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Articoli di riviste sul tema "Thiamine deficiency"

Autore: Grafiati

Pubblicato: 4 giugno 2021

Ultima modifica: 25 luglio 2025

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1

Ritorto, Giovanna, Sara Ussia, Rocco Mollace, et al. "The Pivotal Role of Thiamine Supplementation in Counteracting Cardiometabolic Dysfunctions Associated with Thiamine Deficiency." International Journal of Molecular Sciences 26, no. 7 (2025): 3090. https://doi.org/10.3390/ijms26073090.

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The isolation, structural elucidation, and synthesis of pure thiamin were achieved in 1936, marking a milestone in vitamin research. As an organic compound soluble in water, thiamin is essential for carbohydrate metabolism in plants and animals, and in its active form—as part of the thiamin pyrophosphate coenzyme—performs these functions. Thereby, thiamin represents an essential vitamin to human health and is involved in several pathways that regulate several pathophysiological mechanisms. Cardiovascular disease is significantly impacted by thiamine imbalance and its supplementation offers sub

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2

Olufunso, Adeoye Bayo, Nwachukwu Ijeoma Doris, Olanrewaju Okikiola, and Omobude Aisagbonhi Elizabeth. "Enzyme Thiaminase: A Known Anti-nutritional Enzyme with Unknown Therapeutic Potentials in Cancer Treatment." Cross Current International Journal of Medical and Biosciences 6, no. 06 (2024): 114–19. https://doi.org/10.36344/ccijmb.2024.v06i06.001.

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Thiaminase (EC 2.5.1.2) is an enzyme that cleaves thiamine into its pyrimidine and thiazole moieties resulting in thiamine deficiency in various organisms. It is classified into two main types: Thiaminase I and Thiaminase II defined by the nucleophile used in the mechanism by which the cleavage is accomplished. Thiaminase I employs a variety of nucleophiles including, amines and sulfhydryl compounds while thiaminase II exclusively uses water for hydrolysis of thiamine. The crystal structure of thiaminase I reveals a deep cleft that accommodates thiamine and highlighting important residues that

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3

Dr., G. Sri Manjula, D.V. Ramanjaneyulu, E. Muralinath, et al. "Identification of Early as well as Advanced Symptoms of B1 Deficiency Along with Neurological, Cardiac and Gastrointestinal Manifestations." Journal of Cardiovascular, Cardiopulmonary Rehabilitation Nursing 2, no. 2 (2024): 18–26. https://doi.org/10.5281/zenodo.13756478.

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<em>Thiamine, also </em><em>termed as thiamin and vitamin B1, is one of the 8 B-complex vitamins and is </em><em>categorized as a water-soluble B vitamin. Thiamine </em><em>happens naturally in specific diets, is </em><em>included in food products, and is available in dietary supplements. Rich sources of thiamine include whole grains, brown rice, pig, chicken, soybeans, almonds, peas, dry beans, and fortified or enriched grain products, such as cereals, bread, and baby formulae. Multivitamins supplement an additional 1.5 mg of thiamine to a diet already </em><em>excess in this essential vitami

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4

Pan, Xiaohua, Xuemei Nan, Liang Yang, Linshu Jiang, and Benhai Xiong. "Thiamine status, metabolism and application in dairy cows: a review." British Journal of Nutrition 120, no. 5 (2018): 491–99. http://dx.doi.org/10.1017/s0007114518001666.

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AbstractAs the co-enzyme of pyruvate dehydrogenase andα-ketoglutarate dehydrogenase, thiamine plays a critical role in carbohydrate metabolism in dairy cows. Apart from feedstuff, microbial thiamine synthesis in the rumen is the main source for dairy cows. However, the amount of ruminal thiamine synthesis, which is influenced by dietary N levels and forage to concentrate ratio, varies greatly. Notably, when dairy cows are overfed high-grain diets, subacute ruminal acidosis (SARA) occurs and results in thiamine deficiency. Thiamine deficiency is characterised by decreased ruminal and blood thia

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5

Richter, Catherine A., Allison N. Evans, Maureen K. Wright-Osment, et al. "Paenibacillus thiaminolyticus is not the cause of thiamine deficiency impeding lake trout (Salvelinus namaycush) recruitment in the Great Lakes." Canadian Journal of Fisheries and Aquatic Sciences 69, no. 6 (2012): 1056–64. http://dx.doi.org/10.1139/f2012-043.

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Thiamine (vitamin B1) deficiency is a global concern affecting wildlife, livestock, and humans. In Great Lakes salmonines, thiamine deficiency causes embryo mortality and is an impediment to restoration of native lake trout ( Salvelinus namaycush ) stocks. Thiamine deficiency in fish may result from a diet of prey with high levels of thiaminase I. The discoveries that the bacterial species Paenibacillus thiaminolyticus produces thiaminase I, is found in viscera of thiaminase-containing prey fish, and causes mortality when fed to lake trout in the laboratory provided circumstantial evidence imp

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6

Lonsdale, Derrick. "A Review of the Biochemistry, Metabolism and Clinical Benefits of Thiamin(e) and Its Derivatives." Evidence-Based Complementary and Alternative Medicine 3, no. 1 (2006): 49–59. http://dx.doi.org/10.1093/ecam/nek009.

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Thiamin(e), also known as vitamin B1, is now known to play a fundamental role in energy metabolism. Its discovery followed from the original early research on the ‘anti-beriberi factor’ found in rice polishings. After its synthesis in 1936, it led to many years of research to find its action in treating beriberi, a lethal scourge known for thousands of years, particularly in cultures dependent on rice as a staple. This paper refers to the previously described symptomatology of beriberi, emphasizing that it differs from that in pure, experimentally induced thiamine deficiency in human subjects.

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GOMES, Marleide da Mota, and Marcos Raimundo Gomes de FREITAS. "Probable first report of a motor deafferentation syndrome in the Paraguayan War." Arquivos de Neuro-Psiquiatria 79, no. 6 (2021): 554–56. http://dx.doi.org/10.1590/0004-282x-anp-2020-0479.

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ABSTRACT The Paraguayan War ended 150 years ago. Back then, there were outbreaks of combatants’ limb weakness and tingling related to "palustrian cachexia", not clearly funded at the time on nutritional deficiency, the use of native flora to feed troops, and alcoholism. We report a case of a soldier with ascending paralysis, mental confusion and finally tetraplegia with preserved oculomotricity. This would probably be a case of locked-in syndrome (LIS) due to Gayet-Wernicke's encephalopathy consequent to thiamine deficiency. The role of thiamine in the peripheral or central nervous system expr

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Bourassa, Megan, and Gilles Bergeron. "Advances from the International Thiamine Alliance." Current Developments in Nutrition 4, Supplement_2 (2020): 814. http://dx.doi.org/10.1093/cdn/nzaa053_019.

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Abstract Objectives Thiamine deficiency disorders, including beriberi, remain a pressing public health problem. Without rapid recognition of symptoms and treatment, it is often fatal, especially in infants, and can have lasting neurological effects for survivors. Thus our objective is to create an alliance of country representatives, public health professionals, physicians and researchers who can quantify the prevalence of thiamine deficiency disorders globally and create control and prevention strategies. Methods The first meeting of this international thiamine alliance was held in November 2

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9

Skelton, William P., and Nadine K. Skelton. "Thiamine deficiency neuropathy." Postgraduate Medicine 85, no. 8 (1989): 301–6. http://dx.doi.org/10.1080/00325481.1989.11700760.

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10

van Snippenburg, Wouter, Mariet G. J. Reijnders, Jose G. M. Hofhuis, Rien de Vos, Stephan Kamphuis, and Peter E. Spronk. "Thiamine Levels During Intensive Insulin Therapy in Critically Ill Patients." Journal of Intensive Care Medicine 32, no. 9 (2016): 559–64. http://dx.doi.org/10.1177/0885066616659429.

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Introduction: Thiamine is an essential cofactor in carbohydrate metabolism, and deficiency can therefore cause various organ dysfunctions. Little is known about the prevalence and possible worsening of thiamine deficiency in critically ill patients. In this study, we investigated the prevalence of thiamine deficiency at admission to the intensive care unit (ICU) and hypothesized that intensive insulin therapy, aimed at regulating glucose levels, increases thiamine utilization and therefore might cause or worsen deficiency in patients with limited thiamine stores. Materials and Methods: An obse

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Sweet, Rebecca L., та Jason A. Zastre. "HIF1-α-Mediated Gene Expression Induced by Vitamin B1 Deficiency". International Journal for Vitamin and Nutrition Research 83, № 3 (2013): 188–97. http://dx.doi.org/10.1024/0300-9831/a000159.

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It is well established that thiamine deficiency results in an excess of metabolic intermediates such as lactate and pyruvate, which is likely due to insufficient levels of cofactor for the function of thiamine-dependent enzymes. When in excess, both pyruvate and lactate can increase the stabilization of the hypoxia-inducible factor 1-alpha (HIF-1α) transcription factor, resulting in the trans-activation of HIF-1α regulated genes independent of low oxygen, termed pseudo-hypoxia. Therefore, the resulting dysfunction in cellular metabolism and accumulation of pyruvate and lactate during thiamine

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Mohamed, Ragaa Abd-Elsalam, Ibrahim Mohamed Abu Farag, Marwa Elhady, and Radwa Saeed Ibrahim. "Myocardial dysfunction in relation to serum thiamine levels in children with diabetic ketoacidosis." Journal of Pediatric Endocrinology and Metabolism 32, no. 4 (2019): 335–40. http://dx.doi.org/10.1515/jpem-2018-0320.

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Abstract Background Thiamine deficiency is commonly reported in patients with diabetes especially during diabetic ketoacidosis (DKA) that could attribute to myocardial dysfunction in those patients. However, there is limited data regarding its relation to myocardial function among those patients. This study aimed to explore the association between myocardial function and serum thiamine levels in children with type 1 diabetes mellitus (DM). Methods This cross-sectional comparative study included 25 patients with DKA. Clinical data assessment, echocardiographic examination and measurement of ser

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Buranasakda, Marturod, and Ratrawee Pattanarattanamolee. "Thiamine Level in Out-of-hospital Cardiac Arrest Patients." Open Access Macedonian Journal of Medical Sciences 10, B (2022): 1037–41. http://dx.doi.org/10.3889/oamjms.2022.8015.

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BACKGROUND: Thiamine deficiency is more common in critically ill patients. Administration of thiamine in cardiac arrest mice has improved survival and neurological outcomes. Evidence for thiamine deficiency in cardiac arrest humans is insufficient to support routine use of thiamine in cardiac arrest patients. AIM: This study aimed to determine thiamine blood levels in cardiac arrest patients to understand whether the presence of thiamine deficiency is common in cardiac arrest patients. METHODS: A prospective descriptive study from April 2017 to March 2018, on 24 adult out-of-hospital cardiac a

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Panda, Akhila, Amirmohammad Heidari, Maryam Borumand, Musaab Ahmed, Ahmed Hassan, and Mohamed H. Ahmed. "Thiamine deficiency in diabetes, obesity and bariatric surgery: Recipes for diabetic ketoacidosis." Journal of Family Medicine and Primary Care 13, no. 5 (2024): 1620–27. http://dx.doi.org/10.4103/jfmpc.jfmpc_1413_23.

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ABSTRACT Diabetic ketoacidosis (DKA) is a life-threatening condition affecting individuals with diabetes characterised by hyperglycaemia, metabolic acidosis and ketonemia. The incidence and financial burden of DKA is still high. Thiamine deficiency is well documented in patients with DKA and could be associated with cardiac dysfunction in those patients. Thiamine deficiency leads to cardiac dysfunction, neuronal death and worsens the prognosis of DKA. There is an existing metabolic relationship between thiamine deficiency in diabetes, obesity and bariatric surgery. Careful monitoring of thiami

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15

Reed, Aimee N., Freya E. Rowland, Jennifer A. Krajcik, and Donald E. Tillitt. "Thiamine Supplementation Improves Survival and Body Condition of Hatchery-Reared Steelhead (Oncorhynchus mykiss) in Oregon." Veterinary Sciences 10, no. 2 (2023): 156. http://dx.doi.org/10.3390/vetsci10020156.

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Early rearing of steelhead (Oncorhynchus mykiss) in Oregon hatcheries is often problematic; fry can become emaciated and die during the period between hatch and first feed. Thiamine (vitamin B1) deficiency has caused early mortality in salmonids; however, the thiamine status of Oregon’s steelhead populations is unknown, to date. Of the 26 egg samples from three Oregon hatcheries in 2019, 20 (77%) had thiamine levels < 10 nmol/g, and 13 of those samples (50%) had levels <6.5 nmol/g, suggesting the thiamine deficiency of adult, female steelhead. To investigate if thiamine deficiency was ca

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Boopathy, Deepika, Daniel Grahf, Jacob Ross, et al. "Thiamine Deficiency Is Common and Underrecognized in Emergency Department Oncology Patients." Journal of Clinical Medicine 14, no. 1 (2025): 257. https://doi.org/10.3390/jcm14010257.

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Background: Wernicke’s encephalopathy can occur in oncology patients independent of alcohol use, likely resulting from poor dietary thiamine intake. High metabolic demands, such as those in acute illnesses seen in the emergency department (ED), can exacerbate thiamine deficiency. In this study, our objective was to assess the incidence of thiamine deficiency in ED oncology patients, which could lead to Wernicke’s encephalopathy or other thiamine deficiency disorders if left untreated. Methods: This was a single-center prospective cohort study. We included patients with acute illness and a hist

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17

Pacei, Federico, Antonella Tesone, Nazzareno Laudi, et al. "The Relevance of Thiamine Evaluation in a Practical Setting." Nutrients 12, no. 9 (2020): 2810. http://dx.doi.org/10.3390/nu12092810.

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Thiamine is a crucial cofactor involved in the maintenance of carbohydrate metabolism and participates in multiple cellular metabolic processes. Although thiamine can be obtained from various food sources, some common food groups are deficient in thiamine, and it can be denatured by high temperature and pH. Additionally, different drugs can alter thiamine metabolism. In addition, the half-life of thiamine in the body is between 1 and 3 weeks. All these factors could provide an explanation for the relatively short period needed to develop thiamine deficiency and observe the consequent clinical

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Iimura, Yohei, Shohei Andoh, Toyotaka Kawamata, et al. "Thiamine Deficiency and Neurological Symptoms in Patients with Hematological Cancer Receiving Chemotherapy: A Retrospective Analysis." Journal of Neurosciences in Rural Practice 12, no. 04 (2021): 726–32. http://dx.doi.org/10.1055/s-0041-1735825.

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Abstract Objectives Patients with hematological cancer receiving chemotherapy have a high risk of thiamine deficiency due to accelerated thiamine usage by tumor cells. Mild or severe thiamine deficiency can lead to varying degrees of neurological symptoms. We evaluated the relationship between thiamine deficiency and neurological symptoms, including mild or nonspecific symptoms, and the influence of chemotherapy on thiamine serum levels in patients with hematological cancer receiving chemotherapy. Materials and Methods We retrospectively identified 42 patients diagnosed with hematological canc

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Agedal, Kaitlyn J., Kelly E. Steidl, and Jeni L. Burgess. "An Overview of Type B Lactic Acidosis Due to Thiamine (B1) Deficiency." Journal of Pediatric Pharmacology and Therapeutics 28, no. 5 (2023): 397–408. http://dx.doi.org/10.5863/1551-6776-28.5.397.

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Type B lactic acidosis can occur secondary to several factors, including thiamine deficiency, and is not as common as type A. Recognizing thiamine deficiency–associated lactic acidosis is challenging because serum thiamine concentrations are not routinely obtained, and a thorough and specific history is necessary for clinicians to suspect thiamine deficiency as a root cause. Furthermore, the appropriate dose and duration of thiamine treatment are not well defined. Untreated thiamine deficiency–associated lactic acidosis can lead to critical illness requiring lifesaving extracorporeal therapies

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Kloss, Olena, N. A. Michael Eskin, and Miyoung Suh. "Thiamin deficiency on fetal brain development with and without prenatal alcohol exposure." Biochemistry and Cell Biology 96, no. 2 (2018): 169–77. http://dx.doi.org/10.1139/bcb-2017-0082.

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Adequate thiamin levels are crucial for optimal health through maintenance of homeostasis and viability of metabolic enzymes, which require thiamine as a co-factor. Thiamin deficiency occurs during pregnancy when the dietary intake is inadequate or excessive alcohol is consumed. Thiamin deficiency leads to brain dysfunction because thiamin is involved in the synthesis of myelin and neurotransmitters (e.g., acetylcholine, γ-aminobutyric acid, glutamate), and its deficiency increases oxidative stress by decreasing the production of reducing agents. Thiamin deficiency also leads to neural membran

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Zhao, Yanling, Yiying Wu, Haolu Hu, et al. "Downregulation of Transketolase Activity Is Related to Inhibition of Hippocampal Progenitor Cell Proliferation Induced by Thiamine Deficiency." BioMed Research International 2014 (2014): 1–9. http://dx.doi.org/10.1155/2014/572915.

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In animal experiments, hippocampal neurogenesis and the activity of thiamine-dependent transketolase decrease markedly under conditions of thiamine deficiency. To further investigate the effect of thiamine deficiency on the proliferation of hippocampal progenitor cells (HPCs) and the potential mechanisms involved in this effect, we cultured HPCs in vitro in the absence of thiamine and found that proliferation and transketolase activity were both significantly repressed. Furthermore, specific inhibition of transketolase activity by oxythiamine strongly inhibited HPC proliferation in a dose-depe

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Chhetri, Vishal, Tshering Dorji, Adeep Monger, and Hari Prasad Pokhrel. "Peripheral Neuropathy Outbreaks in Bhutan, 2020–2021." Outbreak, Surveillance, Investigation & Response (OSIR) Journal 16, no. 1 (2023): 1–6. http://dx.doi.org/10.59096/osir.v16i1.261866.

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Thiamine (vitamin B1) deficiency can cause beriberi leading to cardiac involvement or, more commonly, peripheral neuropathy. Common causes of thiamine deficiency are alcohol use, maternal thiamine deficiency, poor dietary diversity, unhygienic food preparation, and unsafe cooking practices. This report presents an analysis of suspected peripheral neuropathy outbreaks recorded at the Royal Centre for Disease Control between 2020 and 2021. The suspected outbreaks were reported from schools and monastic institutes with the main complaints of numbness and swelling of lower limbs. Three of the four

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Ba, Abdoulaye, Valentin N'Douba, Marie-Anne D'Almeida, and Bialli Victor Seri. "Effects of maternal thiamine deficiencies on the pyramidal and granule cells of the hippocampus of rat pups." Acta Neurobiologiae Experimentalis 65, no. 4 (2005): 387–98. http://dx.doi.org/10.55782/ane-2005-1567.

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To understand the implication of thiamine deficiency in the neuronal atrophy and cell death we undertook to induce thiamine (B1 vitamine) deficiency during three essential periods of the ontogenesis of rat central nervous system (CNS). Female rats were fed with a thiamine deprived diet during the gestation and lactation, and the fetuses and pups were alternately exposed to prenatal, perinatal or postnatal thiamine deficiencies. On the 45th postnatal day, histological studies were done on the brains of the pups and the structure of the hippocampus was analyzed. The effects of each treatment wer

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Ton, Angie Nu, Trisha Jethwa, Greta C. Stamper, and Ashley Yenior. "Sensorineural Hearing Loss and Wernicke Encephalopathy: A Case Report and Literature Review." Journal of Audiology and Otology 25, no. 1 (2021): 55–58. http://dx.doi.org/10.7874/jao.2020.00045.

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Sensorineural hearing loss (SNHL) is seldom associated with Wernicke encephalopathy (WE) or thiamine deficiency. While thiamine deficiency and repletion are often considered prior to dextrose infusions in patients with chronic alcohol abuse to prevent WE, they are often overlooked in non-alcoholic patients who are also at risk for malnutrition. In this paper we describe a case of a non-alcoholic 28-year-old female status post-sleeve gastrectomy who developed SNHL in the setting of thiamine deficiency and WE, with ongoing hearing impairment requiring hearing aids despite thiamine repletion.

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Millichap, J. Gordon, and John J. Millichap. "Thiamine Deficiency in Infancy." Pediatric Neurology Briefs 28, no. 10 (2014): 76. http://dx.doi.org/10.15844/pedneurbriefs-28-10-4.

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Holz, Peter H., James R. Phelan, and Ron Slocombe. "Thiamine Deficiency in Honeyeaters." Journal of Avian Medicine and Surgery 16, no. 1 (2002): 21–25. http://dx.doi.org/10.1647/1082-6742(2002)016[0021:tdih]2.0.co;2.

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Suzuki, Takahiro. "Treatable Dementia: Thiamine Deficiency." Journal of Nihon University Medical Association 77, no. 5 (2018): 309–12. http://dx.doi.org/10.4264/numa.77.5_309.

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Pritchard, Peter. "Thiamine deficiency in cats." Veterinary Record 180, no. 10 (2017): 256.4–257. http://dx.doi.org/10.1136/vr.j1202.

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Keyset, A., and B. de Bruijn. "Thiamine deficiency and epilepsy." Clinical Neurology and Neurosurgery 90, no. 3 (1988): 303. http://dx.doi.org/10.1016/0303-8467(88)90083-2.

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Butterworth, RogerF, Christiane Gaudreau, Jean Vincelette, Anne-Marie Bourgault, François Lamothe, and Anne-Marie Nutini. "Thiamine deficiency in AIDS." Lancet 338, no. 8774 (1991): 1086. http://dx.doi.org/10.1016/0140-6736(91)91948-t.

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Wani, Maleeha, Adil Bashir, Masood Tanvir, et al. "A STUDY OF THIAMINE DEFICIENCY IN PATIENTS WITH ACUTE ONSETENCEPHALOPATHY." International Journal of Advanced Research 11, no. 05 (2023): 723–32. http://dx.doi.org/10.21474/ijar01/16925.

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Introduction:Vitamin B1, also known as thiamine, and its crucial role in energy metabolism and proper functioning of the nervous and cardiovascular systems. Thiamine is an essential vitamin that must be obtained through diet since the body does not produce it naturally. It is stored in the liver but only lasts for about 18 days. Thiamine is absorbed in the duodenum and transported across the blood-brain barrier. The recommended daily intake varies based on age, health condition, and life stages.Thiamine plays a vital role in various biochemical pathways in the brain, including energy productio

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Bahat, Hilla, Gad Reisler, Nurit Brandriss, Adina Bar-Chaim, and Michael Goldman. "Thiamine Deficiency in Adolescents with Eating Disorders: A Prospective Cohort Study." Nutrients 12, no. 5 (2020): 1396. http://dx.doi.org/10.3390/nu12051396.

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Background: Pediatric eating disorders (PED) patients are prone to nutritional deficiencies. Thiamine deficiency is well described in other malnutrition states but is not routinely screened for in PED. In the current study we evaluated the prevalence of thiamine deficiency among PED patients on their first admission to an outpatient day hospital for eating disorders (DH). Methods: In this prospective cohort study, we measured whole blood thiamine pyrophosphate concentrations (TPP) in addition to a routine laboratory workup in 69 girls on their first admission to DH. Two subgroup analyses were

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Shehab-Eldin, Mohamed. "The role of thiamine as a resuscitator in patients with nonalcoholic medical and CNS disorders." Southwest Respiratory and Critical Care Chronicles 5, no. 19 (2017): 11. http://dx.doi.org/10.12746/swrccc.v5i19.398.

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Thiamine (vitamin B1), a water-soluble vitamin, is an essential factor in cellular metabolismand fundamental cofactor in important biochemical cycles. Thiamine deficiency is a wellknowncause of neurological and cardiologic disorders, especially in patients with alcoholdependence. Recently, several researchers have studied the role of thiamine deficiency incritically ill patients and the link between thiamine supplementation and changes in lactatelevels in septic shock patients. The role of thiamine in this group of patients is still unclear;however, thiamine supplementation does not cause toxi

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Hassan, Mahmuda, Hamidur Rahman, BH Nazma Yasmeen, et al. "Thiamine deficiency - Beriberi – A forgotten disease." Northern International Medical College Journal 10, no. 1 (2018): 351–54. http://dx.doi.org/10.3329/nimcj.v10i1.39331.

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Thiamine (vitamin B1) was the first B vitamin which has been identified. It serves as a cofactor for several enzymes involved in energy metabolism. The thiamine-dependent enzymes are important for the biosynthesis of neurotransmitters and for the production of reducing substances used in oxidant stress defenses, as well as for the synthesis of pentose used as nucleic acid precursors. Thiamine also plays a central role in cerebral metabolism. There are 2 major manifestations of thiamine deficiency(TD): cardiovascular disease (wet beriberi) and nervous system disease (dry beriberi and Wernicke–K

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Venkat, Cuddapah Gaurav, Vallivedu Chennakesavulu Pujitha, Kanchan S. Channawar, and Vadde Vasavi. "Thiamine responsive pulmonary hypertension: case series." International Journal of Contemporary Pediatrics 8, no. 6 (2021): 1102. http://dx.doi.org/10.18203/2349-3291.ijcp20212056.

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Pulmonary hypertension (PH) is most commonly related either to a cardiac or a pulmonary cause. But less commonly various hematological, hepatic, genetic causes are also associated. Infantile PH due to vitamin deficiencies is very rare though few cases with thiamine deficiencies causing PH have been reported lately. Lack of awareness and late recognition of thiamine deficiency may result in high mortality. A high index of suspicion is required for early diagnosis and management to decrease the severity and morbidity and thereby preventing long term implications on neurological development. Here

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Böhrer, Madeleine, Paige Burgess, Tais Da Costa Sao Pedro, Dana Liza Boctor, and Samantha Boggs. "Shoshin Beriberi in a Child With Intestinal Failure: A Case Report." Critical Care Explorations 6, no. 12 (2024): e1187. https://doi.org/10.1097/cce.0000000000001187.

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Background: Acute illness states with increased metabolic demand can precipitate severe thiamine deficiency if physiologic needs exceed endogenous stores and supplementation. Patients with preexisting risk factors such as parenteral nutrition (PN) dependence, eating disorders, gastrointestinal disorders, or surgeries are especially vulnerable. Case Summary: A 9-year-old girl with short bowel syndrome receiving long-term PN, including standard thiamine supplementation, presented with refractory shock following aspiration. Profound hyperlactatemia, cardiac dysfunction, and vasoactive requirement

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Hilal Ahmad, Sheikh, S. Bashir Ahmad, and Asma Jan. "Thiamine deficiency related peripheral neuropathy in peripartum women of Kashmir, India: a hospital based study." International Journal of Advances in Medicine 6, no. 3 (2019): 662. http://dx.doi.org/10.18203/2349-3933.ijam20191498.

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Background: Beri Beri is still seen in Asian countries due to the large-scale consumption of thiamine depleted polished rice. Thiamine deficiency related disorders are increasingly being reported from Kashmir from last two decades. Specifically, a sensorimotor axonal neuropathy occasionally develops from thiamine deficiency and may occur even without associated Wernicke’s encephalopathy.Methods: This study is a retrospective observational study. All peripartum females referred to our departments from January 2016 to December 2017 with complaints of generalized weakness and or sensory symptoms

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Ushakova, H. O., O. Z. Brazaluk, and O. Yu Yevstafieva. "THIAMINE DEFICIENCY IN EXTREMELY PREMATURE NEWBORNS." Medical and Clinical Chemistry, no. 2 (July 10, 2019): 18–23. http://dx.doi.org/10.11603/mcch.2410-681x.2019.v.i2.10289.

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Introduction. The critical state of extremely premature newborns is accompanied by significant oxidative stress, which leads to an increase in anaerobic metabolism; impaired oxygen utilization in mitochondrial oxidation may occur due to thiamine deficiency. The aim of the study – to learn the thiamine status of extremely premature new-borns in the neonatal period follow-up. Research Methods. The study involved 55 of premature newborns with birth weight less than 1000 g and 35 healthy full-term newborns. The indicated children’s blood tests were made in the first day of life and at

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Kshirsagar, Sujit J., Shivaji Kute, and Pradnya M. Bhalerao. "Anaesthesia and Intensive Care Management of Shoshin Beriberi in Perioperative Period in Young Adult." Annals of Cardiac Anaesthesia 26, no. 3 (2023): 318–20. http://dx.doi.org/10.4103/aca.aca_133_22.

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Thiamine deficiency presents as dry and wet beriberi. Wet beriberi is a complication of the cardiovascular system. Acute form of wet beriberi known as Shoshin beriberi is an acute presentation of cardiogenic shock which is rapidly reversed with thiamine administration. Here we present successful management of intraoperative acute decompensated heart failure, probably due to thiamine deficiency.

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Sharma, Anupama, and Renu Bist. "Alteration in MDA, GSH level and hematological changes due to thiamine deficiency in Mus musculus." Interdisciplinary Toxicology 11, no. 4 (2018): 321–25. http://dx.doi.org/10.2478/intox-2018-0032.

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Abstract It is known that thiamine deficiency may lead to Alzheimer’s diseases in humans. The present study has thus been conducted to understand the role of thiamine deficiency with respect to alteration in the peripheral blood of Swiss albino mice. For this purpose, adult Swiss albino mice (6–8 week old) were divided into three groups. The first group was control; the second (group II) and the third group (group III) were made thiamine deficient for 08 and 10 days respectively. Thiamine deficiency was induced in mice by injecting pyrithiamine (5 µg/10 g bwt) and feeding a thiamine deficient

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Misumida, Naoki, Hisashi Umeda, and Mitsunori Iwase. "Shoshin Beriberi Induced by Long-Term Administration of Diuretics: A Case Report." Case Reports in Cardiology 2014 (2014): 1–5. http://dx.doi.org/10.1155/2014/878915.

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Previous studies have suggested that diuretic therapy for heart failure may lead to thiamine deficiency due to the increased urinary thiamine excretion. Herein, we present the case of a 61-year-old man with shoshin beriberi, a fulminant form of wet beriberi, induced by long-term diuretic therapy. The patient had a history of heart failure with preserved ejection fraction and was receiving furosemide and trichlormethiazide therapy. He presented with worsening exertional dyspnea and was admitted for heart failure exacerbation. His condition failed to improve even after intensive treatment. A hem

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Gul-Hinc, Sylwia, Anna Michno, Marlena Zyśk, Andrzej Szutowicz, Agnieszka Jankowska-Kulawy, and Anna Ronowska. "Protection of Cholinergic Neurons against Zinc Toxicity by Glial Cells in Thiamine-Deficient Media." International Journal of Molecular Sciences 22, no. 24 (2021): 13337. http://dx.doi.org/10.3390/ijms222413337.

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Brain pathologies evoked by thiamine deficiency can be aggravated by mild zinc excess. Cholinergic neurons are the most susceptible to such cytotoxic signals. Sub-toxic zinc excess aggravates the injury of neuronal SN56 cholinergic cells under mild thiamine deficiency. The excessive cell loss is caused by Zn interference with acetyl-CoA metabolism. The aim of this work was to investigate whether and how astroglial C6 cells alleviated the neurotoxicity of Zn to cultured SN56 cells in thiamine-deficient media. Low Zn concentrations did not affect astroglial C6 and primary glial cell viability in

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Todurov, I. M., O. O. Kalashnikov, O. V. Perekhrestenko, S. V. Kosiukhno, G. C. Chervyts, and O. I. O. I. Mytsak. "Thiamine deficiency after sleeve gastrectomy." Clinical Endocrinology and Endocrine Surgery, no. 2 (October 8, 2021): 52–59. http://dx.doi.org/10.30978/cees-2021-2-52.

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The prevalence of morbid obesity has risen to global epidemic proportions. Bariatric surgery has been proven to be a safe and effective treatment for obesity with weight reduction, resolution of obesity-related co-morbidities, improved quality of life and an increased life expectancy. Sleeve gastrectomy (SG) is the most commonly performed bariatric procedure worldwide. Obesity is associated with micronutrient deficiencies that results in the high prevalence of deficient vitamins status prior to bariatric surgery. After bariatric surgery, these micronutrient and vitamins deficiencies increase o

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Doyle, Andrew, Michael E. Barnes, Jeremy L. Kientz, and Micheal H. Zehfus. "Landlocked Fall Chinook Salmon Maternal Liver and Egg Thiamine Levels in Relation to Reproductive Characteristics." Open Fish Science Journal 10, no. 1 (2017): 23–32. http://dx.doi.org/10.2174/1874401x01710010023.

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Object: Landlocked fall Chinook Salmon Oncorhynchus tshawytscha in Lake Oahe, South Dakota, typically experience poor reproductive success. Introduction: Salmon diets consist of rainbow smelt Osmerus mordax and other potentially thiaminase-containing fish that could impact reproduction. Methods: The thiamine levels of spawning female Salmon, eggs, and reproductive characteristics, were measured in 2000, 2001, 2002, 2003, and 2005. Results: Thiamine concentrations varied significantly from year-to-year, with the highest mean values recorded in 2001 at 8.70 nmol/g in maternal livers and 28.80 nm

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Bell, D., C. E. Robertson, and A. L. Muir. "Carbonated Drinks, Thiamine Deficiency and Right Ventricular Failure." Scottish Medical Journal 32, no. 5 (1987): 137–38. http://dx.doi.org/10.1177/003693308703200505.

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A 69 year old male presented with clinical features of right ventricular failure. A dilated poorly contracting right ventricle was confirmed by echocardiography and radionuclide ventriculography, with subsequent improvement following thiamine replacement. Wet beriberi is a result of thiamine deficiency and is uncommon in Europe and North America except in association with chronic alcohol abuse. We report a patient with beriberi presenting unusually with severe right-sided cardiac failure, with documented impairment of right ventricular function, which improved with thiamine replacement. His di

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Ubukata, Masamitsu, Nobuyuki Amemiya, Kosaku Nitta, and Takashi Takei. "Serum Thiamine Values in End-Stage Renal Disease Patients under Maintenance Hemodialysis." International Journal for Vitamin and Nutrition Research 85, no. 5-6 (2015): 348–55. http://dx.doi.org/10.1024/0300-9831/a000242.

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Abstract. Objective: Hemodialysis patients are prone to malnutrition because of diet or many uremic complications. The objective of this study is to determine whether thiamine deficiency is associated with regular dialysis patients. Methods: To determine whether thiamine deficiency is associated with regular dialysis patients, we measured thiamine in 100 patients undergoing consecutive dialysis. Results: Average thiamine levels were not low in both pre-hemodialysis (50.1 ± 75.9 ng/mL; normal range 24 - 66 ng/mL) and post-hemodialysis (56.4 ± 61.7 ng/mL). In 18 patients, post-hemodialysis level

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Ronowska, Anna, Agnieszka Jankowska-Kulawy, Sylwia Gul-Hinc, Marlena Zyśk, Anna Michno, and Andrzej Szutowicz. "Effects of Marginal Zn Excess and Thiamine Deficiency on Microglial N9 Cell Metabolism and Their Interactions with Septal SN56 Cholinergic Cells." International Journal of Molecular Sciences 24, no. 5 (2023): 4465. http://dx.doi.org/10.3390/ijms24054465.

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Abstract (sommario):

Mild thiamine deficiency aggravates Zn accumulation in cholinergic neurons. It leads to the augmentation of Zn toxicity by its interaction with the enzymes of energy metabolism. Within this study, we tested the effect of Zn on microglial cells cultivated in a thiamine-deficient medium, containing 0.003 mmol/L of thiamine vs. 0.009 mmol/L in a control medium. In such conditions, a subtoxic 0.10 mmol/L Zn concentration caused non-significant alterations in the survival and energy metabolism of N9 microglial cells. Both activities of the tricarboxylic acid cycle and the acetyl-CoA level were not

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Laroussi, S., K. S. Moalla, O. Hdiji, et al. "Gayet wernicke encephalopathy: Don’t miss this neuropsychiatric emergency!" European Psychiatry 64, S1 (2021): S361. http://dx.doi.org/10.1192/j.eurpsy.2021.967.

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IntroductionGayet Wernicke Encephalopathy (GWE) is a diagnostic and therapeutic neuropsychiatric emergency due to thiamin deficiency (vitamin B1).ObjectivesThe purpose of our work is to recall some clinical situations suspecting GWE, along with radiological and evolutionary profile.MethodsWe conducted a retrospective study concerning patients who were hospitalized in the neurology department of Habib Bourguiba Hospital between 2013 and 2020 for management of GWE.ResultsThe median age of 7 patients was 39.57 years with sex ratio (H/F):1.33. The most common risk factor found is incoercible vomit

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Nguyen, Ha Ai Phan, C. Pittayawonganon, H. Praekunatham, et al. "Beriberi Outbreak among Myanmar and Thai workers in a Factory in Chachoengsao Province, Thailand, 2012-2013." Outbreak, Surveillance, Investigation & Response (OSIR) Journal 7, no. 3 (2014): 1–7. http://dx.doi.org/10.59096/osir.v7i3.263286.

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Abstract (sommario):

Beriberi is a clinical syndrome which develops from prolonged severe thiamine deficiency in diet. In July 2013, the Ministry of Public Health, Thailand received reports of three deaths among Myanmar workers in a factory. We identified suspect beriberi cases, reviewed clinical history and conducted a survey among both Myanmar and Thai workers in the factory. Blood thiamine levels were measured and foods served in the factory canteen were also examined. Seventeen suspect cases (attack rate = 17%) were identified, with median age of 26 years (range 20-30 years) and male to female ratio of 3.3:1.

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W., Hliwa, Tahiri Y., Afifi M., et al. "Wernicke's Encephalopathy in Crohn's Disease: A Case Report Emphasizing Thiamine Deficiency Risk During Total Parenteral Nutrition." International Journal of Clinical Science and Medical Research 04, no. 11 (2024): 409–13. https://doi.org/10.5281/zenodo.14208079.

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Abstract (sommario):

Abstract Vitamin B1 (thiamine) is vital for carbohydrate metabolism and neurological functions. Thiamine deficiency, often associated with alcoholism, can arise due to absorption issues, increased metabolic demand, or inadequate intake. While rare in industrialized nations, it poses a risk in conditions like Crohn's disease (CD), particularly during prolonged total parenteral nutrition (TPN) without vitamin supplementation. Wernicke's Encephalopathy (WE), a complication characterized by altered mental status, ocular signs, and ataxia, can lead to Wernicke-Korsakoff syndrome (WKS). A severely m

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