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Thematische Bibliographien / Beta adrenergic receptor blockaders / Dissertationen

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Dissertationen zum Thema „Beta adrenergic receptor blockaders“

Autor: Grafiati

Veröffentlicht am 4. Juni 2021

Zuletzt aktualisiert am 21. Februar 2023

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1

Plank, David Michael. "Calcium dynamics, β-adrenergic receptor blockade, and cardiac function in failing and non-failing hearts". University of Cincinnati / OhioLINK, 2003. http://rave.ohiolink.edu/etdc/view?acc_num=ucin1053431441.

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2

Plank, David M. "Calcium dynamics, [beta]-adrenergic receptor blockade, and cardiac function in failing and non-failing hearts." Cincinnati, Ohio : University of Cincinnati, 2003. http://rave.ohiolink.edu/etdc/view?acc%5Fnum=ucin1053431441.

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3

Grogan, Shawn Patrick, of Western Sydney Hawkesbury University, Faculty of Environmental Management and Agriculture, and School of Agriculture and Rural Development. "Endocrine alteration of meat quality and gene expression in rats and deer." THESIS_FEMA_ARD_Grogan_S.xml, 1998. http://handle.uws.edu.au:8081/1959.7/724.

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Stress activates a number of endocrine pathways that alter an animal's physiology in a manner which can result in undesirable meat quality. Animals frequently exhibit meat quality defects, including ecchymosis, at slaughter due to the stress of slaughter. This thesis explores how stress related hormones interact with adrenergic receptors to alter muscle and vascular physiology. Fallow deer were exposed to either a transciptional regulator (hydrocortisone), a beta adrenergic recptor agonist (clenbuterol) or a beta adrenergic receptor antagonist (propranolol). The administration of hydrocortison

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Grogan, Shawn Patrick. "Endocrine alteration of meat quality and gene expression in rats and deer." Thesis, [Richmond, N.S.W.] : CSIRO Animal Production : School of Agriculture, University of Western Sydney, Hawkesbury, 1998. http://handle.uws.edu.au:8081/1959.7/724.

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Stress activates a number of endocrine pathways that alter an animal's physiology in a manner which can result in undesirable meat quality. Animals frequently exhibit meat quality defects, including ecchymosis, at slaughter due to the stress of slaughter. This thesis explores how stress related hormones interact with adrenergic receptors to alter muscle and vascular physiology. Fallow deer were exposed to either a transciptional regulator (hydrocortisone), a beta adrenergic recptor agonist (clenbuterol) or a beta adrenergic receptor antagonist (propranolol). The administration of hydrocortison

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5

Bagley, John Sorrell. "The beta adrenergic receptor in septic shock." Thesis, University of Aberdeen, 1991. http://ethos.bl.uk/OrderDetails.do?uin=uk.bl.ethos.305466.

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The myocardial depression found during septic shock has been a subject of intense study for many years. Many theories exist as to the precise cause of this condition but in this thesis the role of the β-adrenergic receptor is examined. Some investigators have found reduced response to β-adrenergic stimulation in models of septic shock in animals. This is of interest to the clinician since adrenergic stimulation is a key manipulation in the management of septic shock. An animal model of septic shock was established using a comparatively small dose of <i>Escherichia coli</i> endotoxin infused in

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6

Higgins, Thomas John Diatchenko Luda. "Molecular characterization of [beta]2 adrenergic receptor haplotypes." Chapel Hill, N.C. : University of North Carolina at Chapel Hill, 2008. http://dc.lib.unc.edu/u?/etd,1602.

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Thesis (M.S.)--University of North Carolina at Chapel Hill, 2008.<br>Title from electronic title page (viewed Sep. 16, 2008). "... in partial fulfillment of the requirements for the degree of Master's of Science in the Curriculum in Neurobiology." Discipline: Neurobiology; Department/School: Medicine. On title page, [beta] appers as Greek character and 2 appears in subscript.

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Vincent, Karla Kristine. "Transactivation of Beta 2 Adrenergic Receptor by Bradykinin type 2 Receptor via heterodimerization." Diss., Georgia Institute of Technology, 2009. http://hdl.handle.net/1853/37117.

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Although a long standing convention maintained that G Protein Coupled Receptors (GPCRs) exist in the plasma membrane solely as monomers, substantial work over the last two decades has demonstrated that these ubiquitous receptors can and in many cases, preferentially, exist as homodimers, heterodimers, or higher order oligomers. Often, two GPCRs of the same class heterodimerize; it is less common for two GPCRs of different signaling pathways to interact. The work presented here studied the physical and functional interaction of two GPCRs from discrete classes, the Beta 2 Adrenergic Receptor

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8

Snyder, Ellen Amanda Flood Patrick M. "[beta]2-adrenergic receptor modulation of macrophage inflammatory mediator production." Chapel Hill, N.C. : University of North Carolina at Chapel Hill, 2007. http://dc.lib.unc.edu/u?/etd,1344.

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Thesis (M.S.)--University of North Carolina at Chapel Hill, 2007.<br>Title from electronic title page (viewed Apr. 25, 2008). "... in partial fulfillment of the requirements for the degree of Master of Science in the Curriculum of Oral Biology." Discipline: Oral Biology; Department/School: Dentistry. On title page, [beta] appears as Greek character and 2 appears as subscript.

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9

Robson, Alice. "In vitro reconstitution and stability of a beta-adrenergic receptor." Thesis, University of Bristol, 2005. http://ethos.bl.uk/OrderDetails.do?uin=uk.bl.ethos.424005.

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10

Zolty, Ronald. "Beta 1 and Alpha 2C adrenergic receptor polymorphisms and response to beta blockers in heart failure patients /." Connect to full text via ProQuest. Limited to UCD Anschutz Medical Campus, 2007.

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Thesis (Ph.D. in Clinical Science) -- University of Colorado Denver, 2007.<br>Typescript. Includes bibliographical references (leaves 130-142). Free to UCD affiliates. Online version available via ProQuest Digital Dissertations;

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11

Allcock, Stephen M. J. "Interactions between calpain proteases #beta#-adrenergic receptor and skeletal muscle growth." Thesis, University of Nottingham, 1993. http://ethos.bl.uk/OrderDetails.do?uin=uk.bl.ethos.358286.

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12

Clausen, Lisa. "Effects of beta-2 adrenergic receptor agonists in DOK7 congenital myasthenic syndrome." Thesis, University of Oxford, 2015. https://ora.ox.ac.uk/objects/uuid:9360c51b-8497-47ca-bd16-e917a3614a25.

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Congenital myasthenic syndromes (CMS) are a rare group of heterogeneous disorders, characterised by compromised neuromuscular transmission and symptoms of fatiguable muscle weakness. CMS is caused by mutations in genes that affect the structure and function of the neuromuscular junction (NMJ). In about 20% of CMS cases, patients have mutations in the gene DOK7; the protein product, DOK7, is crucial for maintaining the dense aggregation of acetylcholine receptor (AChR) clusters at the NMJ. DOK7-CMS patients do not respond to treatment with acetylcholinesterase inhibitors which are the first lin

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13

Owens, Helen. "The effect of beta-adrenergic receptor antagonists on the temporal accommodative response." Thesis, Aston University, 1991. http://publications.aston.ac.uk/14611/.

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It is well established that the steady-state accommodative response is characterised by temporal changes in lens power having 2 dominant frequency components: a low frequency component (LFC: < 0.6Hz) and a high frequency component (HFC: 1.0-2.2Hz). This thesis investigates various aspects of these microfluctuations of accommodation. The HFC of accommodative fluctuations was shown to be present in central and peripheral lens zones, although the magnitude of the rms of accommodative microfluctuations was found to be reduced in the lens periphery. These findings concur with the proposal that the

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Clausen, Lisa K. J. "Effects of beta-2 adrenergic receptor agonists in DOK7 congenital myasthenic syndromes." Thesis, University of Oxford, 2015. http://ethos.bl.uk/OrderDetails.do?uin=uk.bl.ethos.712079.

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15

Wardle, Robert L. "Functional antagonism between muscarinic receptor and beta-adrenergic receptor agonists in equine trachealis muscle in vitro /." The Ohio State University, 1994. http://rave.ohiolink.edu/etdc/view?acc_num=osu1487858417981441.

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16

Zheng, Lei. "Synthesis and evaluation of a beta-adrenergic receptor ligand: Fluorine-18 labeled fluorocarazolol." Case Western Reserve University School of Graduate Studies / OhioLINK, 1994. http://rave.ohiolink.edu/etdc/view?acc_num=case1061219721.

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17

Camp, Robert M. "Effect of Chronic Stress Exposure on Beta-adrenergic Receptor Signaling and Fear- Learning." Kent State University / OhioLINK, 2015. http://rave.ohiolink.edu/etdc/view?acc_num=kent1449576849.

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18

Mehta, Ratnavali C. "Pharmacological evaluation of trimetoquinol analogs as affinity ligands for beta-adrenergic receptor systems /." The Ohio State University, 1997. http://rave.ohiolink.edu/etdc/view?acc_num=osu1487945015617968.

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19

Potter, Paul Charles. "In Vitro studies of factors which may influence ligand binding, function, immunogenicity and genetic regulation of the Beta-2 adrenergic receptor in asthma." Doctoral thesis, University of Cape Town, 1991. http://hdl.handle.net/11427/26483.

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This thesis records a series of experiments conducted to gain further insight into factors which influence the expression, ligand binding and functional activity of the beta-2 adrenergic receptor. These studies were prompted by previous reports that the postulated beta-2 adrenergic receptor abnormality in allergic asthma could be induced, induced by autoantibodies. I established and optimised beta-2 adrenergic receptor ligand binding and functional assays in guinea pig lung membranes and then conducted an original study of beta adrenergic receptor expression in the guinea pig foetal lung. I fo

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Yu, Esther Jeong. "Cloning and characterization of a novel β-adrenergic like receptor from Drosophila melanogaster". Thesis, University of Cambridge, 2002. http://ethos.bl.uk/OrderDetails.do?uin=uk.bl.ethos.620357.

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21

Salinas, Cristian Andres. "ROBUST EXPERIMENTAL DESIGN FOR ESTIMATING MYOCARDIAL BETA ADRENERGIC RECEPTOR CONCENTRATION USING POSITRON EMISSION TOMOGRAPHY." Case Western Reserve University School of Graduate Studies / OhioLINK, 2006. http://rave.ohiolink.edu/etdc/view?acc_num=case1144088920.

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22

Fraundorfer, Paul F. "Functional and biochemical characterization of trimetoquinol (TMQ) analog interactions with [beta]-adrenergic receptor subtypes /." The Ohio State University, 1993. http://rave.ohiolink.edu/etdc/view?acc_num=osu1487843314696234.

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23

Carie, Adam E. "Tumor suppressive effects of the Beta-2 adrenergic receptor and the small GTPase RhoB." [Tampa, Fla.] : University of South Florida, 2008. http://purl.fcla.edu/usf/dc/et/SFE0002330.

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24

Alsalhin, Aisha Khlani Hassan. "The role of the beta3-adrenergic receptor (β3-AR) in cardioprotection". Thesis, Stellenbosch : Stellenbosch University, 2015. http://hdl.handle.net/10019.1/97812.

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Thesis (MScMedSc)--Stellenbosch University, 2015.<br>ENGLISH ABSTRACT: It is well-established that transient activation of the β-adrenergic signalling pathway with ligands such as isoproterenol, formoterol and dobutamine, elicits cardioprotection against subsequent long periods of ischaemia. Initially the focus was on the β1- and β2-adrenergic receptors (β1-AR, β2-AR), but recently the β3-AR also emerged as a potential target in the treatment of heart disease. In heart failure, β1- and β2-AR are typically known to be down-regulated while β3-ARs, on the other hand, are up-regulated (Moniotte et

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Crumbie, Hayley Elizabeth. "Beta-adrenergic receptor signalling and excitation-contraction coupling in the heart : impact of circadian rhythms and beta-3 receptors." Thesis, University of Leicester, 2016. http://hdl.handle.net/2381/37500.

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A time-of-day variation in myocardial contraction and its response to sympathetic stimulation of β-adrenoceptors (b-ADR) exists, which is reflected by time-of-day variations in intracellular calcium [Ca2+]i regulation and electrical activity. There are three isoforms of β-ADR (β1/β2/β3) and the functional outcome of each receptor differs. β1/β2-ADRs result in positive inotropism, whereas β3-ADR induces a negative inotropic effect, associated with nitric oxide (NO) signalling. The aims of this thesis were to determine the role of the β3-ADR in the time-of-day variation in the response to sympat

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Skrentny, Thomas, та Brittany Traylor. "Influence of Genetic Variation of the β2 Adrenergic Receptor in Patients with Cystic Fibrosis". The University of Arizona, 2010. http://hdl.handle.net/10150/623791.

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Class of 2010 Abstract<br>OBJECTIVES: Cystic fibrosis (CF) is a disease that adversely affects the lung resulting in a reduction of lung diffusion. Stimulation of the β2 adrenergic receptors results in mucocilliary clearance, and therefore, lung diffusion. We sought to determine the influence of an inhaled β-‐agonist on the diffusing capacity of the lungs for carbon monoxide (DLCO), alveolar-‐capillary membrane conductance (DM), pulmonary capillary blood volume (Vc), and peripheral oxygen saturation (SaO2) in subjects with CF and compare the data to matched healthy subjects. METHODS: To det

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27

Moore, Ann Lavinia. "Beta-adrenergic receptor antagonists and exercise radionuclide angiocardiography in patients with proven coronary artery disease." Thesis, Queen's University Belfast, 1997. http://ethos.bl.uk/OrderDetails.do?uin=uk.bl.ethos.361290.

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28

Lundengård, Karin. "De- and Resensitisation of Cardiac β-Adrenergic Receptor Signaling : A Modelling Approach". Thesis, Linköpings universitet, Institutionen för fysik, kemi och biologi, 2011. http://urn.kb.se/resolve?urn=urn:nbn:se:liu:diva-69076.

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Desensitisation is defined as a failure of a signaling pathway to respond to chronic or repeated stimulation. The β-adrenergic receptor signaling pathway of the healthy adult heart is known to desensitise, and then regain the sensitivity to stimulation if given enough time to rest between stimulations (resensitisation). The fetal heart does not desensitise, and in animal models of heart failure, a permanent desensitisation have been observed. No isolated element of the signaling pathway have yet been proven to be the sole modulator of the desensitisation behavior. Therefore a mathematical mode

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29

Adejare, Adeboye. "Part I. fluorinated benzylimidazolines as agents for probing the [alpha]-adrenergic receptor. part II. synthesis, [beta]-adrenergic and antiplatelet activities of trimetoquinol analogs /." The Ohio State University, 1985. http://rave.ohiolink.edu/etdc/view?acc_num=osu1487260135356326.

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30

Hammon, Harald. "Distribution and density of alpha- and beta-adrenergic receptor : binding sites in the bovine mammary gland /." [S.l.] : [s.n.], 1993. http://www.stub.unibe.ch/html/haupt/datenbanken/diss/bestell.html.

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31

Kwok, Wing-yee Winnie, and 郭穎怡. "Polymorphisms of the {221}2-adrenergic receptor gene associated with asthma among Chinese in Hong Kong." Thesis, The University of Hong Kong (Pokfulam, Hong Kong), 2003. http://hub.hku.hk/bib/B29247330.

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32

LaBranche, Timothy Paul. "Characterization of the Beta-2 Adrenergic Receptor Mechanism in Bovine Neutrophils, and Some Effects of Inflammatory Stimuli on its Function." Diss., Virginia Tech, 2005. http://hdl.handle.net/10919/27275.

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The bovine polymorphonuclear leukocyte (neutrophil) is a central component of the acute inflammatory response, and is capable of reacting to a myriad of pro-inflammatory chemical signals that have been characterized in the context of bovine respiratory disease (BRD). Human neutrophils and bovine macrophages are known to react to pro-inflammatory signals as well; however, they are also capable of responding to anti-inflammatory signals from the autonomic nervous system. In particular, activation of the beta2-adrenergic receptor on these cells decreases several aspects of inflammatory activity

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Griffith, D. N. W. "The cerebral circulation in diabetes mellitus and hypertension and its responses to beta adrenergic receptor-blocking drugs." Thesis, University of Cambridge, 1985. http://ethos.bl.uk/OrderDetails.do?uin=uk.bl.ethos.599714.

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34

Herko, Kara, Benjamin Guthrie, and Eric Snyder. "Genetic Variation of the BETA-2 Adrenergic Receptor and the Bronchodilatory Response to Albuterol in Patients with Cystic Fibrosis." The University of Arizona, 2012. http://hdl.handle.net/10150/614494.

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Class of 2012 Abstract<br>Specific Aims: We sought to determine the influence of genetic variation of ADRB2 on the airway response to albuterol in patients with CF when compared to matched healthy controls at baseline and at 60 minutes following the administration of albuterol (2.5mg diluted in 3ml normal saline). Methods: Baseline pulmonary function (forced vital capacity, FVC, forced expiratory flow in 1-second, FEV1, mid-maximal expiratory flow, MMF, and forced expiratory flow at 50% of the FVC) was assessed in 17 patients with CF and 31 healthy subjects. Main Results: As expected, the h

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35

Calderón, Diego Mauricio Bravo. "Migração e invasão do câncer de boca via ativação de receptor beta 2 adrenérgico por mediador do estresse." Universidade de São Paulo, 2015. http://www.teses.usp.br/teses/disponiveis/25/25150/tde-10032016-163155/.

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A ativação do receptor beta 2 adrenérgico (β2-AR), pelos mediadores químicos do estresse, pode induzir efeitos estimuladores ou inibidores na migração e invasão celular, dependendo do tipo de tumor maligno. A importância deste receptor na evolução do câncer de boca não está totalmente esclarecida. O objetivo deste estudo foi verificar a expressão do β2-AR em linhagens de carcinomas espinocelular de boca (SCC-9 e SCC-25), e investigar o papel da ativação deste receptor pela norepinefrina e de seu bloqueio por um antagonista na migração e invasão destas células neoplásicas. As célul

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Calderón, Diego Mauricio Bravo. "Expressão de receptor beta-2 adrenérgico em carcinoma espinocelular de boca e sua associação com a evolução clínica tumoral." Universidade de São Paulo, 2011. http://www.teses.usp.br/teses/disponiveis/25/25150/tde-27072011-095304/.

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Os hormônios produzidos durante o estresse e seus receptores específicos têm sido amplamente envolvidos com a progressão do câncer. O objetivo deste estudo foi avaliar a expressão dos receptores β2 adrenérgicos pelas células malignas de carcinomas espinocelulares de boca (CEC) e sua correlação com as a características clínicas, evolução e o prognóstico dos pacientes. Um total de 106 pacientes portadores de CEC de boca em estádios clínicos II, III e IV, tratados no Departamento de Cirurgia de Cabeça e Pescoço e Otorrinolaringologia do Hospital de Câncer A.C. Camargo, São Paulo, Brasil, no

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Herko, Kara, and Benjamin Guthrie. "Genetic Variation of the BETA-2 Adrenergic Receptor and the Bronchodilatory Response to Albuterol in Patients with Cystic Fibrosis." The University of Arizona, 2012. http://hdl.handle.net/10150/623639.

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Class of 2012 Abstract<br>Specific Aims: We sought to determine the influence of genetic variation of ADRB2 on the airway response to albuterol in patients with CF when compared to matched healthy controls at baseline and at 60 minutes following the administration of albuterol (2.5mg diluted in 3ml normal saline). Methods: Baseline pulmonary function (forced vital capacity, FVC, forced expiratory flow in 1-second, FEV1, mid-maximal expiratory flow, MMF, and forced expiratory flow at 50% of the FVC) was assessed in 17 patients with CF and 31 healthy subjects. Main Results: As expected, the hea

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38

Visser, Thomas Jakob. "Development of new muscarinic and [beta]-adrenergic receptor radiopharmaceuticals for Positron Emission Tomography application to lungs, heart and brain /." [S.l. : [Groningen : s.n.] ; University Library Groningen] [Host], 1997. http://irs.ub.rug.nl/ppn/297496689.

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39

Houle, Melanie Taghon. "Enhanced in vivo and in vitro response to Beta-2 Adrenergic Receptor Stimulation in animals susceptible to Ventricular Fibrillation /." The Ohio State University, 2000. http://rave.ohiolink.edu/etdc/view?acc_num=osu1488192960167032.

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40

Imura, Tetsuya. "Differential expression of small heat shock proteins in reactive astrocytes after focal ischemia : possible role of beta adrenergic receptor." Kyoto University, 2000. http://hdl.handle.net/2433/180858.

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41

Gonçalves, Andrezza Neves. "Investigação do mecanismo bioquímico in vitro da interação da metaloprotease da matriz 2 (MMP-2) com o receptor beta 1 adrenérgico." Universidade de São Paulo, 2018. http://www.teses.usp.br/teses/disponiveis/17/17131/tde-01022019-102035/.

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As metaloproteases da matriz (MMPs) são enzimas proteolíticas que participam da degradação da matriz extracelular no organismo de vertebrados. Estudos mostram a grande importância dessas enzimas no processo de remodelação do tecido cardíaco, além de sugerirem a participação da MMP-2 em doenças cardiovasculares. Em estudo recente foi demonstrado que as MMPs clivam o receptor ?2-adrenérgico, contribuindo para o aumento do tônus arteriolar de ratos espontaneamente hipertensos (SHR). Acredita-se que processo semelhante possa ser verificado em relação ao receptor ?-1 adrenérgico e proteínas das jun

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Chen, Xiaochuan, Amy C. Kelly, Dustin T. Yates, Antoni R. Macko, Ronald M. Lynch, and Sean W. Limesand. "Islet adaptations in fetal sheep persist following chronic exposure to high norepinephrine." BIOSCIENTIFICA LTD, 2017. http://hdl.handle.net/10150/623222.

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Complications in pregnancy elevate fetal norepinephrine (NE) concentrations. Previous studies in NE-infused sheep fetuses revealed that sustained exposure to high NE resulted in lower expression of α2-adrenergic receptors in islets and increased insulin secretion responsiveness after acutely terminating the NE infusion. In this study, we determined if the compensatory increase in insulin secretion after chronic elevation of NE is independent of hyperglycemia in sheep fetuses and whether it is persistent in conjunction with islet desensitization to NE. After an initial assessment of glucose-sti

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Kam, Wan-lung Kenneth. "Effect of ovariectomy and estrogen replacement on the [beta]-Adrenergic receptor signaling pathway and intracellular Ca2+ homeostasis in the rat heart." Click to view the E-thesis via HKUTO, 2005. http://sunzi.lib.hku.hk/hkuto/record/B31473143.

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44

Parsa-Nezhad, Maryam. "Ex vivo evaluation of reduced myocardial beta-adrenergic receptor binding in the streptozotocin-treated hyperglycaemic rats using (S)-[(3)H]CGP12177." Thesis, University of Ottawa (Canada), 2008. http://hdl.handle.net/10393/28013.

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Type II Diabetes Mellitus (DM) is a common metabolic disorder that affects millions of people worldwide. Elevated blood glucose levels in type II DM is associated with altered sympathetic nervous system (SNS) activity and enhanced noradrenaline (NA). Persistent NA release, in turn, leads to alterations in beta-adrenergic receptor (betaAR) density and downstream signalling. A common complication of diabetes is cardiovascular disease. In this project, myocardial betaAR density was evaluated in hyperglycaemic streptozotocin (STZ)-treated rats fed high fat diet (HFD), using betaAR antagonist (S)-[

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Padro, Caroline Jeannette. "A Study of the Distal Molecular Mechanism by which Beta-2 Adrenergic Receptor Stimulation on a B Cell Regulates IgE Production." The Ohio State University, 2013. http://rave.ohiolink.edu/etdc/view?acc_num=osu1384763500.

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46

Enoshiri, Tatsuki. "Beta adrenergic receptor blockers reduce the occurrence of keloids and hypertrophic scars after cardiac device implantation: a single-institution case-control study." Kyoto University, 2017. http://hdl.handle.net/2433/227549.

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47

Muumba, Justice. "Association of Single Nucleotide Polymorphisms in the Beta-2- Adrenergic Receptor Gene with Growth, Carcass and Meat Characteristics in Beef Steers and Heifers." Thesis, The University of Arizona, 2007. http://hdl.handle.net/10150/193363.

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A total of 693 beef steers and heifers of six breeds were utilized in an association study. Traits analyzed were birth weight (BW), weaning age (WA), adjusted weaning weight (WW), hot carcass weight (HCW), marbling score (MS), 12th rib fat thickness (FT), Longissimus dorsi muscle area (LM), estimated kidney, pelvic and heart fat (KPH); and yield grade (YG). Five single nucleotide polymorphisms (SNPs) in the beta-2 adrenergic receptor (ADRB2) gene were evaluated as markers for beef traits. One SNP was uninformative and was discarded. There was an interaction of gender by A11C SNP on WA (P<0.05)

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Majid, Samia. "Synthèse, marquage à l'iode et comportement biologique de [beta]-bloquants potentiels." Université Joseph Fourier (Grenoble), 1994. http://www.theses.fr/1994GRE10074.

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Parmi les differents recepteurs myocardiques, les plus interessants d'un point de vue physiopathologique, sont les recepteurs beta adrenergiques. La synthese de beta-bloquants emetteurs gamma (#1#2#3i) utilisables in vivo dans le denombrement de ces recepteurs, a ete l'objectif essentiel de ce travail. Ce projet nous a conduits a la synthese d'analogues iodes de beta-bloquants connus: analogues du cgp12177 dans lequel le groupement tertiobutyle a ete remplace par le groupement homoveratryle, l'iodation de ce compose l'a rendu trop lipophile. Analogues du h87/07, avec une chaine en position 4 c

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Martinez, Marina G. "Genetic Variation of the Beta-2 Adrenergic Receptor and the Phenylethanolamine N-Methyltransferase Enzyme: Influence on Catecholamines, Cardiovascular Regulation, and the Cardiopulmonary Response to Albuterol." Diss., The University of Arizona, 2014. http://hdl.handle.net/10150/317802.

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Hypertension, or chronic blood pressure elevation, affects approximately a third of American adults and is responsible for $70 billion dollars annually in medical costs. Recent studies have attempted to identify genetic variants that influence cardiopulmonary function, including blood pressure regulation. This study seeks to determine whether a polymorphism in position -182 of the gene encoding the phenylethanolamine N-methyltransferase (PNMT) enzyme, which converts norepinephrine to epinephrine, influences catecholamine levels and cardiovascular function. Secondly, this study seeks to exp

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Santos, Larissa Ferreira dos. "Controle neurovascular do fluxo sanguíneo muscular e da atividade nervosa simpática durante o exercício em pacientes após síndrome isquêmica miocárdica instável com polimorfismos do receptor \'beta\'2-adrenérgico Gln27Glu." Universidade de São Paulo, 2015. http://www.teses.usp.br/teses/disponiveis/39/39132/tde-07042015-144133/.

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A síndrome isquêmica miocárdica instável (SIMI) leva a importantes alterações neurovasculares, tais como à hiperativação simpática e a diminuição do fluxo sanguíneo muscular (FSM) tanto em repouso como durante manobras fisiológicas como o exercício. A presença de alguns polimorfismos na genética humana, como o dos receptores \'beta\'2-adrenérgicos Gln27Glu, apresenta importante associação com a funcionalidade cardiovascular em indivíduos saudáveis. Contudo, não é conhecido se em pacientes com SIMI, a presença dos polimorfismos do receptor \'beta\'2-adrenérgico leva a respostas neurovasculares

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