Thematische Bibliographien / Autoimmune and idiopathic organic disease

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Auswahl der wissenschaftlichen Literatur zum Thema „Autoimmune and idiopathic organic disease“

Autor: Grafiati

Veröffentlicht am 6. Oktober 2023

Zuletzt aktualisiert am 31. Juli 2025

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Zeitschriftenartikel zum Thema "Autoimmune and idiopathic organic disease"

Krug, Susanne M. "Solving the Puzzle: Molecular Research in Inflammatory Bowel Diseases." International Journal of Molecular Sciences 24, no. 17 (2023): 13389. http://dx.doi.org/10.3390/ijms241713389.

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Inflammatory bowel disease (IBD) encompasses chronic idiopathic relapsing and remitting gastrointestinal autoimmune diseases characterized by chronic inflammatory disorders of complex etiology, posing clinical challenges due to their often therapy-refractory nature [...]

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Keda, YM, IV Krjukova, IA Ilovaiskaia, et al. "Antibodies to pituitary surface antigens during various pituitary disease states." Journal of Endocrinology 175, no. 2 (2002): 417–23. http://dx.doi.org/10.1677/joe.0.1750417.

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Autoantibodies to cell surface antigens of human somatotropinoma (ASAS), human prolactinoma (ASAP) and rat adenohypophysis (ASARA) were assayed in the serum of patients with pituitary diseases associated with GH deficiency (GHD), such as pituitary dwarfism and primary empty sella syndrome (ESS), and in the serum of patients with hyperprolactinaemia of different etiologies: idiopathic hyperprolactinaemia, prolactinoma and ESS. The investigation was carried out with a cellular variant of an ELISA. Among children with GHD, the highest percentage of antibody-positive patients was found in the grou

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Meunier, Lucy, and Dominique Larrey. "Hepatotoxicity of Drugs Used in Multiple Sclerosis, Diagnostic Challenge, and the Role of HLA Genotype Susceptibility." International Journal of Molecular Sciences 24, no. 1 (2023): 852. http://dx.doi.org/10.3390/ijms24010852.

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Multiple sclerosis (MS) is a chronic demyelinating disease of the central nervous system and the association with other autoimmune diseases is well-documented. There are many therapeutic options for the treatment of MS. Most of the available drugs cause drug-induced liver injury (DILI) to variable extents with heterogeneous clinical and biological manifestations, including liver injury with or without signs of hypersensitivity and autoimmunity. The diagnosis of DILI may be particularly difficult because MS is frequently associated with idiopathic autoimmune hepatitis. Recent advances suggest t

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Fukushima, Kiyoharu, Kazuyuki Tsujino, Shinji Futami, and Hiroshi Kida. "Natural Autoantibodies in Chronic Pulmonary Diseases." International Journal of Molecular Sciences 21, no. 3 (2020): 1138. http://dx.doi.org/10.3390/ijms21031138.

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In autoantibody-mediated autoimmune diseases, pathogenic autoantibodies generated by a failure of central or peripheral tolerance, have different effects mediated by a variety of mechanisms. Interestingly, even non-autoimmune chronic diseases have a set of disease-specific natural autoantibodies that are maintained for a long time. Because most of these natural autoantibodies target intracellular proteins or long non-coding RNAs, they are speculated to be non-pathological and have some important as yet unrecognized physiological functions such as debris clearance. Recently, we revealed a set o

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Pulito-Cueto, Verónica, Fernanda Genre, Raquel López-Mejías, et al. "Endothelin-1 as a Biomarker of Idiopathic Pulmonary Fibrosis and Interstitial Lung Disease Associated with Autoimmune Diseases." International Journal of Molecular Sciences 24, no. 2 (2023): 1275. http://dx.doi.org/10.3390/ijms24021275.

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The aim of this study was to determine the role of endothelin-1 (ET-1), a molecule involved in multiple vascular and fibrosing abnormalities, as a biomarker of interstitial lung disease (ILD), as well as its use for the differential diagnosis between idiopathic pulmonary fibrosis (IPF) and ILD associated with autoimmune diseases (AD-ILD), using a large and well-defined cohort of patients with ILD. A total of 112 patients with IPF, 91 patients with AD-ILD (28 rheumatoid arthritis (RA), 26 systemic sclerosis, 20 idiopathic inflammatory myositis and 17 interstitial pneumonia with autoimmune featu

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Kageyama, Reiko, Tetsuya Honda, and Yoshiki Tokura. "Acquired Idiopathic Generalized Anhidrosis (AIGA) and Its Complications: Implications for AIGA as an Autoimmune Disease." International Journal of Molecular Sciences 22, no. 16 (2021): 8389. http://dx.doi.org/10.3390/ijms22168389.

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Acquired idiopathic generalized anhidrosis (AIGA) is a rare disorder in which systemic anhidrosis/hypohidrosis occurs without causative dermatological, metabolic or neurological disorder. Most cases of AIGA have been reported in Asia, especially in Japan, but there have been only a few reports in Europe and the United States. Severe AIGA may result in heatstroke and can reduce quality of life due to restriction of exercise and outdoor works. AIGA is often accompanied by cholinergic urticaria (CholU), and it is thought that AIGA and CholU with anhidrosis/hypohidrosis belong to the same spectrum

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Ventura, Ignacio, Gemma Clara Meira-Blanco, María Ester Legidos-García, Marcelino Pérez-Bermejo, and María Teresa Murillo-Llorente. "Is There a Link between the Molecular Basis of Juvenile Idiopathic Arthritis and Autoimmune Diseases? Systematic Review." International Journal of Molecular Sciences 25, no. 5 (2024): 2803. http://dx.doi.org/10.3390/ijms25052803.

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Juvenile Idiopathic Arthritis (JIA) is currently the most common chronic rheumatic disease in children. It is known to have no single identity, but a variety of diagnoses. Under-diagnosis is a barrier to early treatment and reduced complications of the disease. Other immune-mediated diseases may coexist in the same patient, making research in this area relevant. The main objective was to analyse whether links could be established between the molecular basis of JIA and other immune-mediated diseases. Early diagnosis may benefit patients with JIA, which in most cases goes undetected, leading to

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Löfdahl, Anna, Göran Tornling, Jenny Wigén, Anna-Karin Larsson-Callerfelt, Christina Wenglén, and Gunilla Westergren-Thorsson. "Pathological Insight into 5-HT2B Receptor Activation in Fibrosing Interstitial Lung Diseases." International Journal of Molecular Sciences 22, no. 1 (2020): 225. http://dx.doi.org/10.3390/ijms22010225.

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Interstitial lung disease (ILD) encompasses a heterogeneous group of more than 200 conditions, of which primarily idiopathic pulmonary fibrosis (IPF), idiopathic nonspecific interstitial pneumonia, hypersensitivity pneumonitis, ILD associated with autoimmune diseases and sarcoidosis may present a progressive fibrosing (PF) phenotype. Despite different aetiology and histopathological patterns, the PF-ILDs have similarities regarding disease mechanisms with self-sustaining fibrosis, which suggests that the diseases may share common pathogenetic pathways. Previous studies show an enhanced activat

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Kim, Ji-Won, Mi-Hyun Ahn, Ju-Yang Jung, Chang-Hee Suh, and Hyoun-Ah Kim. "An Update on the Pathogenic Role of Neutrophils in Systemic Juvenile Idiopathic Arthritis and Adult-Onset Still’s Disease." International Journal of Molecular Sciences 22, no. 23 (2021): 13038. http://dx.doi.org/10.3390/ijms222313038.

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Neutrophils are innate immune phagocytes that play a key role in immune defense against invading pathogens. The main offensive mechanisms of neutrophils are the phagocytosis of pathogens, release of granules, and production of cytokines. The formation of neutrophil extracellular traps (NETs) has been described as a novel defense mechanism in the literature. NETs are a network of fibers assembled from chromatin deoxyribonucleic acid, histones, and neutrophil granule proteins that have the ability to kill pathogens, while they can also cause toxic effects in hosts. Activated neutrophils with NET

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Rawanduzy, Cameron A., Alexander Winkler-Schwartz, and William T. Couldwell. "Hypophysitis: Defining Histopathologic Variants and a Review of Emerging Clinical Causative Entities." International Journal of Molecular Sciences 24, no. 6 (2023): 5917. http://dx.doi.org/10.3390/ijms24065917.

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Inflammatory disease of the pituitary gland is known as hypophysitis. There are multiple histological subtypes, the most common being lymphocytic, and the pathogenesis is variable and diverse. Hypophysitis can be primary and idiopathic or autoimmune related, or secondary to local lesions, systemic disease, medications, and more. Although hypophysitis was previously accepted as an exceedingly rare diagnosis, a greater understanding of the disease process and new insights into possible etiologic sources have contributed to an increased frequency of recognition. This review provides an overview o

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Dissertationen zum Thema "Autoimmune and idiopathic organic disease"

Malichin, Aikaterini. "Jouissance, écriture et nombre dans les maladies auto-immune et idiopathique : l'assomption de la métaphore subjective par l'organisme." Sorbonne Paris Cité, 2015. http://www.theses.fr/2015USPCC317.

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La recherche clinique et bibliographique présente s’articule sur trois points et constitue l’entreprise tripartite de cette thèse doctorale. Le premier concerne l’étude proche de la théorie freudienne et de l'enseignement lacanien et du concept de phénomène psychosomatique, afin d’aborder la maladie organique auto-immune et idiopathique, la Sclérose par Plaques et la maladie de Crohn, sur deux sujets souffrants au sein d'une cure analytique. Le deuxième concerne l’investigation des objets de construction dans leur discours et de leur place occasionnée, et le troisième l’investigation de savoir

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Liley, Albert James. "Statistical co-analysis of high-dimensional association studies." Thesis, University of Cambridge, 2017. https://www.repository.cam.ac.uk/handle/1810/270628.

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Modern medical practice and science involve complex phenotypic definitions. Understanding patterns of association across this range of phenotypes requires co-analysis of high-dimensional association studies in order to characterise shared and distinct elements. In this thesis I address several problems in this area, with a general linking aim of making more efficient use of available data. The main application of these methods is in the analysis of genome-wide association studies (GWAS) and similar studies. Firstly, I developed methodology for a Bayesian conditional false discovery rate (cFDR)

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Alves, André Oliveira. "Polimiosite - Fisiopatologia e Terapêutica." Master's thesis, 2018. http://hdl.handle.net/10316/84581.

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Relatório de Estágio do Mestrado Integrado em Ciências Farmacêuticas apresentado à Faculdade de Farmácia<br>A Polimiosite é uma doença que se inclui no grupo das Miopatias Inflamatórias Idiopáticase cuja etiologia continua desconhecida até ao momento. Contudo, a presença de anticorpose linfócitos T nos músculos, acompanhada por respostas positivas à terapêutica imunológica,constituem evidência de que podemos estar perante uma doença autoimune.As vias imunitárias e não imunitárias são a base do estudo da Polimiosite, sendo que aevolução destes conceitos foi crucial para uma melhor compreensão e

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Bücher zum Thema "Autoimmune and idiopathic organic disease"

Keith, Lierre. The vegetarian myth: Food, justice and sustainability. Flashpoint Press, 2009.

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Bending, David, Kiran Nistala, and Lucy R. Wedderburn. Pathogenesis of juvenile idiopathic arthritis. Oxford University Press, 2013. http://dx.doi.org/10.1093/med/9780199642489.003.0060.

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Although the term juvenile idiopathic arthritis (JIA) encompasses a heterogeneous group of diseases, they all share a common pathological hallmark: inflammation of the synovium. Highly activated T cells, monocytes, and neutrophils are attracted to the joint and secrete mediators that not only perpetuate inflammation but also may attenuate immune regulation. In the oligoarticular and polyarticular forms of JIA, which are thought to be autoimmune conditions, dysregulated adaptive immunity is a likely factor in disease pathogenesis; the nature of the interactions between T effector (Teff) cells a

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Graham, Andrew. Neurological dementias. Oxford University Press, 2013. http://dx.doi.org/10.1093/med/9780199644957.003.0037.

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Dementia in old age is usually due to Alzheimer’s disease, cerebrovascular disease, or mixed pathology. Dementia due to other neurological disorders is uncommon, but important to recognise because management may be very different to that in primary or vascular dementia. This chapter surveys five neurological conditions that may present with dementia in later life: idiopathic normal pressure hydrocephalus (INPH); Huntington’s disease (HD); multiple sclerosis (MS); autoimmune limbic encephalitis (LE); and prion disease. For each disorder the epidemiology, clinical features, investigations &

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Parkes, Joanna E., Simon Rothwell, and Janine A. Lamb. Aetiology and pathogenesis. Edited by Hector Chinoy and Robert Cooper. Oxford University Press, 2018. http://dx.doi.org/10.1093/med/9780198754121.003.0003.

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The aetiology and pathogenesis of idiopathic inflammatory myopathies (IIM) is poorly understood; IIM are thought to result from exposure to environmental factors in genetically susceptible individuals. Both innate and adaptive immune responses are involved in IIM, and there is increasing evidence that non-inflammatory mechanisms play an important role in disease pathology. Several environmental risk factors, including infectious agents, ultraviolet radiation, cigarette smoking, and exposure to statins, have been implicated. Genetic studies have identified the major histocompatibility complex a

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Steensma, David P. Benign Hematology. Oxford University Press, 2012. http://dx.doi.org/10.1093/med/9780199755691.003.0294.

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The major forms of benign hematologic conditions are anemia, neutropenia, transfusion reactions, Gaucher disease, and porphyria. Anemia is a sign of disease rather than a disease itself. Anemia results from 1 or more of 3 pathologic mechanisms: inadequate production of red blood cells (RBCs) by the bone marrow, blood loss, or premature destruction of RBCs. The major causes of neutropenia include hematologic neoplasm, metastatic neoplasm involving the marrow, irradiation, vitamin B12 deficiency and folate deficiency, drugs, infections, congenital or acquired primary disorders of hematopoiesis,

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Forsyth, Rob, and Richard Newton. Specific conditions. Oxford University Press, 2017. http://dx.doi.org/10.1093/med/9780198784449.003.0004.

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This chapter adopts a systematic approach to common diagnoses in paediatric neurology, aetiologies, management to include investigation and treatment, and outcome. For each condition current knowledge on cause and underlying biology is summarized. A rational approach to investigation and treatment is summarized for each topic. These include: acquired brain injury; autoimmune and autoinflammatory disease of the CNS; cerebral palsy and neurodisability which covers feeding, communication, special senses, and respiratory disease; demyelinating disease; epilepsy including its impact on daily life;

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Limaye, Vidya Sadanand. Overview and epidemiology. Edited by Hector Chinoy and Robert Cooper. Oxford University Press, 2018. http://dx.doi.org/10.1093/med/9780198754121.003.0001.

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The term idiopathic inflammatory myopathies (IIM) encompasses a heterogeneous group of muscle-dominant systemic autoimmune syndromes, including polymyositis (PM), dermatomyositis (DM), sporadic inclusion body myositis (sIBM), and immune-mediated necrotizing myopathy (IMNM). The reported incidence of IIM ranges from 5 to 10 × 10–6. Patients with PM, DM, and IMNM characteristically present with the insidious onset of symmetric proximal weakness, while in sIBM the weakness can be asymmetric, and involve the distal upper limbs and quadriceps. Dermatomyositis may also be accompanied by a range of c

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Clunie, Gavin P. R., Nick Wilkinson, Elena Nikiphorou, and Deepak Jadon, eds. Oxford Handbook of Rheumatology. Oxford University Press, 2018. http://dx.doi.org/10.1093/med/9780198728252.001.0001.

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The Oxford Handbook of Rheumatology, 4th edition, has been expanded and improved to incorporate paediatric and adolescent rheumatology. The format of the book is retained. The first four chapters offer a pragmatic guide to evaluating rheumatic and musculoskeletal diseases, showing how a differential diagnosis can be formed on the basis of symptoms, examination, and investigation findings, both for regional musculoskeletal and systemic generalized conditions. Part II comprises chapters on all the major rheumatic and bone diseases and autoimmune connective tissue diseases, such as rheumatoid art

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Chinoy, Hector, and Robert G. Cooper. Polymyositis and dermatomyositis. Oxford University Press, 2013. http://dx.doi.org/10.1093/med/9780199642489.003.0124.

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Polymyositis (PM), dermatomyositis (DM), and inclusion body myositis (IBM) form part of the idiopathic inflammatory myopathies (IIM), a heterogeneous group of rare autoimmune diseases characterized by an acquired proximal muscle weakness, raised muscle enzymes (including creatine kinase), inflammatory cell infiltrates in muscle biopsy tissue, electrophysiological abnormalities, and presence of circulating myositis-specific/myositis-associated autoantibodies. The underlying aetiology of IIM is poorly understood, but likely involves interactions between environmental and genetic risk factors. My

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Gonder, Ulrike, ed. Ethisch Essen mit Fleisch: Eine Streitschrift über nachhaltige und ethische Ernährung mit Fleisch und die Missverständnisse und Risiken einer streng vegetarischen und veganen Lebensweise. Riva, 2021.

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Buchteile zum Thema "Autoimmune and idiopathic organic disease"

Perez-Rodas, Eli Nancy, Jacobo Sellares, and Fernanda Hernandez-Gonzalez. "Idiopathic Interstitial Pneumonias." In Autoimmune Disease Diagnosis. Springer Nature Switzerland, 2024. https://doi.org/10.1007/978-3-031-69895-8_77.

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Lior, Baraf, Levy Yair, Yehuda Shoenfeld, Gabriela Roque González, and Abihai Lucas Hernández. "Idiopathic Aplastic Anemia." In Autoimmune Disease Diagnosis. Springer Nature Switzerland, 2024. https://doi.org/10.1007/978-3-031-69895-8_105.

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Eisenstein, Eli M., and Yackov Berkun. "Juvenile Idiopathic Arthritis." In Autoimmune Disease Diagnosis. Springer Nature Switzerland, 2024. https://doi.org/10.1007/978-3-031-69895-8_4.

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Gartler, Stanley M., R. Scott Hansen, Vinzenz Oji, et al. "Idiopathic Autoimmune Hamolytic." In Encyclopedia of Molecular Mechanisms of Disease. Springer Berlin Heidelberg, 2009. http://dx.doi.org/10.1007/978-3-540-29676-8_7096.

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Froehlich, Stephan J., Carlo A. Lackerbauer, Guenter Rudolph, et al. "Neuromyotonia, Autoimmune and Idiopathic." In Encyclopedia of Molecular Mechanisms of Disease. Springer Berlin Heidelberg, 2009. http://dx.doi.org/10.1007/978-3-540-29676-8_1276.

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Pretis, Nicolo’ de, Yan Bi, Saurabh Mukewar, and Suresh Chari. "Steroid-Responsive Chronic Pancreatitides: Autoimmune Pancreatitis and Idiopathic Duct-Centric Chronic Pancreatitis." In Pancreas and Biliary Disease. Springer International Publishing, 2016. http://dx.doi.org/10.1007/978-3-319-28089-9_5.

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Brodehl, Johannes, and P. F. Hoyer. "Ciclosporin Treatment of Idiopathic Nephrotic Syndrome in Children (Minimal Change Disease and Focal Segmental Glomerulosclerosis)." In Ciclosporin in Autoimmune Diseases. Springer Berlin Heidelberg, 1985. http://dx.doi.org/10.1007/978-3-642-70607-3_64.

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Ebner, Lukas, Helmut Prosch, and Justus E. Roos. "Fibrosing Interstitial Lung Disease." In IDKD Springer Series. Springer Nature Switzerland, 2025. https://doi.org/10.1007/978-3-031-83872-9_8.

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Abstract This chapter provides an overview of fibrosing interstitial lung disease (ILD), a broad group of over 200 lung conditions, some of which can progress to severe, irreversible fibrosis. ILDs can have various causes, ranging from environmental exposures to autoimmune diseases, and while some may be treatable or reversible, others, such as idiopathic pulmonary fibrosis (IPF), lead to significant lung damage and poor outcomes. Lung fibrosis occurs when abnormal tissue repair leads to scarring, impairing the lungs’ ability to function properly. The chapter emphasizes the importance of chest

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Yucelsen, Can. "Approach to Pericarditis." In Thoracic Infections. Nobel Tip Kitabevleri, 2024. http://dx.doi.org/10.69860/nobel.9786053358930.11.

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Pericarditis is the inflammation of the parietal and visceral layers of the pericardium, presenting with characteristic chest pain, electrocardiographic changes, and a pericardial friction rub. It is the most frequent form of pericardial disease and a common cause of chest pain, particularly affecting males between 20-50 years. The etiology includes viral, bacterial, fungal, and parasitic infections, autoimmune diseases, trauma, and other conditions like uremia and hypothyroidism. The pathophysiology involves the activation of the inflammatory cascade leading to pericardial effusion, which can

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Corbridge, Rogan, and Nicholas Steventon. "The inner ear." In Oxford Handbook of ENT and Head and Neck Surgery. Oxford University Press, 2009. http://dx.doi.org/10.1093/med/9780199550791.003.07.

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Structure and function of the inner ear 110 Hearing loss 114 Presbyacusis 116 Noise-induced hearing loss 118 Idiopathic sudden hearing loss 120 Autoimmune ear disease 122 Ototoxicity 124 Hereditary hearing loss 126 Syndromic hearing loss I 128 Syndromic hearing loss II 130 Non-organic hearing loss (NOHL) ...

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Konferenzberichte zum Thema "Autoimmune and idiopathic organic disease"

Romagnoli, Micaela, Carlotta Nannini, Sara Piciucchi, et al. "Idiopathic Nonspecific Interstitial Pneumonia (NSIP): Early Lung Manifestation Of An Autoimmune Disease?" In American Thoracic Society 2010 International Conference, May 14-19, 2010 • New Orleans. American Thoracic Society, 2010. http://dx.doi.org/10.1164/ajrccm-conference.2010.181.1_meetingabstracts.a6002.

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Hayton, Conal, Dayle Terrington, Waqar Ahmed, et al. "Exhaled volatile organic compounds in idiopathic pulmonary fibrosis and disease progression." In ERS International Congress 2020 abstracts. European Respiratory Society, 2020. http://dx.doi.org/10.1183/13993003.congress-2020.727.

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Conticini, E., P. Cameli, C. V. Cotton, et al. "POS0893 PANEL OF SERUM BIOMARKERS FOR DIFFERENTIAL DIAGNOSIS OF IDIOPATHIC INTERSTITIAL LUNG DISEASE AND INTERSTITIAL LUNG DISEASE-SECONDARY TO AUTOIMMUNE RHEUMATIC DISEASE." In EULAR 2024 European Congress of Rheumatology, 12-15 June. Vienna, Austria. BMJ Publishing Group Ltd and European League Against Rheumatism, 2024. http://dx.doi.org/10.1136/annrheumdis-2024-eular.855.

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Remuzgo-Martínez, S., B. Atienza-Mateo, V. Pulito-Cueto, et al. "Endothelin-1 for the differential diagnosis between interstitial lung disease associated with autoimmune diseases and idiopathic pulmonary fibrosis." In ERS International Congress 2022 abstracts. European Respiratory Society, 2022. http://dx.doi.org/10.1183/13993003.congress-2022.2310.

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Leong, J. Y., P. Kumar, S. H. Tay, et al. "POS0156 CD4+HLADR+ T CELLS WITH AUTOIMMUNE REACTIVITY THAT RESIST REGULATORY CONTROL AND PERPETUATE DISEASE INFLAMMATION IN JUVENILE IDIOPATHIC ARTHRITIS." In EULAR 2024 European Congress of Rheumatology, 12-15 June. Vienna, Austria. BMJ Publishing Group Ltd and European League Against Rheumatism, 2024. http://dx.doi.org/10.1136/annrheumdis-2024-eular.4615.

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Vergara, Karen, Silvana Saavedra, Felipe Reyes, Annelise Goecke, Caterina Chesta, and Sebastian Chavez. "AB1109 CHARACTERIZATION OF PATIENTS WITH INTERSTITIAL PNEUMONIA WITH AUTOIMMUNE FEATURES (IPAF) AND ITS COMPARISON WITH PATIENTS WITH SCLERODERMA-RELATED INTERSTITIAL LUNG DISEASE AND WITH IDIOPATHIC FIBROSIS." In Annual European Congress of Rheumatology, EULAR 2019, Madrid, 12–15 June 2019. BMJ Publishing Group Ltd and European League Against Rheumatism, 2019. http://dx.doi.org/10.1136/annrheumdis-2019-eular.7794.

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Moraes, Marianna, Fabiano Abrantes, José Luiz Pedroso, and Orlando Graziani Povoas Barsottini. "Etiological evaluation of hypertrophic pachymeningitis in a tertiary general neurology department." In XIV Congresso Paulista de Neurologia. Zeppelini Editorial e Comunicação, 2023. http://dx.doi.org/10.5327/1516-3180.141s1.665.

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Introduction: Hypertrophic pachymeningitis (HP) is the thickening of the cranial or spinal dura. This process causes cranial nerve palsy, vascular events (stroke and venous thrombosis), and intracranial hypertension. The diagnosis of HP is disclosed by magnetic resonance imaging, showing dural thickening and contrast enhancement. Several etiologies are possible for HP, but infection and autoimmune diseases are the most frequent. The gold standard for diagnosis is the dural biopsy, but cerebrospinal fluid (CSF), blood and other tissues analysis are essential guides to the correct diagnosis. A q

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Santos, João Vitor Ribeiro dos, Mariana Spitz, and Ana Carolina Andorinho. "Stroke secondary to thrombotic microangiopathy." In XIII Congresso Paulista de Neurologia. Zeppelini Editorial e Comunicação, 2021. http://dx.doi.org/10.5327/1516-3180.300.

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Introduction: Thrombotic thrombocytopenic purpura (TTP) is a hematological disease resulting from the ADAMTS 13 plasmatic protein deficit. It can be congenital or sporadic, and is usually autoimmune. Pathological platelet adhesion occurs, leading to microthrombi in capillary and arterial circulation, microangiopathic anemia and ischemia. The clinical picture includes thrombocytopenia, renal dysfunction, fluctuating neurological symptoms, microangiopathic hemolytic anemia, and fever. Methods: Case report of a 51-year-old male hypertensive patient, diagnosed with idiopathic thrombocytopenic purp

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Berichte der Organisationen zum Thema "Autoimmune and idiopathic organic disease"

Zhang, Yingrong, Sanchun Tan, Jieyu Wang, et al. A scoping review protocol of systematic reviews and meta-analyses to acupuncture for the treatment of peripheral facial paralysis. INPLASY - International Platform of Registered Systematic Review and Meta-analysis Protocols, 2022. http://dx.doi.org/10.37766/inplasy2022.3.0084.

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Review question / Objective: To conduct a systematic comprehensive review for Acupuncture treatment of peripheral facial paralysis and to evaluate the efficacy and safety of acupuncture therapy for peripheral facial paralysis. Condition being studied: Peripheral facial paralysis, known as peripheral facial never palsy, includes Bell’s palsy and Ramsay Hunt syndrome.Any medical conditions such as infection, malignancy and autoimmune issues can result it. Idiopathic Bell's palsy is the most common disease causing peripheral facial nerve palsy, which clinical features include unilateral weakness

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