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Auswahl der wissenschaftlichen Literatur zum Thema „Placenta – Physiologie“
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Zeitschriftenartikel zum Thema "Placenta – Physiologie"
Taher, Shèdy, Yamilette Borja, Lucía Cabanela, Vincent J. Costers, Morgan Carson-Marino, Julie C. Bailes, Biswadeep Dhar et al. „Cholecystokinin, gastrin, cholecystokinin/gastrin receptors, and bitter taste receptor TAS2R14: trophoblast expression and signaling“. American Journal of Physiology-Regulatory, Integrative and Comparative Physiology 316, Nr. 5 (01.05.2019): R628—R639. http://dx.doi.org/10.1152/ajpregu.00153.2018.
Der volle Inhalt der QuelleLien, Yu-Chin, Zhe Zhang, Yi Cheng, Erzsebet Polyak, Laura Sillers, Marni J. Falk, Harry Ischiropoulos, Samuel Parry und Rebecca A. Simmons. „Human Placental Transcriptome Reveals Critical Alterations in Inflammation and Energy Metabolism with Fetal Sex Differences in Spontaneous Preterm Birth“. International Journal of Molecular Sciences 22, Nr. 15 (23.07.2021): 7899. http://dx.doi.org/10.3390/ijms22157899.
Der volle Inhalt der QuelleOsifo, E. O., und V. C. Ezeuko. „Histological Assessment of Placental Development Following Intrauterine Exposure to Caffeine in Adult Wistar Rats“. Journal of Applied Sciences and Environmental Management 28, Nr. 4 (29.04.2024): 1115–20. http://dx.doi.org/10.4314/jasem.v28i4.11.
Der volle Inhalt der QuelleRampon, Christine, Stéphanie Bouillot, Adriana Climescu-Haulica, Marie-Hélène Prandini, Francine Cand, Yves Vandenbrouck und Philippe Huber. „Protocadherin 12 deficiency alters morphogenesis and transcriptional profile of the placenta“. Physiological Genomics 34, Nr. 2 (Juli 2008): 193–204. http://dx.doi.org/10.1152/physiolgenomics.00220.2007.
Der volle Inhalt der QuelleSelvaratnam, Johanna, Haiyan Guan, James Koropatnick und Kaiping Yang. „Metallothionein-I- and -II-deficient mice display increased susceptibility to cadmium-induced fetal growth restriction“. American Journal of Physiology-Endocrinology and Metabolism 305, Nr. 6 (15.09.2013): E727—E735. http://dx.doi.org/10.1152/ajpendo.00157.2013.
Der volle Inhalt der QuelleFlores-Pliego, Arturo, Jael Miranda, Sara Vega-Torreblanca, Yolotzin Valdespino-Vázquez, Cecilia Helguera-Repetto, Aurora Espejel-Nuñez, Héctor Borboa-Olivares et al. „Molecular Insights into the Thrombotic and Microvascular Injury in Placental Endothelium of Women with Mild or Severe COVID-19“. Cells 10, Nr. 2 (10.02.2021): 364. http://dx.doi.org/10.3390/cells10020364.
Der volle Inhalt der QuelleVaughan, Owen R., Fredrick Thompson, Ramón A. Lorca, Colleen G. Julian, Theresa L. Powell, Lorna G. Moore und Thomas Jansson. „Effect of high altitude on human placental amino acid transport“. Journal of Applied Physiology 128, Nr. 1 (01.01.2020): 127–33. http://dx.doi.org/10.1152/japplphysiol.00691.2019.
Der volle Inhalt der QuelleAssad, R. S., F. Y. Lee und F. L. Hanley. „Placental compliance during fetal extracorporeal circulation“. Journal of Applied Physiology 90, Nr. 5 (01.05.2001): 1882–86. http://dx.doi.org/10.1152/jappl.2001.90.5.1882.
Der volle Inhalt der QuelleShanes, Elisheva D., Leena B. Mithal, Sebastian Otero, Hooman A. Azad, Emily S. Miller und Jeffery A. Goldstein. „Placental Pathology in COVID-19“. American Journal of Clinical Pathology 154, Nr. 1 (22.05.2020): 23–32. http://dx.doi.org/10.1093/ajcp/aqaa089.
Der volle Inhalt der QuelleTissot van Patot, M. C., J. Bendrick-Peart, V. E. Beckey, N. Serkova und L. Zwerdlinger. „Greater vascularity, lowered HIF-1/DNA binding, and elevated GSH as markers of adaptation to in vivo chronic hypoxia“. American Journal of Physiology-Lung Cellular and Molecular Physiology 287, Nr. 3 (September 2004): L525—L532. http://dx.doi.org/10.1152/ajplung.00203.2003.
Der volle Inhalt der QuelleDissertationen zum Thema "Placenta – Physiologie"
Ayache, Mohammad. „Application des réseaux de neurones à la classification automatisée des grades placentaires“. Tours, 2007. http://www.theses.fr/2007TOUR3315.
Der volle Inhalt der QuelleThe placenta is a temporary organ joins the mother and the fœtus, which transfers oxygen from the mother to the foetus, allows the evacuation of the carbon dioxide and the products of foetus metabolism. The goal of our work is to study the transfer function of placental development using ultrasound images. A new approach is developed during this work to classify the placental development by image processing techniques based on supervised neural network. The realized model by the wavelet transform based on MLP neural network, represents an effective tool answering our criteria and adapted to our applications concerning the study of placental maturation. The realized model application in the event of placental image processing opens interesting doors in terms of placental grades classification in order to identify the stages of maturation, authorizing the definition of a normal maturation and an abnormal maturation
Routhier, Catherine. „Étude de la voie des polyols dans le placenta en prééclampsie“. Thesis, Université Laval, 2014. http://www.theses.ulaval.ca/2014/30339/30339.pdf.
Der volle Inhalt der QuellePreeclampsia (PE) is a complex obstetrical pathology associated to a defective placentation. The abnormally developed placenta is believed to release factors causing a maternal endothelial dysfunction. We hypothesized that an accumulation of sorbitol, a hyperosmotic sugar produced through the polyol pathway, could induce the release by the placenta of sFlt-1, an antagonist of the angiogenic factor VEGF in PE. We compared mRNA and protein expression levels of AKR1B1 and SORD, the two enzymes of the polyol pathway, in placentas from normotensive (control) and PE pregnancies by quantitative RT-PCR and immunoblotting respectively. Then, we localized the two enzymes by immunohistochemistry. Our results suggest that polyol pathway is altered in amniochorionic membranes from PE pregnancies, and that this phenomenon would promote sorbitol accumulation at the foeto-maternal interface.
Beauchemin, David. „Étude de la synthèse des prostanoïdes vasoactifs et de leurs récepteurs dans le placenta en prééclampsie“. Thesis, Université Laval, 2011. http://www.theses.ulaval.ca/2011/27970/27970.pdf.
Der volle Inhalt der QuelleLe, Duc Kévin. „Physiologie des échanges gazeux et de l'hémodynamique transplacentaire lors d'une réanimation à cordon intact : modèle expérimental d'agneau porteur de hernie diaphragmatique“. Electronic Thesis or Diss., Université de Lille (2022-....), 2024. http://www.theses.fr/2024ULILS079.
Der volle Inhalt der QuelleBirth is a critical period during which numerous mechanisms are engaged to enable the transition from fetal to extrauterine life. Each year, due to poor adaptation to this transition and the persistence of elevated pulmonary vascular resistance, 1 million newborns die within the first 24 hours of life. Ten percent of newborns require medical assistance in the delivery room. Delayed umbilical cord clamping, between 60 and 180 seconds after birth, is now recommended in all situations where the newborn, whether full-term or premature, adapts well to the new environment. This practice notably reduces the risk of iron deficiency anemia in the first months of life.Congenital diaphragmatic hernia (CDH) is a cardiopulmonary malformation caused by a defect in the closure of the diaphragm, leading to high mortality and impairing adaptation to extrauterine life. In delivery room resuscitation scenarios, the lack of physiological and clinical data has not yet allowed for the recommendation of maintaining feto-placental circulation alongside the initiation of resuscitation.In this thesis, we hypothesize that the placenta could contribute to oxygenation and decarboxylation of the newborn until the cardio-pulmonary circulation is established. The aim of this work is to study the physiology of hemodynamics and transplacental gas exchange during intact cord resuscitation (ICR) in a healthy lamb model and in a lamb model with CDH. The specific objectives were: (1) to present the clinical study “CHIC” evaluating the impact of ICR in newborns with CDH; (2) to establish an experimental lamb model of congenital diaphragmatic hernia; (3) to explore the feasibility and maximum duration of intact cord resuscitation in this model; and (4) to study the evolution of hemodynamics and transplacental gas exchange during ICR in both healthy and CDH lamb models.We demonstrated that feto-placental hemodynamics (umbilical venous flow, transplacental vascular resistance) remained stable up to one hour after the initiation of ICR. In the lamb model with diaphragmatic hernia, where the pulmonary exchange system cannot adequately increase arterial partial oxygen pressure (PaO2), the placenta provided sufficient oxygenation and decarboxylation throughout the resuscitation, with stable placental oxygen delivery for one hour (2.7 [2.2-3.3] ml/kg/min). Conversely, in the physiological model, maintaining placental circulation was associated with a 20% decrease in systemic arterial pressure compared to the CDH group (p<0.05). The increase in PaO2 in this group was associated with a decrease in placental oxygen delivery. Cord clamping in this group led to an increase in PaO2 and a decrease in carbon dioxide levels. These findings provide an essential physiological basis for the practice of intact cord resuscitation and highlight the importance of individualized resuscitation strategies based on specific clinical conditions
Bartho, Lucy A. „Mitochondrial Alterations Through Gestation and in Placental Pathologies“. Thesis, Griffith University, 2021. http://hdl.handle.net/10072/411262.
Der volle Inhalt der QuelleThesis (PhD Doctorate)
Doctor of Philosophy (PhD)
School of Pharmacy & Med Sci
Griffith Health
Full Text
Corbel, Tanguy. „Mécanismes toxicocinétiques impliqués dans l'exposition foetale au Bisphénol A“. Thesis, Toulouse, INSA, 2013. http://www.theses.fr/2013ISAT0034/document.
Der volle Inhalt der QuelleBisphenol A (BPA) an endocrine disruptor interfering with developmental processes in rodents, raises the question of risk for human health related to fetal exposure to BPA. The goal of this work was to determine the toxicokinetic mechanisms involved in fetal exposure to BPA. The disposition of BPA and its metabolites in the maternal-placental-fetal unit in an in vivo ovine model enabled us to identify the placental transfer and the fetal-placental metabolism as the major determining factors of fetal exposure to BPA. Bidirectional placental transfer of BPA occurs by passive diffusion leading to a ratio of free BPA between the fetal and maternal plasma concentrations of about 1. By contrast, the permeability of BPA-G is very limited, particularly in materno-to-fetal direction. The hepatic conjugation activities were very low in ovine fetus at an early stage of development and increased throughout pregnancy. Hydrolysis of BPA conjugates observed ex vivo into fetal ovine gonads could lead to local exposure to native BPA. Altogether, these results suggest that the early stage of pregnancy is a critical window of exposure for the developing fetus
Rao, M. Rekha. „Functional Differentiation Of The Human Placenta : Insights From The Expression Of Two Developmentally - Regulated Genes“. Thesis, Indian Institute of Science, 2000. https://etd.iisc.ac.in/handle/2005/177.
Der volle Inhalt der QuelleRao, M. Rekha. „Functional Differentiation Of The Human Placenta : Insights From The Expression Of Two Developmentally - Regulated Genes“. Thesis, Indian Institute of Science, 2000. http://hdl.handle.net/2005/177.
Der volle Inhalt der QuelleGriffith, Oliver William. „Mechanisms of placental evolution: the genetics and physiology of pregnancy in lizards“. Thesis, The University of Sydney, 2015. http://hdl.handle.net/2123/13600.
Der volle Inhalt der QuelleJokhi, Percy Parvez. „Cytokines and their receptors in human placental implantation“. Thesis, University of Cambridge, 1994. http://ethos.bl.uk/OrderDetails.do?uin=uk.bl.ethos.338321.
Der volle Inhalt der QuelleBücher zum Thema "Placenta – Physiologie"
Olga, Genbačev, Klopper Arnold und Beaconsfield Rebecca, Hrsg. Placenta as a model and a source. New York: Plenum Press, 1989.
Den vollen Inhalt der Quelle findenSelʹkov, S. A. Plat︠s︡entarnye makrofagi. Moskva: Tov-vo nauch. izd. KMK, 2007.
Den vollen Inhalt der Quelle findenLise, Cédard, und Firth Anthony, Hrsg. Placental signals: Autocrine and paracine control of pregnancy. Rochester, N.Y: University of Rochester Press, 1992.
Den vollen Inhalt der Quelle finden1946-, Miller Richard K., Thiede Henry A und Rochester Trophoblast Conference. (11th : 1988 : Rochester, N.Y.), Hrsg. Molecular biology and cell regulation of the placenta. Rochester, N.Y: Verav Medical Publishers, 1991.
Den vollen Inhalt der Quelle findenE, Rice Gregory, und Brennecke S. P, Hrsg. Molecular aspects of placental and fetal membrane autacoids. Boca Raton: CRC Press, 1993.
Den vollen Inhalt der Quelle finden1932-, Battaglia Frederick C., Nestlé Nutrition Services und Nestlé Nutrition Workshop (39th : 1996 : East Sussex, England), Hrsg. Placental function & fetal nutrition. [Vevey, Switzerland]: Nestlé Nutrition Services, 1997.
Den vollen Inhalt der Quelle finden1940-, Hata Toshio, Takayama Masaomi 1938-, Taki Ichiro 1919-, Foidart Jean-Michel 1949-, Japanese Placenta Group Meeting und European Placenta Group Meeting, Hrsg. Placental molecules in hemodynamics, transport, and cellular regulation. Rochester, NY: University of Rochester Press, 1997.
Den vollen Inhalt der Quelle findenL, Power Michael, und Schulkin Jay, Hrsg. Birth, distress, and disease: Placental-brain interactions. Cambridge: Cambridge University Press, 2005.
Den vollen Inhalt der Quelle findenWooding, F. B. P. Comparative placentation: Structures, functions and evolution. Berlin: Springer, 2008.
Den vollen Inhalt der Quelle findenNadirashvili, S. A. Iskusstvennoe plat͡s︡entarnoe krovoobrashchenie ploda. Tbilisi: "Met͡s︡niereba", 1986.
Den vollen Inhalt der Quelle findenBuchteile zum Thema "Placenta – Physiologie"
Ebbesen, P. „Placenta physiology“. In Congenital toxoplasmosis, 27–35. Paris: Springer Paris, 2000. http://dx.doi.org/10.1007/978-2-8178-0847-5_3.
Der volle Inhalt der QuelleMcNamara, Jennifer M., und Helen H. Kay. „Placental Hormones: Physiology, Disease, and Prenatal Diagnosis“. In The Placenta, 57–65. Oxford, UK: Wiley-Blackwell, 2011. http://dx.doi.org/10.1002/9781444393927.ch8.
Der volle Inhalt der QuelleDancis, Joseph, und Henning Schneider. „Physiology of the Placenta“. In Human Growth, 221–44. Boston, MA: Springer US, 1986. http://dx.doi.org/10.1007/978-1-4613-2101-9_12.
Der volle Inhalt der QuelleKam, Peter, Ian Power, Michael J. Cousins und Philip J. Siddal. „Physiology of the Placenta“. In Principles of Physiology for the Anaesthetist, 463–66. Fourth edition. | Boca Raton : CRC Press, Taylor & Francis Group, 2020.: CRC Press, 2020. http://dx.doi.org/10.1201/9780429288210-73.
Der volle Inhalt der QuelleKarck, U., und M. Breckwoldt. „Functions of the Placenta“. In Comprehensive Human Physiology, 2291–305. Berlin, Heidelberg: Springer Berlin Heidelberg, 1996. http://dx.doi.org/10.1007/978-3-642-60946-6_118.
Der volle Inhalt der QuelleMounce, Ginny. „The placenta and developing fetus“. In The Anatomy and Physiology Textbook for Midwives, 85–96. London: Routledge, 2025. https://doi.org/10.4324/9781003227571-10.
Der volle Inhalt der QuelleButt, W. R., T. Chard und R. K. Iles. „Hormones of the Placenta: hCG and hPL“. In Marshall’s Physiology of Reproduction, 461–534. Dordrecht: Springer Netherlands, 1994. http://dx.doi.org/10.1007/978-94-011-1286-4_5.
Der volle Inhalt der QuelleLongo, Lawrence D. „Some Aspects of the Physiology of the Placenta“. In The Rise of Fetal and Neonatal Physiology, 137–65. New York, NY: Springer New York, 2013. http://dx.doi.org/10.1007/978-1-4614-7921-5_8.
Der volle Inhalt der QuelleLongo, Lawrence D. „Some Aspects of the Physiology of the Placenta“. In The Rise of Fetal and Neonatal Physiology, 153–94. New York, NY: Springer New York, 2018. http://dx.doi.org/10.1007/978-1-4939-7483-2_8.
Der volle Inhalt der QuelleLongo, Lawrence D. „Some Aspects of Endocrinology of the Placenta“. In The Rise of Fetal and Neonatal Physiology, 195–215. New York, NY: Springer New York, 2018. http://dx.doi.org/10.1007/978-1-4939-7483-2_9.
Der volle Inhalt der QuelleKonferenzberichte zum Thema "Placenta – Physiologie"
Thompson, Jaylyn D., Jenna Zappetti und Clarence Julian II Clark. „Management Strategies for Traumatic Injuries in Pregnant Women: A Comprehensive Literature Review“. In 28th Annual Rowan-Virtua Research Day. Rowan University Libraries, 2024. http://dx.doi.org/10.31986/issn.2689-0690_rdw.stratford_research_day.122_2024.
Der volle Inhalt der QuelleKh ALJUBOURY, Maha, und khawla E ALJUBOURY. „EFFECT OF DEPAKINE ON SOME PHYSIOLOGICAL CHANGES OF PREGNANT MICE AND THEIR EMBRYOS USING FOLIC ACID“. In VII. INTERNATIONAL SCIENTIFIC CONGRESSOF PURE,APPLIEDANDTECHNOLOGICAL SCIENCES. Rimar Academy, 2023. http://dx.doi.org/10.47832/minarcongress7-24.
Der volle Inhalt der QuelleBerichte der Organisationen zum Thema "Placenta – Physiologie"
Bazer, Fuller W., Arieh Gertler und Elisha Gootwine. Role of Placental Lactogen in Sheep. United States Department of Agriculture, Januar 2001. http://dx.doi.org/10.32747/2001.7574339.bard.
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