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Thematische Bibliographien / Mice Reproduction / Zeitschriftenartikel

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Zeitschriftenartikel zum Thema „Mice Reproduction“

Autor: Grafiati

Veröffentlicht am 4. Juni 2021

Zuletzt aktualisiert am 20. Februar 2023

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1

Lafaille, Marie, Patrick Gouat, and Christophe Féron. "Efficiency of delayed reproduction in Mus spicilegus." Reproduction, Fertility and Development 27, no. 3 (2015): 491. http://dx.doi.org/10.1071/rd13130.

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To cope with seasonally varying ecological constraints, some mammals temporally suppress breeding or delay their first reproduction. In field conditions, mound-building mice (Mus spicilegus) born in spring begin to reproduce when 2–3 months old, whereas individuals born at the end of summer delay their first reproduction for 6–8 months until the following spring. In order to test age effects on reproductive performance in M. spicilegus, sexually naïve mice were paired when 2–3 months old or at 6–8 months of age, and surveyed for reproduction. We show here that under laboratory conditions the a

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Munger, James C., and William H. Karasov. "Sublethal parasites in white-footed mice: impact on survival and reproduction." Canadian Journal of Zoology 69, no. 2 (February 1, 1991): 398–404. http://dx.doi.org/10.1139/z91-062.

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The potential impact of two parasites on the population density of host white-footed mice (Peromyscus leucopus) was assessed by measuring effects on survival and reproduction in field populations. Thirty-eight mice infected with larvae of the bot fly Cuterebra angustifrons had their larvae removed, another 41 mice remained infected, and 46 other mice were naturally uninfected during the experiment. No effect of bot larvae removal was detected on either survival (measured as attrition) or reproduction (measured as end of reproductive season). However, contrary to expectation, naturally infected

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Harini, C., S. B. Sainath, and P. Sreenivasula Reddy. "Recovery of suppressed male reproduction in mice exposed to progesterone during embryonic development by testosterone." REPRODUCTION 137, no. 3 (March 2009): 439–48. http://dx.doi.org/10.1530/rep-08-0438.

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The present study aimed to examine whether transplacental exposure to progesterone caused male reproductive abnormalities and whether the changes can be reversed after testosterone administration. Progesterone was injected to mice on day 1, 3, and 7 of pregnancy. The male pups (F1 generation) were allowed to grow for 50 days and assessed for reproductive performance. Gestational exposure to progesterone (7 mg/kg body weight) resulted in significant body weight gain with a decrease in reproductive tissue indices in mice. Total sperm count, viable sperm, and motile sperm decreased in experimenta

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Rollo, C. D., J. Rintoul, and L. J. Kajiura. "Lifetime reproduction of giant transgenic mice: the energy stress paradigm." Canadian Journal of Zoology 75, no. 8 (August 1, 1997): 1336–45. http://dx.doi.org/10.1139/z97-758.

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Lifetime reproduction of female transgenic rat growth hormone (TRrGH) mice and their normal siblings was evaluated on a high-protein (38%) diet, a standard diet (23% protein), and the standard diet supplemented with sucrose cubes. Compared with those on the standard diet, normal mice fed the high-protein diet showed significant increases in litter size, number of litters, and lifetime fecundity. Number of litters and lifetime fecundity were also enhanced in normal mice fed sucrose. TRrGH mice showed no significant improvements in reproduction on the high-protein diet, but they were significant

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Yang, Jichun, Lihong Chen, Xiaoyan Zhang, Yunfeng Zhou, Dongjuan Zhang, Ming Huo, and Youfei Guan. "PPARs and Female Reproduction: Evidence from Genetically Manipulated Mice." PPAR Research 2008 (2008): 1–8. http://dx.doi.org/10.1155/2008/723243.

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Peroxisome proliferator-activated receptors (PPARs) are ligand-activated nuclear receptors controlling many important physiological processes, including lipid and glucose metabolism, energy homeostasis, inflammation, as well as cell proliferation and differentiation. In the past decade, intensive study of PPARs has shed novel insight into prevention and treatment of dyslipidemia, insulin resistance, and type 2 diabetes. Recently, a large body of research revealed that PPARs are also functionally expressed in reproductive organs and various parts of placenta during pregnancy, which strongly sug

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Reese, Jeff, Xuemei Zhao, Wen-Ge Ma, Naoko Brown, Timothy J. Maziasz, and S. K. Dey. "Comparative Analysis of Pharmacologic and/or Genetic Disruption of Cyclooxygenase-1 and Cyclooxygenase-2 Function in Female Reproduction in Mice*." Endocrinology 142, no. 7 (July 1, 2001): 3198–206. http://dx.doi.org/10.1210/endo.142.7.8307.

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Abstract Cyclooxygenase (COX)-derived prostaglandins are critical in female reproduction. Gene targeting studies show that ovulation, fertilization, implantation, and decidualization are defective in COX-2 deficient mice. We used genetic and pharmacologic approaches to perturb COX function and examine the differential and synergistic effects of inhibition of COX-1, COX-2, or of both isoforms on reproductive outcomes during early pregnancy in mice. The results demonstrate that simultaneous inhibition of COX-1 and COX-2 produces more severe effects on early pregnancy events than inhibition of ei

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Harrington, Monica. "Common disinfectants impair reproduction in mice." Lab Animal 43, no. 10 (September 19, 2014): 335. http://dx.doi.org/10.1038/laban.635.

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Yang, Haoxuan, Izhar Hyder Qazi, Bo Pan, Christiana Angel, Shichao Guo, Jingyu Yang, Yan Zhang, et al. "Dietary Selenium Supplementation Ameliorates Female Reproductive Efficiency in Aging Mice." Antioxidants 8, no. 12 (December 11, 2019): 634. http://dx.doi.org/10.3390/antiox8120634.

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Female reproductive (ovarian) aging is distinctively characterized by a markedly reduced reproductive function due to a remarkable decline in quality and quantity of follicles and oocytes. Selenium (Se) has been implicated in playing many important biological roles in male fertility and reproduction; however, its potential roles in female reproduction, particularly in aging subjects, remain poorly elucidated. Therefore, in the current study we used a murine model of female reproductive aging and elucidated how different Se-levels might affect the reproductive efficiency in aging females. Our r

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Hicks, Kristina L., Elysia Roche, James D. Wilkerson, and Krista E. Lindstrom. "Effects of Maternal Fenbendazole on Litter Size, Survival Rate, and Weaning Weight in C57BL/6J Mice." Journal of the American Association for Laboratory Animal Science 60, no. 6 (November 1, 2021): 630–36. http://dx.doi.org/10.30802/aalas-jaalas-21-000056.

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Fenbendazole is a broad-spectrum benzimidazole commonly used in laboratory animal medicine as an anthelmintic for elimination of pinworms. This drug is generally regarded as safe, with minimal side effects. Some data in rodent species indicate multiple physiologic effects of fenbendazole, including changes in immune parameters and behavior, but no studies to date have evaluated possible effects on reproduction in mice. The purpose of the current study was to determine the effects of several treatment regimens of fenbendazole on reproductive parameters in C57BL/6J mice. Uninfected mice were giv

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Roy, Angshumoy, and Martin M. Matzuk. "Deconstructing mammalian reproduction: using knockouts to define fertility pathways." Reproduction 131, no. 2 (February 2006): 207–19. http://dx.doi.org/10.1530/rep.1.00530.

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Reproduction is the sine qua non for the propagation of species and continuation of life. It is a complex biological process that is regulated by multiple factors during the reproductive life of an organism. Over the past decade, the molecular mechanisms regulating reproduction in mammals have been rapidly unraveled by the study of a vast number of mouse gene knockouts with impaired fertility. The use of reverse genetics to generate null mutants in mice through targeted disruption of specific genes has enabled researchers to identify essential regulators of spermatogenesis and oogenesis in viv

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Hollister, Anne, Patricia Okubara, J. Gary Watson, and Sterling Chaykin. "Reproduction in mice: Liver enlargement in mice during pregnancy and lactation." Life Sciences 40, no. 1 (January 1987): 11–18. http://dx.doi.org/10.1016/0024-3205(87)90246-3.

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Luo, Rongrong, Lei Chen, Xingxing Song, Xin Zhang, Wenhao Xu, Dongyang Han, Jianyu Zuo, et al. "Possible Role of GnIH as a Novel Link between Hyperphagia-Induced Obesity-Related Metabolic Derangements and Hypogonadism in Male Mice." International Journal of Molecular Sciences 23, no. 15 (July 22, 2022): 8066. http://dx.doi.org/10.3390/ijms23158066.

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Gonadotropin-inhibitory hormone (GnIH) is a reproductive inhibitor and an endogenous orexigenic neuropeptide that may be involved in energy homeostasis and reproduction. However, whether GnIH is a molecular signal link of metabolism and the reproductive system, and thus, regulates reproductive activity as a function of the energy state, is still unknown. In the present study, we investigated the involvement of GnIH in glycolipid metabolism and reproduction in vivo, and in the coupling between these two processes in the testis level. Our results showed that chronic intraperitoneal injection of

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Nonomura, M., K. Hoshino, T. Harigaya, H. Hashimoto, and O. Yoshida. "Effects of hyperprolactinaemia on reproduction in male mice." Journal of Endocrinology 107, no. 1 (October 1985): 71–76. http://dx.doi.org/10.1677/joe.0.1070071.

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ABSTRACT Hyperprolactinaemia induced by pituitary isografts in male host mice was confirmed by radioimmunoassay, but plasma testosterone levels determined by radioimmunoassay in these mice showed no changes. Immunoenzyme electron microscopic observations revealed large spherical-shaped immunoreactive prolactin granules in pituitary grafts in male hosts, regardless of the sex of the donor mice, indicating the disappearance of sexual dimorphism in prolactin-producing cells in hyperprolactinaemic mice. In hyperprolactinaemic host mice the male accessory sex glands, particularly the seminal vesicl

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Embree-Ku, Michelle, and Kim Boekelheide. "Absence of p53 and FasL Has Sexually Dimorphic Effects on Both Development and Reproduction." Experimental Biology and Medicine 227, no. 7 (July 2002): 545–53. http://dx.doi.org/10.1177/153537020222700720.

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Reproduction and development are highly dependent on apoptosis to balance the proliferation that necessarily occurs during these processes. How the absence of two apoptotic factors in mice would affect reproduction and development was examined. Given previous reports of increased neural tube defects in p53–/– female fetuses, decreased fertility in gld female mice, and altered spermatogenesis in both p53 and gld male mice, the possibility that these phenotypes might be enhanced by the elimination of a second apoptotic factor was investigated. The reproductive vigor and the health of offspring w

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Li, Biao, Zhihui Yang, Jingwen Hou, April McCracken, M. Anita Jennings, and Mark Y. J. Ma. "Compromised Reproductive Function in Adult Female Mice Selectively Expressing Mutant ErbB-1 Tyrosine Kinase Receptors in Astroglia." Molecular Endocrinology 17, no. 11 (November 1, 2003): 2365–76. http://dx.doi.org/10.1210/me.2003-0023.

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Abstract The ErbB-1 tyrosine kinase receptor plays critical roles in regulating physiological functions. This receptor-mediated signaling in astroglia has been implicated in controlling female sexual development via activating neurons that release LH-releasing hormone (LHRH), the neuropeptide required for the secretion of LH. It remains unknown whether astroglial ErbB-1 receptors are necessary for maintaining normal adult reproductive function. Here we provide genetic evidence that astroglia-specific and time-controlled disruption of ErbB-1 receptor signaling by expressing mutant ErbB-1 recept

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Jackson, C., and R. T. F. Bernard. "Effects of supplementary food on the winter inhibition of reproduction in male and female four-striped field mice (Rhabdomys pumilio)." Reproduction, Fertility and Development 17, no. 4 (2005): 393. http://dx.doi.org/10.1071/rd04134.

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The effects of winter food supplementation on reproduction in the seasonally breeding four-striped field mouse Rhabdomys pumilio were investigated at Mountain Zebra National Park in the Eastern Cape Province of South Africa. On both control and supplemented grids, reproductive activity in females was inhibited; no pregnant females were collected and juveniles were only present in the first winter month. The provision of additional food resulted in an increase in body mass and mass of the male and female reproductive organs. However, all males, from both grids, were spermatogenically active. Ov

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Nohara, Kazunari, Suhuan Liu, Matthew S. Meyers, Aurélie Waget, Mathieu Ferron, Gérard Karsenty, Rémy Burcelin, and Franck Mauvais-Jarvis. "Developmental androgen excess disrupts reproduction and energy homeostasis in adult male mice." Journal of Endocrinology 219, no. 3 (October 1, 2013): 259–68. http://dx.doi.org/10.1530/joe-13-0230.

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Polycystic ovary syndrome is a common endocrine disorder in females of reproductive age and is believed to have a developmental origin in which gestational androgenization programs reproductive and metabolic abnormalities in offspring. During gestation, both male and female fetuses are exposed to potential androgen excess. In this study, we determined the consequences of developmental androgenization in male mice exposed to neonatal testosterone (NTM). Adult NTM displayed hypogonadotropic hypogonadism with decreased serum testosterone and gonadotropin concentrations. Hypothalamic KiSS1 neurons

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MCBURNIE, M., J. BLAIRWEST, D. DENTON, and R. WEISINGER. "Sodium Intake and Reproduction in BALB/C Mice." Physiology & Behavior 66, no. 5 (July 1999): 873–79. http://dx.doi.org/10.1016/s0031-9384(99)00034-7.

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Höglund, N. J. "Effects of Ethyl Urethane on Reproduction in Mice." Acta Pharmacologica et Toxicologica 8, no. 1 (March 13, 2009): 82–84. http://dx.doi.org/10.1111/j.1600-0773.1952.tb02887.x.

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Pye, T. "Reproductive biology of the feral house mouse (Mus musculus) on subantarctic Macquarie Island." Wildlife Research 20, no. 6 (1993): 745. http://dx.doi.org/10.1071/wr9930745.

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Reproduction of the feral house mouse (Mus musculus) was studied on subantarctic Macquarie Island and found to be seasonal. Females begin oestrus-cycling in early spring, following a minimum 3-month winter anoestrous period. By late spring all mature females are in breeding condition. Breeding is continuous through spring, summer and into autumn. Postimplantation loss occurs throughout the breeding season. Late autumn pregnancies may fail. Average litter size is 6-7 but litters as large as 10 have been found. Young born in the latter half of the breeding season attain sexual maturity at a late

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Kiyosu, Chiyo, Takehito Tsuji, Kaoru Yamada, Shimpei Kajita, and Tetsuo Kunieda. "NPPC/NPR2 signaling is essential for oocyte meiotic arrest and cumulus oophorus formation during follicular development in the mouse ovary." REPRODUCTION 144, no. 2 (August 2012): 187–93. http://dx.doi.org/10.1530/rep-12-0050.

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Natriuretic peptide type C (NPPC) and its high affinity receptor, natriuretic peptide receptor 2 (NPR2), have been assumed to be involved in female reproduction and have recently been shown to play an essential role in maintaining meiotic arrest of oocytes. However, the overall role of NPPC/NPR2 signaling in female reproduction and ovarian function is still less clear. Here we report the defects observed in oocytes and follicles of mice homozygous for Nppclbab or Npr2cn, mutant alleles of Nppc or Npr2 respectively to clarify the exact consequences of lack of NPPC/NPR2 signaling in female repro

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Kennaway, D. J., A. Voultsios, and M. J. Boden. "241.Reproductive performance in Clock mutant mice." Reproduction, Fertility and Development 16, no. 9 (2004): 241. http://dx.doi.org/10.1071/srb04abs241.

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The relationship between circadian rhythmicity and rodent reproductive cyclicity is well established, but the impact of disrupted clock gene function on reproduction has not been investigated. This study evaluated the reproductive performance of melatonin deficient and proficient mice carrying a mutation in the core circadian gene, Clock. In natural matings, melatonin deficient Clock mutant mice took 2 to 3 days longer to mate and to subsequently deliver pups than their control line. The melatonin proficient mutants (Clock-MEL) had a smaller, but still significant delay (P < 0.05). The Cloc

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Zhang, JinFu, YaPing Gui, Tao Yuan, CuiDong Bian, and LiHe Guo. "Expression of GAT1 in Male Reproductive System and its Effects on Reproduction in Mice." Systems Biology in Reproductive Medicine 55, no. 5-6 (January 2009): 175–80. http://dx.doi.org/10.3109/19396360903030500.

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Ratajczak, Christine K., Katie L. Boehle, and Louis J. Muglia. "Impaired Steroidogenesis and Implantation Failure in Bmal1−/− Mice." Endocrinology 150, no. 4 (December 4, 2008): 1879–85. http://dx.doi.org/10.1210/en.2008-1021.

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Evidence in humans and rodents suggests that normal circadian rhythmicity is important for supporting reproductive function. A molecular clock underlies circadian rhythmicity. Impaired fertility is observed in some genetically altered mice with deficiencies in genes of the molecular clock, suggesting a critical role for these genes in reproduction. Here we systematically characterize the reproductive phenotype of females deficient in the clock gene Bmal1. Bmal1−/− females are infertile. They exhibit progression through the estrous cycle, although these cycles are prolonged. Normal follicular d

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Gouat, Patrick, Christophe Féron, and Simone Demouron. "Seasonal reproduction and delayed sexual maturity in mound-building mice Mus spicilegus." Reproduction, Fertility and Development 15, no. 3 (2003): 187. http://dx.doi.org/10.1071/rd02105.

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In the mound-building mouse, Mus spicilegus, which is found from Central to Eastern Europe, reproduction is seasonal and limited to spring and summer. In autumn, the mice build voluminous mounds composed of vegetable matter covered with earth, where juvenile animals (autumnal individuals) over-winter in groups without reproducing. Autumnal animals delay reproduction until the next spring when they are 6 months old. The influence and interactions of environmental (short light period and cold temperature (C conditions) compared with long light period and temperate temperature (T conditions)) and

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Kinnear, H. M., E. S. Constance, A. David, E. E. Marsh, V. Padmanabhan, A. Shikanov, and M. B. Moravek. "A mouse model to investigate the impact of testosterone therapy on reproduction in transgender men." Human Reproduction 34, no. 10 (October 2019): 2009–17. http://dx.doi.org/10.1093/humrep/dez177.

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Abstract STUDY QUESTION Can mice serve as a translational model to investigate the reproductive effects of testosterone (T) therapy commonly used by transgender men? SUMMARY ANSWER T enanthate subcutaneous injections at 0.45 mg twice weekly can be used in the postpubertal C57BL/6N female mouse to investigate the reproductive effects of T therapy given to transgender men. WHAT IS KNOWN ALREADY Most models of T treatment in female mice involve prenatal or prepubertal administration, which are not applicable to transgender men who often begin T therapy after puberty. Studies that have looked at t

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Rocha, Juliana S., Michael S. Bonkowski, Luiz R. França, and Andrzej Bartke. "Mild Calorie Restriction Does Not Affect Testosterone Levels and Testicular Gene Expression in Mutant Mice." Experimental Biology and Medicine 232, no. 8 (September 2007): 1050–63. http://dx.doi.org/10.3181/0703-rm-52.

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The hypothalamic-pituitary-gonadal (HPG) axis and the somatotropic axis are influenced by nutritional factors. Calorie restriction (CR) extends lifespan but suppresses both the HPG and the somatotropic axes. Since most CR studies use a fairly severe (40%–60%) reduction of calorie intake, we hypothesized that a milder CR (20%) might not be deleterious to reproduction in male mice. To test this hypothesis, we evaluated the effects of 20% CR on testicular testosterone content and on testicular expression of genes that are relevant to testicular function and reproductive competence, including insu

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Huang, Yinqiong, Junping Wen, and Gang Chen. "Role and Mechanism of Chronic Restraint Stress in Regulating Energy Metabolism and Reproductive Function Through Hypothalamic Kisspeptin Neurons." Journal of the Endocrine Society 5, Supplement_1 (May 1, 2021): A551—A552. http://dx.doi.org/10.1210/jendso/bvab048.1124.

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Abstract Objectives: The purpose of this study was to investigate whether stress affected energy metabolism and reproductive function through the glucocorticoid receptor (GR) on Kisspeptin neurons in the hypothalamus. Methods: 1. The first part of this study focused on the effects of chronic restraint stress on energy metabolism and reproductive function in mice. 2. The second part of this study focused on the effects of chronic restraint stress on the expression of serum cortisol and prolactin and the expression of GR and Kiss1 genes in the hypothalamus.3. Based on the above research, we cons

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Shimshek, Derya R., Thorsten Bus, Valery Grinevich, Frank N. Single, Volker Mack, Rolf Sprengel, Daniel J. Spergel, and Peter H. Seeburg. "Impaired Reproductive Behavior by Lack of GluR-B Containing AMPA Receptors But Not of NMDA Receptors in Hypothalamic and Septal Neurons." Molecular Endocrinology 20, no. 1 (January 1, 2006): 219–31. http://dx.doi.org/10.1210/me.2005-0262.

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Abstract The roles of ionotropic glutamate receptors in mammalian reproduction are unknown. We therefore generated mice lacking a major subtype of (S)-α-amino-3-hydroxy-5-methyl-isoxazolepropionic acid (AMPA) receptors or all N-methyl-d-aspartate (NMDA) receptors in GnRH neurons and other mainly limbic system neurons, primarily in hypothalamic and septal areas. Male mice without NMDA receptors in these neurons were not impaired in breeding and exhibited similar GnRH secretion as control littermates. However, male mice lacking GluR-B containing AMPA receptors in these neurons were poor breeders

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Garratt, Michael, Heather Try, Kristina O. Smiley, David R. Grattan, and Robert C. Brooks. "Mating in the absence of fertilization promotes a growth-reproduction versus lifespan trade-off in female mice." Proceedings of the National Academy of Sciences 117, no. 27 (June 22, 2020): 15748–54. http://dx.doi.org/10.1073/pnas.2003159117.

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Trade-offs between growth, reproduction, and lifespan constrain animal life histories, leading to evolutionary diversification of life history cycles in different environments. In female mammals, gestation and lactation are expected to impose the major costs of reproduction, driving reproductive trade-offs, although mating also requires interactions with males that could themselves influence life history. Here we show that a male’s presence by itself leads to lifelong alterations in life history in female mice. Housing C57BL/6J female mice with sterilized males early in life led to an increase

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Antolová, D., and K. Reiterová. "The course of Echinococcus multilocularis infection and pregnancy in mice model." Helminthologia 48, no. 4 (December 1, 2011): 251–55. http://dx.doi.org/10.2478/s11687-011-0035-1.

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AbstractParasitic infection during pregnancy can affect the course of pregnancy, complicate the foeto-maternal relationship and increase abortion rate. The work was aimed to study the course of E. multilocularis secondary infection and reproductive parameters in infected mice. The Balb/c mice were bred in the exponential phase of the metacestode growth, after infection with two different doses (0.5 ml or 1.25 ml) of metacestode material. In comparison to uninfected control group, the effect of infection on reproductive parameters (natality, size of litters) and the course of disease during pre

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Martin, J. Ryan, Sarah B. Lieber, James McGrath, Marya Shanabrough, Tamas L. Horvath, and Hugh S. Taylor. "Maternal Ghrelin Deficiency Compromises Reproduction in Female Progeny through Altered Uterine Developmental Programming." Endocrinology 152, no. 5 (February 15, 2011): 2060–66. http://dx.doi.org/10.1210/en.2010-1485.

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Ghrelin has a well-known role in the regulation of appetite, satiety, energy metabolism, and reproduction; however ghrelin has not been implicated in reproductive tract development. We examined the effect of ghrelin deficiency on the developmental programming of female fertility. We observed that female wild-type mice born of ghrelin heterozygote dams (i.e. exposed in utero to ghrelin deficiency) had diminished fertility and produced smaller litters. We demonstrate that exposure to in utero ghrelin deficiency led to altered developmental programming of the reproductive tract. The number of ova

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Klenke, Ulrike, Carol Taylor-Burds, and Susan Wray. "Metabolic Influences on Reproduction: Adiponectin Attenuates GnRH Neuronal Activity in Female Mice." Endocrinology 155, no. 5 (May 1, 2014): 1851–63. http://dx.doi.org/10.1210/en.2013-1677.

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Metabolic dysfunctions are often linked to reproductive abnormalities. Adiponectin (ADP), a peripheral hormone secreted by white adipose tissue, is important in energy homeostasis and appetite regulation. GnRH neurons are integral components of the reproductive axis, controlling synthesis, and release of gonadotropins. This report examined whether ADP can directly act on GnRH neurons. Double-label immunofluorescence on brain sections from adult female revealed that a subpopulation of GnRH neurons express ADP receptor (AdipoR)2. GnRH/AdipoR2+ cells were distributed throughout the forebrain. To

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Ivanova, Margarita, Klaudia M. Dobrzycka, Shiming Jiang, Kai Michaelis, Rene Meyer, Kaiyan Kang, Brian Adkins, et al. "Scaffold Attachment Factor B1 Functions in Development, Growth, and Reproduction." Molecular and Cellular Biology 25, no. 8 (April 15, 2005): 2995–3006. http://dx.doi.org/10.1128/mcb.25.8.2995-3006.2005.

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ABSTRACT Scaffold attachment factor B1 (SAFB1) is a multifunctional protein that can bind both DNA and RNA and is involved in RNA processing and stress response. In addition, SAFB1 contains a transcriptional repression domain and can bind certain hormone receptors and repress their activity. To assess the role of SAFB1 in vivo, we generated SAFB1 mutant mice through targeted deletion in embryonic stem cells. While viable homozygous mutant (SAFB1−/−) mice were obtained, genotypic distribution indicated that homozygous deficiency resulted in both prenatal and neonatal lethality. Mice lacking SAF

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Polotsky, Alex, and Jasmine Aly. "Paternal Diet and Obesity: Effects on Reproduction." Seminars in Reproductive Medicine 35, no. 04 (July 2017): 313–17. http://dx.doi.org/10.1055/s-0037-1602593.

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AbstractAlthough most research has focused on maternal obesity, there is growing data to indicate that obesity in the father can affect reproduction. Supporting data come from both mouse and human studies. Murine studies found that obese male mice exhibited decreased motility and reduced hyperactivated progression versus lean mice. Obese mice also exhibited sperm with increased levels of intracellular and mitochondrial levels of reactive oxygen species, increased sperm damage, and lower levels of capacitation, which has been shown to be associated with poor fertilization rates following in vit

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Bachelot, Anne, and Nadine Binart. "Reproductive role of prolactin." Reproduction 133, no. 2 (February 2007): 361–69. http://dx.doi.org/10.1530/rep-06-0299.

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The biological actions of prolactin (PRL), a polypeptide hormone, are mostly related to lactation and reproduction. These actions have been clarified by studies of PRL and PRL-deficient receptor mice, which have a clear phenotype of reproductive failure at multiple sites. This review aims to summarize current knowledge about PRL and its receptor, role in reproductive axis and presents information of hyperprolactinemia in reproductive medicine. Our understanding of the physiology and transduction pathway of PRL has largely increased in the past 20 years with the cloning of PRL and its receptor

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TOMIC, D., M. FRECH, J. BABUS, D. SYMONDS, P. FURTH, R. KOOS та J. FLAWS. "Effects of ERα overexpression on female reproduction in mice". Reproductive Toxicology 23, № 3 (квітень 2007): 317–25. http://dx.doi.org/10.1016/j.reprotox.2006.08.004.

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Calle, J., J. Olarte, R. Pinzon, L. F. Ospina, M. C. Mendoza, and M. J. Orozco. "Alterations in the reproduction of mice induced by rapanone." Journal of Ethnopharmacology 71, no. 3 (August 2000): 521–25. http://dx.doi.org/10.1016/s0378-8741(99)00214-7.

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Ishiniwa, Hiroko, Mizuki Sakai, Shimon Tohma, Hidenori Matsuki, Yukio Takahashi, Hideo Kajiwara, and Tsuneo Sekijima. "Dioxin pollution disrupts reproduction in male Japanese field mice." Ecotoxicology 22, no. 9 (September 13, 2013): 1335–47. http://dx.doi.org/10.1007/s10646-013-1120-7.

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Nagai, J., G. Davis, K. Nonaka, and H. Sasada. "Growth and reproduction of mice developed from bisected embryos." Theriogenology 32, no. 3 (September 1989): 475–83. http://dx.doi.org/10.1016/0093-691x(89)90014-9.

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Rocha, Joao L., Eugene J. Eisen, Frank Siewerdt, L. Dale Van Vleck, and Daniel Pomp. "A large-sample QTL study in mice: III. Reproduction." Mammalian Genome 15, no. 11 (November 2004): 878–86. http://dx.doi.org/10.1007/s00335-004-2364-6.

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Ohtsuka, Satoshi, Satoshi Takaki, Masanori Iseki, Kanta Miyoshi, Naomi Nakagata, Yuki Kataoka, Nobuaki Yoshida, Kiyoshi Takatsu, and Akihiko Yoshimura. "SH2-B Is Required for Both Male and Female Reproduction." Molecular and Cellular Biology 22, no. 9 (May 1, 2002): 3066–77. http://dx.doi.org/10.1128/mcb.22.9.3066-3077.2002.

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ABSTRACT Many growth factors and hormones modulate the reproductive status in mammals. Among these, insulin and insulin-like growth factor I (IGF-I) regulate the development of gonadal tissues. SH2-B has been shown to interact with insulin and IGF-I receptors, although the role of SH2-B in these signals has not been clarified. To investigate the role of SH2-B, we generated mice with a targeted disruption of the SH2-B gene. Both male and female SH2-B−/− mice showed slight retardation in growth and impaired fertility. Female knockout mice possess small, anovulatory ovaries with reduced numbers o

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Boden, M. J., and D. J. Kennaway. "297. Reproduction in the arrhythmic Bmal1 knockout mouse." Reproduction, Fertility and Development 17, no. 9 (2005): 126. http://dx.doi.org/10.1071/srb05abs297.

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There is strong epidemiological evidence indicating that disruption of the endogenous circadian rhythms can cause a range of health problems ranging from metabolic and cardiovascular disorders to reproductive failure. Circadian rhythmicity is generated by a suite of genes called ‘clock genes’ that are cyclically expressed in the brain and peripheral tissues. The CLOCK and BMAL1 transcription factors regulate the expression of many genes involved in cell growth, angiogenesis and development. The Bmal1 knockout mouse provides an interesting model to analyse the impact of arrhythmicity on reprodu

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Heideman, Paul D., Julian T. Pittman, Kristin A. Schubert, Christen M. R. Dubois, Jennifer Bowles, Sean M. Lowe, and Matthew R. Price. "Variation in levels of luteinizing hormone and reproductive photoresponsiveness in a population of white-footed mice (Peromyscus leucopus)." American Journal of Physiology-Regulatory, Integrative and Comparative Physiology 298, no. 6 (June 2010): R1543—R1548. http://dx.doi.org/10.1152/ajpregu.00686.2009.

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Natural genetic variation in reproduction and life history strategies is a manifestation of variation in underlying regulatory neuronal and endocrine systems. A test of the hypothesis that genetic variation in luteinizing hormone (LH) level could be related to a life history trait, seasonal reproduction, was conducted on artificial selection lines from a wild-source population of white-footed mice ( Peromyscus leucopus ). Variation exists in the degree of suppression of reproduction by winter short-day photoperiods (SD) in wild-source individuals and in the laboratory population. In this popul

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McKenna, Jarrod, Sally Catt, Mulyoto Pangestu, and Peter Temple-Smith. "Successful sperm cryopreservation in Egyptian spiny mice Acomys cahirinus." Reproduction, Fertility and Development 32, no. 16 (2020): 1293. http://dx.doi.org/10.1071/rd20115.

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The menstruating Egyptian spiny mouse has recently been proposed as a new animal model for reproductive health research. Unfortunately, little is known about reproduction in males. This study compared several characteristics of sperm function before and after cryopreservation. Epididymal spermatozoa were cryopreserved in different concentrations of raffinose and skim milk and tested for motility and membrane integrity (Experiment 1). Further evaluations of motility, plasma membrane and acrosome integrity, mitochondrial membrane potential and DNA integrity were conducted with the addition of l-

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Lee, Sungeun, Dong-Wook Kang, Susan Hudgins-Spivey, Andree Krust, Eun-Young Lee, Youngbum Koo, Yongpil Cheon, Myung Chan Gye, Pierre Chambon та CheMyong Ko. "Theca-Specific Estrogen Receptor-α Knockout Mice Lose Fertility Prematurely". Endocrinology 150, № 8 (7 травня 2009): 3855–62. http://dx.doi.org/10.1210/en.2008-1774.

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Estrogen receptor-α (Esr1) mediates estrogen action in regulating at all levels of the hypothalamic-pituitary-ovarian axis. Whereas the importance of Esr1 in hypothalamus and pituitary has been demonstrated by loss of fertility in the neuron- and pituitary-specific Esr1 knockout mice, whether Esr1 plays a critical role in the ovary remains to be determined. In the ovary, Esr1 is mainly expressed in the theca/interstitial cells and germinal epithelium and thus is believed to mediate estrogen action in these cells. In this study, we assessed the importance of Esr1 in the ovarian theca cells in r

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Bera, Tapan K., and Ira Pastan. "Mesothelin Is Not Required for Normal Mouse Development or Reproduction." Molecular and Cellular Biology 20, no. 8 (April 15, 2000): 2902–6. http://dx.doi.org/10.1128/mcb.20.8.2902-2906.2000.

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ABSTRACT Mesothelin is a glycosylphosphatidylinositol-linked glycoprotein highly expressed in mesothelial cells, mesotheliomas, and ovarian cancer, but the biological function(s) of the protein is not known. We have analyzed the expression of the mouse mesothelin gene in different developmental stages and in various adult tissues by Northern hybridization. The 2.5-kb mesothelin transcript was detected in the mRNA of E 7.0, E 15.0, and E 17.0 stages of mouse development. In adult tissues the mesothelin gene was expressed in lung, heart, spleen, liver, kidney, and testis. To directly assess the

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Starobor, N. N., та O. V. Raskosha. "Reproductive parameters in Аf mice after chronic low-dose gamma radiation". Proceedings of the Komi Science Centre of the Ural Division of the Russian Academy of Sciences 5 (2021): 20–28. http://dx.doi.org/10.19110/1994-5655-2021-5-20-28.

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The results of the study of reproductive parameters in Af mice after chronic low-intensity γ-radiation at doses of 10, 20 and 30 cGy are presented. Males and females of the experimental groups were exposed to external γ-radiation (0.474x106 and 0.451x106 kBq226Ra) for 29, 56 and 84 days, at an average dose rate of 150 µSv/h. Immediately after the end of radiation exposure, pairs for animal reproduction were formed in the experimental and control groups. During the next three months, the number of females participating in reproduction, the number of litters and the number of cubs born were reco

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Hou, SY, L. Zhang, K. Wu, and L. Xia. "Thioglycolic acid inhibits mouse oocyte maturation and affects chromosomal arrangement and spindle configuration." Toxicology and Industrial Health 24, no. 4 (May 2008): 227–34. http://dx.doi.org/10.1177/0748233708095862.

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Previous studies have shown that thioglycolic acid (TGA) leads to potential reproductive toxicology. To clarify the exact effects of this compound on reproduction, mice oocytes were treated with different TGA doses. At the end of the culture period, the nuclear status of mice oocytes was assessed under an inverted microscope. After immunofluorescence staining, the chromosomal arrangement and spindle configuration of oocytes were evaluated. The results indicated that TGA decreases the percentage of first polar body formation but does not influence that of germinal vesicle breakdown. TGA induces

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De Bond, Julie-Ann P., and Jeremy T. Smith. "Kisspeptin and energy balance in reproduction." REPRODUCTION 147, no. 3 (March 2014): R53—R63. http://dx.doi.org/10.1530/rep-13-0509.

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Kisspeptin is vital for the neuroendocrine regulation of GNRH secretion. Kisspeptin neurons are now recognized as a central pathway responsible for conveying key homeostatic information to GNRH neurons. This pathway is likely to mediate the well-established link between energy balance and reproductive function. Thus, in states of severely altered energy balance (either negative or positive), fertility is compromised, as isKiss1expression in the arcuate nucleus. A number of metabolic modulators have been proposed as regulators of kisspeptin neurons including leptin, ghrelin, pro-opiomelanocorti

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