Thematische Bibliographien / Alterations of transcription factors

Inhaltsverzeichnis

  1. Zeitschriftenartikel
  2. Dissertationen
  3. Bücher
  4. Buchteile
  5. Konferenzberichte
  6. Berichte der Organisationen

Auswahl der wissenschaftlichen Literatur zum Thema „Alterations of transcription factors“

Autor: Grafiati
Veröffentlicht am 12. September 2023
Zuletzt aktualisiert am 31. Juli 2025

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Zeitschriftenartikel zum Thema "Alterations of transcription factors"

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Tang, Jinglong, and Masaya Baba. "MiT/TFE Family Renal Cell Carcinoma." Genes 14, no. 1 (2023): 151. http://dx.doi.org/10.3390/genes14010151.

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The microphthalmia-associated transcription factor/transcription factor E (MiT/TFE) family of transcription factors are evolutionarily conserved, basic helix–loop–helix leucine zipper (bHLH-Zip) transcription factors, consisting of MITF, TFEB, TFE3, and TFEC. MiT/TFE proteins, with the exception of TFEC, are involved in the development of renal cell carcinoma (RCC). Most of the MiT/TFE transcription factor alterations seen in sporadic RCC cases of MiT family translocation renal cell carcinoma (tRCC) are chimeric proteins generated by chromosomal rearrangements. These chimeric MiT/TFE proteins
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Lee, J. S., R. H. See, T. Deng, and Y. Shi. "Adenovirus E1A downregulates cJun- and JunB-mediated transcription by targeting their coactivator p300." Molecular and Cellular Biology 16, no. 8 (1996): 4312–26. http://dx.doi.org/10.1128/mcb.16.8.4312.

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Transcription factors and cofactors play critical roles in cell growth and differentiation. Alterations of their activities either through genetic mutations or by viral oncoproteins often result in aberrant cell growth and tumorigenesis. The transcriptional cofactor p300 has recently been shown to be complexed with transcription factors YY1 and CREB. Adenovirus E1A oncoproteins target these transcription complexes via physical interactions with p300, resulting in alterations of transcription mediated by these transcription factors. Here we show that p300 is also critical for repression by E1A
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Wilson, Hannah E., David A. Stanton, Stephanie Rellick, Werner Geldenhuys, and Emidio E. Pistilli. "Breast cancer-associated skeletal muscle mitochondrial dysfunction and lipid accumulation is reversed by PPARG." American Journal of Physiology-Cell Physiology 320, no. 4 (2021): C577—C590. http://dx.doi.org/10.1152/ajpcell.00264.2020.

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The peroxisome proliferator-activated receptors (PPARs) have been previously implicated in the pathophysiology of skeletal muscle dysfunction in women with breast cancer (BC) and animal models of BC. This study investigated alterations induced in skeletal muscle by BC-derived factors in an in vitro conditioned media (CM) system and tested the hypothesis that BC cells secrete a factor that represses PPAR-γ (PPARG) expression and its transcriptional activity, leading to downregulation of PPARG target genes involved in mitochondrial function and other metabolic pathways. We found that BC-derived
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Orzechowska-Licari, Emilia J., Joseph F. LaComb, Aisharja Mojumdar, and Agnieszka B. Bialkowska. "SP and KLF Transcription Factors in Cancer Metabolism." International Journal of Molecular Sciences 23, no. 17 (2022): 9956. http://dx.doi.org/10.3390/ijms23179956.

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Tumor development and progression depend on reprogramming of signaling pathways that regulate cell metabolism. Alterations to various metabolic pathways such as glycolysis, oxidative phosphorylation, lipid metabolism, and hexosamine biosynthesis pathway are crucial to sustain increased redox, bioenergetic, and biosynthesis demands of a tumor cell. Transcription factors (oncogenes and tumor suppressors) play crucial roles in modulating these alterations, and their functions are tethered to major metabolic pathways under homeostatic conditions and disease initiation and advancement. Specificity
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Pazhani, Jayanthi, Vishnu Priya Veeraraghavan, and Selvaraj Jayaraman. "Transcription factors: a potential therapeutic target in head and neck squamous cell carcinoma." Epigenomics 15, no. 2 (2023): 57–60. http://dx.doi.org/10.2217/epi-2023-0046.

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Squamous cell carcinoma is the most common histopathological type of head and neck cancer; it often spreads to and involves the cervical lymph nodes. The tumorigenesis of head and neck squamous cell carcinoma (HNSCC) is a multistep process mediated by various transcription factors involved in progression and metastasis. Alterations in transcription factors such as FOSL1, YY1, FOXD1 and NF-κB have been associated with increased cell proliferation, cell migration and poor survival rates in patients with HNSCC. Stimulation of the NF-κB pathway results in transcriptional activation of other target
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Koch, Marilin, Stefan Czemmel, Felix Lennartz, et al. "CSIG-15. INHIBITION OF THE bHLH TRANSCRIPTIONAL NETWORKS BY A MUTATED E47 PROTEIN LEADS TO A STRONG ANTI-GLIOMA ACTIVITY IN VITRO AND IN VIVO." Neuro-Oncology 21, Supplement_6 (2019): vi47. http://dx.doi.org/10.1093/neuonc/noz175.185.

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Abstract OBJECTIVE The transcription factor E47 heterodimerizes with helix-loop-helix (HLH) and basic helix-loop-helix transcription (bHLH) factors like ID-1 and Olig2 that are overexpressed in glioblastoma. A dominant-negative variant of the E47 (dnE47) lacking the nuclear translocation signal, leads to cytoplasmatic sequestration of HLH and bHLH transcription factors. Here, we investigated combinations of dnE47-mediated inhibition of the bHLH transcriptional network with temozolomide and irradiation and explored the underlying molecular mechanisms. METHODS Long-term and stem cell glioma line
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Zhu, Qian, Xavier Tekpli, Olga G. Troyanskaya, and Vessela N. Kristensen. "Subtype-specific transcriptional regulators in breast tumors subjected to genetic and epigenetic alterations." Bioinformatics 36, no. 4 (2019): 994–99. http://dx.doi.org/10.1093/bioinformatics/btz709.

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Abstract Motivation Breast cancer consists of multiple distinct tumor subtypes, and results from epigenetic and genetic aberrations that give rise to distinct transcriptional profiles. Despite previous efforts to understand transcriptional deregulation through transcription factor networks, the transcriptional mechanisms leading to subtypes of the disease remain poorly understood. Results We used a sophisticated computational search of thousands of expression datasets to define extended signatures of distinct breast cancer subtypes. Using ENCODE ChIP-seq data of surrogate cell lines and motif
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Goodson, Michael, Brian A. Jonas, and Martin A. Privalsky. "Corepressors: Custom Tailoring and Alterations While you Wait." Nuclear Receptor Signaling 3, no. 1 (2005): nrs.03003. http://dx.doi.org/10.1621/nrs.03003.

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A diverse cadre of metazoan transcription factors mediate repression by recruiting protein complexes containing the SMRT (silencing mediator of retinoid and thyroid hormone receptor) or N-CoR (nuclear receptor corepressor) corepressors. SMRT and N-CoR nucleate the assembly of still larger corepressor complexes that perform the specific molecular incantations necessary to confer transcriptional repression. Although SMRT and N-CoR are paralogs and possess similar molecular architectures and mechanistic strategies, they nonetheless exhibit distinct molecular and biological properties. It is now c
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Chen, Shali, Biao Feng, Biju George, Rana Chakrabarti, Megan Chen, and Subrata Chakrabarti. "Transcriptional coactivator p300 regulates glucose-induced gene expression in endothelial cells." American Journal of Physiology-Endocrinology and Metabolism 298, no. 1 (2010): E127—E137. http://dx.doi.org/10.1152/ajpendo.00432.2009.

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Sustained hyperglycemia in diabetes causes alteration of a large number of transcription factors and mRNA transcripts, leading to tissue damage. We investigated whether p300, a transcriptional coactivator with histone acetyl transferase activity, regulates glucose-induced activation of transcription factors and subsequent upregulation of vasoactive factors and extracellular matrix (ECM) proteins in human umbilical vein endothelial cells (HUVECs). HUVECs were incubated in varied glucose concentrations and were studied after p300 small interfering RNA (siRNA) transfection, p300 overexpression, o
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Zhang, Dawn X., and Christopher K. Glass. "Towards an understanding of cell-specific functions of signal-dependent transcription factors." Journal of Molecular Endocrinology 51, no. 3 (2013): T37—T50. http://dx.doi.org/10.1530/jme-13-0216.

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The ability to regulate gene expression in a cell-specific manner is a feature of many broadly expressed signal-dependent transcription factors (SDTFs), including nuclear hormone receptors and transcription factors that are activated by cell surface receptors for extracellular signals. As the most plastic cells of the hematopoietic system, macrophages are responsive to a wide spectrum of regulatory molecules and provide a robust model system for investigation of the basis for cell-specific transcriptional responses at a genome-wide level. Here, focusing on recent studies in macrophages, we rev
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Dissertationen zum Thema "Alterations of transcription factors"

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Morey, Ramonell Lluís. "Chromatin alterations imposed by the oncogenic transcription factor PML-RAR." Doctoral thesis, Universitat Pompeu Fabra, 2008. http://hdl.handle.net/10803/7138.

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En mamíferos, así como en plantas, mutaciones en AND helicasas/ATPasas del la família SNF2, no solo afectan a la estructura de la cromatina, sino que también afectan al patrón global de la metilación del ADN. Sugiriendo una relación funcional entre la estructura de la cromatina y la epigenética. El complejo NuRD, el cual posee una ATPasa de la familía SNF2, está relacionado con la represión de la transcripción y en el remodelamiento de la cromatina. Nuestro laboratorio demostró que la proteína leucémica PML-RARα reprime la transcripción de sus genes diana por el reclutamiento de DNMTs y e
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Ruiz, Emmanuelle. "Coopération entre les inducteurs de l’EMT (EMT-TF/miRNA) et les altérations oncogéniques dans la tumorigenèse mammaire." Thesis, Lyon 1, 2015. http://www.theses.fr/2015LYO10069.

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Les cellules cancéreuses sont capables de réactiver la transition Epithélio-Mésenchymateuse (EMT), mécanisme embryonnaire, pour acquérir une mobilité et une capacité de dédifférenciation. L'EMT conduit à une reprogrammation génétique avec la réactivation d'inducteurs de l'EMT, qui sont en majorité des facteurs de transcription (EMT-TF), et conduit à l'inhibition de miARN. Par ailleurs des stress oncogéniques sont essentiels à la progression tumorale. Le but de mon projet de thèse était de comprendre comment les événements de reprogrammation génétique survenant au cours de l'EMT coopèrent avec
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McElwee, Joshua J. "A comparative analysis of transcriptional alterations in long-lived insulin/IGF-1-like signaling mutants in Caenorhabditis elegans and Drosophila melanogaster /." Thesis, Connect to this title online; UW restricted, 2004. http://hdl.handle.net/1773/4982.

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Jamrog, Laura. "Impact des altérations génétiques de PAX5 sur le développement de la lignée lymphoïde B et dans la leucémogenèse des LAL-B." Electronic Thesis or Diss., Toulouse 3, 2021. http://www.theses.fr/2021TOU30306.

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Le gène PAX5 (Paired boX 5) code un facteur de transcription essentiel pour la différenciation lymphoïde B. Nous avons montré que les deux isoformes PAX5A et PAX5B étaient différentiellement régulées mais pouvaient exercer une fonction équivalente durant l'induction de la différenciation lymphoïde B et pourraient présenter des différences fonctionnelles à la suite de l'activation des lymphocytes B. Le contrôle précis de leur expression peut ainsi refléter un moyen d'ajuster finement le dosage de PAX5 pendant le processus de différenciation des cellules de la lignée lymphoïde B. PAX5 est la cib
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Redondo, Monte Enric [Verfasser], and Philipp [Akademischer Betreuer] Greif. "Investigation of transcription factor alterations in core binding factor leukemia : implications in clonal expansion, cell metabolism and lineage fate decisions / Enric Redondo Monte ; Betreuer: Philipp Greif." München : Universitätsbibliothek der Ludwig-Maximilians-Universität, 2020. http://d-nb.info/122568269X/34.

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Borralleras, Fumaña Cristina 1988. "Correlation between cognitive phenotype, neural morphology and molecular alterations in mouse models of Williams-Beuren syndrome : new therapeutic approaches." Doctoral thesis, Universitat Pompeu Fabra, 2016. http://hdl.handle.net/10803/388032.

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Williams-Beuren syndrome (WBS) is a rare neurodevelopmental disorder caused by a heterozygous deletion of 26-28 contiguous genes in the 7q11.23 region. So far, a great deal of attention has been focused on its unique and distinctive neurocognitive profile. Although important progress has been made with regards to clinical characterization or genotype-phenotype correlations, a much deeper insight into the neuropathological features of WBS would be of great interest. In this thesis project, we have used a WBS mouse model carrying a heterozygous deletion that mimics the most common deletion found
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Chanapai, Seni. "Photocontrol of artificial transcription factors." Thesis, Cardiff University, 2013. http://orca.cf.ac.uk/58014/.

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The design of a photoswitchable homeodomain artificial transcription factor (PATF), modelled on an engrailed homeodomain, for the purpose of controlling DNA binding affinity and controlling the transcription process in cells using light has been investigated. This study was conducted using a 3,3’-bis(sulfo)- 4,4’bis(chloroacetamino)azobenzene crosslinker, alkylated between two cysteine residues with different spacings (i, i+4, i, i+7 and i, i+11) and either a rigid or flexible linker domain. In previous studies, basic leucine zipper transcription activators have been photocontrolled in living
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Pinacho, Garcia Raquel. "SP Transcription factors in psychotic disorders." Doctoral thesis, Universitat de Barcelona, 2015. http://hdl.handle.net/10803/327025.

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Psychotic disorders including bipolar disorder and schizophrenia are a leading cause of disability across the world but the underlying pathophysiological mechanisms remain poorly understood. Available treatments are inadequate for some sets of symptoms as is the case for negative symptoms in schizophrenia. Alterations in brain connectivity, synaptic plasticity, N-methyl D aspartate receptor (NMDAR) signalling and calcium homeostasis have been suggested to contribute to these disorders. However, the particular transcriptional programmes altered in these disorders are not fully characterised. Pr
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Müller, Susanne. "Transcription factors regulating the Btk promoter /." Stockholm, 1997. http://diss.kib.ki.se/1997/91-628-2717-0.

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Paik, Elizabeth Jae-Eun. "Caudal Transcription Factors in Hematopoietic Development." Thesis, Harvard University, 2012. http://dissertations.umi.com/gsas.harvard:10254.

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During embryogenesis, hematopoietic cells arise from the lateral plate mesoderm (LPM) following gastrulation. The transcriptional program required for this LPM to blood switch is not fully understood. Previous work on a zebrafish mutant with a deletion in the cdx4 gene demonstrated the importance of this caudal transcription factor in the LPM to blood transition. To explain how cdx4 regulates embryonic hematopoiesis, two main approaches were taken in this thesis. The first part of the thesis describes a chemical genetics screen that identified cdx4 interacting pathways. To find small molecules
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Bücher zum Thema "Alterations of transcription factors"

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Ravid, Katya, and Jonathan D. Licht, eds. Transcription Factors. John Wiley & Sons, Inc., 2000. http://dx.doi.org/10.1002/0471223883.

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Gossen, Manfred, Jörg Kaufmann, and Steven J. Triezenberg, eds. Transcription Factors. Springer Berlin Heidelberg, 2004. http://dx.doi.org/10.1007/978-3-642-18932-6.

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Higgins, Paul J., ed. Transcription Factors. Humana Press, 2010. http://dx.doi.org/10.1007/978-1-60761-738-9.

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1947-, Locker Joseph, ed. Transcription factors. BIOS, 2001.

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Yamaguchi, Nobutoshi, ed. Plant Transcription Factors. Springer New York, 2018. http://dx.doi.org/10.1007/978-1-4939-8657-6.

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Link, Wolfgang, ed. FOXO Transcription Factors. Springer New York, 2019. http://dx.doi.org/10.1007/978-1-4939-8900-3.

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Yuan, Ling, and Sharyn E. Perry, eds. Plant Transcription Factors. Humana Press, 2011. http://dx.doi.org/10.1007/978-1-61779-154-3.

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Maiese, Kenneth, ed. Forkhead Transcription Factors. Springer New York, 2010. http://dx.doi.org/10.1007/978-1-4419-1599-3.

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Latchman, David S. Eukaryotic transcription factors. 5th ed. Academic Press, 2008.

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Latchman, David S. Eukaryotic transcription factors. 5th ed. Elsevier/Academic Press, 2008.

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Buchteile zum Thema "Alterations of transcription factors"

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Monovich, Alexander C., Aishwarya Gurumurthy, and Russell J. H. Ryan. "The Diverse Roles of ETV6 Alterations in B-Lymphoblastic Leukemia and Other Hematopoietic Cancers." In Transcription factors in blood cell development. Springer Nature Switzerland, 2024. http://dx.doi.org/10.1007/978-3-031-62731-6_13.

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Carlberg, Carsten, and Ferdinand Molnár. "Transcription Factors." In Mechanisms of Gene Regulation. Springer Netherlands, 2016. http://dx.doi.org/10.1007/978-94-017-7741-4_4.

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Arampatzis, Adamantios, Lida Mademli, Thomas Reilly, et al. "Transcription Factors." In Encyclopedia of Exercise Medicine in Health and Disease. Springer Berlin Heidelberg, 2012. http://dx.doi.org/10.1007/978-3-540-29807-6_3139.

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Gooch, Jan W. "Transcription Factors." In Encyclopedic Dictionary of Polymers. Springer New York, 2011. http://dx.doi.org/10.1007/978-1-4419-6247-8_14992.

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Carlberg, Carsten, and Ferdinand Molnár. "Transcription Factors." In Mechanisms of Gene Regulation. Springer Netherlands, 2013. http://dx.doi.org/10.1007/978-94-007-7905-1_4.

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Wong, Henry K. "Transcription Factors." In Principles of Molecular Rheumatology. Humana Press, 2000. https://doi.org/10.1007/978-1-59259-018-6_7.

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Herrera, F. J., D. D. Shooltz, and S. J. Triezenberg. "Mechanisms of Transcriptional Activation in Eukaryotes." In Transcription Factors. Springer Berlin Heidelberg, 2004. http://dx.doi.org/10.1007/978-3-642-18932-6_1.

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Sheen, J. H., and R. B. Dickson. "c-Myc in Cellular Transformation and Cancer." In Transcription Factors. Springer Berlin Heidelberg, 2004. http://dx.doi.org/10.1007/978-3-642-18932-6_10.

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Lasar, A., R. Marienfeld, T. Wirth, and B. Baumann. "NF-κB: Critical Regulator of Inflammation and the Immune Response." In Transcription Factors. Springer Berlin Heidelberg, 2004. http://dx.doi.org/10.1007/978-3-642-18932-6_11.

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Kumar, V., and D. P. Sarkar. "Hepatitis B Virus X Protein: Structure-Function Relationships and Role in Viral Pathogenesis." In Transcription Factors. Springer Berlin Heidelberg, 2004. http://dx.doi.org/10.1007/978-3-642-18932-6_12.

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Konferenzberichte zum Thema "Alterations of transcription factors"

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Sharif, Maria, Peter John, and Attya Bhatti. "Identification of Potential HUB Genes and Associated Transcription Factors in Rheumatoid Arthritis." In 2023 20th International Bhurban Conference on Applied Sciences and Technology (IBCAST). IEEE, 2023. http://dx.doi.org/10.1109/ibcast59916.2023.10713045.

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Zhang, Ji, and Morton H. Friedman. "The Adaptive Response of Endothelial Transcription to Increased Shear Stress In Vitro." In ASME 2010 Summer Bioengineering Conference. American Society of Mechanical Engineers, 2010. http://dx.doi.org/10.1115/sbc2010-19318.

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Previous studies have shown a substantial effect of shear stress on endothelial phenotype and functions such as production of nitric oxide, secretion of growth factors, inflammatory responses, production of reactive oxygen species, permeability to macromolecules and cytoskeletal remodeling [1–3]. However, the dynamics of the endothelial adaptive response to changes in shear stress are largely unknown. The response of vascular endothelial cells to alterations in shear stress is an essential component of normal endothelial physiology, since local shear stress can be altered in vivo by the global
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Kaczmarczyk, B., C. De la Calle-Fabregat, A. C. Duarte, et al. "POS1212 INTERPLAY BETWEEN TRANSCRIPTION FACTORS AND DNA METHYLATION ALTERATIONS SPECIFIC TO PATIENTS WITH RHEUMATOID ARTHRITIS-ASSOCIATED INTERSTITIAL LUNG DISEASE (RA-ILD)." In EULAR 2024 European Congress of Rheumatology, 12-15 June. Vienna, Austria. BMJ Publishing Group Ltd and European League Against Rheumatism, 2024. http://dx.doi.org/10.1136/annrheumdis-2024-eular.4409.

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Collins, Corolyn J., Richard B. Levene, Christina P. Ravera, Marker J. Dombalagian, David M. Livingston, and Dennis C. Lynch. "MOLECULAR CLONING OF THE HUMAN GENE FOR VON WILLEBRAND FACTOR AND IDENTIFICATION OF THE TRANSCRIPTION INITIATION SITE." In XIth International Congress on Thrombosis and Haemostasis. Schattauer GmbH, 1987. http://dx.doi.org/10.1055/s-0038-1642830.

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Most patients with von Willebrand's disease appear to have a defect affecting the level of expression of the von Willebrand factor (vWf) gene. Thus, an understanding of the pathogenesis of von Willebrand's disease will require an analysis of the structure and function of the vWf gene in normals and in patients. To begin such analyses, we have screened a human genomic cosmid library with probes obtained from vWf cDNA and isolated a colinear segment spanning ≈175 kb in five overlapping clones. This segment extends ≈25 kb upstream and ≈5 kb downstream of the transcription start and stop sites for
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Abdel-Sayed, Philippe, Arne Vogel, and Dominique P. Pioletti. "Dissipation Can Act as a Mechanobiological Signal in Cartilage Differentiation." In ASME 2013 International Mechanical Engineering Congress and Exposition. American Society of Mechanical Engineers, 2013. http://dx.doi.org/10.1115/imece2013-62268.

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Knee cartilage is a soft tissue having viscoelastic properties. Under cyclic loadings, viscoelastic materials dissipate mechanical loadings through heat generation. In knee cartilage, this heat might not be convected because of the tissue avascularity, resulting thus to a local temperature increase. As cells are sensitive to temperature, these thermo-mechanical phenomena of energy dissipation could influence their metabolism. The goal of this study is to evaluate the effect of thermogenesis on chondrogenic differentiation. First, we focused our work in quantifying the heat generated in cartila
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Gaykalova, Daria A., Chad A. Glazer, Rajita Vatapalli, et al. "Abstract 2042: Cancer-specific transcription factor BORIS has different effects on expression of its target genes via chromatin structure alterations." In Proceedings: AACR 102nd Annual Meeting 2011‐‐ Apr 2‐6, 2011; Orlando, FL. American Association for Cancer Research, 2011. http://dx.doi.org/10.1158/1538-7445.am2011-2042.

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Hussain, Showket, Neha Singh, Irfana Salam, et al. "Abstract 2722: Transcription factor NF-kB in esophageal squamous cell carcinoma: Alterations in activity and expression during Human Papillomavirus infection." In Proceedings: AACR 102nd Annual Meeting 2011‐‐ Apr 2‐6, 2011; Orlando, FL. American Association for Cancer Research, 2011. http://dx.doi.org/10.1158/1538-7445.am2011-2722.

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Okano, Larissa Miyuki, Alexandre Luiz Korte de Azevedo, Tamyres Mingorance Carvalho, Tathiane Maistro Malta, Mauro Antonio Alves Castro, and Luciane Regina Cavalli. "Characterization of an epigenetic regulatory network on basal-like breast cancer subtype and its impact on signaling pathways and biological processes." In Brazilian Breast Cancer Symposium 2024. Mastology, 2024. http://dx.doi.org/10.29289/259453942024v34s1029.

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Objective: The main objective of this study was to identify DNA methylation at the distal cis-regulatory genomic regions associated with the basal-like breast cancer (BLBC) subtype, construct an epigenetic regulatory network, and determine its impact on cancer-associated signaling pathways and biological processes. Methodology: BLBC (n=134) and non- -tumoral breast (n=84) samples with DNA methylation, mRNA, and miRNA expression data were downloaded from The Cancer Genome Atlas (TCGA) database using a pipeline of computational tools. DNA methylation patterns on cancer- -specific enhancers enric
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Ponomarenko, J., T. Merkulova, G. Orlova, E. Gorshkova, O. Fokin, and M. Ponomarenko. "Mining genome variation to associate disease with transcription factor binding site alteration." In Proceedings 2nd Annual IEEE International Symposium on Bioinformatics and Bioengineering (BIBE 2001). IEEE, 2001. http://dx.doi.org/10.1109/bibe.2001.974424.

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Pires, Bruno Ricardo Barreto, Paulo Rohan, Caroline Borges-de-Almeida, Rafael Cardoso Maciel Costa Silva, Renata Binato, and Eliana Abdelhay. "Comprehensive analysis of TWIST1 in breast cancer and other carcinomas: an association with prognosis and tumor microenvironment." In Brazilian Breast Cancer Symposium 2024. Mastology, 2024. http://dx.doi.org/10.29289/259453942024v34s1063.

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Objective: Metastasis is the main cause of death in patients with carcinomas. This process depends on a phenotypic alteration known as epithelial–mesenchymal transition (EMT), regulated by transcription factors (TFs), including TWIST1, whose increased levels have been described in several carcinomas, including breast cancer. However, a comprehensive analysis of its expression to elucidate its predictive value still needs to be performed. This study aimed to understand the prognostic value of TWIST1 expression and its biological relevance for tumor microenvironment (TME) in breast cancer and ot
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Berichte der Organisationen zum Thema "Alterations of transcription factors"

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Arazi, Tzahi, Vivian Irish, and Asaph Aharoni. Micro RNA Targeted Transcription Factors for Fruit Quality Improvement. United States Department of Agriculture, 2008. http://dx.doi.org/10.32747/2008.7592651.bard.

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Fruits are unique to flowering plants and represent an important component of human and animal diets. Development and maturation of tomato fruit is a well-programmed process, and yet, only a limited number of factors involved in its regulation have been characterized. Micro-RNAs (miRNAs) are small, endogenous RNAs that regulate gene expression in animals and plants. Plant miRNAs have a vital role in the generation of plant forms through post-transcriptional regulation of the accumulation of developmental regulators, especially transcription factors. Recently, we and others have demonstrated th
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Michelotti, Julia M. Identification of Mammary Specific Transcription Factors. Defense Technical Information Center, 1995. http://dx.doi.org/10.21236/ada303179.

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Sederoff, Ronald, Ross Whetten, David O'Malley, and Malcolm Campbell. Transcription Factors in Xylem Development. Final report. Office of Scientific and Technical Information (OSTI), 1999. http://dx.doi.org/10.2172/760586.

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Iyer, Vishwanath R. Genome-Wide Chromosomal Targets of Oncogenic Transcription Factors. Defense Technical Information Center, 2005. http://dx.doi.org/10.21236/ada436905.

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Iyer, Vishwanath R. Genome-Wide Chromosomal Targets of Oncogenic Transcription Factors. Defense Technical Information Center, 2008. http://dx.doi.org/10.21236/ada485280.

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Lyer, Vishwanath R. Genome-Wide Chromosomal Targets of Oncogenic Transcription Factors. Defense Technical Information Center, 2006. http://dx.doi.org/10.21236/ada455791.

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Lyer, Vishwanath R. Genome-Wide Chromosomal Targets of Oncogenic Transcription Factors. Defense Technical Information Center, 2007. http://dx.doi.org/10.21236/ada470576.

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Grafi, Gideon, and Brian Larkins. Endoreduplication in Maize Endosperm: An Approach for Increasing Crop Productivity. United States Department of Agriculture, 2000. http://dx.doi.org/10.32747/2000.7575285.bard.

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The focus of this research project is to investigate the role of endoreduplication in maize endosperm development and the extent to which this process contributes to high levels of starch and storage protein synthesis. Although endoreduplication has been widely observed in many cells and tissues, especially those with high levels of metabolic activity, the molecular mechanisms through which the cell cycle is altered to produce consecutive cycles of S-phase without an intervening M-phase are unknown. Our previous research has shown that changes in the expression of several cell cycle regulatory
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Grotewold, Erich. Engineering phenolics metabolism in the grasses using transcription factors. Office of Scientific and Technical Information (OSTI), 2013. http://dx.doi.org/10.2172/1088660.

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Schaber, John D. Expression and Activation of STAT Transcription Factors in Breast Cancer. Defense Technical Information Center, 1998. http://dx.doi.org/10.21236/ad1012061.

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