Thematische Bibliographien / Lungs Pathophysiology

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Auswahl der wissenschaftlichen Literatur zum Thema „Lungs Pathophysiology“

Autor: Grafiati

Veröffentlicht am 4. Juni 2021

Zuletzt aktualisiert am 28. Januar 2023

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Zeitschriftenartikel zum Thema "Lungs Pathophysiology"

Bland, Richard D. "Pathophysiology of Neonatal Lung Injury." International Journal of Technology Assessment in Health Care 7, S1 (January 1991): 56–60. http://dx.doi.org/10.1017/s0266462300012514.

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Respiratory distress in newborn and young infants often develops as a result of acute lung injury, in which disruption of the normal barrier function of the pulmonary endothelium and epithelium causes protein-rich interstitial and alveolar edema. Several conditions may initiate acute lung injury, including aspiration of meconium or gastric contents, bacterial or viral infection, overzealous resuscitation, and birth associated with incomplete lung development that requires ventilatory support with positivepressure mechanical ventilation and high concentrations of inspired oxygen. The latter con

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Sundar, Isaac K., Hongwei Yao, Michael T. Sellix, and Irfan Rahman. "Circadian molecular clock in lung pathophysiology." American Journal of Physiology-Lung Cellular and Molecular Physiology 309, no. 10 (November 15, 2015): L1056—L1075. http://dx.doi.org/10.1152/ajplung.00152.2015.

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Disrupted daily or circadian rhythms of lung function and inflammatory responses are common features of chronic airway diseases. At the molecular level these circadian rhythms depend on the activity of an autoregulatory feedback loop oscillator of clock gene transcription factors, including the BMAL1:CLOCK activator complex and the repressors PERIOD and CRYPTOCHROME. The key nuclear receptors and transcription factors REV-ERBα and RORα regulate Bmal1 expression and provide stability to the oscillator. Circadian clock dysfunction is implicated in both immune and inflammatory responses to enviro

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van Zanden, Judith E., Henri G. D. Leuvenink, Erik A. M. Verschuuren, Michiel E. Erasmus, and Maximilia C. Hottenrott. "A translational rat model for ex vivo lung perfusion of pre-injured lungs after brain death." PLOS ONE 16, no. 12 (December 2, 2021): e0260705. http://dx.doi.org/10.1371/journal.pone.0260705.

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The process of brain death (BD) detrimentally affects donor lung quality. Ex vivo lung perfusion (EVLP) is a technique originally designed to evaluate marginal donor lungs. Nowadays, its potential as a treatment platform to repair damaged donor lungs is increasingly studied in experimental models. Rat models for EVLP have been described in literature before, yet the pathophysiology of BD was not included in these protocols and prolonged perfusion over 3 hours without anti-inflammatory additives was not achieved. We aimed to establish a model for prolonged EVLP of rat lungs from brain-dead dono

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Levvey, Bronwyn, Kovi Levin, Miranda Paraskeva, Glen Westall, and Gregory Snell. "Donation after Brain Death versus Donation after Circulatory Death: Lung Donor Management Issues." Seminars in Respiratory and Critical Care Medicine 39, no. 02 (March 26, 2018): 138–47. http://dx.doi.org/10.1055/s-0037-1615820.

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AbstractLung transplantation (LTx) has traditionally been limited by a lack of suitable donor lungs. With the recognition that lungs are more robust than initially thought, the size of the donor pool of available lungs has increased dramatically in the past decade. Donation after brain death (DBD) and donation after circulatory death (DCD) lungs, both ideal and extended are now routinely utilized. DBD lungs can be damaged. There are important differences in the public's understanding, legal and consent processes, intensive care unit strategies, lung pathophysiology, logistics, and potential-to

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Naramala, Srikanth, Sharmi Biswas, Sreedhar Adapa, Vijay Gayam, Romeo C. Castillo, Srinadh Annangi, and Venu Madhav Konala. "Pleomorphic Pulmonary Manifestations of IgG4-Related Disease." Case Reports in Rheumatology 2019 (August 20, 2019): 1–4. http://dx.doi.org/10.1155/2019/7572869.

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Immunoglobulin G4-related disease (IgG4-RD) is a fibroinflammatory disorder which has been first reported in 2001 by Hamano and colleagues in a patient with autoimmune sclerosing pancreatitis. Almost every organ in the human body can be affected by IgG4-RD from infiltration with IgG4-positive plasma cells. Involvement of lungs with IgG4 is reported previously, but still, there is no clear picture of the pathophysiology behind lung involvement. Here, we are presenting a patient who has IgG4-RD presenting as pseudotumor of the lungs.

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Agraval, Hina, and Hong Wei Chu. "Lung Organoids in Smoking Research: Current Advances and Future Promises." Biomolecules 12, no. 10 (October 12, 2022): 1463. http://dx.doi.org/10.3390/biom12101463.

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Tobacco smoking has been established to contribute to the pathogenesis of various respiratory diseases including chronic obstructive pulmonary disease (COPD), lung cancer, and asthma. However, major hurdles in mechanistic studies on the role of smoking in human lungs remain in part due to the lack of ex vivo experimental models and ambiguous data from animal models that can best recapitulate the architecture and pathophysiology of the human lung. Recent development of the lung organoid culture system has opened new avenues for respiratory disease research as organoids are proving to be a sophi

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Das, Mita, W. Michael Zawada, James West, and Kurt R. Stenmark. "JNK2 regulates vascular remodeling in pulmonary hypertension." Pulmonary Circulation 8, no. 3 (May 2, 2018): 204589401877815. http://dx.doi.org/10.1177/2045894018778156.

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Pulmonary arterial (PA) wall modifications are key pathological features of pulmonary hypertension (PH). Although such abnormalities correlate with heightened phosphorylation of c-Jun N-terminal kinases 1/2 (JNK1/2) in a rat model of PH, the contribution of specific JNK isoforms to the pathophysiology of PH is unknown. Hence, we hypothesized that activation of either one, or both JNK isoforms regulates PA remodeling in PH. We detected increased JNK1/2 phosphorylation in the thickened vessels of PH patients’ lungs compared to that in lungs of healthy individuals. JNK1/2 phosphorylation parallel

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Frétaud, Maxence, Delphyne Descamps, Daphné Laubreton, Marie-Anne Rameix-Welti, Jean-François Eléouët, Thibaut Larcher, Marie Galloux, and Christelle Langevin. "New Look at RSV Infection: Tissue Clearing and 3D Imaging of the Entire Mouse Lung at Cellular Resolution." Viruses 13, no. 2 (January 28, 2021): 201. http://dx.doi.org/10.3390/v13020201.

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Background: Respiratory Syncytial Virus (RSV) is the major cause of severe acute respiratory tract illness in young children worldwide and a main pathogen for the elderly and immune-compromised people. In the absence of vaccines or effective treatments, a better characterization of the pathogenesis of RSV infection is required. To date, the pathophysiology of the disease and its diagnosis has mostly relied on chest X-ray and genome detection in nasopharyngeal swabs. The development of new imaging approaches is instrumental to further the description of RSV spread, virus–host interactions and r

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Raredon, Micha Sam Brickman, Taylor Sterling Adams, Yasir Suhail, Jonas Christian Schupp, Sergio Poli, Nir Neumark, Katherine L. Leiby, et al. "Single-cell connectomic analysis of adult mammalian lungs." Science Advances 5, no. 12 (December 2019): eaaw3851. http://dx.doi.org/10.1126/sciadv.aaw3851.

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Efforts to decipher chronic lung disease and to reconstitute functional lung tissue through regenerative medicine have been hampered by an incomplete understanding of cell-cell interactions governing tissue homeostasis. Because the structure of mammalian lungs is highly conserved at the histologic level, we hypothesized that there are evolutionarily conserved homeostatic mechanisms that keep the fine architecture of the lung in balance. We have leveraged single-cell RNA sequencing techniques to identify conserved patterns of cell-cell cross-talk in adult mammalian lungs, analyzing mouse, rat,

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Meyerowitz, Glen, and Igor Barjaktarevic. "369 The impact of asymmetric lung injury on gas and pressures distribution in a mechanical ventilation model with implementation of compartmentalized inspiratory hold." Journal of Clinical and Translational Science 6, s1 (April 2022): 69. http://dx.doi.org/10.1017/cts.2022.209.

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OBJECTIVES/GOALS: Asymmetries in lung pathophysiology can result in a maldistribution of gas between regions of the lungs which may generate dangerous pressures that are not observable by clinicians. Our study aims to demonstrate and quantify this through use of high-fidelity simulators to represent a range of commonly encountered clinical pathologies. METHODS/STUDY POPULATION: A benchtop study was performed with two high-fidelity breathing simulators, each representing one lung. This system allows for real-time monitoring of pressure and lung dynamics in a two-lung asymmetric injury model. On

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Dissertationen zum Thema "Lungs Pathophysiology"

McLennan, Geoffrey. "Oxygen toxicity and radiation injury to the pulmonary system." Title page, index and forward only, 1997. http://web4.library.adelaide.edu.au/theses/09PH/09phm164.pdf.

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Bibliography: leaves 168-184. The work in this study encompasses oxygen free radical related inflammation in the peripheral lung and in lung cells. Animal and human studies have been used. Methods include cell culture with function studies, protein chemistry, animal and human physiology, and cell and lung structure through histopathology, and various forms of electron microscopy. The work resulting from this thesis has formed an important basis for understanding acute and chronic lung injury.

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McNamara, Tracy Renee. "Chlamydia pneumoniae and airways inflammation : an investigation of the host cell-pathogen relationship /." Title page, table of contents and abstract only, 2004. http://web4.library.adelaide.edu.au/theses/09PH/09phm4791.pdf.

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Chan, Ching Eunice, and 陳清. "Pathogenetic role of aberrant promoter methylation in lung cancer." Thesis, The University of Hong Kong (Pokfulam, Hong Kong), 2007. http://hub.hku.hk/bib/B39557819.

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Smedley, Jeremy Vance. "A Combined In Vivo and In Vitro Approach to the Study of Endotoxemia in Swine." Thesis, Virginia Tech, 2000. http://hdl.handle.net/10919/33947.

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The cardiopulmonary effects of endotoxin administration (1 microgram/kg) were evaluated in 8-10 week old SPF-derived Yorkshire pigs, both because endotoxemia is a common and important swine problem, and because the pig is a good model for human adult respiratory distress syndrome. Physiological changes included sustained increases in mean pulmonary artery pressure, pulmonary vascular resistance, pulmonary arterial wedge pressure, heart rate, hematocrit, and the arterial partial pressure of carbon dioxide. Transient increases were also observed in central venous pressure and airway pressure.

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Kerckx, Yannick. "Modeling nitric oxide production and transport in the human lung." Doctoral thesis, Universite Libre de Bruxelles, 2009. http://hdl.handle.net/2013/ULB-DIPOT:oai:dipot.ulb.ac.be:2013/210324.

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Le travail présenté ici porte sur l’étude de la production et du transport du monoxyde d’azote (NO) dans le poumon humain. Le NO est une molécule dont l’implication dans des processus physiologiques n’a été mis en évidence qu’en 1987. Depuis, il a été démontré que le NO joue de nombreux rôles dans le corps humain. Le NO est un gaz labile (instable) dans les conditions physiologiques, il diffuse très facilement au travers des parois et il a une grande affinité pour l’hémoglobine. La production du NO est liée à 3 isoformes différentes de la protéine appelées synthases du NO ou NO synthases.\<

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Bondue, Benjamin. "Role of chemerin and its receptor ChemR23 in the physiopathology of inflammatory lung diseases." Doctoral thesis, Universite Libre de Bruxelles, 2010. http://hdl.handle.net/2013/ULB-DIPOT:oai:dipot.ulb.ac.be:2013/209992.

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Chemoattractant agents play a crucial role in the initiation of immune responses, by regulating the traffic and function of leucocyte populations. Their receptors are therefore considered as potential targets for the development of new therapies in the fields of cancer and inflammatory diseases. ChemR23, a previously orphan receptor discovered in the laboratory, is structurally related to receptors for chemoattractant agents. It is expressed on immature myeloid and plasmacytoid dendritic cells (mDCs and pDCs respectively), as well as on adipocytes, macrophages, NK and endothelial cells. Chemer

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Vanderstocken, Gilles. "Caractérisation du rôle des nucléotides extracellulaires et du récepteur purinergique P2Y2 dans la physiopathologie des maladies pulmonaires inflammatoires." Doctoral thesis, Universite Libre de Bruxelles, 2012. http://hdl.handle.net/2013/ULB-DIPOT:oai:dipot.ulb.ac.be:2013/209591.

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Amongst respiratory diseases, inflammatory lung diseases constitute a major part of public health problem. As a consequence, investigating the immune mechanisms that contribute to the pathogenesis of these diseases is essential to identify candidate targets for the development of new therapeutic drugs. Furthermore, over the past 20 years, the growing awareness that purinergic signalling events shape the immune and inflammatory responses to infection and allergic reactions warranted the development of animal models to assess their importance in vivo in acute lung injury and ch

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Wang, Jianpu. "Pathophysiology and treatment of chlorine gas-induced lung injury : an experimental study in pigs /." Linköping : Univ, 2004. http://www.bibl.liu.se/liupubl/disp/disp2004/med877s.pdf.

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Tiev, Kiet Phong. "Rôle du monoxyde d'azote dans la physiopathologie des atteintes pulmonaires de la sclérodermie systémique." Thesis, Paris Est, 2008. http://www.theses.fr/2008PEST0081.

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La pneumopathie interstitielle (PI) est devenue la principale cause de décès de la sclérodermie systémique (ScS). Au cours de PI, l’activation immunitaire déclenche une forte production du monoxyde d’azote (NO) et l’augmentation de la concentration de NO dans l’air expiré des patients atteints de ScS avec PI suggère que cette méthode pourrait détecter précocement l’alvéolite, afin de traiter à temps la PI pour éviter son évolution vers la fibrose pulmonaire. En utilisant le modèle à deux compartiments séparant le NO alvéolaire (CANO) du NO bronchique, nous avons montré que la CANO est : (1) au

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Ridings, Philip Charles. "An investigation into the role of selectins in the pathophysiology of sepsis and sepsis-induced acute lung injury." Thesis, University of Southampton, 1996. http://ethos.bl.uk/OrderDetails.do?uin=uk.bl.ethos.266390.

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Bücher zum Thema "Lungs Pathophysiology"

Pulmonary pathophysiology: A clinical approach. 3rd ed. New York: McGraw-Hill Medical, 2010.

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Pulmonary pathophysiology. Philadelphia: Lippincott, 1995.

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G, Crystal Ronald, and West John B, eds. The Lung: Scientific foundations. New York: Raven Press, 1991.

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West, John B. (John Burnard), ed. Pulmonary pathophysiology: The essentials. 8th ed. Philadelphia: Wolters Kluwer/Lippincott Williams & Wilkins Health, 2012.

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Pulmonary pathophysiology: The essentials. 3rd ed. Baltimore: Williams & Wilkins, 1987.

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Pulmonary pathophysiology: The essentials. 7th ed. Philadelphia: Wolters Kluwer Health/Lippincott Williams & Wilkins, 2008.

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B, West John, ed. Pulmonary pathophysiology--the essentials. 5th ed. Baltimore, Md: Williams & Wilkins, 1998.

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B, West John, ed. Pulmonary pathophysiology--the essentials. 4th ed. Baltimore: Williams & Wilkins, 1992.

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Advances in surgical pathology: Lung cancer. Philadelphia: Wolters Kluwer Health/Lippincott Williams & Wilkins, 2011.

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Analytical lung pathology. Berlin: Springer-Verlag, 1992.

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Buchteile zum Thema "Lungs Pathophysiology"

Christian, Couture. "Lung cancer." In Applied Respiratory Pathophysiology, 207–22. Boca Raton : CRC Press, [2017]: CRC Press, 2017. http://dx.doi.org/10.1201/9781315177052-11.

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Geneviève, Dion, Cormier Yvon, and Boulet Louis-Philippe. "Interstitial lung diseases." In Applied Respiratory Pathophysiology, 177–205. Boca Raton : CRC Press, [2017]: CRC Press, 2017. http://dx.doi.org/10.1201/9781315177052-10.

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Henske, Elizabeth P., Souheil El-Chemaly, Thomas N. Darling, Angelo M. Taveira-DaSilva, and Joel Moss. "Pathophysiology of Lymphangioleiomyomatosis." In Diffuse Cystic Lung Diseases, 101–20. Cham: Springer International Publishing, 2021. http://dx.doi.org/10.1007/978-3-030-63365-3_5.

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Finsterer, U., and K. Peter. "Pathophysiologie thoraxchirurgischer Eingriffe." In Lunge und Mediastinum, 107–15. Berlin, Heidelberg: Springer Berlin Heidelberg, 1991. http://dx.doi.org/10.1007/978-3-662-08433-5_11.

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Kroegel, C., M. Mohorn, and P. R. Grahmann. "Pathophysiologie der Lunge." In Springer Lehrbuch, 201–22. Berlin, Heidelberg: Springer Berlin Heidelberg, 2000. http://dx.doi.org/10.1007/978-3-642-57115-2_14.

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Ward, Chris, Rhys Jones, Mellissa Friel, Eoin Hunt, and Des Murphy. "Pathophysiology in the Lung." In Reflux Aspiration and Lung Disease, 55–69. Cham: Springer International Publishing, 2018. http://dx.doi.org/10.1007/978-3-319-90525-9_5.

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Couture, Christian. "Embryology, anatomy, and histology of the lung." In Applied Respiratory Pathophysiology, 1–14. Boca Raton : CRC Press, [2017]: CRC Press, 2017. http://dx.doi.org/10.1201/9781315177052-1.

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Bhat, Javeed Ahmad, Nawab John Dar, and Wajid Waheed Bhat. "Asthma: Pathophysiology, Current Status, and Therapeutics." In Chronic Lung Diseases, 25–60. Singapore: Springer Singapore, 2020. http://dx.doi.org/10.1007/978-981-15-3734-9_2.

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Grossi, Adalberto, and PierPaolo Giomarelli. "Pathophysiology of adult RDS." In The Surfactant System of the Lung, 183–90. London: Macmillan Education UK, 1991. http://dx.doi.org/10.1007/978-1-349-12553-1_30.

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Johnston, Nikki. "The Pathophysiology of Gastroesophageal Reflux." In Gastroesophageal Reflux and the Lung, 23–41. New York, NY: Springer New York, 2012. http://dx.doi.org/10.1007/978-1-4614-5502-8_2.

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Konferenzberichte zum Thema "Lungs Pathophysiology"

Jagani, Jakin, and Alexandrina Untaroiu. "A Study of TCPC-Stent Conjunction for Cavopulmonary Assist in Fontan Patients With Right Ventricular Dysfunction." In ASME 2016 International Mechanical Engineering Congress and Exposition. American Society of Mechanical Engineers, 2016. http://dx.doi.org/10.1115/imece2016-68760.

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Mechanical circulatory support devices have gained significant importance in recent years as a viable therapeutic option to support paediatric population and children with single functional ventricle. The Fontan operation helps to reroute the deoxygenated blood to the lungs by bypassing the dysfunctional right ventricle. Total Cavopulmonary Connection (TCPC) is usually a method opted by the clinicians to connect the superior vena cava (SVC) and inferior vena cava (IVC) to the left and right pulmonary artery (LPA and RPA). However, the non-physiologic flow patterns created by the Fontan procedu

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Romem, Ayal, Karen Lammers, Aldo T. Iacono, Mohan E. Tulapurkar, Cinthia Dranchenberg, Jeffrey D. Hasday, and Alessio Fasano. "Zonulin - A Novel Player In Human Lung Pathophysiology." In American Thoracic Society 2012 International Conference, May 18-23, 2012 • San Francisco, California. American Thoracic Society, 2012. http://dx.doi.org/10.1164/ajrccm-conference.2012.185.1_meetingabstracts.a4266.

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Bruno, Nicoletta, Aran Singanayagam, Hugo A. Farne, Julia Aniscenko, Nicholas Glanville, Chloe J. Pyle, Dhiren F. Patel, Sebastian L. Johnston, and Robert J. Snelgrove. "G-CSF drives pathophysiology of RV-induced allergic asthma exacerbations by potentiating neutrophilic inflammation and ILC2 function." In ERS Lung Science Conference 2022 abstracts. European Respiratory Society, 2022. http://dx.doi.org/10.1183/23120541.lsc-2022.191.

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Torday, John S. "Evolutionary Biology As A Cipher For Lung Physiology And Pathophysiology." In American Thoracic Society 2010 International Conference, May 14-19, 2010 • New Orleans. American Thoracic Society, 2010. http://dx.doi.org/10.1164/ajrccm-conference.2010.181.1_meetingabstracts.a6416.

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Saha, P., S. Jain, I. Mukherjee, S. Durugkar, M. Das, S. Gokhale, S. S. Sohal, V. Naidu, and P. Sharma. "Effect of Long-Term Particulate Matter (PM10) Exposure on Lung Pathophysiology in Mice." In American Thoracic Society 2020 International Conference, May 15-20, 2020 - Philadelphia, PA. American Thoracic Society, 2020. http://dx.doi.org/10.1164/ajrccm-conference.2020.201.1_meetingabstracts.a2887.

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Vega-Sanchez, Angel Emmanuel, Espiridión Ramos-Martínez, Ivette Buendía-Roldan, Gloría Pérez-Rubio, Ramcés Falfán-Valencia, Mayra Mejía, and Jorge Rojas-Serrano. "Role of the inflammasome in the pathophysiology of antisynthetase-associated interstitial lung disease." In ERS International Congress 2021 abstracts. European Respiratory Society, 2021. http://dx.doi.org/10.1183/13993003.congress-2021.pa2377.

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Basu, S., and A. J. Halayko. "IGFBP3 Modulates Lung Pathophysiology in an Allergen -Specific Manner in Murine Models of Asthma." In American Thoracic Society 2020 International Conference, May 15-20, 2020 - Philadelphia, PA. American Thoracic Society, 2020. http://dx.doi.org/10.1164/ajrccm-conference.2020.201.1_meetingabstracts.a5609.

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He, M., J. MacLeod, S. J. Callahan, K. Qing, N. Tustison, M. Salerno, J. Mata, et al. "Assessment of Pulmonary Pathophysiology in Lung Transplant Recipients With and Without Chronic Lung Allograft Dysfunction Using Hyperpolarized Helium-3 MRI." In American Thoracic Society 2021 International Conference, May 14-19, 2021 - San Diego, CA. American Thoracic Society, 2021. http://dx.doi.org/10.1164/ajrccm-conference.2021.203.1_meetingabstracts.a4604.

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Tan, Yan, and Wei Tan. "Reducing Upstream Compliance Induces Downstream High Pulsatility Flow-Dependent Inflammatory Response in Pulmonary Endothelial Cells via TLR2/NF-KB Pathway." In ASME 2012 Summer Bioengineering Conference. American Society of Mechanical Engineers, 2012. http://dx.doi.org/10.1115/sbc2012-80900.

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Pulmonary arterial hypertension (PAH) is a group of chronic, progressive and fatal diseases, characterized by the dysfunction of the small arteries and microvasculature in the pulmonary circulation. Due to high blood pressure and high resistance in the pulmonary arteries, PAH causes detrimental damage on the lung and right heart ventricle. If left untreated, PAH quickly becomes life threatening. Although the exact pathophysiology remains unknown, there is increasing evidence suggesting that inflammation likely plays an important role in inducing and perpetuating the PAH progress. Although anti

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Sandler, H., B. Gerdin, and T. Saldeen. "STUDIES ON THE ROLE OF THROMBOXANE IN THROMBIN-INDUCED PULMONARY INSUFFICIENCY IN THE RAT." In XIth International Congress on Thrombosis and Haemostasis. Schattauer GmbH, 1987. http://dx.doi.org/10.1055/s-0038-1643379.

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The pathophysiology of the acute pulmonary damage that may occur in association with sepsis, hemorrhagic shock and microembolism seems to involve the activity of cyclooxygenase derived arachidonate metabolites. In the rat, pulmonary microembolism due to infusion of thrombin together with inhibition of fibrinolysis has been found to induce pulmonary insufficiency with similarities to the clinical adult respiratory distress syndrome. The infusion of thrombin results in systemic hypotension, pulmonary hypertension and platelet aggregation, and subsequently, with a certain dealy in time, to increa

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