Einloggen

Thematische Bibliographien / Pulmonary Pathophysiology / Bücher

Um die anderen Arten von Veröffentlichungen zu diesem Thema anzuzeigen, folgen Sie diesem Link: Pulmonary Pathophysiology.

Bücher zum Thema „Pulmonary Pathophysiology“

Autor: Grafiati

Veröffentlicht am 4. Juni 2021

Zuletzt aktualisiert am 19. Februar 2022

Geben Sie eine Quelle nach APA, MLA, Chicago, Harvard und anderen Zitierweisen an

Wählen Sie eine Art der Quelle aus:

Machen Sie sich mit Top-50 Bücher für die Forschung zum Thema "Pulmonary Pathophysiology" bekannt.

Neben jedem Werk im Literaturverzeichnis ist die Option "Zur Bibliographie hinzufügen" verfügbar. Nutzen Sie sie, wird Ihre bibliographische Angabe des gewählten Werkes nach der nötigen Zitierweise (APA, MLA, Harvard, Chicago, Vancouver usw.) automatisch gestaltet.

Sie können auch den vollen Text der wissenschaftlichen Publikation im PDF-Format herunterladen und eine Online-Annotation der Arbeit lesen, wenn die relevanten Parameter in den Metadaten verfügbar sind.

Sehen Sie die Bücher für verschiedene Spezialgebieten durch und erstellen Sie Ihre Bibliographie auf korrekte Weise.

1

Pulmonary pathophysiology. Philadelphia: Lippincott, 1995.

Den vollen Inhalt der Quelle finden

APA, Harvard, Vancouver, ISO und andere Zitierweisen

2

Pulmonary pathophysiology: The essentials. 7th ed. Philadelphia: Wolters Kluwer Health/Lippincott Williams & Wilkins, 2008.

Den vollen Inhalt der Quelle finden

APA, Harvard, Vancouver, ISO und andere Zitierweisen

3

B, West John, ed. Pulmonary pathophysiology--the essentials. 4th ed. Baltimore: Williams & Wilkins, 1992.

Den vollen Inhalt der Quelle finden

APA, Harvard, Vancouver, ISO und andere Zitierweisen

4

West, John B. (John Burnard), ed. Pulmonary pathophysiology: The essentials. 8th ed. Philadelphia: Wolters Kluwer/Lippincott Williams & Wilkins Health, 2012.

Den vollen Inhalt der Quelle finden

APA, Harvard, Vancouver, ISO und andere Zitierweisen

5

Pulmonary pathophysiology: The essentials. 3rd ed. Baltimore: Williams & Wilkins, 1987.

Den vollen Inhalt der Quelle finden

APA, Harvard, Vancouver, ISO und andere Zitierweisen

6

B, West John, ed. Pulmonary pathophysiology--the essentials. 5th ed. Baltimore, Md: Williams & Wilkins, 1998.

Den vollen Inhalt der Quelle finden

APA, Harvard, Vancouver, ISO und andere Zitierweisen

7

Pulmonary pathophysiology: A clinical approach. 3rd ed. New York: McGraw-Hill Medical, 2010.

Den vollen Inhalt der Quelle finden

APA, Harvard, Vancouver, ISO und andere Zitierweisen

8

Workshop on "Chronic Pulmonary Hyperinflation" (1988 Montescano, Italy). Chronic pulmonary hyperinflation. London: Springer-Verlag, 1989.

Den vollen Inhalt der Quelle finden

APA, Harvard, Vancouver, ISO und andere Zitierweisen

9

Pulmonary circulation: Diseases and their treatment. 3rd ed. London: Hodder Arnold, 2011.

Den vollen Inhalt der Quelle finden

APA, Harvard, Vancouver, ISO und andere Zitierweisen

10

Bittar, E. Edward. Pulmonary biology in health and disease. Edited by Springer-Verlag. New York: Springer, 2002.

Den vollen Inhalt der Quelle finden

APA, Harvard, Vancouver, ISO und andere Zitierweisen

11

Belvisi, Maria G., and Jane A. Mitchell, eds. Nitric Oxide in Pulmonary Processes: Role in Physiology and Pathophysiology of Lung Disease. Basel: Birkhäuser Basel, 2000. http://dx.doi.org/10.1007/978-3-0348-8474-7.

Der volle Inhalt der Quelle

APA, Harvard, Vancouver, ISO und andere Zitierweisen

12

Workshop "Respiratory Muscles in C.O.P.D." (1986 Montescano, Italy). Respiratory muscles in chronic obstructive pulmonary disease. London: Springer-Verlag, 1987.

Den vollen Inhalt der Quelle finden

APA, Harvard, Vancouver, ISO und andere Zitierweisen

13

Yuan, Jason X.-J., 1963- and Ward Jeremy P. T, eds. Membrane receptors, channels, and transporters in pulmonary circulation. Dordrecht: Springer, 2010.

Den vollen Inhalt der Quelle finden

APA, Harvard, Vancouver, ISO und andere Zitierweisen

14

Principles of exercise testing and interpretation: Including pathophysiology and clinical applications. 5th ed. Philadelphia: Wolters Kluwer Health/Lippincott Williams & Wilkins, 2012.

Den vollen Inhalt der Quelle finden

APA, Harvard, Vancouver, ISO und andere Zitierweisen

15

Die Wirkung skelettmuskulärer Aktivität auf den Atemwiderstand: Eine psychologische Untersuchung mit gesunden und asthmatischen Personen. Frankfurt am Main: P. Lang, 1996.

Den vollen Inhalt der Quelle finden

APA, Harvard, Vancouver, ISO und andere Zitierweisen

16

West, John B. Pulmonary Pathophysiology: The Essentials (Pulmonary Pathophysiology). 7th ed. Lippincott Williams & Wilkins, 2007.

Den vollen Inhalt der Quelle finden

APA, Harvard, Vancouver, ISO und andere Zitierweisen

17

Pulmonary Pathophysiology: The Essentials (Pulmonary Pathophysiology). 6th ed. Lippincott Williams & Wilkins, 2003.

Den vollen Inhalt der Quelle finden

APA, Harvard, Vancouver, ISO und andere Zitierweisen

18

Juzar, Ali, Summer Warren R, and Levitzky Michael G, eds. Pulmonary pathophysiology. New York: McGraw-Hill, Health Professions Division, 1999.

Den vollen Inhalt der Quelle finden

APA, Harvard, Vancouver, ISO und andere Zitierweisen

19

Juzar, Ali, Summer Warren R, and Levitzky Michael G, eds. Pulmonary pathophysiology. 2nd ed. New York: Lange Medical Books/McGraw-Hill, 2005.

Den vollen Inhalt der Quelle finden

APA, Harvard, Vancouver, ISO und andere Zitierweisen

20

Ali, Juzar. Pulmonary Pathophysiology. McGraw-Hill Professional, 1998.

Den vollen Inhalt der Quelle finden

APA, Harvard, Vancouver, ISO und andere Zitierweisen

21

Ali, Juzar. Pulmonary Pathophysiology. McGraw-Hill Professional, 1998.

Den vollen Inhalt der Quelle finden

APA, Harvard, Vancouver, ISO und andere Zitierweisen

22

Pulmonary Pathophysiology (The Pathophysiology Series). Fence Creek Publishing, 1998.

Den vollen Inhalt der Quelle finden

APA, Harvard, Vancouver, ISO und andere Zitierweisen

23

West's Pulmonary Pathophysiology. Lippincott Williams & Wilkins, 2017.

Den vollen Inhalt der Quelle finden

APA, Harvard, Vancouver, ISO und andere Zitierweisen

24

West, John Jr. Pulmonary Pathophysiology: The Essentials. 3rd ed. Williams & Wilkins, 1987.

Den vollen Inhalt der Quelle finden

APA, Harvard, Vancouver, ISO und andere Zitierweisen

25

Kenneth, Weir E., and Reeves John T, eds. Pulmonary vascular physiology and pathophysiology. New York: Dekker, 1989.

Den vollen Inhalt der Quelle finden

APA, Harvard, Vancouver, ISO und andere Zitierweisen

26

Ali, Juzar, Warren G. Summer, and Michael G. Levitzky. Pulmonary Pathophysiology (Lange Physiology Series). McGraw-Hill Medical, 2004.

Den vollen Inhalt der Quelle finden

APA, Harvard, Vancouver, ISO und andere Zitierweisen

27

Ali, Juzar, Warren G. Summer, and Michael G. Levitzky. Pulmonary Pathophysiology (Lange Physiology Series). 2nd ed. McGraw-Hill Medical, 2004.

Den vollen Inhalt der Quelle finden

APA, Harvard, Vancouver, ISO und andere Zitierweisen

28

J, Whipp Brian, and Wasserman Karlman, eds. Exercise: Pulmonary physiology and pathophysiology. New York: M. Dekker, 1991.

Den vollen Inhalt der Quelle finden

APA, Harvard, Vancouver, ISO und andere Zitierweisen

29

1925-, Morpurgo M., ed. Pathophysiology and treatment of pulmonary circulation. London: Springer-Verlag, 1988.

Den vollen Inhalt der Quelle finden

APA, Harvard, Vancouver, ISO und andere Zitierweisen

30

Price, Laura, and S. John Wort. Pathophysiology and causes of pulmonary hypertension. Oxford University Press, 2016. http://dx.doi.org/10.1093/med/9780199600830.003.0168.

Der volle Inhalt der Quelle

Annotation:

Pulmonary hypertension (PH) in the setting of critical illness may reflect the acute syndrome itself (such as acute massive pulmonary embolism or acute lung injury), and/or pre-existing ‘chronic’ causes of PH. To compound this, iatrogenic factors may also contribute to PH including the effects of positive pressure ventilation and certain vasoactive drugs. The presence of PH, especially when complicated by resulting right ventricular (RV) dysfunction and failure, is a poor prognostic feature in all settings. This chapter reviews the pathophysiology of acute PH in critical illness, and pre-existing chronic causes of PH, including acute decompensation in patients with pre-existing pulmonary arterial hypertension.

APA, Harvard, Vancouver, ISO und andere Zitierweisen

31

Singer, Mervyn. Pathophysiology and causes of pulmonary embolism. Oxford University Press, 2016. http://dx.doi.org/10.1093/med/9780199600830.003.0170.

Der volle Inhalt der Quelle

Annotation:

Pulmonary embolus is predominantly due to thrombus breaking off from deep veins or from within the right heart, lodging within large or small vessels within the pulmonary vasculature, causing a variable degree of clinical features ranging from asymptomatic through to shock and cardiac arrest. Non-thrombotic causes include air or fat embolism. Outcome is predicated by the degree of right ventricular dysfunction. There are multiple risk factors including surgery, arrhythmias, prolonged immobility, venous stasis, pregnancy and an underlying pro-thrombotic tendency, either congenital or acquired. Numerous risk stratification scores have been developed derived from clinical features, imaging findings and biochemical markers of right ventricular strain and myocardial damage.

APA, Harvard, Vancouver, ISO und andere Zitierweisen

32

F, Voelkel Norbert, and Rounds Sharon 1946-, eds. The pulmonary endothelium. Chichester, West Sussex: John Wiley & Sons, 2009.

Den vollen Inhalt der Quelle finden

APA, Harvard, Vancouver, ISO und andere Zitierweisen

33

A, Bray Michael, and Anderson Wayne H, eds. Mediators of pulmonary inflammation. New York: M. Dekker, 1991.

Den vollen Inhalt der Quelle finden

APA, Harvard, Vancouver, ISO und andere Zitierweisen

34

R, Filbin Michael, ed. Blueprints notes & cases, pathophysiology: Pulmonary, gastrointestinal, and rheumatology. Malden, Mass: Blackwell Pub., 2004.

Den vollen Inhalt der Quelle finden

APA, Harvard, Vancouver, ISO und andere Zitierweisen

35

S, Zander Dani, and Farver Carol F, eds. Pulmonary pathology. Philadelphia: Churchill Livingstone/Elsevier, 2008.

Den vollen Inhalt der Quelle finden

APA, Harvard, Vancouver, ISO und andere Zitierweisen

36

Grassino, A. Chronic Pulmonary Hyperinflation. Springer, 2013.

Den vollen Inhalt der Quelle finden

APA, Harvard, Vancouver, ISO und andere Zitierweisen

37

Grassino, A., C. Rampulla, and R. Corsico. Chronic Pulmonary Hyperinflation. Springer, 2014.

Den vollen Inhalt der Quelle finden

APA, Harvard, Vancouver, ISO und andere Zitierweisen

38

P, Lynch Joseph, and DeRemee Richard A. 1933-, eds. Immunologically mediated pulmonary diseases. Philadelphia: Lippincott, 1991.

Den vollen Inhalt der Quelle finden

APA, Harvard, Vancouver, ISO und andere Zitierweisen

39

Pulmonary Physiology and Pathophysiology: An Integrated, Case-Based Approach. Lippincott Williams & Wilkins, 2001.

Den vollen Inhalt der Quelle finden

APA, Harvard, Vancouver, ISO und andere Zitierweisen

40

A, Parent Richard, ed. Treatise on pulmonary toxicology. Boca Raton: CRC Press, 1992.

Den vollen Inhalt der Quelle finden

APA, Harvard, Vancouver, ISO und andere Zitierweisen

41

1926-, Grassi Carlo, and Workshop "Update in Biochemistry of Pulmonary Emphysema" (1990 : Pavia, Italy), eds. Biochemistry of pulmonary emphysema. London: Springer-Verlag, 1992.

Den vollen Inhalt der Quelle finden

APA, Harvard, Vancouver, ISO und andere Zitierweisen

42

Blanchard-Loeb. Heart Failure and Pulmonary Edema: Pathophysiology for Nurses Video Series. Delmar Learning, 2000.

Den vollen Inhalt der Quelle finden

APA, Harvard, Vancouver, ISO und andere Zitierweisen

43

Vergnaud, Sophie, David Dobarro, and John Wort. Pulmonary vasculature. Oxford University Press, 2017. http://dx.doi.org/10.1093/med/9780199657742.003.0017.

Der volle Inhalt der Quelle

Annotation:

A 16-year-old girl with a diagnosis of diffuse cutaneous systemic sclerosis is referred to a specialist pulmonary hypertension centre with a history of progressive breathlessness, reduced exercise tolerance, and raised pulmonary pressures on transthoracic echocardiogram. She is found to have pulmonary arterial hypertension on right cardiac catheterization and is started on sildenafil, a phosphodiesterase-5 inhibitor, which stabilizes her condition. An endothelin receptor antagonist is added, which provides some initial symptomatic improvement. She continues to deteriorate over a period of 5 years, ultimately requiring intravenous prostanoids, the only treatment to provide a real symptomatic and haemodynamic improvement. This chapter explores the physiology and pathophysiology of pulmonary arterial hypertension, its classification, the means of investigation and diagnosis, who to refer to specialist centres, and the concepts behind current and future treatment strategies.

APA, Harvard, Vancouver, ISO und andere Zitierweisen

44

Textbook of pulmonary medicine. 2010.

Den vollen Inhalt der Quelle finden

APA, Harvard, Vancouver, ISO und andere Zitierweisen

45

Tombetti, Enrico, and Justin C. Mason. Pathophysiology of vasculitis. Oxford University Press, 2017. http://dx.doi.org/10.1093/med/9780198755777.003.0017.

Der volle Inhalt der Quelle

Annotation:

Vasculitis represents a spectrum of disorders that are often divided on the basis of the predominant vessel size affected into large-, medium- and small-vessel vasculitides. This chapter will focus on the pathogenesis of the anti-neutrophil cytoplasmic antibody (ANCA)-associated medium- and small-vessel vasculitides (AAV), and large-vessel vasculitis, Takayasu arteritis, and giant cell arteritis. Underlying pathogenic mechanisms in vasculitis remain to be fully understood. In particular, the initiating event(s) are not known. A combination of infectious or other environmental triggers on a susceptible genetic background is currently favoured. In addition to the vessel size affected, the mechanisms of vascular injury vary. Moreover, extravascular granulomatosis may play an important role in disease manifestations. The innate and adaptive immune systems contribute to its pathogenesis. Although pathogenic antibodies have not been identified in large-vessel vasculitis, ANCA are directly implicated in small- and medium-vessel AAV. Disease manifestations are varied and diverse and may include arterial stenosis or aneurysms, glomerulonephritis and renal failure, gastro-intestinal, pulmonary, cutaneous, and neurological complications, visual disturbance, deafness, and nasal bridge collapse. Life-threatening cardiovascular disease is also seen, with myocarditis, pericarditis, valvular heart disease, thrombosis, systemic and pulmonary arterial hypertension, and accelerated coronary heart disease all reported. Despite this, the prognosis for patients with vasculitis has improved significantly in recent decades. Further understanding of the pathogenesis of vasculitis will lead to the discovery of further therapeutic targets and novel, safer biologic therapies.

APA, Harvard, Vancouver, ISO und andere Zitierweisen

46

Denham, Ward, Dahan Albert, and Teppema Luc J, eds. Pharmacology and pathophysiology of the control of breathing. Boca Raton, FL: Taylor & Francis, 2005.

Den vollen Inhalt der Quelle finden

APA, Harvard, Vancouver, ISO und andere Zitierweisen

47

Pulmonary Vascular Physiology and Pathophysiology. Informa Healthcare, 1988.

Den vollen Inhalt der Quelle finden

APA, Harvard, Vancouver, ISO und andere Zitierweisen

48

Modell, Jerome H., and Sean Kiley. Pathophysiology and management of drowning. Oxford University Press, 2016. http://dx.doi.org/10.1093/med/9780199600830.003.0348.

Der volle Inhalt der Quelle

Annotation:

Drowning is a process beginning with airway submergence under a fluid medium, progressing to aspiration, and ultimately death in the absence of intervention. Aspiration of both fresh- and saltwater can cause pulmonary oedema, decreased compliance, intrapulmonary shunting, and severe hypoxia. Devastating neurological injury resulting from prolonged cerebral hypoxia is proportional to the duration of submersion and delay in effective resuscitation and oxygenation. Victims presenting to the emergency department awake and alert, or even stuporous, are likely to have a good neurological outcome with follow-up intensive care. Those presenting comatose are much more likely to have severe neurological deficits. Keys to survival are: timely rescue from the water, immediate initiation of aggressive supportive care regarding airway, cardiovascular and pulmonary function, and optimization of tissue oxygenation.

APA, Harvard, Vancouver, ISO und andere Zitierweisen

49

1952-, Müller B., Wichert P. von, and International Symposium on Lung Surfactant (4th : 1992 : Marburg, Germany), eds. Lung surfactant: Basic research in the pathogenesis of lung disorders. Basel: Karger, 1994.

Den vollen Inhalt der Quelle finden

APA, Harvard, Vancouver, ISO und andere Zitierweisen

50

Chronic pulmonary hyperinflation (Current topics in rehabilitation). Bi & Gi, 1991.

Den vollen Inhalt der Quelle finden

APA, Harvard, Vancouver, ISO und andere Zitierweisen

Wir bieten Rabatte auf alle Premium-Pläne für Autoren, deren Werke in thematische Literatursammlungen aufgenommen wurden. Kontaktieren Sie uns, um einen einzigartigen Promo-Code zu erhalten!