Thematische Bibliographien / Epigenomic regulators

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Auswahl der wissenschaftlichen Literatur zum Thema „Epigenomic regulators“

Autor: Grafiati

Veröffentlicht am 25. Mai 2024

Zuletzt aktualisiert am 31. Juli 2025

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Zeitschriftenartikel zum Thema "Epigenomic regulators"

Al-Janabi, Ismail. "Therapeutic Targeting of the Regulators of Cancer Epigenomes." Al-Rafidain Journal of Medical Sciences ( ISSN 2789-3219 ) 5 (July 1, 2023): 1–13. http://dx.doi.org/10.54133/ajms.v5i.128.

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Aim: To assess the value of targeting the various molecules that regulate the epigenome in the management of cancer. Method: Peer-reviewed articles were examined in PubMed, Google Scholar, and ResearchGate search tools using keywords given in the manuscript. Main points: Three major epigenomic modifications, namely DNA methylation, histone methylation, and histone acetylation, attracted the most research interest and led to a few globally approved drugs for the treatment of various malignancies. The DNA methylation profiles of cancer have been successfully employed in many aspects of the manag

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Paul, Aswathy Mary, Madhavan Radhakrishna Pillai, and Rakesh Kumar. "Prognostic Significance of Dysregulated Epigenomic and Chromatin Modifiers in Cervical Cancer." Cells 10, no. 10 (2021): 2665. http://dx.doi.org/10.3390/cells10102665.

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To broaden the understanding of the epigenomic and chromatin regulation of cervical cancer, we examined the status and significance of a set of epigenomic and chromatin modifiers in cervical cancer using computational biology. We observed that 61 of 917 epigenomic and/or chromatin regulators are differentially upregulated in human cancer, including 25 upregulated in invasive squamous cell carcinomas and 29 in cervical intraepithelial neoplasia 3 (CIN3), of which 14 are upregulated in cervical intraepithelial neoplasia 2 (CIN2). Interestingly, 57 of such regulators are uniquely upregulated in c

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Schmitz, Ulf, Jaynish S. Shah, Bijay P. Dhungel, et al. "Widespread Aberrant Alternative Splicing despite Molecular Remission in Chronic Myeloid Leukaemia Patients." Cancers 12, no. 12 (2020): 3738. http://dx.doi.org/10.3390/cancers12123738.

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Vast transcriptomics and epigenomics changes are characteristic of human cancers, including leukaemia. At remission, we assume that these changes normalise so that omics-profiles resemble those of healthy individuals. However, an in-depth transcriptomic and epigenomic analysis of cancer remission has not been undertaken. A striking exemplar of targeted remission induction occurs in chronic myeloid leukaemia (CML) following tyrosine kinase inhibitor (TKI) therapy. Using RNA sequencing and whole-genome bisulfite sequencing, we profiled samples from chronic-phase CML patients at diagnosis and rem

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Tseng, Yen-Tzu, Hung-Fu Liao, Chih-Yun Yu, Chu-Fan Mo, and Shau-Ping Lin. "Epigenetic factors in the regulation of prospermatogonia and spermatogonial stem cells." REPRODUCTION 150, no. 3 (2015): R77—R91. http://dx.doi.org/10.1530/rep-14-0679.

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Appropriate regulation of epigenome within cells is crucial for the determination of cell fate and contributes to the lifelong maintenance of tissue homeostasis. Epigenomic re-establishment during embryonic prospermatogonia development and fine-tune of the epigenetic landscape in postnatal spermatogonial stem cells (SSCs) are two key processes required for functional male germ cell formation. Repression of re-activated transposons and male germline-specific epigenome establishment occur in prospermatogonia, whereas modulations of the epigenetic landscape is important for SSC self-renewal and d

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Zhou, Huaijun. "97 Dissection of Evolution of Cis-Regulatory Elements and Its Application on Genetic Control of Complex Traits in Farm Animals." Journal of Animal Science 101, Supplement_3 (2023): 51–52. http://dx.doi.org/10.1093/jas/skad281.063.

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Abstract Functional Annotation of Animal Genomes (FAANG) has made significant progress on the improvement of the annotation of the functional elements, particularly non-coding regions in farm animal genomes. Different epigenomes including ChIP-seq for 4 histone marks, ATAC-seq, CTCF-seq, RNA-seq, and Hi-C across different tissues in different animal species were generated. Thousands of cis-regulatory elements including promoters and enhancers across different tissues in different species were identified and annotated. A comprehensive characterization and comparison of cis-regulatory elements a

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Deng, Xian, Xianwei Song, Liya Wei, Chunyan Liu, and Xiaofeng Cao. "Epigenetic regulation and epigenomic landscape in rice." National Science Review 3, no. 3 (2016): 309–27. http://dx.doi.org/10.1093/nsr/nww042.

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Abstract Epigenetic regulation has been implicated in the control of complex agronomic traits in rice (Oryza sativa), a staple food crop and model monocot plant. Recent advances in high-throughput sequencing and the moderately complex genome of rice have made it possible to study epigenetic regulation in rice on a genome-wide scale. This review discusses recent advances in our understanding of epigenetic regulation in rice, with an emphasis on the roles of key epigenetic regulators, the epigenomic landscape, epigenetic variation, transposon repression, and plant development.

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Rada-Iglesias, Alvaro, Ruchi Bajpai, Sara Prescott, Samantha A. Brugmann, Tomek Swigut, and Joanna Wysocka. "Epigenomic Annotation of Enhancers Predicts Transcriptional Regulators of Human Neural Crest." Cell Stem Cell 11, no. 5 (2012): 633–48. http://dx.doi.org/10.1016/j.stem.2012.07.006.

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Smetanina, Mariya A., Valeria A. Korolenya, Alexander E. Kel, et al. "Epigenome-Wide Changes in the Cell Layers of the Vein Wall When Exposing the Venous Endothelium to Oscillatory Shear Stress." Epigenomes 7, no. 1 (2023): 8. http://dx.doi.org/10.3390/epigenomes7010008.

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Epigenomic changes in the venous cells exerted by oscillatory shear stress towards the endothelium may result in consolidation of gene expression alterations upon vein wall remodeling during varicose transformation. We aimed to reveal such epigenome-wide methylation changes. Primary culture cells were obtained from non-varicose vein segments left after surgery of 3 patients by growing the cells in selective media after magnetic immunosorting. Endothelial cells were either exposed to oscillatory shear stress or left at the static condition. Then, other cell types were treated with preconditione

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Boix, Carles A., Benjamin T. James, Yongjin P. Park, Wouter Meuleman, and Manolis Kellis. "Regulatory genomic circuitry of human disease loci by integrative epigenomics." Nature 590, no. 7845 (2021): 300–307. http://dx.doi.org/10.1038/s41586-020-03145-z.

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AbstractAnnotating the molecular basis of human disease remains an unsolved challenge, as 93% of disease loci are non-coding and gene-regulatory annotations are highly incomplete1–3. Here we present EpiMap, a compendium comprising 10,000 epigenomic maps across 800 samples, which we used to define chromatin states, high-resolution enhancers, enhancer modules, upstream regulators and downstream target genes. We used this resource to annotate 30,000 genetic loci that were associated with 540 traits4, predicting trait-relevant tissues, putative causal nucleotide variants in enriched tissue enhance

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Williams, Ruth M., Guneş Taylor, Irving T. C. Ling, et al. "Chromatin remodeller Chd7 is developmentally regulated in the neural crest by tissue-specific transcription factors." PLOS Biology 22, no. 10 (2024): e3002786. http://dx.doi.org/10.1371/journal.pbio.3002786.

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Neurocristopathies such as CHARGE syndrome result from aberrant neural crest development. A large proportion of CHARGE cases are attributed to pathogenic variants in the gene encoding CHD7, chromodomain helicase DNA binding protein 7, which remodels chromatin. While the role for CHD7 in neural crest development is well documented, how this factor is specifically up-regulated in neural crest cells is not understood. Here, we use epigenomic profiling of chick and human neural crest to identify a cohort of enhancers regulating Chd7 expression in neural crest cells and other tissues. We functional

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Dissertationen zum Thema "Epigenomic regulators"

Ferré, Quentin. "Leveraging combinations of epigenomic regulators." Electronic Thesis or Diss., Aix-Marseille, 2021. http://www.theses.fr/2021AIXM0151.

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A régulation cis-génomique chez l’homme est effectuée par des régulateurs de la chromatine, tels que les marques d’histones et les régulateurs de transcription (TR). Ils fonctionnent rarement seuls, mais plutôt en complexes. Le développement du séquençage NGS fournit des méthodes pour étudier cette régulation, incluant le ChIP-seq. Le but de cette thèse est de tirer parti de ces combinaisons avec des méthodes d’apprentissage automatique, efficaces pour apprendre les régularités dans les données. Nous représentons les régions d’intérêt sous forme de listes d’intervalles, converties en représent

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DAS, VIVEK. "LEVERAGING TRANSCRIPTOMIC ANALYSIS TO IDENTIFY TRANSCRIPTION FACTORS ORCHESTRATING CANCER PROGRESSION." Doctoral thesis, Università degli Studi di Milano, 2018. http://hdl.handle.net/2434/559711.

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Next generation sequencing (NGS) technology is currently employed to explore the molecular profiles associated to different biological contexts. The application of this technology provides at same time a high-resolution and global view of the genome and epigenome phenomena, enabling us to study the molecular events underlying many human diseases, including cancer. Our lab tries to exploit the utility of high throughput sequencing technologies generating genomic, transcriptomic and epigenomic data from patient's cohort to study the underlying molecular mechanisms that characterize the specific

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Jhanwar, Shalu 1986. "Computational analysis of epigenomic variability and its effect on regulatory activity." Doctoral thesis, Universitat Pompeu Fabra, 2017. http://hdl.handle.net/10803/580601.

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Epigenetics provides a plausible link between the environment and changes in gene expression that might contribute to disease phenotypes. The main goal of the thesis is to study epigenomic variability and their effect on the regulatory activity underlying chromatin dynamics. With an ultimate aim to identify regulatory variants driving cancer as well as disease specific epigenomic patterns in neurological diseases, the thesis deals with the development and subsequent implementation of a novel supervised machine-learning based enhancer predictor (GEP). Further, to address the role of DNA

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Jené, i. Sanz Alba 1984. "Integrative study of the regulatory and epigenomic programs involved in cancer development." Doctoral thesis, Universitat Pompeu Fabra, 2013. http://hdl.handle.net/10803/113380.

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El càncer ha estat tradicionalment considerat una malaltia fonamentalment genètica, però recentment s'està fent palès que la desregulació de mecanismes epigenètics contribueix en gran manera al desenvolupament tumoral. Al bell mig de la intersecció entre la genètica i l'epigenètica s'hi troben els factors reguladors de la cromatina (CRFs, en anglès), que són un focus important de recerca a causa de la seva potencial utilitat en teràpies contra el càncer. En aquesta tesi, determino l'estat transcriptòmic de cèl·lules normals i tumorals basant-me en informació epigenètica i regulatòria, i d

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Purcaro, Michael J. "Analysis, Visualization, and Machine Learning of Epigenomic Data." eScholarship@UMMS, 2017. https://escholarship.umassmed.edu/gsbs_diss/938.

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The goal of the Encyclopedia of DNA Elements (ENCODE) project has been to characterize all the functional elements of the human genome. These elements include expressed transcripts and genomic regions bound by transcription factors (TFs), occupied by nucleosomes, occupied by nucleosomes with modified histones, or hypersensitive to DNase I cleavage, etc. Chromatin Immunoprecipitation (ChIP-seq) is an experimental technique for detecting TF binding in living cells, and the genomic regions bound by TFs are called ChIP-seq peaks. ENCODE has performed and compiled results from tens of thousands of

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Zhu, Yan. "Microfluidic Technology for Low-Input Epigenomic Analysis." Diss., Virginia Tech, 2018. http://hdl.handle.net/10919/83402.

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Epigenetic modifications, such as DNA methylation and histone modifications, play important roles in gene expression and regulation, and are highly involved in cellular processes such as stem cell pluripotency/differentiation and tumorigenesis. Chromatin immunoprecipitation (ChIP) is the technique of choice for examining in vivo DNA-protein interactions and has been a great tool for studying epigenetic mechanisms. However, conventional ChIP assays require millions of cells for tests and are not practical for examination of samples from lab animals and patients. Automated microfluidic chips off

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Batra, Rajbir Nath. "Decoding the regulatory role and epiclonal dynamics of DNA methylation in 1482 breast tumours." Thesis, University of Cambridge, 2018. https://www.repository.cam.ac.uk/handle/1810/274923.

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Breast cancer is a clinically and molecularly heterogeneous disease displaying distinct therapeutic responses. Although recent studies have explored the genomic and transcriptomic landscapes of breast cancer, the epigenetic architecture has received less attention. To address this, an optimised Reduced Representation Bisulfite Sequencing protocol was performed on 1482 primary breast tumours (and 237 matched adjacent normal tissues). This constitutes the largest breast cancer methylome yet, and this thesis describes the bioinformatics and statistical analysis of this study. Noticeable epigeneti

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Bogatyrova, Olga [Verfasser], and Christoph [Akademischer Betreuer] Plass. "Mutations in regulators of the epigenome and their effects on the DNA methylome / Olga Bogatyrova ; Betreuer: Christoph Plass." Heidelberg : Universitätsbibliothek Heidelberg, 2016. http://d-nb.info/1180617304/34.

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Morikawa, Hiromasa. "Differential roles of epigenetic changes and Foxp3 expression in regulatory T cell-specific transcriptional regulation." Kyoto University, 2013. http://hdl.handle.net/2433/180610.

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Floc'hlay, Swann. "Computational analysis and modelling of regulatory networks controlling embryonic development." Electronic Thesis or Diss., Université Paris sciences et lettres, 2020. http://www.theses.fr/2020UPSLE036.

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La formation d’un embryon est dictée par la séquence ADN propre à cet organisme. La variabilité génétique donne naissance à une grande diversité morphologique, tout en maintenant une organisation générale robuste. Les mutations présentes dans les régions cis-régulatrices impactent la transcription via des mécanismes épigénomiques. La variabilité d’expression génique qui en découle peut être compensée par des mécanismes trans de rétrocontrôle au sein du réseau de régulation. L’organisation précise de ces interactions cis et trans restent encore difficile à déchiffrer. Afin de mieux saisir l’eff

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Bücher zum Thema "Epigenomic regulators"

Lusardi, Theresa A., and Detlev Boison. Ketogenic Diet, Adenosine, Epigenetics, and Antiepileptogenesis. Edited by Detlev Boison. Oxford University Press, 2016. http://dx.doi.org/10.1093/med/9780190497996.003.0023.

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Epilepsy is common, affecting about 1% of the population. Conventional treatments are ineffective in about one third of patients, and current therapies do not prevent epilepsy or its progression. For individuals with drug-refractory epilepsy the ketogenic diet (KD) can provide seizure relief in approximately fifty percent of patients, with complete and permanent remission in some cases, suggesting possible antiepileptogenic effects of the diet. Whereas mechanisms underlying antiseizure effects of KD have been identified, mechanistic links between KD therapy and antiepileptogenesis constitute a

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Buchteile zum Thema "Epigenomic regulators"

Zhu, Yan, and Chang Lu. "Microfluidic Chromatin Immunoprecipitation for Analysis of Epigenomic Regulations." In Microfluidic Methods for Molecular Biology. Springer International Publishing, 2016. http://dx.doi.org/10.1007/978-3-319-30019-1_16.

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Jhanwar, Shalu. "Computational Epigenomics and Its Application in Regulatory Genomics." In Bioinformatics: Sequences, Structures, Phylogeny. Springer Singapore, 2018. http://dx.doi.org/10.1007/978-981-13-1562-6_6.

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Matsumura, Yoshihiro, Timothy F. Osborne, Ryo Ito, Hiroki Takahashi, and Juro Sakai. "β-Adrenergic Signal and Epigenomic Regulatory Process for Adaptive Thermogenesis." In Advances in Experimental Medicine and Biology. Springer Nature Singapore, 2024. http://dx.doi.org/10.1007/978-981-97-4584-5_15.

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Hu, Yongfeng, and Dao-Xiu Zhou. "Rice Epigenomes: Characteristics, Regulatory Functions, and Reprogramming Mechanisms." In Rice Genomics, Genetics and Breeding. Springer Singapore, 2018. http://dx.doi.org/10.1007/978-981-10-7461-5_23.

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Roy Choudhury, Samrat, and Brian A. Walker. "Aberrant Epigenomic Regulatory Networks in Multiple Myeloma and Strategies for Their Targeted Reversal." In RNA Technologies. Springer International Publishing, 2019. http://dx.doi.org/10.1007/978-3-030-14792-1_22.

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Klann, Tyler S., Gregory E. Crawford, Timothy E. Reddy, and Charles A. Gersbach. "Screening Regulatory Element Function with CRISPR/Cas9-based Epigenome Editing." In Methods in Molecular Biology. Springer New York, 2018. http://dx.doi.org/10.1007/978-1-4939-7774-1_25.

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Jones, Peter A., and Minmin Liu. "Introduction to Drug Discovery in Epigenetics." In Epigenetic Drug Discovery. Royal Society of Chemistry, 2024. http://dx.doi.org/10.1039/9781837674916-00001.

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All human tumours contain epigenomic abnormalities which cooperate with genetic mutations to establish and drive the malignant state. Epigenetic therapy seeks to target the epigenome and three drug classes, DNA methyltransferase, histone deacetylase and histone methyltransferase inhibitors, are approved for clinical use. The first two drug classes were discovered unexpectedly in the mid-1970s due to their remarkable abilities to either reprogram cells or induce cellular differentiation in culture. Further development of epigenetic therapy will likely require the design of novel combination the

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Halene, Tobias B., Gregor Hasler, Amanda Mitchell, and Schahram Akbarian. "Epigenomic Exploration of the Human Brain." In Psychiatric Genetics. Oxford University Press, 2018. http://dx.doi.org/10.1093/med/9780190221973.003.0010.

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The exploration of the epigenome has become a flourishing area in the neurosciences. Scientists increasingly appreciate that even the position of genetic material within the nucleus is purposeful, and its spatial orientation conveys information with critical influence on transcription, genome integrity, and stability. Together, epigenetic and three-dimensional genome data hold promise to reveal how DNA variants and mutations come into play in brain disease. Powerful new technologies can now map transcriptome, DNA-methylome, and other epigenetic regulators on the level of single brain cells. Ma

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Zhou, Tong. "Small non-coding RNAs as epigenetic regulators." In Nutritional Epigenomics. Elsevier, 2019. http://dx.doi.org/10.1016/b978-0-12-816843-1.00003-5.

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Beetch, Megan, Sadaf Harandi-Zadeh, Kate Shen, and Barbara Stefanska. "Stilbenoids as dietary regulators of the cancer epigenome." In Nutritional Epigenomics. Elsevier, 2019. http://dx.doi.org/10.1016/b978-0-12-816843-1.00021-7.

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Konferenzberichte zum Thema "Epigenomic regulators"

Takamatsu, Hironori, Naoko Hattori, Naofumi Asano, et al. "Abstract 843: Epigenomic disruption of adipogenic regulators in dedifferentiated liposarcoma." In Proceedings: AACR Annual Meeting 2019; March 29-April 3, 2019; Atlanta, GA. American Association for Cancer Research, 2019. http://dx.doi.org/10.1158/1538-7445.sabcs18-843.

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GEVAERT, OLIVIER, and SYLVIA PLEVRITIS. "IDENTIFYING MASTER REGULATORS OF CANCER AND THEIR DOWNSTREAM TARGETS BY INTEGRATING GENOMIC AND EPIGENOMIC FEATURES." In Proceedings of the Pacific Symposium. WORLD SCIENTIFIC, 2012. http://dx.doi.org/10.1142/9789814447973_0013.

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Worsham, MJ, KM Chen, I. Datta, JK Stephen, D. Chitale, and G. Divine. "Abstract P1-04-06: Network integration of epigenomic data: Leveraging the concept of master regulators in ER negative breast cancer." In Abstracts: 2016 San Antonio Breast Cancer Symposium; December 6-10, 2016; San Antonio, Texas. American Association for Cancer Research, 2017. http://dx.doi.org/10.1158/1538-7445.sabcs16-p1-04-06.

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Battle, Stephanie L., Antti Larjo, Joling Liao, Harri Lähdesmäki, Andre Lieber, and R. David Hawkins. "Abstract AS04: Epigenomic characterization of gene regulatory networks in human ovarian cancer stem cells." In Abstracts: 10th Biennial Ovarian Cancer Research Symposium; September 8-9, 2014; Seattle, WA. American Association for Cancer Research, 2015. http://dx.doi.org/10.1158/1557-3265.ovcasymp14-as04.

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LaFave, Lindsay M., Vinay Kartha, Sai Ma, et al. "Abstract PR08: Leveraging single-cell epigenomics to uncover regulatory programs in lung adenocarcinoma." In Abstracts: AACR Special Conference on the Evolving Landscape of Cancer Modeling; March 2-5, 2020; San Diego, CA. American Association for Cancer Research, 2020. http://dx.doi.org/10.1158/1538-7445.camodels2020-pr08.

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Tricarico, Rossella, Pietro Mancuso, Vikram Bhattacharjee, et al. "Abstract LB-249: TDG, a dual genomic and epigenomic regulator, as a novel antimelanoma target." In Proceedings: AACR 106th Annual Meeting 2015; April 18-22, 2015; Philadelphia, PA. American Association for Cancer Research, 2015. http://dx.doi.org/10.1158/1538-7445.am2015-lb-249.

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XU, Liangliang, Feng WU, Otto K. W. CHEUNG, et al. "Abstract 868: Epigenomic profiling of primary hepatocellular carcinoma reveals super-enhancer-associated chromatin regulator network." In Proceedings: AACR Annual Meeting 2019; March 29-April 3, 2019; Atlanta, GA. American Association for Cancer Research, 2019. http://dx.doi.org/10.1158/1538-7445.sabcs18-868.

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Zacharias, W., M. Morley, D. T. Swarr, P. Senthamarai Kannan, M. C. Basil, and E. E. Morrisey. "Integrated Epigenomic Analysis of the Gene Regulatory Networks Underlying Regenerative Capacity in Alveolar Epithelial Progenitor Cells." In American Thoracic Society 2019 International Conference, May 17-22, 2019 - Dallas, TX. American Thoracic Society, 2019. http://dx.doi.org/10.1164/ajrccm-conference.2019.199.1_meetingabstracts.a4012.

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