Thematische Bibliographien / Antiarrhythmic effect

Inhaltsverzeichnis

  1. Zeitschriftenartikel
  2. Dissertationen
  3. Bücher
  4. Buchteile
  5. Konferenzberichte

Auswahl der wissenschaftlichen Literatur zum Thema „Antiarrhythmic effect“

Autor: Grafiati
Veröffentlicht am 29. Juni 2021
Zuletzt aktualisiert am 29. Juli 2025

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Zeitschriftenartikel zum Thema "Antiarrhythmic effect"

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Calvert, Clay A., and John Brown. "Influence of Antiarrhythmia Therapy on Survival Times of 19 Clinically Healthy Doberman Pinschers With Dilated Cardiomyopathy That Experienced Syncope, Ventricular Tachycardia, and Sudden Death (1985–1998)." Journal of the American Animal Hospital Association 40, no. 1 (2004): 24–28. http://dx.doi.org/10.5326/0400024.

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Overtly healthy Doberman pinschers, having moderate to severe myocardial failure secondary to dilated cardiomyopathy, which experienced ventricular tachycardia, syncope or collapse, and sudden death were studied to determine the effect of antiarrhythmic medication on their clinical outcome. Antiarrhythmia drug therapy may have retarded sudden death in 13 treated dogs compared to the six dogs not administered antiarrhythmia drugs.
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Rusinova, Radda, Roger E. Koeppe, and Olaf S. Andersen. "A general mechanism for drug promiscuity: Studies with amiodarone and other antiarrhythmics." Journal of General Physiology 146, no. 6 (2015): 463–75. http://dx.doi.org/10.1085/jgp.201511470.

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Amiodarone is a widely prescribed antiarrhythmic drug used to treat the most prevalent type of arrhythmia, atrial fibrillation (AF). At therapeutic concentrations, amiodarone alters the function of many diverse membrane proteins, which results in complex therapeutic and toxicity profiles. Other antiarrhythmics, such as dronedarone, similarly alter the function of multiple membrane proteins, suggesting that a multipronged mechanism may be beneficial for treating AF, but raising questions about how these antiarrhythmics regulate a diverse range of membrane proteins at similar concentrations. One
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Rudakova, I. P., and O. V. Gashkova. "Assessment of impact of a new derivative 2-(diethylamino)-N-(o-toluyl) acetamide, manifesting antiarrhythmic effect in heart rhythm disorders of peripheral origin, on cardiovascular system." Perm Medical Journal 39, no. 4 (2022): 58–64. http://dx.doi.org/10.17816/pmj39458-64.

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Objective. To study the effect of a new derivative of arylamides of aminocarboxylic acids, which has a high antiarrhythmic activity on the state of the cardiovascular system.
 Materials and methods. To study the antiarrhythmic activity of the compound, the experiment was carried out on a model of arrhythmia caused by intravenous administration of aconitine. The effect was evaluated by its ability to prevent the occurrence of arrhythmia or to prolong the survival time of animals. In addition, an analysis of the electrocardiogram of awake rats and rabbits was carried out. The test compound
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Pandya, Bejal, and Pier D. Lambiase. "An avoidable antiarrhythmic side effect." British Journal of Hospital Medicine 67, Sup1 (2006): M14—M15. http://dx.doi.org/10.12968/hmed.2006.67.sup1.20338.

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Madakasira, Sudhakar. "Cardiac antiarrhythmic effect of nortriptyline." General Hospital Psychiatry 8, no. 2 (1986): 123–25. http://dx.doi.org/10.1016/0163-8343(86)90098-8.

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Mont, Lluís. "Antiarrhythmic Effect of Cardiac Resynchronization." Revista Española de Cardiología (English Edition) 58, no. 10 (2005): 1139–41. http://dx.doi.org/10.1016/s1885-5857(06)60390-3.

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Martinez-Hernandez, E., and L. A. Blatter. "Effect of carvedilol on atrial excitation-contraction coupling, Ca2+ release, and arrhythmogenicity." American Journal of Physiology-Heart and Circulatory Physiology 318, no. 5 (2020): H1245—H1255. http://dx.doi.org/10.1152/ajpheart.00650.2019.

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Here we show that the clinically widely used β-blocker carvedilol has profound effects on Ca2+ signaling and ion currents, but also antiarrhythmic effects in adult atrial myocytes. Carvedilol inhibits sodium and calcium currents and leads to failure of ECC but also prevents spontaneous Ca2+ release from cellular sarcoplasmic reticulum (SR) Ca2+ stores in form of arrhythmogenic Ca2+ waves. The antiarrhythmic effect occurs by carvedilol acting directly on the SR ryanodine receptor Ca2+ release channel.
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Wang, Jie, Jun Li, and Bo Feng. "Shen Song Yang Xin Capsule Combined with Antiarrhythmic Drugs, a New Integrative Medicine Therapy, for the Treatment of Frequent Premature Ventricular Contractions (FPVC): A Meta-Analysis of Randomized Controlled Trials." Evidence-Based Complementary and Alternative Medicine 2014 (2014): 1–9. http://dx.doi.org/10.1155/2014/976713.

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Objective. To evaluate the beneficial and adverse effects of Shen Song Yang Xin Capsule (SSYX Capsule) combined with antiarrhythmic drugs for the treatment of frequent premature ventricular contractions (FPVC).Methods. Seven electronic databases were searched to retrieve any potential randomized controlled trials (RCTs) designed to evaluate the clinical efficacy of SSYX Capsule combined with Antiarrhythmic Drugs for FPVC reported in any language, with total effect for FPVC and number of ventricular premature contraction as the main outcome measure. The methodological quality of the included st
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Mirzoyan, Ruben S., Antonina I. Turilova, Tamara S. Gan’shina, et al. "New Antiarrhythmic Agent to Stabilize Functional Activity of Rat Heart Sinus Node Cardiomyocytes." Research Results in Pharmacology 6, no. 4 (2020): 19–27. http://dx.doi.org/10.3897/rrpharmacology.6.58520.

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Introduction: The aim of this study was to explore the antiarrhythmic activity of the new antiarrhythmic drug, succinic acid ester of 5-hydroxyadamantane-2-one (ADK-1110) and its effect on the functional activity of rat heart sinus node. Materials and methods: Experiments were performed on 80 non-linear white awake male rats weighing 200 g, using calcium chloride and aconitine arrhythmia models. The ECG was recorded from all the animals in the II standard lead before the start of the experiment. The effect of ADK-1110 on the electrical activity characteristics of rat heart sinus node pacemaker
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Mirzoyan, Ruben S., Antonina I. Turilova, Tamara S. Gan'shina, et al. "New Antiarrhythmic Agent to Stabilize Functional Activity of Rat Heart Sinus Node Cardiomyocytes." Research Results in Pharmacology 6, no. (4) (2020): 19–27. https://doi.org/10.3897/rrpharmacology.6.58520.

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Introduction: The aim of this study was to explore the antiarrhythmic activity of the new antiarrhythmic drug, succinic acid ester of 5-hydroxyadamantane-2-one (ADK-1110) and its effect on the functional activity of rat heart sinus node. Materials and methods: Experiments were performed on 80 non-linear white awake male rats weighing 200 g, using calcium chloride and aconitine arrhythmia models. The ECG was recorded from all the animals in the II standard lead before the start of the experiment. The effect of ADK-1110 on the electrical activity characteristics of rat heart sinus node pacemaker
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Dissertationen zum Thema "Antiarrhythmic effect"

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Das, Kumuda C. "Amelioration of oxidative lung injury by antiarrhythmic agents." Diss., Virginia Tech, 1992. http://hdl.handle.net/10919/39844.

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Crockett, Thomas Robert. "The mechanism(s) underlying the antiarrhythmic effect of drugs acting on endothelin receptors in myocardial ischaemia and reperfusion." Thesis, University of Strathclyde, 2002. http://ethos.bl.uk/OrderDetails.do?uin=uk.bl.ethos.248261.

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El-Eraky, Hala Mohammed Tawfik. "The effect of gender on the pharmacokinetic and pharmacodynamic properties of drugs affecting cardiac repolarization : a study of antiarrhythmic drugs." Thesis, University of Newcastle Upon Tyne, 2002. http://ethos.bl.uk/OrderDetails.do?uin=uk.bl.ethos.394683.

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Sylwan, Gustafsson Magdalena. "Den antiarytmiska effekten av magnesium : En litteraturstudie relaterat till magnesiumform, dos och typ av arytmi." Thesis, Linnéuniversitetet, Institutionen för kemi och biomedicin (KOB), 2021. http://urn.kb.se/resolve?urn=urn:nbn:se:lnu:diva-105440.

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Bakgrund: Arytmi innebär en avvikande rytm hos hjärtat, alltså att det uppstått en avvikelse i impulsbildning och/eller impulsfortledning. Det kan inverka negativt på livskvalitén, orsaka följdsjukdomar eller leda till livshotande tillstånd. Utöver den konventionella behandlingen finns det behov av ytterligare alternativ. Magnesium är ett essentiellt mineralämne som är viktigt för hjärtats elektrofysiologi. Det har i tidigare studier visat sig att magnesium har haft effekt på postoperativt uppkomna arytmier och att låga nivåer av magnesium hos en frisk population har ökat risken för utveckland
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Spiers, James Paul. "The cardiovascular effects of a novel antiarrhythmic drug (UK-52,046)." Thesis, Queen's University Belfast, 1990. http://ethos.bl.uk/OrderDetails.do?uin=uk.bl.ethos.356976.

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Sun, Wei. "Antiarrhythmic effects of ischaemic preconditioning in anaesthetised rats : studies on the roles of bradykinin and nitric oxide." Thesis, University of Strathclyde, 1995. http://ethos.bl.uk/OrderDetails.do?uin=uk.bl.ethos.249783.

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Yong, Sandro Luis. "A series of amino-2-cyclohexyl esters, their electrophysiological and antiarrhythmic effects as related to actions on ischemia-induced arrhythmias." Thesis, National Library of Canada = Bibliothèque nationale du Canada, 2000. http://www.collectionscanada.ca/obj/s4/f2/dsk1/tape4/PQDD_0021/NQ56650.pdf.

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Bugana, Marco. "Mathematical Modeling to Investigate Antiarrhythmic Drug Side Effects: Rate-Dependence Role in Ionic Currents and Action Potentials Shape in the O’Hara Model." Master's thesis, Alma Mater Studiorum - Università di Bologna, 2012. http://amslaurea.unibo.it/3529/.

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Sudden cardiac death due to ventricular arrhythmia is one of the leading causes of mortality in the world. In the last decades, it has proven that anti-arrhythmic drugs, which prolong the refractory period by means of prolongation of the cardiac action potential duration (APD), play a good role in preventing of relevant human arrhythmias. However, it has long been observed that the “class III antiarrhythmic effect” diminish at faster heart rates and that this phenomenon represent a big weakness, since it is the precise situation when arrhythmias are most prone to occur. It is well known that
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Hung, Chi-Feng, and 洪啟峰. "Electromechanical effect of two antiarrhythmic agents, JKL 1067 and N-allylsecoboldine, on cardiac tissue." Thesis, 1994. http://ndltd.ncl.edu.tw/handle/51832110654720344978.

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碩士<br>國立臺灣大學<br>藥理學研究所<br>82<br>JKL 1067 和 N-Allylsecoboldine(STL-1)是兩個合成生物鹼，發現具有 增強收縮力及減慢心跳頻率之作用。 論文主要研究它們在心臟組織之電 生理及增強心收縮力之效果，並且評估它們的抗心律不整的活性。在大白 鼠心房及心室肌組織，1 到 30 uM JKL 1067 會使收縮力隨濃度增加而增 強，然而，其心房自發性跳動頻率則被減慢。 在大白鼠、天竺鼠的實驗 中，會使因冠狀動脈結紮再灌流及ouabain所誘發之心律不整恢復為正常 心律。 之效果，不會因前處理腎腺素阻斷劑而發生改變，但是會被前處 理的鉀管道阻斷劑所減弱 。 在大白鼠心室細胞中， 動作電位期間會隨 JKL 1067 濃度的增加而延長，且減慢伴隨動作電位去極化速率。JKL 1067可減少鈉電流，並且使鈉電流穩定狀態不活化曲線向負電位方向漂移 。 其恢復之時間常數也受影響而延長。 對於瞬時外流鉀電流，可使其尖 峰電流
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Wu, Adonis Zhi-Yang, and 吳智陽. "Underlying Ionic Channel Mechanism of Antiarrhythmic Effect by a Green Tea Polyphenol, (-)-Epicatechin-3-gallate." Thesis, 2012. http://ndltd.ncl.edu.tw/handle/17957081558289252085.

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博士<br>國防醫學院<br>生命科學研究所<br>101<br>The voltage-gated sodium currents (INa) contribute to cardiac spontaneous action potential (sAP) upstroke, influence AP duration (APD), and may be responsible for early afterdepolarization (EAD). (-)-Epicatechin-3-gallate (ECG), a polyphenol extracted from green tea, has been proposed as an effective compound for improving cardiac contractility. However, the therapeutic potential of ECG on arrhythmia remains unknown. I furnished a definition for the temporal involvement of INa on the characterized ontogenic sAP in primary culture of neonatal rat ventricular myo
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Bücher zum Thema "Antiarrhythmic effect"

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Luc, Hondeghem, ed. Molecular and cellular mechanisms of antiarrhythmic agents. Futura Pub. Co., 1989.

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Wijdicks, Eelco F. M., and Sarah L. Clark. Antihypertensives and Antiarrhythmics. Oxford University Press, 2018. http://dx.doi.org/10.1093/med/9780190684747.003.0013.

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Acute brain injury can precipitate a hypertensive response, which for the most part is the result of stress-induced, increased sympathetic activity. Acute stroke with hypertension may not be a response but more often a prior, untreated hypertension or a patient with no access to medication. This hypertensive response may wane quickly, and aggressive treatment of these temporary surges in blood pressure could have unwanted consequences. Important characteristics of most antihypertensive drugs used in the neurosciences intensive care unit are cost, having a rapid onset with a short duration of a
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Billman, George E., ed. The Effects of Omega-3 Polyunsaturated Fatty Acids on Cardiac Rhythm: Antiarrhythmic, Proarrhythmic, Both or Neither? Frontiers Media SA, 2013. http://dx.doi.org/10.3389/978-2-88919-088-1.

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LeMaitre, John, and Jan Kornder. Anti-arrhythmics in critical illness. Oxford University Press, 2016. http://dx.doi.org/10.1093/med/9780199600830.003.0038.

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Anti-arrhythmic drugs (AADs) are an important component of the pharmacological arsenal used in the management of the critically-ill patient. The benefits of AADs, as well as their potential disadvantageous side effects, largely depend on their effects upon the cardiac action potential. AADs can be broadly grouped according to their cellular actions, upon which their clinical effects depend. However, there is substantial cross-over amongst the groups in terms of these actions and efficacy for particular arrhythmias, and also for side effects. Amiodarone exhibits a broad spectrum of antiarrhythm
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Buchteile zum Thema "Antiarrhythmic effect"

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Massarella, J. W., and K. C. Khoo. "Effect of Age on the Clinical Pharmacokinetics of Antiarrhythmic Drugs." In Drug Studies in the Elderly. Springer US, 1986. http://dx.doi.org/10.1007/978-1-4684-1253-6_11.

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Campbell, Ronald W. F. "Sudden Cardiac Death — Failure or Effect of Antiarrhythmic Drug Therapy?" In Cardiac Arrhythmias: New Therapeutic Drugs and Devices. Springer US, 1985. http://dx.doi.org/10.1007/978-1-4613-2595-6_12.

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Arad, Michael, Tatiana Oxman, Ron Leor, and Babeth Rabinowitz. "Protaglandins and the antiarrhythmic effect of preconditioning in the isolated rat heart." In Biochemical Mechanisms in Heart Function. Springer US, 1996. http://dx.doi.org/10.1007/978-1-4613-1279-6_32.

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Jennings, Robert B., and Keith A. Reimer. "Measurement of Infarct Size: Effect of Reperfusion with Arterial Blood." In Risk/Benefit Analysis for the Use and Approval of Thrombolytic, Antiarrhythmic, and Hypolipidemic Agents. Springer US, 1989. http://dx.doi.org/10.1007/978-1-4613-1605-3_1.

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Honerjäger, P. "The contribution of Na channel block to the negative inotropic effect of antiarrhythmic drugs." In Controversial issues in cardiac pathophysiology. Steinkopff, 1986. http://dx.doi.org/10.1007/978-3-662-11374-5_4.

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Raasmaja, Atso, Antti Talo, Heimo Haikala, Erkki Nissinen, Inge-Britt Lindén, and Pentti Pohto. "Biochemical Properties of OR-1259 - A Positive Inotropic and Vasodilatory Compound with an Antiarrhythmic Effect." In Excitation-Contraction Coupling in Skeletal, Cardiac, and Smooth Muscle. Springer US, 1992. http://dx.doi.org/10.1007/978-1-4615-3362-7_63.

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Shenasa, M., W. Haverkamp, H. Shenasa, et al. "Electrophysiologic Effects, Clinical Efficacy and Antiarrhythmic and Proarrhythmic Effects of Sodium Channel Blockers in Patients with Ventricular Tachyarrhythmias." In Antiarrhythmic Drugs. Springer Berlin Heidelberg, 1995. http://dx.doi.org/10.1007/978-3-642-85624-2_16.

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Herzig, S., and H. Lüllmann. "Effects of Cardiac Glycosides at the Cellular Level." In Antiarrhythmic Drugs. Springer Berlin Heidelberg, 1989. http://dx.doi.org/10.1007/978-3-642-73666-7_25.

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Hindricks, G., H. Kottkamp, W. Haverkamp, et al. "Magnesium — Electrophysiological Effects, Antiarrhythmic Properties and Clinical Applications." In Antiarrhythmic Drugs. Springer Berlin Heidelberg, 1995. http://dx.doi.org/10.1007/978-3-642-85624-2_19.

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Cobbe, S. M. "Antiarrhythmic Versus Proarrhythmic Effects of Class III Agents." In Antiarrhythmic Drugs. Springer Berlin Heidelberg, 1995. http://dx.doi.org/10.1007/978-3-642-85624-2_17.

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Konferenzberichte zum Thema "Antiarrhythmic effect"

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Liang, "Cuiping, Zhiyang He, Cunjin Luo, Qince Li, Jun Liu, and Suiping Jiang." "A simulation study on the effect of antiarrhythmic drugs during myocardial infarction." In 2022 Computing in Cardiology Conference. Computing in Cardiology, 2022. http://dx.doi.org/10.22489/cinc.2022.074.

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Sinitsyna, A. P., A. K. Berezhnoy, M. M. Slotvitsky, et al. "INVESTIGATION OF THE EFFECT OF FIBROTIC LESIONS IN THE ATRIAL TISSUE MODEL IN TERMS OF ELECTROPHYSIOLOGICAL CHARACTERISTICS OF THE PATIENT’S CARDIOMYOCYTES." In XI МЕЖДУНАРОДНАЯ КОНФЕРЕНЦИЯ МОЛОДЫХ УЧЕНЫХ: БИОИНФОРМАТИКОВ, БИОТЕХНОЛОГОВ, БИОФИЗИКОВ, ВИРУСОЛОГОВ, МОЛЕКУЛЯРНЫХ БИОЛОГОВ И СПЕЦИАЛИСТОВ ФУНДАМЕНТАЛЬНОЙ МЕДИЦИНЫ. IPC NSU, 2024. https://doi.org/10.25205/978-5-4437-1691-6-158.

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Atrial fibrillation (AF) is one of the most dangerous cardiovascular diseases. AF therapy with medications often proves ineffective in the long-term course of the disease due to changes in the electrophysiology of atrial cells. In this work, we studied the arrhythmogenicity of atrial tissue with fibrous regions depending on the electrophysiological properties of the patient’s cardiomyocytes, including under the action of an antiarrhythmic drug.
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SUZUKI, MAKOTO, MITSUHIRO NISHIZAKI, TETSUO ARAKAWA, et al. "THE SUPPRESSIVE EFFECT OF BEPRIDIL ON ATRIAL FLUTTER ORGANIZED FROM PERSISTENT ATRIAL FIBRILLATION DURING CLASS IC ANTIARRHYTHMIC THERAPY." In Proceedings of the 31st International Congress on Electrocardiology. WORLD SCIENTIFIC, 2005. http://dx.doi.org/10.1142/9789812702234_0084.

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Saiz, J., J. M. Ferrero, M. Monserrat, J. Gomis-Tena, J. Chorro, and A. Ferrero. "Effects of the antiarrhythmic drug dofetilide on myocardial electrical activity: a computer modelling study." In Computers in Cardiology, 2003. IEEE, 2003. http://dx.doi.org/10.1109/cic.2003.1291148.

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Abbasi, Mitra, and Sebastian Polak. "In Silico Assessment of Antiarrhythmic Effects of Drug Ranolazine on Electrical Activity in Human Ventricular Myocardium." In 2016 Computing in Cardiology Conference. Computing in Cardiology, 2016. http://dx.doi.org/10.22489/cinc.2016.309-284.

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ISHII, KUNIAKI, KAZUE NAKASHIMA, and MASAO ENDOH. "EFFECTS OF ANTIARRHYTHMIC DRUGS ON THE CURRENTS OF XENOPUS OOCYTES EXPRESSING HERG AND KvLQT1/minK CHANNELS." In Proceedings of the 31st International Congress on Electrocardiology. WORLD SCIENTIFIC, 2005. http://dx.doi.org/10.1142/9789812702234_0068.

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Saiz, J., M. Monserrat, J. M. Ferrero, et al. "Effects of the antiarrhythmic drug dofetilide on regional heterogeneity of action potential duration: a computer modelling study." In Computers in Cardiology 2004. IEEE, 2004. http://dx.doi.org/10.1109/cic.2004.1442928.

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Tsvelaya, V. A., A. K. Berezhnoy, A. P. Sinitsyna, I. S. Semidetnov, V. D. Naumov, and M. M. Slotvitsky. "ADVANCED CARDIOVASCULAR TOXICITY SCREENING: INTEGRATING HUMAN IPSC-DERIVED CARDIOMYOCYTES WITH IN SILICO MODELS." In XI МЕЖДУНАРОДНАЯ КОНФЕРЕНЦИЯ МОЛОДЫХ УЧЕНЫХ: БИОИНФОРМАТИКОВ, БИОТЕХНОЛОГОВ, БИОФИЗИКОВ, ВИРУСОЛОГОВ, МОЛЕКУЛЯРНЫХ БИОЛОГОВ И СПЕЦИАЛИСТОВ ФУНДАМЕНТАЛЬНОЙ МЕДИЦИНЫ. IPC NSU, 2024. https://doi.org/10.25205/978-5-4437-1691-6-168.

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The pharmaceutical industry is advancing by utilizing hiPSC-derived cardiomyocytes (hiPSC-CM) for in vitro cardiac safety screening. The traditional reliance on QT interval prolongation as the primary marker for cardiotoxicity is being questioned. The Comprehensive In Vitro Proarrhythmia Assay (CiPA) initiative suggests incorporating computer modeling and in silico platforms for a more comprehensive approach to cardiotoxicity testing, alongside hiPSC-CM in vitro screening. Our study introduces an innovative platform that combines in vitro hiPSC-CM propagation testing with in silico models to e
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Beatch, Dickenson, Wood, and Tang. "An Automated, On-Line, PC-based System For Analysis Of The Frequency dependent Effects Of Antiarrhythmic Drugs On Action Potential Duration, Refractory Periods, And Conduction Velocity, And On Energy Requirements For Defibrillation." In Proceedings of the Annual International Conference of the IEEE Engineering in Medicine and Biology Society. IEEE, 1992. http://dx.doi.org/10.1109/iembs.1992.595870.

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10

Beateti, Gregory N., David R. Dickenson, Randall H. Wood, and Anthony S. L. Tang. "An automated, on-line, PC-based system for analysis of the frequency-dependent effects of antiarrhythmic drugs on action potential duration, refractory periods, and conduction velocity, and on energy requirements for defibrillation." In 1992 14th Annual International Conference of the IEEE Engineering in Medicine and Biology Society. IEEE, 1992. http://dx.doi.org/10.1109/iembs.1992.5761256.

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