinical results, we examined LY alone in patients with advanced solid tumors and LY+E in advanced NSCLC patients. Methods: LY monotherapy was administered 20-2,000 mg Q2W IV to 23 patients with advanced solid tumors. Combination therapy with 700-2,000 mg Q2W IV of LY and E (150 mg QD) was completed in 14 pati

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Raveche, E. S., P. Lalor, A. Stall, and J. Conroy. "In vivo effects of hyperdiploid Ly-1+ B cells of NZB origin." Journal of Immunology 141, no. 12 (1988): 4133–39. http://dx.doi.org/10.4049/jimmunol.141.12.4133.

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Abstract Cells with increased chromosome number and DNA content have been found in the spleens of old NZB mice. These hyperdiploid cells are of clonal origin and demonstrate discrete IgH chain gene rearrangements by Southern blot analysis. In this report, hyperdiploid cells were analyzed by three-color flow cytometric techniques and found to be Ly-1+ B cells which were dull for Ly-1 and bright for surface IgM. These cells, unlike typical diploid Ly-1+ B cells, were negative for B220/6B2 and surface IgD. Hyperdiploid Ly-1+ B cells were found to be the predominant splenic subpopulation in animal

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