Thematische Bibliographien / Cerebral ischemia – Treatment

Inhaltsverzeichnis

  1. Zeitschriftenartikel
  2. Dissertationen
  3. Bücher
  4. Buchteile
  5. Konferenzberichte
  6. Berichte der Organisationen

Auswahl der wissenschaftlichen Literatur zum Thema „Cerebral ischemia – Treatment“

Autor: Grafiati
Veröffentlicht am 4. Juni 2021
Zuletzt aktualisiert am 28. Januar 2023

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Zeitschriftenartikel zum Thema "Cerebral ischemia – Treatment"

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Kanazawa, Masato, Tetsuya Takahashi, Masanori Ishikawa, Osamu Onodera, Takayoshi Shimohata, and Gregory J. del Zoppo. "Angiogenesis in the ischemic core: A potential treatment target?" Journal of Cerebral Blood Flow & Metabolism 39, no. 5 (March 6, 2019): 753–69. http://dx.doi.org/10.1177/0271678x19834158.

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The ischemic penumbra is both a concept in understanding the evolution of cerebral tissue injury outcome of focal ischemia and a potential therapeutic target for ischemic stroke. In this review, we examine the evidence that angiogenesis can contribute to beneficial outcomes following focal ischemia in model systems. Several studies have shown that, following cerebral ischemia, endothelial proliferation and subsequent angiogenesis can be detected beginning four days after cerebral ischemia in the border of the ischemic core, or in the ischemic periphery, in rodent and non-human primate models,
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Siesjö, Bo K. "Pathophysiology and treatment of focal cerebral ischemia." Journal of Neurosurgery 77, no. 3 (September 1992): 337–54. http://dx.doi.org/10.3171/jns.1992.77.3.0337.

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✓ The mechanisms that give rise to ischemic brain damage have not been definitively determined, but considerable evidence exists that three major factors are involved: increases in the intercellular cytosolic calcium concentration (Ca++i), acidosis, and production of free radicals. A nonphysiological rise in Ca++i due to a disturbed pump/leak relationship for calcium is believed to cause cell damage by overactivation of lipases and proteases and possibly also of endonucleases, and by alterations of protein phosphorylation, which secondarily affects protein synthesis and genome expression. The
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Wang, Lei, Xu Zhang, Xiaoxing Xiong, Hua Zhu, Ran Chen, Shudi Zhang, Gang Chen, and Zhihong Jian. "Nrf2 Regulates Oxidative Stress and Its Role in Cerebral Ischemic Stroke." Antioxidants 11, no. 12 (November 30, 2022): 2377. http://dx.doi.org/10.3390/antiox11122377.

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Cerebral ischemic stroke is characterized by acute ischemia in a certain part of the brain, which leads to brain cells necrosis, apoptosis, ferroptosis, pyroptosis, etc. At present, there are limited effective clinical treatments for cerebral ischemic stroke, and the recovery of cerebral blood circulation will lead to cerebral ischemia-reperfusion injury (CIRI). Cerebral ischemic stroke involves many pathological processes such as oxidative stress, inflammation, and mitochondrial dysfunction. Nuclear factor erythroid 2-related factor 2 (Nrf2), as one of the most critical antioxidant transcript
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Siesjö, Bo K. "Pathophysiology and treatment of focal cerebral ischemia." Journal of Neurosurgery 108, no. 3 (March 2008): 616–31. http://dx.doi.org/10.3171/jns/2008/108/3/0616.

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✓ This article examines the pathophysiology of lesions caused by focal cerebral ischemia. Ischemia due to middle cerebral artery occlusion encompasses a densely ischemic focus and a less densely ischemic penumbral zone. Cells in the focus are usually doomed unless reperfusion is quickly instituted. In contrast, although the penumbra contains cells “at risk,” these may remain viable for at least 4 to 8 hours. Cells in the penumbra may be salvaged by reperfusion or by drugs that prevent an extension of the infarction into the penumbral zone. Factors responsible for such an extension probably inc
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Siesjö, Bo K. "Pathophysiology and treatment of focal cerebral ischemia." Journal of Neurosurgery 77, no. 2 (August 1992): 169–84. http://dx.doi.org/10.3171/jns.1992.77.2.0169.

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✓ This article examines the pathophysiology of lesions caused by focal cerebral ischemia. Ischemia due to middle cerebral artery occlusion encompasses a densely ischemic focus and a less densely ischemic penumbral zone. Cells in the focus are usually doomed unless reperfusion is quickly instituted. In contrast, although the penumbra contains cells “at risk.” these may remain viable for at least 4 to 8 hours. Cells in the penumbra may be salvaged by reperfusion or by drugs that prevent an extension of the infarction into the penumbral zone. Factors responsible for such an extension probably inc
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Han, Xue Mei, Hong Tao Wei, and Song Yan Liu. "Involvement of Erythropoietin Expression in Acupuncture Preconditioning-Induced Ischemic Tolerance." Advanced Materials Research 554-556 (July 2012): 1650–55. http://dx.doi.org/10.4028/www.scientific.net/amr.554-556.1650.

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Abstract Objective To investigate the expression of erythropoietin (EPO) after acupuncture preconditioning plus focal cerebral ischemia treatment. Methods Rat focal cerebral ischemia model and acupuncture preconditioning model were established. Animals were randomly assigned into different groups: control (focal cerebral ischemia) and acupuncture preconditioning plus focal cerebral ischemia, with 8 rats for each group. The expression of EPO after different treatments was determined by histological examination, immunohistochemistry and in situ hybridization. Results The mRNA and protein express
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Song, Siying, Hao Wu, Xunming Ji, and Ran Meng. "The BE COOL Treatments (Batroxobin, oxygEn, Conditioning, and cOOLing): Emerging Adjunct Therapies for Ischemic Cerebrovascular Disease." Journal of Clinical Medicine 11, no. 20 (October 20, 2022): 6193. http://dx.doi.org/10.3390/jcm11206193.

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Ischemic cerebrovascular disease (ICD), the most common neurological disease worldwide, can be classified based on the onset time (acute/chronic) and the type of cerebral blood vessel involved (artery or venous sinus). Classifications include acute ischemic stroke (AIS)/transient ischemic attack (TIA), chronic cerebral circulation insufficiency (CCCI), acute cerebral venous sinus thrombosis (CVST), and chronic cerebrospinal venous insufficiency (CCSVI). The pathogenesis of cerebral arterial ischemia may be correlated with cerebral venous ischemia through decreased cerebral perfusion. The core
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Spetzler, Robert F. "Treatment of chronic cerebral ischemia." Surgical Neurology 23, no. 2 (February 1985): 201. http://dx.doi.org/10.1016/0090-3019(85)90350-7.

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Ghosh, Nilanjan, Rituparna Ghosh, Zulfiqar A. Bhat, Vivekananda Mandal, Sitesh C. Bachar, Namsa D. Nima, Otimenyin O. Sunday, and Subhash C. Mandal. "Advances in Herbal Medicine for Treatment of Ischemic Brain Injury." Natural Product Communications 9, no. 7 (July 2014): 1934578X1400900. http://dx.doi.org/10.1177/1934578x1400900739.

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Ischemic brain injury is one of the leading causes of death worldwide and has attracted a lot of attention in the field of drug discovery. Cerebral ischemia is a complex pathological process involving a series of mechanisms, including generation of free radicals, oxidative stress, disruption of the membrane function, release of neurotransmitters and apoptosis. Thrombolytic therapy is the most effective therapeutic strategy, but the benefits are far from being absolute. Increased attention in the field of drug discovery has been focused on using natural compounds from traditional medicinal herb
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Han, Xue Mei, Hong Tao Wei та Song Yan Liu. "Functional Role of HIF-1α in Hypoxic Preconditioning-Induced Neuroprotection against Focal Cerebral Ischemia". Advanced Materials Research 554-556 (липень 2012): 1762–67. http://dx.doi.org/10.4028/www.scientific.net/amr.554-556.1762.

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Objective To investigate the expression of HIF-1α after permenent focal cerebral ischemia and to explore the role of HIF-1α in hypoxic preconditioning-induced neuroprotection. Methods Rat focal cerebral ischemia model and hypoxic preconditioning models were established. Animals were randomly divided into four groups: healthy control, hypoxic preconditioning (3 h of 8% O2/92% N2 treatment), focal cerebral ischemia (6 h, 1 d, 3 d or 7 d) and hypoxic preconditioning + focal cerebral ischemia (6 h, 1 d, 3 d or 7 d). The expression of HIF-1α after different treatments was determined by histological
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Dissertationen zum Thema "Cerebral ischemia – Treatment"

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Yang, Di, and 楊荻. "Neuroprotective effects of lycium barbarum extracts in cerebral and retinal ischemia/reperfusion injury." Thesis, The University of Hong Kong (Pokfulam, Hong Kong), 2014. http://hdl.handle.net/10722/206738.

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Ischemic stroke is a devastating cerebrovascular disease resulting in high mortality rate and distressing sequelae such as hemiplegia, ataxia and even visual impairment. Retinal ischemia refers to a common pathological feature shared by many blinding diseases including retinal vascular occlusions, diabetic retinopathy, glaucoma, and retinopathy of prematurity. Ischemia/reperfusion injury is implicated in both of these pathological conditions, which greatly impact on one’s daily life. The eventual consequence of the insult is irreversible neuronal cell death and functional deterioration. Apart
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Chan, Chu-fung, and 陳柱峰. "Neuroprotective effects of granulocyte-colony stimulating factor in a mice stroke model." Thesis, The University of Hong Kong (Pokfulam, Hong Kong), 2007. http://hub.hku.hk/bib/B40687284.

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Xu, Mingjing, and 徐明婧. "Baicalin protects neural cells from cerebral ischemia reperfusion injury by scavenging peroxynitrite." Thesis, The University of Hong Kong (Pokfulam, Hong Kong), 2011. http://hub.hku.hk/bib/B47753110.

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 Ischemic stroke is the leading cause of death and disability in human diseases all around the world. As effective treatment for ischemic stroke is still absent, seeking for new therapy is of great interest. Currently, several key pathological cascades following cerebral ischemia have been explored to develop further therapies. Among them, reactive nitrogen species (RNS) has been indicated to play a critical role in cerebral ischemia reperfusion injury. As one of the RNS, peroxynitrite contributes to the neural cell death and subsequent brain dysfunction in the process. Thus, development of
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Khanasari, Parto S. "An investigation of the neuroprotective properties of fenamate NSAIDs, against experimental model of ischemic stroke." Scholarly Commons, 2007. https://scholarlycommons.pacific.edu/uop_etds/671.

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Stroke is a devastating neurological disease with limited treatment opportunities. Recent advances in understanding the underlying pathogenesis of cerebral ischemia support the involvement of multiple biochemical pathways in the development of the ischemic injury. The work reported in this thesis was undertaken to investigate the hypothesis that fenamate NSAIDs have neuroprotective properties against ischemic stroke and to explore the underlying mechanisms for any efficacy. Fenamates are non-selective inhibitors of cyclooxygenases. In addition, fenamates are antagonists of non-selective cation
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Weiss, Miriam [Verfasser], Gerrit Alexander [Akademischer Betreuer] Schubert, and Martin [Akademischer Betreuer] Wiesmann. "Endovascular rescue treatment for delayed cerebral ischemia after subarachnoid hemorrhage is safe and effective / Miriam Weiss ; Gerrit Alexander Schubert, Martin Wiesmann." Aachen : Universitätsbibliothek der RWTH Aachen, 2019. http://d-nb.info/1215927614/34.

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Boulos, Sherif. "Identification and characterisation of potential neuroprotective proteins induced by erythropoietin (EPO) preconditioning of cortical neuronal cultures." University of Western Australia. School of Biomedical and Chemical Sciences, 2008. http://theses.library.uwa.edu.au/adt-WU2008.0128.

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[Truncated abstract] Clinical therapeutic agents to directly inhibit ischaemic neuronal death are presently unavailable. One approach to developing therapeutics is based upon the identification of proteins up-regulated by 'preconditioning', a natural adaptive response utilised by the neural cells to counter damaging insults, such as ischaemia. Thus, my project aimed to firstly identify proteins differentially expressed following erythropoietin (EPO) mediated neuronal preconditioning and secondly to assess whether any of these proteins possessed neuroprotective activity using in vitro ischaemia
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Rahman, Rosanna, and n/a. "Potential causes of the delayed neural damage observed post-stroke & the effects of epigallocatechin gallate administration." University of Otago. Department of Pharmacology & Toxicology, 2006. http://adt.otago.ac.nz./public/adt-NZDU20070508.122246.

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Stroke is the 3rd leading cause of death and the leading cause of major disability worldwide. Currently, there are no neuroprotective drugs approved for the acute treatment of ischaemic stroke. The vast majority of stroke therapeutics failed in clinical trials due to toxic side effects and/or a clinically irrelevant therapeutic window. This thesis is focused on exploiting the delayed neurodegeneration that occurs in the compromised penumbra, as these cells may be capable of being saved by therapeutic intervention in a clinically obtainable window. In order to investigate the ischaemic cascade
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Pütz, Volker, Matthias Weise, Rüdiger von Kummer, and Georg Gahn. "Effective Treatment with Abciximab for Consecutive Bilateral Middle Cerebral Artery Occlusion." Saechsische Landesbibliothek- Staats- und Universitaetsbibliothek Dresden, 2014. http://nbn-resolving.de/urn:nbn:de:bsz:14-qucosa-135290.

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Pütz, Volker, Matthias Weise, Rüdiger von Kummer, and Georg Gahn. "Effective Treatment with Abciximab for Consecutive Bilateral Middle Cerebral Artery Occlusion." Karger, 2006. https://tud.qucosa.de/id/qucosa%3A27636.

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Brodin, Camille. "De la paillasse au lit du patient, surmonter les problèmes de translation dans le domaine de l'AVC ischémique Single- and two- chain tissue plasminogen activator treatment differentially influence cerebral recovery after stroke Single- and two- chain tissue plasminogen activator treatment differentially influence cerebral recovery after stroke Cerebral blood flow correlates with ischemic brain lesion only when Stroke occurs awake: a preclinical model to bypass the translational roadblocks to clinic." Thesis, Normandie, 2019. http://www.theses.fr/2019NORMC427.

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Les défauts de translation des études précliniques vers les essais cliniques dans le domaine des AVC ischémiques et l'échec des développements thérapeutiques pourraient être expliqués par trois aspects : (1) le manque de compréhension des mécanismes des deux formes de rtPA, le traitement pharmacologique de l'AVC; (2) le manque d'outils adaptés d'imagerie de perfusion chez le petit animal et (3) l'influence de l'anesthésie sur l’effet des traitements testés dans les modèles animaux.Le tPA utilisé en clinique (Actilyse®) est un mélange de deux formes de tPA: une forme chaîne simple (sc-rtPA) et
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Bücher zum Thema "Cerebral ischemia – Treatment"

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Treatment of cerebral infarction: Experimental and clinical study. Wien: Springer-Verlag, 1987.

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Noé, Battistini, ed. Acute brain ischemia: Medical and surgical therapy. New York: Raven Press, 1986.

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1931-, Robertson James T., Nowak Thaddeus S, and Princeton Conference on Cerebrovascular Disease (20th : 1996 : Memphis, Tenn.), eds. Frontiers in cerebrovascular disease: Mechanisms, diagnosis, and treatment. Armonk, NY: Futura Pub. Co., 1998.

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M, Maier Carolina, and Steinberg Gary K, eds. Hypothermia and cerebral ischemia: Mechanisms and clinical applications. Totowa, N.J: Humana Press, 2004.

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Eric, Kasner Scott, and Gorelick Philip B, eds. Prevention and treatment of ischemic stroke. Philadelphia: Butterworth-Heinemann, 2004.

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1946-, Feuerstein Giora Z., ed. Inflammation and stroke. Basel: Birkhäuser, 2001.

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M, Barnett Henry J., and Hachinski Vladimir, eds. Cerebral ischemia: Treatment and prevention. Philadelphia: Saunders, 1992.

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Therapeutic Strategies In Cerebral Ischemia. Clinical Pub, 2011.

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R, Caplan Louis, ed. Brain ischemia: From basic science to treatment. London: Springer-Verlag, 1994.

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(Editor), J. Bogousslavsky, D. Guez (Editor), and Jose Biller (Editor), eds. Cerebral Ischemia: From Pathophysiology to Treatment - Journal: Cerebrovascular Diseases, Suppl. 1, 1991 (Cerebral Ischemia). S. Karger AG (Switzerland), 1991.

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Buchteile zum Thema "Cerebral ischemia – Treatment"

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Hacke, Werner, Herman J. Gelmers, Michael Hennerici, and Günter Krämer. "Treatment and Prophylaxis." In Cerebral Ischemia, 189–222. Berlin, Heidelberg: Springer Berlin Heidelberg, 1991. http://dx.doi.org/10.1007/978-3-642-75548-4_6.

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Hayashi, Nariyuki, and Dalton W. Dietrich. "Global and Focal Cerebral Ischemia." In Brain Hypothermia Treatment, 7–8. Tokyo: Springer Japan, 2004. http://dx.doi.org/10.1007/978-4-431-53953-7_2.

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O’Brien, M. D. "Academic Hemianopia and the Treatment of Cerebrovascular Disease." In Cerebral Ischemia and Calcium, 3–6. Berlin, Heidelberg: Springer Berlin Heidelberg, 1989. http://dx.doi.org/10.1007/978-3-642-85863-5_1.

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Soyka, D. "Treatment of Acute Migraine Attacks with Intravenous Flunarizine." In Cerebral Ischemia and Calcium, 403–9. Berlin, Heidelberg: Springer Berlin Heidelberg, 1989. http://dx.doi.org/10.1007/978-3-642-85863-5_53.

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Meyer, J. S., J. Lotfi, and M. Kobari. "Treatment of Migraine and Cluster Headache with Calcium Antagonists." In Cerebral Ischemia and Calcium, 410–18. Berlin, Heidelberg: Springer Berlin Heidelberg, 1989. http://dx.doi.org/10.1007/978-3-642-85863-5_54.

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Bowen, D. M., P. T. Francis, S. L. Lowe, M. N. Pangalos, A. W. Procter, and J. E. Steele. "Alzheimer’s Disease: Role of Energy Metabolism and Treatment of Symptoms." In Cerebral Ischemia and Dementia, 87–93. Berlin, Heidelberg: Springer Berlin Heidelberg, 1991. http://dx.doi.org/10.1007/978-3-642-76208-6_11.

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Vorstrup, S., O. B. Paulson, and A. Andersen. "CBF Decreases in Ischemic Areas After Calcium Antagonist Treatment in Acute Stroke." In Cerebral Ischemia and Calcium, 378–83. Berlin, Heidelberg: Springer Berlin Heidelberg, 1989. http://dx.doi.org/10.1007/978-3-642-85863-5_49.

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Böker, D. K., and R. Wüllenweber. "Treatment of Symptomatic Cerebral Vasospasm After Subarachnoid Hemorrhage by Intraarterial Perfusion with Nimodipine." In Cerebral Ischemia and Calcium, 547–55. Berlin, Heidelberg: Springer Berlin Heidelberg, 1989. http://dx.doi.org/10.1007/978-3-642-85863-5_68.

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Teasdale, G. M., J. D. Pickard, G. Murray, R. Illingworth, M. D. M. Shaw, P. Foy, P. Humphrey, et al. "Treatment of Subarachnoid Hemorrhage with Oral Nimodipine: Preliminary Report of the British Aneurysm Trial." In Cerebral Ischemia and Calcium, 563–67. Berlin, Heidelberg: Springer Berlin Heidelberg, 1989. http://dx.doi.org/10.1007/978-3-642-85863-5_70.

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Yenari, Midori A., David C. Tong, and Gregory W. Albers. "Glycine Antagonists for Treatment of Ischemic Brain Injury." In Clinical Pharmacology of Cerebral Ischemia, 127–51. Totowa, NJ: Humana Press, 1997. http://dx.doi.org/10.1007/978-1-59259-472-6_6.

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Konferenzberichte zum Thema "Cerebral ischemia – Treatment"

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Киреева, Виктория, Viktoriya Kireeva, Ю. Усольцев, Yu Usolcev, Ж. Капустенская, Zh Kapustenskaya, Е. Кожевникова, et al. "Intermediate results 2016 of a search study of translational diagnostic methods Mitochondrial dysfunction in patients with chronic myocardial ischemia and/or head Brain." In Topical issues of translational medicine: a collection of articles dedicated to the 5th anniversary of the day The creation of a department for biomedical research and technology of the Irkutsk Scientific Center Siberian Branch of RAS. Москва: INFRA-M Academic Publishing LLC., 2017. http://dx.doi.org/10.12737/conferencearticle_58be81ec94893.

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Purpose of the study. To rate prognostic properties of changes in mitochondrial DNA concentration in the blood plasma of patients with chronic cerebral ischemia and ischemic heart disease in relation to the disease and the effectiveness of the therapy. Materials and methods. The study involved patients suffering from coronary heart disease (CHD) and chronic cerebral ischemia (CCI) with stable and unstable atherosclerotic plaques, who have signed informed consent to the data processing within the framework of scientific research. The patients were admitted to the hospital for examination and tr
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Zhu, Liang, and Axel J. Rosengart. "Cooling Penetration Surrounding an Intra-Parenchymal Cooling Probe in Hypothermia Treatment for Ischemia or Head Injury Patients: Theoretical Analyses." In ASME 2004 International Mechanical Engineering Congress and Exposition. ASMEDC, 2004. http://dx.doi.org/10.1115/imece2004-61109.

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Inducing hypothermia to brain tissue after brain ischemia or head injury has been demonstrated beneficial to the patients. Clinical studies have shown that even 1 or 2°C temperature reduction in brain tissue can be protective [Dietrich 1992]. On the contrary, fever-induced hyperthermia can worsen the neurological outcome in an animal model after cerebral ischemia. It is of clinical importance to understand the temperature distribution in brain during brain hypothermia.
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Sazonova, O. B., and E. M. Troshina. "REFLECTION IN THE EEG OF DISORDERS OF CEREBRAL HEMODYNAMICS IN CHRONIC CEREBRAL ISCHEMIA IN CHILDREN." In NOVEL TECHNOLOGIES IN MEDICINE, BIOLOGY, PHARMACOLOGY AND ECOLOGY. Institute of information technology, 2022. http://dx.doi.org/10.47501/978-5-6044060-2-1.368-375.

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The paper presents the results of a study of the bioelectrical activity of the brain in 77 patients aged 2 to 18 years old with chronic cerebral ischemia, due to pathology of the main arteries of the brain, observed and operated in the Institute of Neurosurgery of N.N. Burdenko from 2012. The study showed the role of EEG in assessing the functional state of the brain in chil-dren with chronic cerebrovascular insufficiency of various etiopathogenesis in children, de-pending on the age of the child. Comparison of EEG data in extra- and intracranial cerebral vascular pathology was performed. Reve
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Silva, Tibério Alves da, Ana Carolina Soares de Lira, Bárbara Letícia Barreto Ramos Aragão, and Luciana Karla, Dayanna Grazielle Maia Viana. "Carotid endarterectomy as the treatment of choice for clearing the internal carotid artery in transitional ischemic attacks." In XIII Congresso Paulista de Neurologia. Zeppelini Editorial e Comunicação, 2021. http://dx.doi.org/10.5327/1516-3180.303.

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Introduction: TIAs are ischemia, caused by stenosis of the carotid and vertebral arteries. Those who have a TIA are at risk of ischemic stroke and myocardial infarction, with carotid endarterectomy being an intervention. Objective: To analyze the benefits of carotid endarterectomy using drugs. Methods: Literature review, in bases such as PUBMED, MEDLINE, descriptors: “Endarterectomy”, “Ischemic Attack”, with operator “AND” and “OR”. Those with two descriptors were selected in the summary and date between 2010-2020, English / Portuguese language, resulting in: 17 articles. Results: The internal
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Pastorio, Indianara Keila, Nayara Christina de Lima Curti, Francine de Paula Roberto Domingos, Sayuri Aparecida Hirayama, Lorena Dias Araújo, Rafael de Almeida, Marilia Pires de Sousa e. Silva, and William de Souza Delfim. "Post-traumatic vertebral artery dissection." In XIII Congresso Paulista de Neurologia. Zeppelini Editorial e Comunicação, 2021. http://dx.doi.org/10.5327/1516-3180.515.

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Introduction: Vertebral artery dissections are responsible for 2% of all ischemic strokes and correspond to more than 25% of events in young adults. Its main etiologies, such as spontaneous and traumatic, which compromise the structural integrity of the arterial wall, allowing blood to accumulate between the layers of the vessel as an intramural hematoma, which can progress to stenosis or luminal occlusion in stroke. Case report: We attended a woman, 29 years old, who suffered a mild trauma in the cervical region during weight training in the gym presenting symptoms hours after the accident, a
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Brandão, André Iglesias, Luiz Paulo Bastos Vasconcelos, Carolina de Almeida e. Silva, Raul de Barros Valente, Danilo Jorge da Silva, Pedro Ivo Machado Campos de Araujo Costa, Matheus Henrique Freitas Silva, et al. "Brain Ischemia associated with COVID-19 and PFO - Case Report." In XIII Congresso Paulista de Neurologia. Zeppelini Editorial e Comunicação, 2021. http://dx.doi.org/10.5327/1516-3180.287.

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Context: COVID19 has better known respiratory impacts than cardiovascular1 and high D-dimer as the most significant coagulation parameter2 . Otherwise, paradoxical embolism due to Patent Foramen Ovale (PFO) and Ischaemic Cerebral Vascular Accident (iCVA) mechanisms associated are poorly documented3, 4 . We aimed to report an associated case of iCVA and PFO and highlight COVID19 hypercoagulability triggering thromboembolisms. Case report: CTT, 76y, female, hypertensive, former-smoker was hospitalized with right-hemiplegia, transcortical aphasia, dysarthria by iCVA and evolved with headache, ody
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Strano, A., G. Davi, I. Catalano, G. Francavilla, and A. Notarbartolo. "IS BETATHROMBOGLOBULIN (BTG) OF PROGNOSTIC VALUE IN PATIENTS WITH TRANSIENT CEREBRAL ISCHEMIA (TIA) ?" In XIth International Congress on Thrombosis and Haemostasis. Schattauer GmbH, 1987. http://dx.doi.org/10.1055/s-0038-1643055.

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TIA and stroke have for a long time been recognized as being associated with various abnormalities in platelet function.Plasma levels of BTG, a marker of platelet release reaction, have been found raised in TIA patients. However plasma measurements can be influenced by venipuncture or handling of the samples Therefore we measured urinary BTG levels and compared plasma and urinary levels of this protein in 18 patients with TIA and 18 controls of equivalent age (41-68 years) and sex.Plasma BTG levels were not different between TIA subjects (24.1 ± 8.6 ng/ml) and controls (20.2 ± 6.4 ng/ml). Urin
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Signorelli, C. D., A. Giaquinta, G. Iofrida, G. Donato, Fr Signorelli, C. Bellecci, T. Lo Feudo, P. Gaudio, and M. Gelfusa. "Surgical treatment of cerebral ischemia by means of diode laser: first experimental results and comparison with theoretical model." In European Conference on Biomedical Optics. Washington, D.C.: OSA, 2007. http://dx.doi.org/10.1364/ecbo.2007.6632_7.

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Signorelli, C. D., A. Giaquinta, G. Iofrida, G. Donato, Fr Signorelli, C. Bellecci, T. Lo Feudo, P. Gaudio, and M. Gelfusa. "Surgical treatment of cerebral ischemia by means of diode laser: first experimental results and comparison with theoretical model." In European Conference on Biomedical Optics, edited by Alfred Vogel. SPIE, 2007. http://dx.doi.org/10.1117/12.728256.

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Depieri, Lorena Dellagnesi, and Lorena Souza Viana. "Case report: Intracranial Arterial Dolichoectasia." In XIII Congresso Paulista de Neurologia. Zeppelini Editorial e Comunicação, 2021. http://dx.doi.org/10.5327/1516-3180.739.

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Introduction: Intracranial arterial dolichoectasia (IADE) is a diameter increase and/ or a long and tortuous path in one or more Intracranial arterial. Most patients keep asymptomatic and eventually present neurological complications (ischemia, bleeding or compression of adjacent structures). At around 12% of the patients with stroke present this kind of intracranial dilated, which 80% are in the posterior cerebral circulation, mainly in basilar artery. Objective: Report an unusual case of IADE refractory to the clinical treatment. Method: The information was obtained by reviewing the medical
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Berichte der Organisationen zum Thema "Cerebral ischemia – Treatment"

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Wu, Xiaoqi, Maoxia Fan, Yaobo Pan, and Dona Guo. Quality of Evidence Supporting the Effects of Ginkgo Terpene Lactone Preparations in Ischemic Stroke: An Overview of Systematic Reviews and Meta-Analyses. INPLASY - International Platform of Registered Systematic Review and Meta-analysis Protocols, September 2022. http://dx.doi.org/10.37766/inplasy2022.9.0124.

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Review question / Objective: 2.2.1 Type of studies SRs/MAs of Randomized Controlled Trials (RCTs) of GTLP for IS in any language. 2.2.2 Type of Participants Included patients were diagnosed with IS according to international or national standards, regardless of race, age, gender, time of onset, and source of cases. 2.2.3 Type of Intervention The intervention method in the control group was routine treatment, and the intervention method in the intervention group was GTLP treatment or GTLP combined with the treatment of the control group. 2.2.4 Types of outcomes Conclusions at least need to incl
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2

Yu, Beibei, Yongfeng Zhang, and Shouping Gong. Effects of miRNA-modified exosomes alleviate cerebral ischemic reperfusion injury in Pre-clinical Studies: A Meta-analysis. INPLASY - International Platform of Registered Systematic Review and Meta-analysis Protocols, May 2022. http://dx.doi.org/10.37766/inplasy2022.5.0062.

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Review question / Objective: The purpose of this study was to investigate the effect of miRNA-modified exosomes in alleviating cerebral ischemic reperfusion injury compared with the non-treatment group. The research object is an animal model of middle cerebral artery occlusion. The research method is a controlled study. The primary outcome of this study was infarct volume, and the secondary outcome was neurobehavioral performance. Main outcome(s): The primary outcome of this study was Infarct volumes，which was measured by 2,3,5-triphenyltetranzolium chloride (TTC) staining. And it was calculat
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