Bibliografie tematiche / Neutrophils

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Letteratura scientifica selezionata sul tema "Neutrophils"

Autore: Grafiati

Pubblicato: 4 giugno 2021

Ultima modifica: 25 luglio 2025

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Articoli di riviste sul tema "Neutrophils"

Forlow, S. Bradley, Jill R. Schurr, Jay K. Kolls, Gregory J. Bagby, Paul O. Schwarzenberger, and Klaus Ley. "Increased granulopoiesis through interleukin-17 and granulocyte colony-stimulating factor in leukocyte adhesion molecule–deficient mice." Blood 98, no. 12 (2001): 3309–14. http://dx.doi.org/10.1182/blood.v98.12.3309.

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Abstract Many mutant mice deficient in leukocyte adhesion molecules display altered hematopoiesis and neutrophilia. This study investigated whether peripheral blood neutrophil concentrations in these mice are elevated as a result of accumulation of neutrophils in the circulation or altered hematopoiesis mediated by a disrupted regulatory feedback loop. Chimeric mice were generated by transplanting various ratios of CD18+/+ and CD18−/− unfractionated bone marrow cells into lethally irradiated wild-type mice, resulting in approximately 0%, 10%, 50%, 90%, or 100% CD18 null neutrophils in the bloo

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Gong, Henry H., Matthew J. Worley, Kyle A. Carver, Caleb J. Godin, and Jane C. Deng. "Deficient neutrophil responses early in influenza infection promote viral replication and pulmonary inflammation." PLOS Pathogens 21, no. 1 (2025): e1012449. https://doi.org/10.1371/journal.ppat.1012449.

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Neutrophils play key protective roles in influenza infections, yet excessive neutrophilic inflammation is a hallmark of acute lung injury during severe infections. Phenotypic heterogeneity is increasingly recognized in neutrophil populations; however, how functional variation in neutrophils between individuals determine the diverse outcomes of influenza remains unclear. To examine immunologic responses that may drive varying outcomes in influenza, we infected C57BL/6 (B6) and A/J mice with mouse-adapted influenza A virus A/PR/8/34 H1N1. A self-resolving dose in B6 mice was lethal in A/J mice,

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McGovern, Toby K., Michael Chen, Benoit Allard, Kjell Larsson, James G. Martin, and Mikael Adner. "Neutrophilic oxidative stress mediates organic dust-induced pulmonary inflammation and airway hyperresponsiveness." American Journal of Physiology-Lung Cellular and Molecular Physiology 310, no. 2 (2016): L155—L165. http://dx.doi.org/10.1152/ajplung.00172.2015.

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Airway exposure to organic dust (OD) from swine confinement facilities induces airway inflammation dominated by neutrophils and airway hyperresponsiveness (AHR). One important neutrophilic innate defense mechanism is the induction of oxidative stress. Therefore, we hypothesized that neutrophils exacerbate airway dysfunction following OD exposure by increasing oxidant burden. BALB/C mice were given intranasal challenges with OD or PBS (1/day for 3 days). Mice were untreated or treated with a neutrophil-depleting antibody, anti-Ly6G, or the antioxidant dimethylthiourea (DMTU) prior to OD exposur

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Mizgerd, J. P., B. B. Meek, G. J. Kutkoski, D. C. Bullard, A. L. Beaudet, and C. M. Doerschuk. "Selectins and neutrophil traffic: margination and Streptococcus pneumoniae-induced emigration in murine lungs." Journal of Experimental Medicine 184, no. 2 (1996): 639–45. http://dx.doi.org/10.1084/jem.184.2.639.

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The roles of selectins in the pulmonary margination and emigration of neutrophils were investigated by using mice genetically deficient in both E- and P-selectins (E/P mutants) and/or by intravenous injections of fucoidin (inhibiting both L- and P-selectins). E/P mutants were neutrophilic (14.7 +/- 4.9 x 10(6) vs. 0.8 +/- 0.1 x 10(6) neutrophils/ml). This neutrophilia was associated with increased margination of neutrophils within pulmonary capillaries (39.7 +/- 9.4 vs. 4.6 +/- 1.1 neutrophil profiles per 100 red blood cell profiles) but no change in margination within noncapillary pulmonary m

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Yamasaki, Akira, Ryota Okazaki, and Tomoya Harada. "Neutrophils and Asthma." Diagnostics 12, no. 5 (2022): 1175. http://dx.doi.org/10.3390/diagnostics12051175.

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Although eosinophilic inflammation is characteristic of asthma pathogenesis, neutrophilic inflammation is also marked, and eosinophils and neutrophils can coexist in some cases. Based on the proportion of sputum cell differentiation, asthma is classified into eosinophilic asthma, neutrophilic asthma, neutrophilic and eosinophilic asthma, and paucigranulocytic asthma. Classification by bronchoalveolar lavage is also performed. Eosinophilic asthma accounts for most severe asthma cases, but neutrophilic asthma or a mixture of the two types can also present a severe phenotype. Biomarkers for the d

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Weinmann, Pamela, Karin Scharffetter-Kochanek, S. Bradley Forlow, Thorsten Peters, and Barbara Walzog. "A role for apoptosis in the control of neutrophil homeostasis in the circulation: insights from CD18-deficient mice." Blood 101, no. 2 (2003): 739–46. http://dx.doi.org/10.1182/blood-2002-01-0239.

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The control of neutrophil turnover in the circulation is a key event in homeostasis and inflammation. Using CD18- deficient (CD18−/−) mice that show a 19-fold increase of blood neutrophil counts when compared with wild-type animals (CD18+/+), we found that apoptosis of peripheral neutrophils was significantly reduced from 27.4% in the wild-type to 4.8% inCD18−/− mice within 4 hours after isolation as measured by analysis of DNA content. This was confirmed by detecting CD16 expression, nuclear morphology, and internucleosomal DNA degradation. In contrast, no difference in apoptosis was observed

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Borges, Leandro, Tania Cristina Pithon-Curi, Rui Curi, and Elaine Hatanaka. "COVID-19 and Neutrophils: The Relationship between Hyperinflammation and Neutrophil Extracellular Traps." Mediators of Inflammation 2020 (December 2, 2020): 1–7. http://dx.doi.org/10.1155/2020/8829674.

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Coronavirus disease 2019 (COVID-19) is a virus-induced respiratory disease that may progress to acute respiratory distress syndrome (ARDS) and is triggered by immunopathological mechanisms that cause excessive inflammation and leukocyte dysfunction. Neutrophils play a critical function in the clearance of bacteria with specific mechanisms to combat viruses. The aim of this review is to highlight the current advances in the pathways of neutrophilic inflammation against viral infection over the past ten years, focusing on the production of neutrophil extracellular traps (NETs) and its impact on

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Wang, Guoshun, and Hang Pong Ng. "Myeloid CFTR Loss-of-function Causes Persistent Neutrophilic Inflammation in Cystic Fibrosis." Journal of Immunology 202, no. 1_Supplement (2019): 187.33. http://dx.doi.org/10.4049/jimmunol.202.supp.187.33.

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Abstract Persistent neutrophilic inflammation is a hallmark manifestation of cystic fibrosis (CF). However, the mechanism underlying this phenomenal clinical symptom remains incompletely understood. Here we report a pivotal role of CFTR in myeloid immune cells in control of neutrophilic inflammation. Myeloid CFTR-Knockout (Mye-Cftr−/−) mice and Wild-type (WT) mice were challenged peritoneally with zymosan at different doses. The lethal-dose challenge resulted in significantly higher mortality in Mye-Cftr−/− mice, indicating an intrinsic defect in host protection against inflammation in CF. The

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Inauen, W., D. N. Granger, C. J. Meininger, M. E. Schelling, H. J. Granger, and P. R. Kvietys. "Anoxia-reoxygenation-induced, neutrophil-mediated endothelial cell injury: role of elastase." American Journal of Physiology-Heart and Circulatory Physiology 259, no. 3 (1990): H925—H931. http://dx.doi.org/10.1152/ajpheart.1990.259.3.h925.

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The aim of this study was to assess the role of neutrophilic elastase in anoxia-reoxygenation-induced, neutrophil-mediated injury to microvascular endothelium. Cultured bovine microvascular endothelial cells were grown to confluence and labeled with 51Cr. The endothelial cells were exposed to a 30-min period of anoxia and subsequently reoxygenated. Endothelial cell injury, quantitated as 51Cr release and cell detachment, was determined 8 h after reoxygenation. Addition of neutrophils upon reoxygenation enhanced the anoxia-reoxygenation-induced increase in 51Cr release and cell detachment. The

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Tomar, Bhawna, Hans-Joachim Anders, Jyaysi Desai, and Shrikant R. Mulay. "Neutrophils and Neutrophil Extracellular Traps Drive Necroinflammation in COVID-19." Cells 9, no. 6 (2020): 1383. http://dx.doi.org/10.3390/cells9061383.

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The COVID-19 pandemic is progressing worldwide with an alarming death toll. There is an urgent need for novel therapeutic strategies to combat potentially fatal complications. Distinctive clinical features of severe COVID-19 include acute respiratory distress syndrome, neutrophilia, and cytokine storm, along with severe inflammatory response syndrome or sepsis. Here, we propose the putative role of enhanced neutrophil infiltration and the release of neutrophil extracellular traps, complement activation and vascular thrombosis during necroinflammation in COVID-19. Furthermore, we discuss how ne

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Tesi sul tema "Neutrophils"

Gillis, Caitlin. "Neutrophils in IgG- and endotoxin-induced systemic inflammation : protective or pathological agents ?" Thesis, Paris 6, 2016. http://www.theses.fr/2016PA066279/document.

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Les neutrophiles contribuent à l'inflammation protectrice et pathologique. Ce projet de thèse consiste à déterminer le rôle des neutrophiles dans des modèles d'inflammation systémique graves et potentiellement mortelles, induite par le lipopolysaccharide (LPS, endotoxémie) ou par des complexes immuns antigène-anticorps (anaphylaxie). L'anaphylaxie est une réaction allergique qui peut être IgE- et/ou IgG-dépendante. L’endotoxémie est un modèle pertinent de l'inflammation au cours de maladies graves. Pour étudier les neutrophiles in vivo, nous avons utilisé un nouveau modèle murin de neutropénie

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Garcia, Geoffrey. "Les NETs (Neutrophils Extracellular Traps) et les DNases au cours de la COVID-19." Electronic Thesis or Diss., Bordeaux, 2024. http://www.theses.fr/2024BORD0175.

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Au cours des infections, les polynucléaires neutrophiles libèrent des Neutrophil Extracellular Traps (NETs) pour capturer les agents pathogènes et les détruire. Les NETs; en coordination avec les plaquettes et les cellules immunitaires, activent la coagulation et participent à l’immunothrombose pour piéger les agents pathogènes et éviter leur dissémination. Ils sont dégradés physiologiquement par les DNases et les macrophages. Exacerbée, les NETs sont responsables de dommages cellulaires et tissulaires. Ils sont décrits comme des acteurs centraux de nombreuses pathologies, notamment comme part

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Mawhin, Marie-Anne. "Role of neutrophils and leukotrienes in atherosclerotic plaque destabilisation : implication of endotoxemia." Thesis, Strasbourg, 2017. http://www.theses.fr/2017STRAJ034/document.

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La déstabilisation de la plaque d’athérosclérose reste de nos jours un problème majeur, malgré les progrès récents dans sa compréhension. Les neutrophiles sont des acteurs puissants de l’immunité innée capables d’altérer les plaques. Un chimio-attractant majeur des neutrophiles, le leucotriène B4, pourrait être un des contributeurs potentiels de la déstabilisation des plaques en particulier dans l’endotoxémie, elle-même associée aux accidents cardiovasculaires. L’objectif de ce travail a été de définir le rôle du leucotriène B4 dans l’attraction des neutrophiles dans la plaque au cours de l’en

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Yao, Yi. "Sequential Priming of Neutrophils." University of Toledo Health Science Campus / OhioLINK, 2014. http://rave.ohiolink.edu/etdc/view?acc_num=mco1389612809.

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Manriquez, Rojas Valeria. "Role of the innate immune response in vascular damage caused by Neisseria meningitidis infection Vascular colonization by Neisseria meningitidis triggers a delayed and inefficient neutrophil response Intermittent pili-mediated forces fluidize Neisseria meningitidis aggregates promoting vascular colonization Adhesion to nanofibers drives cell membrane remodeling through 1D wetting." Thesis, Sorbonne Paris Cité, 2018. http://www.theses.fr/2018USPCB076.

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Neisseria meningitidis est un diplocoque à Gram négatif responsable de méningite et de choc septique. Alors que la méningite est la forme d'infection la plus fréquente, la septicémie fulminante est responsable de 90% de la mortalité imputable à N. meningitidis. La septicémie méningococcique est caractérisée par une éruption purpurique due à des lésions vasculaires. Les observations au niveau histologique révèlent des méningocoques associés aux cellules endothéliales, des thromboses, des hémorragies périvasculaires et des infiltrations de cellules inflammatoires. Les mécanismes conduisant à ces

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Fischer, Steven Harold. "Interactions of Neisseria gonorrhoeae with human neutrophils: Gonococcal outer membrane protein II modulates neutrophil responses." Diss., The University of Arizona, 1988. http://hdl.handle.net/10150/184364.

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The disease gonorrhea has plagued mankind at least as long as written records have been kept (Black and Sparling, 1985). N. gonorrhoeae is still an important cause of suffering, infertility, and occasional mortality despite the fact that treatment with antibiotics is relatively easy and highly effective, even with the recent increase in penicillin-resistant isolates (Jephcott, 1986). The continued existence of this public health problem is partly the result of a reservoir of asymptomatic carriers within the community who normally don't seek treatment and continue their usual sexual practices (

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GRIMOLIZZI, FRANCO. "Neutrophils alter placental glucose metabolism in gestational diabetes mellitus via neutrophil elastase mediated IRS1 degradation." Doctoral thesis, Università Politecnica delle Marche, 2017. http://hdl.handle.net/11566/245194.

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La gravidanza è da considerarsi una condizione pro-inflammatoria dove si osserva un’attivazione dei neutrofili circolanti. Con l'avanzare della gravidanza la concentrazione nel sangue di nucleosomi e mieloperossidasi aumenta e riflette la produzione delle trappole extracellulari dei neutrofili (NETs). Abbiamo dimostrato che in corso di diabete mellito gestazionale (GDM) tale produzione è aumentata in confronto ad una gravidanza fisiologica. Elevati livelli di glucosio e TNF-a, segni tipici presenti in corso di GDM, in vitro agiscono in modo sinergico e sono in grado di pre-attivare i neutrof

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Darbousset, Roxane. "Roles of polymorphonuclear neutrophils in thrombosis." Thesis, Aix-Marseille, 2013. http://www.theses.fr/2013AIXM5071.

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L’hémostase est un processus physiologique permettant de préserver l’intégrité du système vasculaire et de prévenir une perte de sang en réponse à une blessure. En situation pathologique, comme dans le cas de cancers, d’infections ou de maladies cardiovasculaires, il peut y avoir activation de la cascade de coagulation entraînant la formation d’un thrombus.Dans cette étude, nous avons utilisé la microscopie intravitale dans un modèle de blessure au rayon laser pour comprendre les mécanismes cellulaires et moléculaires mis en jeu dans la formation d’un thrombus plaquettaire en conditions physio

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Haynes, Andrew Paul. "Metabolic abnormalities in uraemic neutrophils." Thesis, University of Nottingham, 1991. http://ethos.bl.uk/OrderDetails.do?uin=uk.bl.ethos.305133.

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Cross, Andrew. "Molecular properties of inflammatory neutrophils." Thesis, University of Liverpool, 2002. http://ethos.bl.uk/OrderDetails.do?uin=uk.bl.ethos.250467.

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Libri sul tema "Neutrophils"

S, Abramson Jon, and Wheeler J. Gary, eds. The Neutrophil. IRL Press at Oxford University Press, 1993.

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Voisin, Cyr, N. Wierzbicki, and J. P. Revillard. Mucosal immunity: IgA and polymorphonuclear neutrophils. Fondation Franco-Allemande, 1985.

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Tuominen-Gustafsson, Helena Birgitta. Calcium signalling pathways in human neutrophils. Åbo Akademis Förlag, 1998.

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Alonso-Fernández, Patricia. Neutrophils in biological age and longevity. Nova Science Publishers, Inc., 2011.

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Leino, Lasse. Studies on the function and expression of Fc-gamma and complement receptors on human neutrophils. Turun Yliopisto, 1991.

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1952-, Hallett Maurice B., ed. The Neutrophil: Cellular biochemistry and physiology. CRC Press, 1989.

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Hallett, Maurice B. The molecular and ionic signalling of neutrophils. Chapman & Hall, 1997.

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I, Gabrilovich Dmitry, ed. The neutrophils: New outlook for old cells. 2nd ed. Imperial College Press, 2004.

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Brumell, John H. Phosphorylation-dependent signalling mechanisms in human neutrophils. National Library of Canada = Bibliothèque nationale du Canada, 1997.

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Hallett, Maurice B. The molecular and ionic signaling of neutrophils. Landes Bioscience, 1997.

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Capitoli di libri sul tema "Neutrophils"

Whyte, Moira. "Neutrophils." In Cellular Mechanisms in Airways Inflammation. Birkhäuser Basel, 2000. http://dx.doi.org/10.1007/978-3-0348-8476-1_4.

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Zurier, Robert B. "Neutrophils." In Encyclopedia of Medical Immunology. Springer New York, 2014. http://dx.doi.org/10.1007/978-0-387-84828-0_23.

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Cuzzocrea, Salvatore. "Neutrophils." In Inflammation - From Molecular and Cellular Mechanisms to the Clinic. Wiley-VCH Verlag GmbH & Co. KGaA, 2017. http://dx.doi.org/10.1002/9783527692156.ch10.

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Jain, Rohit, Andrew J. Mitchell, Szun S. Tay, Ben Roediger, and Wolfgang Weninger. "Neutrophils." In Immunology of the Skin. Springer Japan, 2016. http://dx.doi.org/10.1007/978-4-431-55855-2_9.

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Zweiman, Burton. "Neutrophils." In Inflammatory Mechanisms in Allergic Diseases. CRC Press, 2023. http://dx.doi.org/10.1201/9780429134432-6.

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Clausen, Torben, José Luis Trejo, Mark P. Mattson, et al. "Neutrophils." In Encyclopedia of Exercise Medicine in Health and Disease. Springer Berlin Heidelberg, 2012. http://dx.doi.org/10.1007/978-3-540-29807-6_2760.

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Gevaert, Elien. "Neutrophils." In Chronic Rhinosinusitis. Springer Singapore, 2022. http://dx.doi.org/10.1007/978-981-16-0784-4_9.

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Kuijpers, Taco W., Timo K. van den Berg, and Dirk Roos. "Neutrophils Forever …" In Phagocyte-Pathogen Interactions. ASM Press, 2014. http://dx.doi.org/10.1128/9781555816650.ch1.

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Oliveira, André Gustavo, Rodrigo Guabiraba, Mauro Martins Teixeira, and Gustavo Batista Menezes. "Tumor-Associated Neutrophils." In Trends in Stem Cell Proliferation and Cancer Research. Springer Netherlands, 2013. http://dx.doi.org/10.1007/978-94-007-6211-4_18.

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Leino, Lasse. "Chemotaxis of Neutrophils." In Encyclopedia of Immunotoxicology. Springer Berlin Heidelberg, 2015. http://dx.doi.org/10.1007/978-3-642-54596-2_251.

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Atti di convegni sul tema "Neutrophils"

Datta, Rupsa, Veronika Miskolci, Gina M. Gallego-López, et al. "Metabolic Characterization of Neutrophil Activation Using Label-Free Fluorescence Lifetime Imaging Microscopy." In Optical Molecular Probes, Imaging and Drug Delivery. Optica Publishing Group, 2025. https://doi.org/10.1364/omp.2025.om1e.4.

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In this work we employ fluorescence lifetime imaging microscopy of intrinsic metabolic co-factor NAD(P)H to quantify metabolic changes in neutrophils upon activation and metabolic pathway perturbations across biological systems.

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Selak, M. A., M. Chignard, and J. B. Smith. "CHARACTERIZATION OF A NEUTROPHIL CPYMOTRYPSIN-LIKE ENZYME THAT ACTIVATES PLATELETS." In XIth International Congress on Thrombosis and Haemostasis. Schattauer GmbH, 1987. http://dx.doi.org/10.1055/s-0038-1643157.

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Communication between neutrophils and platelets was previously investigated by measuring platelet aggregation, serotonin release and changes in cytosolic free calcium subsequent to specific stimulation of neutrophils by fMet-Leu-Phe (FMLP) in a suspension of both cell types. The addition of the chemotactic peptide was shown to elicit secondary platelet activation as a consequence of primary stimulation of neutrophils. Cell-free supernatants from FMLP-stimulated neutrophils were capable of inducing platelet activation thus demonstrating that a factor released bv neutrophils was responsible for

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Teo, Soo Kng, Kim H. Parker, and K. H. Chiam. "Viscoelastic Finite-Element Modelling of Neutrophil Deformation in a Tapered Micropipette." In ASME 2008 Summer Bioengineering Conference. American Society of Mechanical Engineers, 2008. http://dx.doi.org/10.1115/sbc2008-191099.

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In this paper, we discuss the results arising from using a viscoelastic, axisymmetric finite-element model [1] to study the aspiration of neutrophils into a tapered micropipette [2]. The key feature of our model is that we take into account the experimentally observed temporal variations in the rheological properties of deforming neutrophils [3]. We show that our model successfully reproduces the equilibrium state reached by a neutrophil subjected to a constant step pressure [2]. We are also able to extract quantitative information about the rate at which the rheological properties of the neut

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Sounbuli, K., N. L. Mironova, O. V. Markov, and L. A. Alekseeva. "A COMPARATIVE STUDY OF DIFFERENT ISOLATION PROTOCOLS OF MURINE NEUTROPHILS FROM BONE MARROW AND SPLEEN." In OpenBio-2023. ИПЦ НГУ, 2023. http://dx.doi.org/10.25205/978-5-4437-1526-1-52.

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Neutrophils are considered the main player in innate immunity. In the last few years, they were involved in different physiological conditions and diseases. However, progress in the field of neutrophil biology is relatively slow due to several difficulties in neutrophil isolation and studying in culture. Here we compare three protocols based on density-gradient method and immunomagnetic method for mouse neutrophil isolation from the bone marrow and spleen.v

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Marcus, A. J., L. B. Safier, H. L. Ullman, N. Islam, M. J. Broekman, and C. V. Schacky. "NEW EICOSANOIDS FORMED DURING PLATELET-NEUTROPHIL INTERACTIONS." In XIth International Congress on Thrombosis and Haemostasis. Schattauer GmbH, 1987. http://dx.doi.org/10.1055/s-0038-1644626.

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In physiologic and pathologic processes such as hemostasis, thrombosis and inflammation, multiple cell types are brought into close proximity - thereby increasing the possibility of metabolic interactions in the microenvironment. Activated platelets synthesize12-hydroxyeicosatetraenoic acid (12-HETE) in the presence or absence of aspirin. During a cell-cell interaction, platelet 12-HETE is metabolized by a cytochrome P-450 enzyme system in unstimulated neutrophils to 12.20-dihydroxyeicosatetraenoic acid (12,20-DiHETE). Recently, we observed time-dependent formation of a new eicosanoid followin

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Coeffier, E., D. Joseph, and B. B. Vargaftio. "PLATELET-LEUKOCYTE INTERACTION: ACTIVATION OF RABBIT PLATELETS BY FMLP-STIMULATED NEUTROPHILS." In XIth International Congress on Thrombosis and Haemostasis. Schattauer GmbH, 1987. http://dx.doi.org/10.1055/s-0038-1643158.

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The interaction of neutrophils and platelets may be important in inflammation. We have studied the effect of the chemotactic peptide, N-formyl-methionyl-leucy1-phenylalanine (FMLP) on cells in whole rabbit blood or in mixture of purified rabbit platelets and neutrophils. In whole blood, FMLP triggered an aggregation (measured by electrical impedance) dependent upon the concentration of FMLP (9.9± 0.7 and 5.2+1.2 ohms at 1 and 0.01 uM FMLP resp.). This aggregation was accompanied by a strong decrease in platelets counts (54.6± 6.0 and 45.6± 3.8% for 1 and 0.01 uM FMLP resp.) and by a smaller de

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Bell, D., M. Jackson, C. MacRae, A. L. Muir, and J. Dawes. "NEUTROPHIL ELASTASE IS A MARKER OF NEUTROPHIL ACTIVATION IN ACUTE MYOCARDIAL INFARCTION." In XIth International Congress on Thrombosis and Haemostasis. Schattauer GmbH, 1987. http://dx.doi.org/10.1055/s-0038-1643020.

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Elastase is released from human neutrophils as a result of active secretion, phagocytosis or cell lysis. It is a broad-spectrum protease which not only hydrolyses the major components of tissue matrix, but can also affect platelet aggregation, coagulation and fibrinolysis. Neutrophils localise at the site of myocardial infarction and have been implicated in subsequent cell damage. A radioimmunoassay was developed which detects elastase complexed to its inhibitors α2- antitrypsin and α2-macroglobulin as wen as the free enzyme. Plasma concentrations of elastase in 31 healthy controls were 21.3 ±

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Muylle, L., C. Van Brussel, D. R. van Bockstaele, and M. E. Peetermans. "IN VITRO NEUTROPHIL ACTIVATION BY PLASMA OF STORED PLATELET CONCENTRATES." In XIth International Congress on Thrombosis and Haemostasis. Schattauer GmbH, 1987. http://dx.doi.org/10.1055/s-0038-1644686.

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A previous study showed an effect of storage time of platelet concentrates (PLC) on the frequency and severity of reactions to platelet transfusion.As most of the leukocytes were removed from the PLC prior to transfusion, it was suggested that at least part of the reactions were caused by the transfusion of cell products, accumulated during conservation of the PLC.This idea is supported by the finding of high histamine levels in PLC after 5 days storage.In order to test for the presence of substances causing neutrophil activation, samples from 7 PLC were taken at various storage times and incu

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Farache Trajano, Luiza, Rebecca Moore, and Quentin Sattentau. "The Presence of Chemical Cross-Linking Stabilises HIV-1 Envelope Glycoprotein Trimer Antigens in a Model of Intramuscular Immunisation." In Building Bridges in Medical Science 2021. Cambridge Medicine Journal, 2021. http://dx.doi.org/10.7244/cmj.2021.03.001.4.

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Background: The HIV-1 envelope glycoprotein (Env) is the target of antigen design for antibody- based vaccination. In 2019, four trimeric Env vaccines entered an experimental trial: ConM, ConS, and their cross-linked counterparts. The trimers were formulated with MPLA adjuvant. Studies have demonstrated that adjuvants trigger neutrophil infiltration. Neutrophils activate and degranulate releasing proteases, namely elastase and cathepsinG. Aims: To assess the stability and immunogenicity of these vaccines in the presence of adjuvant- recruited neutrophils and their proteolytic enzymes. Methods:

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Alteraifi, Abdullatif M., and Doncho V. Zhelev. "Cytoskeleton Rearrangement in Activated Human Neutrophils." In ASME 1996 International Mechanical Engineering Congress and Exposition. American Society of Mechanical Engineers, 1996. http://dx.doi.org/10.1115/imece1996-1110.

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Abstract The crawling of the neutrophil is essential for its physiological and pathological activity. The neutrophil crawls by projecting pseudopods in the direction of a higher chemoattractant concentration in a process known as chemotaxis. In this process, the cell initially adheres tightly to the present substrate and then rearranges its cytoskeleton. Finally, the ceil body contracts, which results in cell movement. The rearrangement of the cytoskeleton is believed to be a crucial component of cell motility (Cunningham et al., 1992) which determines the overall rate of crawling (Zigmond, 19

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Rapporti di organizzazioni sul tema "Neutrophils"

Boston, Mark E. Effects of Nasal Saline Spray on Human Neutrophils. Defense Technical Information Center, 2002. http://dx.doi.org/10.21236/ada406691.

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Liang, Feixin. Progress in Liquid Biopsy: A possible role of neutrophils. Science Repository, 2018. http://dx.doi.org/10.31487/j.cor.2018.02.004.

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Viksna, Ludmila, Oksana Kolesova, Aleksandrs Kolesovs, Ieva Vanaga, and Seda Arutjunana. Clinical characteristics of COVID-19 patients (Latvia, Spring 2020). Rīga Stradiņš University, 2020. http://dx.doi.org/10.25143/fk2/hnmlhh.

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Data include following variables: Demographics, epidemiological history, comorbidities, diagnosis, complications, and symptoms on admission to the hospital. Also, body’s temperature and SpO2. Blood cells: white cells count (WBC), neutrophils (Neu), lymphocytes (Ly), eosinophils (Eo) and monocytes (Mo), percentages of segmented and banded neutrophils, erythrocytes (RBC), platelet count (PLT), hemoglobin (Hb), and hematocrit (HCT); Inflammatory indicators: erythrocyte sedimentation rate (ESR) and C-reactive protein (CRP); Tissue damage indicators: alanine aminotransferase (ALT), lactate dehydrog

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Boston, Mark E., G. C. Frech, Enrique Chacon-Cruz, E. S. Buescher, and David G. Oelberg. Surfactant Releases Internal Calcium Stores in Neutrophils by G Protein-Mediated Pathway. Defense Technical Information Center, 2002. http://dx.doi.org/10.21236/ada413640.

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Haight, Gary. Inquiry into the causes and significance of cytoplasmic vacuolation of neutrophils in the peripheral circulation. Portland State University Library, 2000. http://dx.doi.org/10.15760/etd.5283.

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Youngman, Sara. The Chemotactic Response of Neutrophils to Components of the Sera of Mice Infected with Trichinella spiralis. Portland State University Library, 2000. http://dx.doi.org/10.15760/etd.6942.

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กาญจนะพังคะ, สุมลยา. การศึกษาเม็ดเลือดขาวและเกล็ดเลือดในกระแสโลหิตของปลาดุก ด้วยกล้องจุลทัศน์แสงสว่างและกล้องจุลทัศน์อิเลคตรอนแบบลำแสงผ่าน. จุฬาลงกรณ์มหาวิทยาลัย, 1998. https://doi.org/10.58837/chula.res.1998.53.

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ศึกษารายละเอียดโครงสร้างเม็ดเลือดขาวและเกล็ดเลือดในกระแสโลหิตของปลาดุก (Clariasbatrachus) ด้วยกล้องจุลทัศน์แสงสว่าง และกล้องจุลทัศน์อิเลคตรอนแบบลำแสงผ่าน พบเกล็ดเลือดลักษณะโครงสร้างต่างกัน 3 ชนิด พบเม็ดเลือดขาวชนิด lymphocyte monocyte และ neutrophil neutrophil เป็น granular leucocyte เพียงชนิดเดียวที่พบ มีทั้งชนิดที่เจริญเต็มที่และยังไม่เจริญเต็มที่ monocyte เกือบทุกเซลล์มีโครงสร้างที่มีลักษณะเฉพาะคือ microfilamentous structure อยู่ชิดรอยหยักของนิวเคลียสในเซลล์ การศึกษาครั้งนี้ได้บรรยายถึงการเปลี่ยนแปลงโครงสร้างของเกล็ดเลือดและ granulocyte ในกระบวนการแข็งตัวของเลือดปลาดุกได้ง่าย

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Bowles, Charles A., and Andrew Baum. Investigation of Stress Induced Alterations in Neutrophil Function. Defense Technical Information Center, 1991. http://dx.doi.org/10.21236/ada237002.

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Trofimenko, A., M. Mamus, S. Bedina, E. Mozgovaya, and S. Spitsina. PARTICIPATION OF NEUTROPHIL EXTRACELLULAR TRAPS IN AUTOIMMUNE RHEUMATIC DISEASES. DOI CODE, 2021. http://dx.doi.org/10.18411/wco-iof-esceo-2021-509.

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Bechev, Blagovest, Moni Magrisso, Stilian Stoeff, and Pavlina Glogovska. Possible Application in Pulmonology of Neutrophil Population Functional State Evaluated by Chemiluminescent Method. "Prof. Marin Drinov" Publishing House of Bulgarian Academy of Sciences, 2019. http://dx.doi.org/10.7546/crabs.2019.03.15.

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