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Tesi sul tema "Pulmonary surfactant"

Autore: Grafiati

Pubblicato: 4 giugno 2021

Ultima modifica: 25 luglio 2025

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1

Lewis, R. W. "Pulmonary surfactant metabolism." Thesis, Cardiff University, 1985. http://ethos.bl.uk/OrderDetails.do?uin=uk.bl.ethos.332108.

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2

Hockey, Peter Morey. "Pulmonary surfactant and asthma." Thesis, University of Southampton, 2000. http://ethos.bl.uk/OrderDetails.do?uin=uk.bl.ethos.274434.

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3

Nilsson, Kristina. "Solute exchange across the alveolo-capillary barrier." Lund : Depts. of Clinical Physiology, Malmö University Hospital and Lund University Hospital, Lund University, 1997. http://books.google.com/books?id=A0hrAAAAMAAJ.

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4

Mo, Young Keun. "Surfactant proteins in extra pulmonary sites /." [St. Lucia, Qld.], 2005. http://www.library.uq.edu.au/pdfserve.php?image=thesisabs/absthe19083.pdf.

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5

Sullivan, Lucy Catherine. "The molecular evolution of vertebrate pulmonary surfactant /." Title page, summary and introduction only, 1996. http://web4.library.adelaide.edu.au/theses/09SB/09sbs949.pdf.

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6

Worthman, Lynn-Ann D. "Surfactant protein A (SP-A) affects pulmonary surfactant morphology and biophysical properties." Thesis, National Library of Canada = Bibliothèque nationale du Canada, 1997. http://www.collectionscanada.ca/obj/s4/f2/dsk2/tape16/PQDD_0014/MQ34241.pdf.

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7

Wood, Philip. "Control of pulmonary surfactant secretion : an evolutionary perspective /." Title page, table of contents and abstract only, 1999. http://web4.library.adelaide.edu.au/theses/09PH/09phw878.pdf.

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8

Mander, Ann. "Pulmonary surfactant and neutrophil function in cystic fibrosis." Thesis, University of Southampton, 1999. http://ethos.bl.uk/OrderDetails.do?uin=uk.bl.ethos.310701.

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9

McCarty, Kenneth Dean. "Characterization of pulmonary surfactant apoproteins in the diabetic mouse." CSUSB ScholarWorks, 1989. https://scholarworks.lib.csusb.edu/etd-project/512.

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10

Espinosa, Frank F. (Frank Francis). "Exogenous surfactant transport through the pulmonary airways : improving surfactant replacement therapy for neonatal respiratory distress syndrome." Thesis, Massachusetts Institute of Technology, 1996. http://hdl.handle.net/1721.1/10974.

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11

Zaltash, Shahparak. "Pulmonary surfactant proteins B and C : molecular organisation and involvement in respiratory disease /." Stockholm, 2000. http://diss.kib.ki.se/2000/91-628-4571-3/.

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12

Johnston, Sonya D. "Development of the pulmonary surfactant system in non-mammalian amniotes /." Title page, contents and abstract only, 2001. http://web4.library.adelaide.edu.au/theses/09PH/09phj737.pdf.

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13

Ocampo, Minette C. "Protein-Lipid Interactions with Pulmonary Surfactant Using Atomic Force Microscopy." The Ohio State University, 2014. http://rave.ohiolink.edu/etdc/view?acc_num=osu1395050693.

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14

Van, Houtte Paul. "Etude expérimentale de l'effet des irradiations et des agents chimiothérapiques sur le poumon et le métabolisme du surfactant." Doctoral thesis, Universite Libre de Bruxelles, 1985. http://hdl.handle.net/2013/ULB-DIPOT:oai:dipot.ulb.ac.be:2013/241286.

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15

Patel, Anjana. "Recombinant synthesis of pulmonary surfactant proteins SP-B and SP-C." Thesis, Aston University, 2018. http://publications.aston.ac.uk/37638/.

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Abstract (sommario):

SP-B and SP-C are two pulmonary surfactant proteins. They were isolated in the tear film in 2006 (Lukovic et al. 2006). The molecular mechanism governing lipid spreading in the tear film is not well understood and, along with their production in yeast (as an alternative to animal derived material) provide the motives for this research. To investigate the involvement of SP-B and SP-C in lipid spreading, SP-B and SP-C have been produced as recombinant proteins in Pichia pastoris yeast. SP-B and SP-C are cleaved from their proproteins to produce mature active proteins and both forms were produced

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16

Dodd, Claire Elizabeth. "Impact of Pulmonary Surfactant on Human Macrophage Susceptibility to Mycobacterium tuberculosis." The Ohio State University, 2017. http://rave.ohiolink.edu/etdc/view?acc_num=osu1491747880967301.

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17

Cai, Jingfei. "Probing the Membrane Association Mechanisms for Pulmonary Collectins and Mammalian Phospholipase C." Thesis, Boston College, 2013. http://hdl.handle.net/2345/3872.

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Thesis advisor: Mary F. Roberts<br>Thesis advisor: Eranthie Weerapana<br>Peripheral proteins from mammals often exhibit multi-domain structures and require metal ions such as calcium as co-factors. This dissertation investigates two types of such proteins -- pulmonary collectins (surfactant proteins A and D) and phosphatidylinositol-specific phospholipase C (PLC) delta1 -- and their interactions with model membranes. One approach to work around the complexity brought upon by such multi-domain protein structure is to use a truncated construct or an isolated single domain. For pulmonary collecti

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18

Rova, M. (Meri). "The significance of surfactant protein gene polymorphisms in multifactorial infantile pulmonary diseases." Doctoral thesis, University of Oulu, 2005. http://urn.fi/urn:isbn:9514277481.

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Abstract (sommario):

Abstract Pulmonary surfactant is a lipid-protein mixture that lines the inner surface of the lung. The main function of surfactant is to reduce surface tension at the air-liquid interface, thus preventing alveolar collapse at the end of expiration. Lack of surfactant is the main cause of respiratory distress syndrome (RDS) in preterm infants. Very preterm babies are at risk of developing a lung disease called bronchopulmonary dysplasia (BPD). The surfactant proteins SP-A, -B, -C and -D have important functions in surfactant structure, homeostasis and innate immunity of the lung. The genes of t

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19

Sallese, Anthony. "Elucidating the Pathogenesis of Pulmonary Alveolar Proteinosis." University of Cincinnati / OhioLINK, 2017. http://rave.ohiolink.edu/etdc/view?acc_num=ucin1491813093586635.

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20

Li, Jing. "Processing, stability and interactions of lung surfactant protein C /." Stockholm, 2005. http://diss.kib.ki.se/2005/91-7140-582-8/.

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21

Kazemi, Taskooh Alireza. "Transport of complex fluids in the human pulmonary airway system." Thesis, Université Paris-Saclay (ComUE), 2019. http://www.theses.fr/2019SACLX077/document.

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Abstract (sommario):

La Thérapie par Substitution de Surfactant (TSS), qui opère par instillation d’une solution de surfactant directement dans l’arbre bronchique, est un traitement essentiel chez les nouveau-nés souffrant de syndrome de détresse respiratoire (SDRN). Cette procédure s’est révélée remarquablement efficace chez les grands prématurés, contribuant à la division par cinq de leur mortalité depuis les années 1980. À l’inverse, son utilisation s’est avérée décevante chez l’adulte dans le traitement du syndrome de détresse respiratoire aigu (SDRA), se soldant par un échec après des premiers essais pourtant

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22

Miller, Natalie J. "The evolution of a physiological system the pulmonary surfactant system in diving mammals /." Connect to this title online, 2005. http://hdl.handle.net/2440/37717.

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Abstract (sommario):

Pulmonary surfactant is a complex mixture of lipids and proteins that lowers surface tension, increases lung compliance, and prevents the adhesion of respiratory surfaces and pulmonary oedema. Pressure can have an enormous impact on respiratory function, by mechanically compressing tissues, increasing gas tension resulting in increased gas absorption and by increasing dissolved gas tensions during diving, resulting in the formation of bubbles in the blood and tissues. The lungs of diving mammals have a huge range of morphological adaptations to enable them to endure the extremely high pressure

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23

Aydın, Evrim Kaya Hakan. "Gebe koyunlara intraamniyotik surfaktan uygulamasının preterm kuzuların akciğer gelişimi üzerine etkilerinin histopatolojik ve biyokimyasal olarak değerlendirilmesi /." Isparta : SDÜ Tıp Fakültesi, 2005. http://tez.sdu.edu.tr/Tezler/TT00186.pdf.

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24

O'Toole, Stuart John. "The pulmonary surfactant system in congenital diaphragmatic hernia and the influence of fetal surgery on its development." Thesis, University of Newcastle Upon Tyne, 2000. http://ethos.bl.uk/OrderDetails.do?uin=uk.bl.ethos.312035.

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25

Wood, Philip. "Functional significance and control of release of pulmonary surfactant in the lizard lung /." Title page and abstract only, 1994. http://web4.library.adelaide.edu.au/theses/09SB/09sbw878.pdf.

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26

Al-Gawhari, Fatima Jalal. "Development of a non-ionic surfactant vesicles formulation of gemcitabine for pulmonary delivery." Thesis, University of Strathclyde, 2013. http://oleg.lib.strath.ac.uk:80/R/?func=dbin-jump-full&object_id=22647.

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Abstract (sommario):

Lung cancer is a major cause of death in the world. Cancer chemotherapy is limited by adverse toxicities to normal tissues. Targeted delivery of anticancer drugs to lung cancer by inhalation would help to reduce these toxicities. Lipid based delivery systems have been shown to be effective in improving the delivery of a number of drugs and the potential of using non-ionic surfactant vesicles (NIV) to improve the delivery of Gemcitabine (Gem) was studied in this project. NIV were used to encapsulate Gem (Gem-NIV) for delivery by the pulmonary route. NIV were prepared using different concentrati

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27

Codd, Jonathan Richard. "Torpor associated fluctuations in the pulmonary surfactant system in Gould's wattled bat Chalinolobus Gouldii /." Title page, contents and abstract only, 2001. http://web4.library.adelaide.edu.au/theses/09SM/09smc669.pdf.

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28

Henning, Lisa Novik. "Pulmonary surfactant protein a regulation of macrophage toll-like receptor expression, activity, and trafficking /." Columbus, Ohio : Ohio State University, 2008. http://rave.ohiolink.edu/etdc/view?acc%5Fnum=osu1211479279.

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29

Nag, Kaushik. "Association and interactions of pulmonary surfactant lipids and proteins in model membranes at the air-water interface." Thesis, National Library of Canada = Bibliothèque nationale du Canada, 1996. http://www.collectionscanada.ca/obj/s4/f2/dsk3/ftp04/NQ56665.pdf.

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30

Bohlin, Kajsa. "Surfactant metabolism in the newborn : the impact of ventilation strategy and lung disease /." Stockholm : Karolinska institutet, 2005. http://diss.kib.ki.se/2005/91-7140-229-2/.

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31

Beresford, Michael William. "The role of pulmonary surfactant proteins and inflammatory cytokines in preterm infants ventilated for respiratory distress receiving natural or synthetic surfactant therapies." Thesis, University of Liverpool, 2002. http://ethos.bl.uk/OrderDetails.do?uin=uk.bl.ethos.268903.

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32

Carlson, Tracy Karin. "The Effects of Pulmonary Surfactant Protein-D on Innate Immune Cells and Tuberculosis Pathogenesis." The Ohio State University, 2011. http://rave.ohiolink.edu/etdc/view?acc_num=osu1299708141.

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33

Kerr, Margaret Heather. "An investigation of the pulmonary surfactant system in children with severe respiratory syncytial virus infection." Thesis, University of Glasgow, 1998. http://ethos.bl.uk/OrderDetails.do?uin=uk.bl.ethos.265640.

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34

Spitler, Grant. "In vitro dissolution of uranium contaminated soil in simulated lung fluid containing a pulmonary surfactant." University of Cincinnati / OhioLINK, 2013. http://rave.ohiolink.edu/etdc/view?acc_num=ucin1377871194.

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35

Liu, Pamela Siumey. "Resposta imune induzida em camundongos por imunização transcutânea com proteína recombinante LipL32 de leptospira." Universidade de São Paulo, 2016. http://www.teses.usp.br/teses/disponiveis/87/87131/tde-24082016-100639/.

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Abstract (sommario):

A imunização transcutânea (TCI) é uma via atrativa para o desenvolvimento de vacinação livre de agulhas, atuando nas APCs da pele, podendo substituir algumas das imunizações convencionais, em termos de facilidade, segurança e eficácia. O presente estudo avaliou a resposta imune da TCI com proteína recombinante de leptospira LipL32, uma proteína altamente conservada em cepas patogênicas e potente candidata vacinal. A TCI com a LipL32 na região abdominal de C57BL-6 foi capaz de primar o sistema imune, suscitando resposta sistêmica com altos níveis de anticorpos contra o antígeno, após reforços s

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36

Bulek, Anna Marta. "Effect of pulmonary surfactant on innate immune responses in influenza virus infected human airway epithelial cells." Thesis, Cardiff University, 2008. http://orca.cf.ac.uk/55755/.

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Overwhelming inflammatory responses leading to neutrophil invasion are hypothesised to be the main cause of mortality in influenza virus induced acute respiratory distress syndrome (ARDS). Previously, pulmonary surfactant has been shown to modulate inflammatory responses to bacterial agents. The aim of the present study was to investigate the effect of pulmonary surfactant on innate immune responses in an in vitro model of influenza virus infected human airway epithelial cells. Human lung type II alveolar epithelial cells A549 and BEAS-2B human bronchial epithelial cells were infected with inf

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37

Munteanu, Bogdan. "Actions de particules d’usure aéroportées sur les propriétés mécaniques et physicochimiques des «films» de surfactant pulmonaire : Conséquences sur la conception de particules tribo-bio-compatibles." Thesis, Lyon, INSA, 2015. http://www.theses.fr/2015ISAL0034/document.

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Paradoxalement, la sécurité routière est assurée entre autre par la production de particules d’usure ! Ainsi, près de 20 000 tonnes de garnitures de frein sont usées par an en France, dont 9 000 tonnes sous forme de particules d’usure aéroportées. Ces particules posent des problèmes de santé car leur composition chimique et leur morphologie font qu’elles interagissent avec la paroi alvéolaire entrainant des pathologies. Au cours de ces pathologies la phase la plus étudiée est la phase inflammatoire qui s’installe une fois que la particule a passé la première barrière de protection qui est le f

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38

Maker, Garth Lucas. "Regulation of surfactant production by fetal type II pneumocytes and characterization of fibroblast-pneumocyte factor /." Access via Murdoch University Digital Theses Project, 2007. http://wwwlib.murdoch.edu.au/adt/browse/view/adt-MU20080430.141113.

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39

Lindwall, Robert B. I. "Respiratory distress syndrome : aspects of inhaled nitric oxide surfactant and nasal CPAP /." Stockholm, 2005. http://diss.kib.ki.se/2005/91-7140-297-7/.

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40

Frangolias, Despina Daisy. "Candidate genes other than the CFTR gene as possible modifiers of pulmonary disease severity in cystic fibrosis." Thesis, University of British Columbia, 2008. http://hdl.handle.net/2429/527.

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Abstract (sommario):

Cystic fibrosis (CF) is a single gene Mendelian disorder characterized by pulmonary disease and pancreatic insufficiency. Pulmonary disease is the major cause of death in CF patients. Although some cystic fibrosis transmembrane conductance regulator (CFTR) genotypes are associated with less severe disease, patients possessing the same genotype show great variation in pulmonary disease severity and progression. Genes involved in modulating the inflammatory response and genes increasing susceptibility to infection are proposed as modifiers of pulmonary disease severity. Polymorphisms selected f

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41

Langman, Carly. "Thermal influences upon the composition and function of pulmonary surfactant in a heterothermic mammal [sic] (Sminthopsis crassicaudata) /." Adelaide, 1995. http://web4.library.adelaide.edu.au/theses/09S.B/09s.bl284.pdf.

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42

Deb, Roona. "An investigation of the roles of lung surfactant proteins -A and -D in the pulmonary allergic response." Thesis, University of Oxford, 2006. http://ethos.bl.uk/OrderDetails.do?uin=uk.bl.ethos.440148.

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43

Fisher, Carolyn E. "Fibres in vitro : the importance of pulmonary surfactant, tumour necrosis factor alpha, nitric oxide and ferric iron." Thesis, Edinburgh Napier University, 1999. http://ethos.bl.uk/OrderDetails.do?uin=uk.bl.ethos.285698.

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44

Souza, João Francisco Ventrici de. "Efeito de materiais particulados em sistemas modelos de biomembranas." Universidade de São Paulo, 2012. http://www.teses.usp.br/teses/disponiveis/59/59138/tde-13042012-144908/.

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Abstract (sommario):

Neste trabalho foram estudadas algumas propriedades de sistemas miméticos de tensoativos pulmonares (TP) constituídos de dipalmitoilfosfatidilcolina (DPPC) e DPPC/colesterol e suas interações com o material particulado (MP) proveniente da queima da palha da cana-de-açúcar, reconhecido por meio de estudos clínicos como agente agravador de diversas patologias respiratórias. O MP utilizado neste trabalho foi analisado por meio de uma série de técnicas: espalhamento dinâmico de luz (DLS), microscopia eletrônica de varredura acrescida do uso de análise de energia dispersiva de raios X (MEV e MEV-ED

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45

au, G. Maker@murdoch edu, and Garth Lucas Maker. "Regulation of surfactant production by fetal type II pneumocytes and the characterization of fibroblast-pneumocyte factor." Murdoch University, 2008. http://wwwlib.murdoch.edu.au/adt/browse/view/adt-MU20080430.141113.

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Abstract (sommario):

The fetal lung undergoes extensive physiological and biochemical maturation prior to birth in preparation for its postnatal function as an organ for gas exchange. Pulmonary surfactant, a substance that reduces surface tension and prevents alveolar collapse, is produced by type II pneumocytes within the lung. Reduced ability to produce surfactant leads to neonatal respiratory distress syndrome. Synthesis of the phospholipid component of surfactant, phosphatidylcholine (PC), is stimulated by fibroblast-pneumocyte factor (FPF), a protein expressed by fibroblast cells within the fetal lung. Althou

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46

Väyrynen, O. (Outi). "Proinflammatory cytokines modify the expression of surfactant proteins:study in perinatal rabbit lung." Doctoral thesis, University of Oulu, 2003. http://urn.fi/urn:isbn:9514270584.

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Abstract Deficiency of pulmonary surfactant is the main cause of respiratory distress syndrome (RDS) in premature newborn infants, which is often complicated by chronic lung disease (CLD). Preterm birth is often associated with intra-amniotic infection (IUI), which is characterized by increased proinflammatory cytokines, such as interleukin-1 (IL-1) and tumor necrosis factor-α (TNF-α), in the amniotic fluid. In very preterm birth due to IUI, the incidence of RDS is decreased, while the incidence of CLD is increased. Maternal glucocorticoids are used in imminent preterm birth to prevent RDS. Th

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47

Dico, Awel Seid. "A ²H-NMR study of interactions in model membranes containing pulmonary surfactant proteins SP-B and SP-C." Thesis, National Library of Canada = Bibliothèque nationale du Canada, 1998. http://www.collectionscanada.ca/obj/s4/f2/dsk2/ftp03/NQ34245.pdf.

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48

Smith, Johan. "A comparison of synthetic surfactants : evaluation of a novel surfactant (1,2-dipalmitoyl-sn-phosphatidycholine and trehalose [C12H22O11]) and comparison with other synthetic formulations." Thesis, Stellenbosch : Stellenbosch University, 2002. http://hdl.handle.net/10019.1/52624.

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Abstract (sommario):

In title 12, 22, 11 are in subscript.<br>Thesis (PhD)--Stellenbosch University, 2002.<br>ENGLISH ABSTRACT: The aim of this study was to test a synthetic protein-free surfactant preparation, LPM-l, with the same chemical composition as commercially available Exosurf (Glaxo Wellcome), but containing in addition, a sugar, trehalose (TRE). Towards this end, a study was designed to firstly test the hypothesis that the true difference in acute physiological effects between a mixture of oppe, tyloxapol, hexadecanol and trehalose (LPM-l), and Exosurf, (Oppe, tyloxapol and hexadecanol) is zero, i

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49

Rausch, Felix [Verfasser], Ludger A. [Akademischer Betreuer] Wessjohann, and Harald [Akademischer Betreuer] Lanig. "3D modeling of the putative human surfactant proteins SP-G and SP-H and simulations in a pulmonary surfactant model system / Felix Rausch. Betreuer: Ludger A. Wessjohann ; Harald Lanig." Halle, Saale : Universitäts- und Landesbibliothek Sachsen-Anhalt, 2015. http://d-nb.info/107850511X/34.

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50

WAN, HUAJING. "CRITICAL ROLES OF FORKHEAD BOX A2 DURING LUNG DEVELOPMENT." University of Cincinnati / OhioLINK, 2004. http://rave.ohiolink.edu/etdc/view?acc_num=ucin1091650603.

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