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Thematische Bibliographien / Rheumatoid arthritis Immunological aspects / Zeitschriftenartikel

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Zeitschriftenartikel zum Thema „Rheumatoid arthritis Immunological aspects“

Autor: Grafiati

Veröffentlicht am 4. Juni 2021

Zuletzt aktualisiert am 31. Januar 2023

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1

Gergely, Peter. "New Immunological Aspects of Rheumatoid Arthritis." Allergy & Clinical Immunology International - Journal of the World Allergy Organization 12, no. 2 (2000): 0077–81. http://dx.doi.org/10.1027/0838-1925.12.2.77.

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2

YOSHINO, KAZUYA. "Immunological aspects of juvenile rheumatoid arthritis." Pediatrics International 35, no. 5 (October 1993): 427–38. http://dx.doi.org/10.1111/j.1442-200x.1993.tb03087.x.

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3

Sugimoto, Masakuni, Yoshihisa Wakabayashi, Shun-Ichi Hirose, and Fumimaro Takaku. "Immunological aspects of the anemia of rheumatoid arthritis." American Journal of Hematology 25, no. 1 (May 1987): 1–11. http://dx.doi.org/10.1002/ajh.2830250102.

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4

Karateev, D. E. "Low activity and remission in rheumatoid arthritis. Clinical, immunological and morphological aspects." Rheumatology Science and Practice, no. 5 (October 15, 2009): 4. http://dx.doi.org/10.14412/1995-4484-2009-582.

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5

Rodríguez, Sandra, Andrés Muñoz, Rosa-Helena Bustos, and Diego Jaimes. "Pharmacovigilance of Biopharmaceuticals in Rheumatic Diseases, Adverse Events, Evolution, and Perspective: An Overview." Biomedicines 8, no. 9 (August 23, 2020): 303. http://dx.doi.org/10.3390/biomedicines8090303.

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Since we have gained an understanding of the immunological pathophysiology of rheumatic diseases such as rheumatoid arthritis and systemic lupus erythematosus, treatment based on biological drugs has become a fundamental axis. These therapies are oriented towards the regulation of cytokines such as tumour necrosis factor-alpha (TNF-α), interleukin (IL)-6, IL-1, and the modulation of cell-mediated immunity (B cells and T cells) by anti CD20 or anti CTAL-4 agents, and can increase the risk of associated infections or adverse events (AE). In this context, the entry of biotherapeutics represented

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6

OTTEN, H. G., M. R. DAHA, M. G. J. MAARL, L. I. HOOGENDOORN, E. M. BEEM, H. H. ROOY, and F. C. BREEDVELD. "IgA rheumatoid factor in mucosal fluids and serum of patients with rheumatoid arthritis: immunological aspects and clinical significance." Clinical & Experimental Immunology 90, no. 2 (June 28, 2008): 256–59. http://dx.doi.org/10.1111/j.1365-2249.1992.tb07938.x.

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7

Pandolfi, Franco, Laura Franza, Valentina Carusi, Simona Altamura, Gloria Andriollo, and Eleonora Nucera. "Interleukin-6 in Rheumatoid Arthritis." International Journal of Molecular Sciences 21, no. 15 (July 23, 2020): 5238. http://dx.doi.org/10.3390/ijms21155238.

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The role of interleukin (IL)-6 in health and disease has been under a lot of scrutiny in recent years, particularly during the recent COVID-19 pandemic. The inflammatory pathways in which IL-6 is involved are also partly responsible of the development and progression of rheumatoid arthritis (RA), opening interesting perspectives in terms of therapy. Anti-IL-6 drugs are being used with variable degrees of success in other diseases and are being tested in RA. Results have been encouraging, particularly when anti-IL-6 has been used with other drugs, such as metothrexate (MTX). In this review we d

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8

Reyes-Castillo, Zyanya, José Francisco Muñoz-Valle, and Mara A. Llamas-Covarrubias. "Clinical and immunological aspects of anti-peptidylarginine deiminase type 4 (anti-PAD4) autoantibodies in rheumatoid arthritis." Autoimmunity Reviews 17, no. 2 (February 2018): 94–102. http://dx.doi.org/10.1016/j.autrev.2017.11.023.

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9

Skovsgaard Itenov, K., N. Søe, E. M. Bartels, H. Bliddal, and M. Andersen. "AB0103 SITE SPECIFICITY OF RHEUMATOID ARTHRITIS INFLAMMATION: A SECONDARY ANALYSIS OF BIOPSIES FROM RADIAL AND ULNAR ASPECTS OF MCP JOINTS." Annals of the Rheumatic Diseases 81, Suppl 1 (May 23, 2022): 1182.2–1182. http://dx.doi.org/10.1136/annrheumdis-2022-eular.5196.

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BackgroundUlnar drift is a common complication of Rheumatoid Arthritis (RA) (1,2). There is no clear consensus regarding the etiology of the hand deformity. Observations from corrective hand surgery and other studies have noted more pronounced inflammation in the radial site of the MCP-joints (3,4). This could partly explain the pathophysiology behind the ulnar deviation.ObjectivesTo determine if there is more pronounced inflammation, measured by increased CD-68 expression (5) and Krenn-synovitis score (6), at the radial side of the MCP joints when compared to the ulnar side, in patients with

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10

Dekhtiarenko, N. О., L. M. Panchenko, M. P. Hrytsai, O. M. Linenko, V. I. Sabadosh, and K. M. Salmanova. "Analysis of Some Immunological Aspects of Joint Infections Developed as a Result of Intra-Articular Glucocorticoid Injection." Visnyk Ortopedii Travmatologii Protezuvannia, no. 4(111) (December 20, 2021): 21–27. http://dx.doi.org/10.37647/0132-2486-2021-111-4-21-27.

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Summary. The work is devoted to the studies of immune status of patients with infectious complications after local glucocorticoid injections. Objective: to assess the state of the immune system of patients with infectious complications after local glucocorticoid injections, to monitor the dynamics of immunological parameters before and after sanitizing surgical treatment, and to reveal factors that are important for predicting the course of the disease and treatment results. Materials and Methods. The immune status of 26 patients with purulent inflammatory processes after local glu

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11

Maeda, Shinji, Yoshihito Hayami, Taio Naniwa, and Ryuzo Ueda. "The Th17/IL-23 Axis and Natural Immunity in Psoriatic Arthritis." International Journal of Rheumatology 2012 (2012): 1–8. http://dx.doi.org/10.1155/2012/539683.

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Psoriatic arthritis (PsA) is a chronic inflammatory skin disease that causes enthesitis and destructive arthritis and significantly lowers patient quality of life. Recognition of the two target organs (the skin and joints) involved in the immunopathophysiology of PsA helped in elucidating the pathology of various systemic autoimmune diseases targeting multiple organs. Recent advances in immunology and genetics have made it clear that acquired immunity, especially that mediated by the Th17/IL-23 axis, plays an important role in the inflammatory pathology observed in psoriasis and PsA. Additiona

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12

RAMOS-CASALS, MANUEL, SANDRA MUÑOZ, FRANCISCO MEDINA, LUIS-JAVIER JARA, JOSÉ ROSAS, JAIME CALVO-ALEN, PILAR BRITO-ZERÓN, XAVIER FORNS, and JOSE-MARIA SÁNCHEZ-TAPIAS. "Systemic Autoimmune Diseases in Patients with Hepatitis C Virus Infection: Characterization of 1020 Cases (The HISPAMEC Registry)." Journal of Rheumatology 36, no. 7 (April 15, 2009): 1442–48. http://dx.doi.org/10.3899/jrheum.080874.

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Objective.To describe the clinical and immunologic characteristics of a large series of patients with systemic autoimmune diseases (SAD) associated with chronic hepatitis C virus (HCV) infection.Methods.The HISPAMEC Registry is a multicenter international study group dedicated to collecting data on patients diagnosed with SAD with serological evidence of chronic HCV infection. The information sources are cases reported by physicians of the HISPAMEC Study Group and periodic surveillance of reported cases by a Medline search updated up to December 31, 2007.Results.One thousand twenty HCV patient

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13

Burlutskaya, A. V., N. V. Savelyeva, and S. V. Polischuk. "THE STRUCTURE AND CLINICAL MANIFESTATIONS OF JUVENILE IDIOPATHIC ARTHRITIS IN CHILDREN IN KRASNODAR." Kuban Scientific Medical Bulletin 25, no. 6 (December 21, 2018): 38–43. http://dx.doi.org/10.25207/1608-6228-2018-25-6-38-43.

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Aim. The research was conducted to study the structure (variants, sexual and age characteristics) and manifestations of articular syndrome in children with juvenile idiopathic arthritis in Krasnodar.Materials and methods. There was conducted a retrospective analysis of 89 case histories in patients aged 2-15 diagnosed with juvenile idiopathic arthritis for the first time.Results. As a result of the study, the following variants of juvenile idiopathic arthritis were diagnosed: oligoarticular – in 62 patients (69,6%), polyarticular – in 24 patients (27%) and systemic – in 3 children (3,4%). Grou

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14

Todorovic-Djilas, Ljiljana, Tijana Icin, Jovanka Novakovic-Paro, and Ivana Bajkin. "Autoimmune thyroid disease and other non-endocrine autoimmune diseases." Medical review 64, no. 3-4 (2011): 183–87. http://dx.doi.org/10.2298/mpns1104183t.

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Introduction, Autoimmune diseases are chronic conditions initiated by the loss of immunological tolerance to self-antigens. They constitute heterogeneous group of disorders, in which multiple alterations in the immune system result in a spectrum of syndromes that either target specific organs or affect the body systematically. Recent epidemiological studies have shown a possible shift of one autoimmune disease to another or the fact that more than one autoimmune disease may coexist in a single patient or in the same family. Numerous autoimmune diseases have been shown to coexist frequently wit

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15

Bogmat, L. F., V. V. Nikonova, N. S. Shevchenko, and I. M. Bessonova. "Features of coagulation system in children with juvenile idiopathic arthritis." CHILD`S HEALTH 16, no. 2 (May 13, 2021): 105–10. http://dx.doi.org/10.22141/2224-0551.16.2.2021.229873.

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Background. It is known that the hemostasis system, inflammation and innate immunity have a common evolutionary origin, which explains the pathogenetic aspects of the relationship between inflammation and disorders in the hemostasis system. When the immune response is disturbed (autoimmune diseases), the inflammation becomes chronic. Unlike thrombosis associated with injury, the inflammatory process can initiate thrombosis in an intact vessel. Therefore, the purpose of this study was to assess the state of the hemostasis system in children with juvenile idiopathic arthritis in the dynamics of

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16

Matuszewska, Agnieszka, Marta Madej, and Piotr Wiland. "Immunological markers of rheumatoid arthritis." Postępy Higieny i Medycyny Doświadczalnej 70 (March 25, 2016): 251–57. http://dx.doi.org/10.5604/17322693.1198270.

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17

Edwards, Christopher J. "Immunological therapies for rheumatoid arthritis." British Medical Bulletin 73-74, no. 1 (January 1, 2005): 71–82. http://dx.doi.org/10.1093/bmb/ldh051.

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18

McGonagle, Dennis, Abdulla Watad, and Sinisa Savic. "Mechanistic immunological based classification of rheumatoid arthritis." Autoimmunity Reviews 17, no. 11 (November 2018): 1115–23. http://dx.doi.org/10.1016/j.autrev.2018.06.001.

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19

Ally, Mahmood M. T. M., and Bridget Hodkinson. "Rheumatoid Arthritis." South African Family Practice 58, no. 2 (March 1, 2016): 5. http://dx.doi.org/10.4102/safp.v58i2.4446.

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Immune-mediated inflammatory disorders include a clinically diverse group of conditions sharing similar pathogenic mechanisms. Conditions such as rheumatoid arthritis, psoriasis, spondyloarthropathy, inflammatory bowel disease and connective tissue diseases are characterised by immune dysregulation and chronic inflammation. This review will focus immuno-pathogenic mechanisms, aspects of early disease, co-morbidity and therapy in rheumatoid arthritis.

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20

Massarotti, Elena M. "MEDICAL ASPECTS OF RHEUMATOID ARTHRITIS." Hand Clinics 12, no. 3 (August 1996): 463–75. http://dx.doi.org/10.1016/s0749-0712(21)00336-x.

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21

Khurana, Ritu, and Seth Mark Berney. "Clinical aspects of rheumatoid arthritis." Pathophysiology 12, no. 3 (October 2005): 153–65. http://dx.doi.org/10.1016/j.pathophys.2005.07.009.

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22

SMITH, CAROLYN A., and FRANK C. ARNETT. "Epidemiologic Aspects of Rheumatoid Arthritis." Clinical Orthopaedics and Related Research &NA;, no. 265 (April 1991): 23???35. http://dx.doi.org/10.1097/00003086-199104000-00004.

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23

Yoshida, Yuji, and Toshio Tanaka. "Interleukin 6 and Rheumatoid Arthritis." BioMed Research International 2014 (2014): 1–12. http://dx.doi.org/10.1155/2014/698313.

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Interleukin-6 (IL-6) is a representative cytokine featuring pleiotropic activity and redundancy. A transient synthesis of IL-6 contributes to host defense against infectious agents and tissue injuries by inducing acute phase reactions and immunological and hematopoietic responses. However, uncontrolled persistent production of IL-6 may lead to the development of several immune-mediated diseases. Rheumatoid arthritis (RA) is a chronic disease with joint and systemic inflammation resulting from immunological abnormalities and it has been found that IL-6 plays a key role in the development of thi

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24

Sany, J. "Prospects in the Immunological Treatments of Rheumatoid Arthritis." Scandinavian Journal of Rheumatology 16, sup66 (January 1987): 129–36. http://dx.doi.org/10.3109/03009748709102530.

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25

Sokolova, V. V., S. V. Lapin, A. V. Moskalev, and V. I. Mazurov. "CLINICAL AND IMMUNOLOGICAL PARAMETERS IN EARLY RHEUMATOID ARTHRITIS." Medical Immunology (Russia) 9, no. 6 (July 18, 2014): 635. http://dx.doi.org/10.15789/1563-0625-2007-6-635-642.

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26

Dibrov, D. A. "ACCP-negative rheumatoid arthritis – clinical and immunological features." Rheumatology Science and Practice 60, no. 3 (July 9, 2022): 314–26. http://dx.doi.org/10.47360/1995-4484-2022-314-326.

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Depending on the presence of laboratory biomarkers: rheumatoid factor IgM and anti-cyclic citrullinated peptide antibodies (ACCP), “seropositive” and “seronegative” variants of rheumatoid arthritis (RA) are distinguished. Immunological subtypes differ in risk factors, immunopathogenesis, and the course of the disease. A review of data concerning immunology and clinical features of ACCP-negative rheumatoid arthritis is presented. The presence of ACCP in the peripheral blood reflects the progressive erosive process with a predominance of the inflammatory component and involvement of the B cells.

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27

Shostak, N. A., A. A. Muradyants, T. K. Loginova, and V. T. Timofeev. "Clinical and immunological features of early rheumatoid arthritis." Rheumatology Science and Practice, no. 1 (February 15, 2004): 15. http://dx.doi.org/10.14412/1995-4484-2004-1376.

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28

McInnes, Iain B., Christopher D. Buckley, and John D. Isaacs. "Cytokines in rheumatoid arthritis — shaping the immunological landscape." Nature Reviews Rheumatology 12, no. 1 (December 10, 2015): 63–68. http://dx.doi.org/10.1038/nrrheum.2015.171.

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29

Toivanen, A., K. Granfors, R. Lahesmaa-Rantala, and P. Toivanen. "Immunological and bacteriological aspects of reactive arthritis." Rheumatology International 9, no. 3-5 (November 1989): 201–3. http://dx.doi.org/10.1007/bf00271881.

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30

Henderson, Carol J., and Daniel J. Lovell. "Nutritional Aspects of Juvenile Rheumatoid Arthritis." Rheumatic Disease Clinics of North America 17, no. 2 (May 1991): 403–13. http://dx.doi.org/10.1016/s0889-857x(21)00580-9.

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31

Guseva, I. A., N. V. Demidova, N. E. Soroka, A. A. Novikov, E. L. Luchikhina, E. N. Aleksandrova, G. V. Lukina та ін. "Immunogenetic Aspects оf Early Rheumatoid Arthritis". Annals of the Russian academy of medical sciences 68, № 4 (2013): 36–43. http://dx.doi.org/10.15690/vramn.v68i4.609.

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32

King, Karen, and Virgil Hanson. "Psychosocial Aspects of Juvenile Rheumatoid Arthritis." Pediatric Clinics of North America 33, no. 5 (October 1986): 1221–37. http://dx.doi.org/10.1016/s0031-3955(16)36117-x.

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33

Leistner, Klaus. "14 Occupational aspects of rheumatoid arthritis." Baillière's Clinical Rheumatology 3, no. 1 (April 1989): 211–18. http://dx.doi.org/10.1016/s0950-3579(89)80046-9.

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34

Miller, Michael L. "Clinical aspects of juvenile rheumatoid arthritis." Current Opinion in Rheumatology 9, no. 5 (September 1997): 423–27. http://dx.doi.org/10.1097/00002281-199709000-00008.

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35

Kuzmina, N. N., and A. V. Shaikov. "JUVENILE RHEUMATOID ARTHRITIS (TERMINOLOGICALAND CLASSIFICATION ASPECTS)." Rheumatology Science and Practice, no. 1 (February 15, 2000): 35. http://dx.doi.org/10.14412/1995-4484-2000-785.

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36

Bugatti, Serena, Emanuele Bozzalla Cassione, Ludovico De Stefano, and Antonio Manzo. "Established rheumatoid arthritis. The pathogenic aspects." Best Practice & Research Clinical Rheumatology 33, no. 5 (October 2019): 101478. http://dx.doi.org/10.1016/j.berh.2019.101478.

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37

Kim, Nam Hyun, Kyu Hyun Yang, and Ick Hwan Yang. "Clinical significance of the immunological tests in rheumatoid arthritis." Yonsei Medical Journal 30, no. 1 (1989): 23. http://dx.doi.org/10.3349/ymj.1989.30.1.23.

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38

Duna, Madalina-Pusa, Denisa Predeteanu, Florian Berghea, Daniela Opris-Belinski, Andra Balanescu, Violeta Bojinca, and Ruxandra Ionescu. "Immunological findings in relatives of patients with rheumatoid arthritis." Romanian Journal of Rheumatology 28, no. 4 (December 31, 2019): 138–43. http://dx.doi.org/10.37897/rjr.2019.4.2.

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39

Mazurov, Vadim I. N., Inna Z. Gaydukova, Aleksandra Yu Fonturenko, Roman A. Bashkinov, Marianna Semenovna Petrova, and Oksana V. Inamova. "Coexistent rheumatoid arthritis and hyperuricemia: clinical and immunological features." HERALD of North-Western State Medical University named after I.I. Mechnikov 13, no. 3 (October 15, 2021): 43–52. http://dx.doi.org/10.17816/mechnikov80731.

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BACKGROUND: Currently, the negative role of asymptomatic hyperuricemia (HU) in the development and progression of cardiovascular pathology, metabolic disorders and chronic kidney disease is generally recognized. There is not much data in the literature on the effect of HU on the course of rheumatoid arthritis (RA), therefore, the study of the relationship of HU with clinical, radiological and immunological features of RA seems relevant. AIM: To study the relationship between HU with the clinical, radiological and immunological features of RA. MATERIALS AND METHODS: The data of 262

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40

Aira, Lazaro E., Patricia Hernández, Dinorah Prada, Araceli Chico, Jorge A. Gómez, Zuyén González, Karla Fuentes, Carmen Viada, and Zaima Mazorra. "Immunological evaluation of rheumatoid arthritis patients treated with itolizumab." mAbs 8, no. 1 (October 15, 2015): 187–95. http://dx.doi.org/10.1080/19420862.2015.1105416.

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41

Abendroth, K., G. Steiner, and G. Wessel. "Immunological mechanism of joint destruction in rheumatoid arthritis: A histomorphological and immunological study." Scandinavian Journal of Rheumatology 17, sup67 (January 1988): 70–72. http://dx.doi.org/10.3109/03009748809105302.

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42

Gao, Dashuang, Xu Gao, Fan Yang, and Qingwen Wang. "Neuroimmune Crosstalk in Rheumatoid Arthritis." International Journal of Molecular Sciences 23, no. 15 (July 24, 2022): 8158. http://dx.doi.org/10.3390/ijms23158158.

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Recent studies have demonstrated that immunological disease progression is closely related to abnormal function of the central nervous system (CNS). Rheumatoid arthritis (RA) is a chronic, inflammatory synovitis-based systemic immune disease of unknown etiology. In addition to joint pathological damage, RA has been linked to neuropsychiatric comorbidities, including depression, schizophrenia, and anxiety, increasing the risk of neurodegenerative diseases in life. Immune cells and their secreted immune factors will stimulate the peripheral and central neuronal systems that regulate innate and a

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43

Popova, Elka, Liliya Kavlakova, and Mariya Panchovska. "Chronic periodontitis and rheumatoid arthritis – microbiological aspects." Journal of Medical and Dental Practice 2, no. 3 (August 3, 2015): 232–44. http://dx.doi.org/10.18044/medinform.201523.232.

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44

Dijkmans, Ben A. C. "Safety Aspects of Cyclosporin in Rheumatoid Arthritis." Drugs 50, Supplement 1 (1995): 41–47. http://dx.doi.org/10.2165/00003495-199500501-00007.

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45

Sinachenko, O. V., and L. M. Pasiyeshvili. "Rheumatoid arthritis: definition, clinical and pathogenetic aspects." Shidnoevropejskij zurnal vnutrisnoi ta simejnoi medicini 2018, no. 1 (December 12, 2018): 4–15. http://dx.doi.org/10.15407/internalmed2018.01.004.

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46

Khobre, Priyanka, and S. Jayachandran. "Temporomandibular joint in rheumatoid arthritis: Clinicoradiological aspects." Indian Journal of Rheumatology 11, no. 3 (2016): 52. http://dx.doi.org/10.4103/0973-3698.187415.

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47

Schmit, P., Anne-Marie Prieur, and Francis Brunelle. "Juvenile rheumatoid arthritis and lymphoedema: lymphangiographic aspects." Pediatric Radiology 29, no. 5 (April 23, 1999): 364–66. http://dx.doi.org/10.1007/s002470050608.

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48

Dulyapin, V. A. "Morphological aspects of chondroclasis in rheumatoid arthritis." Bulletin of Experimental Biology and Medicine 112, no. 1 (July 1991): 1040–44. http://dx.doi.org/10.1007/bf00841168.

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49

Pratt, Arthur G., and John D. Isaacs. "Seronegative rheumatoid arthritis: Pathogenetic and therapeutic aspects." Best Practice & Research Clinical Rheumatology 28, no. 4 (August 2014): 651–59. http://dx.doi.org/10.1016/j.berh.2014.10.016.

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50

Sumathi, Lalitha, Manimekalai Pichaivel, Sabarinath ., and Thilagasundari Kandasamy. "Areview On Rheumatoid Arthiritis." Asian Journal of Pharmaceutical Research and Development 10, no. 6 (December 14, 2022): 104–9. http://dx.doi.org/10.22270/ajprd.v10i6.1200.

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Rheumatoid arthritis is a persistent, painful inflammatory condition characterized by serious destruction of the bone marrow and cartilage in the joints. It can also affect the body as a whole, including the tissues, leading to disorders of the heart, lungs, nervous system, and eyes. It is an extremely painful inflammatory condition that significantly limits movement due to pain and joint deterioration. Systemic disease rheumatoid arthritis usually affects tissues that constrict. Age, race, inheritance, aberrant immunological functioning, and stress are risk factors. Early rheumatoid arthritis

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