Bibliografie tematiche / Melatonin. Nervous system

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Letteratura scientifica selezionata sul tema "Melatonin. Nervous system"

Autore: Grafiati
Pubblicato: 4 giugno 2021
Ultima modifica: 16 febbraio 2022

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Articoli di riviste sul tema "Melatonin. Nervous system"

1

Hardeland, Rudiger. "Melatonin Metabolism in the Central Nervous System." Current Neuropharmacology 8, no. 3 (2010): 168–81. http://dx.doi.org/10.2174/157015910792246164.

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Esposito, Emanuela, and Salvatore Cuzzocrea. "Antiinflammatory Activity of Melatonin in Central Nervous System." Current Neuropharmacology 8, no. 3 (2010): 228–42. http://dx.doi.org/10.2174/157015910792246155.

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Stankov, Bojidar, Franco Fraschini, and Russel J. Reiter. "Melatonin binding sites in the central nervous system." Brain Research Reviews 16, no. 3 (1991): 245–56. http://dx.doi.org/10.1016/0165-0173(91)90008-v.

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Voiculescu, S. E., A. Rosca, C. M. D. Zahiu, C. Badiu, and A. M. Zagrean. "MELATONIN AND MONOAMINERGIC SYSTEM – BEHAVIOURAL ASPECTS." Romanian Journal of Neurology 15, no. 1 (2016): 5–15. http://dx.doi.org/10.37897/rjn.2016.1.1.

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Abstract (sommario):
Melatonin, the hormone synthesized mainly by the pineal gland, is a key member of the complex monoaminergic signaling system, and a circadian regulator with pleiotropic functions. This ubiquitary lipophilic and hydrophilic molecule acts both at cellular and subcellular level, exerting anti-inflammatory, anti-oxidative and anti-apoptotic activities, extremely important in the nervous system, given its high vulnerability to oxidative injury. Melatonin deprivation and the consecutive chronodisruption are associated with multiple behavioural abnormalities, psychiatric disorders and neurodegenerati
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Cardinali, Daniel P., Eleonora S. Pagano, Pablo A. Scacchi Bernasconi, Roxana Reynoso, and Pablo Scacchi. "Melatonin and mitochondrial dysfunction in the central nervous system." Hormones and Behavior 63, no. 2 (2013): 322–30. http://dx.doi.org/10.1016/j.yhbeh.2012.02.020.

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Mahle, Cathy D., and A. John Watson. "Melatonin receptors: potential targets for central nervous system disorders." Expert Opinion on Investigational Drugs 6, no. 4 (1997): 399–406. http://dx.doi.org/10.1517/13543784.6.4.399.

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Lu, Jianan, Yujie Luo, Shuhao Mei, Yuanjian Fang, Jianmin Zhang, and Sheng Chen. "The Effect of Melatonin Modulation of Non-coding RNAs on Central Nervous System Disorders: An Updated Review." Current Neuropharmacology 19, no. 1 (2020): 3–23. http://dx.doi.org/10.2174/1570159x18666200503024700.

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Abstract (sommario):
: Melatonin is a hormone produced in and secreted by the pineal gland. Besides its role in regulating circadian rhythms, melatonin has a wide range of protective functions in the central nervous system (CNS) disorders. The mechanisms underlying this protective function are associated with the regulatory effects of melatonin on related genes and proteins. In addition to messenger ribonucleic acid (RNA) that can be translated into protein, an increasing number of non-coding RNAs in the human body are proven to participate in many diseases. This review discusses the current progress of research o
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Ruddick, Jon P., Andrew K. Evans, David J. Nutt, Stafford L. Lightman, Graham A. W. Rook, and Christopher A. Lowry. "Tryptophan metabolism in the central nervous system: medical implications." Expert Reviews in Molecular Medicine 8, no. 20 (2006): 1–27. http://dx.doi.org/10.1017/s1462399406000068.

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Abstract (sommario):
The metabolism of the amino acid l-tryptophan is a highly regulated physiological process leading to the generation of several neuroactive compounds within the central nervous system. These include the aminergic neurotransmitter serotonin (5-hydroxytryptamine, 5-HT), products of the kynurenine pathway of tryptophan metabolism (including 3-hydroxykynurenine, 3-hydroxyanthranilic acid, quinolinic acid and kynurenic acid), the neurohormone melatonin, several neuroactive kynuramine metabolites of melatonin, and the trace amine tryptamine. The integral role of central serotonergic systems in the mo
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Sadanandan, Nadia, Blaise Cozene, Justin Cho, et al. "Melatonin—A Potent Therapeutic for Stroke and Stroke-Related Dementia." Antioxidants 9, no. 8 (2020): 672. http://dx.doi.org/10.3390/antiox9080672.

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Abstract (sommario):
Secreted by the pineal gland to regulate the circadian rhythm, melatonin is a powerful antioxidant that has been used to combat oxidative stress in the central nervous system. Melatonin-based therapies have been shown to provide neuroprotective effects in the setting of ischemic stroke by mitigating neuroinflammation and accelerating brain tissue restoration. Melatonin treatment includes injection of exogenous melatonin, pineal gland grafting and melatonin-mediated stem cell therapy. This review will discuss the current preclinical and clinical studies investigating melatonin-based therapeutic
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Reiter, Russel J. "Oxidative damage in the central nervous system: protection by melatonin." Progress in Neurobiology 56, no. 3 (1998): 359–84. http://dx.doi.org/10.1016/s0301-0082(98)00052-5.

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Tesi sul tema "Melatonin. Nervous system"

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Maharaj, Deepa Sukhdev. "An investigation into the physico-chemical and neuroprotective properties of melatonin and 6-hydroxymelatonin." Thesis, Rhodes University, 2003. http://eprints.ru.ac.za/71/.

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Parmar, Paresh H. "An investigation into the possible neuroprotective role of melatonin in copper-loading." Thesis, Rhodes University, 2001. http://hdl.handle.net/10962/d1003261.

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Abstract (sommario):
Copper is an extremely toxic metal in biological systems and thus, its availability to the system, must be effectively and efficiently controlled. Copper is vital for life, as it is essential for critical enzymes in biological systems. It is free copper in the biological systems that is toxic, as free copper induces free radical generation, which disrupts lipid membranes, interacts with DNA causing mutations, and eventually leads to cell death. Wilson’s disease is a inherited copper disease, which results in hepatolenticular disease. Copper is unable to be excreted, and thus accumulates, event
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Sjöblom, Markus. "The duodenal mucosal bicarbonate secretion : role of melatonin in neurohumoral control and cellular signaling /." Uppsala : Acta Universitatis Upsaliensis : Univ.-bibl. {[distributör], 2003. http://urn.kb.se/resolve?urn=urn:nbn:se:uu:diva-3521.

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Kadanthode, Rubina John. "An investigation into the neuroprotective effects of melatonin in a model of rotenone-induced neurodegeneration." Thesis, Rhodes University, 2004. http://hdl.handle.net/10962/d1003241.

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Abstract (sommario):
Parkinson’s disease, one of the most common neurodegenerative disorders associated with ageing, is characterised by abnormal and profound loss of nigrostriatal dopaminergic neurons. The cause of Parkinson’s disease is unknown, but epidemiological studies suggest an association with pesticides and other environmental toxins, and biochemical studies implicate oxidative damage and mitochondrial impairment, particularly at the level of complex I enzyme. Recently, rotenone, a commonly used organic pesticide and a classical inhibitor of mitochondrial complex I has been reported to reproduce the spec
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Perreau, Stéphanie Marie. "Control of the daily melatonin rhythm a model of time distribution by the biological clock mediated through the autonomic nervous system /." [S.l. : Amsterdam : s.n.] ; Universiteit van Amsterdam [Host], 2004. http://dare.uva.nl/document/73034.

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Burton, Susan Frances. "A study of the effects of the pineal hormone, melatonin, on dopaminergic transmission in the central nervous system of rats." Thesis, Rhodes University, 1990. http://hdl.handle.net/10962/d1001463.

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Abstract (sommario):
Dopamine mechanisms in the central nervous system are important in the control of both normal and abnormal motor function. The recent observations in both animal and human studies, that melatonin, the principal hormone of the pineal gland, may have a role in the control of movement and the pathophysiology of movement disorders, have given rise to the concept that melatonin may have a modulatory influence on central dopaminergic neurotransmission. This study makes use of three animal behavioural models as well as a biochemical model of central dopaminergic function to further investigate the co
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Lenz, Stéphanie. "Control of the daily melatonin rhythm : A model of time distribution by the biological clock mediated through the autonomic nervous system." Université Louis Pasteur (Strasbourg) (1971-2008), 2004. https://publication-theses.unistra.fr/public/theses_doctorat/2004/LENZ_Stephanie_2004.pdf.

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Abstract (sommario):
Chez les Mammifères, les rythmes circadiens, présentant une rythmicité endogène proche de 24h sont sous le contrôle d'une horloge interne située dans les Noyaux Suprachiasmatiques de l'hypothalamus (NSC). Afin de mieux comprendre la distribution du message temps par les NSC, nous avons étudié spécifiquement le contrôle du rythme journalier de sécrétion de mélatonine, hormone sécrétée de nuit par la glande pinéale, partant du postulat selon lequel les NSC contrôleraient ce rythme en utilisant, pendant la journée, un signal inhibiteur de nature GABAergique pour inhiber la voie polysynaptique rel
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Sommansson, Anna. "Regulation of Duodenal Mucosal Barrier Function and Motility : The Impact of Melatonin." Doctoral thesis, Uppsala universitet, Fysiologi, 2013. http://urn.kb.se/resolve?urn=urn:nbn:se:uu:diva-209669.

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Abstract (sommario):
The duodenal mucosa is regularly exposed to acid, digestive enzymes and ingested noxious agents. It is thus critical to maintain a protective barrier to prevent the development of mucosal injury and inflammation, which are often observed in situations when barrier function is impaired. The rate of mucosal bicarbonate secretion, the regulation of epithelial paracellular permeability and motility are each key components of duodenal barrier function. The hormone melatonin is present in high levels in the gastrointestinal tract and it has been hypothesized that melatonin exerts protective properti
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Lack, Barbara Anne. "Metal interactions with neural substrates and their role in neurodegeneration." Thesis, Rhodes University, 2003. http://hdl.handle.net/10962/d1005709.

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Abstract (sommario):
"Life" may be characterized as a controlled stationary flow equilibrium, maintained by energy consuming chemical reactions. The physiological functioning of these life systems include at least 28 of the elements isolated on the periodic table thus far, most of which are metals. However, as with Paracelsus Principle: "The dose makes the poison", there exists a definite link between metal levels, essential and toxic, and the onset of neurodegenerative diseases. The economic costs of brain dysfunction are enormous, but this pales in comparison to the staggering emotional toll on the victims thems
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Sjöblom, Markus. "The Duodenal Mucosal Bicarbonate Secretion : Role of Melatonin in Neurohumoral Control and Cellular Signaling." Doctoral thesis, Uppsala University, Physiology, 2003. http://urn.kb.se/resolve?urn=urn:nbn:se:uu:diva-3521.

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Abstract (sommario):
<p>The duodenal lumen is exposed to aggressive factors with a high potential to cause damage to the mucosa. Bicarbonate secretion by the duodenal mucosa is accepted as the primary important defense mechanism against the hydrochloric acid intermittently expelled from the stomach.</p><p>The present thesis concerns the influence of the central nervous system and the effects of the hormone melatonin on bicarbonate secretion in anesthetized rats in vivo. Effects of melatonin on intracellular calcium signaling by duodenal enterocyte in vitro were examined in tissues of both human and rat origin. The
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Libri sul tema "Melatonin. Nervous system"

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Switzerland) International Congress Isnim 1999 (Lugano. Neuroimmunomodulation: Perspectives at the New Milennium (Annals of the New York Academy of Sciences). New York Academy of Sciences, 2000.

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Capitoli di libri sul tema "Melatonin. Nervous system"

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Dubocovich, M. L. "Melatonin Receptors in the Central Nervous System." In Advances in Experimental Medicine and Biology. Springer New York, 1991. http://dx.doi.org/10.1007/978-1-4684-5952-4_23.

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Smith, J. A. "Biochemical and Behavioural Studies of Melatonin." In Circadian Rhythms in the Central Nervous System. Palgrave Macmillan UK, 1985. http://dx.doi.org/10.1007/978-1-349-07837-0_1.

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Arendt, Josephine. "The Pineal Hormone Melatonin in Seasonal and Circadian Rhythms." In Circadian Rhythms in the Central Nervous System. Palgrave Macmillan UK, 1985. http://dx.doi.org/10.1007/978-1-349-07837-0_2.

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Checkley, S. A., C. Thompson, C. Franey, and J. Arendt. "Effects of Desipramine on Melatonin and Cortisol in Normal and Depressed Subjects." In Circadian Rhythms in the Central Nervous System. Palgrave Macmillan UK, 1985. http://dx.doi.org/10.1007/978-1-349-07837-0_27.

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Dubocovich, M. L. "Pharmacology and Function of Melatonin Receptors in the Mammalian Central Nervous System." In Serotonin. Palgrave Macmillan UK, 1989. http://dx.doi.org/10.1007/978-1-349-10114-6_32.

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Pang, S. F., H. Yuan, Z. H. Yu, et al. "Melatonin Binding Sites in the Nervous and Immune Systems." In Role of Melatonin and Pineal Peptides in Neuroimmunomodulation. Springer US, 1991. http://dx.doi.org/10.1007/978-1-4615-3756-4_11.

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Anderson, George, and Michael Maes. "Alpha 7 Nicotinic Receptor Agonist Modulatory Interactions with Melatonin: Relevance not only to Cognition, but to Wider Neuropsychiatric and Immune Inflammatory Disorders." In Frontiers in Clinical Drug Research- Central Nervous System. BENTHAM SCIENCE PUBLISHERS, 2016. http://dx.doi.org/10.2174/9781681081892116020006.

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FRASCHINI, FRANCO, and BOJIDAR STANKOV. "Distribution of the Melatonin Receptor in the Central Nervous System of Vertebrates. Kinetic Parameters and Signal Transduction Pathways." In Light and Biological Rhythms in Man. Elsevier, 1993. http://dx.doi.org/10.1016/b978-0-08-042279-4.50012-1.

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Kitzen, Jan M. "Use of Benzodiazepines and Z-Drugs in the Geriatric Population." In The Benzodiazepines Crisis. Oxford University Press, 2020. http://dx.doi.org/10.1093/med/9780197517277.003.0004.

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Abstract (sommario):
The benzodiazepine (BZD) class of drugs has proven to be a useful addition to therapeutic management of anxiety and sleep disorders in the adult population. However, after many years of experience with BZDs in elderly patients (≥65 years), a large body of evidence indicates that BZDs are no longer recommended for use in this segment of the population, except under special conditions. Several aging-related changes in physiology such as decreases in renal and hepatic function, altered central nervous system function and changes in body composition can lead to impaired excretion of drug, higher plasma levels and accumulation of these drugs in the body. Side effects such as sedation, dizziness, cognitive impairment, and diminished control of gait and balance functions place the elderly at greater risk of various adverse events, especially falls and fractures, compared to younger adults. Another class of drugs, known as Z-drugs is structurally dissimilar from the BZDs but able to bind to GABA<sub>A</sub>, receptors making them useful for management of insomnia. These drugs have also been found to pose significant hazards to the elderly and are also not recommended for use in elderly patients. Both of these classes of drugs are currently included in the AGS Beers Criteria<sup>®</sup> of potentially inappropriate medications for use in the elderly. Alternative pharmacologic approaches to managing these conditions include use of either serotonin-norepinephrine reuptake inhibitors or buspirone for anxiety and the use of either low doses of doxepin, melatonin, or the melatonin agonist ramelteon for management of insomnia. Cognitive behavioral therapy and other relaxation techniques offer non-pharmacologic approaches to managing these conditions, thereby decreasing the need for prescribing BZDs or Z-drugs in the elderly.
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Yurt, Kıymet Kübra, Elfide Gizem Kıvrak, Gamze Altun, Abit Aktas, Arife Ahsen Kaplan, and Süleyman Kaplan. "Neuroprotective Effects of Melatonin and Omega-3 on the Central Nervous System Exposed to Electromagnetic Fields in the Pre- and Postnatal Periods." In Omega Fatty Acids in Brain and Neurological Health. Elsevier, 2019. http://dx.doi.org/10.1016/b978-0-12-815238-6.00011-0.

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