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Thematische Bibliographien / Glycosaminoglycans Metabolism / Zeitschriftenartikel

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Zeitschriftenartikel zum Thema „Glycosaminoglycans Metabolism“

Autor: Grafiati

Veröffentlicht am 4. Juni 2021

Zuletzt aktualisiert am 30. Januar 2023

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1

Viola, Manuela, Timothy E. L. Douglas, Laura Alaniz, and Barbara Bartolini. "Glycosaminoglycans Metabolism." Biochemistry Research International 2012 (2012): 1–2. http://dx.doi.org/10.1155/2012/245792.

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2

Mironov, S. P., A. M. Gerasimov, L. N. Furtseva, A. G. Tikhomirov, D. O. Vasiliev, and R. V. Merkurieva. "Oxyprolinuria and Glycosaminoglycansuria in Achilles Tendon Ruptures." N.N. Priorov Journal of Traumatology and Orthopedics 5, no. 2 (June 15, 1998): 51–53. http://dx.doi.org/10.17816/vto104492.

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In 17 athlets and ballet dancers with Achilles tendon ruptures oxyprolinuria and urine content of hexuronic acid were studied. Considerable increase of collagen and glycosaminoglycans decay products was detected. The results of differential spectrophotometry showed that urine glycosaminoglycanes presented by proteoglycans. It was assumed that Achilles tendon ruptures could be caused by excessive mechanical load and/or lack of the connective tissue metabolism. The authors consider that further study of oxyprolinuria as well as enzyme-substrate systems of glycosaminoglycans are perspective for t

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Koźma, Ewa M., Kornelia Kuźnik-Trocha, Katarzyna Winsz-Szczotka, Grzegorz Wisowski, Paweł Olczyk, Katarzyna Komosińska-Vassev, Mariusz Kasperczyk, and Krystyna Olczyk. "Significant Remodeling Affects the Circulating Glycosaminoglycan Profile in Adult Patients with both Severe and Mild Forms of Acute Pancreatitis." Journal of Clinical Medicine 9, no. 5 (May 1, 2020): 1308. http://dx.doi.org/10.3390/jcm9051308.

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Acute pancreatitis (AP) manifests itself either as a mild, self-limiting inflammation or a severe, systemic inflammatory process that is associated with various complications and a high mortality rate. It is unknown whether these two forms of the disease can differ in the profile of circulating glycosaminoglycans, which are molecules with huge biological reactivity due to a high density of negative electric charge. Plasma glycosaminoglycans were characterized/quantified in 23 healthy controls, 32 patients with mild AP, and 15 individuals with severe disease using electrophoresis with enzymatic

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Wolf, Hanna, Andrea Graßmann, Romina Bester, André Hossinger, Christoph Möhl, Lydia Paulsen, Martin H. Groschup, Hermann Schätzl, and Ina Vorberg. "Modulation of Glycosaminoglycans Affects PrPScMetabolism but Does Not Block PrPScUptake." Journal of Virology 89, no. 19 (July 22, 2015): 9853–64. http://dx.doi.org/10.1128/jvi.01276-15.

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ABSTRACTMammalian prions are unconventional infectious agents composed primarily of the misfolded aggregated host prion protein PrP, termed PrPSc. Prions propagate by the recruitment and conformational conversion of cellular prion protein into abnormal prion aggregates on the cell surface or along the endocytic pathway. Cellular glycosaminoglycans have been implicated as the first attachment sites for prions and cofactors for cellular prion replication. Glycosaminoglycan mimetics and obstruction of glycosaminoglycan sulfation affect prion replication, but the inhibitory effects on different st

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Kittlick, P. D. "Inflammation, glycolytic metabolism, and glycosaminoglycans." Experimental pathology 30, no. 1 (January 1986): 1–19. http://dx.doi.org/10.1016/s0232-1513(86)80051-2.

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6

Bower, L., C. Warren, and G. Manley. "Human Serum and Urine Glycosaminoglycans in Health and in Patients with Chronic Renal Failure." Annals of Clinical Biochemistry: International Journal of Laboratory Medicine 29, no. 2 (March 1992): 190–95. http://dx.doi.org/10.1177/000456329202900212.

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Quantitation of uronic acid precipitable by cetylpyridinium chloride (CPC) and electrophoretic separation of glycosaminoglycans were performed on sera from patients with chronic renal failure and compared to normal controls. Serum CPC-precipitable uronic acid (CpUA) levels in patients with renal failure were significantly higher (mean 13·7 mg/L, range 7·1–23·6 mg/L) than normal controls (mean 9·6 mg/L, range 5·1–13·9 mg/L) due to increased concentrations of low sulphated chondroitin sulphate. A positive correlation between serum CpUA and creatinine was found in renal failure patients. Urine Cp

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Kahaly, G., C. Stover, J. Beyer, and E. Otto. "In vitro synthesis of glycosaminoglycans in endocrine ophthalmopathy." Acta Endocrinologica 127, no. 5 (November 1992): 397–402. http://dx.doi.org/10.1530/acta.0.1270397.

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The effects of humoral and cell-mediated immunity on the glycosaminoglycan synthesis of retrobulbar fibroblasts was evaluated in patients with endocrine ophthalmopathy. After incubation with IgG and sera, secreted glycosaminoglycans, radiolabeled with D-6-3H-glucosamine and 35sulfate, were precipitated with cetylpyridinium chloride and ethanol. Hyaluronic acid synthesis of human retrobulbar fibroblasts after incubation with sera and IgG and after co-culture with lymphocytes was assessed by means of a radiometric test. Patients' IgG, compared to controls', accounted for a higher secretory stimu

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Chang, Chih-Cheng, Tien-Chun Chang, Shine CS Kao, Yea-Fhey Kuo, and Li-Fei Chien. "Pentoxifylline inhibits the proliferation and glycosaminoglycan synthesis of cultured fibroblasts derived from patients with Graves' ophthalmopathy and pretibial myxoedema." Acta Endocrinologica 129, no. 4 (October 1993): 322–27. http://dx.doi.org/10.1530/acta.0.1290322.

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Excessive amounts of glycosaminoglycans accumulate in the extraocular muscles of patients with Graves' ophthalmopathy and in the affected skin of patients with pretibial myxoedema. It is widely accepted that fibroblasts are the sources of glycosaminoglycan synthesis. Pentoxifylline, an analogue of the methylxanthine theobromine, inhibits the proliferation and certain biosynthetic activities of fibroblasts derived from normal human skin and from skin of patients with some fibrotic disorders. Our objective was to determine whether pentoxifylline has similar effects on fibroblasts derived from pa

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Bondar', I. A., and V. V. Klimontov. "Glycosaminoglycans and diabetic nephropathy." Problems of Endocrinology 50, no. 2 (April 15, 2004): 29–34. http://dx.doi.org/10.14341/probl11392.

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Diabetic nephropathy (DN) is one of the leading places in the structure of mortality of patients with diabetes mellitus (DM) in Russia and abroad. Despite intensive study, the causes and development mechanisms of this complication are not finally clear. Most often, diabetic kidney damage is seen as the result of a complex interaction of metabolic, hemodynamic, genetic and other mechanisms. At the same time, the majority of researchers give the leading role to hyperglycemia and the metabolic disorders triggered by it. The latter include intensification of non-enzymatic glycation processes, acti

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KAHALY, G., M. SCHULER, A. C. SEWELL, G. BERNHARD, J. BEYER, and U. KRAUSE. "URINARY GLYCOSAMINOGLYCANS IN Graves'OPHTHALMOPATHY." Clinical Endocrinology 33, no. 1 (July 1990): 35–44. http://dx.doi.org/10.1111/j.1365-2265.1990.tb00463.x.

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11

Unnikrishnan, V. S., and P. R. Sudhakaran. "Metabolism of glycosaminoglycans in CCl4-induced liver regeneration." Journal of Biosciences 14, no. 2 (June 1989): 163–72. http://dx.doi.org/10.1007/bf02703168.

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Kurup, Ravi Kumar, and Parameswara Achutha Kurup. "Hypothalamic digoxin-mediated model for epileptogenesis." Acta Neuropsychiatrica 15, no. 3 (June 2003): 115–21. http://dx.doi.org/10.1034/j.1601-5215.2003.00020.x.

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Background/aims:This study assessed the changes in the isoprenoid pathway and its metabolites in seizure disorder (ILAE classification – I generalized – idiopathic generalized epilepsy with age-related onset – epilepsy with generalized tonic clonic seizures on awakening) and the metabolic cascade produced by isoprenoid pathway dysregulation.Methods:The following parameters were assessed in seizure disorder: isoprenoid pathway metabolites, tyrosine and tryptophan catabolites, glycoconjugates metabolism and red blood cell (RBC) membrane composition.Results:There was elevation in plasma HMG-CoA r

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Morozenko, D. V., R. F. Yeromenko, K. V. Gliebova, O. P. Timoshenko, and A. V. Zakharyev. "Disorders of Proteoglycan and Collagen Metabolism in the Kidneys in Diabetes Mellitus: Clinical and Pathogenetic Mechanisms and Laboratory Markers." Ukraïnsʹkij žurnal medicini, bìologìï ta sportu 5, no. 6 (December 12, 2020): 355–61. http://dx.doi.org/10.26693/jmbs05.06.355.

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The article considers the issue of disorders of connective tissue metabolism in diabetes mellitus. Glycosylation of structural components of connective tissue and glucose toxicity have been found to determine the pathogenesis of late complications of diabetes mellitus. The most common concept of the pathogenesis of diabetes is metabolic, according to which all variants of diabetes mellitus, including blood vessels, their basement membrane, are associated with primary disorders of lipid, glycoprotein, protein and carbohydrate metabolism due to complete or partial insufficiency. It has been foun

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TANAKA, Tastuo. "The metabolism of glycosaminoglycans in gingiva of pregnant rat." Nihon Shishubyo Gakkai Kaishi (Journal of the Japanese Society of Periodontology) 28, no. 2 (1986): 516–30. http://dx.doi.org/10.2329/perio.28.516.

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15

Mayer-Sonnenfeld, Tehila, Marsha Zeigler, Michele Halimi, Yael Dayan, Christian Herzog, Corinne I. Lasmezas, and Ruth Gabizon. "The metabolism of glycosaminoglycans is impaired in prion diseases." Neurobiology of Disease 20, no. 3 (December 2005): 738–43. http://dx.doi.org/10.1016/j.nbd.2005.05.009.

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Staprans, I., J. M. Felts, and W. G. Couser. "Glycosaminoglycans and chylomicron metabolism in control and nephrotic rats." Metabolism 36, no. 5 (May 1987): 496–501. http://dx.doi.org/10.1016/0026-0495(87)90050-3.

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17

Steiner, Manfred. "Role of glycosaminoglycans in calcium metabolism of human platelets." Biochimica et Biophysica Acta (BBA) - Molecular Cell Research 886, no. 3 (May 1986): 406–10. http://dx.doi.org/10.1016/0167-4889(86)90176-x.

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Näntö-Salonen, Kirsti, Hannu Larjava, Maija Aalto, and Tarmo Kivimäki. "Urinary glycosaminoglycans in aspartylglycosaminuria: evidence for disturbed proteoglycan metabolism." Clinica Chimica Acta 146, no. 2-3 (March 1985): 111–18. http://dx.doi.org/10.1016/0009-8981(85)90049-x.

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Uijtdewilligen, P. J. E., E. M. Versteeg, E. M. A. van de Westerlo, J. van der Vlag, W. F. Daamen, and T. H. van Kuppevelt. "Dynamic Expression of Genes Involved in Proteoglycan/Glycosaminoglycan Metabolism during Skin Development." BioMed Research International 2018 (August 29, 2018): 1–16. http://dx.doi.org/10.1155/2018/9873471.

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Glycosaminoglycans are important for cell signaling and therefore for proper embryonic development and adult homeostasis. Expressions of genes involved in proteoglycan/glycosaminoglycan (GAG) metabolism and of genes coding for growth factors known to bind GAGs were analyzed during skin development by microarray analysis and real time quantitative PCR. GAG related genes were organized in six categories based on their role in GAG homeostasis, viz. (1) production of precursor molecules, (2) production of core proteins, (3) synthesis of the linkage region, (4) polymerization, (5) modification, and

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Imai, Yumi, Ritsuko Odajima, Yoichi Inoue, and Yoshimasa Shishiba. "Effect of growth factors on hyaluronan and proteoglycan synthesis by retroocular tissue fibroblasts of Graves' ophthalmopathy in culture." Acta Endocrinologica 126, no. 6 (June 1992): 541–52. http://dx.doi.org/10.1530/acta.0.1260541.

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One of the pathological changes seen in Graves' ophthalmopathy is the deposition of glycosaminoglycans such as hyaluronan and proteoglycan in retroocular connective tissue. We analyzed glycosaminoglycans synthesized by retroocular tissue fibroblasts in culture derived from an individual not suffering from thyroid disease and from three patients with Graves' ophthalmopathy. Retroocular tissue fibroblasts synthesized both hyaluronan and proteoglycan, the latter composed mainly of chondroitin sulfate. This contrasts with the proteoglycan synthesized by adult skin fibroblasts which was composed of

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KAHALY, GEORGE, GREGOR FÖRSTER, and CHRISTIANE HANSEN. "Glycosaminoglycans in Thyroid Eye Disease." Thyroid 8, no. 5 (May 1998): 429–32. http://dx.doi.org/10.1089/thy.1998.8.429.

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Vorontsov, A. V., I. I. Dedov, M. V. Shestakova, T. M. Milenkaya, and A. P. Knyazeva. "Glycosaminoglycans in therapy of diabetic nephropathy." Problems of Endocrinology 42, no. 5 (October 15, 1996): 14–18. http://dx.doi.org/10.14341/probl12082.

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Sulodexide, a drug containing glycosaminoglycans, was used in the treatment of patients with type I diabetes. Along with their effects on the blood clotting system, glycosaminoglycans are capable of preventing the mesangial proliferation and hyperproduction of extracellular matrix, as well as thickening of the glomerular basal membrane and impairment of its permeability and charge selection. A reliable antiproteinuric effect of the drug was noted, persisting for 6 weeks after it was discontinued; the excretion of protein with the urine reliably decreased in patients with both, microalbuminuria

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Baud'Huin, M., C. Ruiz Velasco, G. Jego, C. Charrier, M. Maillasson, F. Redini, D. Heymann, and L. Duplomb. "Glycosaminoglycans inhibit rankl-induced osteoclastogenesis." Bone 44 (June 2009): S334. http://dx.doi.org/10.1016/j.bone.2009.03.640.

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Khan, Shaukat, Carlos J. Alméciga-Díaz, Kazuki Sawamoto, William G. Mackenzie, Mary C. Theroux, Christian Pizarro, Robert W. Mason, Tadao Orii, and Shunji Tomatsu. "Mucopolysaccharidosis IVA and glycosaminoglycans." Molecular Genetics and Metabolism 120, no. 1-2 (January 2017): 78–95. http://dx.doi.org/10.1016/j.ymgme.2016.11.007.

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Winsz-Szczotka, Katarzyna, Łukasz Mencner, and Krystyna Olczyk. "Metabolism of glycosaminoglycans in the course of juvenile idiopathic arthritis." Postępy Higieny i Medycyny Doświadczalnej 70 (March 4, 2016): 135–42. http://dx.doi.org/10.5604/17322693.1196355.

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Pinto, D. O. "Biosynthesis and metabolism of sulfated glycosaminoglycans during Drosophila melanogaster development." Glycobiology 14, no. 6 (January 22, 2004): 529–36. http://dx.doi.org/10.1093/glycob/cwh070.

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SCHULER, M., G. KAHALY, A. C. SEWELL, H. SCHMIDT, G. BERNHARD, J. BEYER, and U. KRAUSE. "Urinary glycosaminoglycans in endocrine ophthalmopathy." Acta Endocrinologica 120, no. 3_Suppl (June 1989): S39—S40. http://dx.doi.org/10.1530/acta.0.120s039.

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Vyatkin, V. A., E. G. Butolin, and V. G. Ivanov. "Effect of chondroitin sulfate on the type I collagen metabolism in the compact bone in alloxan-induced rats." Kazan medical journal 96, no. 5 (October 15, 2015): 802–6. http://dx.doi.org/10.17750/kmj2015-802.

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Aim. To study the type I collagen metabolism in the compact bone in rats with alloxan-induced diabetes receiving sulfated glycosaminoglycans. Methods. The study was performed on 67 white outbred male rats with body weight of 180-220 g. Mortality at diabetes reproduction was 44.8%. To clarify the role of exogenous glycosaminoglycans on bone collagen metabolism at diabetes mellitus, 16 animals with alloxan-induced diabetes received 1 mg/kg of chondroitin sulfate intramuscularly every second day. The second group (21 animals) with alloxan-induced diabetes did not received any chondroitin su

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Kahaly, G., Ch Hansen, E. Otto, G. Förster, J. Beyer, and G. Hommel. "Diabetic microangiopathy and urinary glycosaminoglycans." Experimental and Clinical Endocrinology & Diabetes 105, no. 03 (July 14, 2009): 145–51. http://dx.doi.org/10.1055/s-0029-1211743.

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Bishop, R. E., and J. J. Torres. "Leptocephalus energetics: metabolism and excretion." Journal of Experimental Biology 202, no. 18 (September 15, 1999): 2485–93. http://dx.doi.org/10.1242/jeb.202.18.2485.

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Leptocephali are the unusual transparent larvae that are typical of eels, bonefish, tarpon and ladyfish. Unlike the larvae of all other fishes, leptocephali may remain in the plankton as larvae for several months before metamorphosing into the juvenile form. During their planktonic phase, leptocephali accumulate energy reserves in the form of glycosaminoglycans, which are then expended to fuel metamorphosis. The leptocephalus developmental strategy is thus fundamentally different from that exhibited in all other fishes in two respects: it is far longer in duration and energy reserves are accum

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Silbert, Jeremiah. "Metabolism and Structure-Function Relationships of Connective Tissue Glycosaminoglycans and Proteoglycans." Current Organic Chemistry 8, no. 5 (March 1, 2004): 395–411. http://dx.doi.org/10.2174/1385272043485864.

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SHIRAKI, Masafumi, Tatsuo TANAKA, Toshiko HORI, Kiyoshi MIZUNO, Yoshinobu MURAHASHI, Yukio IWAYAMA, and Tie-Zheng TAN. "Autoradiographic studies on the metabolism of gingival glycosaminoglycans in experimental periodontitis." Nihon Shishubyo Gakkai Kaishi (Journal of the Japanese Society of Periodontology) 31, no. 2 (1989): 667–74. http://dx.doi.org/10.2329/perio.31.667.

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Lucon, Marcos, Joao Roberto Martins, Katia Ramos Moreira Leite, Roberto Soler, Helena B. Nader, Miguel Srougi, and Homero Bruschini. "Evaluation of the metabolism of glycosaminoglycans in patients with interstitial cystis." International braz j urol 40, no. 1 (January 2014): 72–79. http://dx.doi.org/10.1590/s1677-5538.ibju.2014.01.11.

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Luikart, SD, JL Sackrison, and CV Thomas. "Altered glycosaminoglycan production by HL-60 cells treated with 4- methylumbelliferyl-beta-D-xyloside." Blood 66, no. 4 (October 1, 1985): 866–72. http://dx.doi.org/10.1182/blood.v66.4.866.866.

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Abstract Glycosaminoglycans, mainly chondroitin 4-sulfate, are located in the primary granules of human myeloid cells. These polyanionic carbohydrates are believed to play an important role in leukocyte maturation and function. To study the effect of altered chondroitin sulfate metabolism on human promyelocytic leukemia cells, we have treated HL-60 cells with 4-methylumbelliferyl-beta-D-xyloside. beta-D- Xylosides initiate the synthesis of free chondroitin sulfate chains. Cytochemical studies of treated cells demonstrated a marked increase in cytoplasmic granules stained with cationic dyes. Th

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Luikart, SD, JL Sackrison, and CV Thomas. "Altered glycosaminoglycan production by HL-60 cells treated with 4- methylumbelliferyl-beta-D-xyloside." Blood 66, no. 4 (October 1, 1985): 866–72. http://dx.doi.org/10.1182/blood.v66.4.866.bloodjournal664866.

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Glycosaminoglycans, mainly chondroitin 4-sulfate, are located in the primary granules of human myeloid cells. These polyanionic carbohydrates are believed to play an important role in leukocyte maturation and function. To study the effect of altered chondroitin sulfate metabolism on human promyelocytic leukemia cells, we have treated HL-60 cells with 4-methylumbelliferyl-beta-D-xyloside. beta-D- Xylosides initiate the synthesis of free chondroitin sulfate chains. Cytochemical studies of treated cells demonstrated a marked increase in cytoplasmic granules stained with cationic dyes. This was co

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Masuda, H., T. Ozeki, I. Takazono, and Y. Tanaka. "Analyses of glycosaminoglycans in human lung cancer." Biochemical Medicine and Metabolic Biology 37, no. 3 (June 1987): 366–73. http://dx.doi.org/10.1016/0885-4505(87)90050-8.

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Masuda, H., T. Ozeki, I. Takazono, and Y. Tanaka. "Composition of glycosaminoglycans in human pancreatic cancer." Biochemical Medicine and Metabolic Biology 41, no. 3 (June 1989): 193–200. http://dx.doi.org/10.1016/0885-4505(89)90026-1.

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Ashhurst, D. E., and M. Page. "Collagens and glycosaminoglycans associated with fracture healing." Bone 7, no. 5 (January 1986): 393. http://dx.doi.org/10.1016/8756-3282(86)90279-6.

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Gambaro, Giovanni, and Bruno Baggio. "Role of glycosaminoglycans in diabetic nephropathy." Acta Diabetologica 29, no. 3-4 (1992): 149–55. http://dx.doi.org/10.1007/bf00573480.

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Maccari, Francesca, Dealba Gheduzzi, and Nicola Volpi. "Anomalous Structure of Urinary Glycosaminoglycans in Patients with Pseudoxanthoma Elasticum." Clinical Chemistry 49, no. 3 (March 1, 2003): 380–88. http://dx.doi.org/10.1373/49.3.380.

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Abstract Background: Pseudoxanthoma elasticum (PXE) is a hereditary connective tissue disease in which proteoglycans have altered properties. We investigated whether altered proteoglycan metabolism occurs in vivo and may be reflected in the urine of PXE individuals by analyzing the excreted polysaccharides. Methods: We measured sulfated glycosaminoglycans in the urine of 10 PXE-affected patients, 12 healthy carriers, and 20 healthy controls by agarose gel electrophoresis. Chondroitin sulfate and heparan sulfate disaccharides were also quantified by treatment with specific lyases and separation

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Hansen, Ch, A. Irmscher, K. Kuhlemann, J. Beyer, and G. Kahaly. "Insulin-Dependent Diabetes Mellitus and Glycosaminoglycans." Hormone and Metabolic Research 27, no. 12 (December 1995): 555–58. http://dx.doi.org/10.1055/s-2007-980024.

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Ysart, G. E., and R. M. Mason. "Serum factors, growth factors and UDP-sugar metabolism in bovine articular cartilage chondrocytes." Biochemical Journal 303, no. 3 (November 1, 1994): 713–21. http://dx.doi.org/10.1042/bj3030713.

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1. The effect of different batches of fetal bovine serum and of growth factors on [35S]sulphate incorporation into glycosaminoglycans and on UDP-sugar pools in explant cultures of bovine articular cartilage was investigated. 2. [35S]Sulphate incorporation was variably stimulated between 1.2- and 3.5-fold by four different batches of serum. The UDP-glucuronate pool size expanded 4.3-6.5-fold in the presence of serum, even in those cultures in which little stimulation of [35S]sulphate incorporation occurred. The UDP-N-acetylhexosamine and UDP-hexose pools expanded by about 1.5- and 2.0-fold resp

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Amendum, Paige, Shaukat Khan, Seiji Yamaguchi, Hironori Kobayashi, Yasuhiko Ago, Yasuyuki Suzuki, Betul Celik, et al. "Glycosaminoglycans as Biomarkers for Mucopolysaccharidoses and Other Disorders." Diagnostics 11, no. 9 (August 28, 2021): 1563. http://dx.doi.org/10.3390/diagnostics11091563.

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Glycosaminoglycans (GAGs) are present in proteoglycans, which play critical physiological roles in various tissues. They are known to be elevated in mucopolysaccharidoses (MPS), a group of rare inherited metabolic diseases in which the lysosomal enzyme required to break down one or more GAG is deficient. In a previous study, we found elevation of GAGs in a subset of patients without MPS. In the current study, we aim to investigate serum GAG levels in patients with conditions beyond MPS. In our investigated samples, the largest group of patients had a clinical diagnosis of viral or non-viral en

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Kubaski, Francyne, Harumi Osago, Robert W. Mason, Seiji Yamaguchi, Hironori Kobayashi, Mikako Tsuchiya, Tadao Orii, and Shunji Tomatsu. "Glycosaminoglycans detection methods: Applications of mass spectrometry." Molecular Genetics and Metabolism 120, no. 1-2 (January 2017): 67–77. http://dx.doi.org/10.1016/j.ymgme.2016.09.005.

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Karlinsky, J. B., P. J. Bucay, D. E. Ciccolella, and M. P. Crowley. "Effects of intratracheal endotoxin administration on hamster lung glycosaminoglycans." American Journal of Physiology-Lung Cellular and Molecular Physiology 261, no. 2 (August 1, 1991): L148—L155. http://dx.doi.org/10.1152/ajplung.1991.261.2.l148.

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Incorporation of [3H]glucosamine and 35S into glycosaminoglycan (GAG) was measured in hamster lung explant cultures at 0, 1, 4, and 24 h after a single endotracheal instillation of Escherichia coli endotoxin. Lung content of GAG was measured in a second group of treated animals over an 8-day period. Albumin was detected after endotoxin treatment in bronchoalveolar lavage fluid at 24 h but was not found in lavage fluid 7 days later or in lavage fluid of saline-treated animals. Over the initial 24 h, increasing amounts of radiolabeled precursor molecules were incorporated into all classes of GAG

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Koźma, E. M., K. Olczyk, A. Głowacki, K. Komosińska, P. Sonecki, T. Najmiec, and M. Jaźwiec. "Glycosaminoglycans of human serum and their alterations in diabetes mellitus." Acta Biochimica Polonica 43, no. 3 (September 30, 1996): 567–74. http://dx.doi.org/10.18388/abp.1996_4492.

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Human serum contains several glycosaminoglycans (GAGs), mainly chondroitin sulphates and significantly less of heparan sulphate + heparin and dermatan sulphate. The non-insulin-dependent diabetes mellitus (with vascular complications) was associated with a significant increase in total serum GAG concentration, mainly of chondroitin sulphates and dermatan sulphate, with a simultaneous decrease in heparan sulphate + heparin level. These alterations were much more evident in patients with poor metabolic control. Hyaluronic acid (undetectable in healthy subjects and in patients with good metabolic

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Wu, V. Y., B. Wilson, and M. P. Cohen. "Disturbances in Glomerular Basement Membrane Glycosaminoglycans in Experimental Diabetes." Diabetes 36, no. 6 (June 1, 1987): 679–83. http://dx.doi.org/10.2337/diab.36.6.679.

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Wu, V. Y., B. Wilson, and M. P. Cohen. "Disturbances in glomerular basement membrane glycosaminoglycans in experimental diabetes." Diabetes 36, no. 6 (June 1, 1987): 679–83. http://dx.doi.org/10.2337/diabetes.36.6.679.

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Hill, D. J., A. Logan, M. McGarry та D. De Sousa. "Control of protein and matrix-molecule synthesis in isolated ovine fetal growth-plate chondrocytes by the interactions of basic fibroblast growth factor, insulin-like growth factors-I and -II, insulin and transforming growth factor-β1". Journal of Endocrinology 133, № 3 (червень 1992): 363–73. http://dx.doi.org/10.1677/joe.0.1330363.

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ABSTRACT Chondrogenesis is thought to be controlled by interactions between circulating anabolic hormones and locally produced peptide growth factors, and involves ordered changes in matrix composition which ultimately allow endochondral calcification. We have used a model of isolated ovine fetal growth-plate chondrocytes to examine the actions and interactions of basic fibroblast growth factor (basic FGF), insulin-like growth factors-I and -II (IGF-I and -II), insulin and transforming growth factor-β1 (TGF-β1) on total protein, collagen or non-collagenous protein and sulphated glycosaminoglyc

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Kawakami, Yutaka, Hiromi Oku, Kazuharu Nomura, Shigeaki Gorie, and Hiromi Ohta. "Metabolism of a Glycosaminoglycan during Metamorphosis in the Japanese Conger eel,Conger myriaster." Research Letters in Biochemistry 2009 (2009): 1–5. http://dx.doi.org/10.1155/2009/251731.

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Hyaluronan (HA) is a linear polysaccharide of high molecular weight that exists as a component of the extracellular matrix. The larvae (leptocephali) of the Japanese conger eel (Anguilliformes:Conger myriaster) have high levels of hyaluronan (HA) which is thought to help control body water content. We isolated glycosaminoglycans (GAGs) from Japanese conger eel leptocephali and measured the changes in tissue HA content during metamorphosis. HA content decreased during metamorphosis. In contrast, neutral sugar content increased during metamorphosis. We hypothesize that the leptocephali utilize a

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