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Gotowe bibliografie tematyczne / Membranoproliferative

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Gotowa bibliografia na temat „Membranoproliferative”

Autor: Grafiati

Data publikacji: 3 czerwca 2025

Data aktualizacji: 31 lipca 2025

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Artykuły w czasopismach na temat "Membranoproliferative"

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Nakopoulou, L. "Membranoproliferative glomerulonephritis." Nephrology Dialysis Transplantation 16, suppl_6 (2001): 71–73. http://dx.doi.org/10.1093/ndt/16.suppl_6.71.

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Urizar, R. E. "Membranoproliferative Glomerulonephritis." Journal of Urology 135, no. 4 (1986): 880. http://dx.doi.org/10.1016/s0022-5347(17)45884-8.

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Fogo, Agnes. "Membranoproliferative Glomerulonephritis." American Journal of Kidney Diseases 31, no. 1 (1998): E1—E2.2. http://dx.doi.org/10.1053/s0272-6386(13)90000-7.

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Alchi, Bassam, and David Jayne. "Membranoproliferative glomerulonephritis." Pediatric Nephrology 25, no. 8 (2009): 1409–18. http://dx.doi.org/10.1007/s00467-009-1322-7.

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Levine, Adam P., Melanie M. Y. Chan, Omid Sadeghi-Alavijeh, et al. "Large-Scale Whole-Genome Sequencing Reveals the Genetic Architecture of Primary Membranoproliferative GN and C3 Glomerulopathy." Journal of the American Society of Nephrology 31, no. 2 (2020): 365–73. http://dx.doi.org/10.1681/asn.2019040433.

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BackgroundPrimary membranoproliferative GN, including complement 3 (C3) glomerulopathy, is a rare, untreatable kidney disease characterized by glomerular complement deposition. Complement gene mutations can cause familial C3 glomerulopathy, and studies have reported rare variants in complement genes in nonfamilial primary membranoproliferative GN.MethodsWe analyzed whole-genome sequence data from 165 primary membranoproliferative GN cases and 10,250 individuals without the condition (controls) as part of the National Institutes of Health Research BioResource–Rare Diseases Study. We examined co

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Goto, Shin, and Ichiei Narita. "6. Membranoproliferative Glomerulonephritis." Nihon Naika Gakkai Zasshi 102, no. 5 (2013): 1145–51. http://dx.doi.org/10.2169/naika.102.1145.

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Rennke, Principal discussant: Helmut G. "Secondary membranoproliferative glomerulonephritis." Kidney International 47, no. 2 (1995): 643–56. http://dx.doi.org/10.1038/ki.1995.82.

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Bakkaloglu, A., O. S�ylemezoglu, K. Tinaztepe, �. Saat�i, and F. S�ylemezoglu. "Familial membranoproliferative glomerulonephritis." Pediatric Nephrology 10, no. 1 (1996): 40. http://dx.doi.org/10.1007/bf00863439.

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Meltem, Sezis Demirci. "Membranoproliferative Glomerulonephritis Preceding Non-Hodgkin Lymphoma Recurrence: A Case Report." Archives of Renal Diseases and Management 2, no. 1 (2016): 024–25. https://doi.org/10.17352/2455-5495.000012.

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<strong>Introduction: </strong>Membranoproliferative glomerulonephritis has been reported to occur in association with non-Hodgkin’s lymphoma but there is few information about glomerulonephritis response to the treatment of non-Hodgkin’s lymphoma. <strong>Case Presentation:</strong> A 67 years-old caucasian female admitted to outpatient clinic with peripheral edema complaint. She had mild proteinuria and microhematuria on admission and tuberculosis, non-Hodgkin lymphoma (cured), diabetes mellitus and hypertension history. Her renal biopsy resulted as  membranoproliferative&nb

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Masani, Naveed, Kenar D. Jhaveri, and Steven Fishbane. "Update on Membranoproliferative GN." Clinical Journal of the American Society of Nephrology 9, no. 3 (2014): 600–608. http://dx.doi.org/10.2215/cjn.06410613.

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Rozprawy doktorskie na temat "Membranoproliferative"

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GOBERT, REGIS. "Glomerulonephrite membranoproliferative et cancer bronchique." Lille 2, 1988. http://www.theses.fr/1988LIL2M210.

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Wong, Edwin Kwan Soon. "Complement abnormalities in membranoproliferative glomerulonephritis and C3 glomerulopathy." Thesis, University of Newcastle upon Tyne, 2016. http://hdl.handle.net/10443/3307.

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Membranoproliferative glomerulonephritis (MPGN) and C3 glomerulopathy (C3G) are rare diseases that associate with dysregulation of the alternative pathway (AP). The earliest abnormalities associated with these diseases were C3 nephritic factor and also rare genetic variants in the gene CFH that caused factor H (FH) deficiency. Since then, other acquired and genetic abnormalities in AP have been reported in MPGN and C3G. The aim of this project was to screen cohorts of MPGN and C3G for such abnormalities. Screening for rare sequence variants in genes encoding proteins involved in AP activity in

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Melin, Pascal. "Hepatite c et glomerulo-nephrite membrano-proliferative : reflexion sur la relation de causalite a partir d'observations recueillies en champagne-ardenne." Reims, 1993. http://www.theses.fr/1993REIMM081.

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Rückert, Franziska [Verfasser], Wolfgang [Gutachter] Rascher, and Kerstin [Gutachter] Benz. "Membranoproliferative Glomerulonephritis - Krankheitsverlauf und Therapie pädiatrischer Patienten / Franziska Rückert ; Gutachter: Wolfgang Rascher, Kerstin Benz." Erlangen : Friedrich-Alexander-Universität Erlangen-Nürnberg (FAU), 2017. http://d-nb.info/1125715464/34.

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Saez, Valérie. "Manifestations extrahépatiques inaugurales de l'infection par le virus de l'hépatite C : à propos d'un cas." Montpellier 1, 1996. http://www.theses.fr/1996MON11176.

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Carneiro, Márcia Fernanda dos Santos Melo. "Contribuição ao estudo das doenças glomerulares na Bahia: estudo prospectivo de glomerulopatias – PROGLOM." reponame:Repositório Institucional da UFBA, 2013. http://www.repositorio.ufba.br/ri/handle/ri/12968.

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Submitted by Hiolanda Rêgo (hiolandar@gmail.com) on 2013-09-17T17:06:21Z No. of bitstreams: 1 Dissertação_ICS_Márcia Fernanda dos Santos Melo Carneiro.pdf: 3252453 bytes, checksum: 8e1c2cfe0185223bd295094f77aa813a (MD5)<br>Approved for entry into archive by Patricia Barroso(pbarroso@ufba.br) on 2013-09-17T17:43:10Z (GMT) No. of bitstreams: 1 Dissertação_ICS_Márcia Fernanda dos Santos Melo Carneiro.pdf: 3252453 bytes, checksum: 8e1c2cfe0185223bd295094f77aa813a (MD5)<br>Made available in DSpace on 2013-09-17T17:43:10Z (GMT). No. of bitstreams: 1 Dissertação_ICS_Márcia Fernanda dos Santos Mel

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Do, Vale Claudia Lewis. "A South African case study of familial membranoproliferative glomerulonephritis." Thesis, 2015. http://hdl.handle.net/10539/18427.

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Membranoproliferative glomerulonephritis (MPGN) is an immune-mediated disease that is characterised by mesangial hypercellularity and endocapilliary proliferation with capillary wall remodeling which results in thickening of the glomerular basement membrane. (1) MPGN was traditionally classified on electron microscopy findings into MPGN type I, II, and III. Multiple genetic risk factors have been identified for MPGN II now referred to as dense deposit disease; however, relatively little is known regarding genetic risk factors for MPGN type I and III. A reclassification scheme that better refl

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Jamal, Sheila Sadik Koch. "Glomerulonefrite membranoproliferativa tipo II : caso clínico." Master's thesis, 2014. http://hdl.handle.net/10451/24563.

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Trabalho Final do Curso de Mestrado Integrado em Medicina, Faculdade de Medicina, Universidade de Lisboa, 2014<br>Membranoproliferative glomerulonephritis type II or dense deposit disease is a rare form of glomerulonephritis, where the main pathophysiological mechanism is excess activation of the alternative pathway of complement, which results in the deposition of complement component in the glomerular basement membrane. This is a pattern of glomerular injury with specific changes in the renal biopsy, namely mesangial hypercelularity, endocapilar proliferation and double contour formation alo

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Heinen, Stefan [Verfasser]. "Die Rolle von Faktor H im atypischen hämolytisch urämischen Syndrom (aHUS) und der membranoproliferativen Glomerulonephritis Typ 2 (MPGN II) / von Stefan Heinen." 2006. http://d-nb.info/981975984/34.

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Książki na temat "Membranoproliferative"

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Gale, Daniel P., and Terry Cook. Membranoproliferative glomerulonephritis and C3 glomerulopathy. Edited by Neil Turner. Oxford University Press, 2015. http://dx.doi.org/10.1093/med/9780199592548.003.0080.

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Membranoproliferative glomerulonephritis (MPGN) is synonymous with mesangiocapillary glomerulonephritis and refers to light microscopic appearances of a kidney biopsy in which there are increased mesangial cells and matrix with thickening of the glomerular capillary walls, often with a double contour appearance. MPGN represents morphological appearances caused by a wide range of diseases, most of which are systemic and involve activation of the immune system. It commonly presents as nephrotic syndrome, alternatively with proteinuria, haematuria, and varying degrees of hypertension and renal dy

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Clayton, Philip, and Steven Chadban. Recurrent renal disease. Edited by Jeremy R. Chapman. Oxford University Press, 2015. http://dx.doi.org/10.1093/med/9780199592548.003.0289.

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Although kidney transplantation restores kidney function, it does not cure the cause of kidney failure when it is glomerulonephritis or a systemic disease. These can recur in the allograft, causing graft dysfunction and ultimately graft failure. This is a challenge in the pre-transplant phase, perioperatively, and in a patient with renal allograft dysfunction. This chapter discusses the prophylaxis, diagnosis, and management of recurrent renal disease, including disease-specific information on focal and segmental glomerulosclerosis, membranous nephropathy, membranoproliferative glomerulonephri

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Fervenza, Fernando C., An S. De Vincent Rajkumar, An S. De Vriese, and Sanjeev Sethi. Other consequences from monoclonal immunoglobulins/fragments. Edited by Giuseppe Remuzzi. Oxford University Press, 2018. http://dx.doi.org/10.1093/med/9780199592548.003.0155_update_001.

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Renal damage secondary to deposition of monoclonal immunoglobulin can occur due to accumulation of either light chains, heavy chains, or both. These include myeloma kidney (cast nephropathy), light-chain and heavy-chain amyloidosis, and light- and heavy-chain deposition disease. Renal damage secondary to deposition of both chains is far less common. In the great majority of these cases the M-component is immunoglobulin G, but the spectrum of renal lesions associated with monoclonal gammopathy is extensive and depends on the physiochemical properties of the immunoglobulin produced.This chapter

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Fabrizi, Fabrizio. Hepatitis C. Edited by Vivekanand Jha. Oxford University Press, 2015. http://dx.doi.org/10.1093/med/9780199592548.003.0186.

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Hepatitis C virus (HCV) is associated with a large spectrum of histopathological lesions in both native and transplanted kidneys. The exact frequency of kidney damage in HCV-infected patients remains unknown, but the most frequent associated renal lesion is membranoproliferative glomerulonephritis (MPGN) with deposition of immunoglobulin and complement (MPGN type 1), usually in the context of type II mixed cryoglobulinaemia associated with a monoclonal IgM which binds IgG.Various approaches have been tried for the treatment of HCV-related glomerulonephritis, including immunosuppressive therapy

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Fabrizi, Fabrizio, and Patrice Cacoub. The patient with cryoglobulinaemia. Edited by Giuseppe Remuzzi. Oxford University Press, 2015. http://dx.doi.org/10.1093/med/9780199592548.003.0151.

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AbstractCryoglobulinaemia is characterized by the presence in the blood of proteins showing the that precipitate when serum is cooled. Clinically recognised cryoprecipitates are predominantly immunoglobulin-containing. In Type 1 cryoglobulinaemia, the precipitate is formed from a monoclonal paraprotein, usually IgG. In Type 2, a monoclonal IgM binds IgG to form a mixed precipitate. Type 3 cryoglobulins do not contain a monoclonal element.Type 1 cryoglobulins are a rare cause of renal disease, but cause a membranoproliferative glomerulonephritis (MPGN) with nephrotic syndrome and haematuria and

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Sakhuja, Vinay, and Harbir Singh Kohli. Malaria. Edited by Vivekanand Jha. Oxford University Press, 2018. http://dx.doi.org/10.1093/med/9780199592548.003.0183_update_001.

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Of the four pathogenic malarial species, clinically significant renal dysfunction is mainly associated with Plasmodium malariae and Plasmodium falciparum infections.P. falciparum infection frequently causes acute kidney injury (AKI). AKI may be the sole manifestation with a complete recovery after treatment or it may be a part of multi-organ failure which is often fatal. AKI due to Plasmodium vivax infection alone or as a result of mixed infection by vivax and falciparum can also occur.‘Quartan malarial nephropathy’ has been attributed to P. malariae infection although this relationship must b

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Rodriguez-Iturbe, Bernardo, and Mark Haas. Glomerulonephritis associated with endocarditis, deep-seated infections, and shunt nephritis. Edited by Neil Turner. Oxford University Press, 2018. http://dx.doi.org/10.1093/med/9780199592548.003.0079_update_001.

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Endocarditis is a cause of glomerulonephritis. Healthcare interventions (prosthetic valves, indwelling catheters, pacemaker wires) and intravenous drug abuse are presently the most common causes of endocarditis and Staphylococcus aureus is frequently the infecting bacteria. Shunt nephritis is a form of glomerulonephritis associated with infection of ventriculoatrial shunts implanted to relieve hydrocephalus and, typically, are caused by prolonged infections of low-pathogenicity microorganisms. This complication led to the replacement of the technique by ventriculoperitoneal shunts. Deep-seated

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Muche, Marion, and Seema Baid-Agrawal. Hepatitis B. Edited by Vivekanand Jha. Oxford University Press, 2018. http://dx.doi.org/10.1093/med/9780199592548.003.0185_update_001.

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Hepatitis B virus (HBV) has been causally linked to a variety of renal diseases, the most common being glomerular diseases and systemic autoimmune disease. Membranous nephropathy (MN) is the commonest HBV-associated glomerulonephritis (HBV-GN), followed by membranoproliferative glomerulonephritis (MPGN), mesangial proliferative glomerulonephritis, immunoglobulin (Ig)-A nephropathy, and focal segmental glomerulosclerosis (FSGS). Polyarteritis nodosa is a rare manifestation. The incidence of HBV-associated renal diseases seems to be decreasing with the introduction of vaccination programmes.HBV-

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Części książek na temat "Membranoproliferative"

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Braun, Michael C., Christoph Licht, and C. Frederic Strife. "Membranoproliferative Glomerulonephritis." In Pediatric Nephrology. Springer Berlin Heidelberg, 2009. http://dx.doi.org/10.1007/978-3-540-76341-3_32.

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Timson, David J., Richard J. Reece, James B. Thoden, et al. "Glomerulonephritis, Membranoproliferative." In Encyclopedia of Molecular Mechanisms of Disease. Springer Berlin Heidelberg, 2009. http://dx.doi.org/10.1007/978-3-540-29676-8_1121.

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Oette, Mark, Marvin J. Stone, Hendrik P. N. Scholl, et al. "Membranoproliferative Glomerulonephritis." In Encyclopedia of Molecular Mechanisms of Disease. Springer Berlin Heidelberg, 2009. http://dx.doi.org/10.1007/978-3-540-29676-8_9270.

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Sethi, Sanjeev, and Fernando C. Fervenza. "Membranoproliferative Glomerulonephritis." In Core Concepts in Parenchymal Kidney Disease. Springer New York, 2013. http://dx.doi.org/10.1007/978-1-4614-8166-9_6.

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Diaz-Encarnación, Montserrat, Gerson Berna, Yolanda Arce, Xoana Barros, and Helena Marco. "Membranoproliferative Glomerulonephritis." In Autoimmune Disease Diagnosis. Springer Nature Switzerland, 2024. https://doi.org/10.1007/978-3-031-69895-8_100.

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Alexander, Mariam P., and Sanjeev Sethi. "Membranoproliferative Glomerulonephritis, Adult." In Glomerulonephritis. Springer International Publishing, 2019. http://dx.doi.org/10.1007/978-3-319-49379-4_22.

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Alexander, Mariam P., and Sanjeev Sethi. "Membranoproliferative Glomerulonephritis, Adult." In Glomerulonephritis. Springer International Publishing, 2017. http://dx.doi.org/10.1007/978-3-319-27334-1_22-1.

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Díaz, Montserrat M., Laia Sans, Yolanda Arce, and José Ballarin. "Membranoproliferative or Mesangiocapillary Glomulonephritis." In Diagnostic Criteria in Autoimmune Diseases. Humana Press, 2008. http://dx.doi.org/10.1007/978-1-60327-285-8_92.

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Floege, Jürgen. "Membranoproliferative Glomerulonephritis und Kryoglobulinämie." In SpringerReference Innere Medizin. Springer Berlin Heidelberg, 2014. http://dx.doi.org/10.1007/978-3-642-54676-1_48-1.

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Argyropoulos, Christos P., Sheldon Bastacky, and John Prentiss Johnson. "HCV-Associated Membranoproliferative Glomerulonephritis." In HCV Infection and Cryoglobulinemia. Springer Milan, 2011. http://dx.doi.org/10.1007/978-88-470-1705-4_22.

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Streszczenia konferencji na temat "Membranoproliferative"

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lorenzoni Grillo, Luiza, Debora Marques Veghini, Anna Carolina Simões Moulin, et al. "TAKAYASU’S ARTERITIS ASSOCIATED TO MEMBRANOPROLIFERATIVE GLOMERULONEPHRITIS: A CASE REPORT." In SBR 2021 Congresso Brasileiro de Reumatologia. Sociedade Brasileira de Reumatologia, 2021. http://dx.doi.org/10.47660/cbr.2021.2164.

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