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Thematische Bibliographien / Chromaffin cells / Dissertationen

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Dissertationen zum Thema „Chromaffin cells“

Autor: Grafiati

Veröffentlicht am 4. Juni 2021

Zuletzt aktualisiert am 27. Januar 2023

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1

Owen, Penelope Jane. "Bradykinin stimulation of bovine adrenal chromaffin cells." Thesis, University of Leicester, 1991. http://hdl.handle.net/2381/33600.

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Cultured bovine adrenal chromaffin cells provide a useful model of stimulus secretion coupling and respond to cholinergic agonists by secreting catecholamines. Work in this thesis concentrates on the responses to a non-cholinergic agonist, bradykinin. Bradykinin as shown to stimulate a two phase, dose dependent increase in catecholamine release which is mediated by a receptor of the B2 subtype. Calcium entry is shown to be required for release to occur but studies with various calcium channel blockers suggest that, in contrast to the response to potassium, a non-voltage sensitive calcium chann

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2

Pappas, Vassilios Konstantinos. "Ca2+ signalling in bovine adrenal chromaffin cells." Thesis, University of Leicester, 1995. http://hdl.handle.net/2381/33634.

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Cells possess two mechanisms, the inositol 1,4,5-trisphosphate (Ins(l,4,5)P3) and ryanodine receptors, by which Ca2+ in intracellular stores can be mobilised. There are also a number of pathways which can mediate Ca2+ entry across the cell plasma membrane. In this study bovine adrenal chromaffin cells were used to investigate the role of intracellular Ca2+ stores in Ca2+ signalling and the relationship between Ca2+ entry and store release. The major part of the exocytotic process in chromaffin cells is due to Ca2+ entry across the plasma membrane. Bradykinin (an Ins(l,4,5)P3 generating agonist

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3

Hagan, Todd. "Finite-difference time-domain modeling of a waveguide-based radiofrequency exposure system for studying non-thermal effects on catecholamine release from chromaffin cells : characterization and optimization /." abstract and full text PDF (free order & download UNR users only), 2005. http://0-wwwlib.umi.com.innopac.library.unr.edu/dissertations/fullcit/1433103.

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Thesis (M.S.)--University of Nevada, Reno, 2005.<br>"May, 2005." Includes bibliographical references. Library also has microfilm. Ann Arbor, Mich. : ProQuest Information and Learning Company, [2005]. 1 microfilm reel ; 35 mm. Online version available on the World Wide Web.

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4

gov, Clearys@ninds nih, and Susannah Cleary. "From chromaffin cells to Phaeochromocytoma : insight into the sympathoadrenal cell lineage." Murdoch University, 2007. http://wwwlib.murdoch.edu.au/adt/browse/view/adt-MU20080526.105525.

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Chromaffin cells are a modified post-ganglionic sympathetic neuron, which synthesise and secrete catecholamines. The neoplastic transformation of chromaffin cells is demonstrated by the tumour phaeochromocytoma, a functional tumour that recapitulates the normal role of chromaffin cells by synthesising, storing and releasing excess catecholamines. Within this thesis we have explored several aspects of chromaffin cell and phaeochromocytoma tumour biology, including the specific expression of key sympathoadrenal markers such as the noradrenaline transporter (NAT), neuropeptide Y (NPY) and chromog

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5

Cleary, Susannah. "From chromaffin cells to Phaeochromocytoma: insight into the sympathoadrenal cell lineage." Thesis, Cleary, Susannah (2007) From chromaffin cells to Phaeochromocytoma: insight into the sympathoadrenal cell lineage. PhD thesis, Murdoch University, 2007. https://researchrepository.murdoch.edu.au/id/eprint/659/.

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Chromaffin cells are a modified post-ganglionic sympathetic neuron, which synthesise and secrete catecholamines. The neoplastic transformation of chromaffin cells is demonstrated by the tumour phaeochromocytoma, a functional tumour that recapitulates the normal role of chromaffin cells by synthesising, storing and releasing excess catecholamines. Within this thesis we have explored several aspects of chromaffin cell and phaeochromocytoma tumour biology, including the specific expression of key sympathoadrenal markers such as the noradrenaline transporter (NAT), neuropeptide Y (NPY) and chromog

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6

Cleary, Susannah. "From chromaffin cells to Phaeochromocytoma: insight into the sympathoadrenal cell lineage." Cleary, Susannah (2007) From chromaffin cells to Phaeochromocytoma: insight into the sympathoadrenal cell lineage. PhD thesis, Murdoch University, 2007. http://researchrepository.murdoch.edu.au/659/.

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Chromaffin cells are a modified post-ganglionic sympathetic neuron, which synthesise and secrete catecholamines. The neoplastic transformation of chromaffin cells is demonstrated by the tumour phaeochromocytoma, a functional tumour that recapitulates the normal role of chromaffin cells by synthesising, storing and releasing excess catecholamines. Within this thesis we have explored several aspects of chromaffin cell and phaeochromocytoma tumour biology, including the specific expression of key sympathoadrenal markers such as the noradrenaline transporter (NAT), neuropeptide Y (NPY) and chromog

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7

Zhu, Jinghua. "The transformation of chromaffin cells into sympathetic neurons." Thesis, University of Oxford, 1994. http://ethos.bl.uk/OrderDetails.do?uin=uk.bl.ethos.386926.

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8

Robinson, Iain Martin. "Ca'2'+ signalling in bovine adrenal chromaffin cells." Thesis, University of Liverpool, 1992. http://ethos.bl.uk/OrderDetails.do?uin=uk.bl.ethos.317281.

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9

Fisher, Richard James. "Amperometric analysis of exocytosis in adrenal chromaffin cells." Thesis, University of Liverpool, 2000. http://ethos.bl.uk/OrderDetails.do?uin=uk.bl.ethos.367144.

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10

Powell, Andrew Dennis. "Modulation of neurotransmitter release from adrenal chromaffin cells." Thesis, University of Bristol, 2000. http://ethos.bl.uk/OrderDetails.do?uin=uk.bl.ethos.310685.

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11

Fournier, Sue. "Calmodulin binding proteins in chromaffin and other neurosecretory cells." Thesis, McGill University, 1988. http://digitool.Library.McGill.CA:80/R/?func=dbin-jump-full&object_id=75881.

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The Ca$ sp{2+}$ binding protein, calmodulin, has been implicated in several Ca$ sp{2+}$ dependent processes during secretion in many different secretory systems. One area where calmodulin is suggested to play a role is the fusion of the secretory granule with the plasma membrane during exocytosis. Calmodulin may mediate the interaction or fusion through specific calmodulin-binding proteins (CMBPs) present in these two membranes.<br>CMBPs present in bovine chromaffin cell granule membranes were characterized using the techniques of calmodulin affinity chromatography and $ sp{125}$I calmodulin o

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12

Walker, Angela. "Electrochemical study of vesicular release in bovine chromaffin cells." Thesis, McGill University, 1995. http://digitool.Library.McGill.CA:80/R/?func=dbin-jump-full&object_id=23431.

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The time course of the spontaneous current spikes produced by the release of the catecholamines from individual vesicles was examined in bovine chromaffin cells by using the carbon filament technique in the amperometric mode.<br>Frequency histograms of the rise and decay times of the current spikes showed a paucity of very short duration events. Scatterograms of the rise and decay times consistently showed a positive relation, and the best fitted lines intercepted the ordinate (the axis of the decay time) at: 16.06 $ pm$ 6.45 msec (n = 11).<br>The effect of temperature changes upon the time co

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13

O'Sullivan, Antony John. "The control of secretion from bovine adrenal chromaffin cells." Thesis, University of Liverpool, 1989. http://ethos.bl.uk/OrderDetails.do?uin=uk.bl.ethos.333504.

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14

Cheek, T. R. "Molecular mechanisms of secretion from bovine adrenal chromaffin cells." Thesis, University of Liverpool, 1987. http://ethos.bl.uk/OrderDetails.do?uin=uk.bl.ethos.382015.

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15

Morgan, Alan. "Molecular mechanisms of exocytosis from bovine adrenal chromaffin cells." Thesis, University of Liverpool, 1993. http://ethos.bl.uk/OrderDetails.do?uin=uk.bl.ethos.385297.

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16

Colville, Caroline Anne. "The interaction of tetanus toxin with adrenal chromaffin cells." Thesis, University of Edinburgh, 1992. http://hdl.handle.net/1842/19641.

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Tetanus toxin exerts its primary biological effect by impairing the release of inhibitory neurotransmitters in the central nervous system; the mechanism of this blockade, however, remains unknown. Studies using adrenal medullary chromaffin cells, which are closely related to the toxin's target neurones but much more accessible to biochemical investigation, have the potential to provide information on various aspects of the intoxication process of tetanus. Nicotine-evoked (but not basal) secretion of catecholamines from intact chromaffin cells was inhibited by tetanus toxin in a dose-dependent

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17

Lawrence, Gary William. "Dissecting exocytosis from chromaffin cells with botulinum and tetanus toxins." Thesis, Imperial College London, 1995. http://ethos.bl.uk/OrderDetails.do?uin=uk.bl.ethos.321807.

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18

Doreian, Bryan William. "Molecular Regulation of the Exocytic Mode in Adrenal Chromaffin Cells." Cleveland, Ohio : Case Western Reserve University, 2009. http://rave.ohiolink.edu/etdc/view?acc_num=case1245785721.

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Thesis (Ph.D.)--Case Western Reserve University, 2009<br>Title from PDF (viewed on 19 August 2009) Department of Physiology and Biophysics Includes abstract Includes bibliographical references Available online via the OhioLINK ETD Center

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19

El-Hajj, Raed Ahmad. "Pharmacological and immunological identification of native [alpha]7 nicotinic receptors: evidence for homomeric and heteromeric [alpha]7 receptors /." Columbus, Ohio : Ohio State University, 2008. http://rave.ohiolink.edu/etdc/view?acc%5Fnum=osu1198155366.

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20

Geertsen, Susanne. "The regulation of nicotinic alpha-bungarotoxin receptors in adrenal chromaffin cells." Thesis, McGill University, 1992. http://digitool.Library.McGill.CA:80/R/?func=dbin-jump-full&object_id=39391.

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The present studies investigate the molecular mechanisms involved in the nicotinic antagonist-induced up-regulation of $ alpha$-bungarotoxin ($ alpha$-BGT) binding sites in bovine adrenal chromaffin cells in culture. To assess the possibility that cellular depolarization and/or the activation of protein kinase C might be involved in the nicotinic antagonist-induced up-regulation, experiments were carried out in which nicotinic antagonists, K$ sp+$ and/or phorbol esters were added to the cells in various combinations. These latter two treatments resulted in increases in the number of toxin bind

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21

Samasilp, Prattana. "MOLECULAR CONTROL OF FUSION PORE EXPANSION IN MOUSE ADRENAL CHROMAFFIN CELLS." Case Western Reserve University School of Graduate Studies / OhioLINK, 2014. http://rave.ohiolink.edu/etdc/view?acc_num=case1396426579.

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22

Wenger, Bryan W. "Expression of multiple populations of nicotinic acetylcholine receptors in bovine adrenal chromaffin cells." Connect to this title online, 2003. http://rave.ohiolink.edu/etdc/view?acc%5Fnum=osu1070496780.

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Thesis (Ph. D.)--Ohio State University, 2003.<br>Title from first page of PDF file. Document formatted into pages; contains xii, 120 p.; also includes graphics. Includes bibliographical references (p. 110-120).

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23

Dumpala, Bindya. "Design, construction, optimization, and characterization of a temperature control system for studying the effects of a rapid and reversible changes in temperature on neurosecretion." abstract and full text PDF (free order & download UNR users only), 2006. http://0-gateway.proquest.com.innopac.library.unr.edu/openurl?url_ver=Z39.88-2004&rft_val_fmt=info:ofi/fmt:kev:mtx:dissertation&res_dat=xri:pqdiss&rft_dat=xri:pqdiss:1438916.

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24

Villanueva, Melissa Wightman R. Mark. "Electrochemical detection of modulation of exocytosis from chromaffin cells monitored with amperometry." Chapel Hill, N.C. : University of North Carolina at Chapel Hill, 2006. http://dc.lib.unc.edu/u?/etd,378.

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Thesis (Ph. D.)--University of North Carolina at Chapel Hill, 2006.<br>Title from electronic title page (viewed Oct. 10, 2007). "... in partial fulfillment of the requirements for the degree of Doctor of Philosophy in the Department of Chemistry." Discipline: Chemistry; Department/School: Chemistry.

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25

Drake, Julie. "Effect of Choline on Ca2+ -activated K+ channels in bovine chromaffin cells." Thesis, McGill University, 1990. http://digitool.Library.McGill.CA:80/R/?func=dbin-jump-full&object_id=60447.

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The actions of internal choline on single "maxi" Ca$ sp{2+}$-activated K$ sp{+}$ channels were studied in excised patches of chromaffin cell membrane. The channels had a unit conductance of approximately 200 pS in a physiological K$ sp{+}$ gradient.<br>Choline (20-70 mM) applied to the intracellular membrane surface dose dependently reduced outward current flow through the channel. The reduction in single channel current amplitude increased with depolarization.<br>These data suggest that choline is a fast blocker that binds within the channel pore. The K$ sb{ rm d}$(0) for the reaction is 90 m

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26

Roth, Dagmar. "The role of proteins in regulated exocytosis from bovine adrenal chromaffin cells." Thesis, University of Liverpool, 1995. http://ethos.bl.uk/OrderDetails.do?uin=uk.bl.ethos.284252.

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27

Jonsson, Anna. "Reference interval for urinary catecholamines and methylated catecholamines analysed using HPLC." Thesis, Uppsala universitet, Institutionen för medicinsk biokemi och mikrobiologi, 2012. http://urn.kb.se/resolve?urn=urn:nbn:se:uu:diva-180252.

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Catecholamines are stress hormones that are produced and released by a rare tumor called pheochromocytoma. This tumor can cause hypertension which if undiagnosed and untreated leads to death. Since good therapy is available, it is important to find the tumor in time. The most common way to diagnose the tumor is measurement of the biochemical markers; catecholamines and their metabolites, methylated catecholamines. After observation that almost all normetanephrine results for women were higher than the upper reference limit and therefore pathological, the accuracy of the present reference inter

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28

Yoon, Jihwan. "Non-thermal effects of pulsed-microwave fields on catecholamine release from chromaffin cells exposure system design and characterization and experimental data /." abstract, 2008. http://0-gateway.proquest.com.innopac.library.unr.edu/openurl?url_ver=Z39.88-2004&rft_val_fmt=info:ofi/fmt:kev:mtx:dissertation&res_dat=xri:pqdiss&rft_dat=xri:pqdiss:3339154.

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29

Al-Rasheed, Nouf. "Receptor-mediated catecholamine release from chromaffin cells : the role of protein kinase C." Thesis, University of Leicester, 2006. http://hdl.handle.net/2381/29950.

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This study reveals that bovine chromaffin cells express PKC-alpha, -beta, -epsilon and -iota and that they were differentially activated by nicotine receptors and a range of Gaq/11-coupled GPCRs. Thus, nicotinic receptor stimulation recruited PKC-alpha, -beta and -epsilon from the cytoplasm to the plasma membrane, indicative of activation. In contrast, activation of Gaq/11-coupled receptors with histamine activated all the expressed PKC isoforms and angiotensin II only activated PKC-alpha and -epsilon.;Inhibition of PKC using general or isoform-selective inhibitors potentiated catecholamine re

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30

Wykes, Robert C. E. "Calmodulin regulation of calcium channels and neurotransmitter release in bovine adrenal chromaffin cells." Thesis, University of Leicester, 2004. http://hdl.handle.net/2381/29943.

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Calmodulin is a molecule implicated in regulating voltage-gated calcium channels (VGCCs) and the exocytotic machinery to fine tune neurotransmitter release. I have investigated the role this molecule plays in stimulus-secretion coupling in bovine adrenal chromaffin cells by over-expressing either wild-type (CaMwt), or a mutated calmodulin (CaM1234), rendered incapable of binding calcium by adenoviral infection. Stimulus-evoked secretion was monitored by combined measurements of membrane capacitance (DeltaC m) and voltage-clamp recording of calcium currents in single cells. Cells were clamped i

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Dry, Katherine L. "Catecholamine release from isolated chromaffin cells under conditions of anoxia or metabolic inhibition." Thesis, University of Edinburgh, 1990. http://hdl.handle.net/1842/18845.

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A significant release of catecholamines within the heart has been observed during myocardial ischaemia. Ischaemia-induced catecholamine release can be markedly inhibited by desipramine and other amine uptake blocking agents, allowing investigation of the importance of such release for arrhythmia production. The mechanism of this release appears to occur by a carrier-mediated efflux from neurones, which is not operative under normal conditions. The aim of the project has been to study this release process in chromaffin cells isolated from the bovine adrenal medulla, which are recognised as a mo

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32

Wenger, Bryan William. "Expression of multiple populations of nicotinic acetylcholine receptors in bovine adrenal chromaffin cells." The Ohio State University, 2003. http://rave.ohiolink.edu/etdc/view?acc_num=osu1070496780.

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33

Förander, Petter. "On the actions of neurotrophic factors on the chromaffin cells of the adrenal medulla /." Stockholm, 2000. http://diss.kib.ki.se/2000/91-628-4145-9/.

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34

Joshi, Atul. "A possible link between calmodulin and calcium-activated potassium channels in bovine chromaffin cells /." Thesis, McGill University, 1989. http://digitool.Library.McGill.CA:80/R/?func=dbin-jump-full&object_id=55663.

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35

Milman, Alexandre. "Contribution à l'étude du rôle physiologique du canal de fuite sodique NALCN dans les cellules excitables : approche sur cellules chromaffines de souris." Thesis, Montpellier, 2018. http://www.theses.fr/2018MONTT060.

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Les cellules chromaffines des glandes surrénales sont des cellules neuroendocrines excitables impliquées dans la sécrétion de catécholamines. En réponse à un stress, ces hormones, parmi les premières à être libérées exercent de multiples actions sur leurs organes-cibles, contribuant à la réponse adaptive de l'organisme. Ainsi, élucider la physiopathologie du stress est un enjeu de santé publique et mieux connaître les mécanismes permettant au tissu médullosurrénalien d'optimiser la sécrétion de catécholamines aux besoins de l'organisme est un défi à relever.La sécrétion des catécholamines est

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36

Karaplis, Andy C. "The role of prostaglandin E2 and other eicosanoids in catecholamine secretion from adrenal chromaffin cells /." Thesis, McGill University, 1987. http://digitool.Library.McGill.CA:80/R/?func=dbin-jump-full&object_id=75418.

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Prostaglandins E$ sb1$ and E$ sb1$ are bound with high affinity and specificity to particulate fractions from bovine, ovine and human adrenal medulla. An identical fraction from fetal bovine and ovine adrenals binds PGEs with similar kinetic properties and specificities. The ${ rm lbrack sp3H rbrack}$PGE$ sb2$-binding site complex dissociates in a biphasic manner, suggesting the presence of both a low and high affinity form of the complex. Gpp(NH)p, a stable GTP analogue, inhibits PGE$ sb2$ binding by promoting the conversion of the high affinity form of the complex to the lower affinity state

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37

Estay, Ahumada Catherine. "Mécanismes moléculaires du couplage exocytose-endocytose dans les cellules neuroendocrines : rôle des protéines Scramblase-1 et Oligophrénine-1." Thesis, Strasbourg, 2016. http://www.theses.fr/2016STRAJ088/document.

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De récentes études ont montré dans les cellules chromaffines que la libération des granules de sécrétion est temporellement et spatialement couplée au processus d’endocytose. Nous avons proposé l’hypothèse que la membrane du granule préserve son intégrité au sein de la membrane plasmique durant l’exocytose avant d’être internalisée ainsi avec ses composants. Cependant, les mécanismes moléculaires de ce processus d’endocytose compensatrice sont encore inconnus. Ainsi, mon projet de thèse vise a répondre à la question suivante : Quels sont les différents mécanismes déclenchant et régulant l’exoc

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38

Tapechum, Sompol. "Role of Doc2β in regulated exocytosis of large dense-core vesicles in bovine adrenal chromaffin cells". Thesis, University of Edinburgh, 2003. http://hdl.handle.net/1842/27506.

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39

Bonifas-Arredondo, Imelda. "Amperometric determination of the flux of neurotransmitters released by chromaffin cells : a biological application of microelectrodes." Paris 7, 2004. http://www.theses.fr/2004PA077207.

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40

Hockman, Dorit. "The development and evolution of vertebrate oxygen-sensing cells." Thesis, University of Cambridge, 2014. https://www.repository.cam.ac.uk/handle/1810/276679.

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Oxygen-sensing cells release neurotransmitters, including serotonin, in response to hypoxia in the blood or surrounding air/water. This stimulates the glossopharyngeal and/or vagal nerves, triggering increased ventilation via the respiratory reflex. In the adult, they are located in the carotid body (glomus cells) and lung epithelia (pulmonary neuroendocrine cells) of amniotes, and in the epithelia of the gills and orobranchial cavity (‘neuroepithelial cells’) of anamniotes. Despite their physiological importance, little is known about the molecular mechanisms of their development, while the e

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41

Lopez, Isabel. "Effects of antimitotic agents on cultured adrenal chromaffin cells : implications for microtubule involvement with adrenal nicotinic receptors /." The Ohio State University, 1992. http://rave.ohiolink.edu/etdc/view?acc_num=osu1487776801318513.

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42

Antypas, Elias Joseph. "The Characterization of Menkes Copper Transporter and Dopamine ß-monooxygenase Carboxy-Terminus in Neuroendocrine Cells." University of Toledo Health Science Campus / OhioLINK, 2008. http://rave.ohiolink.edu/etdc/view?acc_num=mco1213789670.

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43

Jarukanont, Daungruthai [Verfasser]. "Theory of Vesicle Motion and Interpretation of Electrophysiological Experiments in Chromaffin Cells, Hippocampal Neurons and Neuromuscular Junctions / Daungruthai Jarukanont." Kassel : Universitätsbibliothek Kassel, 2018. http://d-nb.info/1163735515/34.

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44

Mason, Helen Sarah. "The effect of fatty acids on calcium-activated and inwardly-rectifying potassium channel activity in bovine chromaffin and endothelial cells." Thesis, University of Aberdeen, 1998. http://ethos.bl.uk/OrderDetails.do?uin=uk.bl.ethos.245247.

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The effect of a variety of fatty acids was examined on two types of potassium channels in bovine chromaffin (BCC) and bovine aortic endothelial cells (BAEC). Large-conductance calcium-activated potassium channels were recorded from inside-out patches excised from cultured BCC. Fatty acids were applied to the intracellular surface of the patch at a concentration of 10μM. All the fatty acids examined (arachidonic acid (C20:4 <I>n-6</I>), eicosapentaenoic acid (C20:5 <I>n-3</I>), docosahexaenoic acid (C22:6 <I>n-3</I>) and myristic acid (C14:0)) increased the patch open probability at a potential

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Dutta, Soumyajit [Verfasser], and Dieter [Akademischer Betreuer] Bruns. "A pHluorin-based method to monitor exocytosis by release of peptidergic cargo molecules from chromaffin cells / Soumyajit Dutta. Betreuer: Dieter Bruns." Saarbrücken : Saarländische Universitäts- und Landesbibliothek, 2016. http://d-nb.info/1099282098/34.

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46

Chen, Xiaohui. "Optical stimulation of quantal exocytosis on transparent microchips." Diss., Columbia, Mo. : University of Missouri-Columbia, 2007. http://hdl.handle.net/10355/4890.

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Thesis (Ph. D.)--University of Missouri-Columbia, 2007.<br>The entire dissertation/thesis text is included in the research.pdf file; the official abstract appears in the short.pdf file (which also appears in the research.pdf); a non-technical general description, or public abstract, appears in the public.pdf file. Title from title screen of research.pdf file (viewed on January 30, 2008) Vita. Includes bibliographical references.

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47

Ramirez-Lavergne, Carmen. "The effect of the second messenger cyclic AMP on scinderin redistribution, F-actin disassembly and catecholamine secretion in bovine adrenal chromaffin cells." Thesis, University of Ottawa (Canada), 1994. http://hdl.handle.net/10393/10241.

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Work in our laboratory has demonstrated that stimulation of bovine chromaffin cells with nicotine elicits as a prelude to exocytosis, (a) redistribution of scinderin (Sc), a novel 80 kD Ca$\sp{2+}$-dependent actin filament severing protein and (b) cortical F-actin disassembly. The work presented in this thesis shows that the second messenger, cyclic AMP (cAMP) modulates Sc redistribution, F-actin disassembly and catecholamine (CA) secretion. Here we show that forskolin (F) produces a concentration-related (10 $\mu$M-50$\mu$M) inhibition of Sc redistribution, F-actin disassembly and CA release

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Maurer, Jennifer A. "Effects of protein kinase inhibitors on cultured bovine adrenal chromaffin cells: evidence for the role of protein phosphorylation reactions in calcium homeostasis /." The Ohio State University, 1994. http://rave.ohiolink.edu/etdc/view?acc_num=osu1487853913101481.

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49

Dembla, Ekta Mayur [Verfasser], and Ute [Akademischer Betreuer] Becherer. "Biogenesis of large dense core vesicles and molecular mechanisms of dead-end docking in mouse chromaffin cells / Ekta Mayur Dembla ; Betreuer: Ute Becherer." Saarbrücken : Saarländische Universitäts- und Landesbibliothek, 2017. http://d-nb.info/1182312969/34.

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Gu, Hyangja. "The effects of antimitotic drugs and mAb35 on ?-tubulin mRNA, microtubules and nicotinic receptor-mediated catecholamine secretion in adrenal chromaffin cells in culture /." The Ohio State University, 1992. http://rave.ohiolink.edu/etdc/view?acc_num=osu1487759914759158.

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