Thematische Bibliographien / CD44 B-Lymphocytes Lymphocyte Activation

Inhaltsverzeichnis

  1. Zeitschriftenartikel
  2. Dissertationen
  3. Bücher
  4. Buchteile
  5. Konferenzberichte

Auswahl der wissenschaftlichen Literatur zum Thema „CD44 B-Lymphocytes Lymphocyte Activation“

Autor: Grafiati
Veröffentlicht am 4. Juni 2021
Zuletzt aktualisiert am 13. Februar 2022

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Zeitschriftenartikel zum Thema "CD44 B-Lymphocytes Lymphocyte Activation"

1

Rivière, Elodie, Juliette Pascaud, Nicolas Tchitchek, et al. "Salivary gland epithelial cells from patients with Sjögren’s syndrome induce B-lymphocyte survival and activation." Annals of the Rheumatic Diseases 79, no. 11 (2020): 1468–77. http://dx.doi.org/10.1136/annrheumdis-2019-216588.

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ObjectivePrimary Sjögren's syndrome (pSS) is characterised by chronic hyperactivation of B lymphocytes. Salivary gland epithelial cells (SGECs) could play a role in promoting B-lymphocyte activation within the target tissue. We aimed to study the interactions between SGECs from patients with pSS or controls and B lymphocytes.MethodsPatients had pSS according to 2016 European League Against Rheumatism/American College of Rheumatology criteria. Gene expression analysis of SGECs and B lymphocytes from pSS and controls isolated from salivary gland biopsies and blood was performed by RNA-seq. SGECs
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Ilangumaran, Subburaj, Anne Briol, and Daniel C. Hoessli. "CD44 Selectively Associates With Active Src Family Protein Tyrosine Kinases Lck and Fyn in Glycosphingolipid-Rich Plasma Membrane Domains of Human Peripheral Blood Lymphocytes." Blood 91, no. 10 (1998): 3901–8. http://dx.doi.org/10.1182/blood.v91.10.3901.

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Abstract CD44 is the major cell surface receptor for the extracellular matrix glycosaminoglycan hyaluronan and is implicated in a variety of biological events that include embryonic morphogenesis, lymphocyte recirculation, inflammation, and tumor metastasis. CD44 delivers activation signals to T lymphocytes, B lymphocytes, natural killer cells, polymorphonuclear leukocytes, and macrophages by stimulating protein tyrosine phosphorylation and calcium influx. The mechanism of signal transduction via CD44 remains undefined, although CD44 was shown to physically associate with intracellular protein
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Ilangumaran, Subburaj, Anne Briol, and Daniel C. Hoessli. "CD44 Selectively Associates With Active Src Family Protein Tyrosine Kinases Lck and Fyn in Glycosphingolipid-Rich Plasma Membrane Domains of Human Peripheral Blood Lymphocytes." Blood 91, no. 10 (1998): 3901–8. http://dx.doi.org/10.1182/blood.v91.10.3901.3901_3901_3908.

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CD44 is the major cell surface receptor for the extracellular matrix glycosaminoglycan hyaluronan and is implicated in a variety of biological events that include embryonic morphogenesis, lymphocyte recirculation, inflammation, and tumor metastasis. CD44 delivers activation signals to T lymphocytes, B lymphocytes, natural killer cells, polymorphonuclear leukocytes, and macrophages by stimulating protein tyrosine phosphorylation and calcium influx. The mechanism of signal transduction via CD44 remains undefined, although CD44 was shown to physically associate with intracellular protein tyrosine
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Shimonaka, Mika, Koko Katagiri, Toshinori Nakayama, et al. "Rap1 translates chemokine signals to integrin activation, cell polarization, and motility across vascular endothelium under flow." Journal of Cell Biology 161, no. 2 (2003): 417–27. http://dx.doi.org/10.1083/jcb.200301133.

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Chemokines arrest circulating lymphocytes within the vasculature through the rapid up-regulation of leukocyte integrin adhesive activity, promoting subsequent lymphocyte transmigration. However, the key regulatory molecules regulating this process have remained elusive. Here, we demonstrate that Rap1 plays a pivotal role in chemokine-induced integrin activation and migration. Rap1 was activated by secondary lymphoid tissue chemokine (SLC; CCL21) and stromal-derived factor 1 (CXCL4) treatment in lymphocytes within seconds. Inhibition of Rap1 by Spa1, a Rap1-specific GTPase-activating protein, a
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Mun, Yeung-Chul, Kyoung-Eun Lee, Jung Mi Kwon, et al. "Establishment of Effective B Lymphocyte Ex Vivo Expansion on Human Cord Blood Using TPO, SCF, FL, IL-4, IL-10, and CD40L." Blood 104, no. 11 (2004): 2882. http://dx.doi.org/10.1182/blood.v104.11.2882.2882.

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Abstract In respect to B lymphocyte-mediated immunity, characteristics of human cord blood are low counts of mature B lymphocytes, deficient expression of CD40L and cytokine production in CD4+ T lymphocytes, defect in the isotype switch of immunoglobulin and the activation of B lymphocytes, and low IgG production of B lymphocytes. These characteristics of the B lymphocyte from human cord blood lead to a delayed B lymphocyte-mediated immune reconstitution and an increased susceptibility to infections after a cord blood transplantation. The mechanism of immunological recostitution after cord blo
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Zhang, Jinyi, Amro Shehabeldin, Luis A. G. da Cruz, et al. "Antigen Receptor–Induced Activation and Cytoskeletal Rearrangement Are Impaired in Wiskott-Aldrich Syndrome Protein–Deficient Lymphocytes." Journal of Experimental Medicine 190, no. 9 (1999): 1329–42. http://dx.doi.org/10.1084/jem.190.9.1329.

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The Wiskott-Aldrich syndrome protein (WASp) has been implicated in modulation of lymphocyte activation and cytoskeletal reorganization. To address the mechanisms whereby WASp subserves such functions, we have examined WASp roles in lymphocyte development and activation using mice carrying a WAS null allele (WAS−/−). Enumeration of hemopoietic cells in these animals revealed total numbers of thymocytes, peripheral B and T lymphocytes, and platelets to be significantly diminished relative to wild-type mice. In the thymus, this abnormality was associated with impaired progression from the CD44−CD
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Zöller, M. "CD44, metastatic tumor spread, lymphocyte activation." Biomedicine & Pharmacotherapy 47, no. 4 (1993): 174. http://dx.doi.org/10.1016/0753-3322(93)90013-b.

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Maltzman, J. S., J. A. Carman, and J. G. Monroe. "Role of EGR1 in regulation of stimulus-dependent CD44 transcription in B lymphocytes." Molecular and Cellular Biology 16, no. 5 (1996): 2283–94. http://dx.doi.org/10.1128/mcb.16.5.2283.

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The immediate-early gene egr-1 encodes a transcription factor (EGR1) that links B-cell antigen receptor (BCR) signals to downstream activation events through the regulation of previously unidentified target genes. Here we identify the gene encoding the lymphocyte homing and migration protein CD44 as a target of EGR1 regulation in B cells. BCR-induced increases in CD44 mRNA expression and transcription levels are shown to occur in EGR1-expressing but not in nonexpressing subclones of the B-cell line WEHI-231. Kinetics of egr-1 transcription and the appearance of nuclear EGR1 protein precede CD4
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Forster, R., T. Emrich, E. Kremmer, and M. Lipp. "Expression of the G-protein--coupled receptor BLR1 defines mature, recirculating B cells and a subset of T-helper memory cells." Blood 84, no. 3 (1994): 830–40. http://dx.doi.org/10.1182/blood.v84.3.830.bloodjournal843830.

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The G-protein-coupled receptor BLR1 related to receptors for chemokines and neuropeptides has been identified as the first lymphocyte-specific member of the gene family characterized by seven transmembrane-spanning regions. Using a high-affinity anti-BLR1 monoclonal antibody (MoAb) and three-color flow cytometry it is shown that BLR1 expression on peripheral blood cells is limited to B cells and to a subset of CD4+ (14%) and CD8+ (2%) lymphocytes. T cells expressing BLR1 were positive for CD45R0, were negative for interleukin-2 receptors, show high levels of CD44, and show low levels of L-sele
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Murakami, S., and H. Okada. "Lymphocyte-Fibroblast Interactions." Critical Reviews in Oral Biology & Medicine 8, no. 1 (1997): 40–50. http://dx.doi.org/10.1177/10454411970080010201.

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Chronic inflammatory reactions are usually characterized by inflammatory cell accumulation in the extravascular connective tissue. In such sites, inappropriate activation of circulating or resident lymphocytes becomes self-perpetuating and can lead to chronic tissue destruction. In addition to that, the locally infiltrated lymphocytes should have an opportunity to interact directly with fibroblasts composing the connective tissue. The direct interactions of those different cell types seem to play important roles in lymphocyte lodging and retention in such sites. Thus, for clarification of the
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Dissertationen zum Thema "CD44 B-Lymphocytes Lymphocyte Activation"

1

Wyant, Tiana L. "Influence of Anti-CD44 on Murine B Cell Activation." VCU Scholars Compass, 2006. http://scholarscompass.vcu.edu/etd/1004.

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Lymphocyte activation and trafficking are indispensable to the immune system. CD44, an adhesion molecule, plays important roles in T cell activation, lymphocyte homing/trafficking, and tumor metastasis. Although the functions of CD44 have been shown in T cells and other leukocytes, little is known about its role in B cells. The effects of CD44 cross-linking on murine B cell activation via CD40L/IL-4 was explored using the anti-CD44 mAbs RK3G9 and IM7. Immobilized RK3G9 and IM7 could strongly inhibit B cell proliferation and Ig production, with IgE inhibition being prominent. Soluble anti-CD44
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Fournier-Conge, Anne-Marie. "Anomalies de l'activation des lymphocytes B circulants au cours de l'infection par le VIH-1 : implications physiopathologiques et cliniques." Montpellier 1, 1996. http://www.theses.fr/1996MON1T025.

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DiSanto, James Philip. "Molecular events in human T cell activation : CD4, CD8 and the human Lyt-3 molecules /." Access full-text from WCMC, 1989. http://proquest.umi.com/pqdweb?did=745024391&sid=1&Fmt=2&clientId=8424&RQT=309&VName=PQD.

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Sutherland, Claire Louise. "Structure/function analysis of CD40, a key activator of B lymphocytes." Thesis, National Library of Canada = Bibliothèque nationale du Canada, 1999. http://www.collectionscanada.ca/obj/s4/f2/dsk1/tape8/PQDD_0027/NQ38986.pdf.

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Delli, Joe. "Coreceptor and costimulatory signals organize proteins within the immunological synapse and augment proximal T cell signaling events /." Connect to full text via ProQuest. IP filtered, 2006.

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Thesis (Ph.D. in Immunology) -- University of Colorado, 2006.<br>Typescript. Includes bibliographical references (leaves 277-285). Free to UCDHSC affiliates. Online version available via ProQuest Digital Dissertations;
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Hermann, Patrice. "Recherche du ligand du CD40 : étude du rôle de son interaction avec le CD40 dans la réponse lymphocytaire B." Lyon 1, 1995. http://www.theses.fr/1995LYO1T120.

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Evans, Dean E. "CD40 Sustains T Cell Activation During Cognate Communication with Resting B Cells: a Dissertation." eScholarship@UMMS, 1998. http://escholarship.umassmed.edu/gsbs_diss/178.

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T and B-lymphocytes play an important role in an adaptive immune response. Communication between these two cells may result in either a humoral immune response or tolerance. Communication between T and B-lymphocytes involves a number of inducible cell surface molecules on both T and B-lymphocytes. It was the aim of this project to gain a greater understanding of the role of CD40 in the dynamic communication that occurs between naïve T-lymphocytes and resting B-lymphocytes during cognate communication. Because in vivo antigen specific T-lymphocytes are at low frequency, it is difficult to exami
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Esquerré, Michael. "Influence des lymphocytes T CD4+ CD25+ régulateurs sur la dynamique de formation de la synapse immunologique entre un lymphocyte T CD4+ effecteur et une cellule présentatrice d'antigène." Toulouse 3, 2007. http://thesesups.ups-tlse.fr/51/.

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La rencontre entre un lymphocyte T et une cellule présentatrice d'antigène (CPA) est un évènement central dans l'initiation et le développement de la réponse immunitaire adaptative. L'interaction entre ces deux cellules entraîne de nombreuses réorganisations moléculaires au niveau de l'aire de contact intercellulaire conduisant à la formation d'une structure dynamique et spécialisée remplissant diverses fonctions biologiques : la Synapse Immunologique (SI). Cette interaction permet à un lymphocyte T CD4+ helper (TH) de s'activer et de mettre en place une signalisation intracellulaire nécessair
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Liu, Anquan. "Proinflammatory factor mediated lymphocyte activation - the pivotal role of leukotriene B4 /." Stockholm, 2007. http://diss.kib.ki.se/2007/978-91-7357-391-7/.

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Jellison, Evan Robert. "CD4 T Cell-Mediated Lysis and Polyclonal Activation of B Cells During Lymphocytic Choriomeningitis Virus Infection: A Dissertation." eScholarship@UMMS, 2008. https://escholarship.umassmed.edu/gsbs_diss/349.

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CD4 T cells and B cells are cells associated with the adaptive immune system. The adaptive immune system is designed to mount a rapid antigen-specific response to pathogens by way of clonal expansions of T and B cells bearing discrete antigen-specific receptors. During viral infection, interactions between CD4 T cells and B cells occur in a dynamic process, where B cells that bind to the virus internalize and degrade virus particles. The B cells then present viral antigens to virus-specific CD4 T cells that activate the B cells and cause them to proliferate and differentiate into virus-specifi
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Bücher zum Thema "CD44 B-Lymphocytes Lymphocyte Activation"

1

Sudhir, Gupta, ed. Mechanisms of lymphocyte activation and immune regulation XI: B cell biology. Springer, 2007.

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Grosschedl, Rudolf, and Harinder Singh. Molecular Analysis of B Lymphocyte Development and Activation. Springer, 2014.

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(Editor), Harinder Singh, and Rudolf Grosschedl (Editor), eds. Molecular Analysis of B Lymphocyte Development and Activation (Current Topics in Microbiology and Immunology, No. 290). Springer, 2005.

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1950-, Grinstein Sergio, and Rotstein Ori D, eds. Mechanisms of leukocyte activation. Academic Press, 1990.

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(Editor), Sudhir Gupta, Frederick W. Alt (Editor), Max D. Cooper (Editor), Fritz Melchers (Editor), and Klaus Rajewsky (Editor), eds. Mechanisms of Lymphocyte Activation and Immune Regulation XI: B Cell Biology (Advances in Experimental Medicine and Biology). Springer, 2007.

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Hartigan-O’Connor, Dennis J., and Christian Brander. Immunology. Oxford University Press, 2017. http://dx.doi.org/10.1093/med/9780190493097.003.0005.

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The key factor in HIV pathogenesis is the decline in CD4+ T cells with resultant immunodeficiency and chronic inflammation. Depletion of CD4+ T cells from the gastrointestinal mucosa followed by microbial translocation and subsequent immune activation are components of disease progression in untreated patients. Symptomatic and occult opportunistic infections including cytomegalovirus contribute to chronic inflammation in persons infected with HIV. Antiretroviral therapy (ART) results in immune reconstitution, with increases in peripheral CD4+ T cell lymphocytes in most persons infected with HI
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Voll, Reinhard E., and Barbara M. Bröker. Innate vs acquired immunity. Oxford University Press, 2013. http://dx.doi.org/10.1093/med/9780199642489.003.0048.

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The innate and the adaptive immune system efficiently cooperate to protect us from infections. The ancient innate immune system, dating back to the first multicellular organisms, utilizes phagocytic cells, soluble antimicrobial peptides, and the complement system for an immediate line of defence against pathogens. Using a limited number of germline-encoded pattern recognition receptors including the Toll-like, RIG-1-like, and NOD-like receptors, the innate immune system recognizes so-called pathogen-associated molecular patterns (PAMPs). PAMPs are specific for groups of related microorganisms
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Buchteile zum Thema "CD44 B-Lymphocytes Lymphocyte Activation"

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Rudd, Christopher E., Elizabeth K. Barber, Kristine E. Burgess, et al. "Molecular Analysis of the Interaction of p56lck with the CD4 and CD8 Antigens." In Mechanisms of Lymphocyte Activation and Immune Regulation III. Springer US, 1991. http://dx.doi.org/10.1007/978-1-4684-5943-2_10.

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Laurence, Jeffrey. "CD4+ and CD8+ T Lymphocyte Activation in HIV Infection." In Advances in Experimental Medicine and Biology. Springer US, 1995. http://dx.doi.org/10.1007/978-1-4615-1995-9_1.

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Ambrus, Julian L., Cynthia H. Jurgensen, Debra L. Bowen, et al. "The Activation, Proliferation, and Differentiation of Human B Lymphocytes." In Mechanisms of Lymphocyte Activation and Immune Regulation. Springer US, 1987. http://dx.doi.org/10.1007/978-1-4684-5323-2_16.

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Melchers, Fritz, Steven R. Bauer, Christoph Berger, et al. "Precursor B Lymphocytes — Specific Monoclonal Antibodies and Genes." In Mechanisms of Lymphocyte Activation and Immune Regulation II. Springer US, 1989. http://dx.doi.org/10.1007/978-1-4757-5803-0_11.

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Sutro, Jeffrey B., Bharathi S. Vayuvegula, Sudhir Gupta, and Michael D. Cahalan. "Voltage-Sensitive Ion Channels in Human B Lymphocytes." In Mechanisms of Lymphocyte Activation and Immune Regulation II. Springer US, 1989. http://dx.doi.org/10.1007/978-1-4757-5803-0_14.

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"Second Signals for T Cell Mitogenesis Provided by a MAbs CD45 (T200) and CD5 (T1)." In Lymphocyte Activation and Differentiation. De Gruyter, 1988. http://dx.doi.org/10.1515/9783110850253-083.

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"Studies Regarding the Interaction of the T4 (CD4) Molecule with the Envelope Protein gp120 of the Human Immunodeficiency Virus (HIV)." In Lymphocyte Activation and Differentiation. De Gruyter, 1988. http://dx.doi.org/10.1515/9783110850253-064.

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"Immunobiology of B Lymphocytes." In Lymphocyte Activation and Differentiation. De Gruyter, 1988. http://dx.doi.org/10.1515/9783110850253-084.

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Petersone, Lina, and Lucy S. K. Walker. "Activation of CD4+ T Lymphocytes." In Cancer Immunotherapy Principles and Practice, 2nd ed. Springer Publishing Company, 2021. http://dx.doi.org/10.1891/9780826137432.0007.

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"Changes in Gene Expression in Differentiating B Lymphocytes." In Lymphocyte Activation and Differentiation. De Gruyter, 1988. http://dx.doi.org/10.1515/9783110850253-092.

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Konferenzberichte zum Thema "CD44 B-Lymphocytes Lymphocyte Activation"

1

Carmona, Eva M., Theodore Kottom, Deanne Hebrink, and Andrew H. Limper. "CD4-Independent Activation Of Human B Lymphocytes By Pneumocystis Beta-Glucans." In American Thoracic Society 2012 International Conference, May 18-23, 2012 • San Francisco, California. American Thoracic Society, 2012. http://dx.doi.org/10.1164/ajrccm-conference.2012.185.1_meetingabstracts.a3298.

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