Thematische Bibliographien / Cell metabolism Regulation

Inhaltsverzeichnis

  1. Zeitschriftenartikel
  2. Dissertationen
  3. Bücher
  4. Buchteile
  5. Konferenzberichte
  6. Berichte der Organisationen

Auswahl der wissenschaftlichen Literatur zum Thema „Cell metabolism Regulation“

Autor: Grafiati
Veröffentlicht am 4. Juni 2021
Zuletzt aktualisiert am 28. Januar 2023

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Zeitschriftenartikel zum Thema "Cell metabolism Regulation"

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GAO, Ping, and HaoRan WEI. "Regulation of cancer cell metabolism." SCIENTIA SINICA Vitae 47, no. 1 (January 1, 2017): 132–39. http://dx.doi.org/10.1360/n052016-00334.

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Hagel-Bradway, S., and R. Dziak. "Regulation of bone cell metabolism." Journal of Oral Pathology and Medicine 18, no. 6 (July 1989): 344–51. http://dx.doi.org/10.1111/j.1600-0714.1989.tb01564.x.

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Sun, Xinghui, and Mark W. Feinberg. "Regulation of Endothelial Cell Metabolism." Arteriosclerosis, Thrombosis, and Vascular Biology 35, no. 1 (January 2015): 13–15. http://dx.doi.org/10.1161/atvbaha.114.304869.

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Cairns, Rob A., Isaac S. Harris, and Tak W. Mak. "Regulation of cancer cell metabolism." Nature Reviews Cancer 11, no. 2 (January 24, 2011): 85–95. http://dx.doi.org/10.1038/nrc2981.

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Brynildsen, M. P., W. W. Wong, and J. C. Liao. "Transcriptional regulation and metabolism." Biochemical Society Transactions 33, no. 6 (October 26, 2005): 1423–26. http://dx.doi.org/10.1042/bst0331423.

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Understanding organisms from a systems perspective is essential for predicting cellular behaviour as well as designing gene-metabolic circuits for novel functions. The structure, dynamics and interactions of cellular networks are all vital components of systems biology. To facilitate investigation of these aspects, we have developed an integrative technique called network component analysis, which utilizes mRNA expression and transcriptional network connectivity to determine network component dynamics, functions and interactions. This approach has been applied to elucidate transcription factor
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Pokotylo, I. V. "Lipoxygenases and plant cell metabolism regulation." Ukrainian Biochemical Journal 87, no. 2 (April 27, 2015): 41–55. http://dx.doi.org/10.15407/ubj87.02.041.

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Spiegel, Sarah, and Alfred H. Merrill. "Sphingolipid metabolism and cell growth regulation." FASEB Journal 10, no. 12 (October 1996): 1388–97. http://dx.doi.org/10.1096/fasebj.10.12.8903509.

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Hough, Kenneth P., Danielle A. Chisolm, and Amy S. Weinmann. "Transcriptional regulation of T cell metabolism." Molecular Immunology 68, no. 2 (December 2015): 520–26. http://dx.doi.org/10.1016/j.molimm.2015.07.038.

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Wang, Yin-Hu, Anthony Y. Tao, Martin Vaeth, and Stefan Feske. "Calcium regulation of T cell metabolism." Current Opinion in Physiology 17 (October 2020): 207–23. http://dx.doi.org/10.1016/j.cophys.2020.07.016.

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Bailey, Shannon M., Uduak S. Udoh, and Martin E. Young. "Circadian regulation of metabolism." Journal of Endocrinology 222, no. 2 (June 13, 2014): R75—R96. http://dx.doi.org/10.1530/joe-14-0200.

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In association with sleep–wake and fasting–feeding cycles, organisms experience dramatic oscillations in energetic demands and nutrient supply. It is therefore not surprising that various metabolic parameters, ranging from the activity status of molecular energy sensors to circulating nutrient levels, oscillate in time-of-day-dependent manners. It has become increasingly clear that rhythms in metabolic processes are not simply in response to daily environmental/behavioral influences, but are driven in part by cell autonomous circadian clocks. By synchronizing the cell with its environment, clo
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Dissertationen zum Thema "Cell metabolism Regulation"

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Tejedor, Vaquero Sonia 1988. "Influence of metabolism in the regulation of T cell differentiation." Doctoral thesis, Universitat Pompeu Fabra, 2018. http://hdl.handle.net/10803/664638.

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Glucose is a key nutrient for T cells. Despite that T cell activation is impaired when they are deprived of glucose, it has also been shown that T effector responses can be elicited in vivo in glucose-poor environments, such as the intratumoral niche, which raises the question of how these cells can maintain their function in nutrient-restricted sites. In this work, we analyzed the ability of T effector cells to be activated by pro-inflammatory polarizing conditions under limiting glucose availability, using an in vitro model in which effector Th0 cells were restimulated to secondary effector
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Babić, Nikolina. "Regulation of energy metabolism of heart myoblasts /." Thesis, Connect to this title online; UW restricted, 2004. http://hdl.handle.net/1773/11563.

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Mathew, Jasmin. "Keratin 8/18 regulation of hepatic cell death and metabolism." Thesis, Université Laval, 2009. http://www.theses.ulaval.ca/2009/26554/26554.pdf.

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Szkolnicka, Dagmara Maria. "MicroRNA regulation of drug metabolism in stem cell-derived hepatocytes." Thesis, University of Edinburgh, 2016. http://hdl.handle.net/1842/23421.

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The liver is a multi-functional and highly regenerative organ. While resilient, the liver is susceptible to organ damage and failure. In both the acute and chronic settings liver disease has dire consequences for health. A common cause of liver damage is adverse reactions to drugs which can lead to drug induced liver injury (DILI). This creates major problems for patients, clinicians, the pharmaceutical industry and regulatory authorities. In the context of drug overdose or serious adverse reactions, liver failure can be acute and life threatening, and in some cases require orthotopic liver tr
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Mukherjee, Abir. "ROLE OF LYSOPHOSPHATIDIC ACID IN REGULATION OF CANCER CELL METABOLISM." VCU Scholars Compass, 2012. http://scholarscompass.vcu.edu/etd/391.

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The simplest phospholipid, lysophosphatidic acid (LPA), is a heat stable component of serum known for its proliferative and migratory activities in cancer cells. Strong evidence suggests that LPA production and expression of its receptors are dysregulated in multiple human malignancies. The mechanism behind LPA-mediated tumor cell growth and oncogenesis remains poorly understood. In this thesis project I used ovarian and other cancer cells as a model system to examine the hypothesis that LPA present in the tumor microenvironment is a pathophysiological determinant of hyperactive de novo lipoge
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Syal, Charvi. "Epigenetic Regulation of Lipid Metabolism in Neural Stem Cell Fate Decision." Thesis, Université d'Ottawa / University of Ottawa, 2019. http://hdl.handle.net/10393/38706.

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Bioactive lipids have emerged as prominent regulators of neural stem and progenitor cell (NPC) function under both physiological and pathological conditions. However, how lipid metabolism is regulated, and its role in modulation of NPC function remains unknown. In this regard, my study defines a novel epigenetic pathway that regulates lipid metabolism to determine NPC proliferation versus differentiation. Specifically, I show that activation of an atypical protein kinase C (aPKC)-mediated Ser436 phosphorylation of CREB binding protein (CBP) by aging, metformin stimulation and continued passagi
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Ng, Shyh Chang. "Regulation of Stem Cell Metabolism by the Lin28/let-7 Axis." Thesis, Harvard University, 2013. http://dissertations.umi.com/gsas.harvard:11217.

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My PhD thesis is focused on two fundamental aspects of stem cell metabolism: (1) the role of Lin28 in programming stem cell metabolism, and (2) how metabolism in turn fuels and governs pluripotency. Our studies led us to discover that the stem cell factor Lin28a promotes gigantism by enhancing glucose metabolism in mice, coinciding with discoveries that LIN28B polymorphisms influence height variation in human GWAS. Subsequently, we discovered that the Lin28/let-7 pathway controls glucose metabolism by orchestrating the upregulation of multiple insulin-PI3K-mTOR components, particularly in skel
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Mofarrahi, Mahroo. "Regulation of skeletal muscle satellite cell proliferation by NADPH oxidase." Thesis, McGill University, 2007. http://digitool.Library.McGill.CA:80/R/?func=dbin-jump-full&object_id=111521.

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Skeletal satellite cells are adult stem cells located among muscle fibers. Proliferation, migration and subsequent differentiation of these cells are critical steps in the repair of muscle injury. We document in this study the roles and mechanisms through which the NAPDH oxidase complex regulates skeletal satellite cell proliferation. The NADPH oxidase subunits Nox2, Nox4, p22phox, p47phox and p67 phox were detected in primary human and murine skeletal muscle satellite cells. In human satellite cells, NADPH oxidase-fusion proteins were localized in the cytosolic and membrane compartments of th
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Aitchison, Robert E. D. "Mammary cell cyclic AMP : regulation of breakdown and influence on protein phosphorylation." Thesis, University of Glasgow, 1987. http://ethos.bl.uk/OrderDetails.do?uin=uk.bl.ethos.303363.

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Beauchamp, Pascal. "The functional role of the RNA-binding protein HuR in the regulation of muscle cell differentiation /." Thesis, McGill University, 2008. http://digitool.Library.McGill.CA:80/R/?func=dbin-jump-full&object_id=111586.

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Muscle tissue development (myogenesis) involves the formation of specific fibers (myotubes) from muscle cells (myoblasts). For this to occur, the sequential expression of Myogenic Regulatory Factors (MRFs), such as MyoD and myogenin, is required. The expression of these MRFs is regulated posttranscriptionally by the RNA-binding protein HuR, whereby HuR associates with the 3'-untranslated regions of MyoD and myogenin mRNA, leading to a significant increase in their half-lives. Here we show that the cleavage of HuR by caspases at the aspartate (D) 226 residue is one of the main regulators of its
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Bücher zum Thema "Cell metabolism Regulation"

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INSERM European Symposium on Hormones and Cell Regulation (15e 1990 Sainte-Odile, France). Hormones and cell regulation. Paris: INSERM, 1990.

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Inserm European Symposium on Hormones and Cell Regulation (13th 1988 Sainte-Odile, France). Hormones and cell regulation. London: Paris : INSERM, 1989.

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G, Thurman Ronald, Kauffman Frederick C, and Jungermann Kurt, eds. Regulation of hepatic metabolism: Intra- and intercellular compartmentation. New York: Plenum Press, 1986.

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Severin, E. S. Izbiratelʹnai͡a︡ reguli͡a︡t͡s︡ii͡a︡ kletochnogo metabolizma: Dolozheno na sorok pi͡a︡tom ezhegodnom Bakhovskom chtenii 17 marta 1989 g. Moskva: "Nauka", 1991.

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Felix, Bronner, ed. Intracellular calcium regulation. New York: Wiley-Liss, 1990.

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J, Clemens Michael, ed. Protein phosphorylation in cell growth regulation. Australia: Harwood Academic Publishers, 1996.

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Regulation of vitamin A homeostasis by the stellate cell (vitamin A-storing cell) system. New York: Nova Biomedical Books, 2011.

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Heinrich, Reinhart. The regulation of cellular systems. New York: Chapman & Hall, 1996.

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1931-, Dumont Jacques E., Nunez J, and Institut national de la santé et de la recherche médicale (France), eds. Hormones and cell regulation =: Hormones et Regulation Cellulaire: Proceedings of the 14th INSERM European Symposium on Hormones and Cell Regulation, held at Mont Sainte-Odile (France), September 25-28, 1989. London: Libbey, 1989.

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Inserm, European Symposium on Hormones and Cell Regulation (11th 1986 Sainte Odile France). Hormones and cell regulation =: Hormones et Regulation Cellulaire: Proceedings of the 11th INSERM European Symposium on Hormones and Cell Regulation, held at Sainte-Odile (France), 29 September-2 October, 1986. Paris: INSERM, 1987.

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Buchteile zum Thema "Cell metabolism Regulation"

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Gouw, Arvin M., Annie L. Hsieh, Zachary E. Stine, and Chi V. Dang. "MYC Regulation of Metabolism and Cancer." In Tumor Cell Metabolism, 101–22. Vienna: Springer Vienna, 2015. http://dx.doi.org/10.1007/978-3-7091-1824-5_5.

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Blackmore, Peter F., Christopher J. Lynch, Stephen B. Bocckino, and John H. Exton. "Regulation of Hepatic Glycogenolysis by Calcium-Mobilizing Hormones." In Cell Calcium Metabolism, 179–85. Boston, MA: Springer US, 1989. http://dx.doi.org/10.1007/978-1-4684-5598-4_19.

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Corkey, Barbara E., Keith Tornheim, Jude T. Deeney, M. Clay Glennon, Janice C. Parker, Franz M. Matschinsky, Neil B. Ruderman, and Marc Prentki. "Metabolic Regulation of Ca2+ Handling in Permeabilized Insulinoma Cells." In Cell Calcium Metabolism, 369–77. Boston, MA: Springer US, 1989. http://dx.doi.org/10.1007/978-1-4684-5598-4_40.

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Vonakis, Becky M., and Jack Y. Vanderhoek. "Role of Calcium in the Regulation of Mammalian Lipoxygenases." In Cell Calcium Metabolism, 387–96. Boston, MA: Springer US, 1989. http://dx.doi.org/10.1007/978-1-4684-5598-4_42.

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Morgan, James I., and Tom Curran. "Regulation of c-fos Expression by Voltage-Dependent Calcium Channels." In Cell Calcium Metabolism, 305–12. Boston, MA: Springer US, 1989. http://dx.doi.org/10.1007/978-1-4684-5598-4_33.

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Trump, Benjamin F., and Irene K. Berezesky. "Role of Ion Regulation in Cell Injury, Cell Death, and Carcinogenesis." In Cell Calcium Metabolism, 441–49. Boston, MA: Springer US, 1989. http://dx.doi.org/10.1007/978-1-4684-5598-4_46.

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Lindros, Kai O., Gunnar Bengtsson, Mikko Salaspuro, and Hannu Väänänen. "Separation of Functionally Different Liver Cell Types." In Regulation of Hepatic Metabolism, 137–58. Boston, MA: Springer US, 1986. http://dx.doi.org/10.1007/978-1-4684-5041-5_6.

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Hansford, Richard G., Rafael Moreno-Sánchez, and James M. Staddon. "Regulation of Pyruvate Dehydrogenase in Isolated Cardiac Myocytes and Hepatocytes by Cytosolic Calcium." In Cell Calcium Metabolism, 331–41. Boston, MA: Springer US, 1989. http://dx.doi.org/10.1007/978-1-4684-5598-4_36.

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Paro, Renato, Ueli Grossniklaus, Raffaella Santoro, and Anton Wutz. "Epigenetics and Metabolism." In Introduction to Epigenetics, 179–201. Cham: Springer International Publishing, 2021. http://dx.doi.org/10.1007/978-3-030-68670-3_9.

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AbstractMost chromatin-modifying enzymes use metabolites as cofactors. Consequently, the cellular metabolism can influence the capacity of the cell to write or erase chromatin marks. This points to an intimate relationship between metabolic and epigenetic regulation. In this chapter, we describe the biosynthetic pathways of cofactors that are implicated in epigenetic and chromatin regulation and provide examples of how metabolic pathways can influence chromatin and epigenetic processes as well as their interplay in developmental and cancer biology.
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Kersten, Sander. "Regulation of Nutrient Metabolism and Inflammation." In Results and Problems in Cell Differentiation, 13–25. Berlin, Heidelberg: Springer Berlin Heidelberg, 2010. http://dx.doi.org/10.1007/978-3-642-14426-4_2.

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Konferenzberichte zum Thema "Cell metabolism Regulation"

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Kasbawati, A. Y. Gunawan, R. Hertadi, and K. A. Sidarto. "Metabolic regulation and maximal reaction optimization in the central metabolism of a yeast cell." In SYMPOSIUM ON BIOMATHEMATICS (SYMOMATH 2014). AIP Publishing LLC, 2015. http://dx.doi.org/10.1063/1.4914436.

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Amitrano, Andrea, Brandon Walling, Kyun Do Kim, Brandon Berry, Adam Trewin, Andrew Wojtovich, and Minsoo Kim. "Abstract A73: Optogenetic regulation of T cell metabolism in the tumor microenvironment." In Abstracts: AACR Special Conference on Tumor Immunology and Immunotherapy; October 1-4, 2017; Boston, MA. American Association for Cancer Research, 2018. http://dx.doi.org/10.1158/2326-6074.tumimm17-a73.

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Blenis, John, Gina Lee, Jamie Dempsey, and Christina England. "Abstract IA03: mTORC1/S6K1: Regulation of RNA biogenesis, protein synthesis, and cell metabolism." In Abstracts: AACR Special Conference on Translational Control of Cancer: A New Frontier in Cancer Biology and Therapy; October 27-30, 2016; San Francisco, CA. American Association for Cancer Research, 2017. http://dx.doi.org/10.1158/1538-7445.transcontrol16-ia03.

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Golovatskaya, I. F., M. V. Nechaeva, and E. V. Boiko. "20E-dependent regulation of growth and secondary metabolism of cell culture Lychnis chalcedonica L." In IX Congress of society physiologists of plants of Russia "Plant physiology is the basis for creating plants of the future". Kazan University Press, 2019. http://dx.doi.org/10.26907/978-5-00130-204-9-2019-124.

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Audet-Walsh, Étienne, David Papadopoli, Julie St-Pierre, and Vincent Giguère. "Abstract 2436: Regulation of breast cancer cell metabolism by the AMPK/ERR/PGC pathway." In Proceedings: AACR Annual Meeting 2014; April 5-9, 2014; San Diego, CA. American Association for Cancer Research, 2014. http://dx.doi.org/10.1158/1538-7445.am2014-2436.

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Burkhart, Richard, Danielle Pineda, Joseph Cozzitorto, Charles Yeo, Jonathan Brody, and Jordan Winter. "Abstract 5144: RNA-binding protein HuR supports post-transcriptional regulation of pancreatic cancer cell metabolism." In Proceedings: AACR 103rd Annual Meeting 2012‐‐ Mar 31‐Apr 4, 2012; Chicago, IL. American Association for Cancer Research, 2012. http://dx.doi.org/10.1158/1538-7445.am2012-5144.

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Vilyanen, D. V., E. Yu Garnik, V. I. Tarasenko, and Yu M. Konstantinov. "STUDY OF CHLOROPHYLL METABOLISM IN A DOUBLE ARABIDOPSIS THALIANA MUTANT GDH1GDH2 DURING A LONG-TERM EXPOSITION OF PLANTS IN THE DARK." In The Second All-Russian Scientific Conference with international participation "Regulation Mechanisms of Eukariotic Cell Organelle Functions". SIPPB SB RAS, 2018. http://dx.doi.org/10.31255/978-5-94797-318-1-22-24.

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Carroll, Patrick A., Daniel Diolaiti, Pei-Feng Cheng, Haiwei Gu, Danijel Djukovic, Daniel Raftery, Donald E. Ayer, Charles H. Muller, and Robert N. Eisenman. "Abstract PR12: Transcriptional regulation of metabolism by MLX and its binding partners is essential for tumor cell survival and spermatogenesis." In Abstracts: AACR Special Conference: Metabolism and Cancer; June 7-10, 2015; Bellevue, WA. American Association for Cancer Research, 2016. http://dx.doi.org/10.1158/1557-3125.metca15-pr12.

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Iwata, Shigeru, Mingzeng Zhang, Maiko Hajime, Naoaki Ohkubo, Hiroko Miyata, Yasuyuki Todoroki, Shingo Nakayamada, and Yoshiya Tanaka. "OP0196 IMBALANCE BETWEEN MEMORY TH1 AND TH1-TREG CELLS DEPENDS ON DIFFERENTIAL REGULATION OF CELL METABOLISM IN PATIENTS WITH SLE." In Annual European Congress of Rheumatology, EULAR 2019, Madrid, 12–15 June 2019. BMJ Publishing Group Ltd and European League Against Rheumatism, 2019. http://dx.doi.org/10.1136/annrheumdis-2019-eular.3409.

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Schnepp, Patricia M., Dennis D. Lee, Ian H. Guldner, Treasa O'Tighearnaigh, Bhavana Palakurthi, Kaitlyn E. Eckert, Tiffany A. Toni, Brandon L. Ashfeld, and Siyuan Zhang. "Abstract 4934: Brain metastatic microenvironment reshapes cancer cell metabolism through epigenetic up-regulation of glutamate decarboxylase 1." In Proceedings: AACR Annual Meeting 2017; April 1-5, 2017; Washington, DC. American Association for Cancer Research, 2017. http://dx.doi.org/10.1158/1538-7445.am2017-4934.

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Berichte der Organisationen zum Thema "Cell metabolism Regulation"

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Granot, David, and Richard Amasino. Regulation of Senescence by Sugar Metabolism. United States Department of Agriculture, January 2003. http://dx.doi.org/10.32747/2003.7585189.bard.

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Research objectives a. Analyze transgenic plants that undergo rapid senescence due to increased expression of hexokinase. b. Determine if hexokinase-induced senescence accelerates natural senescence using senescence specific promoters that drive expression of a reporter gene (GUS) and a cytokinin producing gene (IPT - isopentyl transferase). c. Isolate and analyze plant genes that suppress sugar-induced cell death (SICD) in yeast, genes that potentially are involved in programmed cell death and senescence in plants. Background to the topic Leaf senescence is a regulated process of programmed c
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Ben-Arie, Ruth, John M. Labavitch, and Amos Blumenfeld. Hormonal Regulation of Cell Wall Metabolism During Fruit Ripening. United States Department of Agriculture, August 1987. http://dx.doi.org/10.32747/1987.7568074.bard.

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Meidan, Rina, and Robert Milvae. Regulation of Bovine Corpus Luteum Function. United States Department of Agriculture, March 1995. http://dx.doi.org/10.32747/1995.7604935.bard.

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The main goal of this research plan was to elucidate regulatory mechanisms controlling the development, function of the bovine corpus luteum (CL). The CL contains two different sterodigenic cell types and therefore it was necessary to obtain pure cell population. A system was developed in which granulosa and theca interna cells, isolated from a preovulatory follicle, acquired characteristics typical of large (LL) and small (SL) luteal cells, respectively, as judged by several biochemical and morphological criteria. Experiments were conducted to determine the effects of granulosa cells removal
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Blumwald, Eduardo, and Avi Sadka. Citric acid metabolism and mobilization in citrus fruit. United States Department of Agriculture, October 2007. http://dx.doi.org/10.32747/2007.7587732.bard.

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Accumulation of citric acid is a major determinant of maturity and fruit quality in citrus. Many citrus varieties accumulate citric acid in concentrations that exceed market desires, reducing grower income and consumer satisfaction. Citrate is accumulated in the vacuole of the juice sac cell, a process that requires both metabolic changes and transport across cellular membranes, in particular, the mitochondrial and the vacuolar (tonoplast) membranes. Although the accumulation of citrate in the vacuoles of juice cells has been clearly demonstrated, the mechanisms for vacuolar citrate homeostasi
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Pell, Eva J., Sarah M. Assmann, Amnon Schwartz, and Hava Steinberger. Ozone Altered Stomatal/Guard Cell Function: Whole Plant and Single Cell Analysis. United States Department of Agriculture, December 2000. http://dx.doi.org/10.32747/2000.7573082.bard.

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Original objectives (revisions from original proposal are highlighted) 1. Elucidate the direct effects O3 and H2O2 on guard cell function, utilizing assays of stomatal response in isolated epidermal peels and whole cell gas exchange. 2. Determine the mechanistic basis of O3 and H2O2 effects on the plasma membrane through application of the electrophysiological technique of patch clamping to isolated guard cells. 3. Determine the relative sensitivity of Israeli cultivars of economically important crops to O3 and determine whether differential leaf conductance responses to O3 can explain relativ
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Schuster, Gadi, and David Stern. Integration of phosphorus and chloroplast mRNA metabolism through regulated ribonucleases. United States Department of Agriculture, August 2008. http://dx.doi.org/10.32747/2008.7695859.bard.

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New potential for engineering chloroplasts to express novel traits has stimulated research into relevant techniques and genetic processes, including plastid transformation and gene regulation. This proposal continued our long time BARD-funded collaboration research into mechanisms that influence chloroplast RNA accumulation, and thus gene expression. Previous work on cpRNA catabolism has elucidated a pathway initiated by endonucleolytic cleavage, followed by polyadenylation and exonucleolytic degradation. A major player in this process is the nucleus-encoded exoribonuclease/polymerasepolynucle
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Blumwald, Eduardo, and Avi Sadka. Sugar and Acid Homeostasis in Citrus Fruit. United States Department of Agriculture, January 2012. http://dx.doi.org/10.32747/2012.7697109.bard.

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Citrus fruit quality standards have been determined empirically, depending on species and on the particular growing regions. In general, the TSS (total soluble solids) to total acidity (TA) ratio determines whether citrus fruit can be marketed. Soluble sugars account for most of the TSS during harvest while TA is determined almost solely by the citric acid content, which reaches levels of 1-5% by weight in many cultivated varieties. Acid and sugar homeostasis in the fruit is critical for the management of existing cultivars, the development of new cultivars, the improvement of pre- and post-ha
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Kornbluth, Sally. Metabolic Regulation of Ovarian Cancer Cell Death. Fort Belvoir, VA: Defense Technical Information Center, July 2012. http://dx.doi.org/10.21236/ada570124.

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Kornbluth, Sally. Metabolic Regulation of Ovarian Cancer Cell Death. Fort Belvoir, VA: Defense Technical Information Center, July 2013. http://dx.doi.org/10.21236/ada597625.

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Lers, Amnon, E. Lomaniec, S. Burd, A. Khalchitski, L. Canetti, and Pamela J. Green. Analysis of Senescence Inducible Ribonuclease in Tomato: Gene Regulation and Function. United States Department of Agriculture, February 2000. http://dx.doi.org/10.32747/2000.7570563.bard.

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Natural leaf senescence has a negative influence on yield. Postharvest induced senescence contributes to the losses of quality in flowers, foliage, and vegetables. Strategies designed to control the senescence process in crop plants could therefore have great applied significance. Senescence is regulated by differential gene expression yet, functional characterization of the genes specifically induced and study of their expression control, is still in its infancy. Study of senescence-specific genes is required to allow identification of regulatory elements participating in senescence-induced e
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