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Articoli di riviste sul tema "Peritoneal dialysis"

Autore: Grafiati

Pubblicato: 4 giugno 2021

Ultima modifica: 25 luglio 2025

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1

Nakamoto, Hidetomo, Hiroe Imai, Yuji Ishida, et al. "New Animal Models for Encapsulating Peritoneal Sclerosis—Role of Acidic Solution." Peritoneal Dialysis International: Journal of the International Society for Peritoneal Dialysis 21, no. 3_suppl (2001): 349–53. http://dx.doi.org/10.1177/089686080102103s64.

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Objective Encapsulating peritoneal sclerosis (EPS), in which all or part of the intestine is enveloped in a fibrous ball resembling a cocoon, is a serious complication of peritoneal dialysis (PD). The aim of the present study was to investigate whether pH-neutral or acidic dialysis solutions induce peritoneal fibrosis. Design We divided 18 male Wistar–Kyoto (WKY) rats into three groups and dialyzed them with various solutions as follows: group I, 10 mL acidic dialysis solution (pH 3.8, containing 1.35% glucose), n = 6; group II, 10 mL pH 5.0 dialysis solution, n = 6; and group III, 10 mL neutr

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2

Davenport, Andrew. "Prescribing Peritoneal Dialysis for Elderly Patients Starting Peritoneal Dialysis." Kidney and Dialysis 5, no. 2 (2025): 13. https://doi.org/10.3390/kidneydial5020013.

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Increased availability of dialysis services has led to both an increase in the number of elderly, frail, co-morbid patients with advanced chronic kidney disease now being offered dialysis and starting dialysis with residual kidney function. Traditionally, these patients would have been offered in-centre haemodialysis. However, the introduction of an assisted peritoneal dialysis service has allowed more of these elderly patients to be considered for peritoneal dialysis, a home-based treatment, with the exchanges performed by family members or visiting health care staff. It is now realised that

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3

Lu, Jingyuan, Danye Shi, Xinhui Zhao, Minhui Xi, Hualin Qi та Qiang He. "Crocin ameliorates peritoneal fibrosis in rat induced by peritoneal dialysis via Wnt5a/β-Catenin pathway". Quality Assurance and Safety of Crops & Foods 14, № 4 (2022): 36–44. http://dx.doi.org/10.15586/qas.v14i4.1151.

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Peritoneal dialysis is used in the treatment of patients with kidney diseases. Long-term peritoneal dialysis could result in peritoneal fibrosis and recurrent peritonitis, thus leading to failure of ultrafiltration. Crocin is a bioactive carotenoid and isolated from stigma of Crocus sativus, and ameliorates pulmonary and myocardial fibrosis. The role of crocin in peritoneal fibrosis was assessed. Firstly, rats model with peritoneal dialysis was treated with 4.25% peritoneal dialysate. Results showed that injection with peritoneal dialysate induced obvious hyperplasia and increased thickness in

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4

Andreoli, Maria Claudia Cruz, and Claudia Totoli. "Peritoneal Dialysis." Revista da Associação Médica Brasileira 66, suppl 1 (2020): s37—s44. http://dx.doi.org/10.1590/1806-9282.66.s1.37.

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SUMMARY Peritoneal dialysis (PD) is a renal replacement therapy based on infusing a sterile solution into the peritoneal cavity through a catheter and provides for the removal of solutes and water using the peritoneal membrane as the exchange surface. This solution, which is in close contact with the capillaries in the peritoneum, allows diffusion solute transport and osmotic ultrafiltration water loss since it is hyperosmolar to plasma due to the addition of osmotic agents (most commonly glucose). Infusion and drainage of the solution into the peritoneal cavity can be performed in two ways: m

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5

Wieczorowska-Tobis, K., K. Korybalska, A. Polubinska, M. Radkowski, A. Breborowicz, and D. G. Oreopoulos. "In Vivo Model to Study the Biocompatibility of Peritoneal Dialysis Solutions." International Journal of Artificial Organs 20, no. 12 (1997): 673–77. http://dx.doi.org/10.1177/039139889702001203.

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This study was designed to analyze the complex morphologic and functional effects of dialysis solutions on peritoneum in a rat model on chronic peritoneal dialysis. Peritoneal catheters were inserted into 10 male, Wistar rats and the animals were dialyzed twice daily for 4 weeks with 4.25% Dianeal. During the study we observed two opposite effects: healing of the peritoneum after catheter implantation - decreased cell count in dialysate, decreased permeability of the peritoneum to glucose and total protein, increased volume of drained dialysate; and damage to the membrane due to its exposure t

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6

Wu, George, Katarzyna Wieczorowska Tobis, Alicja Polubinska, et al. "N-Acetylglucosamine Changes Permeability of Peritoneum during Chronic Peritoneal Dialysis in Rats." Peritoneal Dialysis International: Journal of the International Society for Peritoneal Dialysis 18, no. 2 (1998): 217–24. http://dx.doi.org/10.1177/089686089801800212.

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Objective To evaluate the effect of supplementation of dialysis fluid with N-acetylglucosamine (NAG) on the permeability of peritoneum during chronic peritoneal dialysis in rats. Design Experiments were performed on rats with surgically implanted peritoneal catheters. Dialysis solution [DianeaI1.5% (Baxter, Deerfield, IL, U.S.A.) supplemented with either NAG 50 mmol/L or glucose 50 mmol/L (control)] was infused intraperitoneally twice, every day, for 8 weeks. Peritoneal equilibration tests (PET) were performed in all animals at the beginning of the study and after 8 weeks of dialysis. Addition

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7

Kostović, Milica, Milica Cvetkovic, and Dejan Petrovic. "Gastrointestinal Non-Infectious Complications in Patients on Peritoneal Dialysis." Serbian Journal of Experimental and Clinical Research 17, no. 2 (2016): 153–60. http://dx.doi.org/10.1515/sjecr-2015-0039.

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Abstract Gastrointestinal complications are common among patients on peritoneal dialysis. Risk factors for the development of gastrointestinal complications in this patient population include: toxic effects of uremic toxins, frequent use of nonsteroidal anti-inflammatory drugs, Helicobacter pylori infection, angiodysplasia, increased intra-abdominal pressure, use of bioincompatible solution for peritoneal dialysis, increased glucose in solutions for peritoneal dialysis, secondary hyperparathyroidism (hypercalcemia), a disorder of lipid metabolism (hypertriglyceridemia), and the duration of per

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8

Sulowicz, Wladyslaw, Tadeusz Cichocki, and Zygmunt Hanicki. "Changes in Activity of Selected Lysosomal Enzymes in Peritoneal Macrophages of Renal Failure Patients on Peritoneal Dialysis." Peritoneal Dialysis International: Journal of the International Society for Peritoneal Dialysis 9, no. 4 (1989): 313–17. http://dx.doi.org/10.1177/089686088900900417.

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Activity of acid phosphatase (AP), beta-glucuronidase (GR), N-acetyl-beta-D-glucosaminidase (GZ), and peroxidase (P) was assessed using a semiquantitative cytochemical method in peritoneal macro phages of 30 patients with end-stage renal failure treated by intermittent peritoneal dialysis and of 30 control patients with normal renal function. The dialysed patients showed a significantly higher activity of GR and P at the beginning of the treatment as compared with the respective activities observed in the control group and a further significant rise of these activities after 4 months of dialys

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9

Kowalewska, Paulina M., Peter J. Margetts, and Alison E. Fox-Robichaud. "Peritoneal Dialysis Catheter Increases Leukocyte Recruitment in the Mouse Parietal Peritoneum Microcirculation and Causes Fibrosis." Peritoneal Dialysis International: Journal of the International Society for Peritoneal Dialysis 36, no. 1 (2016): 7–15. http://dx.doi.org/10.3747/pdi.2014.00211.

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♦BackgroundThe objective of this study was to examine the effects of a conventional dialysis solution and peritoneal catheter on leukocyte- endothelial cell interactions in the microcirculation of the parietal peritoneum in a subacute peritoneal dialysis (PD) mouse model.♦MethodsAn intraperitoneal (IP) catheter with a subcutaneous injection port was implanted into mice and, after a 2-week healing period, the animals were injected daily for 6 weeks with a 2.5% dextrose solution. Intravital microscopy (IVM) of the parietal peritoneum microcirculation was performed 4 hours after the last injectio

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10

RADULESCU, Daniela, Dragos Adrian GEORGESCU, Andrei ANGELESCU, and Bogdan Florin GEAVLETE. "Abdominal Pseudocyst in the Vicinity of Calcified Renal Allograft in a Patient with Peritoneal Dialysis - Case Report." Medicina Moderna - Modern Medicine 27, no. 3 (2020): 237–40. http://dx.doi.org/10.31689/rmm.2020.27.3.237.

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Abdominal pseudocysts are rarely reported in peritoneal dialysis and usually arise secondary to repeated dialysisrelated peritonitis. We present the case of a patient with end-stage renal disease treated for 9 years by continuous ambulatory peritoneal dialysis that developed an abdominal pseudocyst in the vicinity of the non-functional and calcifi ed renal graft. Because the adequacy of peritoneal dialysis was optimal, surgical removal of the invaginated peritoneum and closure of the breach allowed the patient to continue peritoneal dialysis treatment.

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11

K Nair, Deepak. "Peritoneal Dialysis - An Overview." International Journal of Science and Research (IJSR) 10, no. 8 (2021): 987–88. https://doi.org/10.21275/sr21819111053.

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12

Catalan, Marina Penélope, Jaime Esteban, Dolores Subirá, Jesús Egido, and Alberto Ortiz. "Inhibition of Caspases Improves Bacterial Clearance in Experimental Peritonitis." Peritoneal Dialysis International: Journal of the International Society for Peritoneal Dialysis 23, no. 2 (2003): 123–26. http://dx.doi.org/10.1177/089686080302300205.

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Background Inhibition of caspases improves the antibacterial capacity of leukocytes cultured with peritoneal dialysis solutions, and improves the prognosis of septic, polymicrobial experimental peritonitis. Objective To test whether inhibition of caspases alters the evolution of peritonitis in the presence of peritoneal dialysis solution. Design 32 mice were assigned to therapy with either the pan-caspase inhibitor zVAD or vehicle for 48 hours following infection with Staphylococcus aureus, in the presence of lactate-buffered, 4.25% glucose peritoneal dialysis solution. 16 mice received vehicl

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13

Djurdjevic-Mirkovic, Tatjana. "Peritoneal dialysis - experiences." Medical review 63, no. 11-12 (2010): 753–57. http://dx.doi.org/10.2298/mpns1012753d.

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Peritoneal dialysis is the method of treatment of terminal-stage chronic kidney failure. Nowadays, this method is complementary to haemodialysis. It is based on the principles of the diffusion of solutes and ultrafiltration of fluids across the peritoneal membrane, which acts as a filter. The dialysate is introduced into the peritoneum via the previously positioned peritoneal catheter. The peritoneal dialysis is carried out on daily basis, at home by the patient, and the ?exchange? is repeated 4-5 times during the 24 hours. The first steps in peritoneal dialysis at the Department for Haemodial

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14

Akimoto, Tetsu, Tomoyuki Yamazaki, Marina Kohara, et al. "Pleuroperitoneal Communication and Ovarian Cancer Complicating Peritoneal Dialysis: A Case Report of a Patient with End-Stage Kidney Disease." Clinical Medicine Insights: Case Reports 10 (January 1, 2017): 117954761773581. http://dx.doi.org/10.1177/1179547617735818.

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Peritoneal dialysis has been a widely accepted modality for treating end-stage kidney disease, but a regular dialysis schedule can be seriously disrupted by various comorbid conditions requiring surgical intervention. A 40-year-old woman who had been receiving peritoneal dialysis was sequentially but separately complicated by pleuroperitoneal communication and ovarian cancer. Despite the need for temporary interruption of her peritoneal dialysis schedule, it was successfully resumed after the relevant surgeries for each disease. Several concerns regarding overall postoperative dialytic managem

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15

Hirahara, Ichiro, Eiji Kusano, Satoru Yanagiba, et al. "Peritoneal Injury by Methylglyoxal in Peritoneal Dialysis." Peritoneal Dialysis International: Journal of the International Society for Peritoneal Dialysis 26, no. 3 (2006): 380–92. http://dx.doi.org/10.1177/089686080602600317.

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Background Peritoneal dialysis (PD) is a common treatment for patients with reduced or absent renal function. Long-term PD leads to peritoneal injury with structural changes and functional decline, such as ultrafiltration loss. At worst, peritoneal injury leads to encapsulating peritoneal sclerosis, a serious complication of PD. Glucose degradation products contained in PD fluids contribute to the bioincompatibility of conventional PD fluids. Methylglyoxal (MGO) is an extremely toxic glucose degradation product. The present study examined the injurious effect of MGO on peritoneum in vivo. Meth

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16

Dinarvand, Peyman, Seyed Mahdi Hassanian Mehr, and Alireza R. Rezaie. "Activated Protein C Prevents Peritoneal Fibrosis." Blood 124, no. 21 (2014): 4229. http://dx.doi.org/10.1182/blood.v124.21.4229.4229.

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Abstract Patients with chronic kidney disease require dialysis (hemodialysis or peritoneal dialysis) for treatment. Peritoneal dialysis is an alternative to hemodialysis for the treatment of end-stage renal disease and is based on the use of the peritoneum as a permeable membrane where ultrafiltration and diffusion between dialysate and blood can take place across the peritoneum. Peritoneal fibrosis is one of the main complications of peritoneal dialysis and affects up to 20% of patients undergoing continuous ambulatory peritoneal fibrosis. The exact mechanism of this process has yet to be elu

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17

Slobodan, Krstić, Trbojević-Stanković Jasna, Žunić Snežana, Jovanović Nataša, and Stojimirović Biljana. "Surgical Technique Using An Improvised Peritoneal Catheter In An Experimental Non-Uremic Rabbit Model Of Peritoneal Dialysis." Acta Veterinaria 65, no. 3 (2015): 319–27. http://dx.doi.org/10.1515/acve-2015-0026.

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AbstractExperimental models have strongly contributed to the comprehension of the processes of peritoneal damage that take place during peritoneal dialysis treatment in human patients. A variety of peritoneal dialysis models have been developed, mostly using rats and rabbits.In this study we present the successful development of a custom-made improvised peritoneal catheter for an experimental non-uremic rabbit model of peritoneal dialysis.A detailed description of the surgical technique of peritoneal catheter implantation, care and removal is provided.This innovative approach to constructing a

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18

Breborowicz, Andrzej, and Dimitrios G. Oreopoulos. "Is Normal Saline Harmful to the Peritoneum?" Peritoneal Dialysis International: Journal of the International Society for Peritoneal Dialysis 25, no. 4_suppl (2005): 67–70. http://dx.doi.org/10.1177/089686080502504s09.

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♦ Background Normal saline (0.9% NaCl) is used during various abdominal surgical interventions and during peritoneal dialysis to rinse the peritoneal cavity. Although no clear clinical evidence exists for the bioincompatibility of normal saline, various experimental studies have suggested that 0.9% NaCl solution can initiate fibrosis of peritoneum. ♦ Material and Methods We review the data derived from in vitro and in vivo experimental studies demonstrating the cytotoxic effect of 0.9% NaCl and its ability to initiate peritoneal adhesions. ♦ Results Normal saline reduces the viability and fibr

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19

Gaggiotti, E., A. Arduini, M. Bonomini, et al. "Prevention of Peritoneal Sclerosis: A New Proposal to Substitute Glucose with Carnitine Dialysis Solution (Biocompatibility Testing in Vitro and in Rabbits)." International Journal of Artificial Organs 28, no. 2 (2005): 177–87. http://dx.doi.org/10.1177/039139880502800215.

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Aim Commercial glucose peritoneal dialysis solutions expose the peritoneum to hyperosmolar glucose containing variable amounts of non-enzymic breakdown products of glucose. These solutions are toxic for the peritoneum. The aim of the present study is to compare in vitro and in vivo characteristics of a new dialysis solution containing carnitine, a naturally occurring compound, as substitute of glucose. Material and Methods We compared in vitro and in the rabbit a new peritoneal dialysis solution containing carnitine, with two standard bicarbonate glucose peritoneal dialysis solutions and a sol

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20

Nakamoto, Hidetomo, Hiroe Imai, Rie Fukushima, Yuji Ishida, Yasuhiro Yamanouchi, and Hiromichi Suzuki. "Role of the Renin–angiotensin System in the Pathogenesis of Peritoneal Fibrosis." Peritoneal Dialysis International: Journal of the International Society for Peritoneal Dialysis 28, no. 3_suppl (2008): 83–87. http://dx.doi.org/10.1177/089686080802803s17.

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⋄ Background Although the effects of angiotensin type 1 receptor blocker (ARB) have been studied, little is known about ARBs in hypertensive patients undergoing dialysis. In the present study, we evaluated the effect of an ARB, olmesartan medoxomil (CS866), on the progression of peritoneal fibrosis in peritoneal dialysis by examining its effect in a model of peritoneal fibrosis in hypertensive rats. ⋄ Materials and Methods W e all ocated 40 male Wistar rats with 2-kidney, 1-clip renovascular hypertension (2K1C-RVH) to 4 groups (each n = 10) that were dialyzed using various solutions for 42 day

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21

ITO, TAKAFUMI, NORIAKI YORIOKA, MASAO YAMAMOTO, KATSUKO KATAOKA, and MICHIO YAMAKIDO. "Effect of Glucose on Intercellular Junctions of Cultured Human Peritoneal Mesothelial Cells." Journal of the American Society of Nephrology 11, no. 11 (2000): 1969–79. http://dx.doi.org/10.1681/asn.v11111969.

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Abstract. During continuous ambulatory peritoneal dialysis, the peritoneum is directly and continuously exposed to unphysiologic peritoneal dialysis fluid; the resulting mesothelial damage has been suggested to cause loss of ultrafiltration and dialysis efficacy. The present study investigated the effect of a high glucose concentration on cultured human peritoneal mesothelial cells to clarify the cause of decreased dialysis efficacy during prolonged peritoneal dialysis. High glucose caused a concentration-dependent decrease in cell proliferation, damage to the intercellular junctions, and exce

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22

Korybalska, Katarzyna, Katarzyna Wieczorowska–Tobis, Alicja Polubinska, et al. "L-2-Oxothiazolidine-4-Carboxylate: An Agent that Modulates Lipopolysaccharide-Induced Peritonitis in Rats." Peritoneal Dialysis International: Journal of the International Society for Peritoneal Dialysis 22, no. 3 (2002): 293–300. http://dx.doi.org/10.1177/089686080202200301.

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Objective L-2-Oxothiazolidine-4-carboxylate (OTZ), a cysteine precursor, is a substrate for intracellular glutathione synthesis. As shown previously, OTZ prevents free-radical induced cellular damage during in vitro simulation of peritoneal dialysis. In the present study, we examined the effect of adding OTZ to peritoneal dialysis solution on peritoneal function and structure during lipopolysaccharide (LPS)-induced peritonitis in rats. In addition, we studied the effects of pretreatment with OTZ (given orally) on the effects of LPS-induced peritonitis in rats. Methods Peritonitis was induced i

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23

Tomo, Tadashi. "Peritoneal Dialysis Solutions Low in Glucose Degradation Products—evidence for Clinical Benefits." Peritoneal Dialysis International: Journal of the International Society for Peritoneal Dialysis 28, no. 3_suppl (2008): 123–27. http://dx.doi.org/10.1177/089686080802803s23.

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In Japan, two types of new peritoneal dialysis fluid (PDF) are ordinarily used: two-chambered PDF, and icodextrin PDF. Two-chambered PDF has several biocompatible characteristics, one being low glucose degradation products (GDPs). Of the several GDPs in PDF, 3,4-dideoxyglucosone-3-ene (3,4-DGE) is thought to be strongly associated with the cytotoxicity of standard PDF. Using a PDF low in GDPs may reduce exposure of the peritoneum to 3,4-DGE, helping to preserve peritoneal function in PD patients. Additionally, use of a PDF low in GDPs may reduce plasma levels of advanced glycosylation end-prod

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Hekking, L. H. P., and J. Van Den Born. "Feasibility of Mesothelial Transplantation during Experimental Peritoneal Dialysis and Peritonitis." International Journal of Artificial Organs 30, no. 6 (2007): 513–19. http://dx.doi.org/10.1177/039139880703000609.

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The mesothelial cell layer lining the peritoneum orchestrates peritoneal homeostasis. Continuous exposure to peritoneal dialysis fluids and episodes of peritonitis may damage the monolayer irreversibly, eventually leading to adhesion formation and fibrosis/sclerosis of the peritoneum. Autologous mesothelial cell transplantation is thought to be one of the options to reduce dysfunction of the peritoneal membrane. In this article we will review the mesothelial cell transplantation experiments performed in the field of peritoneal dialysis and peritonitis. In addition we will focus on the trouble

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VIȚALARU, A. B. "Peritoneal dialysis in dogs and cats." Journal of the Hellenic Veterinary Medical Society 71, no. 4 (2021): 2419. http://dx.doi.org/10.12681/jhvms.25914.

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Dialysis represents the separation process of a colloidal dispersion substance from molecular dispersion particles, based on the property of certain membranes to retain only colloidal particles. In veterinary medicine, the most common use for peritoneal dialysis is the therapy of acute kidney injury, although it can be employed for removing dialyzable toxins and treating pancreatitis, electrolyte disorders and acid-base imbalances, refractory congestive heart failure and metabolic congenital disorders. Peritoneal dialysis is contraindicated in patients with peritoneal adhesions, fibrosis or ab

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Pérez-Martínez, J., FC Pérez-Martínez, B. Carrión, et al. "Aliskiren Prevents the Toxic Effects of Peritoneal Dialysis Fluids during Chronic Dialysis in Rats." Plos One 7, no. 4 (2012): e36268. https://doi.org/10.1371/journal.pone.0036268.

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Abstract The benefits of long-term peritoneal dialysis (PD) in patients with end-stage renal failure are short-lived due to structural and functional changes in the peritoneal membrane. In this report, we provide evidence for the in vitro and in vivo participation of the renin-angiotensin-aldosterone system (RAAS) in the signaling pathway leading to peritoneal fibrosis during PD. Exposure to high-glucose PD fluids (PDFs) increases damage and fibrosis markers in both isolated rat peritoneal mesothelial cells and in the peritoneum of rats after chronic dialysis. In both cases, the addition of th

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Opatrna, Sylvie, Marie Korabečná, Věra Křížková, Zbynek Tonar, Jitka Kočová, and Dana Mullerová. "Adipocytes derived fibrinolytic components in peritoneum — a pilot study." Open Medicine 7, no. 5 (2012): 604–9. http://dx.doi.org/10.2478/s11536-012-0042-8.

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AbstractThe proteins of the fibrinolytic system — urokinase plasminogen activator(uPA), tissue plasminogen activator (tPA)and plasminogen activator inhibitor type IPAI-I) — play important roles in fibrotization in various organs and including peritoneum. To study the cellular localization of PAI-1, tPA and uPA within the adipose tissue of the peritoneal membrane in patients at the onset of peritoneal dialysis(PD) we determined the initial expression of these proteins in relationship to multiple clinical variables. Methods: routinely performed parietal peritoneal biopsies in 12 patients undergo

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Kasuga, Hirotake. "After peritoneal dialysis discontinuation: When will we remove peritoneal dialysis catheter?" Journal of Vascular Access 20, no. 1_suppl (2018): 31–34. http://dx.doi.org/10.1177/1129729817751620.

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Most of the peritoneal dialysis patients stop their peritoneal dialysis therapy and transfer to hemodialysis or kidney transplantation. In Japan, most end-stage kidney disease patients select hemodialysis after peritoneal dialysis discontinuation. Peritoneal dialysis catheter will be removed after stopping peritoneal dialysis. If peritoneal dialysis patients suffer from refractory peritonitis or severe tunnel infection, we remove the peritoneal dialysis catheter immediately. However, the causes of peritoneal dialysis discontinuation are ultrafiltration failure or peritoneal membrane dysfunctio

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Krishnan, Rajesh G., Milos V. Ognjanovic, Jean Crosier, and Malcolm G. Coulthard. "Acute Hydrothorax Complicating Peritoneal Dialysis." Peritoneal Dialysis International: Journal of the International Society for Peritoneal Dialysis 27, no. 3 (2007): 296–99. http://dx.doi.org/10.1177/089686080702700315.

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Aim To determine whether gradually increasing the peritoneal dialysate fill volume from 10 to 40 mL/kg over 6 days, rather than commencing at 40 mL/kg, prevents hydrothorax in children and reverses it if present. Methods A review of children peritoneally dialyzed in a single center. Results During the 20 years beginning June 1985, 416 children were peritoneally dialyzed, of which 327 (79%) had acute and 89 had end-stage renal failure. Among 253 children who had gradually increasing fill volumes, none developed acute hydrothoraces, but 13/163 (8%) who began with 40 mL/kg cycles did ( p < 0.0

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Kazancioglu, Rumeyza. "Peritoneal Defense Mechanisms—the Effects of New Peritoneal Dialysis Solutions." Peritoneal Dialysis International: Journal of the International Society for Peritoneal Dialysis 29, no. 2_suppl (2009): 198–201. http://dx.doi.org/10.1177/089686080902902s40.

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It remains to be determined whether the peritoneal dialysis procedure induces abnormalities in the normal host defenses of the abdominal cavity and whether these perturbations are important in the pathogenesis of peritonitis. The peritoneum is a smooth membrane that lines the abdominal cavity and participates in the diffusion of water and solutes during peritoneal dialysis. The diaphragmatic lymphatic uptake and the opsonization of micro-organisms, with phagocytosis and killing by peritoneal macrophages, mesothelial cells, lymphocytes, polymorphonuclear leukocytes, and newly defined proteins s

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Mushahid, Ali, Farooq Aadil, Ghosh Soumyodhriti, et al. "Peritoneal Dialysis Cannulations-Our Experience." Scholars Journal of Applied Medical Sciences 4, no. 6 (2016): 1878–80. http://dx.doi.org/10.21276/sjams.2016.4.6.2.

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Santoboni, Alberto. "Peritoneal dialysis: Marginal dialysis?" Giornale di Tecniche Nefrologiche e Dialitiche 26, no. 5_suppl (2014): 46–47. http://dx.doi.org/10.5301/gtnd.2014.11915.

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Gabella, Paolo, Francesca Bermond, Cristiana Bagnis, and Martino Marangella. "Peritoneal dialysis: Marginal dialysis." Giornale di Tecniche Nefrologiche e Dialitiche 26, no. 5_suppl (2014): 44–45. http://dx.doi.org/10.5301/gtnd.2014.11966.

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Pérez-Díaz, Vicente, Alfonso Pérez-Escudero, Sandra Sanz-Ballesteros, et al. "A New Method to Increase Ultrafiltration in Peritoneal Dialysis: Steady Concentration Peritoneal Dialysis." Peritoneal Dialysis International: Journal of the International Society for Peritoneal Dialysis 36, no. 5 (2016): 555–61. http://dx.doi.org/10.3747/pdi.2016.00007.

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Background Peritoneal dialysis (PD) has limited power for liquid extraction (ultrafiltration), so fluid overload remains a major cause of treatment failure. Methods We present steady concentration peritonal dialysis (SCPD), which increases ultrafiltration of PD exchanges by maintaining a constant peritoneal glucose concentration. This is achieved by infusing 50% glucose solution at a constant rate (typically 40 mL/h) during the 4-hour dwell of a 2-L 1.36% glucose exchange. We treated 21 fluid overload episodes on 6 PD patients with high or average-high peritoneal transport characteristics who

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Kunin, Margarita, and Pazit Beckerman. "The Peritoneal Membrane—A Potential Mediator of Fibrosis and Inflammation among Heart Failure Patients on Peritoneal Dialysis." Membranes 12, no. 3 (2022): 318. http://dx.doi.org/10.3390/membranes12030318.

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Peritoneal dialysis is a feasible, cost-effective, home-based treatment of renal replacement therapy, based on the dialytic properties of the peritoneal membrane. As compared with hemodialysis, peritoneal dialysis is cheaper, survival rate is similar, residual kidney function is better preserved, fluid and solutes are removed more gradually and continuously leading to minimal impact on hemodynamics, and risks related to a vascular access are avoided. Those features of peritoneal dialysis are useful to treat refractory congestive heart failure patients with fluid overload. It was shown that in

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Redmond, Avril, and Elizabeth Doherty. "Peritoneal dialysis." Nursing Standard 19, no. 40 (2005): 55–66. http://dx.doi.org/10.7748/ns.19.40.55.s55.

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Redmond, Avril, and Elizabeth Doherty. "Peritoneal dialysis." Nursing Standard 19, no. 40 (2005): 55–65. http://dx.doi.org/10.7748/ns2005.06.19.40.55.c3893.

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Gould, Caroline. "Peritoneal dialysis." Nursing Standard 20, no. 3 (2005): 67–68. http://dx.doi.org/10.7748/ns.20.3.67.s73.

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Gokal, Ram. "Peritoneal Dialysis." Drugs & Aging 17, no. 4 (2000): 269–82. http://dx.doi.org/10.2165/00002512-200017040-00003.

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Teitelbaum, Isaac. "Peritoneal Dialysis." New England Journal of Medicine 385, no. 19 (2021): 1786–95. http://dx.doi.org/10.1056/nejmra2100152.

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Fung, Winston Wing-Shing, Jack Kit-Chung Ng, and Philip Kam-Tao Li. "Peritoneal Dialysis." Nephrology Self-Assessment Program 20, no. 1 (2021): 19–34. http://dx.doi.org/10.1681/nsap.2021.20.1.2.

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Taskapan, Hulya, Olof Heimburger, Cengiz Utas, and Paul Tam. "Peritoneal Dialysis." International Journal of Nephrology 2011 (2011): 1. http://dx.doi.org/10.4061/2011/218974.

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Twardowski, Zbylut J. "Peritoneal dialysis." Postgraduate Medicine 85, no. 5 (1989): 161–82. http://dx.doi.org/10.1080/00325481.1989.11700663.

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Lum, Gary M. "Peritoneal dialysis." Critical Care Medicine 27, no. 11 (1999): 2595–96. http://dx.doi.org/10.1097/00003246-199911000-00058.

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Kratochwill, K., M. Boehm, R. Herzog, et al. "PERITONEAL DIALYSIS." Nephrology Dialysis Transplantation 29, suppl 3 (2014): iii16—iii18. http://dx.doi.org/10.1093/ndt/gfu114.

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HATHAWAY, LISA. "Peritoneal dialysis." Nursing Made Incredibly Easy! 2, no. 5 (2004): 55–58. http://dx.doi.org/10.1097/00152258-200409000-00011.

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Chaimovitz, Cidio. "Peritoneal dialysis." Kidney International 45, no. 4 (1994): 1226–40. http://dx.doi.org/10.1038/ki.1994.163.

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&NA;. "PERITONEAL DIALYSIS." ASAIO Journal 42, no. 2 (1996): 102–4. http://dx.doi.org/10.1097/00002480-199642020-00022.

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Choi, Peter, and Edwina A. Brown. "Peritoneal Dialysis." Medicine 31, no. 6 (2003): 70–73. http://dx.doi.org/10.1383/medc.31.6.70.28314.

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Padua, Ryan Michael Z. "Peritoneal Dialysis." MEDSURG Nursing 31, no. 2 (2022): 127. http://dx.doi.org/10.62116/msj.2022.31.2.127.

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