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Статті в журналах з теми "Respiratory organs Diseases":

1
.Aleshkin, A. V., and M. V. Zeigarnik. "Phagotherapy of suppurative-inflammatory diseases of the respiratory organs." Infekcionnye bolezni 13, no. 3 (2015): 46–53. http://dx.doi.org/10.20953/1729-9225-2015-3-46-53.
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2
Starevskaya, S. V., M. M. Goloborodko, O. V. Berleva, S. V. Barashkova, I. Yu Melnikova, V. Yu Detkov, and V. P. Molodtsova. "CONDITION OF RESPIRATORY MUCOSA AT CHILDREN WITH RESPIRATORY DISTRESS." HERALD of North-Western State Medical University named after I.I. Mechnikov 7, no. 1 (March 2015): 88–92. http://dx.doi.org/10.17816/mechnikov20157188-92.
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42 children with the diseases of respiratory organs complicated by respiratory failure have been examined to determine the nature of inflammatory changes of a tracheobronchial tree and a condition of mucoсiliary system . At children with acute diseases of respiratory organs the neutrophillic inflammation of tracheobronchial tree mucous has been revealed more often, at children with chronic diseases of lungs has been more often noted lymphocytic eosinophilic an inflammation of tracheobronchial tree mucous , obvious thaumatropy and dystrophy of ciliate epithelium. At 10% of children primary motor disturbance of the mucoсiliary system (total immobility of cilia) has been revealed.
3
Kamaev, I. A., V. M. Levanov, E. A. Perevezentsev, and O. V. Undalova. "Morbidity of Nizhny Novgorod Region population with respiratory organs diseases." Medical almanac, no. 2 (2017): 18–21. http://dx.doi.org/10.21145/2499-9954-2017-2-18-21.
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4
Filimonov, S. N., N. I. Panev, O. Yu Korotenko, N. A. Evseeva, I. P. Danilov, and O. V. Zatsepina. "Prevalence of somatic pathology in coal mine workers with occupational respiratory diseases." Russian Journal of Occupational Health and Industrial Ecology, no. 6 (July 2019): 381–84. http://dx.doi.org/10.31089/1026-9428-2019-6-381-384.
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Introduction. Th e high level of professional and production-related pathology among workers of the coal industry, as well as the frequent development of their combined pathology with an unfavorable prognosis determine the relevance of studying the prevalence of somatic pathology in miners with occupational diseases of the respiratory system.The aim of the study was to explore the prevalence of internal organs pathology in coal industry workers with occupational lung diseases (anthracosilicosis, chronic dust bronchitis, chronic obstructive pulmonary disease).Materials and methods. 788 store miners and shaft sinkers with previously diagnosed occupational respiratory diseases (anthracosilicosis, chronic dust bronchitis, chronic obstructive pulmonary disease) and 161 miners, working for a long time in harmful labour conditions and having no occupational pathology (the control group) were examined.Results. It was revealed that the workers of the coal industry with occupational diseases of the respiratory system pathology of internal organs occurs more often than the workers of the control group, including: diseases of the cardiovascular system (hypertension, angina pectoris I and II functional classes), diseases of the digestive system (nonalcoholic fatty liver disease and chronic pancreatitis), kidney disease (chronic pyelonephritis), as well as a combination of several somatic diseases.Conclusions. In miners with occupational diseases of the respiratory system more oft en than in the control group, there is a pathology of the internal organs: diseases of the cardiovascular system, digestive organs, kidneys, as well as a combination of several somatic diseases. Th e data obtained should be considered during periodic medical examinations and medical examinations for the development of timely therapeutic and preventive and rehabilitation measures.
5
Gordeeva, M. V., M. N. Lyapin, and T. A. Kostyukova. "Means of Respiratory Organs Protection for Work with Agents of Infectious Diseases." Problems of Particularly Dangerous Infections, no. 4 (January 2020): 6–16. http://dx.doi.org/10.21055/0370-1069-2019-4-6-16.
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Chunxi, Li, Liu Haiyue, Lin Yanxia, Pan Jianbing, and Su Jin. "The Gut Microbiota and Respiratory Diseases: New Evidence." Journal of Immunology Research 2020 (July 2020): 1–12. http://dx.doi.org/10.1155/2020/2340670.
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Human body surfaces, such as the skin, intestines, and respiratory and urogenital tracts, are colonized by a large number of microorganisms, including bacteria, fungi, and viruses, with the gut being the most densely and extensively colonized organ. The microbiome plays an essential role in immune system development and tissue homeostasis. Gut microbiota dysbiosis not only modulates the immune responses of the gastrointestinal (GI) tract but also impacts the immunity of distal organs, such as the lung, further affecting lung health and respiratory diseases. Here, we review the recent evidence of the correlations and underlying mechanisms of the relationship between the gut microbiota and common respiratory diseases, including asthma, chronic obstructive pulmonary disease (COPD), cystic fibrosis (CF), lung cancer, and respiratory infection, and probiotic development as a therapeutic intervention for these diseases.
7
Radtsig, Ye Yu, N. V. Yermilova, L. V. Malygina, О. V. Bugaichuk, and Ye P. Sel’kova. "Causal Treatment of Inflammatory Diseases of ENT organs — Complications of Acute Respiratory Infection." Current Pediatrics 13, no. 6 (December 2014): 113–16. http://dx.doi.org/10.15690/vsp.v13i6.1210.
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Andreeva, N. A., T. A. Shumatova, and P. A. Motavkin. "Nitrergic neurons in respiratory organs." Bulletin of Experimental Biology and Medicine 129, no. 2 (February 2000): 190–92. http://dx.doi.org/10.1007/bf02434808.
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Koldibekova, Yu V., Marina A. Zemlyanova, A. M. Ignatova, I. V. Tikhonova, N. I. Markovich, K. V. Chetverkina, and V. M. Ukhabov. "ASSESSMENT OF THE RISK FOR HEALTH DISORDERS IN CHILDREN WHO LIVE IN A TERRITORY OF THE ZONE OF EXPOSURE TO PRODUCTION OF METALLURGICAL ALUMINUM." Hygiene and sanitation 98, no. 2 (April 2019): 135–41. http://dx.doi.org/10.18821/0016-9900-2019-98-2-135-141.
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Introduction. The atmospheric air is known to be the most contaminated on territories where industries are highly developed; non-ferrous metallurgy is one of them that make the greatest contribution into atmospheric air contamination. Data and methods. Our research objects were air samples taken on territories where there was located an enterprise producing metallurgic aluminum and our reference samples were taken on a territory where there was no such production; we also examined databases containing data on morbidity of children population, non-carcinogenic risk assessment, number of exposed population, and the results of epidemiologic examination. Results. Quality of the atmospheric air was unsatisfactory as there were concentrations of suspended substances, PM10 solid particles, nitrogen dioxide, sulfur dioxide, as well as persistence occurrence of manganese, nickel, and copper compounds resulted from activities of enterprises that produced metallurgic aluminum. All the above-mentioned admixtures cause an unacceptable risk of non-carcinogenic effects emerging in the respiratory organs. A significant contribution into hazard index is made by suspended substances, PM10 and PM2.5 particles. Fine-dispersed fraction having a complicated chemical structure (ferric oxides; aluminum, silicon, nickel, chromium, titanium, and manganese oxides), can aggravate negative impacts on the respiratory organs, exerted by the identified risk factors. The prevalence of respiratory organs diseases among children population living on an exposed territory confirms environmental risks to make increased levels of the overall and primary morbidity rate. We proved a probability of respiratory organs diseases to be depended on concentrations of the examined substances in the atmosphere, including suspended substances and fine-dispersed fractions of PM10 and PM2.5, nitrogen oxide, nitrogen dioxide, sulfur dioxide, compounds of nickel, manganese, and copper. Discussion. The detected cause-and-effect relations between a risk of increased morbidity with respiratory organs diseases in children and exposure to chemical risk factors, primarily suspended particles, PM10 and PM2.5, are also confirmed by results obtained in a number of research that concentrates on peculiarities of effects produced by the examined chemical factors on respiratory organs diseases in children. Conclusions. Children living on a territory exposed to the exposure exerted by an enterprise that produces metallurgic aluminum have a chronic aerogenic risk for respiratory organs diseases; this risk becomes apparent due to the elevated morbidity rate of respiratory organs diseases. This risk is proved to be related to impacts exerted by suspended substances and fine-dispersed fractions of PM10 and PM2.5, nitrogen oxide, nitrogen dioxide, sulfur dioxide, manganese, nickel, and copper.
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Lazic, Sava, Tamas Petrovic, Ivan Pusic, and Maja Velhner. "Most frequent calf diseases in industrial breeding." Veterinarski glasnik 58, no. 1-2 (2004): 67–76. http://dx.doi.org/10.2298/vetgl0402067l.
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It is possible to conduct an analysis of the incidence of viral diseases in calves if these diseases are divided into two basic groups. One group comprises diseases of respiratory organs which are manifested by symptoms of a respiratory syndrome, and the second group comprises diseases of digestive tract organs in the form of a gastrointestinal syndrome. It is considered that viruses have the dominant role in the complex etiology of the respiratory syndrome, primarily the IBR virus or the Bovine Herpes Virus-1 (BHV-1), followed by the parainfluenza 3 virus (RSV), the Bovine Viral Diahrrea Virus (BVDV), the bovine Respiratory Syncytial Virus (RSV), but also other viruses, such as adenoviruses, rhinoviruses, coronaviruses, can also influence the appearance of the respiratory syndrome. The respiratory syndrome is rarely caused by a single viral agent, but most frequently by mixed viruses, but also by bacterial infections. Mixed viral infections often have a lethal outcome. Investigations of the etiology of the gastrointestinal syndrome so far indicate that, in addition to bacteria, viruses can also be a significant etiological factor. Rotaviruses, coronaviruses, adenoviruses parvoviruses, herpesviruses (the IBR virus), pestiviruses (BVDV), can be the causes of a gastrointestinal syndrome. It is believed that viruses can be the cause in about 10% cases in the ethiopathogenesis of this syndrome. The paper describes the etiopathogenesis of calf diseases of viral etiology which are most often found in the local conditions of industrial breeding of calves.

Дисертації з теми "Respiratory organs Diseases":

1
Zhao, Hanjun, and 赵旵军. "A study of antiviral peptides with broad activity against respiratory viruses." PG_Thesis, The University of Hong Kong (Pokfulam, Hong Kong), 2010. http://hdl.handle.net/10722/205838.
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A safe, potent and broad-spectrum antiviral is urgently needed to combat emerging viral respiratory diseases such as avian influenza H5N1 and H7N9, severe acute respiratory syndrome (SARS) and Middle East respiratory syndrome (MERS). Previous studies carried out by PhD students in our lab found that mouse β-defenisn 4 (mBD4) shows highly antiviral activity in vitro. However, the recombinant mBD4 (rmBD4) expressed by E.coli is limited to very small scale of production and is very expensive. Thus, in this study, we firstly screened 16 short peptides derived from mBD4 and other mouse and human β-defensins for identifying their antiviral effects. One short peptide P9 (30 amino acids), derived from mBD4, exhibited potent and broad-spectrum antiviral effects against multiple respiratory viruses, including influenza A viruses H1N1, H3N2, H5N1, H7N7 and H7N9, SARS coronavirus (SARS-CoV)and MERS coronavirus (MERS-CoV). This P9 showed very high selectivity index (970), which was higher than that of the full-length peptide of synthetic mBD4 (smBD4) and rmBD4 in vitro. Secondly, the prophylactic and therapeutic effects of P9 against the infection of H1N1 virus were further detected in animal model. The survival rate of P9-pretreated mice challenged by lethal dose of H1N1 virus was 100%. The therapeutic effects of P9 protecting mice from lethal challenge of H1N1 virus were also statistically significant. The survival rate of mice could reach up to 67% by intranasal inoculation and 56% by intraperitoneal injection, respectively. To investigate the antiviral mechanism, we firstly elucidated that P9 could inhibit viral infection but not viral replication or release. Secondly, we detected whether P9 inhibited viral infection by binding to the surface of target cells or viral particles. The results showed that P9 only bound to viral particles but not to the cell surface. It was further identified that P9 bound to viral surface glycoprotein HA but not NA. Thirdly, we demonstrated that P9 did not inhibit virus binding to its receptor and block the virus entry into cells by endocytosis. Instead, P9 inhibited the acidification in late endosomes and thusP9 blocked virus-membrane fusion and subsequent viral disassembly and viral RNA release. Finally, we elucidated that the antiviral activity of P9 was attributed to its high binding affinity to viral HA and the abundance of basic amino acids in its composition. In this study, we have demonstrated that a short peptide P9, which is derived from mBD4, showed potent antiviral activity against multiple respiratory viruses. This peptide can be developed to a new promising prophylactic and therapeutic agent with broad-spectrum antiviral activity and low possibility to cause drug resistance. Moreover, this study has also revealed a novel antiviral mechanism for P9 and paved a path for the development of new antiviral agents with broad-spectrum antiviral activity against emerging respiratory viruses, such as avian influenza H5N1 and H7N9, as well as SARS-CoV and MERS-CoV.
published_or_final_version
Microbiology
Doctoral
Doctor of Philosophy
2
Yip, Ming-shum, and 葉名琛. "Immune responses of human respiratory epithelial cells to respiratory syncytial virus and human metapneumovirus." PG_Thesis, The University of Hong Kong (Pokfulam, Hong Kong), 2007. http://hub.hku.hk/bib/B3955725X.
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3
Chaturvedi, Rakesh K. "Reasoning about therapeutic and patient management plans in respiratory medicine by physicians & medical students." Electronic Thesis or Dissertation, McGill University, 1994. http://digitool.Library.McGill.CA:80/R/?func=dbin-jump-full&object_id=41562.
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Recently, there has been extensive research in the area of diagnostic expertise. The model of diagnostic reasoning and clinical expertise has been well documented (Patel et al., in press). This study attempts to extend this research in order to include therapeutic reasoning. Using the expert-novice paradigm, this study attempts to investigate the use of knowledge, specifically, both biomedical and clinical sciences, and the directionality of reasoning during decision making about patient management and therapeutic planning in respiratory medicine.
Subjects at four levels of expertise were given two clinical problems with the diagnosis and asked (a) to provide therapeutic plans, and (b) describe the underlying pathophysiological explanations of the diseases. Think-aloud protocols were audio-taped and analyzed using methods of protocol analysis. The results showed that the use of basic medical sciences increased as a function of expertise in the procedure-oriented decision-making tasks. The novices generated rule-based prototypical textbook descriptions based on the clinical information, and the diagnosis given in the task. In contrast, the experts' therapeutic responses showed a predominance of causal-level inferences, reflecting more backward-directed inferences than novices. Although both the novices and experts generated forward-directed inferences, the novices were unable to provide accurate and adequate explanations for their decisions. Finally, the pathophysiological explanations of the disease were generated from a different knowledge source than that used to develop therapeutic decisions.
The implications of these findings for development of theory of expertise and for education in the medical domain are discussed.
4
Paudyal, Priyamvada. "Respiratory symptoms and lung function in relation to cotton dust and endotoxin exposure in textile workers in Nepal." Electronic Thesis or Dissertation, University of Aberdeen, 2011. http://digitool.abdn.ac.uk:80/webclient/DeliveryManager?pid=166944.
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Background: Cotton workers are highly exposed to organic dust. Inhalation of cotton based particulate has been associated with various respiratory symptoms and impaired lung function. This study investigates the respiratory health profile of textile mill workers in Nepal in relation to dust and endotoxin exposure. Methods: This study was conducted in four sectors (garment, carpet, weaving and recycling) of the textile industry in Kathmandu, Nepal. A total of 938 individuals completed a health questionnaire and performed spirometry. A subset of 384 workers performed cross-shift spirometry. Personal exposure to inhalable dust and airborne endotoxin was measured during a full-shift for a 114 workers. Results: Geometric mean concentrations of personal exposure to cotton dust and endotoxin were 0.81 mg/m3 and 2160 EU/m3 respectively. Overall prevalence of persistent cough, persistent phlegm, wheeze, breathlessness and chest tightness were 8.5%, 12.5%, 3.2%, 6.5%and 3.6% respectively. Symptoms were most common among the recyclers and less in the garment sector. Exposure to inhalable dust significantly predicted the symptoms of persistent cough and chest tightness. Significant cross-shift reduction in FEV1, FVC, and FEF25_75 were measured in the textile workers (p<0.001 for all); reductions being greater in the recyclers (-143 ml) and smallest in the garment workers (-38 ml) (p=0.012). Cross-shift reduction in FEV1 was significantly predicated by exposure to inhalable dust. Exposure to endotoxin did not correlate with any of the respiratory symptoms nor to lung function. Conclusion: The measured association between exposure to inhalable dust and reporting of respiratory symptoms and lung function suggests that despite high levels of endotoxin exposures, inhalable dust is the driver for these effects and attention should turn to what might be the toxic component in this dust other than endotoxin.
5
岑海音 and Hoi-yum Irma Shum. "Interactions of pseudomonas aeruginosa toxins with respiratory mucosa in vitro." PG_Thesis, The University of Hong Kong (Pokfulam, Hong Kong), 2003. http://hub.hku.hk/bib/B31244725.
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Wai, Chi-wan, and 衛至韻. "Development of shell vial culture assay for the rapid diagnosis of respiratory viruses using the human colorectal adenocarcinoma (CaCo2) cells." PG_Thesis, The University of Hong Kong (Pokfulam, Hong Kong), 2001. http://hdl.handle.net/10722/193551.
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Background: Respiratory diseases are common worldwide, which are caused by various respiratory viruses. As symptoms caused by these viruses are similar, laboratory diagnosis is essential to distinguish the virus. Conventionally, respiratory viruses are isolated by cell culture with a panel of cell lines. However, handling of several cell lines is labour intensive, and the turnaround time of conventional culture is long. In previous study, the use of human colon adeno-carcinoma (Caco-2) in conventional culture was investigated. The study has proven that Caco-2 is generally susceptible to the eight common respiratory viruses, i.e. Adenovirus, Influenza A and B, Respiratory Syncytial virus, Parainfluenza virus 1, 2,3 and 4. As turnaround time of conventional culture is long; therefore, in this study, rapid shell vial culture using Caco-2 cells were evaluated. Moreover, the application of Caco-2 shell vial culture on recovering human metapneumovirus (hMPV) was also investigated. Materials and methods: This study consisted of four stages. First, recovery of viruses by conventional culture and shell vial culture of Caco-2 were compared. Specimens were added to conventional culture and shell vial simultaneously. For conventional culture, formation of CPE was examined daily and IF staining was performed when CPE was indicated; meanwhile, shell vial culture were incubated for seven days and stained with IF to detect infected cells. In stage two, the effect of incubating shell vial culture in rolling drum was investigated. Shell vials inoculated with the same specimen in duplicate were incubated in rolling drum and without rolling drum simultaneously. IF staining was performed in day 2, and results were obtained. For those which are IF negative in day 2, second shell vial was further incubated to seven days before harvest. In the next stage, a large batch of samples was used to evaluate on the use of Caco-2 shell vial culture in day 2 and day 7. Lastly, Caco-2 shell vial and conventional culture and LLC-MK2 conventional culture were tested for isolation of hMPV. Results: Compared to Caco-2 conventional culture, recovery rate of shell vial culture was elevated slightly. When experimenting on the effect of incubation in rolling drum, results showed that recovery rate was raised in shell vial with rolling drum in day 2, moreover, the percentage of positive cells were increased significantly (p value < 0.05). Furthermore, in the evaluation of Caco-2 shell vial in day 2 and day 7, 75% of samples were isolated in day 2 while 85% were recovered in day 7. Lastly, in the investigation on recovery of hMPV, 53%, 42% and 17% hMPV positive cases were isolated by Caco-2 shell vial, Caco-2 conventional culture and LLC-MK2 conventional culture respectively. Conclusion: First, although recovery rate by shell vial and conventional culture were similar, turnaround time was reduced from a week to a few days by shell vial culture. Therefore, Caco-2 shell vial culture is a more efficient than Caco-2 conventional culture in isolating respiratory viruses. The study also showed that incubation of shell vial in rolling drum able to increase the number of positive cells. Furthermore, in this study, Caco-2 cells were also shown to be more efficient in isolating hMPV when compare to LLC-MK2 cells.
published_or_final_version
Microbiology
Master
Master of Medical Sciences
7
胡慧明 and Huie-ming Hou. "Long-term study of sleep apnoea patients treated with MAD." PG_Thesis, The University of Hong Kong (Pokfulam, Hong Kong), 2005. http://hub.hku.hk/bib/B45012222.
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Ratnawati, Ratnawati Prince of Wale Hospital Clinical School UNSW. "Exhaled nitric oxide in asthmatic airway inflammation." Awarded by:University of New South Wales. Prince of Wale Hospital Clinical School, 2006. http://handle.unsw.edu.au/1959.4/25729.
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Measuring the level of exhaled NO (eNO) in the breath is a new method to monitor airway inflammation in asthma and may have a role in the management of asthma. The hypotheses were that eNO will reflect the degree of inflammation in chronic asthma, and will indicate how anti- inflammatory therapy should be altered to improve asthma control. Three studies were performed to test the hypotheses. A cross sectional study was performed to define the normal range of eNO and to compare this range with those who have asthma or atopy. The second study was observational, to compare the level of eNO during and after an exacerbation of asthma. The third study was an interventional study to evaluate eNO in management of paediatric asthma. In this latter study the level of eNO was measured to monitor airway inflammation in asthmatic children with the intention of adjusting antiinflammatory drugs (inhaled glucocorticosteroids) according to the level of eNO. These studies have shown that the mean level of eNO was significantly higher in asthmatic compared with normal subjects, but not significantly different when compared with atopic non-asthmatic subjects. eNO was correlated with the number of positive skin prick tests in atopic subjects whether asthmatic or nonasthmatic. The eNO level was increased during acute exacerbations of asthma and decreased after two weeks with therapy of GCS. In a pilot study eNO appeared to be superior to FEV1 in adjusting the dose of iGCS to control asthmatic children, but this needs to be confirmed with a larger sample size. Another non-invasive method to detect inflammatory markers is the technique of exhaled breath condensate (EBC). Although NO is degraded to NOx, it was found that eNO had no significant correlation with EBC NOx but had a significant correlation with pH. Hypertonic saline challenge, an artificial model of an asthmatic exacerbation was associated with an increase in EBC volume and the release of histamine, implicating mast cell activation. These novel findings suggest that non-invasive markers can be used both for clinical and mechanistic proposes.
9
Lee, Kathleen F. "Clinical competencies required for graduates of an entry-level associate degree respiratory care program to practice competently." Virtual Press, 2002. http://liblink.bsu.edu/uhtbin/catkey/1247887.
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The purpose of this study was to identify the complete scope, cognitive, affective and psychomotor, of clinical competencies required for the successful practice by entry-level associate degree respiratory care graduates entering the workforce. The study was conducted using a Delphi technique. American Association for Respiratory Care House of Delegates nominated a panel of experts to participate in the study. A total of 55 individuals were nominated and 21 agree to participate in the study. The individuals represented both college-based educators and hospital managers and educators. The initial questionnaire requested that each panel member list those cognitive, affective and psychomotor clinical competencies that they believed to be required for successful entrylevel practice. The responses from the first round were grouped into like categories and returned to the panel members to be rated on a four-point scale during the second and third rounds of the study. Consensus was achieved with the third round resulting in a final list of 26 cognitive, 20 affective and 28 psychomotor clinical competencies. The final list of competencies includes the most basic cognitive and psychomotor skills of the profession and did not include many of the areas listed by the National Board for Respiratory Care exam matrix. The study also lists 20 affective attributes as essential for successful practice. There are no current standardized methods in use to evaluate affective skills. Research will need to continue to clarify the skills needed for competent and successful entry-level practice.
Department of Educational Studies
10
Anua, Siti Marwanis. "Workplace and home exposure to respiratory sensitisers : examining the work to home pathway." Electronic Thesis or Dissertation, University of Aberdeen, 2012. http://digitool.abdn.ac.uk:80/webclient/DeliveryManager?pid=203876.
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Background: Contamination of the skin and clothing may lead to workers inadvertently bringing harmful materials home and exposing family members, so called para-occupational exposure. This study investigates whether workplace contamination with respiratory sensitisers such as laboratory animal allergens, flour, isocyanates and enzymes is transported from work to the home environment. Methods: 3 laboratory animal facilities, 92 bakeries, 47 car body workshops, and 2 hospitals in the Grampian region of Scotland were invited to take part in a series of linked studies to increase understanding of the ‘take-home' pathway. Control subjects were recruited from staff and students at the University of Aberdeen. Take-home exposure assessment was carried out using two techniques: surface wipe sampling and vacuum sampling in workplaces, cars and homes. Samples were also collected in the homes of control subjects. Samples from bakers were analysed for total protein, wheat flour antigen (WFA) and fungal alpha amylase (FAA) while samples from laboratory animal workers were analysed for mouse urinary protein (Mus m 1). Enzyme cleaning agents were analysed for subtilisin proteolytic activity. Similar methods using SWYPE™ aliphatic pads for isocyanate contamination assessment were conducted among car body repairers. The pads were scanned and images of SWYPE™ pads were used to estimate contamination against the quantitative assay MDHS 25/3. All analyses were done by the Health and Safety Laboratory (HSL) apart from the SWYPE™ RGB tests and gravimetric measurements. Results: A total of 13 laboratory animal workers in 3 animal facilities, 38 bakers in 5 bakeries, 13 car painters in 5 car body workshops and 20 control subjects participated in the study. Two hospitals were surveyed for enzyme exposures and 3 endoscope cleaning technicians were monitored. Evidence of take-home exposure was found for bakery workers, with potential contamination that could lead to home exposure in the car body repair and hospital workers. Higher levels of Mus m 1 contamination were detected on house door handles of non-exposed controls compared to the exposed laboratory workers (0.62 vs. 0.1 ng/wipe, p<0.001) probably due to exposure variability, might be because exposed laboratory workers being involved in a job that requires more hand washing than the general population, or suggesting widespread environmental contamination with this allergen, and these making it impossible to determine if work-home pathway exists for these workers. There was detectable WFA and FAA found on the hands, forehead, shoes, cars and homes of bakers. Compared to controls, bakers had higher median levels of WFA and FAA in house vacuum samples; the difference was statistically significant for WFA/total protein (516x10-6 vs. 164x10-6, p=0.031), FAA/total protein ratios (1.45x10-6 vs. 0.04x10-6, p<0.001) and FAA loading (1.2 pg/cm2 vs. 0.1 pg/cm2, p<0.001). Among car painters, SWYPE™ colorimetric colour changes score showed three positive SWYPE™ colour changes on skin, and three positive results on shoes of car body workshop workers. However quantitative colour analysis of the SWYPE™ pads proved ineffective for field measurements. Hand wipes of hospital workers during mid-shift and post-shift showed evidence of proteolytic activity, indicating possible spread of contamination from hands, unsatisfactory hygiene practices and the potential for take-home contamination of enzyme. Presence of contamination on footwear indicated that possible transfer of enzyme to other places including homes may occur. Conclusion: These data demonstrate the existence of pathways for take-home exposure of allergens among bakers via skin and clothing from workplaces to cars and workers' homes. The take-home pathway for laboratory animal allergens and isocyanates was not demonstrated and further investigation should be performed for enzyme cleaning agents used in healthcare settings by monitoring dermal take-home exposure with comparison to controls. Further work is needed to ascertain how widespread the take-home of respiratory sensitisers may be and the possible implications to the health of workers' families and the wider community. If parental occupation can lead to take-home exposure to respiratory asthmagens, and consequently to childhood asthma, then this represents a potentially modifiable risk factor for these cases of para-occupational asthma. There is a need for greater understanding of the take-home pathway of exposure to asthmagens and sensitisers and for a programme of education and control measures to limit the transfer of such material from the workplace to the home and wider community.

Книги з теми "Respiratory organs Diseases":

1
Johnson, N. Mcl. Respiratory medicine. 2nd ed. Oxford: Blackwell Scientific Publicatrions, 1990.
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2
Rush, Bonnie. Equine respiratory diseases. Oxford, UK: Blackwell Science, 2004.
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3
National, Heart Lung and Blood Institute Division of Lung Diseases. Respiratory failure. [Bethesda, Md.?]: The Division, 1995.
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4
Gray, Peter. Respiratory disease. London: Allen, 1994.
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5
Wagener, Luke. Respiratory diseases: Causes, treatment, and prevention. Hauppauge, N.Y: Nova Science Publishers, 2011.
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6
Boyda, Ellen K. Respiratory problems. Oradell, N.J: Medical Economics Books, 1985.
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7
Westra, Bonnie. Respiratory problems. Springhouse, Pa: Springhouse Corp., 1987.
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8
Cade, J. F. Essentials of respiratory medicine. Oxford: Blackwell Scientific, 1988.
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9
Johnson, Norman McI. Respiratory medicine. Oxford: Blackwell Scientific, 1986.
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10
Johnson, Norman McI. Respiratory medicine. 2nd ed. Oxford: Blackwell Scientific, 1990.
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Частини книг з теми "Respiratory organs Diseases":

1
Marchuk, Guri I. "Viral And Bacterial Infections of Respiratory Organs." In Mathematical Modelling of Immune Response in Infectious Diseases, 269–89. Dordrecht: Springer Netherlands, 1997. http://dx.doi.org/10.1007/978-94-015-8798-3_9.
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2
Warburg, Michael R. "Respiratory Organs and Respiration." In Evolutionary Biology of Land Isopods, 25–31. Berlin, Heidelberg: Springer Berlin Heidelberg, 1993. http://dx.doi.org/10.1007/978-3-662-21889-1_4.
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3
Jarolim, Dala R. "Respiratory Diseases." In Oklahoma Notes, 27–61. New York, NY: Springer New York, 1996. http://dx.doi.org/10.1007/978-1-4612-2370-2_2.
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4
Tomsic, Jaclyn A., Alireza Ashrafi, Ray English, and Kiara Brown. "Respiratory Diseases." In Oral Board Review for Oral and Maxillofacial Surgery, 371–81. Cham: Springer International Publishing, 2020. http://dx.doi.org/10.1007/978-3-030-48880-2_14.
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5
Schmeltzer, Linda E. "Respiratory Diseases." In Nursing the Feline Patient, 204–7. Ames, Iowa, USA: John Wiley & Sons, Inc, 2016. http://dx.doi.org/10.1002/9781119264910.ch28.
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6
Georgiev, Vassil St. "Respiratory Diseases." In National Institute of Allergy and Infectious Diseases, NIH, 67–74. Totowa, NJ: Humana Press, 2009. http://dx.doi.org/10.1007/978-1-60327-297-1_10.
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Lancaster, H. O. "Respiratory Diseases." In Expectations of Life, 266–71. New York, NY: Springer New York, 1990. http://dx.doi.org/10.1007/978-1-4612-1003-0_26.
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8
Jarolim, Dala R. "Respiratory Diseases." In Oklahoma Notes, 30–61. New York, NY: Springer US, 1993. http://dx.doi.org/10.1007/978-1-4684-0458-6_2.
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9
Jariwalla, G. "Background." In Respiratory Diseases, 1–8. Dordrecht: Springer Netherlands, 1985. http://dx.doi.org/10.1007/978-94-009-4880-8_1.
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10
Jariwalla, G. "Sarcoidosis." In Respiratory Diseases, 162–67. Dordrecht: Springer Netherlands, 1985. http://dx.doi.org/10.1007/978-94-009-4880-8_10.
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Тези доповідей конференцій з теми "Respiratory organs Diseases":

1
Крумкачева А.Ю., Крумкачева А. Ю. "State of respiratory system and peripheral musculature in patients with dust diseases of respiratory organs." In The second international youth Forum "OCCUPATION AND HEALTH". PT "ARIAL", 2018. http://dx.doi.org/10.31089/978-5-907032-51-4-2018-1-141-148.
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2
Savushkina, Olga, Alexander Cherniak, Marina Kameneva, Evgenij Kryukov, and Andrey Zaytsev. "Impulse oscillometry in restrictive respiratory diseases." In ERS International Congress 2018 abstracts. European Respiratory Society, 2018. http://dx.doi.org/10.1183/13993003.congress-2018.pa3394.
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3
Teodoro, M. F., J. N. Garcia, L. M. Coelho, and M. G. Carvalho. "Relating air pollution and respiratory diseases occurrences." In PROCEEDINGS OF THE INTERNATIONAL CONFERENCE ON NUMERICAL ANALYSIS AND APPLIED MATHEMATICS 2014 (ICNAAM-2014). AIP Publishing LLC, 2015. http://dx.doi.org/10.1063/1.4912425.
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4
Muangon, Weenawadee. "A fuzzy information retrieval for respiratory diseases." In ICIET '18: 2018 6th International Conference on Information and Education Technology. New York, NY, USA: ACM, 2018. http://dx.doi.org/10.1145/3178158.3178195.
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5
Fukuda, Yosuke, Shintaro Suzuki, Tetsuya Homma, and Hironori Sagara. "High burden ofA. fumigatusamong chronic respiratory diseases." In ERS International Congress 2016 abstracts. European Respiratory Society, 2016. http://dx.doi.org/10.1183/13993003.congress-2016.pa5022.
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6
Castro-Rodriguez, Jose A., Leticia Jakubson, Oslando Padilla, Doris Gallegos, Rodrigo Fasce, Pablo Beltrand, Ignacio Sanchez, and Cecilia Perret. "Many Respiratory Viruses May Trigger Meningococcal Diseases." In American Thoracic Society 2011 International Conference, May 13-18, 2011 • Denver Colorado. American Thoracic Society, 2011. http://dx.doi.org/10.1164/ajrccm-conference.2011.183.1_meetingabstracts.a4928.
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Mayorga, P., C. Druzgalski, and J. Vidales. "Quantitative models for assessment of respiratory diseases." In 2010 Pan American Health Care Exchanges (PAHCE 2010). IEEE, 2010. http://dx.doi.org/10.1109/pahce.2010.5474607.
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Polyansky, I. Y., F. B. Hrynchuk, S. P. Brodovsky, Bohdan M. Nitsovich, and R. M. Besaha. "Photoluminescent diagnostics of inflammatory-destructive diseases of the abdominal cavity organs." In SPIE Proceedings, edited by Oleg V. Angelsky. SPIE, 2004. http://dx.doi.org/10.1117/12.560069.
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9
Verma, Ajay Kumar, Abhaya Gupta, Arpita Singh, Surya Kant, Ram Awadh Singh Kushwaha, Umesh Pratap Verma, Hemant Kumar, and Anuj Pandey. "Pattern of Periodontal diseases in Hospitalized Patients with Non-Tubercular Respiratory diseases." In ERS International Congress 2019 abstracts. European Respiratory Society, 2019. http://dx.doi.org/10.1183/13993003.congress-2019.pa2794.
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10
Zanolin, M. Elisabetta, Liliya Chamitava, Paolo Degan, Andrea Pasini, Anna Fratta-Pasini, Morena Nicolis, Mario Olivieri, et al. "Biomarkers of oxidative stress in chronic respiratory diseases." In Annual Congress 2015. European Respiratory Society, 2015. http://dx.doi.org/10.1183/13993003.congress-2015.pa1108.
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Звіти організацій з теми "Respiratory organs Diseases":

1
Levin, Myron, Gordon Meiklejohn, and Theodore C. Eickhoff. Prevention of Influenza and Other Respiratory Diseases. Fort Belvoir, VA: Defense Technical Information Center, June 1991. http://dx.doi.org/10.21236/ada239657.
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2
Reecy, James M., and Matthew Schneider. Heritability of Genetic Resistance to Bovine Respiratory Diseases. Ames: Iowa State University, Digital Repository, 2007. http://dx.doi.org/10.31274/farmprogressreports-180814-638.
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3
Meiklejohn, Gordon, and Theodore C. Eickhoff. Prevention of Influenza and Other Respiratory Diseases - Laboratory Studies. Fort Belvoir, VA: Defense Technical Information Center, April 1989. http://dx.doi.org/10.21236/ada208818.
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4
Wise, Kiersten, Carl Bradley, Loren Giesler, Bill Johnson, Travis Legleiter, Mark Licht, Daren Mueller, et al. Soybean Seedling Diseases. United States: Crop Protection Netework, June 2015. http://dx.doi.org/10.31274/cpn-20190620-023.
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5
Anthony Atala, M. D. Regeneration of Tissues and Organs Using Autologous Cells. Office of Scientific and Technical Information (OSTI), October 2012. http://dx.doi.org/10.2172/1052850.
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6
Anthony Atala. Regeneration of Tissues and Organs Using Autologous Cells. Office of Scientific and Technical Information (OSTI), April 2010. http://dx.doi.org/10.2172/977185.
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7
Uyehara, Catherine, and Scott Stewart. Broadband Respiratory Virus Surveillance. Fort Belvoir, VA: Defense Technical Information Center, October 2011. http://dx.doi.org/10.21236/ada555802.
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8
Westergaard, Jørgen M. Wildlife and Infectious Animal Diseases. Nordic Council of Ministers, March 2014. http://dx.doi.org/10.6027/tn2014-508.
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M. Westergaard, Jørgen. Contingency Planning for Animal Diseases. Nordic Council of Ministers, March 2014. http://dx.doi.org/10.6027/tn2014-509.
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10
Philipson, Tomas. Economic Epidemiology and Infectious Diseases. Cambridge, MA: National Bureau of Economic Research, March 1999. http://dx.doi.org/10.3386/w7037.
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