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Letteratura scientifica selezionata sul tema "Myeloid leukemia"

Autore: Grafiati

Pubblicato: 4 giugno 2021

Ultima modifica: 25 luglio 2025

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Articoli di riviste sul tema "Myeloid leukemia"

1

Swatler, Julian, Laura Turos-Korgul, Ewa Kozlowska, and Katarzyna Piwocka. "Immunosuppressive Cell Subsets and Factors in Myeloid Leukemias." Cancers 13, no. 6 (2021): 1203. http://dx.doi.org/10.3390/cancers13061203.

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Both chronic myeloid leukemia and acute myeloid leukemia evade the immune response during their development and disease progression. As myeloid leukemia cells modify their bone marrow microenvironment, they lead to dysfunction of cytotoxic cells, such as CD8+ T cells or NK cells, simultaneously promoting development of immunosuppressive regulatory T cells and suppressive myeloid cells. This facilitates disease progression, spreading of leukemic blasts outside the bone marrow niche and therapy resistance. The following review focuses on main immunosuppressive features of myeloid leukemias. Firs

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2

Namikawa, R., R. Ueda, and S. Kyoizumi. "Growth of human myeloid leukemias in the human marrow environment of SCID-hu mice." Blood 82, no. 8 (1993): 2526–36. http://dx.doi.org/10.1182/blood.v82.8.2526.2526.

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Abstract It has been shown previously that multilineage human hematopoiesis is maintained within human fetal bone marrow (BM) fragments implanted into severe combined immunodeficient (SCID) mice. We describe here an application of this animal model, the SCID-hu mouse, to the study of human myeloid leukemias. BM cells from 8 patients with various types of myeloid leukemias were injected directly into human bone grafts in the SCID-hu mouse. Cells from 7 patients grew in the human marrow without spreading to the mouse marrow. Cells from 6 of these patients were successfully transferred in vivo to

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3

Namikawa, R., R. Ueda, and S. Kyoizumi. "Growth of human myeloid leukemias in the human marrow environment of SCID-hu mice." Blood 82, no. 8 (1993): 2526–36. http://dx.doi.org/10.1182/blood.v82.8.2526.bloodjournal8282526.

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It has been shown previously that multilineage human hematopoiesis is maintained within human fetal bone marrow (BM) fragments implanted into severe combined immunodeficient (SCID) mice. We describe here an application of this animal model, the SCID-hu mouse, to the study of human myeloid leukemias. BM cells from 8 patients with various types of myeloid leukemias were injected directly into human bone grafts in the SCID-hu mouse. Cells from 7 patients grew in the human marrow without spreading to the mouse marrow. Cells from 6 of these patients were successfully transferred in vivo to secondar

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4

Aue, Georg, Yang Du, Susan M. Cleveland, et al. "Sox4 cooperates with PU.1 haploinsufficiency in murine myeloid leukemia." Blood 118, no. 17 (2011): 4674–81. http://dx.doi.org/10.1182/blood-2011-04-351528.

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Abstract Cooperation of multiple mutations is thought to be required for cancer development. In previous studies, murine myeloid leukemias induced by transducing wild-type bone marrow progenitors with a SRY sex determining region Y-box 4 (Sox4)–expressing retrovirus frequently carried proviral insertions at Sfpi1, decreasing its mRNA levels, suggesting that reduced Sfpi1 expression cooperates with Sox4 in myeloid leukemia induction. In support of this hypothesis, we show here that mice receiving Sox4 virus-infected Sfpi1ko/+ bone marrow progenitors developed myeloid leukemia with increased pen

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5

Shvachko, L. P. "EMT-mechanizm induces the leukemic stemness phenotype in myeloid leukemias." Faktori eksperimental'noi evolucii organizmiv 23 (September 9, 2018): 256–60. http://dx.doi.org/10.7124/feeo.v23.1024.

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Aim. To study the targeted expression EMT-induced markers N-cadherin, Snail and Twist in patients with the chronic and acute myeloid leukemias. Methods. RT-PCR, electroforesic in agarose gel, TotalLab v. 2.01 densitometry. Results. Have been investigated the relative levels of mRNA expression of N-cadherin and transcriptional factors Snail and Twist, associated with epithelial-to-mesenchymal induction (EMT) in patients with the essential polycytemia (EP), the chronic mieloid leukemia CML), the acute myeloid leukemia (AML) and the acute lymphoblastic leukemia (ALL). Conclusions. Have been highl

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6

Jamieson, Catriona, Sidd Jaiswal, David Traver, Jason Gotlib, Mark Chao, and Irving L. Weissman. "Increased Expression of CD47 Is a Constant Marker in Mouse and Human Myeloid Leukemias." Blood 106, no. 11 (2005): 3260. http://dx.doi.org/10.1182/blood.v106.11.3260.3260.

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Abstract CD47, also known as integrin associated protein, is a ubiquitously expressed cell surface glycoprotein that interacts with a number of integrins, modulating leukocyte adhesion, migration, cell motility and platelet activation. CD47 is also the ligand for the macrophage inhibitory receptor signal regulatory protein α (SIRP α) and thus, impairs macrophage-mediated phagocytosis. Recent reports suggest that increased CD47 expression may play a role in the pathogenesis of lymphoproliferative disorders such as CLL (Mateo et al, Nat Med.1999; 5:1277) and multiple myeloma, and that a bivalent

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7

Войцеховский, Валерий, Valeriy Voytsekhovskiy, Татьяна Заболотских, et al. "DAMAGE OF THE BRONCHOPULMONARY SYSTEM IN PATIENTS WITH CHRONIC HEMOBLASTOSIS." Bulletin physiology and pathology of respiration 1, no. 69 (2018): 25–35. http://dx.doi.org/10.12737/article_5b975083a62278.59044240.

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In 185 patients with chronic hemoblastosis (chronic lymphocytic leukemia, chronic myeloid leukemia, idiopathic myelofibrosis, multiple myeloma) after autopsy, the pathology of the bronchopulmonary system was studied. It was found out that in addition to immunodeficiency, an important role in the occurrence of respiratory diseases in chronic lymphocytic leukemia, as well as in chronic myeloid leukemia and idiopathic myelofibrosis in the stage of blast crisis is played by specific leukemic infiltration of the lungs, bronchi, pleura and diaphragm; the presence of leukostasis in the vessels of med

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8

Bento, Marta Leal, Luís Carvalho, Zhewei Chen, Ana Coelho, Cong Tang, and Gonçalo Bernardes. "Acute Myeloid and Lymphoblastic Leukemias: A NPM1 Targeting Strategy." Blood 142, Supplement 1 (2023): 7147. http://dx.doi.org/10.1182/blood-2023-172497.

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Leukemia, a wide spectrum of diseases with altered proliferation and differentiation capacity of myeloid and lymphoid blood progenitors, is the most frequent type of cancer in children and one of the most common in adults. We discovered a covalent small-molecule “probe” for the treatment of acute myeloid and lymphoblastic leukemias that allows for post-translational modulation of the proteome in these leukemia cells. The probe dramatically induces apoptosis of leukemic cells at sub-toxic doses and consistently reduces proliferation, impairs cellular metabolism and promotes chemosensitization t

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Longo, Giuseppe S. A., Richard Gorlick, William P. Tong, Emine Ercikan, and Joseph R. Bertino. "Disparate Affinities of Antifolates for Folylpolyglutamate Synthetase From Human Leukemia Cells." Blood 90, no. 3 (1997): 1241–45. http://dx.doi.org/10.1182/blood.v90.3.1241.

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Abstract Previous work showed that acute myelocytic leukemia blasts accumulate less long chain polyglutamates of methotrexate (MTX) than acute lymphocytic leukemia blasts when incubated with this radiolabeled antifolate. This difference likely explains the increased sensitivity of lymphoid leukemias to short-term exposure of MTX as compared with myeloid leukemias. In this study, we examined the basis for differences between long chain MTX polyglutamate accumulation between different leukemia cell types using both leukemia cell lines and blasts freshly isolated from blood of leukemic patients.

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Longo, Giuseppe S. A., Richard Gorlick, William P. Tong, Emine Ercikan, and Joseph R. Bertino. "Disparate Affinities of Antifolates for Folylpolyglutamate Synthetase From Human Leukemia Cells." Blood 90, no. 3 (1997): 1241–45. http://dx.doi.org/10.1182/blood.v90.3.1241.1241_1241_1245.

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Previous work showed that acute myelocytic leukemia blasts accumulate less long chain polyglutamates of methotrexate (MTX) than acute lymphocytic leukemia blasts when incubated with this radiolabeled antifolate. This difference likely explains the increased sensitivity of lymphoid leukemias to short-term exposure of MTX as compared with myeloid leukemias. In this study, we examined the basis for differences between long chain MTX polyglutamate accumulation between different leukemia cell types using both leukemia cell lines and blasts freshly isolated from blood of leukemic patients. The major

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Tesi sul tema "Myeloid leukemia"

1

Cheung, Man-sze, and 張敏思. "Characterization of Leukemic stem cells in acute myeloid Leukemia." Thesis, The University of Hong Kong (Pokfulam, Hong Kong), 2008. http://hub.hku.hk/bib/B40687582.

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Cheung, Man-sze. "Characterization of Leukemic stem cells in acute myeloid Leukemia." Click to view the E-thesis via HKUTO, 2008. http://sunzi.lib.hku.hk/hkuto/record/B40687582.

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3

Yaseen, Mumtaz. "Proteomics of Acute Myeloid Leukemia:." Diss., lmu, 2007. http://nbn-resolving.de/urn:nbn:de:bvb:19-69882.

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4

Gunnarsson, Niklas. "Chronic myeloid leukemia and cancer." Doctoral thesis, Umeå universitet, Medicin, 2017. http://urn.kb.se/resolve?urn=urn:nbn:se:umu:diva-141144.

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Abstract (sommario):

Background Chronic myeloid leukemia (CML) is a relatively rare hematological malignancy with a constant incidence of approximately 90 new cases each year in Sweden (0.9 cases/100 000 inhabitants). The etiology is largely unknown but high doses of ionizing radiation are a known but rare risk factor. The treatment options were for a long time limited to chemotherapies i.e. hydroxyurea and busulfan, interferon’s and allogeneic hematopoietic stem cell transplantation and the median survival were only about four years. Since the beginning of the 21st century a new way of treating CML has been intr

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5

VARINELLI, MARCO. "MODELLING CHRONIC MYELOID LEUKEMIA IN ZEBRAFISH." Doctoral thesis, Università degli studi di Brescia, 2021. http://hdl.handle.net/11379/544088.

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6

Cornforth, Terri Victoria. "Characterising the cell biology of leukemic stem cells in acute myeloid leukemia." Thesis, University of Oxford, 2013. http://ora.ox.ac.uk/objects/uuid:654b2176-fd50-427e-86f2-74e928054bef.

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Acute Myeloid leukemia (AML) is an aggressive haematological malignancy that mainly affects the elderly. Relapse is common and is thought to be due to the presence of chemotherapy resistant leukemic stem cells (LSC). Within the CD34+ disease (>5% of the blast cells expressing CD34) , two subtypes have been identified; an LMPP/GMPlike expanded type and a MPP/CMP-like expanded type, the former is the most common, accounting for around 80% of CD34+ AML. Both the GMP-like and LMPPlike expanded populations show LSC activity. To improve our understanding of the disease and gain better insight in to

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7

Zhang, Lu [Verfasser]. "Immunogenicity of leukemia stem cells in acute myeloid leukemia / Lu Zhang." Ulm : Universität Ulm. Medizinische Fakultät, 2012. http://d-nb.info/1020022574/34.

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García, Montolío Marc 1991. "The Role of PHF19 in myeloid leukemia." Doctoral thesis, Universitat Pompeu Fabra, 2019. http://hdl.handle.net/10803/667911.

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Polycomb group (PcG) of proteins are a group of highly conserved epigenetic regulators involved in many biological functions such as embryonic development, stem cell self-renewal, cell proliferation, and cancer. PHD finger protein 19 (PHF19) is an associated factor of Polycomb Repressor Complex 2 (PRC2) that has been proposed to regulated its activity in embryonic stem cells. PHF19 has been shown to be up-regulated in different human cancers as well as cancer cell lines. In particular, myeloid leukemia cell lines show increased levels of PHF19, yet little is known about its function. Here, we

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9

Palle, Josefine. "Optimizing Chemotherapy in Childhood Acute Myeloid Leukemia." Doctoral thesis, Uppsala University, Department of Women's and Children's Health, 2008. http://urn.kb.se/resolve?urn=urn:nbn:se:uu:diva-9189.

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<p>Despite major advances in our understanding of the biology of childhood acute myeloid leukemia (AML) and the development of new cytotoxic drugs, the prognosis of long-term survival is still only 60-65 %.</p><p>In the present research, we studied the pharmacokinetics of drugs used in the induction therapy of childhood AML and performed in vitro drug sensitivity testing of leukemic cells from children with AML.</p><p>The aims of the studies were to correlate the results of the analysis to biological and clinical parameters and to identify subgroups of AML with specific drug sensitivity profil

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10

Watson, Alexander Scarth. "Autophagy in hematopoiesis and acute myeloid leukemia." Thesis, University of Oxford, 2014. http://ora.ox.ac.uk/objects/uuid:2e66c5c3-4774-44d1-8345-d0dc827da16d.

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Acute myeloid leukemia (AML) develops following oncogenic alterations to hematopoietic stem (HSC) and progenitor cells (HSPCs) in the bone marrow, resulting in dysregulated proliferation of immature myeloid progenitors that interferes with normal hematopoiesis. Understanding the mechanisms of HSPC protection against damage and excessive division, and how these pathways are altered during leukemic progression, is vital for establishing effective therapies. Here, we show that autophagy, a lysosomal degradation pathway, is increased in HSPCs using a novel imaging flow cytometry autophagy assay. L

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Più fonti

Libri sul tema "Myeloid leukemia"

1

Harry, Iland, Hertzberg Mark, and Marlton Paula. Myeloid Leukemia. Humana Press, 2005. http://dx.doi.org/10.1385/1597450170.

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2

Röllig, Christoph, and Gert J. Ossenkoppele, eds. Acute Myeloid Leukemia. Springer International Publishing, 2021. http://dx.doi.org/10.1007/978-3-030-72676-8.

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Hehlmann, Rüdiger, ed. Chronic Myeloid Leukemia. Springer International Publishing, 2021. http://dx.doi.org/10.1007/978-3-030-71913-5.

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4

Li, Shaoguang, and Haojian Zhang, eds. Chronic Myeloid Leukemia. Springer New York, 2016. http://dx.doi.org/10.1007/978-1-4939-4011-0.

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Hehlmann, Rüdiger, ed. Chronic Myeloid Leukemia. Springer International Publishing, 2016. http://dx.doi.org/10.1007/978-3-319-33198-0.

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6

Fortina, Paolo, Eric Londin, Jason Y. Park, and Larry J. Kricka, eds. Acute Myeloid Leukemia. Springer New York, 2017. http://dx.doi.org/10.1007/978-1-4939-7142-8.

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7

E, Cortés F. Jorge, and Deininger Michael, eds. Chronic myeloid leukemia. Informa Healthcare, 2007.

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8

Hughes, Timothy P., David M. Ross, and Junia V. Melo. Handbook of Chronic Myeloid Leukemia. Springer International Publishing, 2014. http://dx.doi.org/10.1007/978-3-319-08350-6.

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9

Harry, Iland, Hertzberg Mark, and Marlton Paula, eds. Myeloid leukemia: Methods and protocols. Humana Press, 2006.

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10

Saglio, Giuseppe, and Carmen Fava. The Treatment of Chronic Myeloid Leukemia. Future Medicine Ltd, 2013. http://dx.doi.org/10.2217/9781780842738.

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Capitoli di libri sul tema "Myeloid leukemia"

1

Voso, Maria Teresa, Eleonora De Bellis, and Tiziana Ottone. "Diagnosis and Classification of AML: WHO 2016." In Acute Myeloid Leukemia. Springer International Publishing, 2021. http://dx.doi.org/10.1007/978-3-030-72676-8_2.

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2

Kayser, Sabine, and Uwe Platzbecker. "Management of Acute Promyelocytic Leukemia." In Acute Myeloid Leukemia. Springer International Publishing, 2021. http://dx.doi.org/10.1007/978-3-030-72676-8_8.

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3

Jaramillo, Sonia, and Richard F. Schlenk. "Treatment of Relapsed and Refractory AML: Intensive Approach in Fit Patients." In Acute Myeloid Leukemia. Springer International Publishing, 2021. http://dx.doi.org/10.1007/978-3-030-72676-8_11.

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Bornhäuser, Martin. "Allogeneic Hematopoietic Cell Transplantation." In Acute Myeloid Leukemia. Springer International Publishing, 2021. http://dx.doi.org/10.1007/978-3-030-72676-8_13.

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5

Itzykson, Raphael, Marco Cerrano, and Jordi Esteve. "Prognostic Factors in AML." In Acute Myeloid Leukemia. Springer International Publishing, 2021. http://dx.doi.org/10.1007/978-3-030-72676-8_7.

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6

Wierzbowska, Agnieszka, and Magdalena Czemerska. "Clinical Manifestation and Diagnostic Workup." In Acute Myeloid Leukemia. Springer International Publishing, 2021. http://dx.doi.org/10.1007/978-3-030-72676-8_6.

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7

Sprute, Rosanne, and Oliver A. Cornely. "Special Clinical Scenarios: Infectious Complications and Prophylaxis." In Acute Myeloid Leukemia. Springer International Publishing, 2021. http://dx.doi.org/10.1007/978-3-030-72676-8_16.

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8

Venditti, Adriano, Peter J. M. Valk, Nigel H. Russell, and Sylvie D. Freeman. "Future Developments: Measurable Residual Disease." In Acute Myeloid Leukemia. Springer International Publishing, 2021. http://dx.doi.org/10.1007/978-3-030-72676-8_18.

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Bug, Gesine, and Halvard Bonig. "Special Clinical Scenarios: Hyperleukocytosis." In Acute Myeloid Leukemia. Springer International Publishing, 2021. http://dx.doi.org/10.1007/978-3-030-72676-8_14.

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Estey, Elihu. "Future Developments: Innovative Trial Design." In Acute Myeloid Leukemia. Springer International Publishing, 2021. http://dx.doi.org/10.1007/978-3-030-72676-8_20.

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Atti di convegni sul tema "Myeloid leukemia"

1

Ahmad, Abdul Rahim, Nuramirah Aiman Zainudin, Muhammad Dinul Ikram Mohd Radzi, Nurul Hazwani Abd Halim, Muhammad Khusairi Osman, and Zuraidi Saad. "CNN-Based Classification of Acute Myeloid Leukemia Blood Samples." In 2024 IEEE 14th International Conference on Control System, Computing and Engineering (ICCSCE). IEEE, 2024. http://dx.doi.org/10.1109/iccsce61582.2024.10696464.

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2

Aby, Aswathy Elma, Salaji S, Anilkumar K K, and Tintu Rajan. "Optimizing Acute Myeloid Leukemia Subtype Classification Through Data Balancing Techniques." In 2025 2nd International Conference on Trends in Engineering Systems and Technologies (ICTEST). IEEE, 2025. https://doi.org/10.1109/ictest64710.2025.11042354.

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3

Chen, Taiyang, Shenghan Hou, Hairuo Wang, and Yifeng Peng. "Machine Learning Based Single-Cell Analysis of Acute Myeloid Leukemia Data." In 2024 4th International Conference on Computer Science and Blockchain (CCSB). IEEE, 2024. http://dx.doi.org/10.1109/ccsb63463.2024.10735469.

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4

DeMarco, B., M. O. Al-Qadi, S. S. Carson, and S. Ghosh. "Leukemic Pleural Effusion in Acute Myeloid Leukemia." In American Thoracic Society 2020 International Conference, May 15-20, 2020 - Philadelphia, PA. American Thoracic Society, 2020. http://dx.doi.org/10.1164/ajrccm-conference.2020.201.1_meetingabstracts.a4863.

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Murray, Kelsey M., and Kishan Patel. "A Rare Presentation: Intracranial Hemorrhage as a Symptom of Acute Leukemic Transformation in a 23-year old Male." In 28th Annual Rowan-Virtua Research Day. Rowan University Libraries, 2024. http://dx.doi.org/10.31986/issn.2689-0690_rdw.stratford_research_day.66_2024.

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This case highlights the urgency of considering acute leukemic transformation in young patients presenting with neurological deficits, emphasizing the importance of prompt evaluation and management to optimize patient outcomes. The case depicted is a tragic complication of Chronic Myeloid Leukemia (CML) and its acute blast crisis. Remarkably, the patient exhibited none of the typical constitutional symptoms associated with CML.

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Rosenbluth, Michael J., Wilbur A. Lam, and Daniel A. Fletcher. "Contribution of Cell Mechanics to Acute Leukemia." In ASME 2004 International Mechanical Engineering Congress and Exposition. ASMEDC, 2004. http://dx.doi.org/10.1115/imece2004-59881.

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Leukostasis is a life-threatening condition that occurs when leukemia cells accumulate in the vasculature of organs such as the brain and lungs. Recent evidence has shown that leukostasis is not simply due to the physical overcrowding of leukemia cells, as previously thought, but may result from specific mechanical properties of the cells and interactions between cells. Using atomic force microscopy (AFM), we obtained direct measurements of two mechanical properties that are likely involved in this condition: (1) stiffness of individual leukemia cells and (2) non-specific adhesion forces betwe

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Kulkarni, Shrinidhi. "CRISPR Solution to Cure Acute Myeloid Leukemia." In 8th North American Conference on Industrial Engineering and Operations Management. IEOM Society International, 2023. http://dx.doi.org/10.46254/na8.20230275.

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Sultonova, Sherozakhon. "CLINICAL-HEMATOLOGICAL FEATURES OF CHRONIC MYELOID LEUKEMIA." In RICERCHE SCIENTIFICHE E METODI DELLA LORO REALIZZAZIONE: ESPERIENZA MONDIALE E REALTÀ DOMESTICHE, chair Din Mohammad, Khamid Karimov, Kodirjon Boboyev, and Khamida Kazakbayeva. European Scientific Platform, 2021. http://dx.doi.org/10.36074/logos-26.11.2021.v3.22.

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Ahmed Mustafa, Srwa, and Gullanar M Hadi. "Automated Leukemia Detection using K-means Clustering for Feature Extraction." In 5TH INTERNATIONAL CONFERENCE ON COMMUNICATION ENGINEERING AND COMPUTER SCIENCE (CIC-COCOS'24). Cihan University-Erbil, 2024. http://dx.doi.org/10.24086/cocos2024/paper.1529.

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Abstract (sommario):

Leukemia is a kind of blood cancer that may cause significant damage to a person's general health. It is characterized by the production of an excessive number of white blood cells. To address leukemia quickly and effectively, it is important to have a diagnosis that is both correct and quick. Not too long ago, experts started using AI methods to help find cancer much earlier. One of the hardest parts of making a method to find leukemia is separating the nuclei from the rest of the picture. When medical staff use quick and accurate division methods, they can find patients faster and treat them

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Verhagen, Han, Marjon Smit, David de Leeuw, et al. "Abstract 2339: IGFBP7 eradicates leukemic stem and progenitor cells in acute myeloid leukemia." In Proceedings: AACR 106th Annual Meeting 2015; April 18-22, 2015; Philadelphia, PA. American Association for Cancer Research, 2015. http://dx.doi.org/10.1158/1538-7445.am2015-2339.

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Rapporti di organizzazioni sul tema "Myeloid leukemia"

1

Jangid, Ajay, Anurag Mishra, Rachit Raj, Sumit Kumar, Priyanka Munjal, and Neha Pandey. Chronic Myeloid Leukemia (CML) as Surgical Emergency. Science Repository, 2024. http://dx.doi.org/10.31487/j.ajscr.2024.01.02.

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Ileal perforation peritonitis is a critical surgical emergency often encountered in developing countries, commonly associated with typhoid fever, tuberculosis, trauma, and non-specific enteritis. This case report presents a unique instance of nonspecific enteritis associated with chronic myeloid leukemia (CML). A 16-year-old girl with a history of pulmonary tuberculosis presented with symptoms, leading to the diagnosis of ileal perforations and CML. Surgical intervention involved ileal resection and double barrel ileostomy. The postoperative course included complications and chemotherapy with

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2

Muller-Sieburg, Christa. Myeloid-Biased Stem Cells as Potential Targets for Chronic Myelogeneous Leukemia. Defense Technical Information Center, 2005. http://dx.doi.org/10.21236/ada447669.

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3

FLORIDA UNIV GAINESVILLE. Dissection of the Pathogenesis of Neurofibromatosis Type 1-Associated myeloid Leukemia. Defense Technical Information Center, 1998. http://dx.doi.org/10.21236/ada359875.

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Brannan, Camilynn I. Dissection of the Pathogenesis of Neurofibromatosis Type 1-Associated Myeloid Leukemia. Defense Technical Information Center, 1999. http://dx.doi.org/10.21236/ada391284.

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Brannan, Camilynn I. Dissection of the Pathogenesis of Neurofibromatosis Type 1-Associated Myeloid Leukemia. Defense Technical Information Center, 2000. http://dx.doi.org/10.21236/ada392474.

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Untaaveesup, Suvijak, Sasinipa Trithiphen, Kamolchanok Kulchutisin, et al. Genetic Alterations in Extramedullary Leukemia among Acute Myeloid Leukemia Patients: Insights from a Cohort Study and Meta-analysis. INPLASY - International Platform of Registered Systematic Review and Meta-analysis Protocols, 2023. http://dx.doi.org/10.37766/inplasy2023.8.0091.

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Zhang, Chengcheng. Dissecting the Role of IGFBP-2 in Development of Acute Myeloid Leukemia. Defense Technical Information Center, 2011. http://dx.doi.org/10.21236/ada555017.

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Zhang, Dong-Er. Protein ISG15 Modification in the Development and the Treatment of Chronic Myeloid Leukemia. Defense Technical Information Center, 2007. http://dx.doi.org/10.21236/ada482354.

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Sorror, Mohamed L., Barry E. Storer, and Elihu H. Estey. Comparing Hematopoietic Cell Transplant versus Other Treatments for Adults with Acute Myeloid Leukemia. Patient-Centered Outcomes Research Institute (PCORI), 2021. http://dx.doi.org/10.25302/01.2021.ce.13047451.

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Tremblay, Michel. Contribution of Protein Tyrosine Phosphateses to the Ontogeny and Progression of Chronic Myeloid Leukemia. Defense Technical Information Center, 2006. http://dx.doi.org/10.21236/ada462811.

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