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Tesi sul tema "Receptors de tipus Toll"

Autore: Grafiati

Pubblicato: 4 giugno 2021

Ultima modifica: 12 febbraio 2022

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1

Montserrat, i. Sangrà Sara. "Ex vivo and in vitro studies on Toll like receptors in canine Leishmania infection." Doctoral thesis, Universitat Autònoma de Barcelona, 2018. http://hdl.handle.net/10803/665953.

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La leishmaniosis canina (CanL) es una enfermedad causada por el protozoa Leishmania infantum. Las manifestaciones clínicas en el perro son muy variables, abarcan desde una infección subclínica a un cuadro severo, que puede ser fatal. Al ser una enfermedad compleja, es difícil de diagnosticar. La respuesta inmunitaria de cada individuo juega un papel muy importante, tanto para el diagnóstico como para su tratamiento. Los receptores tipo Toll (TLRs) son los principales iniciadores de la respuesta inmunitaria innata cuya función es frenar cualquier intrusión de agentes extraños al cuerpo. Sin emb

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2

Estévez, Medina Javier. "Toll-like receptors as modulators of intestinal barrier function." Doctoral thesis, Universitat Autònoma de Barcelona, 2016. http://hdl.handle.net/10803/400146.

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Las patologías funcionales (síndrome del intestino irritable, IBS) e inflamatorias gastrointestinales (enfermedad inflamatoria intestinal, IBD) se caracterizan por alteraciones de la función barrera epitelial, con un aumento de la permeabilidad, y cambios en la microbiota intestinal. Los receptores de tipo Toll (TLRs) participan en el reconocimiento bacteriano en el intestino y en el control neuroinmune local, estando, por tanto, implicados en la regulación de la función barrera del epitelio intestinal. El objetivo de este trabajo ha sido caracterizar la implicación de los receptores TLR5 y

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3

Weber, Alexander Norman Rainer. "Structural and functional studies of Drosophila toll and human toll-like receptors." Thesis, University of Cambridge, 2004. http://ethos.bl.uk/OrderDetails.do?uin=uk.bl.ethos.615987.

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4

Irvine, Katherine Lucy. "The pharmacology of equine toll-like receptors." Thesis, University of Cambridge, 2013. http://ethos.bl.uk/OrderDetails.do?uin=uk.bl.ethos.608103.

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5

Offord, Victoria Anne. "Toll-like receptors : from sequence to structure." Thesis, Royal Veterinary College (University of London), 2015. http://ethos.bl.uk/OrderDetails.do?uin=uk.bl.ethos.669195.

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6

McIlroy, Graham William. "Toll-7 and Toll-6 : central nervous system functions as Drosophila neurotrophin receptors." Thesis, University of Birmingham, 2012. http://etheses.bham.ac.uk//id/eprint/3098/.

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The Drosophila Toll receptor is crucial for dorsoventral patterning in embryos, and for innate immunity. Toll also functions during central nervous system development, promoting neuronal survival and targeting. There are nine Toll paralogues in Drosophila, and it is unknown whether any of these also function in the CNS. Toll’s ligand, Spz, has an NGF domain. NGF is a vertebrate neurotrophin - a growth factor that regulates the development and function of the nervous system. Drosophila Neurotrophin 1 (DNT1), identified by homology to the vertebrate neurotrophin BDNF, and DNT2 are paralogues of

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7

Dessing, Mark Christianus. "Toll-like receptors and innate immunity in pneumonia." [S.l. : Amsterdam : s.n.] ; Universiteit van Amsterdam [Host], 2007. http://dare.uva.nl/document/47971.

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8

Kassem, Ali. "Toll-like receptors (TLRs) and inflammatory bone modeling." Doctoral thesis, Umeå universitet, Institutionen för odontologi, 2015. http://urn.kb.se/resolve?urn=urn:nbn:se:umu:diva-110296.

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Patients with inflammatory or infectious conditions such as periodontitis, peri-implantitis, osteomyelitis, rheumatoid arthritis, septic arthritis and loosened joint prosthesis display varying severity of destruction in the adjacent bone tissue. Bone loss in inflammatory diseases is considered a consequence of cytokine induced RANKL and subsequent enhanced osteoclast formation. Hence, osteotropic cytokines and their receptors have been suggested to be important for the pathogenesis of inflammation-induced osteolysis. It is, here, suggested that bacterial components, so called “pathogen associa

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9

Philbin, Victoria Jane. "Identification & characterisation of avian toll-like receptors." Thesis, University of Oxford, 2006. http://ethos.bl.uk/OrderDetails.do?uin=uk.bl.ethos.425894.

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10

Cant, Rachel. "Modulation of toll like receptors by naltrexone hydrochloride." Thesis, St George's, University of London, 2017. https://ethos.bl.uk/OrderDetails.do?uin=uk.bl.ethos.753993.

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Abstract (sommario):

Naltrexone is an opioid antagonist usually used in the treatment of patients addicted to drugs or alcohol. However, since the 1980s naltrexone has been used at lower doses to treat patients with cancer and autoimmune diseases such as multiple sclerosis and Crohn’s disease. The mechanism of action of naltrexone in treating these diseases is unknown, however, evidence suggests that the drug has immune modulating effects. Toll like receptors (TLR) are type I membrane receptors that are crucial in the innate immune response. TLR recognise exogenous and endogenous ligands to induce the production o

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11

Patole, Prashant. "Nucleic acid specific Toll-like receptors in lupus nephritis." Diss., lmu, 2006. http://nbn-resolving.de/urn:nbn:de:bvb:19-52560.

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12

Gong, Jing. "Databases and computational interaction models of Toll-like receptors." Diss., lmu, 2010. http://nbn-resolving.de/urn:nbn:de:bvb:19-115612.

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13

Braedel-Ruoff, Sibylla. "Toll-like receptors - link between innate and adaptive immunity." [S.l. : s.n.], 2007.

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14

Lalanne, Ana. "Regulation of murine β lymphopoiesis by Toll like receptors". Paris 7, 2008. http://www.theses.fr/2008PA077220.

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Abstract (sommario):

Les récepteurs Toll like sont connus pour protéger de l'invasion des pathogènes et pour renforcer les réponses immunitaires. Notre objectif était d'étudier si le signal des TLRs est capable de réguler la lymphopoïèse B chez la souris. Nos résultats ont montré que les ligands de TLR2, TLR7 et TLR9 inhibent l'expansion in vitro des cellules pro-B. Nous avons étudié plus spécifiquement le TLR9, récepteur à l'ADN non méthylé contenant des motifs CpG. Le CpG inhibe l'expansion des cellules pro-B, mais n'a aucun effet sur les cellules pré-B. Le CpG inhibe l'expansion des cellules pro-B non pas par d

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15

Aboussahoud, Wedad. "The role of toll-like receptors in assisted conception." Thesis, University of Manchester, 2016. https://www.research.manchester.ac.uk/portal/en/theses/the-role-of-tolllike-receptors-in-assisted-conception(230bd2c9-e8bb-490f-b63f-9376667da89c).html.

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Background: In the last two decades the discovery of the Toll-Like Receptor (TLR) family in humans revealed the importance of innate immunity in providing host defence against invading pathogens. Moreover, TLRs have a vital role in mediating the interactions between the reproductive and immune systems. Similar to TLRs, (NOD)-like receptors (NLRs); retinoic acid-inducible gene-1, RIG-1-like receptors (RLRs) are also important pattern recognition receptors in the female reproductive system. Successful implantation requires effective reciprocal interactions between receptive endometrium and compe

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16

Eckl-Dorna, J. "How B cell receptors and Toll-like receptors collaborate in shaping B cell responses." Thesis, University College London (University of London), 2009. http://discovery.ucl.ac.uk/18762/.

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Antigen recognition by B cells results in their activation followed by specific antibody production. These events are initiated by antigen binding to their surface B cell receptors (BCR) which triggers both signalling and internalization of the receptor bound antigen to the endosome. However B cells also express features of the innate immune system such as Toll like receptors (TLRs), that can be located either on the surface of the cell or intracellularly where they recognize bacterial and viral nucleic acids. Engagement of these receptors within B cells is associated with enhancement of humor

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17

Chang, Serena Soyoung Yunmee. "Toll-Like Receptors: Target of Hepatitis C Virus: A Dissertation." eScholarship@UMMS, 2008. https://escholarship.umassmed.edu/gsbs_diss/386.

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Hepatitis C Virus (HCV) is the primary cause of liver transplantation due to its chronic nature in up to eighty percent of infected cases. Around 3 percent of the world’s population is infected with HCV. Treatment for HCV is a combined Ribavirin and interferon-α (IFN-α) therapy effective in only fifty to eighty percent of patients depending on HCV genotype. The growing health concern with this disease is the lack of a cure despite liver transplantation. HCV targets hepatocytes, liver cells, but is not cytolytic. HCV has been shown to induce end stage liver disease through sustained inflammatio

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18

Burkey, Thomas Edward. "Expression of toll-like receptors in porcine immune cells and tissues." Diss., Manhattan, Kan. : Kansas State University, 2006. http://hdl.handle.net/2097/153.

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19

Cao, Fei. "Chlamydia pneumoniae, toll-like receptors and pathogenesis of atherosclerotic heart disease." View the abstract Download the full-text PDF version (on campus access only), 2007. http://etd.utmem.edu/ABSTRACTS/2007-022-Cao-index.html.

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Thesis (Ph.D. )--University of Tennessee Health Science Center, 2007.<br>Title from title page screen (viewed on May 16, 2008 ). Research advisor: Gerald I. Byrne, Ph.D. Document formatted into pages (xi, 114 p. : ill.). Vita. Abstract. Includes bibliographical references (p. 65-107).

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20

Sang, Yongming. "Porcine innate antiviral immunity : host defense peptides and toll-like receptors." Diss., Manhattan, Kan. : Kansas State University, 2008. http://hdl.handle.net/2097/960.

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21

Kanczkowski, Waldemar, Kai Zacharowski, and Stefan R. Bornstein. "Role of Toll-Like Receptors and Inflammation in Adrenal Gland Insufficiency." Saechsische Landesbibliothek- Staats- und Universitaetsbibliothek Dresden, 2014. http://nbn-resolving.de/urn:nbn:de:bsz:14-qucosa-135727.

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Adrenal gland insufficiency – the clinical manifestation of deficient production or action of adrenal steroids – is a life-threatening disorder. Among many factors which can predispose to primary adrenal failure, an autoimmune adrenalitis and infectious agents play a major role. The initial host defense against bacterial infections is executed primarily by the pattern recognition receptors, e.g. Toll-like receptors (TLRs), expressed in cells from the innate immune system. Upon activation, TLRs have been found to regulate various levels of innate and adaptive immunity as well as control tissue

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22

Fiola, Stéphanie. "Reconnaissance moléculaire du virus Epstein-Barr par les «Toll-like receptors»." Thesis, Université Laval, 2010. http://www.theses.ulaval.ca/2010/26899/26899.pdf.

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23

Oakes, N. "The expression of toll-like receptors in B-chronic lymphocytic leukaemia." Thesis, University of the West of England, Bristol, 2015. http://eprints.uwe.ac.uk/25452/.

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Chronic Lymphocytic Leukaemia (CLL) is the most common form of Leukaemia in the Western world and has a highly variable clinical course. Continuing advances in the range of therapeutic options available to clinicians require reliable prognostic indicators that can be used to group patients accurately according to their risk of disease progression, thereby allowing meaningful comparisons of treatments. The expression of Toll-Like Receptors (TLR) on cells involved with the disease process in CLL was studied to establish links between levels of expression and the disease process. The expression l

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24

Asquith, Mark. "Toll-like receptors in the induction and regulation of intestinal inflammation." Thesis, University of Oxford, 2010. http://ethos.bl.uk/OrderDetails.do?uin=uk.bl.ethos.526567.

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25

Kanczkowski, Waldemar, Kai Zacharowski, and Stefan R. Bornstein. "Role of Toll-Like Receptors and Inflammation in Adrenal Gland Insufficiency." Karger, 2010. https://tud.qucosa.de/id/qucosa%3A27670.

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Abstract (sommario):

Adrenal gland insufficiency – the clinical manifestation of deficient production or action of adrenal steroids – is a life-threatening disorder. Among many factors which can predispose to primary adrenal failure, an autoimmune adrenalitis and infectious agents play a major role. The initial host defense against bacterial infections is executed primarily by the pattern recognition receptors, e.g. Toll-like receptors (TLRs), expressed in cells from the innate immune system. Upon activation, TLRs have been found to regulate various levels of innate and adaptive immunity as well as control tissue

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26

Huhta, H. (Heikki). "Toll-like receptors in Alimentary tract -special reference to Barrett’s esophagus." Doctoral thesis, Oulun yliopisto, 2016. http://urn.fi/urn:isbn:9789526213392.

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Abstract Incidence of esophageal adenocarcinoma is rising rapidly in Western countries. The main risk factor for esophageal adenocarcinoma is Barrett’s esophagus. Barrett’s esophagus results from long-term gastroesophageal reflux disease. The gastrointestinal tract is colonized by bacteria, fungi and viruses forming the alimentary tract microbiome. Microbiome transformation is involved in pathogenesis of alimentary tract cancer and also in the development of Barrett’s metaplasia. Toll-like receptors (TLR) are molecules of the innate immune system and they are involved in bacterial and viral re

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27

Mathew, Manu. "Characterisation of antimicrobial peptides and toll-like receptors in amniotic membranes." Thesis, University of Nottingham, 2016. http://ethos.bl.uk/OrderDetails.do?uin=uk.bl.ethos.716469.

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PURPOSE- Antimicrobial peptides (AMPs) are cationic host defence peptides with microbicidal properties providing a "chemical barrier" in response to pathogens and various pathological states. They show promise as potential therapeutic agents. Amniotic membrane (AM) transplantation is indicated in ocular surface surgery procedure. However, little is known of the AMP profile and variability within the AM. In addition, the effects of processing, storage, and preoperative preparation on AMPs in the transplant ready amniotic membrane (TRAM) are not clear. This can alter the therapeutic potential of

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28

Wang, Jixin. "Bioinformatic analysis of chicken chemokines, chemokine receptors, and Toll-like receptor 21." Texas A&M University, 2006. http://hdl.handle.net/1969.1/4212.

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Chemokines triggered by Toll-like receptors (TLRs) are small chemoattractant proteins, which mainly regulate leukocyte trafficking in inflammatory reactions via interaction with G protein-coupled receptors. Forty-two chemokines and 19 cognate receptors have been found in the human genome. Prior to this study, only 11 chicken chemokines and 7 receptors had been reported. The objectives of this study were to identify systematically chicken chemokines and their cognate receptor genes in the chicken genome and to annotate these genes and ligand-receptor binding by a comparative genomics approach.

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29

Burgueño, Banús Juan Francisco. "Understanding the role of Toll-like receptors in the lower gastrointestinal tract." Doctoral thesis, Universitat Autònoma de Barcelona, 2014. http://hdl.handle.net/10803/134668.

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La mucosa del tracte gastrointestinal (TGI) constitueix la superfície de contacte més àmplia de l’organisme amb el medi extern. A la llum del TGI, la barrera epitelial permet la separació entre els bilions de microorganismes residents i els immunòcits. En aquest context, el manteniment de la tolerància a la microbiota resident és essencial per a preservar l’homeòstasi; qualsevol esdeveniment que causi la desregulació d’aquestes interaccions pot desencadenar respostes pro-inflamatòries com les observades a la malaltia inflamatòria intestinal (MII). Els receptors Toll-like (TLR) regulen el diàle

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30

Seibl, Reinhart. "Toll-like receptors: Studies on cellular activation and involvement in rheumatoid arthritis /." Zürich, 2003. http://e-collection.ethbib.ethz.ch/show?type=diss&nr=15257.

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31

Lunazzi, Giulia 1981. "Analysis of NFAT5 expression and activity in response to toll-like receptors." Doctoral thesis, Universitat Pompeu Fabra, 2013. http://hdl.handle.net/10803/666120.

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Stimulation of Toll-like receptors (TLRs) in cells of the innate immune system activates the expression of a proinflammatory and antimicrobial gene program controlled by a network of transcriptional regulators. We show that NFAT5, which belongs to the Rel family of transcription factors and was previously characterized as an osmostress responsive factor, is required for the expression of a group of TLR-responsive genes in macrophages, such as Nos2, Il6 and Tnf. NFAT5 recruitment to its target genes is dependent on IKKβ activity, de novo protein synthesis and is sensitive to histone deacetylase

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32

Jorgenson, Rebecca L. "The innate immune response and toll-like receptors in the human endometrium." Diss., Columbia, Mo. : University of Missouri-Columbia, 2005. http://hdl.handle.net/10355/4178.

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Thesis (Ph. D.)--University of Missouri-Columbia, 2005.<br>The entire dissertation/thesis text is included in the research.pdf file; the official abstract appears in the short.pdf file (which also appears in the research.pdf); a non-technical general description, or public abstract, appears in the public.pdf file. Vita. "December 2005" Includes bibliographical references.

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33

Bäckhed, Fredrik. "Role of toll-like receptors in host responses to mucosal bacterial infections /." Stockholm, 2002. http://diss.kib.ki.se/2002/91-7349-367-8/.

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34

Pupjain, Srijan. "The impact of Toll-like receptors on antigen presentation by mouse macrophages." Thesis, University of Newcastle Upon Tyne, 2008. http://ethos.bl.uk/OrderDetails.do?uin=uk.bl.ethos.491833.

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Toll-like receptors (TLRs) are a group of trans-membrane proteins playing important roles as pattern-recognition receptors (PRRs) of innate immunity that are mainly expressed on professional antigen presenting cells (APCs). Stimulation of APC with TLR ligands, a variety of Jnolecular components derived from microorganisms, triggers innate immunity and establishes adaptive immune responses. I studied the impact of TLR specific for bacterial components on antigen processing and presentation of protective antigen (PA) of B. anthracis by bone marrow macrophages from mice.

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35

Morris, Gavin Edward. "Mechanisms of airway smooth muscle activation by agonists of toll-like receptors." Thesis, University of Sheffield, 2005. http://ethos.bl.uk/OrderDetails.do?uin=uk.bl.ethos.425196.

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36

Lacavé-Lapalun, Jean-Victor. "Réponse immunitaire induite par l’irradiation colorectale : manipulation thérapeutique des « toll like receptors »." Paris 6, 2013. http://www.theses.fr/2013PA066249.

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L’exposition aux rayonnements ionisants de la sphère abdomino-pelvienne est associée à une haute incidence de complications. Lors des cas les plus importants, l’apparition d’ulcérations peut entrainer le décès des patients en absence de lourds traitements. Dans ces cas, des essais cliniques sont réalisés avec des Cellules Souches Mésenchymateuses (CSM). Les radiothérapies abdomino-pelvienne peuvent provoquer à court et/ou long terme des effets délétères. Des études démontrent que l’injection de motifs bactériens confère une radioprotection au niveau intestinal. Ils stimulent des récepteurs (To

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37

Chumble, Anuja. "Epigenetic Alterations of Toll-Like Receptors by TET2 in Spontaneous Preterm Labor." VCU Scholars Compass, 2014. http://scholarscompass.vcu.edu/etd/3469.

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Increasing evidence implicates the presence of bacteria in intrauterine tissues as an important risk factor for spontaneous preterm labor. Epigenetic alterations of innate immunity genes may increase the mother’s sensitivity to subclinical levels of bacteria. This study examined the presence of TET2, TLR-2, and TLR-9 in intrauterine tissue, and evaluated whether epigenetic alterations of these genes, as well as IL-8, changed their expression in human decidual tissue and a macrophage cell culture. Immunohistochemicalstaining was used to detect the presence of these proteins in intrauterine tiss

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38

Ahmadi, Farhana. "Interaction of toll-like receptor 4 with the adaptor proteins MAL and TRAM." Thesis, University of Cambridge, 2012. http://ethos.bl.uk/OrderDetails.do?uin=uk.bl.ethos.610209.

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Lange, Margaret J. "Hormonal regulation of innate immune responses and toll-like receptors in the human endometrium." Diss., Columbia, Mo. : University of Missouri-Columbia, 2008. http://hdl.handle.net/10355/5606.

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Thesis (Ph. D.)--University of Missouri-Columbia, 2008.<br>"May 2008" The entire dissertation/thesis text is included in the research.pdf file; the official abstract appears in the short.pdf file (which also appears in the research.pdf); a non-technical general description, or public abstract, appears in the public.pdf file. Vita. Includes bibliographical references.

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Lin, Miao, and 林苗. "The critical role of toll-like receptor 4 in diabetic nephropathy." Thesis, The University of Hong Kong (Pokfulam, Hong Kong), 2011. http://hub.hku.hk/bib/B47246637.

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Baoprasertkul, Puttharat Liu Zhanjiang. "Characterization of innate immune genes of catfish CXC chemokines and toll-like receptors /." Auburn, Ala., 2006. http://repo.lib.auburn.edu/2006%20Spring/doctoral/BAOPRASERTKUL_PUTTHARAT_14.pdf.

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42

Dzaye, Omar [Verfasser]. "Investigating the role of toll-like receptors in the glioma microenvironment / Omar Dzaye." Berlin : Medizinische Fakultät Charité - Universitätsmedizin Berlin, 2016. http://d-nb.info/1119803799/34.

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Hu, Feng [Verfasser]. "Dissecting the role of Toll-like receptors in microglia-glioma crosstalk / Feng Hu." Berlin : Medizinische Fakultät Charité - Universitätsmedizin Berlin, 2014. http://d-nb.info/1062537041/34.

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44

Sadangi, Chinmaya [Verfasser], and G. [Akademischer Betreuer] Schratt. "Role of endosomal toll-like receptors in epilepsy / Chinmaya Sadangi ; Betreuer: G. Schratt." Marburg : Philipps-Universität Marburg, 2018. http://d-nb.info/1156461723/34.

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45

Voss, Söhnke. "Molecular investigations of agonists and antagonists of Toll-like receptors 2 and 4." [S.l. : s.n.], 2006.

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46

Morale, Mirian Galliote. "Efeito da infecção por HPV nas vias de sinalização por toll like receptors." Universidade de São Paulo, 2016. http://www.teses.usp.br/teses/disponiveis/46/46131/tde-30112016-100636/.

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Abstract (sommario):

As oncoproteínas E6 e E7 do Papilomavírus Humano (HPV) estão envolvidas na desregulação do sistema imune inato, provocando alterações na expressão dos receptores do tipo Toll (TLR). Considerando-se a função da via de sinalização iniciada por TLR, haveria uma vantagem para o vírus capaz de manipular a resposta desta via de modo que possa persistir nas células sem ser detectado pelo sistema imune ou ainda modulando essa resposta e criando um ambiente mais propício à manutenção da infecção. No entanto, muitos dos mecanismos que levam à eliminação da infecção ou persistência do HPV ainda são pouco

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47

Whitmore, Laura Christine. "Neutrophil priming and host inflammation: The roles of NOX2 and toll-like receptors." Diss., University of Iowa, 2014. https://ir.uiowa.edu/etd/3215.

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Neutrophils, essential innate immune cells, recognize danger signals through receptors on their surface. Upon receptor ligation, neutrophils may undergo priming, a process involving limited reactive oxygen species (ROS) generation and partial degranulation. Priming facilitates neutrophil migration and prepares the cell for an enhanced response to a secondary stimulus, including a spike in ROS generation by NADPH oxidase 2 (NOX2). It is well established that NOX2-derived oxidants are involved in pathogen killing and that off-target effects can cause host tissue damage; however, several lines of

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48

Lionel, Sagayaraj Linushiya Ranjani. "Gene Expression Analysis of Loto-Type Toll Receptors in the Onychophoran Euperipatoides kanangrensis." Thesis, Uppsala universitet, Institutionen för geovetenskaper, 2017. http://urn.kb.se/resolve?urn=urn:nbn:se:uu:diva-334158.

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The expression and role of Loto genes was previously investigated in arthropods and it was concluded that the morphogenetic functions of Loto genes were conserved in the last common ancestors of arthropods. In this paper, possible orthologs of Loto clade genes are analysed and it was found that they possibly play a conserved role in convergent extension of cells from the blastoderm stage to germ band extension stage. The differences and similarities in the number of and expression of Loto clade genes in arthropods and onychophorans are compared and analysed. Unlike in arthropods, the expressio

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Jackson, Leila J. "The dynamic regulation of the low affinity IGE receptor by toll like receptor and B cell receptor agonists /." Connect to full text via ProQuest. Limited to UCD Anschutz Medical Campus, 2008.

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Abstract (sommario):

Thesis (Ph.D. in Immunology) -- University of Colorado Denver, 2008.<br>Typescript. Includes bibliographical references (leaves 122-129). Free to UCD Anschutz Medical Campus. Online version available via ProQuest Digital Dissertations;

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50

Liang, Chaofan, and 梁超凡. "Toll-like receptor 4 plays a key role in insulin resistance and endothelial dysfunction." Thesis, The University of Hong Kong (Pokfulam, Hong Kong), 2011. http://hub.hku.hk/bib/B47028920.

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