Thematische Bibliographien / Corn – Metabolsim

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Auswahl der wissenschaftlichen Literatur zum Thema „Corn – Metabolsim“

Autor: Grafiati

Veröffentlicht am 4. Juni 2021

Zuletzt aktualisiert am 14. Februar 2022

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Zeitschriftenartikel zum Thema "Corn – Metabolsim"

Farhoudi, R., und D. J. Lee. „Halopriming corn seeds improves seed emergence and carbohydrate metabolism under salinity stress“. Seed Science and Technology 42, Nr. 3 (01.12.2014): 461–65. http://dx.doi.org/10.15258/sst.2014.42.3.13.

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Man, Andy W. C., Yawen Zhou, Ning Xia und Huige Li. „Involvement of Gut Microbiota, Microbial Metabolites and Interaction with Polyphenol in Host Immunometabolism“. Nutrients 12, Nr. 10 (06.10.2020): 3054. http://dx.doi.org/10.3390/nu12103054.

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Immunological and metabolic processes are inextricably linked and important for maintaining tissue and organismal health. Manipulation of cellular metabolism could be beneficial to immunity and prevent metabolic and degenerative diseases including obesity, diabetes, and cancer. Maintenance of a normal metabolism depends on symbiotic consortium of gut microbes. Gut microbiota contributes to certain xenobiotic metabolisms and bioactive metabolites production. Gut microbiota-derived metabolites have been shown to be involved in inflammatory activation of macrophages and contribute to metabolic diseases. Recent studies have focused on how nutrients affect immunometabolism. Polyphenols, the secondary metabolites of plants, are presented in many foods and beverages. Several studies have demonstrated the antioxidant and anti-inflammatory properties of polyphenols. Many clinical trials and epidemiological studies have also shown that long-term consumption of polyphenol-rich diet protects against chronic metabolic diseases. It is known that polyphenols can modulate the composition of core gut microbiota and interact with the immunometabolism. In the present article, we review the mechanisms of gut microbiota and its metabolites on immunometabolism, summarize recent findings on how the interaction between microbiota and polyphenol modulates host immunometabolism, and discuss future research directions.

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Takahashi, Kei, Tetsuya Yamada, Sohei Tsukita, Keizo Kaneko, Yuta Shirai, Yuichiro Munakata, Yasushi Ishigaki et al. „Chronic mild stress alters circadian expressions of molecular clock genes in the liver“. American Journal of Physiology-Endocrinology and Metabolism 304, Nr. 3 (01.02.2013): E301—E309. http://dx.doi.org/10.1152/ajpendo.00388.2012.

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Chronic stress is well known to affect metabolic regulation. However, molecular mechanisms interconnecting stress response systems and metabolic regulations have yet to be elucidated. Various physiological processes, including glucose/lipid metabolism, are regulated by the circadian clock, and core clock gene dysregulation reportedly leads to metabolic disorders. Glucocorticoids, acting as end-effectors of the hypothalamus-pituitary-adrenal (HPA) axis, entrain the circadian rhythms of peripheral organs, including the liver, by phase-shifting core clock gene expressions. Therefore, we examined whether chronic stress affects circadian expressions of core clock genes and metabolism-related genes in the liver using the chronic mild stress (CMS) procedure. In BALB/c mice, CMS elevated and phase-shifted serum corticosterone levels, indicating overactivation of the HPA axis. The rhythmic expressions of core clock genes, e.g., Clock, Npas2, Bmal1, Per1, and Cry1, were altered in the liver while being completely preserved in the hypothalamic suprachiasmatic nuculeus (SCN), suggesting that the SCN is not involved in alterations in hepatic core clock gene expressions. In addition, circadian patterns of glucose and lipid metabolism-related genes, e.g., peroxisome proliferator activated receptor ( Ppar) α, Pparγ-1, Pparγ-coactivator-1α, and phosphoenolepyruvate carboxykinase, were also disturbed by CMS. In contrast, in C57BL/6 mice, the same CMS procedure altered neither serum corticosterone levels nor rhythmic expressions of hepatic core clock genes and metabolism-related genes. Thus, chronic stress can interfere with the circadian expressions of both core clock genes and metabolism-related genes in the liver possibly involving HPA axis overactivation. This mechanism might contribute to metabolic disorders in stressful modern societies.

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Clark, Teresa J., Longyun Guo, John Morgan und Jorg Schwender. „Modeling Plant Metabolism: From Network Reconstruction to Mechanistic Models“. Annual Review of Plant Biology 71, Nr. 1 (29.04.2020): 303–26. http://dx.doi.org/10.1146/annurev-arplant-050718-100221.

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Mathematical modeling of plant metabolism enables the plant science community to understand the organization of plant metabolism, obtain quantitative insights into metabolic functions, and derive engineering strategies for manipulation of metabolism. Among the various modeling approaches, metabolic pathway analysis can dissect the basic functional modes of subsections of core metabolism, such as photorespiration, and reveal how classical definitions of metabolic pathways have overlapping functionality. In the many studies using constraint-based modeling in plants, numerous computational tools are currently available to analyze large-scale and genome-scale metabolic networks. For 13C-metabolic flux analysis, principles of isotopic steady state have been used to study heterotrophic plant tissues, while nonstationary isotope labeling approaches are amenable to the study of photoautotrophic and secondary metabolism. Enzyme kinetic models explore pathways in mechanistic detail, and we discuss different approaches to determine or estimate kinetic parameters. In this review, we describe recent advances and challenges in modeling plant metabolism.

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Baumler, David J., Bing Ma, Jennifer L. Reed und Nicole T. Perna. „Inferring ancient metabolism using ancestral core metabolic models of enterobacteria“. BMC Systems Biology 7, Nr. 1 (2013): 46. http://dx.doi.org/10.1186/1752-0509-7-46.

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Kerou, Melina, Pierre Offre, Luis Valledor, Sophie S. Abby, Michael Melcher, Matthias Nagler, Wolfram Weckwerth und Christa Schleper. „Proteomics and comparative genomics of Nitrososphaera viennensis reveal the core genome and adaptations of archaeal ammonia oxidizers“. Proceedings of the National Academy of Sciences 113, Nr. 49 (18.11.2016): E7937—E7946. http://dx.doi.org/10.1073/pnas.1601212113.

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Ammonia-oxidizing archaea (AOA) are among the most abundant microorganisms and key players in the global nitrogen and carbon cycles. They share a common energy metabolism but represent a heterogeneous group with respect to their environmental distribution and adaptions, growth requirements, and genome contents. We report here the genome and proteome of Nitrososphaera viennensis EN76, the type species of the archaeal class Nitrososphaeria of the phylum Thaumarchaeota encompassing all known AOA. N. viennensis is a soil organism with a 2.52-Mb genome and 3,123 predicted protein-coding genes. Proteomic analysis revealed that nearly 50% of the predicted genes were translated under standard laboratory growth conditions. Comparison with genomes of closely related species of the predominantly terrestrial Nitrososphaerales as well as the more streamlined marine Nitrosopumilales [Candidatus (Ca.) order] and the acidophile “Ca. Nitrosotalea devanaterra” revealed a core genome of AOA comprising 860 genes, which allowed for the reconstruction of central metabolic pathways common to all known AOA and expressed in the N. viennensis and “Ca. Nitrosopelagicus brevis” proteomes. Concomitantly, we were able to identify candidate proteins for as yet unidentified crucial steps in central metabolisms. In addition to unraveling aspects of core AOA metabolism, we identified specific metabolic innovations associated with the Nitrososphaerales mediating growth and survival in the soil milieu, including the capacity for biofilm formation, cell surface modifications and cell adhesion, and carbohydrate conversions as well as detoxification of aromatic compounds and drugs.

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Cherifi, Myriam, Muriel Raveton, Antoine Picciocchi, Patrick Ravanel und Michel Tissut. „Atrazine metabolism in corn seedlings“. Plant Physiology and Biochemistry 39, Nr. 7-8 (Juli 2001): 665–72. http://dx.doi.org/10.1016/s0981-9428(01)01281-5.

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Lamoureux, Gerald L., Donald G. Rusness und Fred S. Tanaka. „Chlorimuron ethyl metabolism in corn“. Pesticide Biochemistry and Physiology 41, Nr. 1 (September 1991): 66–81. http://dx.doi.org/10.1016/0048-3575(91)90061-p.

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Skelton, Joshua J., David M. Simpson, Mark A. Peterson und Dean E. Riechers. „Comparative Analysis of 2,4-D Uptake, Translocation, and Metabolism in Non–AAD-1 Transformed and 2,4-D–Resistant Corn“. Weed Science 65, Nr. 5 (05.07.2017): 567–78. http://dx.doi.org/10.1017/wsc.2017.22.

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The Enlist™ traits provide 2,4-D resistance in several crops. Though corn is naturally tolerant to 2,4-D, the engineered trait conferred by the aryloxyalkanoate dioxygenase-1 (AAD-1) enzyme provides enhanced 2,4-D tolerance and confers resistance to the graminicide herbicide family, the aryloxyphenoxypropionates. The objectives of this research were 2-fold: (1) measure and compare uptake, translocation, and metabolism of 2,4-D in Enlist™ (E, +AAD1) and non–AAD-1 transformed (NT, −AAD1) isogenic corn hybrids; and (2) and investigate the effect of glyphosate and/or the Enlist™ adjuvant system (ADJ) on these factors and corn injury. Uptake of radiolabeled 2,4-D acid applied alone in corn was not altered by the addition of ADJ when tank mixed at 24 h after application (HAA). By contrast, uptake of radiolabeled 2,4-D was significantly lower (69%) compared with 2,4-D plus ADJ (89%) at 24 HAA with a premixed formulation of 2,4-D choline plus glyphosate-dimethylamine (Enlist Duo™ herbicide [EDH]). Translocation of 2,4-D between the two corn hybrids was not different. E corn metabolized more 2,4-D (100% of absorbed) than NT corn (84%), and glyphosate did not alter 2,4-D metabolism. Furthermore, the metabolism of 2,4-D to nonphytotoxic dichlorophenol (DCP) and subsequent DCP-derived metabolites formed in E corn was examined. Injury to E corn is not typically observed in the field; however, injury symptoms were clearly evident in E corn (within 24 HAA) when formulated acetochlor was tank mixed with EDH, which correlated with an increase in 2,4-D uptake during this time period. In summary, the lack of injury in E corn following EDH applied alone may be attributed to a relatively low amount of 2,4-D uptake and the combination of natural and engineered 2,4-D metabolic pathways.

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Tani, Satoshi, Shokei Yamada und Robert S. Knighton. „Extensibility of the lumbar and sacral cord“. Journal of Neurosurgery 66, Nr. 1 (Januar 1987): 116–23. http://dx.doi.org/10.3171/jns.1987.66.1.0116.

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✓ Tethered spinal cord, or tethered cord syndrome, describes a disorder manifested by progressive motor and sensory deficit in the legs and by incontinence. Tethered cord syndrome occurs when the elongated spinal cord is anchored by a thick filum terminale or other pathological structures. The underlying mechanism is impairment of oxidative metabolism in the lumbosacral cord. The authors studied the extensibility of various parts of lumbar, sacral, and coccygeal segments in experimental animals and correlated this with the oxidative metabolism in these segments. The filum terminale possesses far greater extensibility than any spinal cord segments and functions as a buffer in preventing the cord from overstretching. The lumbar, sacral, and coccygeal segments elongate under traction only below the attachment of the lowest pair of dentate ligaments. The lower the cord segment, the greater the percentage of elongation in spite of limited elasticity of the cord tissue; this greater percentage of elongation of the spinal cord correlates with increasing impairment of the oxidative metabolism and more severe neurological deficit. These findings explain such symptoms and signs as motor and sensory deficits in the legs associated with the human tethered cord syndrome, and correspond with the high clinical incidence of incontinence. The lower spinal cord segments elongated promptly within 3 seconds after the start of traction. This implies that repeated acute hyperextension and hyperflexion, as occurs in humans, may accentuate oxidative metabolic changes that have already been caused by chronic cord tethering. The authors conclude that the elongation of the spinal cord under traction parallels the degree of metabolic dysfunction.

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Dissertationen zum Thema "Corn – Metabolsim"

Nogemane, Noluyolo. „Symplasmic pathway in phloem loading and unloading in source and sink leaves of Zea mays L. as evidenced under normal and elevated CO₂ conditions“. Thesis, Rhodes University, 2003. http://hdl.handle.net/10962/d1007813.

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Zea mays plants kept at ambient (ca 375ppm) and elevated CO₂ (ca 650 to 700ppm) were used to examine the possibility of a symplasmic loading, unloading and transport pathway in dark-adapted and illuminated (200μmolm⁻²sec⁻¹ ) sink and source leaves. 5,6-carboxyfluorescein diacetate was introduced into the mesophyll cells and symplasmic transfer observed 3h after application. In sink and source leaves exposed to ambient CO₂ and illuminated at 200 molm-2sec-1, the fluorescence front was observed approximately 3cm from the point of application, while in dark-adapted plants, the fluorescence front was observed approximately 1cm from the point of application. Under elevated CO₂ conditions the fluorescence front in illuminated plants appeared to transport faster moving approximately 5cm from the point of application, and in dark-adapted plants, only 3cm from the point of application. Based on the increase in 5,6-CF accumulation under elevated CO₂ conditions, the present study suggests that there was an increase in capacity for assimilate loading and transport under elevated CO₂ conditions. In source leaves, 5,6-CFDA was taken up into the mesophyll cells, loaded symplasmically and transported basipetally. In sink leaves 5,6- CFDA was taken up from basal mesophyll and after symplasmic loading, was transported acropetally where it was offloaded into the younger immature sink region. Transport in the sieve tubes was confirmed by using aniline blue, which was applied 3h after 5,6-CF transport. Aniline blue coupled with 5,6-CF transport studies showed that the sieve tubes of both cross and longitudinal veins are involved in symplasmic unloading, loading and transport processes in sink and source leaves. Apoplasmic uptake of 5,6-CFDA by cut leaves showed that after apoplasmic transport via the transpiration stream, 5,6-CFDA was offioaded to the xylem parenchyma where it was metabolically cleaved , releasing fluorescent 5,6-CF into the xylem parenchyma. Transverse sections cut after 3h of uptake were observed after 120 and 180 min suggesting that a retrieval of solutes occurs from the xylem to the xylem parenchyma, bundle sheath, phloem parenchyma and to the th in-walled sieve tubes. It was not possible to determine if the thick-walled sieve tubes were involved or if they took up 5,6-CF. Given the available data on loading and offioading of assimilates in sink and source leaves respectively, this study demonstrated that a slow symplasmic pathway exists from the mesophyll to the phloem, and that offloading from the phloem in sink leaves can occur via a symplasmic route.

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Álvarez, Guardia David. „Estudi dels mecanismes moleculars implicats en l’associació entre inflamació i alteracions metabòliques en cèl∙lules cardíaques“. Doctoral thesis, Universitat de Barcelona, 2011. http://hdl.handle.net/10803/31985.

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El canvi en l’estil de vida que s’ha produït en les societats desenvolupades els darrers anys ha tingut com a contrapartida l’aparició de conductes sedentàries i modificacions en la dieta. Com a conseqüència d’aquests factors s’han produït diverses alteracions metabòliques que han causat un augment de la prevalença de l’obesitat. Aquesta obesitat té una sèrie d’efectes adversos sobre la fisiologia cardiovascular i és considerada un important factor de risc pel desenvolupament de la insuficiència cardíaca. De fet, el consum de dietes amb un elevat contingut en greixos (HFD) s’ha relacionat amb una sèrie d’alteracions cardíaques directes com són la inflamació, la hipertròfia i la disfunció contràctil. Durant el procés inflamatori que es produeix en les esmentades malalties cardiovasculars, les cèl•lules cardíaques humanes secreten citocines i quimiocines proinflamatòries com el TNF-α, MCP-1, i IL-6, molècules que es troben sota el control transcripcional del factor de transcripció ubic i induïble anomenat NF-κB. Aquestes citocines exerceixen diversos efectes pleiotròpics autocrins en les cèl•lules cardíaques, tot produint un efecte de retroalimentació positiva del procés inflamatori que contribueix al desenvolupament d’aquestes malalties. En condicions normals, el cor de mamífers adults obté energia en forma d’ATP principalment a partir de la β-oxidació d’àcids grassos en el mitocondri, encara que aquest orgànul és capaç de catabolitzar altres substrats com la glucosa o el lactat per tal d’assegurar una font constant d’energia. Ara bé, en determinades circumstàncies, com és el cas de la hipertròfia i la insuficiència cardíaques, aquesta flexibilitat de substrat es veu compromesa i la β-oxidació d’àcids grassos es redueix degut a que la font principal d’energia passa a ser la glucosa. Aquests canvis metabòlics comporten una desregulació del control transcripcional de gens relacionats amb el transport, captació i catabolisme dels àcids grassos i la glucosa. En el miocardi, els factors de transcripció implicats en el control d’aquests gens inclouen ERRα i PPARβ/δ. Ambdós factors de transcripció, participen en l’activació de la PDK4, enzim clau en la modulació homeostàtica de la glucosa. Aquesta cinasa regula l’activitat de la PDC, enzim que catalitza la reacció de descarboxilació del piruvat a acetil-CoA, tot limitant l’ús de carbohidrats com a font d’energia en mitocondris i afavorint així la β-oxidació d’àcids grassos. En l’activació de la transcripció de PDK4 també hi participa PGC-1α, que interacciona amb ERRα i PPARβ/δ, tot incrementant-ne la seva activitat transcripcional. Estudis recents però, semblen indicar que no només aquests dos factors de transcripció participen en la regulació de PDK4. És el cas d’E2F1, un factor de transcripció clau en la regulació de la transició de la fase G1 a la fase S del cicle cel•lular i del qual la regió promotora del gen que codifica per PDK4 en presenta dos llocs d’unió. Estudis recents suggereixen que PPARβ/δ, que és la forma predominant en les cèl•lules cardíaques, pot atenuar les vies de senyalització inflamatòries i, per tant, interferir en la remodelació cardíaca. Aquesta funció és en gran mesura deguda a la capacitat dels PPAR, un cop han estat activats per agonistes, de formar complexos amb altres factors de transcripció activats, com ara NF-κB i STAT resultant així en la inhibició de l’activitat transcripcional d’aquests últims. Així l’ús d’agonistes PPARβ/δ podria ser un camí força interessant de cara a trobar potencials fàrmacs per tal de pal•liar les afeccions cardíaques derivades d’alteracions metabòliques i amb un rerefons inflamatori. En conjunt en aquest treball es presenten una sèrie de resultats destinats a conèixer de forma més detallada els mecanismes moleculars que relacionen les alteracions metabòliques i els processos inflamatoris en cor, per tal de poder buscar potencials dianes farmacològiques amb l’objectiu de prevenir i tractar aquests estats patològics.
The change in lifestyle that has occurred in developed societies in recent years has been accompanied by the rise of sedentary behavior and changes in diet that have caused an increasing obesity prevalence. Obesity has a huge number of adverse effects on cardiovascular physiology and is considered an important risk factor for heart failure developement. In fact, high fat diets have been linked with direct cardiac abnormalities such as inflammation, hypertrophy and contractile dysfunction. During the inflammatory process that occurs in these diseases, human cardiac cells secrete proinflammatory cytokines and chemokines such as TNF-α, MCP-1, and IL-6, molecules that are under the control of the ubiquitous and inducible transcription factor NF-κB. In certain circumstances, such in hypertrophy and heart failure, the substrate flexibility in heart is compromised and the fatty acids β-oxidation is reduced because the main source of energy becomes the glucose. These metabolic changes lead to a deregulation on the transcriptional control of genes associated with transport, uptake and catabolism of fatty acids and glucose. In the myocardium, among the transcription factors involved in the control of these genes we found ERRα and PPARβ/δ. Both transcription factors, are involved in PDK4 activation, an important enzyme in the homeostatic modulation of glucose. This kinase regulates PDC activity, an enzyme that catalyzes the decarboxylation from pyruvate to acetyl-CoA, limiting the use of carbohydrates as energy source in mitochondria and thus favoring the fatty acid β-oxidation. In the PDK4 transcription activation also participates PGC-1α, which interacts with ERRα and PPARβ/δ, increasing its transcriptional activity. However recent studies, suggest that not only these two transcription factors are involved in PDK4 regulation. Other transcription factors as E2F1, which is crucial for cell cycle control, may regulate PDK4 expression. Overall, the results shown in this work are aimed to learn in more detail the molecular mechanisms linking the metabolic disorders and inflammatory processes in heart, in order to find potential drug targets to prevent and treat these pathological states.

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Martins, Carolina Guerra. „Níveis de inclusão de bagaço de cana-de-açúcar in natura associados a milho grão inteiro ou floculado em dietas de terminação de bovinos Nelore“. Universidade de São Paulo, 2013. http://www.teses.usp.br/teses/disponiveis/11/11139/tde-13112013-145253/.

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O objetivo deste trabalho foi a avaliação metabólica de dietas contendo níveis de inclusão de bagaço de cana-de-açúcar in natura (0, 3, 6, 9% da MS da dieta) associados a milho grão inteiro ou floculado (360g/L), perfazendo um total de cinco tratamentos. Foram utilizados cinco bovinos machos castrados da raça Nelore, com peso médio inicial igual a 479,2kg; contendo cânulas no rúmen, distribuídos em um delineamento Quadrado Latino 5x5. Tratamentos aplicados: MI-0: milho inteiro sem inclusão de BIN (bagaço de cana-de-açúcar in natura); MI-3: milho inteiro com 3% de BIN; MI-6: milho inteiro com 6% de BIN; MI-9: milho inteiro com 9% de BIN; MF-6: milho floculado com 6% de BIN. O método de processamento não alterou o consumo de MS e nutrientes (p>0,05) e não houve efeito de nível de inclusão de BIN (p>0,05), exceto para o consumo de FDN que aumentou linearmente (p=0,0145) com a inclusão de BIN. Os coeficientes de digestibilidade não diferiram entre os métodos de processamento e nem entre os níveis de BIN (p>0,05), apesar do efeito positivo da inclusão de BIN no pH ruminal (p=0,0001). O milho duro utilizado no presente estudo quando fornecido inteiro para bovinos Nelore em dietas com altos teores de concentrado apresentou ao redor de 96% da concentração de ELm e de ELg (2,12 e 1,44 Mcal/Kg de MS) em relação ao valor tabular do NRC (1996) para milho dentado (2,18 e 1,50 Mcal/Kg de MS). Os maiores teores de BIN na dieta ocasionaram maiores tempos gastos na atividade de ingestão (p<0,05) e mastigação (p<0,05) e, também, houve uma tendência (p=0,0702) para maior tempo de alimentação à medida que o teor de BIN foi aumentando. O método de floculação não alterou os VfCF e VfCNF, no entanto, diminuiu a KdCF e aumentou a KdCNF e o lag time.
The objective of this study was to evaluate diets containing levels of fresh sugar cane bagasse (0, 3, 6, 9% DM) associated with whole or steam flaked corn grain (360g/L) in metabolic parameters of cattle. Five rumen cannulated Nellore steers, weighing 479.2 kg at the beginning of the experiment, were distributed in five treatments in a 5x5 Latin Square design. Experimental treatments were the following finishing diets: WC-0: whole corn with no addition of sugar cane bagasse (SCB); WC-3: whole corn with 3% SCB; WC-6: whole corn with 6% SCB, WC-9: whole corn with 9% SCB; SFC-6: steam flaked corn with 6% SCB. Despite the increased in vitro Kd of non-fibrous carbohidrates (NFC), steam flaked corn did not improve the nutritional value of corn grain in comparison with whole grain based on in vivo metabolic assay. The inclusion of dietary SCB did not improve DM intake and digestibility of nutrients, but resulted in positive effects on ruminal pH. The addition of whole flint corn used in the present study in the diet of Nellore steers fed high concentrate finishing diets, had approximately 96% of NEm and NEg concentrations (2.11 and 1.44 Mcal / kg DM) in comparison to tabular values from (1996) for dent corn (2.18 and 1.50 Mcal / kg DM), for DM intake restricted to 85% of potential intake.

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Lahiguera, Belenguer Álvaro. „Defectos en la reparación homóloga producen un aumento del metabolismo oxidativo en cáncer de ovario: relevancia para los tratamientos con inhibidores de PARP“. Doctoral thesis, Universitat de Barcelona, 2020. http://hdl.handle.net/10803/671620.

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Hipótesis: ¿El daño crónico en el ADN o bloqueo de la HR crónico produce un shift o una selección de células que han cambiado su metabolismo para poder sobrevivir a la situación? Objetivos: El principal objetivo de esta tesis doctoral ha sido estudiar la correlación entre los defectos de reparación en la recombinación homóloga o daño crónico en el ADN y la adaptación metabólica que se lleva a cabo para poder sobrevivir en esa situación en el cáncer de ovario seroso de alto grado. Como objetivos concretos de la tesis tenemos: A) Generación y caracterización de modelos que padezcan daño crónico en el ADN o sufran defectos en la reparación por recombinación homóloga. B) Estudiar los mecanismos de adaptación metabólica de estos modelos celulares al daño crónico y los fallos en la recombinación homologa C) Ver el papel que pueden jugar inhibidores de la cadena respiratoria como posibles tratamientos a tumores de estas características y las interacciones con otros tratamientos que se utilizan actualmente.

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Casas, Maria I. „BIOCHEMICAL AND GENETIC CHARACTERIZATION OF SPECIALIZED FLAVONOID METABOLISM IN MAIZE“. The Ohio State University, 2015. http://rave.ohiolink.edu/etdc/view?acc_num=osu1431071650.

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Paranhos, Vivian Maria dos Santos 1984. „Taxa metabólica basal em atletas usuários de cadeira de rodas : comparação entre dois métodos de avaliação“. [s.n.], 2014. http://repositorio.unicamp.br/jspui/handle/REPOSIP/275064.

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Orientador: Edison Duarte
Dissertação (mestrado) - Universidade Estadual de Campinas, Faculdade de Educação Física
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Resumo: Considerada atualmente um problema de saúde pública, as lesões na medula espinhal na população aumentam a cada ano e com ela muitos problemas que irão acompanhar o indivíduo ao longo de toda a vida. Algumas dúvidas surgem ao trabalhar com essas pessoas pelos profissionais nutricionistas, visto as alterações fisiológicas decorrentes da lesão. Como calcular o gasto energético total diário e especificar, com maior precisão, as necessidades de calorias totais e de macronutrientes (proteínas, lipídeos, carboidratos e todas as vitaminas e minerais)? Como calcular esse gasto em atletas com lesão medular usuários de cadeira de rodas em suas práticas esportivas? O método comumente empregado para cálculo de gasto energético como a fórmula de Harris-Benedict, por exemplo, elaborada para indivíduos hígidos. Outro método utilizado para esse tipo de avaliação é o de calorimetria indireta, bastante preciso, porém inviável muitas vezes, quer pelo custo, quer pela dificuldade de aplicação. Esse estudo teve como o objetivo analisar o gasto energético basal através de um método de calorimetria indireta em atletas usuários de cadeira de rodas e comparar os resultados com a fórmula comumente utilizada (Harris-Benedict) buscando comparar os valores apresentados entre esses dois métodos. O estudo foi realizado com 14 indivíduos praticantes da modalidade de rugby em cadeira de rodas, com lesão da medula espinhal cervical (C5-C7), do gênero masculino, com idade entre 25 e 36 anos. Observou-se uma correlação significativa de 0,58 entre a comparação dos dois métodos e concordância significativa com intervalo de confiança entre eles de +154,3 kcal/dia até -115,5 kcal/dia. No entanto a fórmula de Harris & Benedict não prevê e não calcula o gasto relacionado à espasmos musculares, comum nessa população, dentre outras especificidades. Concluiu-se que a calorimetria indireta é um método mais preciso, validado e mais indicado para essa população quando comparado à fórmula de predição de Harris & Benedict, devido as especificidades da lesão da medula espinhal
Abstract: Currently considered a public health problem , injuries to the spinal cord in the population increase every year and with it many problems that will accompany the individual throughout life . Some questions arise when working with these people by professional nutritionists , since the physiological changes resulting from injury. How to calculate the total daily energy expenditure and to specify more precisely the needs of total calories and macronutrients ( proteins , lipids , carbohydrates and all the vitamins and minerals) ? How to calculate this expense in athletes with spinal cord injury wheelchair users in their sports practices ? The common method for calculating energy expenditure as the Harris-Benedict formula , for example , designed to healthy individuals . Another method used for this type of assessment is to indirect calorimetry quite accurate , but often impractical , either by cost or by the difficulty of implementation . This study aimed to analyze the basal energy expenditure through a method of indirect calorimetry in athletes of wheelchair users and compare the results with the commonly used formula ( Harris -Benedict ) trying to compare the values ??presented between these two methods . The study was conducted with 14 subjects practicing the sport of rugby in wheelchairs with cervical spinal cord ( C5 - C7 ) , male , aged between 25 and 36 years. There was a significant correlation of 0.58 between the two methods and a significant agreement with a confidence interval between them +154.3 kcal / day to -115.5 kcal / day. However the formula of Harris & Benedict does not provide and does not calculate the expense related to muscle , spasms common in this population , among other specifics . It was concluded that indirect calorimetry is a more accurate , validated and most appropriate method for this population when compared to the prediction formula of Harris & Benedict , because the specifics of spinal cord injury
Mestrado
Atividade Fisica Adaptada
Mestra em Educação Física

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Costa, Amina Chain. „Influência do exercício físico sobre a massa e o metabolismo ósseo de indivíduos com lesão medular cervical“. Universidade do Estado do Rio de Janeiro, 2011. http://www.bdtd.uerj.br/tde_busca/arquivo.php?codArquivo=3702.

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Indivíduos que permanecem longo tempo em cadeira de rodas apresentam importante perda de massa óssea, principalmente nos membros inferiores, possivelmente agravada pela baixa ingestão de cálcio dietético e pelo inadequado estado nutricional de vitamina D. O exercício físico pode contribuir para a manutenção ou aumento da massa óssea em diferentes populações e nos indivíduos com lesão medular pode contribuir para atenuar a perda de massa óssea. O objetivo do presente estudo foi avaliar a influência da prática regular de exercício físico sobre a adequação da massa óssea, indicadores bioquímicos do metabolismo ósseo e estado nutricional de vitamina D em indivíduos com lesão medular cervical há pelo menos um ano. Em vinte e cinco homens de 19 a 56 anos sendo 15 fisicamente ativos e 10 sedentários, foi realizada análise sérica de cálcio, PTH, 25(OH)D, IGF-1, osteocalcina e NTx. As medidas do conteúdo mineral ósseo, densidade mineral óssea (DMO), massa magra e massa gorda foram realizadas por DXA. A pigmentação da pele (constitutiva e por bronzeamento) foi determinada por colorimetria com o objetivo de investigar sua influência sobre o estado de vitamina D. A ingestão habitual de cálcio foi registrada em um questionário de frequência alimentar direcionado para alimentos fonte. As comparações entre os dois grupos foram realizadas pela aplicação do Teste t de Student exceto para as variáveis ósseas que foram realizadas após ajustes pela massa corporal total, tempo de lesão e ingestão de cálcio utilizando-se análise de co-variância. Associações entre as variáveis estudadas foram avaliadas através de análise de correlação de Pearson. Valores de p<0.05 foram considerados significativos. Não foram observadas diferenças estatisticamente significativas entre os grupos para nenhuma variável óssea com exceção do z-score da DMO da coluna lombar, que foi significativamente maior no grupo de indivíduos sedentários (0,9 1,7 vs -0,7 0,8; p<0,05). No entanto, entre os indivíduos ativos, aqueles que iniciaram a prática de exercício físico com menos tempo decorrido após a lesão apresentaram maior DMO do fêmur (r=-0,60; p<0,05). Nos indivíduos ativos, a freqüência do exercício apresentou associação negativa com a concentração sérica de i-PTH (r = -0,50; p =0,05) e positiva com a concentração de 25(OH)D (r= 0,58; p <0,05). Após ajustes pela massa corporal total e tempo de lesão foram observadas associações positivas entre a ingestão diária de cálcio e z-score da DMO da coluna lombar (r = 0,73 e p <0,01) e DMO do rádio (r = 0,56 e p <0,05). Os resultados do presente estudo apontam para um efeito benéfico do exercício físico sobre a massa óssea e o perfil hormonal relacionado ao metabolismo ósseo. O início da prática regular de exercício físico o quanto antes após a lesão parece contribuir para atenuar a perda de massa óssea nos membros inferiores. Além disso, os resultados deste estudo sugerem uma possível potencialização do efeito osteogênico do exercício físico quando combinado a uma adequada ingestão de cálcio.
Individuals who stay long time in a wheelchair have significant bone loss, especially in lower limbs that may be aggravated by low calcium intake and inadequate vitamin D status. Physical exercise contributes to maintain or increase bone mass in different populations and may be useful in reducing bone loss in spinal cord injured individuals. The aim of this study was to evaluate the influence of regular physical exercise on bone mass adequacy, biochemical markers of bone metabolism and vitamin D status in individuals with cervical spinal cord injury. Twenty five male adults (19-56 years) with cervical spinal cord injury for at least one year, were recruited and divided into physically active (n=15) and sedentary (n=10) groups. Fasting blood samples were collected and serum samples were stored at -20oC until analysis of calcium, PTH, 25(OH)D, IGF-1, osteocalcin and NTx. Bone mineral content and density (BMD), lean mass and fat mass were evaluated by DXA. Skin pigmentation (constitutive and facultative) was evaluated by reflectance colorimetry in order to investigate its influence on vitamin D status. Habitual calcium intake was recorded using a food frequency questionnaire directed to calcium food sources. Comparisons between groups were performed using Students t test except for bone variables that were performed after adjustments for total body mass, duration of injury and calcium intake by analysis of covariance. Associations between variables were evaluated using Pearson's correlation analysis. P values <0.05 were considered significant. There were no significant differences between groups for bone measurements except for lumbar spine Z-score, that was significantly higher in the sedentary group (0.9 1.7 vs -0.7 0.8; p< 0.05). However, in the active group, it was observed that subjects who initiated the practice of physical exercise with less time after injury had higher BMD at the femur (r=-0.60; p<0.05). In active subjects, exercise frequency was negatively associated with serum i-PTH (r = -0.50, p = 0.05) and positively associated with serum 25(OH)D (r= 0.58; p <0.05). After adjustments for total body mass and duration of injury daily calcium intake was positively associated with lumbar spine Z-score (r = 0.73 and p < 0.01) and with radius BMD (r = 0.56 and p <0.05). The results of this study suggest a beneficial effect of regular exercise practice on bone mass and bone-related hormonal profile. The earlier initiation of regular physical exercise after the injury appears to contribute to attenuate the loss of bone mass in lower limbs. Moreover, our results suggest that the osteogenic effect of exercise may be potentiated when combined with an adequate calcium intake.

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Quiles, Caroline Luísa. „Iluminação artificial : efeito do fotoperíodo e do espectro de cor sobre os ritmos biológicos e metabolismo“. reponame:Biblioteca Digital de Teses e Dissertações da UFRGS, 2017. http://hdl.handle.net/10183/163577.

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Objetivo: Avaliar a influencia da iluminação artificial nos ritmos biológicos e metabolismo por meio de dois experimentos em ratos Wistar. O primeiro avalia mudanças de fotoperíodo que mimetizem a sazonalidade; o segundo, a qualidade da iluminação artificial (espectro de cor) no ciclo claro/escuro (LD). Métodos: Experimento1 - Três grupos de animais: Controle (CL; n=6, ciclo LD de 12/12); grupo que inicia com fotoperíodo longo (LP/SP; n=7; LD 16.5:7.5); grupo que inicia com fotoperíodo curto (SP/LP; n=7; LD 7.5:16.5). Os grupos experimentais passaram por 18 dias no fotoperíodo inicial, 17 dias de redução ou aumento gradual do fotoperíodo, 18 dias no fotoperíodo inverso ao que iniciou. Experimento 2 - 36 animais foram mantidos 108 dias em ciclo LD 16:8h, divididos em 2 grupos: Standard Light (SL; n=18), mantidos sob iluminação com espectro de cor padrão (LED, 4000K); e Circadian Light (CL; n=18) com alterações de espectro de cor ao longo do dia (LED, 2700-6500K). Em ambos os estudos, níveis de atividade e temperatura, além de melatonina e corticosterona sérica, foram mensurados. No Experimento 2, além das pesagens semanais, após eutanásia a gordura visceral foi medida. Os parâmetros circadianos foram obtidos por meio da análise de séries temporais. Na análise estatística foram usados os testes paramétricos ou não paramétricos, de acordo com a normalidade dos dados. Resultados: A quantidade de atividade noturna, além dos níveis de corticosterona foram menores no grupo SP/LP (p<0.05). Portanto, os animais demonstraram pior adaptação dos ritmos à transição do fotoperíodo de dia curto para longo (SP/LP). A qualidade de iluminação também influenciou o comportamento animal. O grupo CL apresentou melhores parâmetros rítmicos que o grupo SL, por exemplo, menor intracyclevariability, maior amplitude e quantidade de atividade (p<0.05). Apesar de o peso corporal ter sido similar, o grupo SL apresentou maior quantidade de gordura visceral (p<0.05). Parâmetros rítmicos de atividade correlacionaram com a concentração de melatonina somente no grupo CL, enquanto que parâmetros rítmicos correlacionaram com a concentração de corticosterona principalmente no grupo SL. Conclusões: Nosso estudo reforça a relevância da iluminação como um fator importante na regulação do comportamento e metabolismo, sugerindo que o a iluminação artificial comumente utilizada, sem variação de espectro de cor, é um forte fator facilitador do processo de cronodisrupção e aumento de gordura visceral. Ainda, o sistema de iluminação utilizado frequentemente nos alojamentos experimentais podem ser subótimas para simular o ambiente natural. Apoio: FIPE/HCPA, CNPq, CAPES e Luxion Iluminação.
Objective:To evaluate the influence of artificial illumination on biological rhythms and metabolism by two experiments whit Wistar rats. The first one evaluated changes in photoperiod that mimetics seasonality; the second one, the quality of artificial light (color spectrum) on light/dark cycle (LD). Methods: Experiment 1 – Three animal groups: Control (CL; n=6, LD cycle 12/12); group that started with long photoperiod (LP/SP; n = 7; LD 16.5:7.5); group that started with short photoperiod (SP/LP; n=7; LD 7.5:16.5). Experimental groups passed for 18 day in a start photoperiod, 17 days of gradual increase or decrease of photoperiod, 18 days on inverse photoperiod to what start.Experiment 2 – 36 animals were kept for 108 days in a LD cycle of 16:8h, divided in 2 groups: Standard Light (SL; n=18), kept under illumination with standard color spectrum (LED, 4000K); and Circadian Light (CL; n=18) with changes of color spectrum during the day (LED, 2700-6500K). In both studies, activity and temperature levels, as well as serum melatonin and corticosterone, were measured. On Experiment 2, in addition to weekly weighing, after euthanasia the visceral fat was measures. The circadian parameters were obtained by temporal series analyses. In statistical analyses were used parametric or non-parametric tests, according the normality of data. Results:Amount of activity on dark, besides corticosterone levels were lower on SL/LP group (p<0.05). So, animals showed low rhythms adaptation to photoperiod transitions from short to long light (SL/LP). The quality of illumination also influenced in animal behavior. The CL group presented better rhythmic parameters than SL group, for example, low intracycle variability, high amplitude and quantity of activity (p<0.05). Although body weight was similar, SL group presented higher amount of visceral fat (p<0.05). Rhythmic parameters of activity correlated with the melatonin concentration just in CL group, whereas rhythmic parameters correlated whit corticosterone concentration principally in SL group. Conclusions: Our study reinforces the relevance of illumination as an important factor on metabolic and behavioral regulation, suggesting that artificial illumination commonly used, without color spectrum variation, is a strong facilitating factor on the process of chronodisruption and increase of visceral fat. Thus, the illumination system frequently used in experimental accommodation could be suboptimal for to simulate the natural environment. Support:FIPE/HCPA, CNPq, CAPES andLuxionIluminação.

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Saunders, Royal Duane. „Arachidonic acid and lipid metabolism following spinal cord injury /“. The Ohio State University, 1985. http://rave.ohiolink.edu/etdc/view?acc_num=osu1487260859496213.

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Chiriboga, Varea María de los Ángeles. „Aspectos bioquímicos del bloqueo de la maduración de peras ‘Conference’ tratadas con 1-metilciclopropeno (1-MCP) y aplicación de sistemas de control“. Doctoral thesis, Universitat de Lleida, 2012. http://hdl.handle.net/10803/108177.

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El objetivo de esta tesis fue investigar cómo el 1-metilciclopropeno (1-MCP) afecta a peras “Conference”. El tratamiento con 1-MCP es muy eficaz para retardar la maduración y senescencia pero su aplicación puede resultar en una inhibición permanente de la maduración y se asocia con una alta concentración de 1-MCP (600 ppb), mientras que una concentración más baja (300 ppb) da resultados muy variables. Este comportamiento se ve influenciado principalmente por la madurez a la cosecha y especialmente, por la capacidad de la fruta para producir etileno en el momento del tratamiento. La capacidad de la fruta para madurar en el periodo de después del tratamiento con 1-MCP ve determinat por parámetros fisiológicos relacionados principalmente con el metabolismo del etileno, como el grado de inhibición de la enzima ACC sintasa durante la conservación frío y la reactivación de la misma durante la vida útil. El tratamiento también permite un mejor mantenimiento de los sistemas antioxidantes de la fruta. Finalmente, esta tesis propone un sistema para prevenir el bloqueo de la maduración debido al tratamiento con 1-MCP. La combinación de una alta dosis de 1-MCP y etileno exógeno aplicado al mismo tiempo inmediatamente después de la cosecha permite un considerable retraso del proceso de maduración sin bloquearlo por completo y dando lugar a una homogeneización de la maduración entre los frutos de diferentes fechas de cosecha.
L'objectiu d'aquesta Tesi ha estat investigar com l’ 1-metilciclopropè (1-MCP) afecta a les peres ‘Conference’. El tractament amb 1-MCP es molt eficaç per retrasar la maduració i la senescència, però pot arribar a provocar una inhibició permanent de la maduració i va associat amb una alta concentració d’1-MCP (600 nL L-1), mentre que una concentració més baixa (300 nL L-1) pot donar resultats molt variables. L'efecte d'1 MCP estan influenciats principalment per la maduresa de collita i especialment per la capacitat de la fruita per produir etilè en el moment del tractament. El comportament de la maduració desprès del tractament amb 1-MCP ve determinada per par‡metres fisiològics relacionats principalment amb el metabolisme de l'etilË i ve determinada pel grau d'inhibició d'ACS durant l'emmagatzematge en fred i la reactivació d'aquest enzim durant la vida útil. El tractament amb 1 MCP pot també tenir un efecte positiu sobre el metabolisme antioxidant. Finalment, aquesta tesi proposa un sistema que evita aquest bloqueig de la maduració. La combinació d'una alta dosi d'1-MCP i etilè aplicats simultàniament e immediatament després de la collita, permet aconseguir un considerable retard de la maduració, sense bloquejar-la completament, a la vegada que dona lloc a una homogeneïtzació de la maduració entre els fruits de diverses dates de collita.
The aim of this thesis was to investigate how 1-methylcyclopropene (1-MCP) affects ëConferenceí pears. The treatment with 1-MCP was very effective to reduce ripening and senescence but can result in a permanent inhibition of ripening. This ripening blockage was associated with a high concentration of 1-MCP (600 ppb) whereas a lower concentration (300 ppb) gave very variable results. This behaviour was mainly influenced by the harvest maturity and especially by the ability of the fruit to produce ethylene at the moment of treatment. The softening behaviour and the effectiveness of 1-MCP are determined by physiological parameters related principally with the ethylene metabolism and is determined by the extent of the inhibition of ACC synthase during cold storage and the reactivation of the enzyme during shelf life. 1-MCP treatment also allows a better maintenance of the fruitís antioxidant system. Finally, this thesis proposes a system that permits the prevention of the evergreen behaviour. The combination of a high dose of 1-MCP and ethylene applied simultaneously immediately after harvest permitted a considerable delay of the ripening process without completely blocking it and resulted in a homogenization of ripening between fruit from different harvest dates.

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Bücher zum Thema "Corn – Metabolsim"

C, Shannon Jack, Knievel Daniel P. 1943-, Boyer Charles D und Pennsylvania State University. Intercollege Graduate Program in Plant Physiology., Hrsg. Regulation of carbon and nitrogen reduction and utilization in maize: Proceedings of the first annual Penn State Symposium in Plant Physiology (May 22-23, 1986), The Pennsylvania State University. Rockville, Md: American Society of Plant Physiologists, 1986.

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Nadjmi, Maschallah, Hrsg. Imaging of Brain Metabolism Spine and Cord Interventional Neuroradiology Free Communications. Berlin, Heidelberg: Springer Berlin Heidelberg, 1989. http://dx.doi.org/10.1007/978-3-642-74337-5.

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Genevieve, Morgan, Hrsg. The core balance diet: 4 weeks to boost your metabolism and lose weight for good. Carlsbad, Calif: Hay House, 2009.

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Genevieve, Morgan, Hrsg. The core balance diet: 28 days to boost your metabolism and lose weight for good. 5. Aufl. Carlsbad, CA: Hay House, 2013.

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Congress, European Society of Neuroradiology. Imaging of brain metabolism, spine and cord interventional neuroradiology free communications: XVth Congress of the European Society of Neuroradiology, Würzburg, September 13th-17th, 1988. Berlin: Springer-Verlag, 1989.

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Marzok, Mohamed Ahmed Tony Ahmed. Detection of genetically modified soybeans and maize in Egypt as well as comparative nutritional safety investigations of isogenic and transgenic (Bt) maize in broiler nutrition: Broiler performance, degradation and metabolic fate of maize DNA in some tissues and organs. Berlin: Mensch & Buch Verlag, 2004.

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Mohamed Ahmed Tony Ahmed Marzok. Detection of genetically modified soybeans and maize in Egypt as well as comparative nutritional safety investigations of isogenic and transgenic (Bt) maize in broiler nutrition: Broiler performance, degradation and metabolic fate of maize DNA in some tissues and organs. Berlin: Mensch & Buch Verlag, 2004.

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V, Khotyleva L., Instytut henetyki i tsytalohii (Akadėmii͡a︡ navuk Belarusi) und Belorusskoe obshchestvo genetikov i selekt͡s︡ionerov., Hrsg. Bioėnergeticheskie prot͡s︡essy pri geterozise. Minsk: "Navuka i tėkhnika", 1991.

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Miller, Keith Douglas. The effects of the herbicide safener benoxacor on glutathione s-transferase activity and the metabolism of benoxacor in suspension cultures of Zea mays. 1994.

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Kjelgaard, James F. Comparison of models for estimating evapotranspiration from maize. 1993.

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Buchteile zum Thema "Corn – Metabolsim"

Macdonald, Ian A., und Michael J. Gibney. „Core Concepts of Nutrition“. In Nutrition and Metabolism, 1–6. Oxford, UK: Wiley-Blackwell, 2011. http://dx.doi.org/10.1002/9781444327779.ch1.

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Gafarova, Tatiana E., und Dmitry M. Tyshkivsky. „Formation of Ketols of 9-Hydroxylinolenic Acid in Corn Seeds“. In Plant Lipid Metabolism, 283–85. Dordrecht: Springer Netherlands, 1995. http://dx.doi.org/10.1007/978-94-015-8394-7_78.

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Bauman, William A., und Christopher P. Cardozo. „Spinal Cord Injury: Skeletal Pathophysiology and Clinical Issues“. In Primer on the Metabolic Bone Diseases and Disorders of Mineral Metabolism, 1018–27. Ames, USA: John Wiley & Sons, Inc., 2013. http://dx.doi.org/10.1002/9781118453926.ch122.

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Tappy, Luc, Leonie Egli und Christel Tran. „Metabolism of Nutritive Sweeteners in Humans“. In Fructose, High Fructose Corn Syrup, Sucrose and Health, 35–50. New York, NY: Springer New York, 2014. http://dx.doi.org/10.1007/978-1-4899-8077-9_3.

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Marsala, M. „Spinal cord blood flow and metabolism in transient spinal ischemia“. In Spinal Cord Monitoring, 3–25. Vienna: Springer Vienna, 1998. http://dx.doi.org/10.1007/978-3-7091-6464-8_1.

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Träger, Karl, Peter Radermacher und Xavier Leverve. „The adrenergic coin: perfusion and metabolism“. In Applied Physiology in Intensive Care Medicine 2, 339–42. Berlin, Heidelberg: Springer Berlin Heidelberg, 2012. http://dx.doi.org/10.1007/978-3-642-28233-1_36.

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Ko, Hyun-Yoon. „Electrolyte and Metabolic Disorder“. In Management and Rehabilitation of Spinal Cord Injuries, 275–96. Singapore: Springer Singapore, 2019. http://dx.doi.org/10.1007/978-981-10-7033-4_21.

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Gennari, F. John. „Metabolic Alkalosis“. In Core Concepts in the Disorders of Fluid, Electrolytes and Acid-Base Balance, 275–96. Boston, MA: Springer US, 2012. http://dx.doi.org/10.1007/978-1-4614-3770-3_9.

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Thurman, Joshua M., und Tomas Berl. „Disorders of Water Metabolism“. In Core Concepts in the Disorders of Fluid, Electrolytes and Acid-Base Balance, 29–48. Boston, MA: Springer US, 2012. http://dx.doi.org/10.1007/978-1-4614-3770-3_2.

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Teoh, H., M. Malcangio und N. G. Bowery. „GABAB Receptor Control of Transmitter Release in the Spinal Cord“. In GABA: Receptors, Transporters and Metabolism, 95–102. Basel: Birkhäuser Basel, 1996. http://dx.doi.org/10.1007/978-3-0348-8990-2_11.

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Konferenzberichte zum Thema "Corn – Metabolsim"

Winter, Jordan M., Richard A. Burkhart, Danielle M. Pineda, Saswati Chand, Joseph Cozzitorto, Charles J. Yeo und Jonathan R. Brody. „Abstract 5394: HuR affects pancreatic cancer metabolism through post-transcriptional control of core metabolic enzymes.“ In Proceedings: AACR 104th Annual Meeting 2013; Apr 6-10, 2013; Washington, DC. American Association for Cancer Research, 2013. http://dx.doi.org/10.1158/1538-7445.am2013-5394.

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Lapierre, H., H. F. Tyrrell, G. Pelletier und D. R. Ouellet. „Variations of 13C enrichment of milk and milk components from cows fed barley- or corn-based diets“. In 6th EAAP International Symposium on Energy and Protein Metabolism and Nutrition. The Netherlands: Wageningen Academic Publishers, 2019. http://dx.doi.org/10.3920/978-90-8686-891-9_135.

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López Zazueta, Claudia, Olivier Bernard und Jean-Luc Gouze. „Reduction of Metabolic Networks Keeping Core Dynamics“. In 9th Vienna Conference on Mathematical Modelling. ARGESIM Publisher Vienna, 2018. http://dx.doi.org/10.11128/arep.55.a55218.

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Ding De-wu und He Xiao-qing. „Analyzing the core of E. coli metabolism with flux balance“. In 2010 Second International Conference on Computational Intelligence and Natural Computing (CINC). IEEE, 2010. http://dx.doi.org/10.1109/cinc.2010.5643859.

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Hui, Du. „Analysis of Human Metabolic Core Using Hub-Based Centrality“. In 2009 ETP International Conference on Future Computer and Communication (FCC). IEEE, 2009. http://dx.doi.org/10.1109/fcc.2009.18.

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Hu, Yi. „Nitrogen metabolism in an ancient nutritional symbiosis betweenCephalotesants and core gut bacteria“. In 2016 International Congress of Entomology. Entomological Society of America, 2016. http://dx.doi.org/10.1603/ice.2016.113575.

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Mneimneh, Farah, Nesreen Ghaddar, Kamel Ghali, Charbel Moussalem und Ibrahim Omeis. „Experiment Study for Evaluation of Phase Change Material Cooling Vest’s Effectiveness at Two Melting Points Used by People With Paraplegia During Exercise“. In ASME 2020 International Mechanical Engineering Congress and Exposition. American Society of Mechanical Engineers, 2020. http://dx.doi.org/10.1115/imece2020-23083.

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Abstract Phase change material (PCM) cooling vests were tested on people with thoracic (T1-T12) spinal cord injury (SCI), also called people with paraplegia (PA), during exercise in heat. The purpose was to reduce heat stress, limit the increase in core temperature, and improve thermal comfort for PA under high metabolic rates and hot ambient conditions. This health risk was a result of thoracic SCI and disruption of thermoregulatory responses in PA. The current study aims to evaluate the efficacy of cooling vest on PA during arm-crank exercise at two melting points, 20°C (V20) and 14°C (V14) compared to no vest test (NV). Eleven participants with high- (T1-T3) and mid-thoracic SCI (T4-T8) were selected to participate in three tests. Core and skin temperatures and heart rate values were measured during 15-min precondition, 30-min exercise and 15-min recovery. Subjective voting of thermal comfort, sensation, skin wettedness and perceived exertion were recorded during exercise only. The main findings revealed significant reduction in change in core temperature (0.42±0.3°C;0.38±0.2°C) in V20 and V14 compared to NV tests for mid-thoracic group. For high-thoracic group, V20 and V14 were least effective in reducing core temperature (p > 0.05). Improvements in thermal comfort was established when using V14 and V20 compared to NV by 85% and 30% for high-thoracic group and by 72% and 53% for mid-thoracic group.

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Massone, Anna Maria, Cristina Campi, Mauro C. Beltrametti und Cecilia Marini. „FDG-PET and the assessment of spinal cord metabolism in amyotrophic lateral sclerosis (ALS)“. In 2016 IEEE Nuclear Science Symposium, Medical Imaging Conference and Room-Temperature Semiconductor Detector Workshop (NSS/MIC/RTSD). IEEE, 2016. http://dx.doi.org/10.1109/nssmic.2016.8069573.

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Samatova, N. F., A. Geist, G. Ostrouchov und A. Melechko. „Parallel out-of-core algorithm for genome-scale enumeration of metabolic systemic pathways“. In Proceedings 16th International Parallel and Distributed Processing Symposium. IPDPS 2002. IEEE, 2002. http://dx.doi.org/10.1109/ipdps.2002.1016588.

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Zeilig, G., H. Weingarden, A. Obuchov, A. Bloch, M. Gaides, R. Reuveny und I. Ben-Dov. „Lokomat walking results in increased metabolic markers in individuals with high spinal cord injury“. In 2015 International Conference on Virtual Rehabilitation (ICVR). IEEE, 2015. http://dx.doi.org/10.1109/icvr.2015.7358604.

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Berichte der Organisationen zum Thema "Corn – Metabolsim"

Durán, Raúl V. Metabolismo del cáncer: comiendo con tu enemigo. Sociedad Española de Bioquímica y Biología Molecular, November 2019. http://dx.doi.org/10.18567/sebbmdiv_rpc.2019.11.1.

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