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Auswahl der wissenschaftlichen Literatur zum Thema „Lungs Inflammation“
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Zeitschriftenartikel zum Thema "Lungs Inflammation"
Kumar, Rajiv. „SARS-CoV-2, Inflammation, Allergy of the Lungs and Nanotherapeutics“. International Journal of Clinical Case Reports and Reviews 11, Nr. 1 (04.04.2022): 01–02. http://dx.doi.org/10.31579/2690-4861/208.
Der volle Inhalt der QuelleLe, Nguyen Phuong Khanh, Shankaramurthy Channabasappa, Mokarram Hossain, Lixin Liu und Baljit Singh. „Leukocyte-specific protein 1 regulates neutrophil recruitment in acute lung inflammation“. American Journal of Physiology-Lung Cellular and Molecular Physiology 309, Nr. 9 (01.11.2015): L995—L1008. http://dx.doi.org/10.1152/ajplung.00068.2014.
Der volle Inhalt der QuelleLorenzo, Erica, Jacob Hopkins, Julie Lefebvre und Laura Haynes. „Vaccination does not protect aged mice from influenza-induced lung inflammation (VAC9P.1062)“. Journal of Immunology 194, Nr. 1_Supplement (01.05.2015): 145.2. http://dx.doi.org/10.4049/jimmunol.194.supp.145.2.
Der volle Inhalt der QuelleSmall, Donna M., Ryan R. Brown, Declan F. Doherty, Anthony Abladey, Zhe Zhou-Suckow, Rebecca J. Delaney, Lauren Kerrigan et al. „Targeting of cathepsin S reduces cystic fibrosis-like lung disease“. European Respiratory Journal 53, Nr. 3 (17.01.2019): 1801523. http://dx.doi.org/10.1183/13993003.01523-2018.
Der volle Inhalt der QuelleRamos-Ramírez, Patricia, Carina Malmhäll, Kristina Johansson, Mikael Adner, Jan Lötvall und Apostolos Bossios. „Lung Regulatory T Cells Express Adiponectin Receptor 1: Modulation by Obesity and Airway Allergic Inflammation“. International Journal of Molecular Sciences 21, Nr. 23 (26.11.2020): 8990. http://dx.doi.org/10.3390/ijms21238990.
Der volle Inhalt der QuelleChapoval, Svetlana P., Ann E. Kelly-Welch, Elizabeth Smith und Achsah D. Keegan. „Complex role of STAT6 in allergic airway inflammation (39.11)“. Journal of Immunology 178, Nr. 1_Supplement (01.04.2007): S27. http://dx.doi.org/10.4049/jimmunol.178.supp.39.11.
Der volle Inhalt der QuelleHerbein, Joel F., und Jo Rae Wright. „Enhanced clearance of surfactant protein D during LPS-induced acute inflammation in rat lung“. American Journal of Physiology-Lung Cellular and Molecular Physiology 281, Nr. 1 (01.07.2001): L268—L277. http://dx.doi.org/10.1152/ajplung.2001.281.1.l268.
Der volle Inhalt der QuelleAhn, So Yoon, Dong Kyung Sung, Yun Sil Chang und Won Soon Park. „Intratracheal Transplantation of Mesenchymal Stem Cells Attenuates Hyperoxia-Induced Microbial Dysbiosis in the Lungs, Brain, and Gut in Newborn Rats“. International Journal of Molecular Sciences 23, Nr. 12 (13.06.2022): 6601. http://dx.doi.org/10.3390/ijms23126601.
Der volle Inhalt der QuelleMarín-Corral, Judith, Leticia Martínez-Caro, José A. Lorente, Marta de Paula, Lara Pijuan, Nicolas Nin, Joaquim Gea, Andrés Esteban und Esther Barreiro. „Redox Balance and Cellular Inflammation in the Diaphragm, Limb Muscles, and Lungs of Mechanically Ventilated Rats“. Anesthesiology 112, Nr. 2 (01.02.2010): 384–94. http://dx.doi.org/10.1097/aln.0b013e3181c38bed.
Der volle Inhalt der QuelleBai, Jing, Shi-Lin Qiu, Xiao-Ning Zhong, Qiu-Ping Huang, Zhi-Yi He, Jian-Quan Zhang, Guang-Nan Liu, Mei-Hua Li und Jing-Min Deng. „Erythromycin EnhancesCD4+Foxp3+Regulatory T-Cell Responses in a Rat Model of Smoke-Induced Lung Inflammation“. Mediators of Inflammation 2012 (2012): 1–9. http://dx.doi.org/10.1155/2012/410232.
Der volle Inhalt der QuelleDissertationen zum Thema "Lungs Inflammation"
McLennan, Geoffrey. „Oxygen toxicity and radiation injury to the pulmonary system“. Title page, index and forward only, 1997. http://web4.library.adelaide.edu.au/theses/09PH/09phm164.pdf.
Der volle Inhalt der QuelleCorsino, Betsy Ann 1962. „THE PULMONARY RESPONSE INDUCED BY GLASS FIBERS (INFLAMMATION, SILICOSIS, MURINE MODEL)“. Thesis, The University of Arizona, 1986. http://hdl.handle.net/10150/291468.
Der volle Inhalt der QuelleDokka, Sujatha. „IL-10 gene therapy for the treatment of pulmonary inflammation“. Morgantown, W. Va. : [West Virginia University Libraries], 2000. http://etd.wvu.edu/templates/showETD.cfm?recnum=1421.
Der volle Inhalt der QuelleTitle from document title page. Document formatted into pages; contains ix, 132 p. : ill. (some col.) Vita. Includes abstract. Includes bibliographical references.
Finlay, Alison. „Kinetics of pulmonary eosinophilia in a mouse model“. Thesis, University of York, 1998. http://ethos.bl.uk/OrderDetails.do?uin=uk.bl.ethos.245971.
Der volle Inhalt der QuelleKarandashova, Sophia. „The Role of Ceramide in Neutrophil Elastase Induced Inflammation in the Lungs“. VCU Scholars Compass, 2018. https://scholarscompass.vcu.edu/etd/5468.
Der volle Inhalt der QuelleMcDaniel, Dylan K. „Characterization of Biomedical and Incidental Nanoparticles in the Lungs and Their Effects on Health“. Diss., Virginia Tech, 2018. http://hdl.handle.net/10919/86128.
Der volle Inhalt der QuellePh. D.
Over the years, nanoparticles have become more common in medicine, technology, and engineering due to their unique properties. Many of these properties allow for increased interactions with biological materials. Organs such as the lungs are at increased risk of exposure because they naturally encounter microorganisms and airborne particles on a daily basis. However, the lungs are also a highly desirable site for drug delivery using nanoparticles, due to ease of access. Inflammatory diseases such as asthma and emphysema could potentially benefit from nanoparticle-mediated delivery. Additionally, harmful nanoparticles can enter the lungs and cause or even exacerbate these diseases. Unfortunately, there is a lack of knowledge pertaining to this subject. Our work focused on assessing the interactions of nanoparticles in the lungs. First, we looked at nanoparticles that could be used for drug delivery. We found that fluorescentlylabeled nanoparticles were taken up by phagocytic white blood cells called macrophages. Furthermore, these particles did not induce cell death or inflammation in the lungs. Therefore, we found that these particles could be useful for drug delivery in the lungs. Secondly, we investigated potentially harmful nanoparticles and their effects on the lungs. The titanium-based particles called Magnéli phases, have been shown to be produced through coal burning. We found that while these particles are non-inflammatory in the lungs, they do lead to programmed death of macrophages as well as the increase in genes associated with fibrosis. Ultimately these particles led to a decrease in lung function after long-term exposure.
Zheng, Ling 1958. „Airway inflammation and remodelling post human lung transplantation“. Monash University, Dept. of Medicine, 2002. http://arrow.monash.edu.au/hdl/1959.1/8099.
Der volle Inhalt der QuelleLewis, Joshua B. „Alterations in Tight Junctional Proteins and Their Effects on Pulmonary Inflammation“. BYU ScholarsArchive, 2017. https://scholarsarchive.byu.edu/etd/6308.
Der volle Inhalt der QuelleLau, Kwok-wai, und 劉國威. „The involvement of serotoninergic system in cigarette smoke-induced oxidative stress and inflammation: relevantto chronic obstructive pulmonary disease“. Thesis, The University of Hong Kong (Pokfulam, Hong Kong), 2012. http://hub.hku.hk/bib/B47869616.
Der volle Inhalt der Quellepublished_or_final_version
Medicine
Doctoral
Doctor of Philosophy
McNamara, Tracy Renee. „Chlamydia pneumoniae and airways inflammation : an investigation of the host cell-pathogen relationship /“. Title page, table of contents and abstract only, 2004. http://web4.library.adelaide.edu.au/theses/09PH/09phm4791.pdf.
Der volle Inhalt der QuelleBücher zum Thema "Lungs Inflammation"
1940-, Olivieri D., und Bianco S, Hrsg. Airway obstruction and inflammation: Present status and perspectives. Basel: Karger, 1990.
Den vollen Inhalt der Quelle findenCriner, Gerard J., William D. Cornwell und Thomas J. Rogers. Smoking and lung inflammation: Basic, pre-clinical, and clinical research advances. New York: Springer, 2013.
Den vollen Inhalt der Quelle findenL, Kradin Richard, und Robinson Bruce W. S, Hrsg. Immunopathology of lung disease. Boston: Butterworth-Heinemann, 1996.
Den vollen Inhalt der Quelle findenBertolini, Renzo. Animal and vegetable dusts as a cause of deep lung inflammation. Hamilton: CCOHS, 1988.
Den vollen Inhalt der Quelle findenMichel, Chignard, und Conference on Cytokines and Adhesion Molecules in Lung Inflammation (1995 : Paris, France), Hrsg. Cytokines and adhesion molecules in lung inflammation. New York: New York Academy of Sciences, 1996.
Den vollen Inhalt der Quelle findenF, Donner C., Hrsg. COPD is/is not a systemic disease? Hauppauge, NY: Nova Science, 2009.
Den vollen Inhalt der Quelle finden1952-, Strieter Robert M., Kunkel S. L und Standiford Theodore J, Hrsg. Chemokines in the lung. New York: Marcel Dekker, Inc., 2003.
Den vollen Inhalt der Quelle findenRogers, Thomas J., Gerard J. Criner und William D. Cornwell, Hrsg. Smoking and Lung Inflammation. New York, NY: Springer New York, 2013. http://dx.doi.org/10.1007/978-1-4614-7351-0.
Der volle Inhalt der QuelleDubinett, Steven M., Hrsg. Inflammation and Lung Cancer. New York, NY: Springer New York, 2015. http://dx.doi.org/10.1007/978-1-4939-2724-1.
Der volle Inhalt der QuelleAlper, Scott, und William J. Janssen, Hrsg. Lung Innate Immunity and Inflammation. New York, NY: Springer New York, 2018. http://dx.doi.org/10.1007/978-1-4939-8570-8.
Der volle Inhalt der QuelleBuchteile zum Thema "Lungs Inflammation"
Marleau, S., C. Fortin und P. Borgeat. „In Vivo Desensitization to LTB4-Induced Neutrophil Sequestration in Rabbit Lungs“. In Eicosanoids and Other Bioactive Lipids in Cancer, Inflammation and Radiation Injury, 261–63. Boston, MA: Springer US, 1993. http://dx.doi.org/10.1007/978-1-4615-3520-1_51.
Der volle Inhalt der QuelleUlges, Alexander, Edgar Schmitt, Tobias Bopp und Matthias Klein. „Messenger RNA Sequencing of Rare Cell Populations in the Lung and Lung-Draining Lymph Nodes“. In Inflammation, 199–219. New York, NY: Springer New York, 2017. http://dx.doi.org/10.1007/978-1-4939-6786-5_14.
Der volle Inhalt der QuelleSharma, Sherven, Siwen Hu-Lieskovan, Steven M. Dubinett und Jay Moon Lee. „Inflammation and Lung Cancer: Addressing Inflammation with Immunotherapy“. In Inflammation and Lung Cancer, 191–209. New York, NY: Springer New York, 2015. http://dx.doi.org/10.1007/978-1-4939-2724-1_7.
Der volle Inhalt der QuellePorsbjerg, Celeste, und Unnur Björnsdóttir. „Biomarkers of eosinophilic inflammation“. In Eosinophilic Lung Diseases, 37–50. Sheffield, United Kingdom: European Respiratory Society, 2022. http://dx.doi.org/10.1183/2312508x.10029520.
Der volle Inhalt der QuelleKennedy, Paul A., und Laurie E. Kilpatrick. „Neutrophil Inflammation in COPD“. In Smoking and Lung Inflammation, 59–79. New York, NY: Springer New York, 2013. http://dx.doi.org/10.1007/978-1-4614-7351-0_3.
Der volle Inhalt der QuelleHoughton, A. McGarry, und Steven D. Shapiro. „Inflammation and Lung Cancer: The Relationship to Chronic Obstructive Pulmonary Disease“. In Inflammation and Lung Cancer, 1–21. New York, NY: Springer New York, 2015. http://dx.doi.org/10.1007/978-1-4939-2724-1_1.
Der volle Inhalt der QuelleWalser, Tonya C., Stacy J. Park, Jane Yanagawa und Steven M. Dubinett. „Inflammation and Lung Cancer: The Role of Epithelial–Mesenchymal Transition“. In Inflammation and Lung Cancer, 23–68. New York, NY: Springer New York, 2015. http://dx.doi.org/10.1007/978-1-4939-2724-1_2.
Der volle Inhalt der QuelleKadara, Humam, Wilbur A. Franklin und Ignacio I. Wistuba. „Inflammation and Lung Cancer: Molecular Pathology“. In Inflammation and Lung Cancer, 69–93. New York, NY: Springer New York, 2015. http://dx.doi.org/10.1007/978-1-4939-2724-1_3.
Der volle Inhalt der QuelleBauer, Alison K., York E. Miller und Robert L. Keith. „Inflammation and Lung Cancer: Prevention“. In Inflammation and Lung Cancer, 95–136. New York, NY: Springer New York, 2015. http://dx.doi.org/10.1007/978-1-4939-2724-1_4.
Der volle Inhalt der QuelleEl Rayes, Tina, Raul Catena, Shahin Rafii, Nasser Altorki und Vivek Mittal. „Inflammation and Lung Cancer: The Link to Angiogenesis“. In Inflammation and Lung Cancer, 137–59. New York, NY: Springer New York, 2015. http://dx.doi.org/10.1007/978-1-4939-2724-1_5.
Der volle Inhalt der QuelleKonferenzberichte zum Thema "Lungs Inflammation"
Kuroda, E., und KJ Ishii. „1707c Inhaled fine particles induce allergic inflammation in the lungs“. In 32nd Triennial Congress of the International Commission on Occupational Health (ICOH), Dublin, Ireland, 29th April to 4th May 2018. BMJ Publishing Group Ltd, 2018. http://dx.doi.org/10.1136/oemed-2018-icohabstracts.145.
Der volle Inhalt der QuelleTorres-Gonzalez, Edilson, Jeffrey D. Ritzenthaler und Jesse Roman-Rodriguez. „Modulation Of Inflammation By Mid-Cervical Vagotomy In Murine Lungs“. In American Thoracic Society 2012 International Conference, May 18-23, 2012 • San Francisco, California. American Thoracic Society, 2012. http://dx.doi.org/10.1164/ajrccm-conference.2012.185.1_meetingabstracts.a5131.
Der volle Inhalt der QuelleWall, Wolfgang A., Andrew Comerford, Lena Wiechert und Sophie Rausch. „Coupled and Multi-Scale Building Blocks for a Comprehensive Computational Lung Model“. In ASME 2009 Summer Bioengineering Conference. American Society of Mechanical Engineers, 2009. http://dx.doi.org/10.1115/sbc2009-206407.
Der volle Inhalt der QuelleShirai, Atsushi, und Toshiyuki Hayase. „Effect of Neutrophils Retention Time in Capillaries on Increase in Their Concentration in a Lattice Capillary Network Model“. In ASME 2007 Summer Bioengineering Conference. American Society of Mechanical Engineers, 2007. http://dx.doi.org/10.1115/sbc2007-176717.
Der volle Inhalt der QuelleGlasser, Stephan W., Melissa D. Maxfield, Teah L. Witt, John E. Baatz, Henry T. Akinbi und Tom Korfhagen. „LPS Exposure Increases Inflammation In The Lungs Of SP-C Deficient Mice“. In American Thoracic Society 2010 International Conference, May 14-19, 2010 • New Orleans. American Thoracic Society, 2010. http://dx.doi.org/10.1164/ajrccm-conference.2010.181.1_meetingabstracts.a2454.
Der volle Inhalt der QuelleOdom, C., Y. Kim, F. T. Korkmaz, E. Na, L. Baird, M. R. Jones, J. P. Mizgerd, K. Traber und L. J. Quinton. „Liver Activity Reprograms the Lungs During Systemic Inflammation to Fortify Pulmonary Defense“. In American Thoracic Society 2021 International Conference, May 14-19, 2021 - San Diego, CA. American Thoracic Society, 2021. http://dx.doi.org/10.1164/ajrccm-conference.2021.203.1_meetingabstracts.a1262.
Der volle Inhalt der QuelleZhou, Haiying, Shawn He, Sean Gunsten, Steven Brody, Walter Akers und Mikhail Y. Berezin. „NIR fluorescent contrast agents for detection of inflammation of lungs in vivo“. In CLEO: Applications and Technology. Washington, D.C.: OSA, 2014. http://dx.doi.org/10.1364/cleo_at.2014.am2p.1.
Der volle Inhalt der QuelleChan, Y. L., B. Wang, H. Chen, K. F. Ho und B. Oliver. „Impact of Traffic Related Air Pollutant Exposure on Lung Inflammation and Mitochondrial Wellbeing in Mouse Lungs“. In American Thoracic Society 2019 International Conference, May 17-22, 2019 - Dallas, TX. American Thoracic Society, 2019. http://dx.doi.org/10.1164/ajrccm-conference.2019.199.1_meetingabstracts.a1832.
Der volle Inhalt der QuelleTitova, Olga, Natalya Kuzubova, Elena Lebedeva, Tatiana Preobrajenskaya und Elizaveta Volchkova. „Influence of induced immunosupression on inflammation and lungs remodeling on the COPD model“. In ERS International Congress 2019 abstracts. European Respiratory Society, 2019. http://dx.doi.org/10.1183/13993003.congress-2019.pa3853.
Der volle Inhalt der QuelleHamburg, Brian, Mei Hulver, Zeyu Xiong, Jeffrey Isenberg und Janet S. Lee. „A Lack Of Thrombospondin-1 Predisposes The Lungs To Inflammation Following Exposure To Endotoxin“. In American Thoracic Society 2011 International Conference, May 13-18, 2011 • Denver Colorado. American Thoracic Society, 2011. http://dx.doi.org/10.1164/ajrccm-conference.2011.183.1_meetingabstracts.a1087.
Der volle Inhalt der QuelleBerichte der Organisationen zum Thema "Lungs Inflammation"
Lin, Hongwei, Yanjun Gao, Kang Sun und Faguang Jin. Association between PM2.5 pollution and outpatient visits for respiratory diseases in China: a systematic review and meta-analysis. INPLASY - International Platform of Registered Systematic Review and Meta-analysis Protocols, Mai 2022. http://dx.doi.org/10.37766/inplasy2022.5.0144.
Der volle Inhalt der QuelleBurgess, Jefferey L. Acute Lung Injury Following Smoke Inhalation: Predictive Value of Sputum Biomarkers and Time Course of Lung Inflammation. Fort Belvoir, VA: Defense Technical Information Center, Mai 2005. http://dx.doi.org/10.21236/ada446886.
Der volle Inhalt der QuelleBurgess, Jefferey L. Acute Lung Injury Following Smoke Inhalation: Predictive Value of Sputum Biomarkers and Time Course of Lung Inflammation. Fort Belvoir, VA: Defense Technical Information Center, Mai 2004. http://dx.doi.org/10.21236/ada424038.
Der volle Inhalt der QuelleBurgess, Jefferey L. Acute Lung Injury Following Smoke Inhalation: Predictive Value of Sputum Biomarkers and Time Course of Lung Inflammation. Fort Belvoir, VA: Defense Technical Information Center, Mai 2007. http://dx.doi.org/10.21236/ada472079.
Der volle Inhalt der QuelleSun, Muzhen, Mei Cao, Yulian Zhang und Xipeng Kang. Systemic immune inflammation index is a prognostic factor for small cell lung cancer: a systematic review and meta-analysis. INPLASY - International Platform of Registered Systematic Review and Meta-analysis Protocols, Dezember 2021. http://dx.doi.org/10.37766/inplasy2021.12.0083.
Der volle Inhalt der QuelleCzerwaty, Katarzyna, Karolina Dżaman, Krystyna Maria Sobczyk und Katarzyna Irmina Sikrorska. The Overlap Syndrome of Obstructive Sleep Apnea and Chronic Obstructive Pulmonary Disease: A Systematic Review. INPLASY - International Platform of Registered Systematic Review and Meta-analysis Protocols, November 2022. http://dx.doi.org/10.37766/inplasy2022.11.0077.
Der volle Inhalt der QuelleMa, He, Jifu Zhao und Zhilei Wang. Efficacy and safety of HuaYu TongFu Method combined with acupuncture in the treatment of Acute Exacerbation of Chronic Obstructive Pulmonary Disease:A protocol for systematic review and meta-analysis. INPLASY - International Platform of Registered Systematic Review and Meta-analysis Protocols, September 2022. http://dx.doi.org/10.37766/inplasy2022.9.0114.
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