Letteratura scientifica selezionata sul tema "Skeletal Adaptation"
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Articoli di riviste sul tema "Skeletal Adaptation"
Hibbitt, Catherine. "Using Skeleton Typograms to Explore Comparative Anatomy". American Biology Teacher 82, n. 2 (1 febbraio 2020): 120–22. http://dx.doi.org/10.1525/abt.2020.82.2.120.
Testo completoTurner, Charles H. "Skeletal Adaptation to Mechanical Loading". Clinical Reviews in Bone and Mineral Metabolism 5, n. 4 (dicembre 2007): 181–94. http://dx.doi.org/10.1007/s12018-008-9010-x.
Testo completoRöckl, Katja S. C., Michael F. Hirshman, Josef Brandauer, Nobuharu Fujii, Lee A. Witters e Laurie J. Goodyear. "Skeletal Muscle Adaptation to Exercise Training". Diabetes 56, n. 8 (18 maggio 2007): 2062–69. http://dx.doi.org/10.2337/db07-0255.
Testo completoMcleod, Kenneth J., Clinton T. Rubin, Mark W. Otter e Yi-Xian Qin. "Skeletal Cell Stresses and Bone Adaptation". American Journal of the Medical Sciences 316, n. 3 (settembre 1998): 176–83. http://dx.doi.org/10.1016/s0002-9629(15)40398-2.
Testo completoWang, Y., e J. M. Winters. "Predictive model for skeletal muscle adaptation". Journal of Biomechanics 39 (gennaio 2006): S43. http://dx.doi.org/10.1016/s0021-9290(06)83047-2.
Testo completoBurton, H. W., B. M. Carlson e J. A. Faulkner. "Microcirculatory Adaptation to Skeletal Muscle Transplantation". Annual Review of Physiology 49, n. 1 (marzo 1987): 439–51. http://dx.doi.org/10.1146/annurev.ph.49.030187.002255.
Testo completoCarter, Dennis R., e Tracy E. Orr. "Skeletal development and bone functional adaptation". Journal of Bone and Mineral Research 7, S2 (dicembre 1992): S389—S395. http://dx.doi.org/10.1002/jbmr.5650071405.
Testo completoWarden, Stuart J. "Extreme Skeletal Adaptation to Mechanical Loading". Journal of Orthopaedic & Sports Physical Therapy 40, n. 3 (marzo 2010): 188. http://dx.doi.org/10.2519/jospt.2010.0404.
Testo completoMcLEOD, KENNETH J., CLINTON T. RUBIN, MARK W. OTTER e YI-XIAN QIN. "Skeletal Cell Stresses and Bone Adaptation". American Journal of the Medical Sciences 316, n. 3 (settembre 1998): 176–83. http://dx.doi.org/10.1097/00000441-199809000-00005.
Testo completoYuan, Chong-Xi, Qiang Ji, Qing-Jin Meng, Alan R. Tabrum e Zhe-Xi Luo. "Earliest Evolution of Multituberculate Mammals Revealed by a New Jurassic Fossil". Science 341, n. 6147 (15 agosto 2013): 779–83. http://dx.doi.org/10.1126/science.1237970.
Testo completoTesi sul tema "Skeletal Adaptation"
Ellman, Rachel. "Skeletal adaptation to reduced mechanical loading". Thesis, Massachusetts Institute of Technology, 2014. http://hdl.handle.net/1721.1/107612.
Testo completoCataloged from PDF version of thesis.
Includes bibliographical references (pages 126-139).
Bone adapts its mass and architecture in response to its mechanical environment. Yet control of this process by mechanical cues is poorly understood, particularly for unloading. Defining the fundamental mechano-regulation of bone adaptation is critical for the better understanding and mitigation of bone loss in astronauts as well as clinical conditions such as spinal cord injury, stroke, muscular dystrophy, and bed rest. The overall goal of this work was to study skeletal adaptation to varying amounts of reduced loading to help delineate the relationship between mechanical stimuli and skeletal adaptation. We first examined the relative contribution of muscle and gravitational forces to the maintenance of skeletal health in mice, using botulinum toxin (BTX) to induce muscle paralysis and hindlimb unloading to eliminate external loading on the hindlimbs, alone and in combination. BTX led to greater bone loss than hindlimb unloading, while the combination of interventions led to the most detrimental effects overall, suggesting that both muscle and gravitational forces play a role in skeletal maintenance, with greater contributions from muscle forces. We then characterized skeletal adaptation to controlled reductions in mechanical loading of varying degrees employing a novel model that enables long-term exposure of mice to partial weightbearing (PWB). We found that declines in bone mass and architecture were linearly related to the degree of unloading. Even mice bearing 70% of their body weight exhibited significant bone loss, suggesting that the gravity of the moon (0.16 G) and Mars (0.38 G) will not be sufficient to prevent bone loss on future exploration missions. Finally, since bone remodeling is highly site-specific, we used gait analysis and inverse dynamics to determine the mechanical environment during PWB, and then developed a finite element model of the tibia to resolve the local strain-related stimulus proposed to drive changes in bone mass. We found modest correlations between cortical bone architecture at different PWB levels and strain energy density. Altogether this work provides a critical foundation and rationale for future studies that incorporate detailed quantification of the mechanical stimuli and longitudinal changes in bone architecture to further advance our understanding of the skeletal response to reduced loading.
by Rachel Ellman.
Ph. D. in Medical Engineering and Bioastronautics
Eliman, Rachel. "Skeletal adaptation to reduced mechanical loading". Thesis, Massachusetts Institute of Technology, 2014. http://hdl.handle.net/1721.1/95861.
Testo completoCataloged from PDF version of thesis.
Includes bibliographical references (pages 126-139).
Bone adapts its mass and architecture in response to its mechanical environment. Yet control of this process by mechanical cues is poorly understood, particularly for unloading. Defining the fundamental mechanoregulation of bone adaptation is critical for the better understanding and mitigation of bone loss in astronauts as well as clinical conditions such as spinal cord injury, stroke, muscular dystrophy, and bed rest. The overall goal of this work was to study skeletal adaptation to varying amounts of reduced loading to help delineate the relationship between mechanical stimuli and skeletal adaptation. We first examined the relative contribution of muscle and gravitational forces to the maintenance of skeletal health in mice, using botulinum toxin (BTX) to induce muscle paralysis and hindlimb unloading to eliminate external loading on the hindlimbs, alone and in combination. BTX led to greater bone loss than hindlimb unloading, while the combination of interventions led to the most detrimental effects overall, suggesting that both muscle and gravitational forces play a role in skeletal maintenance, with greater contributions from muscle forces. We then characterized skeletal adaptation to controlled reductions in mechanical loading of varying degrees employing a novel model that enables long-term exposure of mice to partial weightbearing (PWB). We found that declines in bone mass and architecture were linearly related to the degree of unloading. Even mice bearing 70% of their body weight exhibited significant bone loss, suggesting that the gravity of the moon (0.16 G) and Mars (0.38 G) will not be sufficient to prevent bone loss on future exploration missions. Finally, since bone remodeling is highly site-specific, we used gait analysis and inverse dynamics to determine the mechanical environment during PWB, and then developed a finite element model of the tibia to resolve the local strain-related stimulus proposed to drive changes in bone mass. We found modest correlations between cortical bone architecture at different PWB levels and strain energy density. Altogether this work provides a critical foundation and rationale for future studies that incorporate detailed quantification of the mechanical stimuli and longitudinal changes in bone architecture to further advance our understanding of the skeletal response to reduced loading.
by Rachel Eliman.
Ph. D.
Kohn, Tertius A. "Characteristics and adaptation of skeletal muscle to endurance exercise". Thesis, Stellenbosch : University of Stellenbosch, 2011. http://hdl.handle.net/10019.1/16517.
Testo completoENGLISH ABSTRACT: Skeletal muscle adapts to stimuli by modifying structural and metabolic protein expression. Furthermore, a muscle group may vary within itself to accommodate specialisation in regions. Structural and metabolic characteristics of an individual are regulated partly by genotype, but contraction duration and intensity may play a greater role in muscle phenotype. The aims of this dissertation were to investigate: structural and metabolic regionalisation in a muscle group, possible relationships between training volume and intensity and hybrid fibres, muscle characteristics of athletes from two different ethnic groups, and muscle adaptation in already well-trained athletes subjected to high intensity interval training. Myosin heavy chain (MHC) isoform content and citrate synthase (CS) activities were measured in the Quadriceps femoris (QF) muscle of 18 female rats. Muscle was divided into superficial, middle and deep, distal, central and proximal parts. MHC IIb and IIx were more abundant in superficial regions (P < 0.05) with low CS activities compared to deeper parts. Isoform content varied along the length of deep regions. This study showed that the QF has regional specialisation. Therefore, standardisation of sampling site is important. Hybrid fibre proportions in muscle biopsies of 12 middle distance runners and 12 non-runners were investigated. MHC IIa/IIx correlated with training volume/week in runners (r = -0.66, P < 0.05) and MHC IIa/IIx correlated with exercise hours/week in non-runners (r = -0.72, P < 0.01). Average preferred racing distance (PRDA) correlated better with MHC IIa/IIx in runners (r = -0.85, P < 0.001). MHC IIa/IIx may therefore be more closely related to exercise intensity than previously thought. Fibre type characteristics and performance markers were investigated in 13 Xhosa and 13 Caucasian distance runners, matched for performance, training volume and PRDA. Xhosa runners had less MHC I and more MHC IIa fibres in muscle biopsies than Caucasian runners (P < 0.05). Xhosa runners had lower plasma lactate at 80% peak treadmill speed (PTS) (P < 0.05), but higher lactate dehydrogenase (LDH) (P < 0.01) and phosphofructokinase (P = 0.07) activities in homogenate muscle samples. LDH activities in MHC I (P = 0.05) and IIa (P < 0.05) fibre pools were higher in Xhosa runners. Xhosa athletes may thus have a genetic advantage or they may have adapted to running at a higher intensity. Six weeks of individually standardised high intensity interval treadmill training (HIIT) were investigated in 15 well-trained runners. PTS increased after HIIT (P < 0.01), while maximum oxygen consumption (VO2max) only showed a tendency to have increased as a result of HIIT (P = 0.06). Sub-maximal tests showed lower plasma lactate at 64% PTS (P = 0.06), with lower heart rates at workloads from 64% to 80% PTS (P < 0.01) after HIIT. No changes were observed for cross-sectional area, capillary supply and enzyme activities in homogenates muscle samples. LDH activity showed a trend (P = 0.06) to have increased in MHC IIa pools after HIIT. Higher HIIT speed was related to decreases in MHC I fibres, but increases in MHC IIa/IIx fibres (r = -0.70 and r = 0.68, respectively, P < 0.05). Therefore, HIIT may alter muscle fibre composition in well-trained runners, with a concomitant improvement in performance markers.
AFRIKAANSE OPSOMMING: Skeletspier kan adapteer deur strukturele en metaboliese protein ekspressie te verander as gevolg van stimulante. ‘n Spiergroep kan ook intern verskil om spesialisering in spierdele toe te laat. Strukturele en metaboliese karaktereienskappe van ‘n individu word deels gereguleer deur gene, maar kontraksie tydperk en intensiteit mag ‘n groter rol speel in spierfenotipe. Die doelwitte van hierdie tesis was om ondersoek in te stel in: strukturele en metaboliese eienskappe in spiergroepstreke, moontlike verhoudings tussen oefeningsvolume of intensiteit en baster vesels, spier eienskappe in atlete van twee etniese groepe, en spier adaptasie in goed geoefende atlete blootgestel aan hoë intensiteit interval oefening. Miosien swaar ketting (MSK) isovorm inhoud en sitraat sintase (SS) aktiwiteite is gemeet in die Quadriceps femoris (QF) spier van 18 wyfie rotte. Spiere was opgedeel in oppervlakkig, middel en diep, asook distaal, sentraal en proksimale dele. MSK IIb en IIx was meer oorvloedig in oppervlakkige dele (P < 0.05) met lae SS aktiwiteite in vergelyking met dieper dele. Isovorm inhoud het ook verskil oor die lengte van diep dele. Dus bevat die QF gespesialiseerde streke en is die area van monsterneming belangrik. Baster vesel proporsies is ondersoek in spiermonsters van 12 middel afstand hardlopers en 12 niehardlopers. MSK IIa/IIx van hardlopers het met oefeningsvolume/week gekorreleer (r = -0.66, P < 0.05), asook MSK IIa/IIx van nie-hardlopers met oefeningsure/week (r = -0.72, P < 0.01). Gemiddelde voorkeur wedloop afstand (VWAG) het beter met MSK IIa/IIx gekorreleer in hardlopers (r = -0.85, P < 0.001). MSK IIa/IIx mag dus meer verwant wees aan oefeningsintensiteit. Veseltipe eienskappe en prestasie merkers was ondersoek in 13 Xhosa en 13 Caucasian langafstand atlete, geëweknie vir prestasie, oefeningsvolume en VMAG. Xhosa hardlopers het minder tipe I en meer tipe IIA vesels in hul spiermonsters gehad as die Caucasian hardlopers (P < 0.05). Xhosa hardlopers het laer plasma laktaat by 80% van hul maksimale trapmeul spoed (MTS) (P < 0.05), maar hoër laktaat dihidrogenase (LDH) (P < 0.01) en fosfofruktokinase (P = 0.07) aktiwiteite in homogene spiermonsters gehad. LDH aktiwiteite in MSK I (P = 0.05) en IIa (P < 0.05) veselbondels was hoër in Xhosa hardlopers. Xhosa atlete mag dus ‘n genetiese voorsprong geniet, of hulle het geadapteer om by hoër intensiteite te hardloop. Ses weke van geïndividualiseerde gestandardiseerde hoë intensiteit interval trapmeul oefening (HIIT) was ondersoek in 15 goed geoefende hardlopers. MTS het verhoog na HIIT (P < 0.01), en maksimale surrstof verbruik (VO2max) het ‘n neiging getoon om te verhoog het na HIIT (P = 0.07). Submaksimale toetse het laer plasma laktaat by 64% MTS getoon (P = 0.06), met laer harttempos by werkladings 64% tot 80% MTS (P < 0.01). Geen veranderings was gemerk vir deursnit area, kapillêre toevoer en ensiem aktiwiteite in homogene spiermonsters nie. LDH aktiwiteit het ‘n neiging getoon om te verhoog het (P = 0.06) in MSK IIa veselbondels na HIIT. Hoër HIIT snelhede was verwant aan ‘n daling in MSK I vesels, maar ‘n verhoging in MSK IIa/IIx vesels (r = -0.70 en r = 0.68, respektiwelik, P < 0.05). HIIT mag dus spier veseltipe verander in goed geoefende hardlopers, met gevolglike verbetering in prestasie merkers.
Beckitt, Timothy. "Skeletal muscle adaptation following a supervised exercise programme for claudication". Thesis, University of Bristol, 2010. http://ethos.bl.uk/OrderDetails.do?uin=uk.bl.ethos.539766.
Testo completoWiebe, Peter N., e res cand@acu edu au. "Effects of Different Loading Intensities on Skeletal Adaptation to Exercise in Prepubertal Girls". Australian Catholic University. School of Exercise Science, 2004. http://dlibrary.acu.edu.au/digitaltheses/public/adt-acuvp62.29082005.
Testo completoHirschberg, Jens. "Simulations of mechanical adaptation and their relationship to stress bearing in skeletal tissue". University of Western Australia. School of Anatomy and Human Biology, 2005. http://theses.library.uwa.edu.au/adt-WU2005.0095.
Testo completoChen, Ting. "LKB1 Regulation of High-Fat Diet-induced Adaptation in Mouse Skeletal Muscle". BYU ScholarsArchive, 2017. https://scholarsarchive.byu.edu/etd/6682.
Testo completoIsaacs, Ashwin Wayne. "Muscle damage and adaptation in response to plyometric jumping". Thesis, Stellenbosch : Stellenbosch University, 2012. http://hdl.handle.net/10019.1/20384.
Testo completoENGLISH ABSTRACT: The aim of the study was to investigate skeletal muscle changes induced by an acute bout of plyometric exercise before and after plyometric training. The study consisted of an acute study and training intervention study. The acute study, investigated whether direct evidence of ultrastructural damage and identification of indirect factors were more evident in subjects presenting with rhabdomyolysis. Moreover the training intervention study investigated whether plyometric training would protect the muscle from ultrastructural damage and rhabdomyolysis. During the acute intervention, twenty six healthy untrained individuals completed an acute bout of plyometric exercise (10 x 10 squat-jumps, 1 min rest). After, thirteen subjects continued with the training intervention. Eight of these subjects completed 8 weeks of plyometric jump training, while five subjects were instructed to rest from physical activity for 8 weeks. Seven days after the final training session the training and rest group repeated a second acute bout of plyometric exercise. Acute Study: Creatine kinase (CK) activity increased significantly following the single bout of plyometric exercise in all subjects (baseline: 129 to day 4: 5348 U/l). This was accompanied by an increase in perceived pain, C-reactive protein (CRP) a marker of inflammation as well as white blood cells (WBCs). Electron micrographs of muscle biopsies taken 3 days post exercise showed evidence of ultrasructural damage and membrane damage was apparent by immunofluorescence by the loss of dystrophin staining. A stretch of the c-terminus of titin was observed by immunogold, and western blot analysis indicated an increase in calpain-3 autolysis. Based on individual CK responses (CK range: 153-71,024 U/L at 4days after exercise) the twenty six subjects were divided into two groups, namely the high (n=10) and low responders (n=16). Training intervention: Following training the trained group did not experience: a rise of CK activity (110.0 U/l), perceived pain, CRP, WBCs, Z-line streaming, a stretch of titin or calpain-3 activation; while in the control group only two subjects presented with Z-line streaming. The results indicate that high responders have a more pronounced inflammatory response compared to low responders after eccentric exercise, therefore more WBCs and more specifically neutrophils are recruited to damaged areas resulting in greater membrane damage by respiratory burst in high responders. This damage can be limited with training by remodelling sarcomeric proteins via calpain activation resulting in the stable assembly of proteins in the sarcomere preventing the release of proteins.
AFRIKAANSE OPSOMMING: Die doel van die studie was om skeletspier veranderinge wat teweeggebring is deur voor en na afloop van akute pleometriese oefening, te ondersoek. Die studie bestaan uit ‘n akute intervensie en ‘n oefeningsintervensie gedeelte. Die akute intervensie het ondersoek ingestel na die direkte bewyse van ultrastrukturele skade en identifikasie van indirekte faktore meer sigbaar is in proefpersone wat met rhabdomiolose presenteer. Meerso het die oefningsintervensie die moontlikheid dat pleometriese oefening die spier van ultrastrukturele skade en rhabdomiolose beskerm, ondersoek. Tydens die akute intervensie is 26 gesonde ongeoefende individue die akute pleometriese oefeningsessie (10 x 10 hurkspronge, 1 min rus) voltooi. Hierna het 13 proefpersone voortgegaan met die oefeningsintervensie. Agt van hierdie proefpersone het agt weke pleometriese sprongsessie oefeninge voltooi, terwyl vyf proefpersone gevra is om vir 8 weke geen oefeninge te doen nie. Sewe dae na afloop van die finale oefeningssessie het die oefening en kontrole groep in ‘n tweede herhaalde akute pleometriese oefeningsessie deelgeneem. Akute intervensie: kreatienkinase (KK) aktiwiteit het betekenisvol verhoog na die enkel pleometriese oefeningsessie in all proefpersone (basislyn: 129 tot op dag vier: 5348 U/l). Hierdie is vergesel met ‘n toename in die persepsie van pyn, c-reaktiewe proteïen (CRP) ‘n merker van inflammasie sowel as witbloedselle (WBS). Elektronmikrograwe van spierbiopsies wat geneem is drie dae na afloop van die oefeninge, het tekens van ultrastrukturele skade en membraanskade getoon wat ook deur immunofluoresensie duidelik warneembaar was deur die verlies van distrofienverkleuring. ‘n Verrekking van die c-terminus van titin is ook waargeneem deur middel van immunogold. Westernblot analyse het ‘n toename in calpain-3 outolise getoon. Gegrond op individuele KK response (KK grense: 153-71,024 U/L na vier dae post oefening) is 26 proefpersone verdeel in twee groepe naamlik ‘n hoë (n=10) en lae responders (n=16). Oefeningintervensie:: Na oefening het die geoefende groep nie ‘n toename in KK aktiwiteit getoon nie (KK aktiwiteit (110.0 U/l)), pynervaring, CRP, WBS, Z-lynstroming, ‘n strekking van titin of calpain-3 aktivering; terwyl in die kontrole groep daar slegs twee proefpersone met Z-lynstroming geïdentifiseer is. Die resultate wyse daarop dat hoë responders ‘n meer uitgesproke inflammatoriese reaksie toon vergeleke met die lae responders na afloop van essentriese oefening. Daar word dus meer WBS en spesifiek meer neutrofiele na beskadigde areas gelokaliseer wat in grootter membraanskade deur respiratoriese inspanning in die hoë responders. Hierdie skade kan beperk word deur oefening waardeur hermodulering van sarkomeriese proteïene via calpain aktivering tot stabiele rangskiking van proteïene in die sarcomere lei en daardeur proteïen vrystelling verhinder.
The NRF for financial assistance
Owino, Dorcas Vivian Apiyo. "Evaluation of role of paracrine/autocrine IGF-1 system in skeletal muscle adaptation". Thesis, University College London (University of London), 2003. http://ethos.bl.uk/OrderDetails.do?uin=uk.bl.ethos.406510.
Testo completoVarley, I. "Association of genotype with bone metabolism, skeletal adaptation and stress fracture injury occurrence". Thesis, Nottingham Trent University, 2014. http://irep.ntu.ac.uk/id/eprint/223/.
Testo completoLibri sul tema "Skeletal Adaptation"
Ruff, Christopher B., a cura di. Skeletal Variation and Adaptation in Europeans. Hoboken, NJ, USA: John Wiley & Sons, Inc., 2018. http://dx.doi.org/10.1002/9781118628430.
Testo completoMacPherson, Laura Lynn. Adaptations of skeletal muscle pyruvate dehydrogenase kinase in response to food-restriction in mitochondrial subpopulations. St. Catharines, Ont: Brock University, Faculty of Applied Health Sciences, 2007.
Cerca il testo completoMechanobiology: Osteoarthritis and Skeletal Regeneration, and Osteoporosis and Bone Functional Adaptation. Diane Pub Co, 2000.
Cerca il testo completoRuff, Christopher B. Skeletal Variation and Adaptation in Europeans: Upper Paleolithic to the Twentieth Century. Wiley & Sons, Incorporated, John, 2017.
Cerca il testo completoSkeletal Variation and Adaptation in Europeans: Upper Paleolithic to the Twentieth Century. Wiley-Blackwell, 2018.
Cerca il testo completoRuff, Christopher B. Skeletal Variation and Adaptation in Europeans: Upper Paleolithic to the Twentieth Century. Wiley & Sons, Incorporated, John, 2017.
Cerca il testo completoAdaptation of skeletal muscle to spaceflight: COSMOS Rhesus project, COSMOS 2044 and 2229 : final report. [Washington, DC: National Aeronautics and Space Administration, 1994.
Cerca il testo completo(Editor), Neil Spurway, e Henning Wackerhage (Editor), a cura di. Genetics and Molecular Biology of Muscle Adaptation (Advances in Sport and Exercise Science). Churchill Livingstone, 2006.
Cerca il testo completoSkeletal muscle and hepatic enzyme adaptation to physical training under beta-adrenergic blockade in the rat. 1985.
Cerca il testo completoSkeletal muscle and hepatic enzyme adaptation to physical training under beta-adrenergic blockade in the rat. 1987.
Cerca il testo completoCapitoli di libri sul tema "Skeletal Adaptation"
Ruff, Christopher B. "Quantifying Skeletal Robusticity". In Skeletal Variation and Adaptation in Europeans, 39–47. Hoboken, NJ, USA: John Wiley & Sons, Inc., 2017. http://dx.doi.org/10.1002/9781118628430.ch3.
Testo completoRuff, Christopher B. "Introduction". In Skeletal Variation and Adaptation in Europeans, 1–13. Hoboken, NJ, USA: John Wiley & Sons, Inc., 2017. http://dx.doi.org/10.1002/9781118628430.ch1.
Testo completoRuff, Christopher B., e Heather Garvin. "Iberia". In Skeletal Variation and Adaptation in Europeans, 281–314. Hoboken, NJ, USA: John Wiley & Sons, Inc., 2017. http://dx.doi.org/10.1002/9781118628430.ch10.
Testo completoSládek, Vladimír, Margit Berner, Eliška Makajevová, Petr Velemínský, Martin Hora e Christopher B. Ruff. "Central Europe". In Skeletal Variation and Adaptation in Europeans, 315–54. Hoboken, NJ, USA: John Wiley & Sons, Inc., 2017. http://dx.doi.org/10.1002/9781118628430.ch11.
Testo completoNiskanen, Markku, Heli Maijanen, Juho-Antti Junno, Sirpa Niinimäki, Anna-Kaisa Salmi, Rosa Vilkama, Tiina Väre, Kati Salo, Anna Kjellström e Petra Molnar. "Scandinavia and Finland". In Skeletal Variation and Adaptation in Europeans, 355–96. Hoboken, NJ, USA: John Wiley & Sons, Inc., 2017. http://dx.doi.org/10.1002/9781118628430.ch12.
Testo completoRuff, Christopher B., e Brigitte Holt. "The Balkans". In Skeletal Variation and Adaptation in Europeans, 397–418. Hoboken, NJ, USA: John Wiley & Sons, Inc., 2017. http://dx.doi.org/10.1002/9781118628430.ch13.
Testo completoRuff, Christopher B., Brigitte Holt, Markku Niskanen, Vladimir Sládek e Margit Berner. "Conclusions". In Skeletal Variation and Adaptation in Europeans, 419–26. Hoboken, NJ, USA: John Wiley & Sons, Inc., 2017. http://dx.doi.org/10.1002/9781118628430.ch14.
Testo completoNiskanen, Markku, e Christopher B. Ruff. "Body Size and Shape Reconstruction". In Skeletal Variation and Adaptation in Europeans, 15–37. Hoboken, NJ, USA: John Wiley & Sons, Inc., 2017. http://dx.doi.org/10.1002/9781118628430.ch2.
Testo completoNiskanen, Markku, Christopher B. Ruff, Brigitte Holt, Vladimir Sládek e Margit Berner. "Temporal and Geographic Variation in Body Size and Shape of Europeans from the Late Pleistocene to Recent Times". In Skeletal Variation and Adaptation in Europeans, 49–89. Hoboken, NJ, USA: John Wiley & Sons, Inc., 2017. http://dx.doi.org/10.1002/9781118628430.ch4.
Testo completoHolt, Brigitte, Erin Whittey, Markku Niskanen, Vladimir Sládek, Margit Berner e Christopher B. Ruff. "Temporal and Geographic Variation in Robusticity". In Skeletal Variation and Adaptation in Europeans, 91–132. Hoboken, NJ, USA: John Wiley & Sons, Inc., 2017. http://dx.doi.org/10.1002/9781118628430.ch5.
Testo completoAtti di convegni sul tema "Skeletal Adaptation"
Qin, Yi-Xian, Hoyan Lam e Murtaza Malbari. "The Effects of Loading Rate and Duration on Mitigation of Osteopenia by Dynamic Muscle Stimulation". In ASME 2009 Summer Bioengineering Conference. American Society of Mechanical Engineers, 2009. http://dx.doi.org/10.1115/sbc2009-206685.
Testo completoQin, Yi-Xian, e Hoyan Lam. "Bone Formation and Inhibition of Bone Loss by Dynamic Muscle Stimulation With Altered Interstitial Fluid Pressure". In ASME 2007 Summer Bioengineering Conference. American Society of Mechanical Engineers, 2007. http://dx.doi.org/10.1115/sbc2007-176607.
Testo completoAli, Adiba, e Yi-Xian Qin. "Inhibition of Bone Loss and Muscle Atrophy by Dynamic Muscle Contractions With Rest Periods in a Functional Disuse Mouse Model". In ASME 2008 Summer Bioengineering Conference. American Society of Mechanical Engineers, 2008. http://dx.doi.org/10.1115/sbc2008-193066.
Testo completoTaylor, Rebecca E., Chunhua Zheng, Ryan P. Jackson, Joey C. Doll, Julia Chen, Katherine R. S. Holzbaur, Thor Besier e Ellen Kuhl. "Critical Loading During Serve: Modeling Stress-Induced Bone Growth in Performance Tennis Players". In ASME 2008 Summer Bioengineering Conference. American Society of Mechanical Engineers, 2008. http://dx.doi.org/10.1115/sbc2008-192005.
Testo completoBonivtch, Amber Rath, Lynda F. Bonewald e Daniel P. Nicolella. "Tissue Strain Transduction and Amplification at the Osteocyte as a Result of Microstructural Changes in the Bone Matrix". In ASME 2007 Summer Bioengineering Conference. American Society of Mechanical Engineers, 2007. http://dx.doi.org/10.1115/sbc2007-176139.
Testo completoHu, M., J. Cheng, S. Ferreri, F. Serra-Hsu, W. Lin e Y. X. Qin. "Induced Intramedullary Pressure by Dynamic Hydraulic Stimulation and Its Potential in Attenuation of Bone Loss". In ASME 2011 Summer Bioengineering Conference. American Society of Mechanical Engineers, 2011. http://dx.doi.org/10.1115/sbc2011-54015.
Testo completoQin, Y. X., M. Hu, F. Serra-Hsu, J. Cheng, S. Ferreri, Y. Huang, Z. K. Zhang, L. Lin e D. Evangelista. "Local and Distant Intramedullary Pressure and Bone Strain by Dynamic Hydraulic Stimulation". In ASME 2011 Summer Bioengineering Conference. American Society of Mechanical Engineers, 2011. http://dx.doi.org/10.1115/sbc2011-54017.
Testo completoKlinder, Tobias, Hannes Wendland, Irina Wachter-Stehle, David Roundhill e Cristian Lorenz. "Adaptation of an articulated fetal skeleton model to three-dimensional fetal image data". In SPIE Medical Imaging, a cura di Sébastien Ourselin e Martin A. Styner. SPIE, 2015. http://dx.doi.org/10.1117/12.2081139.
Testo completoRapporti di organizzazioni sul tema "Skeletal Adaptation"
Turner, Russel. Bone-97 Alcohol and Skeletal Adaptation to Mechanical Usage. Fort Belvoir, VA: Defense Technical Information Center, ottobre 2002. http://dx.doi.org/10.21236/ada415959.
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