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Статті в журналах з теми "Flux coronaire":
Meimoun, P., S. Sayah, B. Maitre, A. L. Bore, T. Benali, M. Beausoleil, and J. Bailly. "Mesure du flux et de la réserve coronaire par échographie transthoracique : un vieux concept, un outil moderne, des intérêts multiples." Annales de Cardiologie et d'Angéiologie 53, no. 6 (November 2004): 325–34. http://dx.doi.org/10.1016/j.ancard.2004.09.009.
Pinaquy, J. B., P. Ferenczi, F. Debordeaux, L. Bordenave, H. Douard, T. Couffinhal, and Y. Pucheu. "Corrélation entre les flux myocardiques, la réserve coronaire et les scores de risque cardiovasculaire Framingham et ESC-SCORE(+ Running poster)." Médecine Nucléaire 45, no. 4 (July 2021): 203. http://dx.doi.org/10.1016/j.mednuc.2021.06.056.
Bailly, M., F. Thibault, M. Courtehoux, G. Metrard, and M. J. Santiago Ribeiro. "Effet de la correction d’atténuation sur le calcul des flux et de la réserve coronaire en scintigraphie myocardique sur caméra CZT." Médecine Nucléaire 44, no. 2 (March 2020): 101–2. http://dx.doi.org/10.1016/j.mednuc.2020.01.118.
Huang, Qiaobing, Mac Wu, Cynthia Meininger, Katherine Kelly, and Yuan Yuan. "Neutrophil-dependent augmentation of PAF-induced vasoconstriction and albumin flux in coronary arterioles." American Journal of Physiology-Heart and Circulatory Physiology 275, no. 4 (October 1, 1998): H1138—H1147. http://dx.doi.org/10.1152/ajpheart.1998.275.4.h1138.
ROGERS, S., A. KHALATBARI, B. DATTA, S. ELLERY, V. PAUL, M. FRENNEAUX, and P. JAMES. "NO metabolite flux across the human coronary circulation." Cardiovascular Research 75, no. 2 (July 15, 2007): 434–41. http://dx.doi.org/10.1016/j.cardiores.2007.04.019.
SYRRIS, Petros, Nicholas D. CARTER, James C. METCALFE, Paul R. KEMP, David J. GRAINGER, Juan C. KASKI, David C. CROSSMAN та ін. "Transforming growth factor-β1 gene polymorphisms and coronary artery disease". Clinical Science 95, № 6 (1 грудня 1998): 659–67. http://dx.doi.org/10.1042/cs0950659.
Jardine, Moira M. "Topology of stellar coronae." Proceedings of the International Astronomical Union 2, no. 14 (August 2006): 289–90. http://dx.doi.org/10.1017/s1743921307010642.
Schrijver, Carolus J., and Alan M. Title. "The Heating of Cool‐Star Coronae: From Individual Loops to Global Flux‐Flux Scalings." Astrophysical Journal 619, no. 2 (February 2005): 1077–83. http://dx.doi.org/10.1086/426709.
Noll, T., A. Hempel, and H. M. Piper. "Neuropeptide Y reduces macromolecule permeability of coronary endothelial monolayers." American Journal of Physiology-Heart and Circulatory Physiology 271, no. 5 (November 1, 1996): H1878—H1883. http://dx.doi.org/10.1152/ajpheart.1996.271.5.h1878.
Honig, C. R., J. L. Frierson, and T. E. Gayeski. "Anatomical determinants of O2 flux density at coronary capillaries." American Journal of Physiology-Heart and Circulatory Physiology 256, no. 2 (February 1, 1989): H375—H382. http://dx.doi.org/10.1152/ajpheart.1989.256.2.h375.
Дисертації з теми "Flux coronaire":
Adjedj, Julien. "Circulation coronaire : Principes et méthodes de mesure invasive du flux coronaire segmentaire en pratique clinique." Thesis, Paris Est, 2017. http://www.theses.fr/2017PESC0006/document.
Coronary circulation is complex and highly regulated while invasive coronary flow measurements techniques allow the assessment of coronary physiology in clinical practice. Material et methods We describe in two reviews the principles and methods of different invasive coronary flowmeasurements techniques in clinical practice. We study the clinical impact of fractional flow reserve(FFR) in intermediate coronary stenosis, the hyperemic agents and dosage to measure FFR and FFRcorrelation with angiographic indices according to risk factors accumulation. Finally, we describe the principle and method of coronary flow and microvascular resistances measurements with a dedicated infusion microcatheter for coronary thermodilution to obtain assessment of macro and microvascular components of coronary circulation. Results We recommend the FFR cut off value of 0.80 to guide revascularization based on our study showing higher myocardial infarction and death rate in patients treated with medical therapy and FFR<0.80compared to those with FFR>0.80, respectively 9.4 versus 4.8%, P=0.06 and 7,5 versus 3,2%, P=0.06. We studied different hyperemic agents and dosages and showed that intracoronary adenosine at 100μg to 200 μg induce maximal hyperemia while contrast medium induce 65% of maximal hyperemia. Therefore, FFR measurements with contrast medium is feasible and has better accuracy than restindices compared to FFR. We establish the weak correlation between FFR and angiographic indicesand weakens correlation as risk factors accumulates, especially in diabetic patients. Finally, we described in three studies the method of absolute coronary flow and microvascular resistancesmeasurements based on thermodilution principle with a dedicated infusion catheter. We showed anaccurate measurement with this technique (R=0.98), which induces maximal hyperemia without theneed of hyperemic agent with reproducible measurements in humans (R=0,91).Conclusion The use of invasive coronary flow measurements to study the coronary circulation is essential inclinical practice and in research
Taurel, Marianne. "Evaluation de la réserve coronaire et/ou de la fonction endothéliale dans l'insuffisance cardiaque et l'athérosclérose expérimentales : effets des inhibiteurs du système rénine-angiotensine." Paris 11, 2001. http://www.theses.fr/2001PA11T021.
Lethimonnier, Franck. "Evaluation des cardiopathies ischémiques par IRM quantification du flux et de la réserve coronaire : visualisation des sténoses par angiographie 3D : étude du retentissement fonctionnel par analyse de la fonction ventriculaire gauche." Angers, 1999. http://www.theses.fr/1999ANGE0508.
Rasser, Charlotte. "Biocompatibility improvement conferred by the immobilization of a CD31 peptide on endovascular stents." Thesis, Sorbonne Paris Cité, 2017. http://www.theses.fr/2017USPCC317/document.
Over the last decades, coronary stents and intracranial flow diverting stents have revolutionized the endovascular treatment of two different arterial pathologies: coronary artery disease and intracranial aneurysms. The working mechanisms of these metallic endoprostheses are different but both are associated with complications stemming from biocompatibility issues. In particular, the rapid covering by endothelial cells presenting an anti-inflammatory and anti-thrombotic phenotype is key to the integration of the endoprosthesis at the blood/vessel interface. Thus, the development of solutions to improve the endothelialization and the integration of these two types of stents in the vessel wall would represent a major progress in their respective field.In this context, this thesis work deals with the immobilization of a bioactive molecule on coronary stents and flow diverting stents in order to solve their biocompatibility issues. The bioactive molecule that we used is a synthetic peptide, named P8RI, which promotes the regulatory functions of the transmembrane glycoprotein CD31 : the inhibition of platelets and leukocytes activation, as well as the enhancement of endothelial cell survival, migration and barrier function.The first part of this thesis work consisted in the development of a process for the immobilization of P8RI on metallic stents. We adopted three successive approaches: the direct immobilization of the peptide on plasma-functionalized alloy surfaces; the plasma-enhanced chemical vapor deposition of an intermediate polymeric layer; and the deposition of a polydopamine coating by self-polymerization, followed with the immobilization of a linker and the binding of P8RI by copper-free click chemistry.We then carried out an in vitro evaluation of the biocompatibility of the resulting coated alloy surfaces, in terms of anti-thrombotic, anti-inflammatory, and pro-endothelialization properties. The surfaces on which P8RI had been immobilized were shown to exhibit a tendency to decrease platelet adhesion, increase endothelial cell adhesion and barrier function, and promote an anti-inflammatory and anti-thrombotic phenotype in human vascular endothelial cells.Finally, coronary stents and flow diverting stents were evaluated in vivo. Coronary stents were implanted in the coronary arteries of farm pigs, and preliminary results showed a more complete endothelialization and a lesser density of adherent leukocytes on ‘P8RI-coated’ stents than on the controls. ‘P8RI-coated’ flow diverting stents were implanted in a rabbit elastase-induced carotid aneurysm model. Compared with the controls, they were associated with the formation of a thicker and better organized neointima, in particular on the stent struts in front of the aneurysm neck, which implies lesser risks of persistence of blood flow and aneurysm rupture
Gibb, Gordon P. S. "The formation and eruption of magnetic flux ropes in solar and stellar coronae." Thesis, University of St Andrews, 2015. http://hdl.handle.net/10023/7069.
Abdalla, Luiz Antonio 1953. "Efeitos da vitamina E sobre o fluxo coronario endotelio-dependente em cães hipercolesterolemicos." [s.n.], 2002. http://repositorio.unicamp.br/jspui/handle/REPOSIP/313462.
Tese (doutorado) - Universidade Estadual de Campinas, Faculdade de Ciencias Medicas
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Resumo: Foram estudados os efeitos da vitamina E sobre o fluxo coronário endotélio-dependente em cães hipercolesterolêmicos. A pesquisa foi realizada em 21 cães machos adultos, pesando em média 7,4±1,0 kg, que foram divididos em três grupos: controle, hipercolesterolêmico e vitamina E. Os animais do grupo hipercolesterolêmico foram alimentados com uma dieta rica em colesterol (5% g/g) e óleo de coco (10% g/g) por 40 dias. O grupo vitamina E recebeu a mesma dieta, acrescida de 400 Ui de vitamina E, durante os últimos 15 dias do experimento. Os níveis de colesterol total sérico foram avaliados no começo e no final do estudo, por meio de "kits" enzimáticos e espectrofotômetro Beckman 700. O fluxo coronário foi medido por um fluxômetro eletromagnético (Caroline Instruments, Inc.), usando-se um probe, posicionado na artéria coronária descendente anterior esquerda, próximo ao óstio. Uma agulha, conectada a uma bomba de infusão, foi introduzida na artéria coronária para administração de acetilcolina (ACH) e nitroprussiato de sódio (NPS), à velocidade de 5 mg/kg/min. A aorta foi canulada para medida da pressão arterial por intermédio de transdutor acoplado a polígrafo da marca Siemmens (Mingograph 804). Os teores de colesterol e malonodialdeído (MOA) foram, também, medidos em segmentos de vaso coronário.Após 40 dias, os níveis de colesterol sérico haviam aumentado em 203%, no grupo hipercolesterolêmico e, em 198%, no grupo da vitamina E. Entretanto, esta diferença não foi significante (P>0,05). A pressão sangüínea na aorta e a freqüência cardíaca permaneceram sem alteração, durante a administração de ACH. Contudo, a pressão sistólica e a diastólica diminuíram e a freqüência cardíaca aumentou durante a infusão de NPS.O teor de colesterol tecidual e MOA aumentaram, significativamente, (P<0,05) nas amostras de artéria coronária do grupo hipercolesterolêmico, comparativamente aos animais do grupo-controle. A vitamina E reduziu o colesterol tecidual e o MDA nos animais hipercolesterolêmicos (P<0,05).A elevação do percentual do fluxo coronário, durante a administração da ACH, foi significativamente menor no grupo hipercolesterolêmico, quando comparada com an1ma1_ do grupo-controle (P
Abstract: The effects of vitamin E on endothelium-dependent coronary flow were studied in hypercholesterolernic dogs. Adult mongrel dogs weighing 7.4±1.0 kg were divided into control, hypercholesterolernic and vitamin E groups. The animals in the hypercholesterolernic group were fed a diet enriched with cholesterol (5%w/w) and coconut oil (l0%w/w) for 40 days. The vitamin E group received the same diet plus 400 IU of vitamin E during the last 15 days of the experiment. Total serum cholesterol levels were evaluated at the beginning and at the end of the experiment using a commercial enzyme kit and a Beckman analyzer. The coronary flow was determined byelectromagnetic flowmetry using a probe positioned in the left anterior descending coronary artery, near the ostium. A needle connected to a perfusion pump was introduced into the coronary artery for the administration of acetycholine and sodium nitroprusside at a rate of 5 ug!kg per min. The aorta was cannulated for the measurement of arterial blood pressure via a pressure transducer coupled to a Siemens multi-channel recorder. The tissue cholesterol content and malonic dialdehyde (MDA) were also measured in isolated coronary vessel specimens. At the end of 40 days, the serum cholesterol levels had increased by 203% and 198% in the hypercholesterolernic and vitamin E groups, respectively. However, the difference in the levels of these two groups was not significant (P>0.05). The aortic blood pressure and heart rate remained inchanged during acetylcholine administration. In contrast, systolic and diastolic pressure fell and the heart rate increased during the infusion of sodium nitroprusside. The tissue cholesterol content and MDA were significantly (P<0.05) increased in coronary artery specimens from the hypercholesterolernic compared to control animals. Vitamin E was able to reduce these increases in cholesterol treated animals (P<0.O5). The percent change in coronary flow during acetylcholine administration was significantly lower in the hypercholesterolernic group when compared with control animals (P<0.05) but was unaltered in the vitamin E group (P>0.05). During sodium nitroprusside administration, the coronary flow increased in the vitamin E group (P
Medicina Interna
Doutor em Ciências Médicas
Melis, Riera Gaspar. "Papel de la disfunción endotelial en la respuesta a la cocaína del flujo coronario y la contractilidad miocárdica." Doctoral thesis, Universitat de Barcelona, 2003. http://hdl.handle.net/10803/2174.
Nuestra hipótesis es que la cocaína causa una depresión de la función ventricular independiente de su efecto sobre el flujo coronario y que esta depresión se agrava en presencia de disfunción endotelial. Analizamos los efectos de la administración sistémica de cocaína sobre el flujo coronario y la función ventricular en un modelo experimental porcino, y si la inhibición de la síntesis de óxido nítrico mediante L-NAME intracoronaria modifica la acción de la cocaína sobre la función ventricular.
2. METODOLOGIA
Se utilizaron 57 animales, en un modelo experimental con anestesia general y con un protocolo quirúrgico que permite analizar las siguientes variables:
a)Parámetros hemodinámicos: Tensión arterial, frecuencia cardíaca, doble producto y presión telediastólica del ventrículo izquierdo.
b)Fracción de acortamiento de la fibra miocárdica
c)Flujo coronario y resistencias coronarias
d)Parámetros adicionales: dP/dt del ventrículo izquierdo, gasto cardíaco y resistencias vasculares sistémicas.
Los animales se aleatorizarón en seis grupos.
Grupo control: salino i.v. (n=5). Grupo 1: cocaína i.v. 10 mg kg -1 durante 20 min. (n=17); Grupo 2: la misma dosis de cocaina después de una infusión intracoronaria de L-NAME 20 mg/kg min -1 (n=16); grupo 3:la misma dosis de cocaína administrada después de la reducción mecánica del 20% del flujo coronario de la arteria descendente anterior mediante una oclusión mecánica externa (n=9); grupo 4: la misma dosis de cocaína después de la administración simultanea de L-arginina y L-NAME intracoronaria (n=5); grupo 5: la misma dosis de cocaína administrada después de la administración simultanea de L-NAME y nitroglicerina intracoronaria ( n=5).
3. RESULTADOS
La cocaína no altero el flujo coronario, mientras que indujo una reducción significativa del gasto cardíaco, de la dp/dt del ventrículo izquierdo y de la fracción de acortamiento. Cuando se administró la cocaína después de la infusión i.c. de L-NAME durante 30 min se produjo una mayor reducción de la fracción de acortamiento, este efecto se abolió con la administración simultanea al L-NAME de L-arginina y nitroglicerina. Los resultados de la administración de cocaína después de la reducción mecánica del 20% del flujo coronario no difieren de los producidos con cocaína sola.
4. CONCLUSIÓN
La cocaína produce una depresión directa de la contractilidad miocárdica. Al inhibir la síntesis de óxido nítrico con L-NAME se produce una vasoconstricción de la microcirculación coronaria que aumenta el efecto depresor de la cocaína sobre el miocardio.
"Role of endotelial dysfuntion on cocaine effects in coronary flow and myocardial contractility "
1. Hypothesis
Cocaine has been associated with myocardial ischaemia and left ventricular dysfunction. Whether nitric oxide (NO) inhibition may potentiate the effects of cocaine on coronary circulation and ventricular function is still unknown.
2.Material and methods
In order to test this hipótesis, 57 pentobarbital-anaesthetized pigs were instrumenteted for systolic blood pressure, coronary blood flow, left ventricular dp/dt, cardiac output, left ventricular end-diastolic and end-systolic leghts and shortening fraction.
The pigs were randomized into six groups: control group: i.v. saline(n=5); group 1: cocaine, 10 mg kg-1 over 20 min (n=17); group 2 : the same dosis of cocaine 30 min after i.c. L-NAME 20 mg/kg min-1 infusion( n=16); group 3: The same dosis of cocaine after the proximal left anterior descending (LAD) flow was reduced to around 20% of the basl value by means of a mechanical occluder (n=9); group 4: the same dosis of cocaine after i.c. simultaneously L-arginina and L-NAME infusion (n=5); group 5: the same dosis of cocaine after simultaneously i.c. L-NAME and nitroglicerine infusion.
3.Results
Cocaine i.v. did not change the coronary blood flow, while it induced a significant reduction in cardiac output, left ventricular dp/dt and shortening fraction. When cocaine was administered after L-NAME infused i.c. a significant more severe reduction of the shortening fraction was induced; this effect was abolished by simultaneosus perfusion of L-arginine and NAG i.c.. The results when cocaine was administered after 20% LAD flow reduction by mechanical occluder did not differ from those of cocaine alone.
4.Conclusions
NO inhibition intesifies the cocaine-induced left ventricular funtion.
Sant'Anna, Fernando Mendes. "Influência da avaliação rotineira do fluxo fracionado de reserva durante intervenções coronárias percutâneas na estratégia terapêutica." Universidade de São Paulo, 2006. http://www.teses.usp.br/teses/disponiveis/5/5131/tde-06092006-192836/.
BACKGROUND: In complex and multivessel coronary artery disease, it is often difficult to assess which lesions are associated with reversible ischemia and should be stented. Fractional flow reserve (FFR) is a well established methodology to indicate which lesions are culprit or not. Yet, frequently the selection of lesions to be stented is based on the angiogram alone. The main aim of this study in patients admitted for elective percutaneous coronary intervention (PCI) was to evaluate the percentage of change in the initial therapeutic plan if decision is based on FFR measurement rather than on angiographic assessment. METHODS: All patients scheduled for elective PCI between October 2004 and April 2005 were included in the study except those with chronic total occlusion. Two hundred and fifty patients and 471 arteries with a stenosis ≥ 50% by visual estimation and initially selected to be stented were assessed by FFR measurements. Before PCI, 3 cardiologists independently reviewed the diagnostic angiogram and classified lesions as those that should be treated by PCI by visual assessment and those that should not be treated. Next, the decision to stent was based upon FFR measurement. If FFR was < 0.75, actual stenting was performed; if FFR was ≥ 0.75, no interventional treatment was given. RESULTS: It was possible to perform optimal pressure measurements and FFR determinations in 452 (96%) lesions. Mean diameter stenosis was 62 ± 12% and average FFR 0.67 ± 0.17 for the entire group. In 68% of the stenoses initial therapeutic strategy as assessed from the angiogram was followed and in 32% there was a change in the planned approach based on FFR. In 100 stenoses (22%) PCI planned on the basis of angiography was deferred, and in 44 stenoses (10%) revascularization was performed although such stenosis was not considered as ischemia-related on the angiogram. In 48% of the patients there was at least one lesion in which the treatment decision was changed after physiologic measurements. CONCLUSIONS: In this prospective, non-selective, but complete study representing the real world of PCI, 32% of the coronary stenoses and 48% of patients would have received a different treatment if solely the visual assessment by angiography was followed, stressing the utility of physiologic assessment in refining decision making during PCI.
Redondo, Fernanda Roberta Roque. "Efeitos do uso de esteróides anabolizantes associados ao treinamento físico de natação sobre o fluxo sangüíneo para o miocárdio de ratos normotensos." Universidade de São Paulo, 2007. http://www.teses.usp.br/teses/disponiveis/39/39132/tde-20042007-100232/.
The abusive use of ergogenic resources as the anabolic steroid became an increasing problem in several segments of the population, beyond the athletical way, searching for better performance or physical appearance, without being worried about the risks of the collateral effects promoted by this practice. In the present work we studied the effects of the use of supraphysiological doses of anabolic steroids associated with aerobic swimming training on the coronary blood flow of normotensive rats, investigating the participation of adenosine as one of the possible mechanisms of blood flow regulation, besides the cardiac structural alterations that could influence the coronary blood perfusion. The effect of the physical training was efficient to promote beneficial adaptations of the cardiovascular system, as the presence of physiological cardiac hypertrophy and improves the coronary blood flow at rest, probably mediated by a higher circulating and cardiac adenosine production. The use of anabolic steroids associated with the swimming training attenuated the beneficial effect promoted by training, being observed the presence of cardiac hypertrophy, followed by reduction of cardiac output and coronary blood flow, mediated by lower circulating adenosine production, besides the impairment of the vasodilator response to the acetylcholine, demonstrating a probable endothelial dysfunction and reduction of the cardiac capillary density, characterizing in this way, a pathological state
Lima, Marta Fernandes. "Valor prognóstico da reserva de fluxo coronário e miocárdico obtida pela ecocardiografia contrastada em pacientes com cardiomiopatia dilatada de origem não isquêmica." Universidade de São Paulo, 2012. http://www.teses.usp.br/teses/disponiveis/5/5131/tde-30072012-081401/.
Despite advances in understanding of the pathophysiology and therapeutic approaches, dilated cardiomyopathy (DCM) remains as a condition with high mortality and one of the mechanisms involved in this process seems to be microvascular dysfunction. Recently, new echocardiographic techniques have been incorporated in the clinical practice and used for the assessment of microvascular dysfunction, including evaluation of left anterior descending coronary artery (LAD) by Doppler and quantitative analysis of myocardial flow by real-time myocardial perfusion echocardiography (RTMPE). In the present study, we evaluated the prognostic value of coronary flow velocity reserve (CFVR), obtained by transthoracic echocardiography, and parameters of myocardial flow reserve (MFR), obtained by RTMPE for predicting cardiac death and heart transplantation in patients with nonischemic DCM. In addition, we sought to detect the incremental value of flow reserve over clinical and echocardiographic parameters already known as predictors of events in this population. We studied 195 patients with DCM (130 men, mean age 54 ± 12 years) who had left ventricular ejection fraction (LVEF) less than 35% by echocardiography and no signs of obstructive coronary artery disease by invasive coronary angiography or noninvasive methods (nuclear medicine or angiography by computed tomography). We assessed conventional echocardiographic parameters of systolic and diastolic left ventricular function at rest. Coronary flow velocity was determined by pulsed Doppler in LAD and dynamics of microbubbles in the myocardium was measured by RTMPE using specific computer programs, both at rest and during dipyridamole stress (0.84 mg/kg). CFVR, MFR and the reserve of velocity of microbubbles in the myocardium ( reserve) were obtained as the ratio between parameters of flow during hyperemia and at rest, being considered abnormal when these were below 2.0. The mean follow-up was 29 months (ranging from 6 to 69 months). During this period, 45 patients (24%) had events, 43 cardiac deaths and 2 heart transplantations. In the univariate analysis, predictors of events were: Chagas disease, New York Heart Association functional class, use of angiotensin-converting enzyme inhibitors/angiotensin II receptor blocker (protective effect), left ventricular and atrial diameters, left ventricular volumes, LVEF, diastolic dysfunction, degree of mitral regurgitation, CVFR, MFR and reserve. By multivariate analysis, the only independent predictors of events were the left atrial diameter (OR = 1.16, 95% confidence interval = 1.078 to 1.264, p <0.001) and reserve 2.0 (OR = 3.219, confidence interval 95 % = 1.178 to 8.795, p <0.001). In the sequential model of events prediction, LVEF and left atrial diameter added prognostic value over clinical factors (chi-square from 15.2 to 58.5; p<0.001). reserve added power to the model (chi-square = 70.2, p<0.001). We concluded that CVFR, obtained by Doppler in LAD, MFR and reserve, obtained by dipyridamole stress RTMPE, are predictors of cardiac death and heart transplantation in patients with nonischemic DCM. However, only left atrial diameter and depressed reserve showed independent and incremental predictive value beyond that provided by current known prognostic clinical and echocardiographic factors
Частини книг з теми "Flux coronaire":
Isenberg, Gerrit, and Vladimir Ganitkevich. "Na-Ca Exchange in Isolated Coronary Myocytes: A Study Combining Voltage Clamp and Ca2+ Fluorometry." In Ion Flux in Pulmonary Vascular Control, 57–66. Boston, MA: Springer US, 1993. http://dx.doi.org/10.1007/978-1-4615-2397-0_5.
Kellner-Weibel, Ginny, Margarita de la Llera-Moya, Sandhya Sankaranarayanan, and George H. Rothblat. "In Vitro Studies and Mass Flux of Cholesterol Between Serum and Macrophages." In High Density Lipoproteins, Dyslipidemia, and Coronary Heart Disease, 83–88. New York, NY: Springer New York, 2010. http://dx.doi.org/10.1007/978-1-4419-1059-2_10.
Тези доповідей конференцій з теми "Flux coronaire":
Fayssal, Iyad, and Fadl Moukalled. "A Numerical Analysis of the Hemodynamic Functionality of Human Coronary Stenosis Under Different Physiologic Conditions and Boundary Condition Formulations." In ASME-JSME-KSME 2019 8th Joint Fluids Engineering Conference. American Society of Mechanical Engineers, 2019. http://dx.doi.org/10.1115/ajkfluids2019-4820.