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Articoli di riviste sul tema "Pylori"

Autore: Grafiati

Pubblicato: 4 giugno 2021

Ultima modifica: 25 luglio 2025

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1

Chiba, N., A. Matisko, P. Sinclair, and ABR Thomson. "Helicobacter pylori: From Bench to Bedside." Canadian Journal of Gastroenterology 11, no. 7 (1997): 589–96. http://dx.doi.org/10.1155/1997/975469.

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With the exponential increase in research in the field ofHelicobacter pyloria paradigm shift has occurred. It is now recognized thatH pyloriis a chronic infection of the stomach causing inflammation. Some patients remain asymptomatic, while others may develop dyspepsia, duodenal or gastric ulcer, gastric cancer or a mucosa-associated lymphoid tissue lymphoma. However, the role ofH pyloriin contributing to nonulcer dyspepsia or nonsteroidal anti-inflammatory drug gastropathy remains controversial. An effective vaccine againstH pyloriis years away. Major interest has focused on the questions "wh

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2

Sakaguchi, Takuki, Takaaki Sugihara, Ken Ohnita, et al. "Pyloric Incompetence Associated with Helicobactor pylori Infection and Correlated to the Severity of Atrophic Gastritis." Diagnostics 12, no. 3 (2022): 572. http://dx.doi.org/10.3390/diagnostics12030572.

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Duodenogastric reflux (DGR) causes bile reflux gastritis (BRG) and may develop into gastric cancer. DGR is classified as primary in non-operated stomachs or secondary to surgical intervention. Primary DGR and Helicobacter pylori (H. pylori) infection are reportedly related. However, the mechanism is not fully understood. This study aimed to elucidate the relationship between H. pylori infection and pyloric incompetence in a non-operated stomach. A total of 502 non-operated participants who underwent an upper intestinal endoscopy were prospectively enrolled. Endoscopic findings (EAC, endoscopic

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3

Assumpção, Paulo Pimentel de, Robert M. Genta, Maria Constanza Camargo, Jessica Manoelli Costa da Silva, Manuel Ricardo Amieva, and Massimo Rugge. "The Landscape of Helicobacter pylori-related Gastric Carcinogenesis." Journal of Gastrointestinal and Liver Diseases 33, no. 4 (2024): 524–34. https://doi.org/10.15403/jgld-5959.

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The relationship between Helicobacter pylori (H. pylori) and humans remains a complex enigma. While other factors contribute to gastric cancer (GC), their impact pales in comparison to the central role of H. pylori. Various cofactors, such as dietary carcinogens and Epstein-Barr virus infection, can lead to GC independently of H. pylori. However, it is likely the combination of mechanisms, especially those driven by H. pylori, that represents the primary force behind GC development. Identifying individuals at high risk of developing H. pylori-related GC or detecting the disease in its earliest

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Garcés-Duran, R., S. Kindt, K. Kotilea, et al. "Belgian consensus for Helicobacter pylori management 2023." Acta Gastro Enterologica Belgica 86, no. 1 (2023): 74–91. http://dx.doi.org/10.51821/86.1.11327.

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Helicobacter pylori (H. pylori) infection causes chronic gastritis, peptic ulcers and gastric cancer. Although H. pylori prevalence is decreasing worldwide, regional variations exist in Europe, with the lowest infection prevalence in Northern Europe, and the highest in Eastern and Southern Europe (1). Changes in the treatment recommendations and the increasing available evidence have justified the implementation of new recommendations since last Belgian consensus in 1998 (2). Several non-H. pylori Helicobacter species (NH.PYLORI-H), colonizing the stomach of domestic animals, also have the abi

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5

Chen, Yang. "The overview of Clarithromycin treatment for Helicobacter pylori infection." Theoretical and Natural Science 46, no. 1 (2024): 142–48. http://dx.doi.org/10.54254/2753-8818/46/20240604.

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Helicobacter pylori (H. pylori) is a Gram-negative bacillus that causes infections worldwide. Helicobacter pylori's drug resistance and infectivity are vital factors causing Helicobacter pylori infection. The virulence factors of Helicobacter pylori include acid escape virulence factors and epithelial cell colonizing factors, which can directly or indirectly cause chronic gastritis or gastric ulcer. As the resistance of Helicobacter pylori increases, a combination of multiple antibiotics is now the recommended therapy, and clarithromycin is one of them. The production process of clarithromycin

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6

Asonuma, Sho, Akira Imatani, Naoki Asano, et al. "Helicobacter pylori induces gastric mucosal intestinal metaplasia through the inhibition of interleukin-4-mediated HMG box protein Sox2 expression." American Journal of Physiology-Gastrointestinal and Liver Physiology 297, no. 2 (2009): G312—G322. http://dx.doi.org/10.1152/ajpgi.00518.2007.

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Helicobacter pylori is a major cause of the transdifferentiation into intestinal metaplasia that may develop gastric cancer. However, the molecular pathogenesis of this transdifferentiation is poorly understood. A SRY-related HMG box protein Sox2 is an essential transcription factor of organ development in brain, lung, and stomach. Our aim of this study was to investigate the mechanism responsible for regulation of Sox2 in host Th1-dominant response to H. pylori. Sox2 protein was immunohistochemically expressed in both human oxyntic and pyloric glands with H. pylori infection, but not in intes

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7

Takatsuna, Masafumi, Rie Azumi, Takeshi Mizusawa, et al. "A case of Helicobacter pylori-negative early gastric adenocarcinoma with gastrointestinal phenotype." Endoscopy International Open 09, no. 06 (2021): E863—E866. http://dx.doi.org/10.1055/a-1396-3854.

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AbstractA 40-year-old man with slightly depressed (0-IIc) type gastric cancer of the pyloric anterior gastric area underwent pre-operative screening for tetralogy of Fallot and endoscopic submucosal dissection (ESD) and was tested for Helicobacter pylori antigens and antibodies. Both tests were negative. He did not have a history of eradication. Pathological diagnosis of ESD showed a well-differentiated adenocarcinoma. The tumor was CD10-positive, MUC5AC-negative, and MUC6-confocal positive; it showed differentiation with gastrointestinal phenotype. Moreover, the tumor cells were lysozyme-posi

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8

Saler, Tayyibe, Şakir Özgür Keşkek, Sibel Kırk, Süleyman Ahbab, and Gülay Ortoğlu. "H. pyloriMay Not Be Associated with Iron Deficiency Anemia in Patients with Normal Gastrointestinal Tract Endoscopy Results." Advances in Hematology 2014 (2014): 1–4. http://dx.doi.org/10.1155/2014/375915.

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Background. The aim of this study was to investigate the association between iron deficiency anemia andH. pyloriin patients with normal gastrointestinal tract endoscopy results.Materials and Methods. A total of 117 male patients with normal gastrointestinal tract endoscopy results were included in this retrospective study. The study and control groups included 69 and 48 patients with and without iron deficiency anemia, respectively. The prevalence ofH. pylori, the number of RBCs, and the levels of HGB, HTC, MCV, iron, and ferritin were calculated and compared.Results. There was no statisticall

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9

Kosai, Nik Ritza, Hardip Singh Gendeh, Abdul Rashid Norfaezan, Jamin Razman, Paul Anthony Sutton, and Srijit Das. "Prolapsing Gastric Polyp Causing Intermittent Gastric Outlet Obstruction." International Surgery 100, no. 6 (2015): 1148–52. http://dx.doi.org/10.9738/intsurg-d-14-00205.1.

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Gastric polyps are often an incidental finding on upper gastrointestinal endoscopy, with an incidence up to 5%. The majority of gastric polyps are asymptomatic, occurring secondary to inflammation. Prior reviews discussed Helicobacter pylori (H pylori)–associated singular gastric polyposis; however, we present a rare and unusual case of recurrent multiple benign gastric polyposis post H pylori eradication resulting in intermittent gastric outlet obstruction. A 70-year-old independent male, Chinese in ethnicity, with a background of diabetes mellitus, hypertension, and a simple renal cyst prese

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10

Öztekin, Merve, Birsen Yılmaz, Duygu Ağagündüz, and Raffaele Capasso. "Overview of Helicobacter pylori Infection: Clinical Features, Treatment, and Nutritional Aspects." Diseases 9, no. 4 (2021): 66. http://dx.doi.org/10.3390/diseases9040066.

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Helicobacter pylori (H. pylori) is a 0.5–1 µm wide, 2–4 µm long, short helical, S-shaped Gram-negative microorganism. It is mostly found in the pyloric region of the stomach and causes chronic gastric infection. It is estimated that these bacteria infect more than half of the world’s population. The mode of transmission and infection of H. pylori is still not known exactly, but the faecal–oral and oral–oral routes via water or food consumption are thought to be a very common cause. In the last three decades, research interest has increased regarding the pathogenicity, microbial activity, genet

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Blasiak, Janusz, Jan Chojnacki, and Cezary Chojnacki. "Inflammation, oxidative stress, DNA damage response and epigenetic modifications interact behind the beneficial actions of melatonin on H. pylori-mediated gastric disorders." Melatonin Research 6, no. 2 (2023): 135–47. http://dx.doi.org/10.32794/mr112500145.

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Helicobacter pylori (H. pylori) infection is associated with several disorders of the gastrointestinal tract, including gastric cancer. Studies of ours and others suggest that H. pylori infection may affect melatonin synthesis in the gastric epithelial cells. On the other hand, melatonin ameliorates gastric disorders as shown in clinical trials and experimental studies. Moreover, melatonin not only suppresses the DNA-damaging reaction of diet-related mutagens that can initiate carcinogenesis in gastric mucosa, but also the oxidative DNA damage evoked by reactive oxygen and nitrogen species pro

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12

Omata, Fumio, Takuro Shimbo, Sachiko Ohde, Gautam A. Deshpande, and Tsuguya Fukui. "Cost-Effectiveness Analysis ofHelicobacter pyloriDiagnostic Methods in Patients with Atrophic Gastritis." Gastroenterology Research and Practice 2017 (2017): 1–10. http://dx.doi.org/10.1155/2017/2453254.

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Background. There are several diagnostic methods forHelicobacter pylori (H. pylori)infection. A cost-effective analysis is needed to decide on the optimal diagnostic method. The aim of this study was to determine a cost-effective diagnostic method in patients with atrophic gastritis (AG).Methods. A decision-analysis model including seven diagnostic methods was constructed for patients with AG diagnosed by esophagogastroduodenoscopy. Expected values of cost and effectiveness were calculated for each test.Results. If the prevalence ofH. pyloriin the patients with AG is 85% and CAM-resistantH. py

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Schneider, Sabine, Gert Carra, Ugur Sahin, Benjamin Hoy, Gabriele Rieder, and Silja Wessler. "Complex Cellular Responses of Helicobacter pylori-Colonized Gastric Adenocarcinoma Cells." Infection and Immunity 79, no. 6 (2011): 2362–71. http://dx.doi.org/10.1128/iai.01350-10.

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ABSTRACTHelicobacter pyloriis an important class I carcinogen that persistently infects the human gastric mucosa to induce gastritis, gastric ulceration, and gastric cancer.H. pyloripathogenesis strongly depends on pathogenic factors, such as VacA (vacuolating cytotoxin A) or a specialized type IV secretion system (T4SS), which injects the oncoprotein CagA (cytotoxin-associated gene A product) into the host cell. Since access to primary gastric epithelial cells is limited, many studies on the complex cellular and molecular mechanisms ofH. pyloriwere performed in immortalized epithelial cells o

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Fiorini, Giulia, Ilaria Maria Saracino, Angelo Zullo, et al. "Antibiotic Resistance and Therapy for H. pylori Infection in Immigrant Patients Treated in Italy." Journal of Clinical Medicine 9, no. 5 (2020): 1299. http://dx.doi.org/10.3390/jcm9051299.

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Background: Helicobacter pylori (H. pylori) infection is the leading cause of both peptic ulcers and gastric tumors, including low-grade MALT-lymphoma and adenocarcinoma. Although it is decreasing in developed countries, H. pylori prevalence remains high in developing areas, mainly due to low socio-economic levels, and the potential consumption of contaminated water. Moreover, a different pattern of primary antibiotic resistance is expected in their H. pylori isolates, potentially affecting the efficacy of standard eradication therapies. Indeed, a previous study showed the eradication rate fol

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15

Di Ciaula, Agostino, Giuseppe Scaccianoce, Marino Venerito, et al. "Eradication Rates in Italian Subjects Heterogeneously Managed for Helicobacter pylori Infection. Time to Abandon Empiric Treatments in Southern Europe." Journal of Gastrointestinal and Liver Diseases 26, no. 2 (2017): 129–37. http://dx.doi.org/10.15403/jgld.2014.1121.262.itl.

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Background & Aims: H. pylori eradication is strongly affected by various factors, including the ongoing antibiotic resistance. We describe a “real life” scenario in patients managed for H. pylori-related conditions, living in a southern Italian region (Apulia), an area with clarithromycin resistance >15%.Methods: 2,224 subjects were studied in two tertiary referral centers in Apulia. Analyses included: reason for referral, H. pylori infection rates (13C-urea breath test – UBT or upper endoscopy), and eradication rates following distinct regimens previously prescribed or prospectively pr

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I.M., Saracino, Pavoni M., Zullo A., et al. "Antibiotic Resistance and Therapy Outcome in H. pylori Eradication Failure Patients." Antibiotics 9, no. 3 (2020): 121. http://dx.doi.org/10.3390/antibiotics9030121.

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Helicobacter pylori (H. pylori) eradication fails in a definite amount of patients despite one or more therapeutic attempts. Curing these patients is progressively more difficult, due to development of antibiotic resistance. Current guidelines suggest testing antibiotic susceptibility in H. pylori isolates following two therapeutic attempts. Aim: to evaluate the development of antibiotic resistance, MIC values trends and therapeutic outcomes in patients who failed at least one H. pylori eradication therapy. Methods: consecutive patients, referred to perform upper gastrointestinal endoscopy (UG

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German, Serafima V., A. V. Modestova, I. E. Zykova, I. P. Bobrovnitsky, and M. Yu Yakovlev. "ON THE POSSIBLE PARTICULARITY OF HELICOBACTER PYLORI INFECTION TO GALLSTONE DISEASE." Hygiene and sanitation 97, no. 11 (2018): 1076–79. http://dx.doi.org/10.18821/0016-9900-2018-97-11-1076-79.

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Introduction. Up to now, it has not been established whether Helicobacter pylori, the most common bacterial pathogen of human, is involved in cholelithiasis. Material and Methods. Based on the analysis of prophylactic medical examination of working people in the Moscow region, the determination the pyloric Helicobacter infection and assessment of the virulence of bacteria there were studied the associations of H. pylori infection and gallstones. The infection was detected by the presence of serum specific antibodies of IgG class, the virulence of the strain H. pylori - by the presence of total

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Adewumi, F. A., J. O. Ipinlaye, C. O. Esan, and A. O. Oluyege. "Rapid detection of <i>Helicobacter pylori</i> infection among participants presenting with dyspepsia in a primary health care facility in Ado-Ekiti, Ekiti State." World Journal of Applied Science & Technology 16, no. 1 (2025): 98–102. https://doi.org/10.4314/wojast.v16i1.98.

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Helicobacter pylori (H. pylori) is a Gram-negative bacterium capable of colonizing the pyloric region of the stomach, and induces chronic gastric infection. It is closely related to gastrointestinal diseases such as gastritis, gastric ulcer, and gastric cancer. Notably, this bacterium has achieved pandemic magnitudes, infecting over half of the global population. This study sought to investigate the prevalence of Helicobacter pylori infection among patients presenting with dyspepsia at a primary healthcare facility in Ado-Ekiti, Ekiti State, utilizing non-invasive diagnostic procedures. Two hu

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Haruna, Sani Ado. "Prevalence and Risk Factors of Helicobacter Pylori Infection among Patients with Peptic Ulcer Disease Attendeing Muhammad Abdullahi Wase Teaching Hospital." Scholars Academic Journal of Biosciences 11, no. 12 (2023): 467–73. http://dx.doi.org/10.36347/sajb.2023.v11i12.008.

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Duodenal (DU) and gastric (GU) ulcers are both included in the category of peptic ulcer disease (PUD). The first section of the duodenum or the stomach's pre-pyloric region (antrum) are the most common sites for duodenal ulcers. Most cases of gastric ulcers occur in the stomach's lesser curvature, which connects to the body's antrum (angularis). The purpose of the study is to ascertain the prevalence and risk factors of Helicobacter pylori infections in Muhammad Abdullahi Wase Teaching Hospital patients suffering from peptic ulcer disease. Patients with PUD who had an H. pylori infection test

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Hassan, Md Ali, MA Ahad, MH Rahman, MSH Bhuiyan, and MAH Khan. "Association between cytotoxin producing H. pylori and gastric carcinoma." Bangladesh Medical Journal Khulna 49, no. 1-2 (2017): 13–17. http://dx.doi.org/10.3329/bmjk.v49i1-2.31819.

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Background: Enormous studies have been conducted worldwide regarding CagA+ status of H. pylori in gastric carcinoma Objective: No study relating CagA+ status and gastric carcinoma has been carried out in our country yet. This study has been designed to see the association between CagA+ H. pylorl strain and gastric carcinomaMethods: For this purpose, a total number of 80 (eighty) patients were selected. Of the 80 (eighty) patients, 40 (forty) were selected as cases (malignant) and the remainder 40 (forty) were selected as controls (non malignant). H. pylori was detected by applying non invasive

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Malik, G. M., M. Mubarik, and S. A. Kadla. "Helicobacter pylori Infection in Endoscopic Biopsy Specimens of Gastric Antrum: Laboratory Diagnosis and Comparative Efficacy of Three Diagnostic Tests." Diagnostic and Therapeutic Endoscopy 6, no. 1 (1999): 25–29. http://dx.doi.org/10.1155/dte.6.25.

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Aims and objectives The present study was undertaken to compare the diagnostic yield of three available test procedures for detecting Helicobacter pylori (H. pylori) infection in endoscopic biopsies.Methods H. pylori infection was sought in 150 patients referred for upper gastrointestinal (GI) endoscopy. Multiple (about six) biopsy specimens were taken from pyloric antrum in each patient. Two biopsy specimens were subjected to one minute endoscopy room test – OMERT (a modified form of urease test), two were sent for histopathological analysis, where multiple sections were subjected to Giemsa s

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Jaafar, Salim Shamkhi, Ehab Y. Jabber, Amer Abdul Husain Alwish, Raheem Tuama Obayes Al- Mammori та Hasanain Khaleel Shareef. "THE IMMUNOLOGICAL ROLE OF ANTI-H. PYLORI ANTIBODIES LEVEL, INF-Γ, AND TNF-Α IN A NUMBER OF PATIENTS WITH A TUMOR IN THE DIGESTIVE SYSTEM IN BABYLON GOVERNORATE". International Journal Multidisciplinary (IJMI) 1, № 3 (2024): 168–76. https://doi.org/10.61796/ijmi.v1i3.184.

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General Background: Helicobacter pylori is a well-known bacterium responsible for chronic infections, significantly contributing to gastric and mucosa-associated lymphoid tissue cancers, as well as peptic ulcer disease. Specific Background: The link between H. pylori infection and gastrointestinal (GIT) tumors remains under-explored, particularly concerning immunological parameters. Knowledge Gap: The role of immune markers, such as anti-H. pylori IgG antibodies, TNF-α, and IFN-γ, in distinguishing between malignant and benign GIT tumors is unclear. Aims: This study aimed to assess the relatio

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Lee, Woo-Kon, Keiji Ogura, John T. Loh, Timothy L. Cover, and Douglas E. Berg. "Quantitative Effect of luxS Gene Inactivation on the Fitness of Helicobacter pylori." Applied and Environmental Microbiology 72, no. 10 (2006): 6615–22. http://dx.doi.org/10.1128/aem.01291-06.

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ABSTRACT Furanone metabolites called AI-2 (autoinducer 2), used by some bacterial species for signaling and cell density-regulated changes in gene expression, are made while regenerating S-adenosyl methionine (SAM) after its use as a methyl donor. The luxS-encoded enzyme, in particular, participates in this activated methyl cycle by generating both a pentanedione, which is transformed chemically into these AI-2 compounds, and homocysteine, a precursor of methionine and SAM. Helicobacter pylori seems to contain the genes for this activated methyl cycle, including luxS, but not genes for AI-2 up

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Bahrami, Ahmad Reza, Ebrahim Rahimi, and Hajieh Ghasemian Safaei. "Detection ofHelicobacter pyloriin City Water, Dental Units' Water, and Bottled Mineral Water in Isfahan, Iran." Scientific World Journal 2013 (2013): 1–5. http://dx.doi.org/10.1155/2013/280510.

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Helicobacter pyloriinfection in human is one of the most common infections worldwide. However, the origin and transmission of this bacterium has not been clearly explained. One of the suggested theories is transmission via water. This study was conducted to determine the prevalence rate ofH. pyloriin tap water, dental units' water, and bottled mineral water in Iran. In the present study, totally 200 water samples were collected in Isfahan province and tested forH. pyloriby cultural method and polymerase chain reaction (PCR) by the detection of theureC (glmM)gene. Using cultural method totally

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Coconnier, Marie-Helene, Vanessa Lievin, Elisabeth Hemery, and Alain L. Servin. "Antagonistic Activity againstHelicobacter Infection In Vitro and In Vivo by the HumanLactobacillus acidophilus Strain LB." Applied and Environmental Microbiology 64, no. 11 (1998): 4573–80. http://dx.doi.org/10.1128/aem.64.11.4573-4580.1998.

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ABSTRACT The purpose of the present study was to examine the activity of the human Lactobacillus acidophilus strain LB, which secretes an antibacterial substance(s) against Helicobacter pyloriin vitro and in vivo. The spent culture supernatant (SCS) of the strain LB (LB-SCS) dramatically decreased the viability of H. pylori in vitro independent of pH and lactic acid levels. Adhesion of H. pylori to the cultured human mucosecreting HT29-MTX cells decreased in parallel with the viability of H. pylori. In conventional mice, oral treatment with the LB-SCS protected against infection with Helicobac

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Watanabe, Tomohiro, Naoki Asano, Atsushi Kitani, Ivan J. Fuss, Tsutomu Chiba, and Warren Strober. "NOD1-Mediated Mucosal Host Defense againstHelicobacter pylori." International Journal of Inflammation 2010 (2010): 1–6. http://dx.doi.org/10.4061/2010/476482.

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Infection of the stomach withHelicobacter pyloriis an important risk factor for gastritis, peptic ulcer, and gastric carcinoma. Although it has been well established that persistent colonization byH. pyloriis associated with adaptive Th1 responses, the innate immune responses leading to these Th1 responses are poorly defined. Recent studies have shown that the activation of nucleotide-binding oligomerization domain 1 (NOD1) in gastric epithelial cells plays an important role in innate immune responses againstH. pylori. The detection ofH. pylori-derived ligands by cytosolic NOD1 induces several

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Nisreen Jawad Kadhim and Saeed Hameed Lafta. "Helicobacter pylori: Challenges and Advances in Diagnosis, Treatment, and Eradication." Academic International Journal of Medical Update 2, no. 1 (2024): 43–52. https://doi.org/10.59675/u216.

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A common and extensively distributed "Helicobacter pylori" still poses a serious risk to public health. "H. pylori" infections can cause various stomach problems, such as inflammation, gastroduodenal ulcers, and cancers; therefore, it is essential to discover and treat them early to stop their progression. To reduce or perhaps completely eradicate "H. pylori," people must be informed of the bacteria and the health risks it poses. Raising the bar for the present diagnostic, eradication, and alternate treatment approaches is therefore essential. Furthermore, a succinct review of conventional and

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Zullo, Angelo, Vincenzo De Francesco, Annamaria Bellesia, et al. "Bismuth-based Quadruple Therapy Following H. pylori Eradication Failures: a Multicenter Study in Clinical Practice." Journal of Gastrointestinal and Liver Diseases 26, no. 3 (2017): 225–29. http://dx.doi.org/10.15403/jgld.2014.1121.263.zul.

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Background & Aims: Helicobacter pylori (H. pylori) eradication in patients who failed one or more therapeutic attempts remains challenging. This study aimed to assess the efficacy of three-in-one capsules bismuth-based quadruple therapy (Pylera®) in these patients managed in clinical practice. Methods: This was a prospective, open-label, multicenter study enrolling consecutive, adult patients with persistent H. pylori infection following at least one standard therapy. All patients received a rescue quadruple therapy with Pylera (3 capsules four times daily) and esomeprazole 20 mg (1

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Dahshan, Ahmed, Kevin G. Donovan, Issam M. Halabi, Richard Ranne, Mary Li, and William P. Illig. "Helicobacter pylori and Infantile Hypertrophic Pyloric Stenosis." Journal of Pediatric Gastroenterology and Nutrition 42, no. 3 (2006): 262–64. http://dx.doi.org/10.1097/01.mpg.0000189359.76545.b8.

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Chicherin, I. Yu, I. P. Pogorelskiy, E. P. Kolevatykh, I. A. Lundovskikh, and M. R. Shabalina. "First experience with the metaprebiotic Stimbifid plus used for eradication of Helicobacter pylori in patients with gastric ulcer." Infekcionnye bolezni 19, no. 3 (2021): 92–103. http://dx.doi.org/10.20953/1729-9225-2021-3-92-103.

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Objective. To analyze the relationships between probiotic microorganisms and H. pylori KM-11 (RifR) and evaluate their effect on the structural organization of the pathogen and natural colonization resistance of the stomach both independently and with the metaprebiotic Stimbifid plus using bacteriological methods and electron microscopy. Our findings can be potentially used for effective eradication of H. pylori KM-11 (RifR) and treatment of gastric ulcer in volunteers during metaprebiotic therapy. Materials and methods. The following strains of microorganisms were used in this study: Lactobac

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Zohaib Hassan, Riaz Ahmed, Sajid Hussain, Muhammad Khalil Surani, Asif Karim, and Mubina Ifat. "Helicobacter pylori infection in children with recurrent abdominal pain." Professional Medical Journal 29, no. 05 (2022): 681–85. http://dx.doi.org/10.29309/tpmj/2022.29.05.6822.

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Objective: To find out the frequency of Helicobacter Pylori infection in children with recurrent abdominal pain. Study Design: Cross Sectional study. Setting: Pediatric Medicine, Children Hospital and Institute of Child Health, Multan. Period: November 2020 to May 2021. Material & Methods: A total of 148 children of both genders aged 4-12 years presenting with recurrent abdominal pain were included. Detailed history and physical examination was conducted. Frequency of helicobacter pylori was noted and it association with characteristics of the children studied was noted employing chi squar

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Kharchenko, Aleksandr V., Nataliya V. Kharchenko, Petro M. Makarenko, Lyudmyla M. Sakharova, Pavlo V. Khomenko та Olga V. Kvak. "Statistical analysis of the сhronic gastritis in students". Wiadomości Lekarskie 73, № 2 (2020): 360–64. http://dx.doi.org/10.36740/wlek202002129.

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The aim: The aim of the study is a statistical analysis of the mucosa of the stomach affected by Helicobacter pylori in young people studying at the university. Materials and methods: The work contains the results of the study of chronic gastritis of type B in university volunteer students. The study was attended by students of 1-4 courses, aged 17 to 25 years, a total of 50 people. Among them were 28 men and 22 women. Results: Various forms of chronic gastritis were found in the mucosa of the topographic-anatomical sections of the stomach, 90% of which were associated with Helicobacter pylori

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Krishnamurthy, Partha, Mary Parlow, Jason B. Zitzer, et al. "Helicobacter pylori Containing Only Cytoplasmic Urease Is Susceptible to Acid." Infection and Immunity 66, no. 11 (1998): 5060–66. http://dx.doi.org/10.1128/iai.66.11.5060-5066.1998.

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ABSTRACT Helicobacter pylori, an important etiologic agent in a variety of gastroduodenal diseases, produces large amounts of urease as an essential colonization factor. We have demonstrated previously that urease is located within the cytoplasm and on the surface of H. pylori both in vivo and in stationary-phase culture. The purpose of the present study was to assess the relative contributions of cytoplasmic and surface-localized urease to the ability of H. pylori to survive exposure to acid in the presence of urea. Toward this end, we compared the acid resistance in vitro of H. pylori cells

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Manyakina, O. M., I. S. Akkuratova-Maksimova, T. G. Pukhova, and A. S. Shitova. "Role of genetic structure of Helicobacter Pylori in formation of chronic inflammatory process in gastric mucosa." Perm Medical Journal 38, no. 1 (2021): 87–99. http://dx.doi.org/10.17816/pmj38187-99.

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Abstract (sommario):

The literature review highlights the questions of the interaction of Helicobacter pylori and the human body. Modern data on the structure of the pathogenicity island in the Helicobacter pylori genome are presented. There is given a detailed description of both well-known virulence and pathogenicity factors of the infection (genes encoding the formation of urease subunits, in particular urel, cytotoxin associated gene A, vacuolating cytotoxin gen A, blood group associated binding adhesion, induced by contact with epithelium) and less studied ones (sialic acid-binding adhesion, adhesion-associat

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Goto, Takayuki, Akira Nishizono, Toshio Fujioka, Junko Ikewaki, Kumato Mifune, and Masaru Nasu. "Local Secretory Immunoglobulin A and Postimmunization Gastritis Correlate with Protection against Helicobacter pylori Infection after Oral Vaccination of Mice." Infection and Immunity 67, no. 5 (1999): 2531–39. http://dx.doi.org/10.1128/iai.67.5.2531-2539.1999.

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ABSTRACT C57BL/6 mice were orally immunized with five weekly doses of 2 mg, 200 μg, or 2 μg of Helicobacter pylori (Sydney strain) whole-cell sonicate combined with cholera toxin. One week after the last vaccination, mice were challenged with 5 × 107CFU of live H. pylori three times at 2-day intervals. At 6 or 18 weeks after the challenge, mice were sacrificed and bacterial cultures and histological studies of the stomach were performed. Vaccination with 2 mg/session or 200 μg/session inhibited H. pylori colonization by 90 and 100%, respectively. These mice were considered protected. Lower lev

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Yamaoka, Yoshio, Kazuyoshi Yamauchi, Hiroyoshi Ota, et al. "Natural History of Gastric Mucosal Cytokine Expression in Helicobacter pylori Gastritis in Mongolian Gerbils." Infection and Immunity 73, no. 4 (2005): 2205–12. http://dx.doi.org/10.1128/iai.73.4.2205-2212.2005.

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ABSTRACT Data regarding the chronological changes in gastric mucosal cytokines in the different phases of Helicobacter pylori infection are unavailable. We examined Mongolian gerbils for up to 52 weeks after H. pylori (ATCC 43504) inoculation. Levels of mRNAs of mucosal cytokines (interleukin-1β [IL-1β], gamma interferon [IFN-γ], IL-4, IL-6, and IL-10) were assessed using real-time reverse transcription-PCR. Starting 26 weeks after H. pylori inoculation, two clinicohistologic patterns appeared: gastric ulcers in 32% and hyperplastic polyps in 68% of gerbils. High levels of mucosal IL-1β mRNA w

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Terebiznik, M. R., C. L. Vazquez, K. Torbicki, et al. "Helicobacter pylori VacA Toxin Promotes Bacterial Intracellular Survival in Gastric Epithelial Cells." Infection and Immunity 74, no. 12 (2006): 6599–614. http://dx.doi.org/10.1128/iai.01085-06.

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Abstract (sommario):

ABSTRACT Helicobacter pylori colonizes the gastric epithelium of at least 50% of the world's human population, playing a causative role in the development of chronic gastritis, peptic ulcers, and gastric adenocarcinoma. Current evidence indicates that H. pylori can invade epithelial cells in the gastric mucosa. However, relatively little is known about the biology of H. pylori invasion and survival in host cells. Here, we analyze both the nature of and the mechanisms responsible for the formation of H. pylori's intracellular niche. We show that in AGS cells infected with H. pylori, bacterium-c

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FAISAL, NABIHA, MUHAMMAD MANSOOR UL HAQ, HAFEEZULLAH SHAIKH, Pervez Ashraf, and Jamila H. Esmail. "HELICOBACTER PYLORI INFECTION;." Professional Medical Journal 19, no. 02 (2012): 202–7. http://dx.doi.org/10.29309/tpmj/2012.19.02.2011.

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Objective: To determine the frequency of H. pylori infection in dyspeptic patients undergoing endoscopy at a tertiary care centerin Karachi. Data source: Patients undergoing endoscopy at Liaquat National Hospital, Karachi. Design of study: Cross sectional descriptivestudy. Setting: Department of Gastroenterology, Liaquat National Hospital, Karachi. Period: May 2008–October 2008. Material andmethods: All adult patients with symptoms of dyspepsia for more than 1 month duration were included. Patients with upper gastrointestinalbleed, anemia or weight loss were excluded. Upper gastrointestinal en

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Alomar, Hatun A., Noha Fathallah, Marwa M. Abdel-Aziz, Taghreed A. Ibrahim, and Wafaa M. Elkady. "GC-MS Profiling, Anti-Helicobacter pylori, and Anti-Inflammatory Activities of Three Apiaceous Fruits’ Essential Oils." Plants 11, no. 19 (2022): 2617. http://dx.doi.org/10.3390/plants11192617.

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Abstract (sommario):

Eradication of Helicobacter pylori is a challenge due to rising antibiotic resistance and GIT-related disorders. Cuminum cyminum, Pimpinella anisum, and Carum carvi are fruits belonging to the Apiaceae family. Their essential oils were extracted, analyzed using GC-MS, tested for anti-H. pylori activity by a micro-well dilution technique, identified for potential anti-H. pylori inhibitors by an in-silico study, and investigated for anti-inflammatory activity using a COX-2 inhibition assay. Results showed that the main components of C. cyminum, P. anisum, and C. carvi were cumaldehyde (41.26%),

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Lee, Sun Eun, Joo Weon Lim, Jung Mogg Kim, and Hyeyoung Kim. "Anti-Inflammatory Mechanism of Polyunsaturated Fatty Acids inHelicobacter pylori-Infected Gastric Epithelial Cells." Mediators of Inflammation 2014 (2014): 1–12. http://dx.doi.org/10.1155/2014/128919.

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Abstract (sommario):

Helicobacter pyloriis an important risk factor for gastric inflammation, which is mediated by multiple signaling pathways. The aim of this study was to investigate the effects of polyunsaturated fatty acids (PUFAs), such as linoleic acid (LA), alpha-linolenic acid (ALA), and docosahexaenoic acid (DHA), on the expression of the proinflammatory chemokine interleukin-8 (IL-8) inH. pylori-infected gastric epithelial AGS cells. To investigate whether PUFAs modulateH. pylori-induced inflammatory signaling, we determined the activation of epidermal growth factor receptor (EGFR), protein kinase C-δ(PK

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Maylyan, Eduard A., Elena S. Zhadan, Andrey E. Bagriy, Olga A. Trunova та Evgeny V. Prokhorov. "Serum levels of 25-hydroxyvitamin D and interleukin-1β in Helicobacter pylori-infected women with postmenopausal osteoporosis". Человек и его здоровье 26, № 2 (2023): 59–65. http://dx.doi.org/10.21626/vestnik/2023-2/07.

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Abstract (sommario):

Dysfunction of the gastrointestinal system in Helicobacter pylori lesions significantly reduces the absorption of not only calcium (Ca), but also vitamin D and other nutrients, the intake of which is critical for bone formation. H. pylory can contribute to the formation of osteoporosis and due to the development of the inflammatory process. The inflammatory process is an important component of the pathogenesis of the above bone disease. Objective: to study of the levels of 25(OH)D and interleukin-1β in blood serum in postmenopausal OP in women with a positive test for antibodies to the Helicob

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Yeniova, Abdullah Özgür, Metin Kucukazman, Naim Ata, et al. "The Relationship betweenHelicobacter pyloriand Beta-2 Microglobulin in Humans." BioMed Research International 2014 (2014): 1–5. http://dx.doi.org/10.1155/2014/615089.

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Abstract (sommario):

H. pyloriis related to various gastrointestinal diseases.β2Microglobulin (β2M) is an intrinsic element of major histocompatibility complex (MHC I). Serumβ2M level may increase in inflammatory states. The aim of current study is to evaluate the relationship betweenβ2M andH. pyloribearing CagA strains.Methods.H. pyloristatus was determined by histopathology of samples taken from stomach. CagA status andβ2M level were measured from blood samples of patients. Eradication therapy was administered to the patients withH. pyloriinfection.β2Microglobulin levels were measured before and after treatment.

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Nisa, Intan Chairun, and Brilliant Margalin. "Optimasi dan uji efektivitas ekstrak Ganoderma lucidum sebagai anti-Helicobacter pylori." Bioma : Jurnal Ilmiah Biologi 10, no. 2 (2021): 217–28. http://dx.doi.org/10.26877/bioma.v10i2.8236.

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Abstract (sommario):

Helicobacter pylori diketahui sebagai penyebab utama tukak lambung dengan melemahkan lapisan pelindung pada lambung dan duodenum. Sejumlah obat anti tukak lambung yang sering digunakan dapat menyebabkan resistensi pada H. pylori. Ganoderma lucidum diketahui dapat menghambat dan mendukung penyembuhan tukak lambung yang disebabkan oleh asam asetat. Akan tetapi, kemampuan G. lucidum dalam menghambat tukak lambung yang disebabkan H. pylori belum banyak diungkap. Penelitian ini bertujuan untuk mengetahui efektivitas ekstrak G. lucidum dalam menghambat pertumbuhan H. pylori penyebab tukak lambung. P

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. "NSAID, gebruikNSAID-gebruik en H. pylori, tetracycline H. pylori, naproxen H. pylori, metronidazol H. pylori, bismutoxideH. Pylori-eradicatie." Medisch-Farmaceutische Mededelingen 38, no. 4 (2000): 72. http://dx.doi.org/10.1007/bf03057514.

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Shahbazi, Shaahin, Zahra Vahdat Shariatpanahi, and Nourkhoda Sadeghifard. "Effect of Proton Pump Inhibitor Based Triple Therapy with Apple Cider Vinegar on Helicobacter Pylori Eradication." Galen Medical Journal 3, no. 2 (2014): 90–94. http://dx.doi.org/10.31661/gmj.v3i2.97.

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Abstract (sommario):

Background: The purpose of this study was to evaluate the effect of apple cider vinegar plus PPI-based triple therapy on eradication of Helicobacter Pylori (H.pylori) infection.Materials and Methods: 116 patients with H. pylori infection were included in this randomized clinical trial. Patients in control group (n=59) received amoxicillin, clarithromycin, and pantaprazole twice daily. Patients in study group (57 patients) received the same regimen plus 30 mL of apple cider vinegar twice daily. The duration of therapy for study and control were 10 and 14 days respectively. Four weeks after the

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NICOLAYSEN, J. "Carcinoma pylori; resectio pylori; död." Nordiskt Medicinskt Arkiv 13, no. 27 (2009): 1–12. http://dx.doi.org/10.1111/j.0954-6820.1881.tb00952.x.

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47

Kuo, Chao-Hung, Chien-Yu Lu, Yuan-Chieh Yang, et al. "Does Long-Term Use of Silver Nanoparticles Have Persistent Inhibitory Effect onH. pyloriBased on Mongolian Gerbil’s Model?" BioMed Research International 2014 (2014): 1–7. http://dx.doi.org/10.1155/2014/461034.

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Abstract (sommario):

Background. It is urgent to find alternative agents due to increasing failure rate ofHelicobacter pylori (H. pylori) eradication. The study surveyed the long-term effect of silver nanoparticles (AgNP) onH. pyloribased on Mongolian gerbil’s model.Materials and Methods. Fifty gerbils were randomly allocated to six groups (A–F). Group (Gr) A: the gerbils were fed with broth; Gr B and D: the gerbils were fed with AgNP/clay complex (0.1% of weight); Gr C and E: the gerbils were fed with AgNP/clay complex(1% of weight); and Gr D, E, and F: the gerbils were inoculated withH. pylori. At the 20th exper

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Bauer, Bianca, and Thomas F. Meyer. "The Human Gastric Pathogen Helicobacter pylori and Its Association with Gastric Cancer and Ulcer Disease." Ulcers 2011 (July 18, 2011): 1–23. http://dx.doi.org/10.1155/2011/340157.

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Abstract (sommario):

With the momentous discovery in the 1980's that a bacterium, Helicobacter pylori, can cause peptic ulcer disease and gastric cancer, antibiotic therapies and prophylactic measures have been successful, only in part, in reducing the global burden of these diseases. To date, ~700,000 deaths worldwide are still attributable annually to gastric cancer alone. Here, we review H. pylori's contribution to the epidemiology and histopathology of both gastric cancer and peptic ulcer disease. Furthermore, we examine the host-pathogen relationship and H. pylori biology in context of these diseases, focusin

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O'Connor, Pamela M., Tamia K. Lapointe, Shannon Jackson, Paul L. Beck, Nicola L. Jones, and Andre G. Buret. "Helicobacter pylori Activates Calpain via Toll-Like Receptor 2 To Disrupt Adherens Junctions in Human Gastric Epithelial Cells." Infection and Immunity 79, no. 10 (2011): 3887–94. http://dx.doi.org/10.1128/iai.05109-11.

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Abstract (sommario):

ABSTRACTHelicobacter pyloriis a risk factor for the development of gastritis, gastroduodenal ulcers, and gastric adenocarcinoma.H. pylori-induced disruption of epithelial adherens junctions (AJs) is thought to promote the development of severe disease; however, the mechanisms wherebyH. pylorialters AJ structure remain incompletely understood. The present study demonstrates thatH. pyloriinfection in human patients is associated with elevated serum levels of an 80-kDa E-cadherin ectodomain, whose presence is independent of the presence of serum antibodies against CagA.In vitro, a heat-labileH. p

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Turdieva, Sh T., and E. A. Shamansurova. "Features of the endoscopic picture in paediatric gastroduodenal diseases caused by Helicobacter pylori." Russian Journal of Infection and Immunity 10, no. 3 (2020): 543–50. http://dx.doi.org/10.15789/2220-7619-fot-1249.

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Abstract (sommario):

The aim of the study was to examine the features of the endoscopic picture in the upper digestive tract mucosa in paediatric chronic gastroduodenal pathology (CGDP) associated with Helicobacter pylori. Materials and methods. There were examined 286 patients, aged 6–15 years. Diagnostic criteria for chronic gastroduodenal pathology were anamnestic as well as instrumental and functional studies data: gastric fractional intubation, esophagogastroduodenoscopy (EPGDS) with endoscopic pH-metry without biopsy, and ultrasound examination of abdominal organs. H. pylori testing was carried out by two un

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