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Thematische Bibliographien / Placenta; transporter; transportér

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Auswahl der wissenschaftlichen Literatur zum Thema „Placenta; transporter; transportér“

Autor: Grafiati

Veröffentlicht am 28. Juni 2021

Zuletzt aktualisiert am 29. Juli 2025

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Zeitschriftenartikel zum Thema "Placenta; transporter; transportér"

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Granitzer, Sebastian, Isabella Ellinger, Rumsha Khan, et al. "In vitro function and in situ localization of Multidrug Resistance-associated Protein (MRP)1 (ABCC1) suggest a protective role against methyl mercury-induced oxidative stress in the human placenta." Archives of Toxicology 94, no. 11 (2020): 3799–817. http://dx.doi.org/10.1007/s00204-020-02900-5.

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Abstract Methyl mercury (MeHg) is an organic highly toxic compound that is transported efficiently via the human placenta. Our previous data suggest that MeHg is taken up into placental cells by amino acid transporters while mercury export from placental cells mainly involves ATP binding cassette (ABC) transporters. We hypothesized that the ABC transporter multidrug resistance-associated protein (MRP)1 (ABCC1) plays an essential role in mercury export from the human placenta. Transwell transport studies with MRP1-overexpressing Madin-Darby Canine Kidney (MDCK)II cells confirmed the function of

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Shearman, Lauren P., Alison M. McReynolds, Feng C. Zhou, and Jerrold S. Meyer. "Relationship between [125I]RTI-55-labeled cocaine binding sites and the serotonin transporter in rat placenta." American Journal of Physiology-Cell Physiology 275, no. 6 (1998): C1621—C1629. http://dx.doi.org/10.1152/ajpcell.1998.275.6.c1621.

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We investigated the characteristics of cocainelike binding sites in rat placenta using [125I]RTI-55. [3H]paroxetine binding and immunocytochemical staining for serotonin [5-hydroxytryptamine (5-HT)] and for the 5-HT transporter were also used to obtain evidence for rat placental 5-HT uptake. [125I]RTI-55 saturation analyses with membranes from normal gestational day 20 placentas yielded curvilinear Scatchard plots that were resolved into high- and low-affinity components (mean dissociation constants of 0.29 and 7.9 nM, respectively). Drug competition studies with various monoamine uptake inhib

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do Imperio, Guinever Eustaquio, Enrrico Bloise, Mohsen Javam, et al. "Chorioamnionitis Induces a Specific Signature of Placental ABC Transporters Associated with an Increase of miR-331-5p in the Human Preterm Placenta." Cellular Physiology and Biochemistry 45, no. 2 (2018): 591–604. http://dx.doi.org/10.1159/000487100.

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Background/Aims: The ATP-binding cassette (ABC) transporters mediate drug biodisposition and immunological responses in the placental barrier. In vitro infective challenges alter expression of specific placental ABC transporters. We hypothesized that chorioamnionitis induces a distinct pattern of ABC transporter expression. Methods: Gene expression of 50 ABC transporters was assessed using TaqMan® Human ABC Transporter Array, in preterm human placentas without (PTD; n=6) or with histological chorioamnionitis (PTDC; n=6). Validation was performed using qPCR, immunohistochemistry and Western blo

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Xu, Jie, Jiao Wang, Yang Cao, et al. "Downregulation of Placental Amino Acid Transporter Expression and mTORC1 Signaling Activity Contributes to Fetal Growth Retardation in Diabetic Rats." International Journal of Molecular Sciences 21, no. 5 (2020): 1849. http://dx.doi.org/10.3390/ijms21051849.

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Alterations in placental transport may contribute to abnormal fetal intrauterine growth in pregnancies complicated by diabetes, but it is not clear whether the placental amino acid transport system is altered in diabetic pregnancies. We therefore studied the changes in the expressions of placental amino acid transporters in a rat model of diabetes induced by streptozotocin, and tested the effects of hyperglycemia on trophoblast amino acid transporter in vitro. Our results showed that the expressions for key isoforms of system L amino acid transporters were significantly reduced in the placenta

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Roos, S., Y. Kanai, P. D. Prasad, T. L. Powell, and T. Jansson. "Regulation of placental amino acid transporter activity by mammalian target of rapamycin." American Journal of Physiology-Cell Physiology 296, no. 1 (2009): C142—C150. http://dx.doi.org/10.1152/ajpcell.00330.2008.

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The activity of placental amino acid transporters is decreased in intrauterine growth restriction (IUGR), but the underlying regulatory mechanisms have not been established. Inhibition of the mammalian target of rapamycin (mTOR) signaling pathway has been shown to decrease the activity of the system L amino acid transporter in human placental villous fragments, and placental mTOR activity is decreased in IUGR. In the present study, we used cultured primary trophoblast cells to study mTOR regulation of placental amino acid transporters in more detail and to test the hypothesis that mTOR alters

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Cleal, J. K., P. E. Day, C. L. Simner, et al. "Placental amino acid transport may be regulated by maternal vitamin D and vitamin D-binding protein: results from the Southampton Women's Survey." British Journal of Nutrition 113, no. 12 (2015): 1903–10. http://dx.doi.org/10.1017/s0007114515001178.

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Both maternal 25-hydroxyvitamin D (25(OH)D) concentrations during pregnancy and placental amino acid transporter gene expression have been associated with development of the offspring in terms of body composition and bone structure. Several amino acid transporter genes have vitamin D response elements in their promoters suggesting the possible linkage of these two mechanisms. We aimed to establish whether maternal 25(OH)D and vitamin D-binding protein (VDBP) levels relate to expression of placental amino acid transporters. RNA was extracted from 102 placental samples collected in the Southampt

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Ericsson, Anette, Bengt Hamark, Nina Jansson, Bengt R. Johansson, Theresa L. Powell, and Thomas Jansson. "Hormonal regulation of glucose and system A amino acid transport in first trimester placental villous fragments." American Journal of Physiology-Regulatory, Integrative and Comparative Physiology 288, no. 3 (2005): R656—R662. http://dx.doi.org/10.1152/ajpregu.00407.2004.

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Alterations in placental nutrient transfer have been implicated in fetal growth abnormalities. In pregnancies complicated by diabetes and accelerated fetal growth, upregulations of glucose transporter 1 (GLUT1) and amino acid transporter system A have been shown in the syncytiotrophoblast of term placenta. In contrast, intrauterine growth restriction is associated with a downregulation of placental system A transporters. However, underlying mechanisms of transporter regulation are poorly understood, particularly in early pregnancy. In this study, hormonal regulation of placental glucose and sy

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Taggi, Valerio, Mario Riera Romo, Micheline Piquette-Miller, Henriette E. Meyer zu Schwabedissen, and Sibylle Neuhoff. "Transporter Regulation in Critical Protective Barriers: Focus on Brain and Placenta." Pharmaceutics 14, no. 7 (2022): 1376. http://dx.doi.org/10.3390/pharmaceutics14071376.

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Drug transporters play an important role in the maintenance of chemical balance and homeostasis in different tissues. In addition to their physiological functions, they are crucial for the absorption, distribution, and elimination of many clinically important drugs, thereby impacting therapeutic efficacy and toxicity. Increasing evidence has demonstrated that infectious, metabolic, inflammatory, and neurodegenerative diseases alter the expression and function of drug transporters. However, the current knowledge on transporter regulation in critical protective barriers, such as the brain and pl

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Chen, Xin, Zsuzsanna Gáborik, Qingcheng Mao та Jashvant D. Unadkat. "Are Δ9-Tetrahydrocannabinol and Its Major Metabolites Substrates or Inhibitors of Placental or Human Hepatic Drug Solute-Carrier Transporters?" International Journal of Molecular Sciences 25, № 22 (2024): 12036. http://dx.doi.org/10.3390/ijms252212036.

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Δ9-Tetrahydrocannabinol (THC) is the primary psychoactive component of cannabis which is being increasingly consumed by pregnant people. In humans, THC is sequentially metabolized in the liver to its circulating metabolites 11-hydroxy-THC (11-OH-THC, psychoactive) and 11-nor-9-carboxy-THC (THC-COOH, non-psychoactive). Human and macaque data show that fetal exposure to THC is considerably lower than the corresponding maternal exposure. Through perfused human placenta studies, we showed that this is due to the active efflux of THC (fetal-to-maternal) by a placental transporter(s) other than P-gl

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Audette, Melanie C., John R. G. Challis, Rebecca L. Jones, Colin P. Sibley, and Stephen G. Matthews. "Synthetic Glucocorticoid Reduces Human Placental System A Transport in Women Treated With Antenatal Therapy." Journal of Clinical Endocrinology & Metabolism 99, no. 11 (2014): E2226—E2233. http://dx.doi.org/10.1210/jc.2014-2157.

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Context: Synthetic glucocorticoids (sGCs) are routinely given to women with threatened preterm labor and have been linked to fetal growth restriction and developmental programming. Reductions in fetal growth are likely to be mediated by placental dysfunction, including altered nutrient transport. sGCs modify the system A neutral amino acid transporter in vitro, but there are no in vivo comparable data in human placenta. Objective: Because ∼30% of women who receive sGCs carry to term, our objective was to examine the short- and longer-term consequences of antenatal sGCs on placental system A tr

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Dissertationen zum Thema "Placenta; transporter; transportér"

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Russi, Rachel Mary. "The molecular regulation of placental zinc transporters." Thesis, University of Newcastle Upon Tyne, 2004. http://ethos.bl.uk/OrderDetails.do?uin=uk.bl.ethos.402646.

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Peres, Kenya Costa [UNESP]. "Transporte placentário via cavéola na placenta de bovinos clonados e transgênicos." Universidade Estadual Paulista (UNESP), 2014. http://hdl.handle.net/11449/115903.

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Made available in DSpace on 2015-03-03T11:52:41Z (GMT). No. of bitstreams: 0 Previous issue date: 2014-02-26Bitstream added on 2015-03-03T12:06:08Z : No. of bitstreams: 1 000809964.pdf: 1108463 bytes, checksum: 43fa89dc624ca99b382ecaa200b42518 (MD5)<br>A utilização da transgenia com a proteína fluorescente verde (GFP) como marcador de células de origem fetal nas placentas de clones bovinos servirá de modelo inédito para estudo morfofisiológico e imunológico da interação materno-fetal, visto que possibilitará o seu mapeamento, diferenciando as células fetais das maternas. Tal modelo terá apli

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Peres, Kenya Costa. "Transporte placentário via cavéola na placenta de bovinos clonados e transgênicos/." Dracena, 2014. http://hdl.handle.net/11449/115903.

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Orientador: Flavia Thomaz Verechia Pereira<br>Banca: Cristiana Andrighetto<br>Banca: Vanessa Gomes Ueno<br>Resumo: A utilização da transgenia com a proteína fluorescente verde (GFP) como marcador de células de origem fetal nas placentas de clones bovinos servirá de modelo inédito para estudo morfofisiológico e imunológico da interação materno-fetal, visto que possibilitará o seu mapeamento, diferenciando as células fetais das maternas. Tal modelo terá aplicação direta, principalmente porque estes são animais que apresentam problemas em relação ao seu desenvolvimento. Com o auxílio deste modelo

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Evseenko, Denis. "Regulation and functional significance of ATP binding cassette transporters in human placenta." Thesis, University of Auckland, 2008. http://hdl.handle.net/2292/2348.

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The aim of this project was to study ATP binding cassette (ABC) transporters in the human placenta, in particular their regulation and role in trophoblast differentiation and survival. The presence and localisation of four major placental drug transporters, multidrug resistance gene product 1 and 3 (MDR1 and 3)/ABC subfamily B members 1 and 4 (ABCB1 and 4), multidrug resistance associated proteins 1 and 2 (MRP1 and 2)/ABCC1 and 2 and breast cancer resistance protein (BCRP)/ABCG2 was initially studied in term human placenta, cultured primary trophoblast and BeWo and Jar trophoblast-like cell li

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Hirst, Chloe. "Placental taurine transport in pre-eclampsia." Thesis, University of Manchester, 2015. https://www.research.manchester.ac.uk/portal/en/theses/placental-taurine-transport-in-preeclampsia(85a6b6e1-f0be-46f4-bec4-22c03183ff19).html.

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Pre-eclampsia (PE) is a serious disease affecting approximately 5% of pregnancies per annum. The disease etiology is complex but its origin lies in abnormal placental development and function. PE is associated with inflammation, increased nitrative stress and abnormal renewal of syncytiotrophoblast (STB), the transporting epithelium of the placenta. STB is renewed by cytotrophoblasts (CTBs) that proliferate, differentiate and fuse with STB and this is balanced by apoptosis. The amino acid taurine facilitates proliferation, differentiation and apoptosis in non-placental tissues. Taurine is also

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Taylor, Louise. "Comparative analyses of ABC transporters and metabolising enzymes in human and rat placental models." Thesis, University of Manchester, 2013. https://www.research.manchester.ac.uk/portal/en/theses/comparative-analyses-of-abc-transporters-and-metabolising-enzymes-in-human-and-rat-placental-models(3daff296-0364-4e4b-89f8-337dac6dbf10).html.

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The placenta provides a protective barrier for the developing foetus during gestation. Physiological barriers including the placenta, liver, kidney, intestine and blood-brain barrier are known to express ATP-Binding cassette transporters (ABC transporters) and metabolising enzymes. These specialised proteins have the ability to transport or metabolise xenobiotics. There is evidence to suggest that ABC transporters and metabolising enzymes are located at the interface between the maternal and foetal blood supplies (a cell layer referred to as the syncytiotrophoblast) and therefore may help prot

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Wang, Meng. "Enhancement of the Placental Transmission of Lopinavir Using a Transporter Targeted Prodrug Strategy." VCU Scholars Compass, 2015. http://scholarscompass.vcu.edu/etd/3973.

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Lopinavir (LPV) is a potent protease inhibitor specific for HIV-1. However, LPV has poor placental penetration due to substrate activity for efflux transporter by P-glycoprotein (P-gp). Since fatty acid transporters are highly expressed in the placenta during pregnancy, we designed fatty acid ester prodrug of lopinavir as substrates of fatty acid transporter in order to improve their uptake into placenta. Seven dicarboxylic acid esters of lopinavir have been made in our lab. The structures were characterized by 1H-NMR, 13C-NMR, LC-MS/MS, HRMS, IR and melting points. After making the prodrugs,

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Jobarteh, Modou Lamin. "The effect of prenatal nutritional intervention on placental nutrient transporter expression and feto-placental outcome in rural Gambian women." Thesis, University of Aberdeen, 2015. http://digitool.abdn.ac.uk:80/webclient/DeliveryManager?pid=225784.

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Iron and zinc deficiency during pregnancy is common among women in low-income nations. In such settings, prenatal nutritional intervention is encouraged to improve pregnancy outcome. The impact of the intervention on transporter proteins involved in fetal nutrient supply is unexplored. This study investigated gene expression of some transporter proteins involved in fetal nutrient supply in the placenta. In a trial in rural Gambia, pregnant women at <20weeks of gestation were randomised to 4 nutritional intervention arms: i) Iron and folic acid (FeFol), representing the usual care ii) Multiple

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Sivaprasadarao, A. "Studies on the transport and uptake of vitamin A by placenta." Thesis, University of Leeds, 1987. http://ethos.bl.uk/OrderDetails.do?uin=uk.bl.ethos.384722.

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Vasilopoulou, Elisavet. "The role of thyroid hormones in placental development and the importance of the thyroid hormone transporter MCT8." Thesis, University of Birmingham, 2010. http://etheses.bham.ac.uk//id/eprint/1146/.

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Thyroid hormones (THs) are important for fetoplacental development. Human intrauterine growth restriction (IUGR) is associated with malplacentation and reduced fetal circulating concentration of THs. The expression of the plasma membrane TH transporters MCT8, MCT10, LAT1, LAT2, OATP1A2 and OATP4A1 was characterised in human placental biopsies across gestation. The protein expression of MCT8 was increased in samples from severe IUGR compared with normal pregnancies. In vitro, triiodothyronine (T\(_3\)) decreased survival and increased apoptosis of IUGR compared with normal cytotrophoblasts, whi

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Bücher zum Thema "Placenta; transporter; transportér"

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1932-, Battaglia Frederick C., Nestlé Nutrition Services, and Nestlé Nutrition Workshop (39th : 1996 : East Sussex, England), eds. Placental function & fetal nutrition. Nestlé Nutrition Services, 1997.

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(Editor), Paul Bischoff, Henning Schneider (Editor), and Rudolf Leiser (Editor), eds. Fetal Growth and the Placenta-From Implantation to Delivery: From Implantation to Delivery (Trophoblast Research, Vol 7). Univ of Rochester Pr, 1993.

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Moléculas que participan en el transporte de hierro materno-fetal : importancia del receptor 1 de transferrina y de la ferroportina en la placenta humana. Editorial Académica Española, 2012.

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Allegaert, Karel, and Kristel Van Calsteren. Maternal, fetal, and neonatal pharmacokinetics. Oxford University Press, 2016. http://dx.doi.org/10.1093/med/9780198713333.003.0005.

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Most drugs are not thoroughly evaluated for use during pregnancy, delivery, or postpartum (e.g. breastfeeding). The same holds true for early infancy, and results in extensive off-label, unlicensed pharmacotherapy in these specific subpopulations. At present, most drug labels do not contain any instructions for use during pregnancy, in infancy, or during breastfeeding, yet these are the main concerns of healthcare providers considering medical treatment. Anaesthetists commonly treat pregnant women with similar dosing regimens recommended for use in adults and subsequently titrate to effect. Th

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Malyszko, Jolanta, and Iain C. Macdougall. Iron metabolism in chronic kidney disease. Edited by David J. Goldsmith. Oxford University Press, 2015. http://dx.doi.org/10.1093/med/9780199592548.003.0125.

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While whole-body (‘absolute’) iron deficiency is common and probably increased in frequency in chronic kidney disease (CKD), functional iron deficiency is a particular problem in CKD. Absolute iron deficiency is likely to be present in advanced CKD when the ferritin falls below 100 ng/mL and the TSAT falls below 20%. Functional iron deficiency is characterized by the presence of adequate iron stores (as defined by conventional criteria), but with an inability to mobilize this iron rapidly enough to adequately support erythropoiesis with the administration of erythropoietin. Among such patients

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Buchteile zum Thema "Placenta; transporter; transportér"

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Mason, Clifford W., and Carl P. Weiner. "Placental Drug Transport." In The Placenta. Wiley-Blackwell, 2011. http://dx.doi.org/10.1002/9781444393927.ch40.

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Mao, Qingcheng, Vadivel Ganapathy, and Jashvant D. Unadkat. "Drug Transport in the Placenta." In Drug Transporters. John Wiley & Sons, Inc., 2014. http://dx.doi.org/10.1002/9781118705308.ch17.

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Gruber, Michael, Birgit Hirschmugl, Carolin Schliefsteiner, and Christian Wadsack. "Placental Function—Nutrient Transport—Gas Exchange." In The Placenta. Springer Berlin Heidelberg, 2023. http://dx.doi.org/10.1007/978-3-662-66256-4_4.

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Dubé, Evemie, Guillaume Desparois, and Julie Lafond. "Placental Lipid Transport." In Preeclampsia. Springer New York, 2017. http://dx.doi.org/10.1007/978-1-4939-7498-6_24.

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Bendon, Robert W. "Amnion Transport: Histologic Features." In Pathology of the Placenta. Springer International Publishing, 2018. http://dx.doi.org/10.1007/978-3-319-97214-5_40.

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Dicke, Jeffrey M. "Placental transport and metabolism." In Clinical Maternal-Fetal Medicine Online, 2nd ed. CRC Press, 2021. http://dx.doi.org/10.1201/9781003222590-63.

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Cetin, Irene, and Chiara Mandò. "Stable Isotope Methodologies for the Study of Transport and Metabolism In Vivo." In The Placenta. Wiley-Blackwell, 2011. http://dx.doi.org/10.1002/9781444393927.ch27.

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Xia, Qinglan, Carolyn Salafia, and Simon Morgan. "Optimal Transport and Placental Function." In Springer Proceedings in Mathematics & Statistics. Springer International Publishing, 2015. http://dx.doi.org/10.1007/978-3-319-12307-3_73.

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Maguire, D. J., R. Blums, R. Morgan, J. Collie, and G. R. Cannell. "A Placental Perfusion pO2 Logger." In Oxygen Transport to Tissue XIV. Springer US, 1992. http://dx.doi.org/10.1007/978-1-4615-3428-0_77.

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Brunette, Michèle G., Sylvie Letendre, and Serge Allard. "Phosphate Transport Through Placenta Brush Border Membrane." In Phosphate and Mineral Homeostasis. Springer US, 1986. http://dx.doi.org/10.1007/978-1-4684-5206-8_68.

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Konferenzberichte zum Thema "Placenta; transporter; transportér"

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Bukowski, Michael, Brij Singh, James Roemmich, and Kate Larson. "Lipidomic analysis of TRPC1 Ca2+-permeable channel-knock out mouse demonstrates a vital role in placental tissue sphingolipid and triacylglycerol homeostasis under high-fat diet." In 2022 AOCS Annual Meeting & Expo. American Oil Chemists' Society (AOCS), 2022. http://dx.doi.org/10.21748/tjdt4839.

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Placental function including oxygen delivery and nutrient transport are critical determinants of fetal growth, moderating the risks of obesity and metabolic diseases later in life. Previously, we demonstrated in a mouse model that parental diet and exercise play important roles in placental lipid content and inflammation. Transient receptor potential canonical channel 1 (TRPC1) is a Ca2+-permeable integral membrane protein. We have demonstrated that TRPC1 increases total body adiposity in mice by decreasing the efficacy of exercise to limit adipose accumulation under a high fat (HF) diet. Impo

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Venturi, Christinni Machado, Fernando Hartmann Barazzetti, Marcos André Schörner, et al. "Abordagem molecular para detecção de patógenos em tecido placentário humano." In XIV Congresso da Sociedade Brasileira de DST - X Congresso Brasileiro de AIDS - V Congresso Latino Americano IST/HIV/AIDS. Zeppelini Editorial e Comunicação, 2023. http://dx.doi.org/10.5327/dst-2177-8264-202335s1065.

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Introdução: Placenta é um órgão transitório que funciona como sistema de transporte de substâncias da mãe para o feto. A placentação remodela a circulação sanguínea materna, havendo contato direto com a placenta, expondo o feto a possíveis patógenos maternos. Durante a pandemia causada por SARS-CoV-2, estudou-se a possibilidade de infecção congênita em virtude da expressão dos mediadores da entrada viral em vários compartimentos da placenta. Como a placenta forma barreira entre os compartimentos fetais e maternos durante a gravidez, o cordão umbilical é a provável via de infecção materno-fetal

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Perazzolo, S., B. Hirschmugl, C. Wadsack, G. Desoye, R. M. Lewis, and B. G. Sengers. "Computational modelling of fatty acid transport in the human placenta." In 2015 37th Annual International Conference of the IEEE Engineering in Medicine and Biology Society (EMBC). IEEE, 2015. http://dx.doi.org/10.1109/embc.2015.7320262.

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Jalali, Seyedaydin, and Ponnambalam Selvaganapathy. "A TUBULAR PLACENTAL VILI BARRIER MODEL FOR TRANSPORT STUDIES." In The 28th International Conference on Miniaturized Systems for Chemistry and Life Sciences - Micro-Total Analysis Systems. Chemical and Biological Microsystems Society, 2024. https://doi.org/10.70477/xesy6031.

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Lüscher, B., D. Surbek, P. Schneider, and M. Baumann. "Placental uric acid transport system and its impact on fetal development." In 62. Kongress der Deutschen Gesellschaft für Gynäkologie und Geburtshilfe – DGGG'18. Georg Thieme Verlag KG, 2018. http://dx.doi.org/10.1055/s-0038-1671120.

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Martin, John T., and Virginia L. Ferguson. "Regional Similarities in the Mechanical Properties of the Human Umbilical Artery." In ASME 2009 Summer Bioengineering Conference. American Society of Mechanical Engineers, 2009. http://dx.doi.org/10.1115/sbc2009-206800.

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The human umbilical cord (UC) bridges the blood flow gap between baby and mother, protecting the blood supply in a way that allows the fetus freedom to move within the amniotic sac. Once the blood supply has been oxygenated by the maternal blood pool via the placenta, the umbilical vein (UV) provides a transport pathway to the fetus. Two umbilical arteries (UA) return the blood supply to the pool to eliminate CO2 and other metabolic wastes [1]. The walls of the UA’s and UV are made up of an intima composed a single layer of large, elongated endothelial cells [2], and a media composed of random

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Moulais, Tamires Lima, Vanessa Carneiro Gabriel, Gabriela Ansiliero De Lima, and Alcione Oliveira Dos Santos. "FATORES DE RISCO DA TRANSMISSÃO VERTICAL EM GESTANTES SOROPOSITIVAS PARA HIV E MEDIDAS PROFILÁTICAS." In I Congresso Brasileiro de Doenças Infectocontagiosas On-line. Revista Multidisciplinar em Saúde, 2021. http://dx.doi.org/10.51161/rems/2213.

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Introdução: A transmissão vertical refere-se a transmissão da doença, nesse caso o Vírus da Imunodeficiência Humana (HIV), da mãe para o bebê em três principais formas de transmissão: durante a gestação, no parto e na amamentação do bebê. Sendo assim, é preciso que todas as gestantes realizem testes para diagnósticos de infecções sexualmente transmissíveis (IST) para, a partir de um diagnóstico positivo, efetuar as medidas necessária para a prevenção. Objetivo: Compreender os fatores de risco da transmissão vertical em gestantes soropositivas para HIV. Metodologia: No presente estudo, foi feit

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McCarthy, P., G. Gau, and M. Shearer. "PLASMA AND LIVER LEVELS OF VITAMIN K IN THE NEWBORN." In XIth International Congress on Thrombosis and Haemostasis. Schattauer GmbH, 1987. http://dx.doi.org/10.1055/s-0038-1643607.

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Few measurements have been made of vitamin K in neonatal tissues. Using an assay based on HPLC with dual-electrode electrochemical detection we have investigated the vitamin K status of the newborn from analyses of paired cord-maternal plasma samples and liver samples obtained at post-mortem. For vitamin K1 (K1) the median value in cord plasma (16 pg/ml, range 4-45 pg/ml) in 20 babies was some 30 fold lower than that in maternal plasma (median 0.47 ng/ml , range 0.14-2.42 ng/ml). This is the highest maternal-cord gradient of all the fat-soluble vitamins and together with the lack of correlatio

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Berichte der Organisationen zum Thema "Placenta; transporter; transportér"

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Splitter, Gary A., Menachem Banai, and Jerome S. Harms. Brucella second messenger coordinates stages of infection. United States Department of Agriculture, 2011. http://dx.doi.org/10.32747/2011.7699864.bard.

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Aim 1: To determine levels of this second messenger in: a) B. melitensiscyclic-dimericguanosinemonophosphate-regulating mutants (BMEI1448, BMEI1453, and BMEI1520), and b) B. melitensis16M (wild type) and mutant infections of macrophages and immune competent mice. (US lab primary) Aim 2: To determine proteomic differences between Brucelladeletion mutants BMEI1453 (high cyclic-dimericguanosinemonophosphate, chronic persistent state) and BMEI1520 (low cyclicdimericguanosinemonophosphate, acute virulent state) compared to wild type B. melitensisto identify the role of this second messenger in esta

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