Thematische Bibliographien / Novel NSAID

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Auswahl der wissenschaftlichen Literatur zum Thema „Novel NSAID“

Autor: Grafiati

Veröffentlicht am 7. Juni 2025

Zuletzt aktualisiert am 2. August 2025

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Zeitschriftenartikel zum Thema "Novel NSAID"

Wallace, John L. "NSAID Gastroenteropathy: Past, Present and Future." Canadian Journal of Gastroenterology 10, no. 7 (1996): 451–59. http://dx.doi.org/10.1155/1996/850710.

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The toxicity of nonsteroidal anti-inflammatory drugs (NSAIDs) in the gastrointestinal tract continues to be a major limitation to their use in the treatment of inflammatory disorders. Better understanding of the pathogenesis of NSAID enteropathy has facilitated the development of novel NSAIDs that spare the gastrointestinal tract. In particular, identification and characterization of the inducible form of prostaglandin synthase has led to the design of novel NSAIDs that specifically target that enzyme. The pathogenesis of NSAID gastroenteropathy is reviewed, as are the strategies that have bee

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Johnson, Annie, та Michael Myers. "NSAIDs inhibit NFkβ activation to prevent inflammation (IRC4P.602)". Journal of Immunology 194, № 1_Supplement (2015): 57.19. http://dx.doi.org/10.4049/jimmunol.194.supp.57.19.

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Abstract Non-steroidal anti-inflammatory drugs (NSAIDs) control inflammation through reductions in Prostaglandin E2 (PGE2) levels. Recent work by this laboratory identified NSAID-induced changes in inflammatory gene expression that appear to be independent of changes in PGE2; however, the molecular mechanism is unknown. Our goal is to determine whether NSAIDs affect gene expression via inhibition of NFkβ. Knowledge of NSAID gene control will provide a mechanism for screening novel drugs claiming anti-inflammatory properties. RAW 264.7 cells transfected with a plasmid under NFkβ control to expr

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&NA;. "Novel NSAID highly specific." Inpharma Weekly &NA;, no. 907 (1993): 10. http://dx.doi.org/10.2165/00128413-199309070-00020.

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Rigas, B. "Novel agents for cancer prevention based on nitric oxide." Biochemical Society Transactions 35, no. 5 (2007): 1364–68. http://dx.doi.org/10.1042/bst0351364.

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NO (nitric oxide) biology has provided the impetus for the development of anticancer agents based on their ability to release NO. NO-NSAIDs (NO-donating non-steroidal anti-inflammatory drugs), consisting of a conventional NSAID to which an NO-releasing moiety is covalently attached, are promising chemopreventive agents against cancer. Compared with their parent compounds, NO-NSAIDs are up to several hundred times more potent in inhibiting the growth of cancer cell lines and prevent colon and pancreatic cancer in animal models. Their chemopreventive effect is due to inhibition of proliferation,

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Hoggatt, Jonathan, Pratibha Singh, Amber Hoggatt, Jennifer M. Speth, and Louis M. Pelus. "Inhibition of Prostaglandin E2 (PGE2) Signaling by Non-Steroidal Anti-Inflammatory Drugs (NSAIDs) or EP4 Receptor Antagonism Expands Hematopoietic Stem and Progenitor Cells (HSPC) and Enhances Their Mobilization to Peripheral Blood in Mice and Baboons." Blood 114, no. 22 (2009): 84. http://dx.doi.org/10.1182/blood.v114.22.84.84.

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Abstract Abstract 84 PGE2 is a physiological regulator of HSPC and exhibits both stimulatory and inhibitory activities. We recently reported that PGE2 increases CXCR4 expression and homing of HSPC to bone marrow (Hoggatt et al, Blood 2009). Since PGE2 is produced within the marrow microenvironment, we hypothesized that blocking PGE2 synthesis in vivo via inhibition of cyclooxygenases (COX) by NSAIDs would facilitate HSPC mobilization and serve as an adjunct to current mobilization regimens. Treatment of mice with the NSAID indomethacin b.i.d. along with G-CSF for 4 days resulted in an increase

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Tachecí, Ilja, Marcela Kopáčová, Stanislav Rejchrt, and Jan Bureš. "Non-steroidal Anti-inflammatory Drug Induced Injury to the Small Intestine." Acta Medica (Hradec Kralove, Czech Republic) 53, no. 1 (2010): 3–11. http://dx.doi.org/10.14712/18059694.2016.56.

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Non-steroidal anti-inflammatory drug (NSAIDs) induced enteropathy represents an important complication of one of the most commonly used drugs worldwide. Due to previous diagnostics difficulties the real prevalence of this disease was underestimated for a long time. The pathogenesis of NSAID-enteropathy is more multifactorial and complex than formerly assumed but has still not been fully uncovered. A combination of the local and systemic effect plays an important role in pathogenesis. Thanks to novel enteroscopy methods (wireless capsule endoscopy, double balloon enteroscopy), small bowel lesio

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Kirk, Julienne K., Jennifer M. Hamilton, and Kathy C. Phelps. "Evaluation of Novel New NSAIDs: A Review of COX-2 Inhibitors, with an Emphasis on Gastrointestinal Toxicity." Journal of Pharmacy Practice 12, no. 5 (1999): 401–11. http://dx.doi.org/10.1177/089719009901200506.

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Identification of two isoforms of cyclooxygenase, COX-1 and COX-2, has initiated a revolution in the approach to pharmacologie pain management. It has been further determined that inhibition of COX-2 reduces inflammation, and inhibition of COX-1 compromises gastrointestinal mucosal integrity. As traditional nonsteroidal anti-inflammatory drugs (NSAIDs) inhibit both COX-1 and COX-2, gastrointestinal ulceration can develop in association with the use of these agents to control pain and inflammation. An ideal NSAID would, therefore, inhibit COX-2 to provide anti-inflammatory effects while leaving

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MECHCATIE, ELIZABETH. "Arthritis Advisory Panel Rejects Novel NSAID." Rheumatology News 9, no. 6 (2010): 10. https://doi.org/10.1016/s1541-9800(10)70288-x.

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PLAYFORD, Raymond J., Christopher E. MACDONALD, Denis P. CALNAN, et al. "Co-administration of the health food supplement, bovine colostrum, reduces the acute non-steroidal anti-inflammatory drug-induced increase in intestinal permeability." Clinical Science 100, no. 6 (2001): 627–33. http://dx.doi.org/10.1042/cs1000627.

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Non-steroidal anti-inflammatory drugs (NSAIDs) are effective analgesics but cause gastrointestinal injury. Present prophylactic measures are suboptimal and novel therapies are required. Bovine colostrum is a cheap, readily available source of growth factors, which reduces gastrointestinal injury in rats and mice. We therefore examined whether spray-dried, defatted colostrum could reduce the rise in gut permeability (a non-invasive marker of intestinal injury) caused by NSAIDs in volunteers and patients taking NSAIDs for clinical reasons. Healthy male volunteers (n = 7) participated in a random

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Leitch, Sharon, Jiaxu Zeng, Alesha Smith, and Tim Stokes. "Avoiding anti-inflammatories: a randomised controlled trial testing the effect of an eHealth information package on primary healthcare patient medication knowledge and behaviour in Aotearoa New Zealand." BMJ Open 14, no. 11 (2024): e081545. http://dx.doi.org/10.1136/bmjopen-2023-081545.

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BackgroundPatient medication knowledge and health literacy affect patient safety. Taking angiotensin-converting enzyme inhibitors (ACE-i) or angiotensin II receptor blockers (ARBs), with diuretics and non-steroidal anti-inflammatory medications (NSAIDs) is nephrotoxic. Patients may not know of this risk. An eHealth information package was developed to inform patients at risk of taking this combination of medication.ObjectiveTo assess the impact of the eHealth information package on patient knowledge and behaviour.DesignThis was a two-arm, parallel, randomised control trial. A knowledge quiz an

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Dissertationen zum Thema "Novel NSAID"

DELL'OMO, GIULIA. "IDENTIFICATION AND CHARACTERIZATION OF SIRT1 AS A NOVEL TARGET OF NSAIDS CHEMOPREVENTION." Doctoral thesis, Università degli Studi di Milano, 2018. http://hdl.handle.net/2434/605551.

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Several epidemiological, clinical and experimental studies proposed non-steroidal antiinflammatory drugs (NSAIDs) as promising chemopreventive agents for many types of cancer. However, their use in chronic treatment is hampered by gastrointestinal, renal and cardiovascular side effects mainly due to the inhibition of cyclooxygenase (COX) isoenzymes. The development of NSAIDs as chemopreventive agents is prevented by the limited knowledge of the mechanism underlying the anti-cancer properties of these drugs. Inhibition of COX has been proposed as the mechanism underlying their anti-neopla

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Mohamed, Aymen I. I. "The effects of NO-NSAID and their novel analogues on bone metabolism in vitro and in vivo." Thesis, University of Aberdeen, 2004. http://digitool.abdn.ac.uk/R?func=search-advanced-go&find_code1=WSN&request1=AAIU179527.

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Accelerated osteoclastic bone resorption plays an important role in the pathogenesis of osteoporosis and other common bone diseases such as Paget's disease of bone, rheumatoid arthritis and cancer-associated bone disease. The prevention and treatment of these diseases is based on agents that inhibit osteoclast (OC) formation and resorption. Nitrated non-steroidal anti-flammatory drugs (NO-NSAID) are a recently developed group of compounds that contain a conventional NSAID linked to a nitric oxide (NO)-donor group. As prostaglandins and NO can both stimulate and inhibit OC formation and bone re

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Zhou, Ziyuan. "RATIONAL DESIGN, SYNTHESIS, AND CHARACTERIZATION OF NOVEL mPGES-1 INHIBITORS AS NEXT GENERATION OF ANTI-INFLAMMATORY DRUGS." UKnowledge, 2017. http://uknowledge.uky.edu/pharmacy_etds/75.

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Aspirin and other nonsteroidal anti-inflammatory drugs (NSAIDs) are currently widely used as fever and pain relief in patients with arthritis and other inflammatory symptoms. NSAIDs effect by inhibiting cyclooxygenase-1 (COX-1) and/or cyclooxygenase-2 (COX-2). COX isozymes (COXs) are key enzymes in the biosynthesis of prostaglandin H2 (PGH2) from arachidonic acid (AA). It is now clear that prostaglandin E2 (PGE2), one of the downstream products of PGH2, is the main mediator in both chronic and acute inflammation. Microsomal prostaglandin E synthase (mPGES-1) is the terminal enzyme of COX-2 in

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Cheng, Yen-Ling, and 鄭雁玲. "Design and Development of Novel Injectable COX-2 NSAIDs." Thesis, 2005. http://ndltd.ncl.edu.tw/handle/53813491214274258725.

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碩士<br>高雄醫學大學<br>藥學研究所碩士班<br>93<br>ABSTRACT Nonsteroidal anti-inflammatory drugs (NSAIDs) have been most commonly available in the treatment of acute and chronic pain, and so has been in the inflammatory response. NSAIDs generally can be divided into two types: traditional and COX-2-specific. The latter is with fewer side effects on the whole body, and only one of its kinds is clinically available for intravenous and intamuscular infections. The research and development of injectable COX-2 NSAIDs will be conducive to the clinical treatment. The pharmacodynemics study was to investigate

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Bücher zum Thema "Novel NSAID"

Roddy, Edward, and Michael Doherty. Calcium pyrophosphate crystal deposition (CPPD). Oxford University Press, 2013. http://dx.doi.org/10.1093/med/9780199642489.003.0142.

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Calcium pyrophosphate crystal deposition (CPPD) in articular cartilage is a common age-related phenomenon. Recent important advances in our understanding of the pathophysiology of pyrophosphate metabolism include the identification of a mutation within the ANK gene which associates with familial CPPD, and elucidation of the interleukin-1β‎ (IL-1β‎)-dependent mechanisms by which crystals invoke an inflammatory response. Risk factors for CPPD include age, prior joint damage and osteoarthritis, genetic factors, and occasionally metabolic diseases (hyperparathyroidism, haemochromatosis, hypomagnes

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Bannwarth, Bernard, and Francis Berenbaum. Systemic analgesics (including paracetamol and opioids). Oxford University Press, 2016. http://dx.doi.org/10.1093/med/9780199668847.003.0029.

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Apart from non-steroidal anti-inflammatory drugs (NSAIDs), there are only two categories of systemic analgesics, namely paracetamol (acetaminophen) and opioids, that are currently available worldwide for clinical use. Paracetamol is poorly effective in relieving pain and improving function in patients with symptomatic osteoarthritis (OA). Furthermore, its safety profile is less favourable than classically thought. In fact, there is evidence paracetamol acts as a weak inhibitor of the cyclooxygenase enzymes. Given that paracetamol poses a lower risk of severe adverse events than NSAIDs while be

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Buchteile zum Thema "Novel NSAID"

Del Soldato, P., G. Cirino, and J. L. Wallace. "NO-NSAID: A Novel Class of Anti-Inflammatory Drugs with Reduced Gastrointestinal and Renal Toxicity." In Cell Injury and Protection in the Gastrointestinal Tract. Springer Netherlands, 1997. http://dx.doi.org/10.1007/978-94-011-5392-8_3.

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Serhan, Charles N., Clary B. Clish, Jessica Brannon, Sean P. Colgan, Karsten Gronert, and Nan Chiang. "Antimicroinflammatory Lipid Signals from Vascular Cyclooxygenase-2: A Novel Mechanism for NSAID and N-3 PUFA Therapeutic Actions." In Advances in Prostaglandin and Leukotriene Research. Springer Netherlands, 2001. http://dx.doi.org/10.1007/978-94-015-9721-0_9.

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Szabo, S., E. Gyomber, R. E. Morales, L. Nagy, and P. Vattay. "Novel strategies of gastric and duodenal mucosal protection against NSAID injury: role of protease inhibitors, muscle relaxants and growth factors." In Side-Effects of Anti-Inflammatory Drugs 3. Springer Netherlands, 1992. http://dx.doi.org/10.1007/978-94-011-2982-4_15.

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Okuyan, Erhan, and Mulaim Sizer. "Dysmenorrhoea Its Treatment and Relationship with Nutrition." In Obstetrics & Gynecology and Nutrition. Nobel Tip Kitabevleri, 2024. http://dx.doi.org/10.69860/nobel.9786053359494.5.

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Dysmenorrhea, causing significant discomfort for many women, is divided into primary (without underlying pathology) and secondary (due to conditions like endometriosis). Diagnosis requires detailed history and examination. Treatment includes NSAIDs, acetaminophen, and hormonal contraceptives. In some cases, surgical interventions may be necessary.. Nutritional changes, such as low-fat vegan diets, vitamin supplements, and the Mediterranean diet, can help manage symptoms. Complementary therapies like exercise, stress management, and acupuncture also offer relief. Overall, dietary and lifestyle

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Yucelsen, Can. "Approach to Pericarditis." In Thoracic Infections. Nobel Tip Kitabevleri, 2024. http://dx.doi.org/10.69860/nobel.9786053358930.11.

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Pericarditis is the inflammation of the parietal and visceral layers of the pericardium, presenting with characteristic chest pain, electrocardiographic changes, and a pericardial friction rub. It is the most frequent form of pericardial disease and a common cause of chest pain, particularly affecting males between 20-50 years. The etiology includes viral, bacterial, fungal, and parasitic infections, autoimmune diseases, trauma, and other conditions like uremia and hypothyroidism. The pathophysiology involves the activation of the inflammatory cascade leading to pericardial effusion, which can

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Wallace, John L., Quentin J. Pittman, and Giuseppe Cirino. "Nitric Oxide-Releasing Nsaids: a Novel Class of Gi-Sparing Anti-Inflammatory Drugs." In Novel Molecular Approaches to Anti-Inflammatory Therapy. Birkhäuser Basel, 1995. http://dx.doi.org/10.1007/978-3-0348-7276-8_12.

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Del Soldato, P., L. Cuzzolin, A. Adami, A. Conforti, F. Crivellente, and G. Benoni. "Nitric Oxide-Releasing NSAIDs, a Novel Class of Safe and Effective Anti-Inflammatory Agents." In Side Effects of Anti-Inflammatory Drugs IV. Springer Netherlands, 1997. http://dx.doi.org/10.1007/978-94-011-5394-2_25.

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M. Heffernan, Shane, and Gillian E. Conway. "Nutraceutical Alternatives to Pharmaceutical Analgesics in Osteoarthritis." In Pain Management - Practices, Novel Therapies and Bioactives. IntechOpen, 2021. http://dx.doi.org/10.5772/intechopen.95919.

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Chronic pain is a considerable health concern worldwide, effecting almost 30% of all European adults. Osteoarthritis (OA), a progressive pro-inflammatory condition, is one of the leading causes of chronic pain (effecting 13% of all those over 50 years, globally) and is the most common cause of joint pain. The prevalence of non-steroidal anti-inflammatory drug (NSAIDs) and analgesic use has been well studied and is abundant throughout the western world, with women being the greatest users and ibuprofen generally being the most reported NSAID. In the US, 65% of all OA patients are prescribed N

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Göbel, Hartmut, Axel Heinze, Katja Heinze-Kuhn, and Vera Zumbroich. "Triptan plus NSAID with a long half-life: increase of efficacy and reduction of headache recurrence in acute migraine." In The Triptans: Novel Drugs for Migraine. Oxford University PressNew York, NY, 2001. http://dx.doi.org/10.1093/oso/9780192632142.003.0046.

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Abstract Triptans have been proved to be effective and well-tolerated drugs for the treatment of acute migraine attacks. Whereas the onset of action is rapid, however, a certain percentage of patients—ranging between 20% and 40% in studies depending on the kind of triptan used—report headache recurrence1,2. Headache recurrence is defined as a return of migraine headache of at least moderate intensity within 24 h of the first—effective—treatment of a migraine attack.

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Gómez-Escudero, Octavio. "Drug-Related Enteropathy." In Diarrhea - Novel Advances and Future Perspectives in the Etiological Diagnosis and Management [Working Title]. IntechOpen, 2022. http://dx.doi.org/10.5772/intechopen.103734.

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Over 700 drugs have been implicated as cause of chronic diarrhea and potential enteral damage. Pathophysiologic mechanisms include intrinsic malabsorption as their main mode of action (i.e., acarbose or orlistat), increased risk of microscopic colitis/enteritis (proton-pump inhibitors (PPI), non-steroidal anti-inflammatory drugs (NSAID), selective serotonin reuptake inhibitors (SSRI)), dysbiosis (antibiotics, metformin, PPI), and microscopic or overt enteropathy (angiotensin inhibitors, antineoplastic agents, targeted therapy and check-point inhibitors). According to type, diarrhea can be mala

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Konferenzberichte zum Thema "Novel NSAID"

Chan, Daniel C., Lajos Gera, Zhiyong Zhang, et al. "Abstract 1941: Development of novel NSAID conjugated molecules for lung cancer therapy." In Proceedings: AACR 103rd Annual Meeting 2012‐‐ Mar 31‐Apr 4, 2012; Chicago, IL. American Association for Cancer Research, 2012. http://dx.doi.org/10.1158/1538-7445.am2012-1941.

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Ramos-Inza, Sandra, Ignacio Encío, Arun K. Sharma, Carmen Sanmartín, and Daniel Plano. "Abstract 1287: Novel NSAID derivatives containing selenium as potent cytotoxic agents against cancer." In Proceedings: AACR Annual Meeting 2021; April 10-15, 2021 and May 17-21, 2021; Philadelphia, PA. American Association for Cancer Research, 2021. http://dx.doi.org/10.1158/1538-7445.am2021-1287.

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Plano, Daniel, Deepkamal Karelia, Manoj Pandey, Shantu Amin, and Arun K. Sharma. "Abstract 5553: Development of novel selenium-NSAIDs as potential cancer therapeutics." In Proceedings: AACR 104th Annual Meeting 2013; Apr 6-10, 2013; Washington, DC. American Association for Cancer Research, 2013. http://dx.doi.org/10.1158/1538-7445.am2013-5553.

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Cho, Dong Ha, Ki Kwang Oh, and Md Adnan. "Potential non-steroidal anti-inflammatory drugs (NSAIDs) and novel mechanism insights against COVID-19 through network pharmacology." In 6th International Electronic Conference on Medicinal Chemistry. MDPI, 2020. http://dx.doi.org/10.3390/ecmc2020-07362.

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Patil, Vipul M., and Harinath N. More. "Enlarging the NSAIDs Family: Molecular Docking of Designed Pyrazole and Oxadiazole Derivatives as Novel Anti-Inflammatory Agents." In IECBM 2022. MDPI, 2022. http://dx.doi.org/10.3390/iecbm2022-13390.

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Nagaraj, Anil Belur, QuanQiu Wang, Olga Kovalenko, et al. "Abstract NTOC-081: PHARMACOLOGIC ANALYSIS OF HIGH–GRADE SEROUS OVARIAN CANCER TCGA DATASET IDENTIFIES A NOVEL CHEMO–ADJUVANT ROLE FOR NON–STEROIDAL ANTI–INFLAMMATORY DRUGS (NSAIDS)." In Abstracts: 11th Biennial Ovarian Cancer Research Symposium; September 12-13, 2016; Seattle, WA. American Association for Cancer Research, 2017. http://dx.doi.org/10.1158/1557-3265.ovcasymp16-ntoc-081.

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