Thematische Bibliographien / Gastrointestinal system Motility

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Auswahl der wissenschaftlichen Literatur zum Thema „Gastrointestinal system Motility“

Autor: Grafiati

Veröffentlicht am 4. Juni 2021

Zuletzt aktualisiert am 30. Juli 2024

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Zeitschriftenartikel zum Thema "Gastrointestinal system Motility"

Wood, Jackie D. "Enteric Nervous System: Neuropathic Gastrointestinal Motility." Digestive Diseases and Sciences 61, no. 7 (May 3, 2016): 1803–16. http://dx.doi.org/10.1007/s10620-016-4183-5.

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BURKS, THOMAS F. "Central Nervous System Regulation of Gastrointestinal Motility." Annals of the New York Academy of Sciences 597, no. 1 Neurobiology (July 1990): 36–42. http://dx.doi.org/10.1111/j.1749-6632.1990.tb16156.x.

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Wang, Po-Min, Genia Dubrovsky, James C. Y. Dunn, Yi-Kai Lo, and Wentai Liu. "A Wireless Implantable System for Facilitating Gastrointestinal Motility." Micromachines 10, no. 8 (August 9, 2019): 525. http://dx.doi.org/10.3390/mi10080525.

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Gastrointestinal (GI) electrical stimulation has been shown in several studies to be a potential treatment option for GI motility disorders. Despite the promising preliminary research progress, however, its clinical applicability and usability are still unknown and limited due to the lack of a miniaturized versatile implantable stimulator supporting the investigation of effective stimulation patterns for facilitating GI dysmotility. In this paper, we present a wireless implantable GI modulation system to fill this technology gap. The system consists of a wireless extraluminal gastrointestinal

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Alqudah, M., O. Al-Shboul, A. Al-Dwairi, D. G. Al-U´Dat, and A. Alqudah. "Progesterone Inhibitory Role on Gastrointestinal Motility." Physiological Research, no. 2 (April 30, 2022): 193–98. http://dx.doi.org/10.33549/physiolres.934824.

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Progesterone is a steroidal hormone that is produced from the corpus luteum of the ovaries and from the placenta. The main function of progesterone is to promote the secretory differentiation in the endometrium of the uterus and to maintain pregnancy by inhibiting uterine contractions throughout pregnancy. Progesterone performs its actions by activating the classical progesterone nuclear receptors that affect gene transcription and by the non-classical activation of cell surface membrane receptors that accounts for the rapid actions of progesterone. Besides the reproductive roles of progestero

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Stern, H. Patrick, Suzanne E. Stroh, Stephen C. Fiedorek, Kelly Kelleher, Michael W. Mellon, Sandra K. Pope, and Phillip L. Rayford. "Increased Plasma Levels of Pancreatic Polypeptide and Decreased Plasma Levels of Motilin in Encopretic Children." Pediatrics 96, no. 1 (July 1, 1995): 111–17. http://dx.doi.org/10.1542/peds.96.1.111.

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Objective. Abnormalities of hormones affecting gastrointestinal motility have been found in "functional" disorders of the gastrointestinal system in adults. One such disorder of childhood, encopresis, is frequently associated with constipation, the treatment of which often eliminates the soiling. We hypothesized that hormones affecting gastrointestinal motility were different between encopretic patients and matched controls. Methods. Ten encopretic patients were matched by age, race, and sex with controls who had no history of encopresis or constipation. After an overnight fast, each child con

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Lee, Yunna, Jeongbin Jo, Hae Young Chung, Charalabos Pothoulakis, and Eunok Im. "Endocannabinoids in the gastrointestinal tract." American Journal of Physiology-Gastrointestinal and Liver Physiology 311, no. 4 (October 1, 2016): G655—G666. http://dx.doi.org/10.1152/ajpgi.00294.2015.

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The endocannabinoid system mainly consists of endogenously produced cannabinoids (endocannabinoids) and two G protein-coupled receptors (GPCRs), cannabinoid receptors 1 and 2 (CB1 and CB2). This system also includes enzymes responsible for the synthesis and degradation of endocannabinoids and molecules required for the uptake and transport of endocannabinoids. In addition, endocannabinoid-related lipid mediators and other putative endocannabinoid receptors, such as transient receptor potential channels and other GPCRs, have been identified. Accumulating evidence indicates that the endocannabin

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Plourde, Victor. "Stress-Induced Changes in the Gastrointestinal Motor System." Canadian Journal of Gastroenterology 13, suppl a (1999): 26A—31A. http://dx.doi.org/10.1155/1999/320626.

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Several autonomic, hormonal, behavioural and neuropeptidergic bodily responses to stressful stimuli have been described over the past few decades. Both animal models and human paradigms have been explored. It is acknowledged that stress modulates gastrointestinal (GI) motility through central mechanisms including corticotropin-releasing-factor. This process requires the integrity of autonomic neural pathways. It has become evident that the effects of stress on GI motility vary according to the stressful stimulus, its intensity, the animal species under study and the time course of the study. R

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Milla, PJ. "Acquired Motility Disorders in Childhood." Canadian Journal of Gastroenterology 13, suppl a (1999): 76A—84A. http://dx.doi.org/10.1155/1999/610486.

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Acquired motility disorders in childhood cause a number of gastrointestinal symptoms – principally, recurrent vomiting, abdominal pain and distension, constipation and loose stools. Gastrointestinal motility disorders result from disturbances of the control mechanisms of gut motor activity, which may be produced by organic disease involving enteric nerves and muscle, perturbation of the humoral environment of the nerves and muscle, and altered central nervous system input. In children, both congenital and acquired disease processes may produce these pathogenetic mechanisms, resulting in syndro

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López-Pingarrón, Laura, Henrique Almeida, Marisol Soria-Aznar, Marcos C. Reyes-Gonzales, Ana B. Rodríguez-Moratinos, Antonio Muñoz-Hoyos, and Joaquín J. García. "Interstitial Cells of Cajal and Enteric Nervous System in Gastrointestinal and Neurological Pathology, Relation to Oxidative Stress." Current Issues in Molecular Biology 45, no. 4 (April 18, 2023): 3552–72. http://dx.doi.org/10.3390/cimb45040232.

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The enteric nervous system (ENS) is organized into two plexuses—submucosal and myenteric—which regulate smooth muscle contraction, secretion, and blood flow along the gastrointestinal tract under the influence of the rest of the autonomic nervous system (ANS). Interstitial cells of Cajal (ICCs) are mainly located in the submucosa between the two muscle layers and at the intramuscular level. They communicate with neurons of the enteric nerve plexuses and smooth muscle fibers and generate slow waves that contribute to the control of gastrointestinal motility. They are also involved in enteric ne

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Pan, H. L., Z. B. Zeisse, and J. C. Longhurst. "Mechanical stimulation is not responsible for activation of gastrointestinal afferents during ischemia." American Journal of Physiology-Heart and Circulatory Physiology 272, no. 1 (January 1, 1997): H99—H106. http://dx.doi.org/10.1152/ajpheart.1997.272.1.h99.

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Abdominal ischemia reflexly excites the cardiovascular system through activation of visceral sympathetic afferents. Although a number of ischemic metabolites are known to stimulate sympathetic afferents, the contribution of mechanical stimulation to activation of afferents during abdominal ischemia remains uncertain. Thus the present study examined the role of changes in motility in activation of gastrointestinal afferents during ischemia. Single-unit activity of C fiber afferents located on the stomach, duodenum, jejunum, or colon was recorded from the right sympathetic chain of anesthetized

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Dissertationen zum Thema "Gastrointestinal system Motility"

Andrews, Jane Mary. "Relationships between motor and sensory function in the proximal gut, appetite, & nutrients in healthy human subjects." Title page, contents and summary only, 1999. http://web4.library.adelaide.edu.au/theses/09PH/09pha567.pdf.

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Bibliography: leaves 206-251. The motor and sensory interactions between nutrients and proximal gut in humans are not well understood, despite the pivotal importance of these interactions on appetite, absorption and thus, nutrition. In part, this lack of knowledge results from technical difficulties in studying motor function in the human gut. In particular, the inability to continuously measure intraluminal flow with any degree of temporal resolution, has impeded progress in this field. The studies described in this thesis focus on nutrient-gut interactions, and also on the development of nov

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Spear, Estelle Trego. "Altered Gastrointestinal Motility in Multiple Sclerosis." ScholarWorks @ UVM, 2018. https://scholarworks.uvm.edu/graddis/837.

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Multiple sclerosis (MS) is an autoimmune disease of the central nervous system that causes motor, visual, and sensory symptoms. Patients also experience constipation, which is not yet understood, but could involve dysfunction of the enteric nervous system (ENS). Autoimmune targeting of the ENS occurs in other autoimmune diseases that exhibit gastrointestinal (GI) symptoms, and similar mechanisms could lead to GI dysfunction in MS. Here, we characterize GI dysmotility in the experimental autoimmune encephalomyelitis (EAE) model of MS and test whether autoantibodies targeting the ENS are present

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Jones, Karen Louise. "Studies of normal and disordered gastric motility in humans /." Title page, table of contents and summary only, 1997. http://web4.library.adelaide.edu.au/theses/09PH/09phj777.pdf.

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Boillat, Carol Simone. "Investigation of gastrointestinal motility in dogs using a wireless capsule system /." [S.l.] : [s.n.], 2009. http://www.ub.unibe.ch/content/bibliotheken_sammlungen/sondersammlungen/dissen_bestellformular/index_ger.html.

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HIRNING, LANE DURAND. "MULTIPLE PEPTIDE RECEPTORS AND SITES OF ACTION IN THE CANINE SMALL INTESTINE (OPIOIDS, MOTILIN, TACHYKININS, INTESTINAL MOTILITY, SUBSTANCE P)." Diss., The University of Arizona, 1986. http://hdl.handle.net/10150/188150.

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Motility of the small intestine is a result of complex neurochemical and hormonal interactions within the intestine. The net motility (contraction) of the intestine is a balance of the influences from the central nervous system, enteric nervous system and hormonal changes in the body. Recently, the discovery of several peptide neurotransmitters common to the brain and the intestine has stimulated new research into the influence of these novel neurotransmitter candidates on intestinal motility at the level of the enteric (intestinal) nervous system. The present studies examined the contractile

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Lo, Wing-joe. "Effects of neurotransmitters and peptides on gastrointestinal motility in the shark, hemiscyllium plagiosum (Bennett) /." [Hong Kong : University of Hong Kong], 1993. http://sunzi.lib.hku.hk/hkuto/record.jsp?B13597322.

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羅穎祖 and Wing-joe Lo. "Effects of neurotransmitters and peptides on gastrointestinal motilityin the shark, hemiscyllium plagiosum (Bennett)." Thesis, The University of Hong Kong (Pokfulam, Hong Kong), 1993. http://hub.hku.hk/bib/B3123382X.

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Zhao, Ling. "Increased bile acid-metabolizing bacteria contributes to enhanced gastrointestinal motility in irritable bowel syndrome." HKBU Institutional Repository, 2018. https://repository.hkbu.edu.hk/etd_oa/561.

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Irritable bowel syndrome (IBS), majorly characterized by irregular bowel movements and abdominal pain, is one of the most prevalent functional gastrointestinal disorders (FGIDs) in the world. Disturbance of gut microbiota, closely linking with gut dysfunction, has been regarded as one of important pathogenetic factors for IBS. However, gut microbiota-driven mechanism underlying IBS remains unclear, which leads to inefficient and non-specific effects of current microbiota-oriented therapy. In this thesis, function-based microbiota investigation with combination of metagenomic and metabolomic an

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Fone, David R. "Studies of the function of the human pylorus : and its role in the regulation of gastric emptying / David R. Fone." Title page, contents and summary only, 1990. http://web4.library.adelaide.edu.au/theses/09MD/09mdf673.pdf.

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Branstutter, Joseph W. "The role of nitric oxide in altering intestinal motility in lipopolysaccharide-injected rats : a morphological and functional assessment." Virtual Press, 1999. http://liblink.bsu.edu/uhtbin/catkey/1136700.

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Nitric oxide, a short-lived free radical and neurotransmitter, is responsible for decreased smooth muscle contractility in vitro. When in excess, NO can cause hypotension and is believed to mediate altered intestinal motility. Not enough evidence is available for morphological changes in gastrointestinal smooth muscle and its correlation with motility disorders caused by Escherichia coli-induced NO production. Male Lewis rats were treated with injections of 10 mg/kg LPS from E. coli with or without 12.5 mg/kg of NOS inhibitor, LNMMA. Eighteen to 24 hours following injection, duodenum, ileum, c

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Bücher zum Thema "Gastrointestinal system Motility"

W, Read N., ed. Gastrointestinal motility : which test? Peterfield: Wrightson Biomedical, 1989.

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Hongō, Michio, editor of compilation, ed. Functional and GI motility disorders. Basel: Karger, 2014.

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G, Schultz Stanley, Rauner Brenda B, Wood Jack D, and American Physiological Society (1887- ), eds. The Gastrointestinal system. Bethesda, Md: American Physiological Society, 1989.

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Devinder, Kumar, and Gustavsson Sven, eds. An Illustrated guide to gastrointestinal motility. 2nd ed. Edinburgh: Churchill Livingstone, 1993.

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Michael, Camillieri, and Stanghellini Vincenzo, eds. Manometric diagnosis of gastrointestinal motility disorders. New York: Thieme, 1986.

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S, Gaginella Timothy, ed. Regulatory mechanisms in gastrointestinal function. Boca Raton: CRC Press, 1995.

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Cheng, L. K. New Advances in Gastrointestinal Motility Research. Dordrecht: Springer Netherlands, 2013.

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S, Davison J., ed. Gastrointestinal secretion. London: Wright, 1989.

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International Workshop on Stress and Digestive Motility (1988 Mont Gabriel, Canada). Stress and digestive motility: Proceedings of the International Workshop on Stress and Digestive Motility. Edited by Bueno Lionel, Collins Stephen M, and Junien Jean-Louis. London: John Libbey, 1989.

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A, Kurygin A., ed. Motornai͡a︡ funkt͡s︡ii͡a︡ tonkoĭ kishki: V norme i pri nekotorykh patologicheskikh sostoi͡a︡nii͡a︡kh. Sankt-Peterburg: "Nauka", 1994.

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Buchteile zum Thema "Gastrointestinal system Motility"

Chang, Eugene B., and Po Sing Leung. "Gastrointestinal Motility." In The Gastrointestinal System, 35–62. Dordrecht: Springer Netherlands, 2014. http://dx.doi.org/10.1007/978-94-017-8771-0_2.

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Klaus, Jennifer. "Gastrointestinal system motility and integrity." In Monitoring and Intervention for the Critically Ill Small Animal, 267–83. Hoboken, NJ, USA: John Wiley & Sons, Inc., 2016. http://dx.doi.org/10.1002/9781118923870.ch15.

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Bredenoord, Albert J., André Smout, and Jan Tack. "Biliary System." In A Guide to Gastrointestinal Motility Disorders, 103–7. Cham: Springer International Publishing, 2016. http://dx.doi.org/10.1007/978-3-319-26938-2_9.

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Kini, Usha. "Pathology of the Gut Motility Disorders: Hirschsprung’s Disease." In Surgical Pathology of the Gastrointestinal System, 339–74. Singapore: Springer Singapore, 2022. http://dx.doi.org/10.1007/978-981-16-6395-6_10.

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Das, Prasenjit, and Prateek Kinra. "Pathology of Gut Motility Disorders: Chronic Intestinal Pseudoobstruction and Entities Other than Hirschsprung’s Disease." In Surgical Pathology of the Gastrointestinal System, 375–403. Singapore: Springer Singapore, 2022. http://dx.doi.org/10.1007/978-981-16-6395-6_11.

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Sanger, Gareth J., John Broad, Brid Callaghan, and John B. Furness. "Ghrelin and Motilin Control Systems in GI Physiology and Therapeutics." In Gastrointestinal Pharmacology, 379–416. Cham: Springer International Publishing, 2016. http://dx.doi.org/10.1007/164_2016_104.

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Pocock, Gillian, Christopher D. Richards, and David A. Richards. "Introduction to the gastrointestinal system." In Human Physiology. Oxford University Press, 2017. http://dx.doi.org/10.1093/hesc/9780198737223.003.53.

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This chapter describes the gross anatomical organization of the gastrointestinal system, the internal structure of the gut wall, and its blood supply. It provides an overview of the nervous and hormonal mechanisms that regulate the motility and secretory activity of the gut. It also presents a detailed examination of the structure and functions of the mouth and oesophagus, the stomach, and the small and large intestine, together with the digestive functions of the pancreas. The chapter outlines key aspects of nutrition and energy balance, noting how food is required by the body both for the production of energy and for the growth and repair of tissues. Each day an average adult consumes around 1 kg of solid food and 1-2 litres of fluid. The majority of this material is in a form that first must be broken down into simple molecules.

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Mischopoulou, Magdalini, and Gianluca Cipriani. "Muscularis Macrophages in Healthy and Diseased Gut." In Phagocytosis - Main Key of Immune System [Working Title]. IntechOpen, 2023. http://dx.doi.org/10.5772/intechopen.109889.

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Muscularis macrophages are a newly discovered population of macrophages distributed within the smooth muscle layers of the gastrointestinal tract. Muscularis macrophages are emerging as essential cell keepers of homeostatic gastrointestinal function, and when affected, can lead to functional gastrointestinal disorders. In this chapter, we briefly introduce the phenotype, the distribution of muscularis macrophages, and the difference compared with other tissue-resident macrophages. We next describe how they contribute to normal gastrointestinal function by interacting with cells required for gastrointestinal motility, such as enteric neurons. Finally, we highlight the increasing pieces of evidence suggesting the contribution of muscularis macrophages to gastrointestinal function diseases, such as gastrointestinal inflammation, gastroparesis and post operative ileus.

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Camilleri, Michael, and Adil E. Bharucha. "Disturbances of Gastrointestinal Motility and the Nervous System." In Neurology and General Medicine, 293–310. Elsevier, 2008. http://dx.doi.org/10.1016/b978-044306707-5.50019-5.

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Camilleri, Michael, and Adil E. Bharucha. "Disturbances of Gastrointestinal Motility and the Nervous System." In Aminoff's Neurology and General Medicine, 255–71. Elsevier, 2014. http://dx.doi.org/10.1016/b978-0-12-407710-2.00014-x.

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Konferenzberichte zum Thema "Gastrointestinal system Motility"

Yoshimoto, Kayo, Kenji Yamada, Kenji Watabe, Maki Takeda, Takahiro Nishimura, Michiko Kido, Toshiaki Nagakura, et al. "Evaluation of the three-dimensional endoscope system for assessing the gastrointestinal motility." In SPIE BiOS, edited by Tuan Vo-Dinh, Anita Mahadevan-Jansen, and Warren S. Grundfest. SPIE, 2014. http://dx.doi.org/10.1117/12.2041652.

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Pinheiro, Amanda Pereira Sindeaux, Pedro Vitor Ferreira Rodrigues, Raoni de Oliveira da Silva Domingues, and Leonardo José Rodrigues Araújo Melo. "Gastrointestinal dysmotility associated with Parkinson’s disease’s mechanism." In XIV Congresso Paulista de Neurologia. Zeppelini Editorial e Comunicação, 2023. http://dx.doi.org/10.5327/1516-3180.141s1.461.

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Introduction: Parkinson’s Disease (PD) is a condition of the brain that consiste of the death of dopaminergic neurons in the substantia nigra, therefore causing dyskinesias and dystonias. Besides the motor symptoms, the neurogastro motility is affected by the disease, since gastrointestinal dysfunction is a frequent and clinically relevant symptom of PD. Objectives: To link the neural pathways and neurotransmitters that involve the neuroenteric system control and the PD’s pathology. Methods: A systematic literature review was performed based on data extraction through the advanced research eng

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Nobe, Kazuki, Kayo Yoshimoto, Kenji Yamada, and Hideya Takahashi. "3D registration method for assessing the gastrointestinal motility using spectral reflectance estimation." In Advanced Biomedical and Clinical Diagnostic and Surgical Guidance Systems XVI, edited by Tuan Vo-Dinh, Anita Mahadevan-Jansen, and Warren S. Grundfest. SPIE, 2018. http://dx.doi.org/10.1117/12.2288383.

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