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Letteratura scientifica selezionata sul tema "Noradrenaline"

Autore: Grafiati

Pubblicato: 4 giugno 2021

Ultima modifica: 25 luglio 2025

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Articoli di riviste sul tema "Noradrenaline"

1

Murtazina, A. R., Yu O. Nikishina, L. K. Dil’mukhametova, A. Ya Sapronova, and M. V. Ugrumov. "The role of the brain in the regulation of peripheral noradrenaline-producing organs in rats during morphogenesis." Доклады Академии наук 486, no. 6 (2019): 748–52. http://dx.doi.org/10.31857/s0869-56524866748-752.

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This work represents one part of our research project, in which we try to prove, that in the perinatal period exist a humoral regulation between noradrenaline producing organs. In this study we used a rat model that allowed blocking synthesis of noradrenalin in the brain and evaluated gene expression and protein levels of noradrenaline key synthesis enzymes such as tyrosine hydroxylase (TH) and dopamine beta-hydroxylase (DBH) in peripheral noradrenaline producing organs. As a result we showed increased gene expression of TH and DBH in adrenal glands. This data indicate that if neonatal rat bra

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2

Rudzite, Vera, Edite Jurika, Gilbert Reibnegger, Günter Weiss, Helmut Wachter, and Dietmar Fuchs. "Influence of Kynurenine, Neopterin, Noradrenaline and Pyridoxal-5-Phosphate on Cholesterol and Phospholipid Content and Phospholipid Biosynthesis in vitro." Pteridines 4, no. 3 (1993): 126–30. http://dx.doi.org/10.1515/pteridines.1993.4.3.126.

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Summary Incorporation of fatty acids into phospholipids has been investigated using samples of rat liver tissue homogenate, Krebs-Ringer-phosphate buffer (pH = 7.4) containing 0.3% albumin, fatty acid mixture and glyceroL The addition of L-kynurenine (4 nmol/g wet weight), D-eryhro-neopterin (5 and 30 pmol/g wet weight) and noradrenaline (4 nmol/g wet weight) to incubation medium induced an increase of saturated (palmitic acid) and decrease of poly-unsaturated (linoleic and arachidonic acid) fatty acids incorporation into phospholipids. The increase of saturated fatty acids incorporation into

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3

Gushcha, V. K., S. V. Lelevich, and V. M. Sheibak. "Neurotransmitter disturbances in some parts of the rat brain and their correction under chronic and intermittent alcohol intoxication." Biomeditsinskaya Khimiya 65, no. 1 (2019): 21–27. http://dx.doi.org/10.18097/pbmc20196501021.

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The pool of key neuromediators and some neurotransmitter amino acids in cerebellum, hypothalamus and midbrain of rats exposed to chronic and different variants of interrupted alcohol intoxication was investigated. The most pronounced changes were recorded in midbrain. Chronic alcohol intoxication caused an increase in the concentrations of tyrosine, dopamine, 3,4-dihydroxyphenylacetic acid (DOPAC), noradrenaline, tryptophan, serotonin, GABA and aspartate in this part of the rat brain. Interrupted alcohol intoxication with 4 days interval is accompanied by an increase in the content of tyrosine

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4

Eisenhofer, Graeme, Murray D. Esler, Ian T. Meredith, Claudia Ferrier, Gavin Lambert, and Garry Jennings. "Neuronal Re-Uptake of Noradrenaline by Sympathetic Nerves in Humans." Clinical Science 80, no. 3 (1991): 257–63. http://dx.doi.org/10.1042/cs0800257.

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1. Plasma concentrations of [3H]dihydroxyphenylglycol, the intraneuronal metabolite of noradrenaline, were examined during intravenous infusion of [3H]noradrenaline in 43 subjects, to assess the nature of its formation. Noradrenaline re-uptake by sympathetic nerves was estimated in 11 subjects from the effects of neuronal uptake blockade with desipramine on noradrenaline clearance and plasma concentrations of [3H]dihydroxyphenylglycol and endogenous dihydroxyphenylglycol. In seven subjects noradrenaline re-uptake and spillover into plasma were examined before and during mental arithmetic or ha

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5

D’Andrea, Giovanni, Massimo Leone, Gennaro Bussone, et al. "Abnormal tyrosine metabolism in chronic cluster headache." Cephalalgia 37, no. 2 (2016): 148–53. http://dx.doi.org/10.1177/0333102416640502.

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Objective Episodic cluster headache is characterized by abnormalities in tyrosine metabolism (i.e. elevated levels of dopamine, tyramine, octopamine and synephrine and low levels of noradrenalin in plasma and platelets.) It is unknown, however, if such biochemical anomalies are present and/or constitute a predisposing factor in chronic cluster headache. To test this hypothesis, we measured the levels of dopamine and noradrenaline together with those of elusive amines, such as tyramine, octopamine and synephrine, in plasma of chronic cluster patients and control individuals. Methods Plasma leve

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6

RONGEN, Gerard A., Jacques W. M. LENDERS, Paul SMITS, and John S. FLORAS. "Comparison of two indices for forearm noradrenaline release in humans." Clinical Science 99, no. 5 (2000): 363–69. http://dx.doi.org/10.1042/cs0990363.

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Abstract (sommario):

Although there is as yet no method which measures directly the neuronal release of noradrenaline in humans in vivo, the isotope dilution technique with [3H]noradrenaline has been applied to estimate forearm neuronal noradrenaline release into plasma. Two different equations have been developed for this purpose: one to estimate the spillover of noradrenaline into the venous effluent, and a modified formula (often referred to as the appearance rate) which may reflect more closely changes in the neuronal release of noradrenaline into the synaptic cleft, particularly during interventions that alte

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7

Lang, Chim C., Abdul R. Rahman, David J. K. Balfour, and Allan D. Struthers. "Effect of Noradrenaline on Renal Sodium and Water Handling in Euhydrated and Overhydrated Man." Clinical Science 85, no. 4 (1993): 487–94. http://dx.doi.org/10.1042/cs0850487.

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Abstract (sommario):

1. The renal effects of incremental doses of intravenously infused noradrenaline were evaluated in normal subjects during two different water loads, 5 ml/kg (n = 6) and 20 ml/kg (n = 9), producing conditions of euhydration and overhydration, respectively. 2. Noradrenaline infusion rates ranged from 0.015 to 0.075 μg min−1 kg−1. In the euhydrated subjects, noradrenaline caused a dose-dependent fall in urinary sodium excretion and an increase in urinary flow rate. During overhydration similar doses of noradrenaline caused a fall in urinary sodium excretion but a decrease in urinary flow rate. 3.

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8

Eisenhofer, Graeme, David S. Goldstein, and Irwin J. Kopin. "Plasma dihydroxyphenylglycol for estimation of noradrenaline neuronal re-uptake in the sympathetic nervous system in vivo." Clinical Science 76, no. 2 (1989): 171–82. http://dx.doi.org/10.1042/cs0760171.

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Abstract (sommario):

1. Neuronal re-uptake is the primary means for terminating the actions of endogenously released noradrenaline. A portion of the recaptured noradrenaline is deaminated to form dihydroxyphenylglycol. The present report describes a technique using plasma dihydroxyphenylglycol for estimation of the rate of neuronal reuptake of endogenous noradrenaline in vivo. 2. Neuronal re-uptake of noradrenaline in the sympathetic nervous system of the rat was estimated from the effects of neuronal uptake blockade with desipramine on three variables: (i) the plasma clearance of intravenously infused 3H-labelled

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9

Lamontagne, Daniel, Nobuharu Yamaguchi, Christophe Ribuot, Jacques de Champlain, and Réginald Nadeau. "Reduction of tissue noradrenaline content in the isolated perfused rat heart during ischemia: importance of monoamine oxidation." Canadian Journal of Physiology and Pharmacology 69, no. 8 (1991): 1190–95. http://dx.doi.org/10.1139/y91-174.

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The effect of ischemia on myocardial noradrenaline concentration and endogenous noradrenaline output was studied in the isolated perfused rat heart. Following a 15-min stabilization period, regional ischemia was produced by coronary artery ligation. After 60 min of ischemia, noradrenaline concentrations were significantly reduced in the interventricular septum and left ventricle but not in the right ventricle. The reduction in tissue noradrenaline concentration was not prevented when the 60-min ischemia was replaced by a 10-min ischemia followed by a 50-min perfusion. No modification in noradr

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10

Hammond, James R., Wenda F. MacDonald, and Thomas D. White. "Evoked secretion of [3H]noradrenaline and ATP from nerve varicosities isolated from the myenteric plexus of the guinea pig ileum." Canadian Journal of Physiology and Pharmacology 66, no. 3 (1988): 369–75. http://dx.doi.org/10.1139/y88-062.

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Abstract (sommario):

Neuronal varicosities, isolated from the myenteric plexus of guinea pig ileum longitudinal muscle, were incubated with [3H]noradrenaline to label the contents of the noradrenergic secretory vesicles. Exposure of these varicosities to KCl, nicotine, or acetylcholine resulted in the Ca2+-dependent release of [3H]noradrenaline. Veratridine also evoked a large efflux of [3H] from this preparation, but this release was only partially Ca2+ dependent. The α2-adrenoceptor agonist, clonidine, inhibited the K+-, nicotine-, and acetylcholine-induced release of [3H]noradrenaline. Similarly, exogenously ad

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Tesi sul tema "Noradrenaline"

1

Smith, Nigel. "Plasma protein binding of noradrenaline." Thesis, University of Nottingham, 1990. http://ethos.bl.uk/OrderDetails.do?uin=uk.bl.ethos.252951.

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2

Scriven, Anthony James Ivor. "Plasma noradrenaline and cardiovascular sympathetic activity." Thesis, Imperial College London, 1990. http://hdl.handle.net/10044/1/46541.

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3

Haywood, S. A. "GnRH network : noradrenaline and sex steroids." Thesis, University of Cambridge, 2000. http://ethos.bl.uk/OrderDetails.do?uin=uk.bl.ethos.603906.

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Abstract (sommario):

The first studies in this thesis use <I>in situ</I> hybridisation to investigate if temporal changes occur in cellular GnRH gene expression during the preovulatory phase of the oestrous cycle and further investigation gene expression in other neurotransmitter systems in the GnRH network within the preoptic area and the brainstem. It is evident from these studies that, during the oestrous cycle, transcriptional changes within GnRH, enkphalin, nitric oxide synthase and noradrenaline containing neural populations, in the preoptic area and brainstem respectively, are small, and are perhaps not per

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4

Field, Brent A. "The effects of noradrenaline on cortical signal processing /." view abstract or download file of text, 2000. http://wwwlib.umi.com/cr/uoregon/fullcit?p9978587.

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Thesis (Ph. D.)--University of Oregon, 2000.<br>Typescript. Includes vita and abstract. Includes bibliographical references (leaves 140-159). Also available for download via the World Wide Web; free to University of Oregon users.

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Kelly, Christopher Brendan. "Noradrenergic function in anxiety and depressive states." Thesis, Queen's University Belfast, 1994. http://ethos.bl.uk/OrderDetails.do?uin=uk.bl.ethos.261770.

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6

Roberts, Veronica J. "Corticosterone modulation of noradrenaline stimulated cyclic AMP formation in hippocampus." Thesis, University of Ottawa (Canada), 1985. http://hdl.handle.net/10393/4949.

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7

Brice, Carolyn F. "Caffeine : consumption, behavioural effects and the role of central noradrenaline." Thesis, University of Bristol, 2000. http://ethos.bl.uk/OrderDetails.do?uin=uk.bl.ethos.302061.

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8

Popovik, Elvira. "The role of noradrenaline in the development of rat neocortex." Thesis, University of Bristol, 2003. http://ethos.bl.uk/OrderDetails.do?uin=uk.bl.ethos.289632.

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9

DONG, WEN-XIN. "Metabolisme pre-synaptique peripherique de la noradrenaline chez le rat." Paris 6, 1997. http://www.theses.fr/1997PA066067.

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Abstract (sommario):

Nous avons etudie le metabolisme pre-synaptique de la noradrenaline (na) chez l'animal entier ; cette voie metabolique etant la voie la plus importante pour la na synaptique. Un dosage radio-enzymatique, approprie pour mesurer les concentrations plasmatiques des derives desamines de la na (3,4-dihydroxyphenyl glycol : dhpg et 3,4-dihydroxymandelique acide : doma) a ete developpe et valide au plan physiologique. Une reduction de l'activite sympathique par la clonidine ou la guanethidine provoque une baisse de la na mais aussi du dhpg ; la reponse du doma est moins claire. Dans une deuxieme etap

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10

MAIGNAN, EMMANUELLE. "Systeme sympathique : la cinetique de la noradrenaline chez le rat." Paris 6, 2001. http://www.theses.fr/2001PA066331.

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L'objectif de ce travail a ete d'etudier la physiologie du systeme sympathique chez le rat par la technique de la dilution isotopique (di). Nous avons mesure la cinetique de la noradrenaline (na), definie par son trop plein synaptique (tps) et sa clairance metabolique (cm). Nous montrons que l'anesthesie provoque des alterations majeures et heterogenes de la cinetique de la na. Nous avons d'abord retenu que l'etat experimental qui s'imposait etait le rat conscient, non restreint. Dans ce contexte, son tps est environ 10 fois plus eleve que celui de l'homme, suggerant une activite sympathique b

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Libri sul tema "Noradrenaline"

1

1960-, Fuchs Stefan, and Auer Max 1962-, eds. Biochemistry and histocytochemistry research developments. Nova Science, 2009.

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2

J, Heal David, and Marsden C. A, eds. The Pharmacology of noradrenaline in the central nervous system. Oxford University Press, 1990.

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3

Ward, Valerie Lynn. Oxygen-derived free radicals and the pathogenesis of catecholamine cardiomyopathy. s.n.], 1993.

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4

Singh, Lakhbir. Modulation of a 5-hydroxytryptamine-related behaviour by noradrenaline and gaba. University of Aston. Department of Pharmaceutical Sciences, 1985.

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5

A, Ordway Gregory, Schwartz Michael A, and Frazer Alan, eds. Brain norepinephrine: Neurobiology and therapeutics. Cambridge University Press, 2007.

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6

Pernow, John. Co-release and functional interactions of neuropeptide Y and noradrenaline in peripheral sympathetic vascular control. Published for the Scandinavian Physiological Society by Blackwell Scientific Publications, 1988.

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7

Ichishima, Eiji. Unique enzymes of Aspergillus fungi used in Japanese bioindustries. Nova Science Publishers, 2011.

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8

Kirkup, Anthony Joseph. Modulation of membrane currents and mechanical activity by noradrenaline and other agents in vascular smooth muscle. University of Manchester, 1995.

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9

Hentschel, Erwin. Occurence and function of biogenic amines (5-hydroxytryptamine, dopamine, noradrenaline, adrenaline and octopamine) in invertebrates: Bibliography = Vorkommen und Funktion biogener Amine (5-hydroxytryptamin, Dopamin, Noradrenalin, Adrenalin und Octopamin) bei wirbellosen Tieren : Bibliographie. Universitätsbibliothek, 1986.

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10

Hentschel, Erwin. Occurrence and function of biogenic amines (5-hydroxytryptamine, dopamine, noradrenaline, adrenaline and octopamine) in invertebrates: Bibliography = Vorkommen und Funktion biogener Amine (5-hydroxytryptamin, Dopamin, Noradrenalin, Adrenalin und Octopamin) bei wirbellosen Tieren : Bibliographie. Universitätsbibliothek, 1986.

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Capitoli di libri sul tema "Noradrenaline"

1

Andrews, Anne M., Greg A. Gerhardt, Lynette C. Daws, et al. "Noradrenaline." In Encyclopedia of Psychopharmacology. Springer Berlin Heidelberg, 2010. http://dx.doi.org/10.1007/978-3-540-68706-1_715.

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2

Kruk, Zygmunt L., and Christopher J. Pycock. "Noradrenaline." In Neurotransmitters and Drugs. Springer Netherlands, 1991. http://dx.doi.org/10.1007/978-94-011-3132-2_3.

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3

Kruk, Zygmunt L., and Christopher J. Pycock. "Noradrenaline." In Neurotransmitters and Drugs. Springer Netherlands, 1991. http://dx.doi.org/10.1007/978-94-011-3134-6_3.

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4

Pannu, Neesh, Xiaoyan Wen, John A. Kellum, et al. "Noradrenaline." In Encyclopedia of Intensive Care Medicine. Springer Berlin Heidelberg, 2012. http://dx.doi.org/10.1007/978-3-642-00418-6_1963.

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5

Anagnostou, Evdokia, Deepali Mankad, Joshua Diehl, et al. "Noradrenaline." In Encyclopedia of Autism Spectrum Disorders. Springer New York, 2013. http://dx.doi.org/10.1007/978-1-4419-1698-3_100948.

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6

Amemiya, Seiichiro. "Noradrenaline." In Encyclopedia of Personality and Individual Differences. Springer International Publishing, 2020. http://dx.doi.org/10.1007/978-3-319-24612-3_785.

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7

Amemiya, Seiichiro. "Noradrenaline." In Encyclopedia of Personality and Individual Differences. Springer International Publishing, 2017. http://dx.doi.org/10.1007/978-3-319-28099-8_785-1.

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8

Segal, Sabrina. "Norepinephrine/Noradrenaline." In Encyclopedia of Behavioral Medicine. Springer International Publishing, 2020. http://dx.doi.org/10.1007/978-3-030-39903-0_39.

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9

Tiro, Jasmin, Simon J. Craddock Lee, Steven E. Lipshultz, et al. "Norepinephrine/Noradrenaline." In Encyclopedia of Behavioral Medicine. Springer New York, 2013. http://dx.doi.org/10.1007/978-1-4419-1005-9_39.

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10

Froehlich, Stephan J., Carlo A. Lackerbauer, Guenter Rudolph, et al. "Noradrenaline Deficiency." In Encyclopedia of Molecular Mechanisms of Disease. Springer Berlin Heidelberg, 2009. http://dx.doi.org/10.1007/978-3-540-29676-8_8067.

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Atti di convegni sul tema "Noradrenaline"

1

Verisokin, Andrey Yu, Darya V. Verveyko, Alexey R. Brazhe, and Stanislav V. Lagosha. "Simple model of neurogliovascular unit activity with noradrenaline leading force." In 2024 8th Scientific School Dynamics of Complex Networks and their Applications (DCNA). IEEE, 2024. http://dx.doi.org/10.1109/dcna63495.2024.10718403.

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2

Van Diest, M. J., T. J. Verbeuren, and A. G. Herman. "RELAXATIONS INDUCED BY LIPOXYGENASE METABOLITES OF ARACHI-DONIC ACID IN SPLENIC ARTERIES OF THE DOG." In XIth International Congress on Thrombosis and Haemostasis. Schattauer GmbH, 1987. http://dx.doi.org/10.1055/s-0038-1643796.

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The lipoxygenase metabolites of arachidonic acid, 15 hydroperoxyeicosatetraenoic acid (15HPETE) and its hydroxy derivative (15HETE) evoke contractions in a variety of isolated blood vessels. We recently were able to show that thromboxane A2 (TXA2)-receptor antagonists BM13177 and BM13505 suppress the contractions induced by lipoxygenase metabolites in canine splenic arteries, and that these compounds also inhibit the contractile effects of PGF2α and of the TXA2-mimetic U46619. We also reported that 15HETE and 15HPETE cause relaxations of isolated dog arteries when the tissues are contracted wi

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3

Carrere, Maxime, and Frederic Alexandre. "Modeling the sensory roles of noradrenaline in action selection." In 2016 Joint IEEE International Conference on Development and Learning and Epigenetic Robotics (ICDL-EpiRob). IEEE, 2016. http://dx.doi.org/10.1109/devlrn.2016.7846814.

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4

Ishikawa, G., X. Peng, J. Mcgovern, et al. "Role of Noradrenaline and Macrophage Dynamics in Pulmonary Fibrosis." In American Thoracic Society 2024 International Conference, May 17-22, 2024 - San Diego, CA. American Thoracic Society, 2024. http://dx.doi.org/10.1164/ajrccm-conference.2024.209.1_meetingabstracts.a5206.

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5

Hesse, S., C. Ulke, M. Rullmann, et al. "Prefrontal-thalamic noradrenaline transporter availability is reduced in adults with ADHD." In NuklearMedizin 2019. Georg Thieme Verlag KG, 2019. http://dx.doi.org/10.1055/s-0039-1683515.

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6

Chung, Hui-Kuan, and Philippe Tobler. "Boosting noradrenaline improves economic rationality by enhancing attention in healthy humans." In 2023 Conference on Cognitive Computational Neuroscience. Cognitive Computational Neuroscience, 2023. http://dx.doi.org/10.32470/ccn.2023.1463-0.

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7

Sun, H., C. Perry, A. Ghincea, T. Baernthaler, and E. Herzog. "Reduction in Systemic Noradrenaline Does Not Suppress Bleomycin Induced Lung Fibrosis." In American Thoracic Society 2022 International Conference, May 13-18, 2022 - San Francisco, CA. American Thoracic Society, 2022. http://dx.doi.org/10.1164/ajrccm-conference.2022.205.1_meetingabstracts.a1943.

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BRINK, K., F. DERKX, E. BROMMER, J. STIBBE, H. KOLSTEE, and M. SCHALEKAMP. "THE FIBRINOLYTIC, FACTOR VIII:C, VON WILLEBRAND FACTOR AND HEMODYNAMIC RESPONSES TO DDAVP IN PATIENTS WITH HEREDITARY NEPHROGENIC DIABETES INSIPIDUS." In XIth International Congress on Thrombosis and Haemostasis. Schattauer GmbH, 1987. http://dx.doi.org/10.1055/s-0038-1644709.

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The pressor response of vasopressin (AVP) is mediated by a calcium-dependent mechanism (VI-receptor), whereas its antidiuretic effect depends on c-ANP (V2-receptor). DDAVP (1-desamino-8-D-arginine vasopressin) is a synthetic V2 analog of AVP. AVP and DDAVP also increase FVIII:C vWF:Ag and tissue-type plasminogen activator (t-PA) in plasma. The mechanism by which AVP and DDAVP elevate these factors is unclear. Patients with X-linked nephrogenic diabetes insipidus (NDI) are resistant to the V2-mediated antidiuretic action of AVP and DDAVP. We therefore have studied the effect of DDAVP (0.4 ug/kg

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9

Schmidt, E., C. Schinke, M. Rullmann, et al. "Changes of central noradrenaline transporter availability in immunotherapy-naïve multiple sclerosis patients." In NuklearMedizin 2020. © Georg Thieme Verlag KG, 2020. http://dx.doi.org/10.1055/s-0040-1708189.

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10

Morais, Camila, Dora Kassisse, Filipy Borghi, Carolina da Silva, and Priscila da Silva. "Signaling downstream cascade crosstalk induced by noradrenaline on angiotensin II metabolic activity in adipocytes." In Congresso de Iniciação Científica UNICAMP. Universidade Estadual de Campinas, 2019. http://dx.doi.org/10.20396/revpibic2720191878.

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Rapporti di organizzazioni sul tema "Noradrenaline"

1

Arias Quiroz, Kevin Josué, and Ariel Enrique Pastrana Arias. Efectos en el sistema nervioso de recién nacidos por consumo de cocaína en la vida intrauterina. Universidad de Cartagena, 2023. https://doi.org/10.32997/11227/18440.

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Si bien en la actualidad no es que ese vean muchos casos de niños afectados por el consumo de la droga durante la vida intra uterina, pero cada vez se es más frecuente por lo cual es un tema de investigación importante. Para que se hagan de cuanto está aumentando el consumo en mujeres en edad fértil algún tipo de sustancia en Colombia, el Observatorio de Drogas de Colombia (ODC) reportó un aumentó en un 3,5% en el consumo de sustancias como marihuana, pasta básica de coca, éxtasis o heroína en las personas entre los 15 y 65 años. Desde hace muchos años se han descrito numerosos efectos secunda

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2

Noradrenaline confirmed as first-choice vasopressor for septic shock. National Institute for Health Research, 2015. http://dx.doi.org/10.3310/signal-000165.

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