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Thematische Bibliographien / Anti-immunoglobulin E autoantibodies

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Auswahl der wissenschaftlichen Literatur zum Thema „Anti-immunoglobulin E autoantibodies“

Autor: Grafiati

Veröffentlicht am 4. Juni 2021

Zuletzt aktualisiert am 29. Juli 2025

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Zeitschriftenartikel zum Thema "Anti-immunoglobulin E autoantibodies"

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Kawasaki, Eiji. "Anti-Islet Autoantibodies in Type 1 Diabetes." International Journal of Molecular Sciences 24, no. 12 (2023): 10012. http://dx.doi.org/10.3390/ijms241210012.

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Anti-islet autoantibodies serve as key markers in immune-mediated type 1 diabetes (T1D) and slowly progressive T1D (SPIDDM), also known as latent autoimmune diabetes in adults (LADA). Autoantibodies to insulin (IAA), glutamic acid decarboxylase (GADA), tyrosine phosphatase-like protein IA-2 (IA-2A), and zinc transporter 8 (ZnT8A) are currently employed in the diagnosis, pathological analysis, and prediction of T1D. GADA can also be detected in non-diabetic patients with autoimmune diseases other than T1D and may not necessarily reflect insulitis. Conversely, IA-2A and ZnT8A serve as surrogate

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MORITA, Kazumasa, Akihiro FUJISAWA, Yosuke YAGI, et al. "Immunoglobulin A anti-BP230 autoantibodies in linear immunoglobulin A/immunoglobulin G bullous dermatosis." Journal of Dermatology 38, no. 10 (2011): 1030–32. http://dx.doi.org/10.1111/j.1346-8138.2010.01158.x.

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von Gunten, Stephan, Monique Vogel, Alexander Schaub, et al. "Intravenous immunoglobulin preparations contain anti–Siglec-8 autoantibodies." Journal of Allergy and Clinical Immunology 119, no. 4 (2007): 1005–11. http://dx.doi.org/10.1016/j.jaci.2007.01.023.

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Di Mario, Umberto, Riccardo Perfetti, Emanuela Anastasi, et al. "Autoantibodies to insulin do appear in non-diabetic patients with autoimmune disorders: Comparison with anti-immunoglobulin antibodies and other autoimmune phenomena." Acta Endocrinologica 122, no. 2 (1990): 303–8. http://dx.doi.org/10.1530/acta.0.1220303.

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Abstract Insulin- and anti-immunoglobulin-antibodies have been recently reported in pre-diabetic subjects: the former has been proposed as a predictive marker of Type I diabetes in non-diabetic-subjects. To evaluate the diabetes-related specificity of these antibodies, the presence of insulin autoantibodies, using a recently developed and highly sensitive competitive radioimmune assay, and of anti-immunoglobulin antibodies together with that of immune complexes and of other autoantibodies has been investigated in patients with organ- or non-organ-specific autoimmune diseases. One hundred and e

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Dietrich, G., M. Algiman, Y. Sultan, UE Nydegger, and MD Kazatchkine. "Origin of anti-idiotypic activity against anti-factor VIII autoantibodies in pools of normal human immunoglobulin G (IVIg)." Blood 79, no. 11 (1992): 2946–51. http://dx.doi.org/10.1182/blood.v79.11.2946.2946.

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Abstract Therapeutic preparations of polyspecific IgG obtained from plasma pools of a large number of normal donors (IVIg) express anti-idiotypic activity against a wide spectrum of natural and disease-associated autoantibodies. The present study investigated the origin of anti- idiotypic activity against autoantibodies to factor VIII. The neutralizing activity of pools of IgG against patients' anti-factor VIII autoantibodies was not influenced by the presence of individuals with natural anti-factor VIII antibodies among donors contributing to the pool. A higher frequency of neutralizing antib

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Dietrich, G., M. Algiman, Y. Sultan, UE Nydegger, and MD Kazatchkine. "Origin of anti-idiotypic activity against anti-factor VIII autoantibodies in pools of normal human immunoglobulin G (IVIg)." Blood 79, no. 11 (1992): 2946–51. http://dx.doi.org/10.1182/blood.v79.11.2946.bloodjournal79112946.

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Therapeutic preparations of polyspecific IgG obtained from plasma pools of a large number of normal donors (IVIg) express anti-idiotypic activity against a wide spectrum of natural and disease-associated autoantibodies. The present study investigated the origin of anti- idiotypic activity against autoantibodies to factor VIII. The neutralizing activity of pools of IgG against patients' anti-factor VIII autoantibodies was not influenced by the presence of individuals with natural anti-factor VIII antibodies among donors contributing to the pool. A higher frequency of neutralizing antibodies aga

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Hattori, Naoki, Katsuji Ikekubo, Yasuhisa Nakaya, Kaori Kitagawa, and Chiyoko Inagaki. "Immunoglobulin G Subclasses and Prolactin (PRL) Isoforms in Macroprolactinemia Due to Anti-PRL Autoantibodies." Journal of Clinical Endocrinology & Metabolism 90, no. 5 (2005): 3036–44. http://dx.doi.org/10.1210/jc.2004-1600.

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Although macroprolactinemia due to antiprolactin (anti-PRL) autoantibodies is not uncommon among hyperprolactinemic patients, the pathogenesis of such macroprolactinemia is still unknown. We examined IgG subclasses of anti-PRL autoantibodies by enzyme immunoassay, and PRL phosphorylation and isoforms by Western blotting, mass spectrometry, and two-dimensional electrophoresis in six patients with anti-PRL autoantibodies and in 29 controls. PRL-specific IgG subclasses in patients with anti-PRL autoantibodies were heterogeneous, but five of six patients showed IgG4 predominance, which is known to

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Senécal, Jean-Luc, Sabrina Hoa, Roger Yang, and Martial Koenig. "Pathogenic roles of autoantibodies in systemic sclerosis: Current understandings in pathogenesis." Journal of Scleroderma and Related Disorders 5, no. 2 (2019): 103–29. http://dx.doi.org/10.1177/2397198319870667.

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The potential pathogenic role for autoantibodies in systemic sclerosis has captivated researchers for the past 40 years. This review answers the question whether there is yet sufficient knowledge to conclude that certain serum autoantibodies associated with systemic sclerosis contribute to its pathogenesis. Definitions for pathogenic, pathogenetic and functional autoantibodies are formulated, and the need to differentiate these autoantibodies from natural autoantibodies is emphasized. In addition, seven criteria for the identification of pathogenic autoantibodies are proposed. Experimental evi

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de Carvalho, Jozélio Freire, Yaniv Sherer, and Yehuda Shoenfeld. "The fine-tuning of anti-oxidized low-density lipoprotein antibodies in cardiovascular disease and thrombosis." Thrombosis and Haemostasis 98, no. 12 (2007): 1157–59. http://dx.doi.org/10.1160/th07-11-0652.

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SummaryAnti-oxidised low-density lipoprotein (anti-oxLDL) antibodies are a heterogeneous group of autoantibodies including both pathogenic and protective subsets. Whereas in most studies the levels of anti-oxLDL antibodies were associated with enhanced atherosclerosis as evaluated by different methods, immunization with oxLDL leads to elevated levels of anti-oxLDL and protection against atherosclerosis. Anti-oxLDL can also be used for immunomodulation of atherosclerosis (i.e. possible therapeutic use of intravenous immunoglobulin, oral tolerance). More specific autoantibodies out of total anti

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Sui, Jingzhe, Haiwei Li, Yanqiong Liu, et al. "Correlation study between clinical phenotypes and autoantibodies in systemic sclerosis in Southwest China." Medicine 104, no. 23 (2025): e42639. https://doi.org/10.1097/md.0000000000042639.

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This study investigates the characteristics of baseline data and the correlation between autoantibodies and clinical phenotypes in patients with systemic sclerosis (SSc) from Southwest China. A total of 346 patients diagnosed with SSc between 2021 and 2023 were included. Clinical data and laboratory data were collected to analyze the relationship between autoantibodies and clinical phenotypes. The prevalence of hypertension and osteoporosis in female patients with SSc was significantly higher than that in male patients. Anti-Scl-70 antibody was positively correlated with Raynaud phenomenon, fa

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Dissertationen zum Thema "Anti-immunoglobulin E autoantibodies"

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Holfelder, Christina Ingrid. "Effects of the glutamic acid decarboxylase (GAD) inhibitor semicarbazide and anti-GAD autoantibodies-containing immunoglobulin G on neuronal network activity within the motor cortex." Diss., lmu, 2010. http://nbn-resolving.de/urn:nbn:de:bvb:19-123547.

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Holfelder, Christina Ingrid [Verfasser]. "Effects of the glutamic acid decarboxylase (GAD) inhibitor semicarbazide and anti-GAD autoantibodies-containing immunoglobulin G on neuronal network activity within the motor cortex / vorgelegt von Christina Ingrid Holfelder." 2010. http://d-nb.info/1009568817/34.

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Bücher zum Thema "Anti-immunoglobulin E autoantibodies"

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F, Shakib, ed. Autoantibodies to immunoglobulins. Karger, 1989.

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Noutsias, Michel, and Bernhard Maisch. Myocarditis and pericarditis. Oxford University Press, 2015. http://dx.doi.org/10.1093/med/9780199687039.003.0058.

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Transition of acute myocarditis to dilated cardiomyopathy occurs in approximately 20% of patients within a follow-up period of 33 months. Recent research has revealed the adverse prognostic impact of several clinical parameters for this scenario. Acute myocarditis and its sequelae dilated cardiomyopathy and inflammatory cardiomyopathy are often caused by viral infections. Histological evaluation of endomyocardial biopsies is critical for the diagnosis of the cardiomyopathy entity and for the clinical management of around 20% of the patients. Additionally, contemporary diagnostic procedures of

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Noutsias, Michel, and Bernhard Maisch. Myocarditis and pericarditis. Oxford University Press, 2017. http://dx.doi.org/10.1093/med/9780199687039.003.0058_update_001.

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Transition of acute myocarditis to dilated cardiomyopathy occurs in approximately 20% of patients within a follow-up period of 33 months. Recent research has revealed the adverse prognostic impact of several clinical parameters for this scenario. Acute myocarditis and its sequelae dilated cardiomyopathy and inflammatory cardiomyopathy are often caused by viral infections. Histological evaluation of endomyocardial biopsies is critical for the diagnosis of the cardiomyopathy entity and for the clinical management of around 20% of the patients. Additionally, contemporary diagnostic procedures of

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Buchteile zum Thema "Anti-immunoglobulin E autoantibodies"

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Maltsev, Dmytro. "Efficacy of rituximab in autism spectrum disorders associated with genetic deficiency of the folate cycle with signs of antineuronal autoimmunity." In IMMUNODIAGNOSTICS AND IMMUNOTHERAPY OF NEUROPSYCHIATRIC DISORDERS IN CHILDREN. TECHNOLOGY CENTER PC, 2025. https://doi.org/10.15587/978-617-8360-21-4.ch12.

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Advances in genetics, molecular biology, and immunology over the past decades have significantly changed our understanding of the etiology and pathogenesis of autism spectrum disorders (ASD) in children. One of the key advances in this direction is the elucidation of the association of genetic deficiency of the folate cycle (GDFC) with ASD, evidence for which is based on the results of at least 5 meta-analyses of randomized controlled clinical trials and a number of additional controlled trials, the data of which have not yet been properly summarized. It has been established that GDFC leads to

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Molnár, Ildikó. "Deiodinase Enzymes and Their Activities in Graves’ Hyperthyroidism." In Graves' Disease [Working Title]. IntechOpen, 2021. http://dx.doi.org/10.5772/intechopen.97007.

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The origin of hyperthyroidism in Graves’ disease was displayed demonstrating the complexity of the processes. The role of stimulating TSH receptor antibodies is the one factor for the production of increased thyroidal T3 and T4. The T3 and T4 formation in colloid-embedded thyroglobulin and the activities of thyroidal deiodinases [type 1 (DIO1) and type 2 (DIO2)] play a crucial role in that. The findings of different authors were summarized with respect to highlighting the role of tissue-specific deiodinase activities. Apart from the results of experimental studies, the clinical results were br

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Zivelonghi, Cecilia, and Andrew McKeon. "A Girl With Back Pain, Paresthesias, and Painful Vision Loss." In Mayo Clinic Cases in Neuroimmunology, edited by Andrew McKeon, B. Mark Keegan, and W. Oliver Tobin. Oxford University Press, 2021. http://dx.doi.org/10.1093/med/9780197583425.003.0014.

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A 12-year-old girl sought care for subacute onset of cramping back pain, along with paresthesias in her lower limbs up to the waistline, both hands, upper back, and chest, followed by rapidly progressive (over a few hours) painful vision loss affecting initially the right eye with subsequent involvement of the left eye. She underwent neuroophthalmologic evaluation and was diagnosed with bilateral optic neuritis. A positive Lhermitte sign was also present. The patient was tested for aquaporin-4-immunoglobulin G autoantibodies, which were positive in both serum and cerebrospinal fluid. A diagnos

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Spalter, Sergio H., Srini Kaveri, and Michel D. Kazatchkine. "Anti-Idiotypes to Autoantibodies in Therapeutic Preparations of Normal Polyspecific Human IgG (Intravenous Immunoglobulin, IVIg)." In Idiotypes in Medicine: Autoimmunity, Infection and Cancer. Elsevier, 1997. http://dx.doi.org/10.1016/b978-044482807-1/50021-1.

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Konferenzberichte zum Thema "Anti-immunoglobulin E autoantibodies"

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Beardsley, D. S. "IMMUNE THROMBOCYTOPENIA (ITP) : PLATELET TARGET ANTIGENS OF THE ANTIBODIES IN DIFFERENT CLINICAL SETTINGS." In XIth International Congress on Thrombosis and Haemostasis. Schattauer GmbH, 1987. http://dx.doi.org/10.1055/s-0038-1644757.

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Autoantibodies against platelet antigens are important in the pathogenesis of ITP. We have studied the antigenic targets of these autoantibodies by immunoblotting using electrophoretically separated proteins from normal, Glanzmann's Thrombasthenic, and Bernard-Soulier Syndrome platelets immobilized on nitrocellulose paper. Incubation of these proteins with ITP patient serum, immunoglobulin, or F(ab*>2, followed by labeled antiglobulin, allowed identification of the antigenic target glycoproteins (GPfs). Patients with ITP of several categories were studied: A) Chronic ITP (> 1 yr duration

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Bajwa, Wardah, Mary Kosciuk, Randel Swanson, et al. "Extravasated Brain-Reactive Autoantibodies Perturb Neuronal Surface Protein Expression in Alzheimer's Pathology." In 27th Annual Rowan-Virtua Research Day. Rowan University Libraries, 2023. https://doi.org/10.31986/issn.2689-0690_rdw.stratford_research_day.75_2023.

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Background: Increased blood-brain barrier (BBB) permeability is reported in both the neuropathological and in vivo studies in both Alzheimer’s Disease (AD) and age matched cognitively normal, no cognitive impairment (NCI), subjects. Impaired BBB allows various vascular components such as immunoglobulin G (IgG) to extravasate into the brain and specifically bind to various neuronal surface proteins (NSP), also known as brain reactive autoantibodies (BrABs). This interaction is predicted to further enhance deposition of amyloid plaques. Hypothesis: Interaction between extravasated BrABs and its

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Oliveira, Daniel Alves de, Arthur Cesário de Holanda, Vitor Maia Arca, Marcos Eugênio Ramalho Bezerra, and Renata Amaral Andrade. "Fluctuating spastic paraparesis with “owls sign” in spine MRI: a diagnostic challenge." In XIV Congresso Paulista de Neurologia. Zeppelini Editorial e Comunicação, 2023. http://dx.doi.org/10.5327/1516-3180.141s1.775.

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Case presentation: A 42-year-old female patient, with no previous comorbidities, complained of tremors and gait disturbance that had started two years before. She mentioned that her legs had fluctuating moving difficulties, associated with pain. At her first evaluation at clinical onset, the case was interpreted as episodic myoclonus, and clonazepam was prescribed with partial improvement. In the initial physical examination, it was observed pathologic glabellar reflex, bilateral hyperreflexia in the lower limbs, and bilateral foot and knee clonus, with no findings in tonus or muscle weakness.

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