Bibliografie tematiche / Skeletal muscle fibrosis

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Letteratura scientifica selezionata sul tema "Skeletal muscle fibrosis"

Autore: Grafiati
Pubblicato: 14 marzo 2023

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Articoli di riviste sul tema "Skeletal muscle fibrosis"

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Mahdy, Mohamed A. A. "Skeletal muscle fibrosis: an overview." Cell and Tissue Research 375, no. 3 (2018): 575–88. http://dx.doi.org/10.1007/s00441-018-2955-2.

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Amani, Majid, Masoud Rahmati, Mohammad Fathi, and Hasan Ahmadvand. "Reduce Muscle Fibrosis through Exercise via NRG1/ErbB2 Modification in Diabetic Rats." Journal of Diabetes Research 2020 (May 14, 2020): 1–8. http://dx.doi.org/10.1155/2020/6053161.

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Abstract (sommario):
Diabetic myopathy refers to the manifestations in the skeletal muscle as a result of altered glucose homeostasis which reflects as fibrosis. Since physical exercise has been indicated a protective strategy for improving glucose metabolism in skeletal muscle, we tested a hypothesis under which the endurance exercise training could reverse the produced skeletal muscle fibrosis by diabetes. Eight-week-old male Wistar rats were randomly assigned into four groups including healthy control (HC), healthy trained (HT), diabetic control (DC), and diabetic trained (DT) groups. Diabetes was induced by a
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Meyer, Gretchen A., and Richard L. Lieber. "Skeletal muscle fibrosis develops in response to desmin deletion." American Journal of Physiology-Cell Physiology 302, no. 11 (2012): C1609—C1620. http://dx.doi.org/10.1152/ajpcell.00441.2011.

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Skeletal muscle is a dynamic composite of proteins that responds to both internal and external cues to facilitate muscle adaptation. In cases of disease or altered use, these messages can be distorted resulting in myopathic conditions such as fibrosis. In this work, we describe a mild and progressive fibrotic adaptation in skeletal muscle lacking the cytoskeletal intermediate filament protein desmin. Muscles lacking desmin become progressively stiffer, accumulate increased collagen, and increase expression of genes involved in extracellular matrix turnover. Additionally, in the absence of desm
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Zhao, Na, Bo Liu, Si-Wen Liu, et al. "The Combination of Electroacupuncture and Massage Therapy Alleviates Myofibroblast Transdifferentiation and Extracellular Matrix Production in Blunt Trauma-Induced Skeletal Muscle Fibrosis." Evidence-Based Complementary and Alternative Medicine 2021 (July 7, 2021): 1–10. http://dx.doi.org/10.1155/2021/5543468.

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Complementary therapies, such as acupuncture and massage, had been previously reported to have therapeutic effects on skeletal muscle contusions. However, the recovery mechanisms on skeletal muscles after blunt trauma via the combination of electroacupuncture (EA) and massage therapy remain unclear. In the present study, a rat model of the skeletal muscle fibrosis following blunt trauma to rat skeletal muscle was established, and the potential molecular mechanisms of EA + massage therapy on the skeletal muscle fibrosis were investigated. The results suggested that EA + massage therapy could si
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Lieber, Richard L., and Samuel R. Ward. "Cellular Mechanisms of Tissue Fibrosis. 4. Structural and functional consequences of skeletal muscle fibrosis." American Journal of Physiology-Cell Physiology 305, no. 3 (2013): C241—C252. http://dx.doi.org/10.1152/ajpcell.00173.2013.

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Abstract (sommario):
Skeletal muscle fibrosis can be a devastating clinical problem that arises from many causes, including primary skeletal muscle tissue diseases, as seen in the muscular dystrophies, or it can be secondary to events that include trauma to muscle or brain injury. The cellular source of activated fibroblasts (myofibroblasts) may include resident fibroblasts, adult muscle stem cells, or inflammatory or perivascular cells, depending on the model studied. Even though it is likely that there is no single source for all myofibroblasts, a common mechanism for the production of fibrosis is via the transf
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Moyer, Adam L., and Kathryn R. Wagner. "Regeneration versus fibrosis in skeletal muscle." Current Opinion in Rheumatology 23, no. 6 (2011): 568–73. http://dx.doi.org/10.1097/bor.0b013e32834bac92.

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Li, Zhao Bo, Helen D. Kollias, and Kathryn R. Wagner. "Myostatin Directly Regulates Skeletal Muscle Fibrosis." Journal of Biological Chemistry 283, no. 28 (2008): 19371–78. http://dx.doi.org/10.1074/jbc.m802585200.

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Pidlisetskyy, Andriy, Serhii Savosko, Igor Gayovich, Oleksii Dolhopolov, and Volodymyr Biliavskyi. "THE ULTRASONOGRAPHY EXAMINATION OF SKELETAL MUSCLES IN TRAUMATIC ISCHEMIA (EXPERIMENTAL STUDY)." Wiadomości Lekarskie 76, no. 1 (2023): 175–81. http://dx.doi.org/10.36740/wlek202301124.

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Abstract (sommario):
The aim: To establish indicators and significance of sonography in the evaluation of muscle necrosis in ischemia of the limb acording to quantitative ultrasonographic indicators and density of collagen by histological method. Materials and methods: In experiments, rabbits modeled with 6-hour limb ischemia by applying an elastic tourniquet. On days 5, 15, and 30, ultrasound and histological studies of the muscles and correlation analysis were performed between the muscles’ entropy and the degree of their damage (atrophy, fibrosis and necrosis). Results: The relative amount of structurally alter
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Chen, Wan-Jing, I.-Hsuan Lin, Chien-Wei Lee, and Yi-Fan Chen. "Aged Skeletal Muscle Retains the Ability to Remodel Extracellular Matrix for Degradation of Collagen Deposition after Muscle Injury." International Journal of Molecular Sciences 22, no. 4 (2021): 2123. http://dx.doi.org/10.3390/ijms22042123.

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Abstract (sommario):
Aging causes a decline in skeletal muscle function, resulting in a progressive loss of muscle mass, quality, and strength. A weak regenerative capacity is one of the critical causes of dysfunctional skeletal muscle in elderly individuals. The extracellular matrix (ECM) maintains the tissue framework structure in skeletal muscle. As shown by previous reports and our data, the gene expression of ECM components decreases with age, but the accumulation of collagen substantially increases in skeletal muscle. We examined the structural changes in ECM in aged skeletal muscle and found restricted ECM
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Tonogai, Ichiro, and Ichiro Tonogai. "Influence of Platelet Rich Plasma on the Skeletal Muscle Fibrosis after Limb Lengthening in Mice." Foot & Ankle Orthopaedics 5, no. 4 (2020): 2473011420S0046. http://dx.doi.org/10.1177/2473011420s00468.

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Category: Basic Sciences/Biologics Introduction/Purpose: Skeletal muscle fibrosis induced by the increase of collagen occurs after limb lengthening which is also called distraction osteogenesis. Although there are studies about influence of platelet rich plasma (PRP) on tissues healing process, its effectiveness is still controversial. The aim of this study was to examine whether PRP decreased the skeletal muscle fibrosis induced by limb lengthening. Methods: Tibial osteotomy was done to 8-week-old wild type mice. Tibia was lengthened at a rate of 0.42 mm/day during 2 weeks, launching 1 week a
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Tesi sul tema "Skeletal muscle fibrosis"

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Smith, Cheryl A. "Skeletal muscle injury, fibrosis and transforming growth factor-[beta]." Morgantown, W. Va. : [West Virginia University Libraries], 2000. http://etd.wvu.edu/templates/showETD.cfm?recnum=1744.

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Abstract (sommario):
Thesis (Ph. D.)--West Virginia University, 2000.<br>Title from document title page. Document formatted into pages; contains xii, 146 p. : ill. (some col.). Includes abstract. Includes bibliographical references.
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van, Erp Christel. "Modifying function and fibrosis of cardiac and skeletal muscle from mdx mice." University of Southern Queensland, Faculty of Sciences, 2005. http://eprints.usq.edu.au/archive/00001521/.

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Abstract (sommario):
Duchenne Muscular Dystrophy (DMD) is a fatal condition occurring in approximately 1 in 3500 male births and is due to the lack of a protein called dystrophin. Initially DMD was considered a skeletal myopathy, but the pathology and consequences of cardiomyopathy are being increasingly recognised. Fibrosis, resulting from continual cycles of degeneration of the muscle tissues followed by inadequate regeneration of the muscles, is progressive in both cardiac and skeletal dystrophic muscle. In the heart fibrosis interferes with contractility and rhythm whereas it affects contractile function and c
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Puliti, Elisa. "Role of sphingosine 1-phosphate metabolism and signalling in skeletal muscle atrophy and fibrosis." Doctoral thesis, Università di Siena, 2022. http://hdl.handle.net/11365/1195603.

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In the last 30 years, multiple roles of S1P have been demonstrated in the regulation of skeletal muscle biology. The presented study is focused on the role of S1P metabolism in myogenic differentiation, where SPL was found playing a crucial role in regulating S1P cellular levels and responsible for onset of myogenic program. The role of S1P axis was also confirmed in skeletal muscle atrophy induced by TNF-alpha. S1P signalling pathwayplays a crucial role in the development and maintenance of the fibrotic process. New S1P3 antagonists were tested to antagonise the receptor involved in fibrosis,
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Cai, Weisong, and 蔡蔚松. "Cystic fibrosis transmembrane conductance regulator is involved in therelease of ATP from contracting skeletal muscle." Thesis, The University of Hong Kong (Pokfulam, Hong Kong), 2012. http://hub.hku.hk/bib/B49618088.

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Abstract (sommario):
Contracting skeletal muscle releases ATP into the interstitial space where it is subsequently broken down to adenosine by the action of ecto-5’-nucleotidase. Both ATP and adenosine are vasodilators that contribute to the exercise hyperaemia. However, the mechanism for the release of ATP from muscle during exercise remains unknown. Cystic fibrosis transmembrane conductance regulator (CFTR) is involved in ATP release from muscle at low intracellular pH: this study was performed to investigate whether CFTR was involved in the ATP release from skeletal muscle during contractions. Experiments were
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Lu, Lin, and 鹿琳. "The involvement of connexin hemichannels and cystic fibrosis transmembrane conductance regulator in acidosis-induced ATP release from skeletal myocytes." Thesis, The University of Hong Kong (Pokfulam, Hong Kong), 2014. http://hdl.handle.net/10722/208017.

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Abstract (sommario):
The cystic fibrosis transmembrane conductance regulator (CFTR) was identified to be involved in acidosis-induced ATP release from skeletal myocytes in vitro and from contracting muscle in vivo. My PhD studies aimed to investigate the underlying mechanism and identify the pathway for ATP release in acidosis-induced CFTR-regulated ATP release. Lactic acid (10 mM) decreased the intracellular pH of L6 skeletal myocytes to 6.87 ± 0.12 after 3 hours, and the lowered pH resulted in the elevation of ATP release from skeletal myocytes. The acidosis-induced ATP release was totally abolished by GlyH-1
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Pinto, Priscilla Avelino Ferreira. "Treino de baixa intensidade retarda a deposi??o de fibras col?genas no m?sculo gastrocn?mio distr?fico de modelo mdx." UFVJM, 2017. http://acervo.ufvjm.edu.br/jspui/handle/1/1608.

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Submitted by Jos? Henrique Henrique (jose.neves@ufvjm.edu.br) on 2018-03-13T19:28:36Z No. of bitstreams: 2 license_rdf: 0 bytes, checksum: d41d8cd98f00b204e9800998ecf8427e (MD5) priscilla_avelino_ferreira_pinto.pdf: 1920663 bytes, checksum: c9bd6e512c60dc9f7254711669d80fe0 (MD5)<br>Approved for entry into archive by Rodrigo Martins Cruz (rodrigo.cruz@ufvjm.edu.br) on 2018-03-29T11:52:20Z (GMT) No. of bitstreams: 2 license_rdf: 0 bytes, checksum: d41d8cd98f00b204e9800998ecf8427e (MD5) priscilla_avelino_ferreira_pinto.pdf: 1920663 bytes, checksum: c9bd6e512c60dc9f7254711669d80fe0 (MD5)<br>M
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Hauck, James Spencer. "Mineralocorticoid Receptor Signaling in Acute and Chronic Muscle Injury." The Ohio State University, 2019. http://rave.ohiolink.edu/etdc/view?acc_num=osu1565089935933727.

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Neves, Juliana de Carvalho. "Envolvimento da neuraminidase-1 na regeneração muscular." Universidade de São Paulo, 2014. http://www.teses.usp.br/teses/disponiveis/5/5138/tde-06052014-091743/.

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A neuraminidase-1 (Neu1) participa da regulação do catabolismo de sialoglicoconjugados nos lisossomos. A deficiência congênita da Neu1 é a base da sialidose, doença neurossomática grave associada a deformidades osteoesqueléticas, hipotonia e fraqueza muscular. Camundongos com deficiência de Neu1 (Neu1-/-) desenvolvem uma forma atípica de degeneração muscular caracterizada por expansão da matriz extracelular (MEC), invasão das fibras musculares por fibroblastos, fragmentação do citoplasma, formação vacuolar e atrofia muscular. Apesar de a degeneração muscular estar bem caracterizada nestes anim
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Ranasinghesagara, Janaka C. Yao Gang. "Optical reflectance in fibrous tissues and skeletal muscles." Diss., Columbia, Mo. : University of Missouri--Columbia, 2008. http://hdl.handle.net/10355/6629.

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Abstract (sommario):
Title from PDF of title page (University of Missouri--Columbia, viewed on March 8, 2010). The entire thesis text is included in the research.pdf file; the official abstract appears in the short.pdf file; a non-technical public abstract appears in the public.pdf file. Dissertation advisor: Dr. Gang Yao. Vita. Includes bibliographical references.
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Huber, Adrian Thomas. "Multi-organ non-invasive tissue characterization of fibrosis, adipose tissue, edema and inflammation with magnetic resonance (MR) imaging : applications to myocardium, skeletal muscle and liver interactions Cardiac MR strain: a noninvasive biomarker of fibro-fatty remodeling of the left atrial myocardium Comparison of MR T1 and T2 mapping parameters to characterize myocardial and skeletal muscle involvement in systemic Idiopathic Inflammatory Myopathy (IIM) Non-invasive differentiation of acute viral myocarditis and idiopathic inflammatory myopathy with cardiac involvement using magnetic resonance imaging T1 and T2 mapping CT predicts liver fibrosis: Prospective evaluation of morphology- and attenuationbased quantitative scores in routine portal venous abdominal scans." Thesis, Sorbonne université, 2019. http://www.theses.fr/2019SORUS135.

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Cette thèse réalise une preuve de concept pour quantifier la déformation de l’oreillette gauche (OG) en IRM, ainsi que la relaxométrie IRM dans le myocarde, dans les muscles squelettiques et dans le foie. Grâce à l’interaction entre radiologues et ingénieurs, deux logiciels différents ont été développés, appliqués et validés pour l'analyse de la déformation myocardique multi-chambre et pour la cartographie quantitative du T1 multi-organes. La première publication a montré une forte corrélation de la déformation de l’OG, avec le degré de remplacement fibro-graisseux en histologie. Ce biomarqueu
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Capitoli di libri sul tema "Skeletal muscle fibrosis"

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Pessina, Patrizia, and Pura Muñoz-Cánoves. "Fibrosis-Inducing Strategies in Regenerating Dystrophic and Normal Skeletal Muscle." In Methods in Molecular Biology. Springer New York, 2016. http://dx.doi.org/10.1007/978-1-4939-3810-0_7.

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Elhussieny, Ahmed, Ken’ichiro Nogami, Fusako Sakai-Takemura, et al. "Mesenchymal Stem Cells for Regenerative Medicine for Duchenne Muscular Dystrophy." In Muscular Dystrophy - Research Updates and Therapeutic Strategies. IntechOpen, 2020. http://dx.doi.org/10.5772/intechopen.92824.

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Abstract (sommario):
Mesenchymal stem cells (MSCs) are multipotent stem cells that can be isolated from both foetal and adult tissues. Several groups demonstrated that transplantation of MSCs promoted the regeneration of skeletal muscle and ameliorated muscular dystrophy in animal models. Mesenchymal stem cells in skeletal muscle, also known as fibro-adipogenic progenitors (FAPs), are essential for the maintenance of skeletal muscle. Importantly, they contribute to fibrosis and fat accumulation in dystrophic muscle. Therefore, MSCs in muscle are a pharmacological target for the treatment of muscular dystrophies. In this chapter, we briefly update the knowledge on mesenchymal stem/progenitor cells and discuss their therapeutic potential as a regenerative medicine treatment of Duchenne muscular dystrophy.
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Lambrechts, Mark. "Musculoskeletal Abnormalities Caused by Cystic Fibrosis." In Advances in Skeletal Muscle Health and Disease [Working Title]. IntechOpen, 2022. http://dx.doi.org/10.5772/intechopen.104591.

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Cystic Fibrosis (CF) can affect all organs of the human body including the musculoskeletal system. Although the musculoskeletal aspects of CF are less commonly studied, fractures (predominantly spinal), muscle injuries, and joint pain are more commonly seen in the CF population compared to the general public due to their lower bone mineral density, dysfunctional skeletal muscle, and elevated levels of pro-inflammatory cytokines. Additionally, due to elevated levels of inflammation in the CF population diagnosis of musculoskeletal injuries can be difficult to pinpoint. As treatment for CF evolves, an increased understanding of how CF affects the musculoskeletal system is imperative. We will discuss the orthopedic aspects of CF and provide potential insights into the future direction of orthopedic care in the CF population.
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Lambrechts, Mark. "Musculoskeletal Abnormalities Caused by Cystic Fibrosis." In Advances in Skeletal Muscle Health and Disease [Working Title]. IntechOpen, 2022. http://dx.doi.org/10.5772/intechopen.104591.

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Abstract (sommario):
Cystic Fibrosis (CF) can affect all organs of the human body including the musculoskeletal system. Although the musculoskeletal aspects of CF are less commonly studied, fractures (predominantly spinal), muscle injuries, and joint pain are more commonly seen in the CF population compared to the general public due to their lower bone mineral density, dysfunctional skeletal muscle, and elevated levels of pro-inflammatory cytokines. Additionally, due to elevated levels of inflammation in the CF population diagnosis of musculoskeletal injuries can be difficult to pinpoint. As treatment for CF evolves, an increased understanding of how CF affects the musculoskeletal system is imperative. We will discuss the orthopedic aspects of CF and provide potential insights into the future direction of orthopedic care in the CF population.
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Serrano, Antonio L., Christopher J. Mann, Berta Vidal, Esther Ardite, Eusebio Perdiguero, and Pura Muñoz-Cánoves. "Cellular and Molecular Mechanisms Regulating Fibrosis in Skeletal Muscle Repair and Disease." In Current Topics in Developmental Biology. Elsevier, 2011. http://dx.doi.org/10.1016/b978-0-12-385940-2.00007-3.

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Bundgaard, Henning, Anna Axelsson, Alex Christensen, and Helle Petri. "The heart in neuromuscular disease: myotonic dystrophy." In ESC CardioMed. Oxford University Press, 2018. http://dx.doi.org/10.1093/med/9780198784906.003.0370.

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Myotonic dystrophy type 1 (DM1) is a multisystemic disease, mainly involving skeletal muscles, the central nervous system, the gastrointestinal tract, and the heart. DM1 is inherited as an autosomal dominant trait. Patients with DM1 have a reduced life expectancy with a mean age at death of approximately 53 years. Fibrosis, fat infiltration, and degeneration are seen in both skeletal muscles and in the myocardium. Cardiac involvement in DM1 patients is a major concern as the cause of death is of cardiac origin in 30% of patients. The major cardiac manifestations, including conduction abnormalities, supraventricular and ventricular arrhythmias, and reduced left ventricular systolic function, may lead to sudden cardiac death (SCD) or death from progressive heart failure. The increased risk of SCD underscores the need for assessment of cardiac involvement in order to prevent SCD. Clinical evaluation at the time of diagnosis and life-long repeated follow-up should include clinical assessment, electrocardiogram, Holter monitoring, and echocardiography. Generally, cardiac manifestations in DM1 patients should be treated according to general guidelines. An important topic to be resolved is whether a subset of patients, not fulfilling traditional criteria for pacemaker implantation, but who are estimated to be at high risk of severe conduction abnormalities, benefit from pacemaker or defibrillator implantation on a primary prophylactic basis.
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Atkinson, Martin E. "Introduction and surface anatomy." In Anatomy for Dental Students. Oxford University Press, 2013. http://dx.doi.org/10.1093/oso/9780199234462.003.0029.

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The head and neck contain the structures that are the most significant to the practice of dental surgery. These regions are not as easy to study from dissection as other areas because an ‘onion skin’ approach has to be adopted. Layers are dissected from the most superficial subcutaneous structures to the deepest internal structures, the brain, and spinal cord; structures that appear at one level may not show up again until the dissection has advanced to much deeper layers. It is important to have a general understanding of the structures forming the head and neck to build up a coherent picture of their relationship to each other. The skull is the structural basis of the head. The skull comprises the cranium, formed from 27 bones joined together by fibrous joints known as sutures, and the separate mandible that articulates with the cranium at the temporomandibular joints (TMJ). The skull houses and protects the brain in the cranial cavity. It also protects other delicate structures vital for the reception of the special senses; the orbital cavities contain the eyes and dense bones in the cranial base house the internal ears. The entrance to the respiratory tract is the bony and cartilaginous nasal cavity; it can also be accessed together with the gastrointestinal tract through the oral cavity between the cranium and mandible. The major skeletal component of the neck is the cervical part of the vertebral column formed by seven vertebrae. The lower five cervical vertebrae conform to the general pattern of vertebrae outlined in Section 10.1.1, but the upper two cervical vertebrae are specialized; the atlas articulates with the underside of the skull for nodding movements and the second vertebra, the axis, articulates with the atlas for shaking movements of the head. The hyoid bone in the upper anterior neck and the laryngeal cartilages below it form the laryngeal skeleton. There are several important muscle groups in the head. The muscles of facial expression are small superficial muscles beneath the skin of the face; they alter facial expression in response to emotion, but also play a part in chewing, swallowing, and speech.
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Shpadaruk, Volha, and Karen E. Harman. "Cutaneous vasculitis, connective tissue diseases, and urticaria." In Oxford Textbook of Medicine, edited by Roderick J. Hay. Oxford University Press, 2020. http://dx.doi.org/10.1093/med/9780198746690.003.0556.

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Abstract (sommario):
Vasculitis (angiitis) denotes necrotizing inflammation of the blood vessels; occlusive vasculopathy implies vascular occlusion without significant vascular inflammation. A small-vessel cutaneous vasculitis is the most common vasculitis affecting the skin, and may be the first sign of a systemic vasculitis, but 50% of patients have no systemic disease. Systemic lupus erythematosus is diagnosed if four or more of the American College of Rheumatology revised criteria for the classification of this disease are present, either sequentially or simultaneously. Meanwhile, dermatomyositis is an uncommon multisystem autoimmune disease in which inflammatory skin changes are associated with polymyositis of skeletal muscle. Scleroderma means thickened, fibrotic, bound-down skin. It might develop in association with a systemic connective tissue disease (systemic sclerosis) or present as a localized cutaneous problem. Panniculitis is inflammation of the subcutaneous fat, sometimes associated with vasculitis. It presents with erythematous subcutaneous nodules, most often on the lower leg.
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Atti di convegni sul tema "Skeletal muscle fibrosis"

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Rozenberg, D., M. Sussman, R. G. L. Koh, et al. "Skeletal Muscle Size and Fat Infiltration of the Limb Muscles in Idiopathic Pulmonary Fibrosis." In American Thoracic Society 2022 International Conference, May 13-18, 2022 - San Francisco, CA. American Thoracic Society, 2022. http://dx.doi.org/10.1164/ajrccm-conference.2022.205.1_meetingabstracts.a3179.

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Balañá, Ana, Juana Martínez-Llorens, Diego Agustin Rodríguez, et al. "Skeletal muscle function and structure in patients with non-cystic fibrosis bronchiectasis." In ERS International Congress 2016 abstracts. European Respiratory Society, 2016. http://dx.doi.org/10.1183/13993003.congress-2016.oa265.

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Verges, Samuel, Nicolas Decorte, Mathieu Gruet, et al. "Skeletal muscle metabolism in active cystic fibrosis (CF) patients with light/moderate pulmonary dysfunction." In Annual Congress 2015. European Respiratory Society, 2015. http://dx.doi.org/10.1183/13993003.congress-2015.pa2242.

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Wang, Xuejie, Ana Balañá Corberó, Juana Martínez Llorens, et al. "Skeletal Muscle Dysfunction and Body Composition Alterations in Non-Cystic Fibrosis Bronchiectasis Patients: Gender Differences." In ERS International Congress 2020 abstracts. European Respiratory Society, 2020. http://dx.doi.org/10.1183/13993003.congress-2020.1836.

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Huang, Alice H., Spencer S. Watson, and Ronen Schweitzer. "Lineage Tracing Reveals a New Model for Tendon Growth and Elongation During Development." In ASME 2012 Summer Bioengineering Conference. American Society of Mechanical Engineers, 2012. http://dx.doi.org/10.1115/sbc2012-80915.

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Tendons are dense, fibrous tissues connecting muscle to bone, and their primary function is to transmit muscle forces to the appropriate skeletal elements, thereby enabling movement. In the limb, flexion and extension of the hand (autopod) and wrist are controlled by long tendons that insert into muscles in the arm (zeugopod) [1]. Although tendons are critically important in mediating joint movement, the cellular and molecular events underlying tendon formation remain largely unknown. Using the transcription factor Scleraxis (Scx), which labels all tendon progenitors, we previously showed that
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Uslu, Nazlı Zeynep, Derya Kocakaya, Sehnaz Olgun Yıldızeli, et al. "Does cystic fibrosis impact skeletal muscles and diaphragm function?" In ERS International Congress 2019 abstracts. European Respiratory Society, 2019. http://dx.doi.org/10.1183/13993003.congress-2019.pa343.

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Marillier, Mathieu, Anne-Catherine Bernard, Onofre Moran-Mendoza, Denis E. O'Donnell, Samuel Verges, and J. Alberto Neder. "Beyond the lungs in fibrotic interstitial lung disease: does supplemental O2 improve skeletal muscle oxygenation and fatigue?" In ERS International Congress 2020 abstracts. European Respiratory Society, 2020. http://dx.doi.org/10.1183/13993003.congress-2020.4405.

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