Thematische Bibliographien / Deoxynivalenol

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Auswahl der wissenschaftlichen Literatur zum Thema „Deoxynivalenol“

Autor: Grafiati

Veröffentlicht am 4. Juni 2021

Zuletzt aktualisiert am 11. März 2023

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Zeitschriftenartikel zum Thema "Deoxynivalenol"

Xu, Hua, Bidong Wu, Lei Guo, Jia Chen, Nini Lin, Lingling Qin und Jianwei Xie. „Preparation of deoxynivalenol and mask deoxynivalenol“. Toxicon 158 (Februar 2019): S65—S66. http://dx.doi.org/10.1016/j.toxicon.2018.10.224.

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Vidal, Arnau, Nabila Bouzaghnane, Sarah De Saeger und Marthe De Boevre. „Human Mycotoxin Biomonitoring: Conclusive Remarks on Direct or Indirect Assessment of Urinary Deoxynivalenol“. Toxins 12, Nr. 2 (24.02.2020): 139. http://dx.doi.org/10.3390/toxins12020139.

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Deoxynivalenol is one of the most ubiquitous mycotoxins in the Western diet through its presence in cereals and cereal products. A vast amount of studies indicate the worrying level of exposure to this toxin, while even high percentages of the population exceed the tolerable daily intake. To evaluate and assess dietary exposure, analysis of urinary levels of deoxynivalenol and its glucuronides has been proposed as a reliable methodology. An indirect preliminary method was used based on the cleavage of deoxynivalenol glucuronides through the use of enzymes (β-glucuronidase) and subsequent determination of "total deoxynivalenol" (sum of free and released mycotoxins by hydrolysis). Next, a direct procedure for quantification of deoxynivalenol-3-glucuronide and deoxynivalenol-15-glucuronide was developed. As deoxynivalenol glucuronides reference standards are not commercially available, the indirect method is widely applied. However, to not underestimate the total deoxynivalenol exposure in urine, the direct and indirect methodologies need to be compared. Urinary samples (n = 96) with a confirmed presence of deoxynivalenol and/or deoxynivalenol glucuronides were analysed using both approaches. The indirect method clarified that not all deoxynivalenol glucuronides were transformed to free deoxynivalenol during enzymatic treatment, causing an underestimation of total deoxynivalenol. This short communication concludes on the application of direct or indirect assessment of urinary deoxynivalenol.

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Curtui, V., C. Seidler, E. Schneider und E. Usleber. „Bestimmung von Deoxynivalenol und Deepoxy-Deoxynivalenol in Milch“. Mycotoxin Research 21, Nr. 1 (März 2005): 40–42. http://dx.doi.org/10.1007/bf02954814.

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HUFF, WILLIAM E., und WINSTON M. HAGLER. „Density Segregation of Corn and Wheat Naturally Contaminated with Aflatoxin, Deoxynivalenol and Zearalenone“. Journal of Food Protection 48, Nr. 5 (01.05.1985): 416–20. http://dx.doi.org/10.4315/0362-028x-48.5.416.

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Density segregation was used to reduce mycotoxin levels of corn samples naturally contaminated with aflatoxin or deoxynivalenol, and wheat samples naturally contaminated with deoxynivalenol or zearalenone. Corn kernels which were buoyant in saturated sodium chloride represented 3% of the total sample, yet contained 74% of the aflatoxin. Corn buoyant in water contained 51 and 14% of the total deoxynivalenol present in two naturally contaminated corn samples. Subsequent segregation of corn non-buoyant in water with 30% sucrose removed additional deoxynivalenol-contaminated kernels, resulting in the combined removal of 59 and 79% of the deoxynivalenol. Removal of deoxynivalenol-contaminated corn kernels with both water and 30% sucrose reduced the concentration of deoxynivalenol by 53 and 77%. Removing wheat buoyant in water and 30% sucrose decreased the deoxynivalenol present by 96 and 68%, and reduced the deoxynivalenol concentration by 96 and 67%. Removing wheat naturally contaminated with zearalenone buoyant in water and 30% sucrose combined resulted in no detectable zearalenone remaining in the non-buoyant fraction of the samples.

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Simsek, Senay, Kimberly Burgess, Kristin L. Whitney, Yan Gu und Steven Y. Qian. „Analysis of Deoxynivalenol and Deoxynivalenol-3-glucoside in wheat“. Food Control 26, Nr. 2 (August 2012): 287–92. http://dx.doi.org/10.1016/j.foodcont.2012.01.056.

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Faixová, Z., Š. Faix, R. Bořutová und Ľ. Leng. „Efficacy of Dietary Selenium to Counteract Toxicity of Deoxynivalenol in Growing Broiler Chickens“. Acta Veterinaria Brno 76, Nr. 3 (2007): 349–56. http://dx.doi.org/10.2754/avb200776030349.

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The aim of this study was to evaluate the effect of deoxynivalenol on plasma indicators and efficacy of dietary selenium to counteract toxicity of deoxynivalenol in growing broiler chicks. Three groups of broilers were formed with 14 birds in each group. Three diets included control (0.2 ppm deoxynivalenol, 0.4 mg selenium/kg diet), deoxynivalenol-contaminated (3 ppm deoxynivalenol, 0.4 mg selenium/kg diet) and deoxynivalenol-contaminated (3 ppm deoxynivalenol) plus selenium-enriched yeast (1.4 mg selenium/kg diet). After 6 weeks of feeding all birds were sacrifi ced and blood samples for chemical analyses were collected. Plasma calcium, chloride and alanine aminotransferase activity were signifi cantly elevated and magnesium, total proteins, triglycerides and free glycerol were decreased in chicks fed deoxynivalenol-contaminated diet compared with those fed the control diet. Supplementation of selenium-enriched yeast to the diet reversed plasma levels of calcium, magnesium and alanine aminotransferase activity in chicks induced by dietary deoxynivalenol. Phosphorus, albumin and cholesterol levels and alkaline phosphatase, aspartate aminotransferase and lactate dehydrogenase activities were not affected by diets. The inclusion of selenium to DON-contaminated diet, however, did not completely alleviate toxic effect on protein and lipid metabolism by the liver. Supplementation of selenium-enriched yeast product counteracted most of the plasma indicator alterations caused by deoxynivalenol-contaminated diet in chicks.

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Dall'Asta, C., A. Dall'Erta, P. Mantovani, A. Massi und G. Galaverna. „Occurrence of deoxynivalenol and deoxynivalenol-3-glucoside in durum wheat“. World Mycotoxin Journal 6, Nr. 1 (01.02.2013): 83–91. http://dx.doi.org/10.3920/wmj2012.1463.

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The occurrence of deoxynivalenol and deoxynivalenol-3-glucoside in durum wheat samples (n=150; 25 lines × 2 reps × 3 environments) collected in 2010 from 3 areas located in north-central Italy was evaluated. In addition, the co-occurrence of other trichothecenes was considered. An optimised extraction method based on the use of salts followed by ultra-high performance liquid chromatography-mass spectrometry analysis was used for the quantification of the mycotoxins. All samples were found positive for deoxynivalenol at concentrations ranging between 47 and 3,715 μg/kg. A ubiquitous occurrence of deoxynivalenol-3-glucoside was found; 85% of the analysed samples contained this masked mycotoxin at concentrations varying between 46 and 842 μg/kg. In addition to glycosylated deoxynivalenol, acetylated forms of deoxynivalenol (3- and 15-acetyldeoxynivalenol) were also found in most of the durum wheat samples. The deoxynivalenol-3-glucoside/deoxynivalenol ratio, reaching up to 30% in many samples, was similar to that already found in other cereals such as soft wheat and barley. These data open the way for further investigations on the role of glycosylating activity as a possible Fusarium head blight-resistance mechanism in durum wheat, as already proved in the case of soft wheat.

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Scarpino, Valentina, und Massimo Blandino. „Effects of Durum Wheat Cultivars with Different Degrees of FHB Susceptibility Grown under Different Meteorological Conditions on the Contamination of Regulated, Modified and Emerging Mycotoxins“. Microorganisms 9, Nr. 2 (16.02.2021): 408. http://dx.doi.org/10.3390/microorganisms9020408.

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The enhancement of Fusarium head blight (FHB) resistance is one of the best options to reduce mycotoxin contamination in wheat. This study has aimed to verify that the genotypes with high tolerance to deoxynivalenol could guarantee an overall minimization of the sanitary risk, by evaluating the contamination of regulated, modified and emerging mycotoxins on durum wheat cvs with different degrees of FHB susceptibility, grown under different meteorological conditions, in 8 growing seasons in North-West Italy. The years which were characterized by frequent and heavy rainfall in spring were also those with the highest contamination of deoxynivalenol, zearalenone, moniliformin, and enniatins. The most FHB resistant genotypes resulted in the lowest contamination of all the mycotoxins but showed the highest deoxynivalenol-3-glucoside/deoxynivalenol ratio and moniliformin/deoxynivalenol ratio. An inverse relationship between the amount of deoxynivalenol and the deoxynivalenol-3-glucoside/deoxynivalenol ratio was recorded for all the cvs and all the years. Conversely, the enniatins/deoxynivalenol ratio had a less intense relationship with cv tolerance to FHB. In conclusion, even though the more tolerant cvs, showed higher relative relationships between modified/emerging mycotoxins and native/target mycotoxins than the susceptible ones, they showed lower absolute levels of contamination of both emerging and modified mycotoxins.

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Curtui, V., A. Brockmeyer, R. Dietrich, O. Kappenstein, H. Klaffke, J. Lepschy, E. Märtlbauer et al. „Deoxynivalenol in Lebensmitteln“. Mycotoxin Research 21, Nr. 2 (Juni 2005): 83–88. http://dx.doi.org/10.1007/bf02954424.

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Gagiu, Valeria, Elena Mateescu, Alina Alexandra Dobre, Irina Smeu, Mirela Elena Cucu, Oana Alexandra Oprea, Daniel Alexandru, Enuța Iorga und Nastasia Belc. „Deoxynivalenol Occurrence in Triticale Crops in Romania during the 2012–2014 Period with Extreme Weather Events“. Toxins 13, Nr. 7 (29.06.2021): 456. http://dx.doi.org/10.3390/toxins13070456.

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This article aims to evaluate deoxynivalenol occurrence in triticale crops in Romania in years with extreme weather events (2012: Siberian anticyclone with cold waves and heavy snowfall; 2013 and 2014: “Vb” cyclones with heavy precipitation and floods in spring). The deoxynivalenol level in triticale samples (N = 236) was quantified by ELISA. In Romania, the extreme weather events favoured deoxynivalenol occurrence in triticale in Transylvania and the southern hilly area (44–47° N, 22–25° E) with a humid/balanced-humid temperate continental climate, luvisols and high/very high risk of floods. Maximum deoxynivalenol contamination was lower in the other regions, although heavy precipitation in May–July 2014 was higher, with chernozems having higher aridity. Multivariate analysis of the factors influencing deoxynivalenol occurrence in triticale showed at least a significant correlation for all components of variation source (agricultural year, agricultural region, average of deoxynivalenol, average air temperature, cumulative precipitation, soil moisture reserve, aridity indices) (p-value < 0.05). The spatial and geographic distribution of deoxynivalenol in cereals in the countries affected by the 2012–2014 extreme weather events revealed a higher contamination in Central Europe compared to southeastern and eastern Europe. Deoxynivalenol occurrence in cereals was favoured by local and regional agroclimatic factors and was amplified by extreme weather events.

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Dissertationen zum Thema "Deoxynivalenol"

Pierron, Alix. „Toxicity of three biological derivatives of deoxynivalenol : deepoxy-deoxynivalenol, 3-epi-deoxynivalenol and deoxynivalenol-3-glucoside on pigs“. Thesis, Toulouse 3, 2016. http://www.theses.fr/2016TOU30096/document.

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Les mycotoxines sont des métabolites secondaires de moisissures contaminant de façon naturelle de nombreuses denrées alimentaires, notamment les céréales. Le déoxynivalénol (DON), produit par Fusarium sp., est la mycotoxine la plus répandue dans le monde. Du fait de sa grande stabilité chimique, le DON est difficile à éliminer, et se retrouve dans les céréales et les produits finis ou il induit des effets toxiques pour l'homme et l'animal. De nouvelles stratégies de lutte sont mises en places, telle la transformation biologique utilisant des bactéries ou des plantes. En effet certaines bactéries possèdent des enzymes capables de transformer le DON en de nouveaux composés, le déepoxy-déoxynivalénol (DOM-1) et le 3-épi-déoxynivalénol (3-epi-DON). De plus, certaines plantes sont naturellement capables de transformer le DON dans le but de l'éliminer et de le détoxifier, formant ainsi le deoxynivalénol-3-ß-D-glucoside (D3G). L'objectif de cette thèse était d'évaluer la toxicité de ces dérivés du DON au niveau de l'intestin et du système immunitaire par le biais d'analyses in silico, in vitro, ex vivo et in vivo. Les tests de toxicité in vitro sur la lignée humaine intestinale cellulaire Caco-2 montrent que le DOM-1, le 3-epi-DON et le D3G n'étaient pas cytotoxiques, ils ne modifiaient ni la viabilité, ni la fonction de barrière des cellules, mesurée par la résistance électrique transépithéliale. Les tests de toxicité ex vivo sur des explants jéjunum porcin ont montré que le DOM-1, le 3-epi-DON ou le D3G n'induisaient pas de modifications histomorphologiques. En revanche, les explants exposés au DON montraient des lésions morphologiques et une régulation positive de l'expression des cytokines pro-inflammatoires. L'impact de ces trois dérivés a été également analysé sur l'expression de l'ensemble des gènes du tissu, avec une analyse microarray. Ceci a montré que ces dérivés du DON n'induisaient aucun changement dans l'expression des gènes par rapport au groupe contrôle. Le DON quand a lui exprimait différentiellement 747 sondes, correspondantes à 333 gènes impliqués dans l'immunité, la réponse inflammatoire, le stress oxydatif, la mort cellulaire, le transport moléculaire et la fonction mitochondriale. L'analyse in silico a montré que le D3G, contrairement au DON était incapable de se lier au site-A du ribosome, principale cible de la toxicité pour le DON. Les deux dérivés microbiens eux, étaient capables de se fixer au site-A au sein du ribosome, mais contrairement au DON ils ne formaient que deux liaisons hydrogènes au lieu de trois. De plus, ces trois dérivés n'induisaient pas de stress ribotoxique, d'activation des MAPKs (mitogen-activated protein kinases), et de réponse pro-inflammatoire. Une étude complémentaire a été menée in vivo pour évaluer la toxicité du DOM-1 chez le porc (gavage pendant 21 jours avec .0.14mg / kg de poids vif). Les résultats ont montré que le DOM-1, contrairement au DON n'induisait pas les effets toxiques du DON au niveau des paramètres zootechniques (pas de vomissements, aucune diminution de la consommation alimentaire ou de perte de poids), sur l'intestin et le foie (pas de dommages tissulaires), ou sur la réponse immunitaire (pas de réponse inflammatoire induite). En conclusion, nos résultats montrent l'efficacité de ces transformations enzymatiques. La déepoxydation et l'épimérisation bactérienne, ainsi que la glycosylation par les plantes permettent de sensiblement diminuer la toxicité du DON, passant par une absence de toxicité sur le ribosome avec une absence d'activation des MAPKs et de réponses inflammatoires. Dans ce contexte de contamination par les mycotoxines, ces méthodes de luttes alternatives semblent être des approches prometteuses
The Fusarium sp. mycotoxin deoxynivalenol (DON) is one of the most frequently widespread mycotoxin worldwide. Due to its high structural stability, the elimination of DON, once present in cereals or feed materials, becomes difficult. Thereby, it is present in many cereals and final feed products, inducing several toxic effects on human and animals, and causing big economic losses. New strategies of to fight against mycotoxins were developed, as biological transformation, either by the use of bacteria or plants. Indeed, some microorganisms are able to transform DON in new products, by enzymatic reaction, forming the deepoxy-deoxynivalenol (DOM-1) and the 3-epi-deoxynivalenol (3-epi-DON). Moreover, some plants naturally own the capacity to glycosylate DON in the aim to detoxify it, forming the deoxynivalenol-3-ß-D-glucoside (D3G). The aim of this thesis was to assess the toxicity of these DON derivatives, on the intestine and immune response, using several approaches such as in silico, in vitro, ex vivo and in vivo models. On the human intestinal Caco-2 cell line, DOM-1, 3-epi-DON and D3G were not cytotoxic; they did not alter its viability and barrier function, as measured by the trans epithelial electrical resistance. The expression profile of DOM-1, 3-epi-DON and D3G-treated jejunal explants was similar to that of controls and these explants did not show any histomorphology alteration. On the other hand, the treatment of intestinal explants with DON, induced morphological lesions and upregulated the expression of proinflammatory cytokines. The impact of these three derivatives was also studied on intestinal explants with a pan-genomic transcriptomic analysis. Results show that the derivatives of DON did not induce any change on the gene expression in comparison to the control-treated explants. In contrary, DON-treated explants differentially expressed 747 probes, representing 323 genes involved in immune and inflammatory responses, oxidative stress, cell death, molecular transport and mitochondrial function. In silico analysis revealed that D3G, opposing to DON, was unable to bind to the A site of the ribosome, which is the main target for DON toxicity. Both DOM-1 and 3-epi-DON were able to fit into the pockets of the A site of the ribosome but only by forming two hydrogen bonds, while in this position, DON forms three hydrogen bonds. Moreover, the three derivatives do not elicit a ribotoxic stress, MAPKinase activation, and inflammatory response. Then, an in vivo study was carried out to assess the toxicity of DOM-1 on pig (feed forced during 21 days at 0.14 mg/Kg BW). The results showed that DOM-1 does not have as much toxic effects as DON on zootechnical parameters (no emesis induced, no decrease of food consumption or weight loss observed), on intestine and liver (no tissues damages), or on the immune response (no inflammatory response induced). Our data demonstrate that bacterial de-epoxidation or epimerization of deepoxy-DON modified its interaction with the ribosome, leading to an absence of MAPKinase activation and toxicity; and that the glycosylation of DON suppresses its ability to bind to the ribosome and decreases its intestinal toxicity. The mycotoxin deoxynivalenol (DON) remains an important challenge in many regions in the world. Thus, these biological detoxifications of DON seem to represent a new promising approach helping manage the problem of its contamination

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Burgess, Kimberly. „Analysis of Deoxynivalenol and Deoxynivalenol-3-glucoside in Wheat“. Thesis, North Dakota State University, 2012. https://hdl.handle.net/10365/26454.

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Deoxynivalenol (DON), a mycotoxin produced in cereal grains infected by Fusarium Head Blight produced by Fusarium graminearium and Deoxynivalenol-3-?-D-glucopyranoside (DON-3G), were studied during processing using LC-MS-MS and GC. DON reduced significantly (P<0.05) 61.8% during milling into flour. Therefore, DON was concentrated mostly in the bran and germ. DON increased 40.8% during the fermentation stage of baking. DON increased in dough more than flour and mixed dough. Milling reduced by 23.7% but fermentation did not. But bread was significantly lower in DON-3G at 0.15 ppm than flour and dough at 0.31 ppm. The baking increased DON and decreased DON-3G showing a difference in stability of the mycotoxins during processing. Enzyme hydrolysis on DON using ?-amylase, cellulase, protease, and xylanase, showed a significant increase with cellulase (20.8%), protease (11.4%), and xylanase (35.6%) compared to wheat composite. DON may be bound to the cell wall or protein component of the kernel.

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Jiang, Wei. „Fate of Deoxynivalenol and Deoxynivalenol-3-Glucoside during the Malting Process“. Thesis, North Dakota State University, 2015. https://hdl.handle.net/10365/27868.

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Deoxynivalenol (DON) is commonly found on small grains and causes food safety issues. Deoxynivalenol-3-Glucoside (DON-3-G) is a conjugate, formed as a defense response by the host plant. Past studies have shown both to be present in Fusarium infected small grains, and processed products like beer, but there is limited information on DON-3-G in malt. Objectives were to determine the levels of DON-3-G in barley and wheat, and to study its fate during malting of inoculated and commercial samples. Commercial barley and wheat samples were used to determine levels in naturally infected grain. During malting, barley DON declined 48% on average, but DON-3-G increased by 115%. Both compounds increased in malted wheat. The genotype x crop year interactions were significant for both toxins, indicating that the genotypes did not respond similarly in the two years. The potential for large amounts of DON-3-G to be formed during malting has not been reported.

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Nogueira, da Costa Andre. „Mechanism-based biomarkers for deoxynivalenol“. Thesis, University of Leeds, 2010. http://ethos.bl.uk/OrderDetails.do?uin=uk.bl.ethos.531519.

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Magallanes, Lopez Ana Maria. „Fate of Deoxynivalenol during Wet Milling“. Thesis, North Dakota State University, 2018. https://hdl.handle.net/10365/29010.

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The fungal disease Fusarium head blight affects cereal grains and can produce mycotoxins, like the water-soluble deoxynivalenol (DON). Wheat wet milling process begins with ground endosperm obtained by dry milling and ends with the separation of starch from gluten. Research was conducted on hard red spring wheat and durum wheat samples naturally contaminated with DON. The fate of DON in wheat dry milled fractionations (farina/semolina, shorts, and bran) during wet milling was investigated. Three wet milling processes were evaluated. DON levels were assessed by GC-ECD. Results showed that DON was present in all dry milled fractions. DON concentration in farina and semolina exceeded the safety threshold for human consumption. After wet milling farina and semolina, nearly all the DON was found in the water-soluble fraction, regardless the wet milling process. A negligible level of DON was found in the gluten extracted from HRSW with Martin wet milling process.

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Seidler, Caroline. „Nachweis der Fusarientoxine Deoxynivalenol und Zearalenon in Lebensmitteln“. Giessen : VVB Laufersweiler, 2007. http://geb.uni-giessen.de/geb/volltexte/2007/4728/index.html.

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Wippermann, Wolf. „Diaplazentare Deoxynivalenolintoxikation bei Schweinefeten. Lassen sich am 70. Trächtigkeitstag histomorphologisch und immunhistologisch diagnostisch verwertbare Befunde erheben?“ Doctoral thesis, Universitätsbibliothek Leipzig, 2011. http://nbn-resolving.de/urn:nbn:de:bsz:15-qucosa-67849.

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Dawlatana, Mamtaz. „Control of mycotoxins in major food commodities in Bangladesh“. Thesis, University of Portsmouth, 1997. http://ethos.bl.uk/OrderDetails.do?uin=uk.bl.ethos.338351.

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Spindelböck, Bernd Ulrich. „Untersuchung zum Vorkommen und zur Häufigkeit von Deoxynivalenol in Lebensmitteln“. Diss., lmu, 2004. http://nbn-resolving.de/urn:nbn:de:bvb:19-27258.

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Nielsen, Carina. „Untersuchungen zur Toxizität und zu den molekularen Wirkungsmechanismen von Deoxynivalenol“. Diss., lmu, 2009. http://nbn-resolving.de/urn:nbn:de:bvb:19-107564.

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Bücher zum Thema "Deoxynivalenol"

Chŏn, Hyang-suk. Sikpʻum chung teoksiniballenol e taehan anjŏnsŏng pʻyŏngka =: Safety evaluation for deoxynivalenol in foods. [Seoul]: Sikpʻum Ŭiyakpʻum Anjŏnchʻŏng, 2007.

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Ernährung und Landwirtschaft Germany. Bundesministerium für Verbraucherschutz. Analytik und Vorkommen wichtiger Fusarientoxine (Deoxynivalenol, Zeralenon) sowie Aufnahme dieser Toxine durch den deutschen Verbraucher: Verbundforschungsprojekt 00HS 055 : Abschlussbericht : Projektzeitraum: 1. August 2001 - 31 Dezember 2004. Münster: Landwirtschaftsverlag, 2006.

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Recent Advances and Perspectives in Deoxynivalenol Research. MDPI, 2017. http://dx.doi.org/10.3390/books978-3-03842-471-0.

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M. Ebrahem, Susanne Kersten, G. Breves, A. Beineke, Kathrin Hermeyer und S. Dänicke. Effect of increasing concentrations of deoxynivalenol (DON) in diet on health and performance of laying hens of different genetic background. Verlag Eugen Ulmer, 2013. http://dx.doi.org/10.1399/eps.2013.4.

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Buchteile zum Thema "Deoxynivalenol"

Whitaker, Thomas B., Winston M. Hagler, Francis G. Giesbrecht und Anders S. Johansson. „Sampling Wheat for Deoxynivalenol“. In Advances in Experimental Medicine and Biology, 73–83. Boston, MA: Springer US, 2002. http://dx.doi.org/10.1007/978-1-4615-0629-4_8.

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Trigo-Stockli, Dionisia M. „Effect of Processing on Deoxynivalenol and other Trichothecenes“. In Advances in Experimental Medicine and Biology, 181–88. Boston, MA: Springer US, 2002. http://dx.doi.org/10.1007/978-1-4615-0629-4_18.

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Schmitt, K., Erwin Märtlbauer, Ewald Usleber, R. Gessler, J. Lepschy und David Abramson. „Detection of Acetylated Deoxynivalenol by Enzyme-Linked Immunosorbent Assay“. In Immunoassays for Residue Analysis, 314–21. Washington, DC: American Chemical Society, 1996. http://dx.doi.org/10.1021/bk-1996-0621.ch023.

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Pestka, James J., M. A. Moorman, R. L. Warner, M. F. Witt, J. H. Forsell und J.-H. Tai. „Immunoglobulin a Nephropathy as a Manifestation of Vomitoxin (Deoxynivalenol) Immunotoxicity“. In Microbial Toxins in Foods and Feeds, 427–40. Boston, MA: Springer US, 1990. http://dx.doi.org/10.1007/978-1-4613-0663-4_40.

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Selvakumar, Raman, Dalasanuru Chandregowda Manjunathagowda, Arun Kumar Pandey, Dipendra Kumar Mahato, Akansha Gupta, Shikha Pandhi, Raveena Kargwal, Madhu Kamle und Pradeep Kumar. „Detection and Management Strategies for Deoxynivalenol in Food and Feed“. In Mycotoxins in Food and Feed, 119–55. Boca Raton: CRC Press, 2023. http://dx.doi.org/10.1201/9781003242208-5.

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Lombaert, Gary A. „Methods for the Determination of Deoxynivalenol and other Trichothecenes in Foods“. In Advances in Experimental Medicine and Biology, 141–53. Boston, MA: Springer US, 2002. http://dx.doi.org/10.1007/978-1-4615-0629-4_14.

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Pieters, Moniek N., Jan Freijer, Bert-Jan Baars, Daniëlle C. M. Fiolet, Jacob van Klaveren und Wout Slob. „Risk Assessment of Deoxynivalenol in Food: Concentration Limits, Exposure and Effects“. In Advances in Experimental Medicine and Biology, 235–48. Boston, MA: Springer US, 2002. http://dx.doi.org/10.1007/978-1-4615-0629-4_25.

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Snijders, C. H. A., und C. F. Krechting. „Inhibition of Deoxynivalenol Translocation and Fungal Colonization in Fusarium Head Blight Resistant Wheat“. In Durability of Disease Resistance, 348. Dordrecht: Springer Netherlands, 1993. http://dx.doi.org/10.1007/978-94-011-2004-3_74.

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Whitlow, Lon W., Ray L. Nebel und Winston M. Hagler. „The Association of Deoxynivalenol in Grain with Milk Production Loss in Dairy Cows“. In Mycotoxins, Wood Decay, Plant Stress, Biocorrosion, and General Biodeterioration, 131–39. Boston, MA: Springer US, 1994. http://dx.doi.org/10.1007/978-1-4757-9450-2_11.

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Birzele, B., A. Meier, H. Hindorf, J. Krämer und H. W. Dehne. „Epidemiology of Fusarium infection and deoxynivalenol content in winter wheat in the Rhineland, Germany“. In Mycotoxins in Plant Disease, 667–73. Dordrecht: Springer Netherlands, 2002. http://dx.doi.org/10.1007/978-94-010-0001-7_9.

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Konferenzberichte zum Thema "Deoxynivalenol"

Yang, Hong, Wen-Ming Cao, Xue-Qing Wei und Xuebing Xu. „Simultaneous determination of deoxynivalenol and deoxynivalenol-3-glucoside in wheat and its products by HPLC-UV with immunoaffinity cleanup“. In Virtual 2021 AOCS Annual Meeting & Expo. AOCS, 2021. http://dx.doi.org/10.21748/am21.505.

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Su, Wen-Hao. „Rapid Assessment of Deoxynivalenol Content in Barley Using Hyperspectral imaging“. In 2021 ASABE Annual International Virtual Meeting, July 12-16, 2021. St. Joseph, MI: American Society of Agricultural and Biological Engineers, 2021. http://dx.doi.org/10.13031/aim.202100348.

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Chen, Zhenzhen, Lixin Zhu, Renrong Liu, Wei Meng, Na Wu, Fanfan Yang, Kaihong Li und Yifan Lang. „Determination of deoxynivalenol in grain sorghum by chemiluminescence enzyme immunoassay“. In 2016 3rd International Conference on Materials Engineering, Manufacturing Technology and Control. Paris, France: Atlantis Press, 2016. http://dx.doi.org/10.2991/icmemtc-16.2016.19.

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Sanchis, Vicente, Arnau Vidal, Antonio J. Ramos und Sonia Marin. „Stability of Deoxynivalenol and Ochratoxin A Through the Bread-Making Process“. In XII Latin American Congress on Food Microbiology and Hygiene. São Paulo: Editora Edgard Blücher, 2014. http://dx.doi.org/10.5151/foodsci-microal-347.

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Bondalapati, Krishna D., Jeffrey M. Stein und Kathleen M. Baker. „Neural network model to predict deoxynivalenol (DON) in barley using historic and forecasted weather conditions“. In 2012 First International Conference on Agro-Geoinformatics. IEEE, 2012. http://dx.doi.org/10.1109/agro-geoinformatics.2012.6311618.

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Mignani, A. G., L. Ciaccheri, A. A. Mencaglia, A. De Girolamo, V. Lippolis und M. Pascale. „Rapid screening of wheat bran contaminated by deoxynivalenol mycotoxin using Raman spectroscopy: a preliminary experiment“. In Sixth European Workshop on Optical Fibre Sensors (EWOFS'2016), herausgegeben von Elfed Lewis. SPIE, 2016. http://dx.doi.org/10.1117/12.2235910.

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K. H. S. Peiris, Y. Dong, W. W. Bockus und F. E. Dowell. „Estimation of Bulk Deoxynivalenol and Moisture Content of Wheat Grain Samples by FT-NIR Spectroscopy“. In 2013 Kansas City, Missouri, July 21 - July 24, 2013. St. Joseph, MI: American Society of Agricultural and Biological Engineers, 2013. http://dx.doi.org/10.13031/aim.20131593402.

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Yang, Xudong, Siyao Ju, Mengjie Liu, Yuqi Wang, Yupan Zhu, Ruonan Ma und Zhen Jiao. „Comparative Analysis of Cold Atmospheric Plasma on the Degradation of Deoxynivalenol in Solid and Aqueous State“. In 2021 IEEE International Conference on Plasma Science (ICOPS). IEEE, 2021. http://dx.doi.org/10.1109/icops36761.2021.9588420.

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Mateo, F., A. Medina, Eva M. Mateo, F. M. Valle-Algarra und M. Jiménez. „Capacity of neural network models to predict deoxynivalenol build-up in barley grain contaminated in vitro with Fusarium culmorum“. In Proceedings of the III International Conference on Environmental, Industrial and Applied Microbiology (BioMicroWorld2009). WORLD SCIENTIFIC, 2010. http://dx.doi.org/10.1142/9789814322119_0146.

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Seah, Stephen, Ting Zhou, Nadine Abraham und Jason Carere. „Engineering the NADPH specificity of DepB, a novel aldo-keto reductase involved in the detoxification of the agroeconomic mycotoxin deoxynivalenol (DON)“. In 1st International Electronic Conference on Toxins. Basel, Switzerland: MDPI, 2021. http://dx.doi.org/10.3390/iect2021-09171.

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Berichte der Organisationen zum Thema "Deoxynivalenol"

Webster, Richard W., Maxwell O. Chibuogwu, Hannah Reed, Brian Mueller, Carol L. Groves, Albert U. Tenuta, Martin I. Chilvers, Kiersten A. Wise und Damon Smith. Disease Development and Deoxynivalenol Accumulation in Silage Corn. United States of America: Crop Protection Netework, November 2021. http://dx.doi.org/10.31274/cpn-20211130-000.

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Pereboom, D. P. K. H., R. C. J. Dam, T. C. van Rijk, M. de Nijs und J. G. J. Mol. Proficiency test for deoxynivalenol (DON), acetyl-DONs and DON-3G in cereals : EURL-PT_MP01 (2018). Wageningen: RIKILT Wageningen University & Research, 2019. http://dx.doi.org/10.18174/476812.

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Zhou, Ting, Roni Shapira, Peter Pauls, Nachman Paster und Mark Pines. Biological Detoxification of the Mycotoxin Deoxynivalenol (DON) to Improve Safety of Animal Feed and Food. United States Department of Agriculture, Juli 2010. http://dx.doi.org/10.32747/2010.7613885.bard.

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Annotation:

The trichothecene deoxynivalenol (DON, vomitoxin), one of the most common mycotoxin contaminants of grains, is produced by members of the Fusarium genus. DON poses a health risk to consumers and impairs livestock performance because it causes feed refusal, nausea, vomiting, diarrhea, hemolytic effects and cellular injury. The occurrence of trichothecenes contamination is global and they are very resistant to physical or chemical detoxification techniques. Trichothecenes are absorbed in the small intestine into the blood stream. The overall objective of this project was to develop a protecting system using probiotic bacteria that will express trichothecene 3-O-acetyltransferase (Tri101) that convert T-2 to a less toxic intermediate to reduce ingested levels in-situ. The major obstacle that we had faced during the project is the absence of stable and efficient expression vectors in probiotics. Most of the project period was invested to screen and isolate strong promoter to express high amounts of the detoxify enzyme on one hand and to stabilize the expression vector on the other hand. In order to estimate the detoxification capacity of the isolated promoters we had developed two very sensitive bioassays.The first system was based on Saccharomyces cerevisiae cells expressing the green fluorescent protein (GFP). Human liver cells proliferation was used as the second bioassay system.Using both systems we were able to prove actual detoxification on living cells by probiotic bacteria expressing Tri101. The first step was the isolation of already discovered strong promoters from lactic acid bacteria, cloning them downstream the Tri101 gene and transformed vectors to E. coli, a lactic acid bacteria strain Lactococcuslactis MG1363, and a probiotic strain of Lactobacillus casei. All plasmid constructs transformed to L. casei were unstable. The promoter designated lacA found to be the most efficient in reducing T-2 from the growth media of E. coli and L. lactis. A prompter library was generated from L. casei in order to isolate authentic probiotic promoters. Seven promoters were isolated, cloned downstream Tri101, transformed to bacteria and their detoxification capability was compared. One of those prompters, designated P201 showed a relatively high efficiency in detoxification. Sequence analysis of the promoter region of P201 and another promoter, P41, revealed the consensus region recognized by the sigma factor. We further attempted to isolate an inducible, strong promoter by comparing the protein profiles of L. casei grown in the presence of 0.3% bile salt (mimicking intestine conditions). Six spots that were consistently overexpressed in the presence of bile salts were isolated and identified. Their promoter reigns are now under investigation and characterization.

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