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Thematische Bibliographien / Anticancer chemotherapy / Zeitschriftenartikel

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Zeitschriftenartikel zum Thema „Anticancer chemotherapy“

Autor: Grafiati

Veröffentlicht am 28. Juni 2021

Zuletzt aktualisiert am 1. Februar 2022

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1

Hayashi, S., S. Maruoka, N. Takahashi, and S. Hashimoto. "Carotidynia after anticancer chemotherapy." Singapore Medical Journal 55, no. 09 (2014): e142-e144. http://dx.doi.org/10.11622/smedj.2014127.

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2

Kvolik, S., L. Glavas-Obrovac, K. Sakic, D. Margaretic, and I. Karner. "Anaesthetic implications of anticancer chemotherapy." European Journal of Anaesthesiology 20, no. 11 (2005): 859–71. http://dx.doi.org/10.1017/s026502150300139x.

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3

Kvolik, S., L. Glavas-Obrovac, K. Sakic, D. Margaretic, and I. Karner. "Anaesthetic implications of anticancer chemotherapy." European Journal of Anaesthesiology 20, no. 11 (2003): 859–71. http://dx.doi.org/10.1097/00003643-200311000-00002.

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4

F. Goncalves, R., G. Kriechammer, and M. P.M. Marques. "MGBG in Combined Anticancer Chemotherapy." Letters in Drug Design & Discovery 8, no. 10 (2011): 897–903. http://dx.doi.org/10.2174/157018011797655197.

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5

Kratz, Felix, Ivonne A Müller, Claudia Ryppa, and André Warnecke. "Prodrug Strategies in Anticancer Chemotherapy." ChemMedChem 3, no. 1 (2008): 20–53. http://dx.doi.org/10.1002/cmdc.200700159.

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6

Kris, M. G., Fausto Roila, Pieter H. M. De Mulder, and Michel Marty. "Delayed emesis following anticancer chemotherapy." Supportive Care in Cancer 6, no. 3 (1998): 228–32. http://dx.doi.org/10.1007/s005200050158.

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7

Kris, M. G., K. M. W. Pisters, and L. Hinkley. "Delayed emesis following anticancer chemotherapy." Supportive Care in Cancer 2, no. 5 (1994): 297–300. http://dx.doi.org/10.1007/bf00365581.

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8

Hennes, Emily R., Michael Reed, Mary Mably, et al. "Implementation of a chemotherapy stewardship process." American Journal of Health-System Pharmacy 77, no. 15 (2020): 1243–48. http://dx.doi.org/10.1093/ajhp/zxaa157.

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Abstract Purpose To design and implement a chemotherapy stewardship process to optimize the location of chemotherapy administration in an effort to decrease the number of inappropriate inpatient anticancer regimen administrations and decrease institutional costs associated with inpatient administration. Summary As the costs of anticancer agents continue to rise, it is crucial that multidisciplinary efforts are aimed at managing anticancer medication utilization; this is especially important for high-cost medications, medications whose use requires increased monitoring due to safety concerns, a

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9

Ben Kridis, W., A. Khanfir, and M. Frikha. "ACUTE PANCREATITIS INDUCED BY ANTICANCER CHEMOTHERAPY." Acta Clinica Belgica 68, no. 4 (2013): 309–10. http://dx.doi.org/10.2143/acb.3351.

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10

Ma, Yuting, Oliver Kepp, François Ghiringhelli, et al. "Chemotherapy and radiotherapy: Cryptic anticancer vaccines." Seminars in Immunology 22, no. 3 (2010): 113–24. http://dx.doi.org/10.1016/j.smim.2010.03.001.

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11

Popruzhenko, Т. V., and S. P. Boris. "Salivation in children during anticancer chemotherapy." Stomatologiya 95, no. 2 (2016): 30. http://dx.doi.org/10.17116/stomat201695230-33.

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12

Bacherikov, V. A. "ANTIFOLATES FOR ANTICANCER CHEMOTHERAPY. PART IІ". Odesa National University Herald. Chemistry 18, № 4(48) (2015): 26. http://dx.doi.org/10.18524/2304-0947.2013.4(48).37019.

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13

Locher, Clara, Rosa Conforti, Laetitia Aymeric, et al. "Desirable cell death during anticancer chemotherapy." Annals of the New York Academy of Sciences 1209, no. 1 (2010): 99–108. http://dx.doi.org/10.1111/j.1749-6632.2010.05763.x.

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14

Kumagai, Kazuhide, Kouji Shimizu, Kouki Masuo, Kennichi Yamagata, and Takayuki Tanaka. "Anticancer Effects ofPreoperative Chemotherapy onColorectal Carcinoma." Digestive Surgery 15, no. 4 (1998): 337–41. http://dx.doi.org/10.1159/000018649.

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15

Hryvkova, L. V. "Anticancer chemotherapy with high-dose methotrexate." Practical oncology 4, no. 1 (2021): 30–38. http://dx.doi.org/10.22141/2663-3272.4.1.2021.229869.

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The literature review is devoted to the use of high doses of methotrexate in oncology. There is a brief historical overview of the use of methotrexate for the treatment of malignant neoplasms; the main mechanism of its cytotoxic effect on tumor cells, the development of resistance to methotrexate, the main indications for the use of high doses of methotrexate and the main side effects of this treatment are considered. The review details the results of using high doses of methotrexate in the treatment of patients with osteosarcoma, non-Hodgkin’s lymphomas, located mainly in the central nervous

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16

Yu, Yue, Masahiro Nishikawa, Ming Liu, et al. "Self-assembled nanodiamond supraparticles for anticancer chemotherapy." Nanoscale 10, no. 19 (2018): 8969–78. http://dx.doi.org/10.1039/c8nr00641e.

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Chemically functionalized nanodiamonds (NDs) were transformed into supraparticle (SP) nanoclusters via self-assembly. The ND–SP nanoclusters were biocompatible and internalized by cancer cells, and markedly enhanced anticancer drug efficacy compared to conventional nanomedicines.

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17

Shurin, Michael R. "Dual role of immunomodulation by anticancer chemotherapy." Nature Medicine 19, no. 1 (2013): 20–22. http://dx.doi.org/10.1038/nm.3045.

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18

Ben-Aharon, Irit, and Ruth Shalgi. "What lies behind chemotherapy-induced ovarian toxicity?" REPRODUCTION 144, no. 2 (2012): 153–63. http://dx.doi.org/10.1530/rep-12-0121.

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Seminal advances in anticancer therapy as well as supportive care strategies have led to improved survival rates, posing an emphasis on preserving an optimum quality of life after cancer treatment. This recognition has paved the way to an increasing research of long-term side effects, both clinical and preclinical and to an ongoing design of a supportive care system to evaluate and treat long-term adverse effects of anticancer treatments, including the impact on fertility. As with many adverse effects induced by anticancer treatments, the literature comprised mostly clinical data with regard t

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19

Shih, Ya-Chen Tina, Fabrice Smieliauskas, Daniel M. Geynisman, Ronan J. Kelly, and Thomas J. Smith. "Trends in the Cost and Use of Targeted Cancer Therapies for the Privately Insured Nonelderly: 2001 to 2011." Journal of Clinical Oncology 33, no. 19 (2015): 2190–96. http://dx.doi.org/10.1200/jco.2014.58.2320.

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Purpose This study sought to define and identify drivers of trends in cost and use of targeted therapeutics among privately insured nonelderly patients with cancer receiving chemotherapy between 2001 and 2011. Methods We classified oncology drugs as targeted oral anticancer medications, targeted intravenous anticancer medications, and all others. Using the LifeLink Health Plan Claims Database, we studied and disaggregated trends in use and in insurance and out-of-pocket payments per patient per month and during the first year of chemotherapy. Results We found a large increase in the use of tar

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Li, Fan, Xinqing Fu, Qingqing Huo, and Wantao Chen. "Research Progress on the Nano-Delivery Systems of Antitumor Drugs." Nano LIFE 10, no. 01n02 (2020): 2040006. http://dx.doi.org/10.1142/s1793984420400061.

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To date, chemotherapy, the main treatment for malignant tumors, still fails to provide ideal therapeutic efficacy, which is deeply rooted in various physiological barriers, either temporal or spatial, to the delivering of anticancer drugs to solid tumor sites during chemotherapy. In the meantime, the therapeutic efficacy of anticancer drugs is affected by inherent cancer characteristics, drug transport, cellular uptake and other complex interactions. Recently, advances have been constantly achieved on nanoscale drug delivery systems (NDDSs) for anticancer drug delivery, driven by their excelle

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21

Xu, Tengyan, Chunhui Liang, Debin Zheng, et al. "Nuclear delivery of dual anticancer drug-based nanomedicine constructed by cisplatinum-induced peptide self-assembly." Nanoscale 12, no. 28 (2020): 15275–82. http://dx.doi.org/10.1039/d0nr00143k.

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22

Tiligada, E. "Chemotherapy: induction of stress responses." Endocrine-Related Cancer 13, Supplement_1 (2006): S115—S124. http://dx.doi.org/10.1677/erc.1.01272.

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Eukaryotic cells, from yeast to mammals, respond and adapt to environmental and microenvironmental stressors by evolutionary conserved multicomponent endogenous systems that utilise a network of signal transduction pathways to regulate the adaptive and protective phenotype. The balance between cell survival and cell death is decisive for sensitivity or resistance to DNA-damaging chemotherapeutic agents. Anticancer drugs may themselves act as stressors to induce adaptive signals that could limit their clinical value. Related research has been focused on the modulation of the expression and func

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23

Kaufmann, S. H., and R. Hancock. "Topoisomerase II as a target for anticancer chemotherapy." Acta Biochimica Polonica 42, no. 4 (1995): 381–93. http://dx.doi.org/10.18388/abp.1995_4892.

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Type II DNA topoisomerases are required for the segregation of genomic DNA at cell division in prokaryotic and eukaryotic cells, and inhibitors of these enzymes are potential cytotoxic agents in both prokaryotes and eukaryotes. The bacterial member of the topoisomerase II family, DNA gyrase, and the chemotherapeutic agents which target it are the subject of a recent review (Maxwell, A. et al., 1993, in Molecular Biology of DNA Topoisomerases, Andoh, T. et al., eds., pp. 21-30, CRC Press, Boca Raton). Here we present an overview of current knowledge of eukaryotic topoisomerase II and the antica

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Bansal, DivyaBhargava, Dipankar Bhadra, Umesh Gupta, and NK Jain. "PEGylated methotrexate based micellar conjugates for anticancer chemotherapy." Asian Journal of Pharmaceutics 9, no. 1 (2015): 60. http://dx.doi.org/10.4103/0973-8398.150042.

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25

Klinkert, M., and V. Heussler. "The Use of Anticancer Drugs in Antiparasitic Chemotherapy." Mini-Reviews in Medicinal Chemistry 6, no. 2 (2006): 131–43. http://dx.doi.org/10.2174/138955706775475939.

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26

Wang, Xiaoyong, and Zijian Guo. "The Role of Sulfur in Platinum Anticancer Chemotherapy." Anti-Cancer Agents in Medicinal Chemistry 7, no. 1 (2007): 19–34. http://dx.doi.org/10.2174/187152007779314062.

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27

Ploylearmsaeng, Su-arpa, Uwe Fuhr, and Alexander Jetter. "How may Anticancer Chemotherapy with Fluorouracil be Individualised?" Clinical Pharmacokinetics 45, no. 6 (2006): 567–92. http://dx.doi.org/10.2165/00003088-200645060-00002.

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28

Maeta, Michio, Kiyoaki Mizusawa, and Shigemasa Koga. "Induction of diffuse necrotizing enterocolitis by anticancer chemotherapy." Gastroenterologia Japonica 22, no. 3 (1987): 370–73. http://dx.doi.org/10.1007/bf02774263.

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29

Han, Xiang Y., and Jeffrey J. Tarrand. "Moraxella osloensisBlood and Catheter Infections During Anticancer Chemotherapy." American Journal of Clinical Pathology 121, no. 4 (2004): 581–87. http://dx.doi.org/10.1309/qbb3avcmgwa3k1xk.

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30

Shi, Huayun, Cinzia Imberti, and Peter J. Sadler. "Diazido platinum(iv) complexes for photoactivated anticancer chemotherapy." Inorganic Chemistry Frontiers 6, no. 7 (2019): 1623–38. http://dx.doi.org/10.1039/c9qi00288j.

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31

Zaffaroni, N., G. Fiorentini, and U. De Giorgi. "Hyperthermia and hypoxia: new developments in anticancer chemotherapy." European Journal of Surgical Oncology (EJSO) 27, no. 4 (2001): 340–42. http://dx.doi.org/10.1053/ejso.2000.1040.

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32

Robaey, Philippe, Maja Krajinovic, Sophie Marcoux, and Albert Moghrabi. "Pharmacogenetics of the neurodevelopmental impact of anticancer chemotherapy." Developmental Disabilities Research Reviews 14, no. 3 (2008): 211–20. http://dx.doi.org/10.1002/ddrr.29.

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33

Zunino, Barbara, and Jean-Ehrland Ricci. "Hyperthermic intra-peritoneal chemotherapy and anticancer immune response." OncoImmunology 5, no. 1 (2015): e1060392. http://dx.doi.org/10.1080/2162402x.2015.1060392.

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34

Fulda, S., and K.-M. Debatin. "Extrinsic versus intrinsic apoptosis pathways in anticancer chemotherapy." Oncogene 25, no. 34 (2006): 4798–811. http://dx.doi.org/10.1038/sj.onc.1209608.

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35

Costantini, P. "Mitochondrion as a Novel Target of Anticancer Chemotherapy." Journal of the National Cancer Institute 92, no. 13 (2000): 1042–53. http://dx.doi.org/10.1093/jnci/92.13.1042.

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36

Potanovich, Lisa M., Katherine M. W. Pisters, Mark G. Kris, et al. "Midazolam in patients receiving anticancer chemotherapy and antiemetics." Journal of Pain and Symptom Management 8, no. 8 (1993): 519–24. http://dx.doi.org/10.1016/0885-3924(93)90080-f.

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37

Ranchon, Florence, Céline Moch, Benoît You, et al. "Predictors of prescription errors involving anticancer chemotherapy agents." European Journal of Cancer 48, no. 8 (2012): 1192–99. http://dx.doi.org/10.1016/j.ejca.2011.12.031.

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38

Mazzei, Teresita, and Enrico Mini. "Clinical application of biochemical modulation in anticancer chemotherapy." Pharmacological Research 26 (September 1992): 225. http://dx.doi.org/10.1016/1043-6618(92)91160-i.

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39

Basilotta, Rossella, Deborah Mannino, Alessia Filippone, et al. "Role of Calixarene in Chemotherapy Delivery Strategies." Molecules 26, no. 13 (2021): 3963. http://dx.doi.org/10.3390/molecules26133963.

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Since cancer is a multifactorial disease with a high mortality rate, the study of new therapeutic strategies is one of the main objectives in modern research. Numerous chemotherapeutic agents, although widely used, have the disadvantage of being not very soluble in water or selective towards cancerous cells, with consequent side effects. Therefore, in recent years, a greater interest has emerged in innovative drug delivery systems (DDSs) such as calixarene, a third-generation supramolecular compound. Calixarene and its water-soluble derivatives show good biocompatibility and have low cytotoxic

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40

Cao, Shuwen, Chunhao Lin, Shunung Liang, Chee Hwee Tan, Phei Er Saw, and Xiaoding Xu. "Enhancing Chemotherapy by RNA Interference." BIO Integration 1, no. 2 (2020): 64–81. http://dx.doi.org/10.15212/bioi-2020-0003.

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Abstract Small interfering RNA (siRNA) has shown tremendous potential for treating human diseases in the past decades. siRNA can selectively silence a pathological pathway through the targeting and degradation of a specific mRNA, significantly reducing the off-target side effects of anticancer drugs. However, the poor pharmacokinetics of RNA significantly restricted the clinical use of RNAi technology. In this review, we examine in-depth the siRNA therapeutics currently in preclinical and clinical trials, multiple challenges faced in siRNA therapy, feasibility of siRNA treatment with anticance

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41

Maor, Y., and S. Malnick. "Liver Injury Induced by Anticancer Chemotherapy and Radiation Therapy." International Journal of Hepatology 2013 (2013): 1–8. http://dx.doi.org/10.1155/2013/815105.

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Cytotoxic chemotherapy prolongs survival of patients with advanced and metastatic tumors. This is, however, a double-edged sword with many adverse effects. Since the liver has a rich blood supply and plays an active role in the metabolism of medications, it is not surprising that there can be hepatic injury related to chemotherapy. In addition, radioembolization may affect the parenchyma of normal and cirrhotic livers. We review chemotherapy-associated liver injury in patients with colorectal liver metastases, including downsizing chemotherapy and neoadjuvant chemotherapy. We discuss the mecha

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42

Savage, P., C. Holloway, G. Lindsay, et al. "Cancer referral and treatment activity 2010–2015: a population-based study from Vancouver Island." Current Oncology 23, no. 6 (2016): 626. http://dx.doi.org/10.3747/co.23.3306.

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Introduction The years since 2005 have seen major changes in cancer treatment and significant increases in the number of anticancer drugs available. However, there are relatively few published data to reflect how those changes are affecting the activity and workload of oncology services. To explore the effects of those changes, we reviewed the population-based cancer treatment activity on Vancouver Island for the period 2010–2015.Methods Information about new patient referrals, radiation courses, new chemotherapy cycles commenced, total intravenous (IV) chemotherapy treatment visits, and pharm

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43

Yin, Feng, Bobo Gu, Jingxu Li, et al. "In vitro anticancer activity of AIEgens." Biomaterials Science 7, no. 9 (2019): 3855–65. http://dx.doi.org/10.1039/c9bm00881k.

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AIEgens possess superior cytotoxicity, tumor invasion, and hemolysis against cancer cells and cancer stem cells. Simple structural modifications enable them as highly biocompatible, image-guided chemotherapy agents.

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44

Wang, Yan, Wei Xie, Juliette Humeau, et al. "Autophagy induction by thiostrepton improves the efficacy of immunogenic chemotherapy." Journal for ImmunoTherapy of Cancer 8, no. 1 (2020): e000462. http://dx.doi.org/10.1136/jitc-2019-000462.

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BackgroundImmunogenic cell death (ICD) is a peculiar modality of cellular demise that elicits adaptive immune responses and triggers T cell-dependent immunity.MethodsFluorescent biosensors were employed for an unbiased drug screen approach aiming at the identification of ICD enhancers.ResultsHere, we discovered thiostrepton as an enhancer of ICD able to boost chemotherapy-induced ATP release, calreticulin exposure and high-mobility group box 1 exodus. Moreover, thiostrepton enhanced anticancer immune responses of oxaliplatin (OXA) in vivo in immunocompetent mice, yet failed to do so in immunod

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45

Doundoua, D. P., A. V. Staferov, A. V. Sorokin, and A. G. Kedrova. "CARDIOONCOLOGY: CHEMOTHERAPY DRUGS AND RADIATION THERAPY IMPACT ON CARDIOVASCULAR SYSTEM." Journal of Clinical Practice 7, no. 4 (2016): 41–48. http://dx.doi.org/10.17816/clinpract7441-48.

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Anticancer therapy can cause angina pectoris, acute coronary syndrome, stroke, critical limb ischemia, arterial hypertension, arrhythmias and heart failure. The new specialization in cardiology, called cardiooncology, exploring the complications of cardiovascular system, arising during the treatment of cancer. The first part of the review, based on the publications of the last decades, is dedicated to the definition of cardiooncology, mechanisms of cardiac and vascular toxicity with a number of anticancer drugs affecting the cardiovascular system.

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46

Jiang, Zhi-Gang, Steven A. Fuller, and Hossein A. Ghanbari. "PAN-811 Blocks Chemotherapy Drug-InducedIn VitroNeurotoxicity, While Not Affecting Suppression of Cancer Cell Growth." Oxidative Medicine and Cellular Longevity 2016 (2016): 1–11. http://dx.doi.org/10.1155/2016/9392404.

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Chemotherapy often results in cognitive impairment, and no neuroprotective drug is now available. This study aimed to understand underlying neurotoxicological mechanisms of anticancer drugs and to evaluate neuroprotective effects of PAN-811. Primary neurons in different concentrations of antioxidants (AOs) were insulted for 3 days with methotrexate (MTX), 5-fluorouracil (5-FU), or cisplatin (CDDP) in the absence or presence of PAN-811·Cl·H2O. The effect of PAN-811 on the anticancer activity of tested drugs was also examined using mouse and human cancer cells (BNLT3 and H460) to assess any nega

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47

Sheng Sow, Heng, and Stephen R. Mattarollo. "Combining low-dose or metronomic chemotherapy with anticancer vaccines." OncoImmunology 2, no. 12 (2013): e27058. http://dx.doi.org/10.4161/onci.27058.

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48

Gohji, Kazuo, and Sadao Kamidono. "STUDY OF ANTICANCER CHEMOTHERAPY FOR HUMAN RENAL CELL CARCINOMA." Japanese Journal of Urology 78, no. 6 (1987): 982–90. http://dx.doi.org/10.5980/jpnjurol1928.78.6_982.

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49

Focher, F., and S. Spadari. "Thymidine Phosphorylase: A Two-Face Janus in Anticancer Chemotherapy." Current Cancer Drug Targets 1, no. 2 (2001): 141–53. http://dx.doi.org/10.2174/1568009013334232.

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50

Nakashima, Makoto, Takuya Goto, Sachika Maeta, et al. "Development of Anticancer Chemotherapy Support Software and Its Evaluation." Iryo Yakugaku (Japanese Journal of Pharmaceutical Health Care and Sciences) 37, no. 12 (2011): 721–27. http://dx.doi.org/10.5649/jjphcs.37.721.

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