Bibliografie tematiche / PF4

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Letteratura scientifica selezionata sul tema "PF4"

Autore: Grafiati

Pubblicato: 4 giugno 2021

Ultima modifica: 11 marzo 2023

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Articoli di riviste sul tema "PF4"

Esan, A. M., Z. Khan, R. Kiran, T. O. Omolekan, K. A. Aremu, and H. R. Y. Adeyemi. "Ameliorative Effects of Pseudomonas fluorescence Strains on Growth and Antioxidant Potential of Okra (Abelmoschus esculentus) Plant under Nematode Infection." European Journal of Biology and Biotechnology 2, no. 3 (June 5, 2021): 50–56. http://dx.doi.org/10.24018/ejbio.2021.2.3.168.

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Meloidogyne incognita is a plant pathogen causing root-knot nematodes disease in many crops worldwide. Due to the environmental threat on the use of chemical fumigants, there is a need for a biological control method using microbial antagonists on root-knot nematodes disease. Therefore, this study was conducted to screen and evaluate the biocontrol potential of P. fluorescens strains against root-knot nematodes. The effectiveness of six P. fluorescens strains viz., Pf1, Pf2, Pf3, Pf4, Pf5and Pf6 were tested in vitro and also in pots experiment for their inhibitory activities and biocontrol pot

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González-Serrano, Diego J., Milad Hadidi, Matin Varcheh, Aniseh Zarei Jelyani, Andres Moreno, and Jose M. Lorenzo. "Bioactive Peptide Fractions from Collagen Hydrolysate of Common Carp Fish Byproduct: Antioxidant and Functional Properties." Antioxidants 11, no. 3 (March 6, 2022): 509. http://dx.doi.org/10.3390/antiox11030509.

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Collagen isolated from byproducts of common carp was hydrolyzed with alcalase enzyme to obtain peptide fractions. The resulting >30 kDa (PF1), 10–30 kDa (PF2), 3–10 kDa (PF3) and <1 kDa (PF4) fractions were studied for their antioxidant and functional properties. All peptide fractions illustrated antioxidant activity at different concentrations (1, 5, and 10 mg/mL). Although PF4 indicated the highest DPPH radical-scavenging activity (87%) at a concentration of 1 mg/mL, the highest reducing power (0.34) and hydroxyl radical scavenging activity (95.4%) were also observed in PF4 at a concen

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Kakkar, Preeti Handa, R. M. Saxena, and Mamta Joshi. "Histopathological analysis of liver in Puntius ticto exposed to water soluble fraction (WSF) of petrol." Journal of Applied and Natural Science 2, no. 2 (December 1, 2010): 290–92. http://dx.doi.org/10.31018/jans.v2i2.136.

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The fresh water fish Puntius ticto were exposed to lethal concentration of water soluble fraction (WSF) of petrol (5%-PF1, 10%-PF2, 15%-PF3, 20%-PF4 and 25%-PF5) for 96 hours. The exposure of WSF produced some conspicuous histopathological changes in liver. The swelling of hepatocytes, degeneration, necrosis, hemolysis, dilation, congestion and fibrosis in blood sinusoids were the prominent changes observed. The histological analysis showed increasing damages dose-dependents and time-dependents.

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FELLOWES, R. A., A. G. MAULE, N. J. MARKS, T. G. GEARY, D. P. THOMPSON, and D. W. HALTON. "Nematode neuropeptide modulation of the vagina vera of Ascaris suum: in vitro effects of PF1, PF2, PF4, AF3 and AF4." Parasitology 120, no. 1 (January 2000): 79–89. http://dx.doi.org/10.1017/s0031182099005260.

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Ascaris suum possesses a large number of FMRFamide-related peptides (FaRPs) of which KNEFIRFamide (AF1), KHEYLRFamide (AF2) and KSAYMRFamide (AF8/PF3) have been shown to modulate the intrinsic, rhythmic activity of the vagina vera of A. suum in vitro. In the present study, the effects of the nematode FaRPs, SDPNFLRFamide (PF1), SADPNFLREamide (PF2) and KPNFIRFamide (PF4) (from Panagrellus redivivus) and AVPGVLRFamide (AF3) and GDVPGVLRFamide (AF4) (from A. suum) on the in vitro activity of the vagina vera were examined. The effects of each of the peptides were qualitatively and quantitatively

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Lecomte-Raclet, Laurence, Mònica Alemany, Anabelle Sequeira-Le Grand, Jean Amiral, Gérard Quentin, Anne Marie Vissac, Jacques P. Caen, and Zhong Chao Han. "New Insights Into the Negative Regulation of Hematopoiesis by Chemokine Platelet Factor 4 and Related Peptides." Blood 91, no. 8 (April 15, 1998): 2772–80. http://dx.doi.org/10.1182/blood.v91.8.2772.2772_2772_2780.

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Platelet factor 4 (PF4) has been recognized as an inhibitor of myeloid progenitors. However, the mechanism of action of this chemokine remains poorly understood. The present study was designed to determine its structure/function relationship. A series of peptides overlapping the C-terminal and central regions of PF4 were analyzed in vitro for their action on murine hematopoietic progenitor growth to assess the minimal sequence length required for activity. The peptides p17-58 and p34-58 possessed an increased hematopoietic inhibitory activity when compared with PF4, whereas the shorter peptide

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Karuppaija, Sinthuja, Kapilan Ranganathan, and Vasantharuba Seevaratnam. "Characterization of best naringinase producing fungus strain isolated from palmyrah (Borrasus flabellifer) fruit pulp." International Journal of Biological Research 4, no. 2 (July 19, 2016): 97. http://dx.doi.org/10.14419/ijbr.v4i2.6289.

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Background: The Palmyrah (Borrasus flabellifer L.) fruit pulp has the bitter compound flabelliferin (a tetraglycoside) which can be hydrolyzed by naringinase enzyme. The diverse groups of filamentous fungi and bacteria that live in different substrates have the capacity of producing extracellular naringinase enzyme which is of tremendous industrial value.Objective: The objective of the study was to isolate the naringinase producing fungal strains from Palmyrah and to identify the best naringinase producer under liquid and solid state fermentation systems.Methods: Fungal strains isolated from P

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PURCELL, J., A. P. ROBERTSON, D. P. THOMPSON, and R. J. MARTIN. "The time-course of the response to the FMRFamide-related peptide PF4 in Ascaris suum muscle cells indicates direct gating of a chloride ion-channel." Parasitology 124, no. 6 (June 2002): 649–56. http://dx.doi.org/10.1017/s0031182002001695.

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We investigated the effects of PF4 on Ascaris suum somatic muscle cells using a 2 electrode current-clamp technique. PF4 is a FaRP (FMRFamide-related peptide), originally isolated from the free-living nematode Panagrellus redivivus. PF4 caused hyperpolarization and an increase in chloride ion conductance when it was applied to the muscle cells of the Ascaris body wall. The delay between the application of the peptide and the appearance of the response was measured and compared with that of gamma-amino butyric acid (GABA), a compound that directly gates ion channels, and with PF1, a FaRP that a

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Bokka, Chandra Sekhar, Ganesh Kumar Veeramachaneni, V. B. S. C. Thunuguntla, Janakiram Bobbillapati, and Jayakumar Singh Bondili. "Peptide Mapping, In Silico and In Vivo Analysis of Allergenic Sorghum Profilin Peptides." Medicina 55, no. 5 (May 21, 2019): 178. http://dx.doi.org/10.3390/medicina55050178.

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Background and objectives: Nearly 20–30% of the world’s population suffers from allergic rhinitis, among them 15% are progressing to asthma conditions. Sorghum bicolor profilin (Sorb PF), one of the panallergens, was identified, but the allergen specificity is not yet characterized. Materials and Methods: To map the antigenic determinants responsible for IgE binding, the present study is focused on in silico modeling, simulation of Sorb PF and docking of the Sorb PF peptides (PF1-6) against IgG and IgE, followed by in vivo evaluation of the peptides for its allergenicity in mice. Results: Pept

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Ismail, Muhammad Hafiz, Katharine A. Michie, Yu Fen Goh, Parisa Noorian, Staffan Kjelleberg, Iain G. Duggin, Diane McDougald, and Scott A. Rice. "The Repressor C Protein, Pf4r, Controls Superinfection of Pseudomonas aeruginosa PAO1 by the Pf4 Filamentous Phage and Regulates Host Gene Expression." Viruses 13, no. 8 (August 15, 2021): 1614. http://dx.doi.org/10.3390/v13081614.

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It has been shown that the filamentous phage, Pf4, plays an important role in biofilm development, stress tolerance, genetic variant formation and virulence in Pseudomonas aeruginosa PAO1. These behaviours are linked to the appearance of superinfective phage variants. Here, we have investigated the molecular mechanism of superinfection as well as how the Pf4 phage can control host gene expression to modulate host behaviours. Pf4 exists as a prophage in PAO1 and encodes a homologue of the P2 phage repressor C and was recently named Pf4r. Through a combination of molecular techniques, ChIPseq an

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Hara, Francisco Adilson dos Santos, and Luiz Antonio de Oliveira. "Características fisiológicas e ecológicas de isolados de rizóbios oriundos de solos ácidos e álicos de Presidente Figueiredo, Amazonas." Acta Amazonica 34, no. 3 (September 2004): 343–57. http://dx.doi.org/10.1590/s0044-59672004000300002.

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Alguns isolados de rizóbio, além de fixarem o N2, são capazes de solubilizar fosfatos pouco solúveis, disponibilizando o P para as plantas e para si mesmos. No entanto, o Al e a acidez dos solos da Amazônia podem diminuir a população desses microrganismos. O presente trabalho avaliou a capacidade nodulífera, a tolerância à acidez e ao Al tóxico, bem como a capacidade de solubilizar fosfatos de Ca e de Al de 88 isolados de rizóbio de solos agrícolas, do município de Presidente Figueiredo, AM. Amostras de solo sob cultivos agrícolas foram coletadas e utilizadas como fontes de inóculo para planta

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Tesi sul tema "PF4"

Brousseau-Nault, Mathieu. "Chronic periodontitis is associated with platelet factor 4 (PF4) secretion." Thesis, University of British Columbia, 2016. http://hdl.handle.net/2429/59016.

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Aim: Platelets contribute to chronic inflammation but their role in periodontitis is not well understood. The aim of this study was to compare platelet recruitment and activation in healthy and inflamed periodontium. Materials and Methods: Gingival crevicular fluid (GCF) samples were obtained from sites of healthy periodontium, gingivitis and periodontitis. Platelets were quantified in the GCF by staining and microscopy. GCF concentrations of platelet factor 4 (PF4) [PF4]GCF and glycoprotein IIbIIIa ([GPIIbIIIa]GCF) were determined by ELISA. Blood samples were obtained from the 3 patient gro

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XI, XIAODONG. "Role du pf4 et de l'il-13 dans la regulation de l'hematopoiese." Paris 7, 1995. http://www.theses.fr/1995PA077261.

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De nombreux regulateurs (positifs ou negatifs) sont impliques dans la regulation de l'hematopoiese. Deux de ces regulateurs, le pf4 et l'il-13, font l'objet de cette these. Le chapitre i presente les techniques utiles developpees pour la realisation du travail et les resultats obtenus a partir de ces techniques. Le chapitre ii porte sur les effets differenciateurs de l'il-13 sur la croissance des progeniteurs hematopoietiques: stimulation de la lignee megacaryocytaire et inhibition de la lignee granulomacrophagique. C'est un rapport original concernant l'effet de l'il-13 sur les megacaryocytes

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Purcell, Jenny. "The effects of PF4 on Ascaris suum somatic muscle cells : an electrophysiological study." Thesis, University of Edinburgh, 2001. http://hdl.handle.net/1842/29954.

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PF4 is a FaRP which was isolated from the free-living nematode Panagrellus redivivus and which has been shown to have activity in other nematodes, including Ascaris suum. A. suum, the large parasitic nematode found in domestic pigs, is often used as an experimental model in studying nematodes due to its large size and easy availability. The nematodes used in this study were obtained from three different sources. The action of PF4 on the somatic muscle cells of A.suum was investigated using the two electrode current clamp technique. PF4 caused these cells to hyperpol

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Dubrac, Alexandre. "Analyse fonctionnelle et structurale du facteur antiangiogénique pf4v1." Thesis, Bordeaux 1, 2008. http://www.theses.fr/2008BOR13751/document.

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De nombreuses équipes se sont intéressées aux fonctions antiangiogéniquesde PF4 (Platelet Factor 4 ou Facteur Plaquettaire 4). Ses capacités inhibitrices vis-àvisde la prolifération et de la migration des cellules endothéliales in vitro et son effetinhibiteur sur lʼangiogenèse in vivo ne sont plus à démontrer. En revanche, il existeencore de nombreuses interrogations sur les mécanismes dʼaction responsables deson activité antiangiogénique. La chimiokine PF4v1 (Platelet Factor 4 variant 1)mature ne diverge de PF4 que par trois acides aminés mais son potentielangiostatique est beaucoup plus élev

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El, Golli Nargès. "Etude du trafic intracellulaire de la protéine alpha-granulaire plaquettaire : le facteur plaquettaire (PF4)." Paris 7, 2005. http://www.theses.fr/2005PA077136.

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Lazar, Noureddine. "Structure secondaire et activité biologique : relations structure-fonction dans le PF4 Humain et la Prosomatostatine." Paris 6, 2003. http://www.theses.fr/2003PA066181.

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Pertuy, Fabien. "Etude des mécanismes de formation des plaquettes sanguines : rôle de l'environnement médullaire." Thesis, Strasbourg, 2014. http://www.theses.fr/2014STRAJ092/document.

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Les mécanismes de formation des plaquettes sanguines à partir des mégacaryocytes ne sont pas totalement compris, mais l’environnement médullaire semble y avoir une influence cruciale. Dans ce travail nous montrons que i) les intégrines β3, récepteurs de protéines de matrice extracellulaire, semblent impliquées dans la mégacaryopoïèse et la formation des plaquettes, ii) la différenciation des cellules hématopoïétiques dans un environnement 3D de rigidité comparable à la moelle osseuse améliore la maturation des mégacaryocytes différenciés in vitro et iii) la myosine IIA est impliquée dans la di

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Schulze, Annika [Verfasser]. "Charakterisierung der anti-PF4/Heparin-Antikörper-sezernierenden B-Zellen im Mausmodell nach systemischer Immunisierung / Annika Schulze." Greifswald : Universitätsbibliothek Greifswald, 2015. http://d-nb.info/1068082143/34.

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AIDOUDI, SALLOUHA. "Effet protecteur du facteur plaquettaire 4 (pf4) en comparaison avec un myeloprotecteur, le tetrapeptide acsdkp, sur l'hematopoiese de souris traitees par la chimiotherapie." Paris 7, 1997. http://www.theses.fr/1997PA077354.

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Le pf4 est une proteine synthetisee par la lignee megacaryocytes/plaquettes dont l'activite inhibitrice a ete recemment montree sur la lignee megacaryocytaire humaine et murine. Il a ete montre que l'effet du pf4 est predominant sur la lignee megacaryocytaire, mais a plus forte doses, il inhibe aussi la proliferation des autres lignees hematopoietiques. Le but de ce travail a consiste a etudier l'effet du pf4, en comparaison avec un inhibiteur et myeloprotecteur connu de l'hematopoiese l'acsdkp, sur l'hematopoiese des souris traitees avec deux chimiotherapies : le 5-fu ou ara-c. Les resultats

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Libraire, Julie. "Le facteur 4 plaquettaire (PF4/CXCL4) prévient la formation du complexe initial de l’inhibiteur de l’activateur du plasminogène (PAI-1) avec sa cible d’origine tissulaire (t-PA)." Thesis, Paris 5, 2012. http://www.theses.fr/2012PA05P654.

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Le facteur 4 plaquettaire (PF4/CXCL4) est un tétramère constitué de quatre sous-unités identiques de 7,8 kDa qui est libéré en grande quantité par les plaquettes lors de l’hémostase primaire (ensemble des phénomènes permettant un colmatage initial d’une lésion vasculaire). L’étude de la formation d’un caillot de fibrine en présence de PF4 montre une augmentation de la turbidité finale du caillot : le PF4 modifie le réseau formé. Etant donné que la plupart des acteurs de la fibrinolyse se lie au caillot de fibrine et que le PF4 modifie sa structure, nous avons pensé qu’il serait intéressant de

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Libri sul tema "PF4"

Borčić, Barbara. P74 20 years: P74 20 let. Ljubljana: Zavod P.A.R.A.S.I.T.E., 2020.

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Fieled, Adam. Gauntlet: (PFS). Edited by Adam Fieled. Conshohocken, Pa: Internet Archive, 2013.

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O PFL. São Paulo, SP: Publifolha, 2001.

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Fieled, Adam. Gauntlet: (PFS). Edited by Adam Fieled. Conshohocken, Pa: Internet Archive, 2013.

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Fieled, Adam. Gauntlet: (PFS). Edited by Adam Fieled. Conshohocken, Pa: Internet Archive, 2013.

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Fieled, Adam. Gauntlet: (PFS). Edited by Adam Fieled. Conshohocken, Pa: Internet Archive, 2013.

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Fieled, Adam. Gauntlet: (PFS). Conshohocken, Pa: Internet Archive, 2013.

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Crowley, W. Robert. Using the IBM PC: PFS,file/PFS,report. New York: Holt, Rinehart and Winston, 1985.

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Terry, Neville, Association of Chartered Certified Accountants., and Certified Accountants Educational Trust, eds. PFI: Practical perspectives. London: Association of Chartered Certified Accountants for the Certified Accountants Educational Trust, 2002.

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Labour Research Department. Profiting from PFI. London: UNISON, 2003.

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Capitoli di libri sul tema "PF4"

Stöcker, W., and C. Krüger. "Antikörper gegen Heparin/PF4." In Lexikon der Medizinischen Laboratoriumsdiagnostik, 1–2. Berlin, Heidelberg: Springer Berlin Heidelberg, 2017. http://dx.doi.org/10.1007/978-3-662-49054-9_234-1.

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Stöcker, W., and C. Krüger. "Antikörper gegen Heparin/PF4." In Springer Reference Medizin, 157–58. Berlin, Heidelberg: Springer Berlin Heidelberg, 2019. http://dx.doi.org/10.1007/978-3-662-48986-4_234.

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Abbadessa, V., P. Citarrella, R. Perricone, and P. Di Marco. "β-TG and PF4 in the Diagnosis of Thrombosis." In Advances in Hemostasis and Thrombosis, 315–21. Boston, MA: Springer US, 1985. http://dx.doi.org/10.1007/978-1-4615-9424-6_30.

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Gass, Saul I., and Carl M. Harris. "PFI." In Encyclopedia of Operations Research and Management Science, 614. New York, NY: Springer US, 2001. http://dx.doi.org/10.1007/1-4020-0611-x_751.

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Gooch, Jan W. "PFA." In Encyclopedic Dictionary of Polymers, 528. New York, NY: Springer New York, 2011. http://dx.doi.org/10.1007/978-1-4419-6247-8_8615.

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Donato, Dominique M., Steven K. Hanks, Kenneth A. Jacobson, M. P. Suresh Jayasekara, Zhan-Guo Gao, Francesca Deflorian, John Papaconstantinou, et al. "PFT." In Encyclopedia of Signaling Molecules, 1364. New York, NY: Springer New York, 2012. http://dx.doi.org/10.1007/978-1-4419-0461-4_101022.

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Braun-Falco, Markus, Henry J. Mankin, Sharon L. Wenger, Markus Braun-Falco, Stephan DiSean Kendall, Gerard C. Blobe, Christoph K. Weber, et al. "PFO." In Encyclopedia of Molecular Mechanisms of Disease, 1607. Berlin, Heidelberg: Springer Berlin Heidelberg, 2009. http://dx.doi.org/10.1007/978-3-540-29676-8_5128.

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Braun-Falco, Markus, Henry J. Mankin, Sharon L. Wenger, Markus Braun-Falco, Stephan DiSean Kendall, Gerard C. Blobe, Christoph K. Weber, et al. "PFO." In Encyclopedia of Molecular Mechanisms of Disease, 1633. Berlin, Heidelberg: Springer Berlin Heidelberg, 2009. http://dx.doi.org/10.1007/978-3-540-29676-8_8674.

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Villars, P., K. Cenzual, J. Daams, R. Gladyshevskii, O. Shcherban, V. Dubenskyy, V. Kuprysyuk, and I. Savysyuk. "In2Ti6(PO4)6[Si2O(PO4)6]." In Structure Types. Part 9: Space Groups (148) R-3 - (141) I41/amd, 71. Berlin, Heidelberg: Springer Berlin Heidelberg, 2010. http://dx.doi.org/10.1007/978-3-642-02702-4_33.

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Villars, P., K. Cenzual, J. Daams, R. Gladyshevskii, O. Shcherban, V. Dubenskyy, N. Melnichenko-Koblyuk, et al. "Tl3[PO4]." In Structure Types. Part 5: Space Groups (173) P63 - (166) R-3m, 55. Berlin, Heidelberg: Springer Berlin Heidelberg, 2007. http://dx.doi.org/10.1007/978-3-540-46933-9_10.

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Atti di convegni sul tema "PF4"

Rucinski, B., G. J. Stewart, G. Boden, and S. Niewiarowski. "INTERACTION OF PLATELET FACTOR 4 WITH HEPATOCYTES AND ITS POSSIBLE SIGNIFICANCE IN HEMOSTASIS." In XIth International Congress on Thrombosis and Haemostasis. Schattauer GmbH, 1987. http://dx.doi.org/10.1055/s-0038-1643500.

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Previous experiments have demonstrated the rapid clearance of himan platelet factor 4 (PF4) from rabbit and rat blood, its accumulation in the liver and its elimination of PF4 degradation products with urine. Injection of heparin resulted in a rapid loss of 125 i-pF4-radioactivity from the liver (Rucinski et al Amer. J. Physiol. 251, H800, 1986). Current experiments demonstrate the uptake of human 125i-pf4 by hepatocytes reaching maximum at 180 min. This uptake is 2-3 times greater at 37°C than at 4°C. At 37°C degradation of 125I-PF4 by hepatocytes was also observed as indicated by the increas

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Morrison, J. EJ, A. B. Latif, C. Mason, P. Bramley, and T. R. Criag. "THE PROFILE OF IN VIVO PLATELET ACTIVATION IN NOCTURNAL ASTHMA." In XIth International Congress on Thrombosis and Haemostasis. Schattauer GmbH, 1987. http://dx.doi.org/10.1055/s-0038-1643495.

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The precise pathophysiology of nocturnal asthma still remains to be elucidated. Activated platelets have the ability to release potent broncho- and vaso-constrictors and therefore, have been implicated in asthma. However, there is no information on the status of in vivo platelet activation in patients with nocturnal asthmaIn a randomised controlled study five healthy volunteers and five asthmatics were investigated during a period of 24h after acclimatisation for one day. Both peak flow rate (PFR) and blood samples were obtained at 4 hourly intervals. Plasma levels of platelet factor 4 (PF4) a

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Gewirtz, A., W. Y. Xu, B. Rucinski, and S. Niewiarowski. "SELECTIVE INHIBITION OF HUMAN MEGAKARYOCYTOPOIESIS IN VITRO BY HIGHLY PURIFIED PLATELET FACTOR 4." In XIth International Congress on Thrombosis and Haemostasis. Schattauer GmbH, 1987. http://dx.doi.org/10.1055/s-0038-1644621.

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Platelet (plt) factor 4 (PF4) is an alpha granule protein which can modulate T lymphocyte function. T cells may help regulate megakaryocytopoiesis. Therefore, we hypothesized that T cell-PF4 interactions might play a role in autoregulating marrow megakaryocyte (MEG) production. To test this idea, we studied MEG colony formation in plasma clot cultures containing human serum derived solely from pit poor normal AB plasma, enriched hematopoietic progenitor cells (HPC), autologous T cells, and exogenous PF4. Highly purified PF4 (single band on SDS gel) was prepared from outdated human pits by a co

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Celia, G., M. Prosdocimi, and A. A. Sasahara. "IN VIVO INTERACTION BETWEEN HUMAN PLATELET FACTOR 4 (PF4) AND PROTAMINE SULPHATE (PS) IN THE PRESENCE OF DIFFERENT GLYCOSAMI-NOGLYCANS (GAGs)." In XIth International Congress on Thrombosis and Haemostasis. Schattauer GmbH, 1987. http://dx.doi.org/10.1055/s-0038-1643501.

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Anti-heparin substances, like PF4 or PS, have been studied largely in reference to their ability to neutralize the anticoagulant activity of heparin. On the other hand, few data are available concerning the relationship between GAGs and anti-heparin proteins clearance. We studied the action of PS on human PF4 kinetics in anesthetized rabbits pre-treated with heparin (H, 1000 I.U.), heparan sulphate (HS, 30 mg) and derma tan sulphate (DS, 30 mg). PF4 (given at a dose of 45 μgAg) disappearance reflected its different affinity for the GAGs, with the following half lives (min): control 2.09±0.28,

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Boschetti, C., A. Vicari, E. Cofrancesco, A. Della Volpe, G. Moreo, E. Po-gliani, G. Pozza, and E. Polli. "HEPARIN-RELEASED PLATELET FACTOR 4 (HR-PF4) IN DIABETIC MICR0VAS-CULAR DISEASE." In XIth International Congress on Thrombosis and Haemostasis. Schattauer GmbH, 1987. http://dx.doi.org/10.1055/s-0038-1643498.

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When heparin is injected i.v. as a bolus, PF4 but not (β-throm boglobulin ((βTG) is released immediately. HR-PF4 is not liberated from platelets but from the endothelial cells of vessels which serve as storage sites. The role of platelet activation in diabetic microvascular disease is still controversial, however there is experimental evidence of vascular injury and hemostatic activation preceding the appearance of microvascular disease. The contradictory results so far obtained in man may be partly attributed to the heterogeneity of the diabetic patients studied. We studied 20 insulin-depende

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Liu, Chenglian, Kun Lu, Chen Liu, Qingqing Du, Yujun Dong, Jun Wang, Kaizhong Ding, Xiaoshi Tang, Xiongyi Huang, and Yuntao Song. "ITER PF4 CFT 80 K Cold Test at ASIPP." In 2018 IEEE International Conference on Applied Superconductivity and Electromagnetic Devices (ASEMD). IEEE, 2018. http://dx.doi.org/10.1109/asemd.2018.8558989.

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Kotze, H. F., P. N. Badenhorst, A. du, P. Heyns, P. Heyns, and V. van Wyk. "THE EFFECT OF ARTERIAL DE-ENDOTHELIALIZATION ON IN VIVO AND EX VIVO PLATELET FUNCTION TESTS IN THE BABOON." In XIth International Congress on Thrombosis and Haemostasis. Schattauer GmbH, 1987. http://dx.doi.org/10.1055/s-0038-1643950.

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The effect of endothelial cell injury on platelet function was evaluated in eight baboons with a normal vasculature. Endothelium was removed with a balloon catheter. On each baboon, the left carotid artery was de-endothelialized on day 0 and the right carotid artery and abdominal aorta on day 7. Mean platelet density (MPD; gm/ml), circulating platelet aggregate ratio (CPA), the number of electron-dense bodies per platelet (DBP) , and the plasma concentration (ng/ml) of platelet factor 4 x(PF4) and beta thromboglobulin (bTG) were determined on day -7 (baseline). These ex vivo measurements were

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Teruya, J., N. Shimizu, J. Matsuda, M. Kazama, and T. Abe. "PROFILE DIAGNOSIS OF HEMOSTATIC DISORDERS BY SIMULTANEOUS ASSAY OF HEMOSTATIC MOLECULAR MARKERS." In XIth International Congress on Thrombosis and Haemostasis. Schattauer GmbH, 1987. http://dx.doi.org/10.1055/s-0038-1643052.

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Simultaneous measurements of molecular markers of platelet, coagulation, fibrinolysis, and vascular system give us precise-and comprehensive information about hemostatic profile of various diseases. We adopted 6-thromboglobulin(B-TG) and platelet factor 4(PF4) for platelet function, fibrinopeptide A(FPA) and soluble fibrin monomer complex(SFMC) for coagulation, fibrinopeptide BB15-42(BB) and fibrin degradation product(FDP), for fibrinolysis, and tissue plasminogen activator(TPA) antigen for vascular system.(l).The results of normal values were as follow(n=20); 6-TG 40.6ng/ml, PF4 9.9ng/ml, FPA

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Gralnick, H. R., L. M. Magurder, K. Hansmann, M. Vail, G. Marti, R. McEver, and S. Williams. "THE SURFACE EXPRESSION OF ALPHA GRANULE PROTEINS FOLLOWING THROMBIN STIMULATION." In XIth International Congress on Thrombosis and Haemostasis. Schattauer GmbH, 1987. http://dx.doi.org/10.1055/s-0038-1643859.

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We have studied the platelet glycoproteins (GP) GPIb and the GPIIb/IIIa and the expression of alpha granule proteins (AGP) on the platelet (P) surface following thrombin (T) stimulation. The platelets were separated from plasma proteins on a arabinogalactan gradient. The P were stimulated with purified alpha T 0.1u/108p. Either monospecific polyclonal or murine monoclonal antibodies were used to detect the P glycoprotein and AGP. The platelets were analyzed on an EPICS V Flow Cytometer. Resting P had small amounts of AGP (2-8%) present on their surface. Within 1-3 min. after T stimulation sign

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Fujii, S., and T. Kariya. "PLATELET FUNCTION AND LIPOPROTEINS IN PATIENTS WITH HYPOTHYROIDISM." In XIth International Congress on Thrombosis and Haemostasis. Schattauer GmbH, 1987. http://dx.doi.org/10.1055/s-0038-1643474.

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Platelet function and serum lipoprotein levels were studied in ten patients (two males and eight females) with hypothyroidism. Platelet aggregation and ATP release were determined by Lumi-aggregometer using ADP , collagen and epinephrine as stimulants. Platelet factor 4 (PF4) and thromboxane B2 (TXB2) were determined by radioimmunoassay. High density lipoproteincholesterol (HDL-C) was determined by heparin-manga-nese method. HDL subfractions were separated by gradient gel electrophoresis (PAA 4/30). Apolipopro-teins were measured by single radial immunodiffusion. Platelet aggregation increased

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Rapporti di organizzazioni sul tema "PF4"

Drypolcher, Katherine Carr. Pu Workers in PF4. Office of Scientific and Technical Information (OSTI), April 2019. http://dx.doi.org/10.2172/1511208.

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Schanken, Mary D., and John W. Taylor. Evaluation Report of Sytek PFX A2000 and PFX A2100. Fort Belvoir, VA: Defense Technical Information Center, November 1986. http://dx.doi.org/10.21236/ada208048.

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Aftanas, B. L. PFP solution stabilization. Office of Scientific and Technical Information (OSTI), April 1996. http://dx.doi.org/10.2172/362484.

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Chen, Cheng-Che, and Hao-En Lin. Survival Benefits and Bleeding Risk of Anti-VEGF Agents for Renal Cell Carcinoma (RCC): A Updated Systematic Review and Meta-Analysis of Phase 2 and 3 Randomized Clinical Trials. INPLASY - International Platform of Registered Systematic Review and Meta-analysis Protocols, March 2023. http://dx.doi.org/10.37766/inplasy2023.3.0007.

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Review question / Objective: To investigate the survival benefits (PFS, DFS, OS) and bleeding risk of the anti-VEGF agents compared with placebo or interferon alpha (IFNa) in patients with RCC. Condition being studied: Part 1. The hazard ratio (HR) of the progression-free survival (PFS) and overall survival (OS) of anti-VEGF agents vs. non/placebo for patients with unresectable, advanced, metastatic, renal cell carcinoma (RCC). Part 2. The HR of the disease-free survival (PFS) and OS of anti-VEGF agents vs. non/placebo for patients with post-nephrectomy RCC (adjuvant use). Part 3. The HR of th

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Price, Roz. Climate Adaptation: Lessons and Insights for Governance, Budgeting, and Accountability. Institute of Development Studies (IDS), December 2020. http://dx.doi.org/10.19088/k4d.2022.008.

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This rapid review draws on literature from academic, policy and non-governmental organisation sources. There is a huge literature on climate governance issues in general, but less is known about effective support and the political-economy of adaptation. A large literature base and case studies on climate finance accountability and budgeting in governments is nascent and growing. Section 2 of this report briefly discusses governance of climate change issues, with a focus on the complexity and cross-cutting nature of climate change compared to the often static organisational landscape of governm

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Khan, Mahreen. Public Financial Management and Transitioning out of Aid. Institute of Development Studies, September 2022. http://dx.doi.org/10.19088/k4d.2022.145.

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This rapid review found an absence of literature focused specifically on measuring the impact of PFM and governance systems in countries that have transitioned from aid, by moving up the income ladder. However, there are a few academic publications and a limited number of studies by multilateral, such as the World Bank, that examine the role of PFM and governance systems in countries that are transitioning or have moved away from aid. However, the importance of public financial management (PFM) and governance systems in development is well established and seen as a pre-requisite for economic g

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Silvan, G. R. PFP MICON maintenance manual. Office of Scientific and Technical Information (OSTI), November 1994. http://dx.doi.org/10.2172/10106293.

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Hirzel, D. R. PFP Wastewater Sampling Facility. Office of Scientific and Technical Information (OSTI), May 1995. http://dx.doi.org/10.2172/80949.

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BUSCH, M. S. PFP Emergency Lighting Study. Office of Scientific and Technical Information (OSTI), February 2000. http://dx.doi.org/10.2172/801369.

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Morley, J. M. PFP Harrington lever hoists. Office of Scientific and Technical Information (OSTI), August 1994. http://dx.doi.org/10.2172/10185637.

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