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Thematische Bibliographien / Sensitivity of the cancer cells / Dissertationen

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Dissertationen zum Thema „Sensitivity of the cancer cells“

Autor: Grafiati

Veröffentlicht am 28. Juni 2021

Zuletzt aktualisiert am 29. Juli 2025

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1

Tong, Zhimin. "The mechanisms of cellular sensitivity to photodynamic therapy /." *McMaster only, 2001.

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2

Landry, Benjamin D. "Tumor-stroma interactions differentially alter drug sensitivity based on the origin of stromal cells." eScholarship@UMMS, 2018. https://escholarship.umassmed.edu/gsbs_diss/1011.

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Tumor heterogeneity observed between patients has made it challenging to develop universal or broadly effective cancer therapies. Therefore, an ever-growing movement within cancer research aims to tailor cancer therapies to individual patients or specific tumor subtypes. Tumor stratification is generally dictated by the genomic mutation status of the tumor cells themselves. Importantly, non-genetic influences – such as interactions between tumor cells and other components of the tumor microenvironment – have largely been ignored. Therefore, in an effort to increase treatment predictability and

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3

Ferguson, Paul R. "The role of thymidylate synthase in modulating sensitivity to chemotherapeutic agents." Thesis, Queen's University Belfast, 2000. http://ethos.bl.uk/OrderDetails.do?uin=uk.bl.ethos.326399.

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4

Fung, Ka-lai. "Significance of MAD2 in mitotic checkpoint control and cisplatin sensitivity of testicular germ cell tumour cells." View the Table of Contents & Abstract, 2007. http://sunzi.lib.hku.hk/hkuto/record/B38588912.

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5

Kanwal, Shahzina. "Effect of O-GlcNAcylation on tamoxifen sensitivity in breast cancer derived MCF-7 cells." Phd thesis, Université René Descartes - Paris V, 2013. http://tel.archives-ouvertes.fr/tel-00912341.

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One of the hallmarks of cancer cells is to exhibit increased uptake and consumption of glucose.3-5% of the glucose entering into the cell leads to a minor pathway of the glucose metabolismknown as the hexosamine biosynthetic pathway (HBP). UDP-N-acetylglucosamine is the endproduct of HBP and is used as substrate by OGT (O-GlcNAc transferase) to modify diverserange of nuclear and cytoplasmic proteins with a recently characterized post-translationalmodification called O-GlcNAcylation. It corresponds to the addition of sugar moiety O-linked β-N-acetylglucosamine (O-GlcNAc) on serine or threonine

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6

Sureechatchaiyan, Paichat [Verfasser]. "Role of Purinergic Ligands to Enhance Cisplatin Sensitivity in Ovarian Cancer Cells / Paichat Sureechatchaiyan." Düsseldorf : Universitäts- und Landesbibliothek der Heinrich-Heine-Universität Düsseldorf, 2017. http://d-nb.info/1148066756/34.

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7

Fung, Ka-lai, and 馮家禮. "Significance of MAD2 in mitotic checkpoint control and cisplatin sensitivity of testicular germ cell tumour cells." Thesis, The University of Hong Kong (Pokfulam, Hong Kong), 2007. http://hub.hku.hk/bib/B39357727.

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8

Zhang, Pingde, та 张萍德. "TAp73α enhances the cellular sensitivity to cisplatin in ovarian cancer cells via the JNK signaling pathway". Thesis, The University of Hong Kong (Pokfulam, Hong Kong), 2011. http://hub.hku.hk/bib/B47752944.

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Ovarian cancer is the most lethal gynecological malignancy. Most of ovarian cancer patients relapse and subsequently die due to the development of resistance to chemotherapy. P73 belongs to the tumor suppressor p53 family. Like p53, the transcriptionally active TAp73 can bind specifically to p53 responsive elements and transactivates some of the p53 target genes, and finally leads to cell cycle arrest and apoptosis. TAp73 can be induced by DNA damage to enhance cellular sensitivity to anticancer agents in human cancer cells. However, the functions of TAp73 in ovarian cancer cells and t

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9

Newbury, Golnar. "A Numerical Study of a Delay Differential Equation Model for Breast Cancer." Thesis, Virginia Tech, 2007. http://hdl.handle.net/10919/34420.

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In this thesis we construct a new model of the immune response to the growth of breast cancer cells and investigate the impact of certain drug therapies on the cancer. We use delay differential equations to model the interaction of breast cancer cells with the immune system. The new model is constructed by combining two previous models. The first model accounts for different cell cycles and includes terms to evaluate drug treatments, but ignores quiescent tumor cells. The second model includes quiescent cells, but ignores the immune response and drug treatments. The new model is obtained by co

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10

Jokinen, E. (Elina). "Targeted therapy sensitivity and resistance in solid malignancies." Doctoral thesis, Oulun yliopisto, 2014. http://urn.fi/urn:isbn:9789526205755.

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Abstract Cancer is a major global killer and a challenge for the healthcare worldwide. Earlier cancer has been treated with surgery, radiation, chemotherapy and hormonal therapy. Unfortunately the efficiency of these therapies has shown to be limited and this has raised an enthusiasm for development of new, targeted cancer therapies that are based on activated oncogenes. The challenge of the targeted therapies is therapy resistance, de novo, adaptive and acquired. This work investigated targeted therapy sensitivity and resistance in lung cancer, breast cancer, colorectal cancer, and melanoma c

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11

Dilruba, Shahana [Verfasser]. "Impact of ERK signaling and its spatial regulation on cisplatin sensitivity in ovarian cancer cells / Shahana Dilruba." Bonn : Universitäts- und Landesbibliothek Bonn, 2019. http://d-nb.info/1220912638/34.

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12

Chan, Dana C. "THE EFFECT OF SILENCING THE WILMS' TUMOR 1 GENE ON THE RADIATION SENSITIVITY OF GLIOBLASTOMA CELLS." VCU Scholars Compass, 2006. http://hdl.handle.net/10156/1483.

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13

Meacham, Amy Marie. "Relationship of dna methyltransferases, dnmt3a and dnmt3b, and 5-aza-2'-deoxycytidine sensitivity among various cancer cell lines." [Gainesville, Fla.] : University of Florida, 2005. http://purl.fcla.edu/fcla/etd/UFE0010443.

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Thesis (M.S.)--University of Florida, 2005.<br>Typescript. Title from title page of source document. Document formatted into pages; contains 50 pages. Includes Vita. Includes bibliographical references.

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14

Ladha, Shabirali. "The effect of changes in plasma membrane lipid composition on the heat sensitivity of hepatoma tissue culture cells and selected plasma membrane enzymes." Thesis, Durham University, 1990. http://etheses.dur.ac.uk/6197/.

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Hepatoma Tissue Culture (HTC) cells grown in the presence of 60µM arachidonic acid for 24, 36 and 48 hours became progressively more thermosensitive than control cells. However, this difference in thermal sensitivity was only detectable with the clonogenic assay and not with the colorimetric assay. Attempts were also made to manipulate cellular cholesterol levels. Firstly, some cells were incubated with phosphatidylcholine liposomes to deplete the plasma membrane of cholesterol: Secondly, another group of cells were treated with 25 hydroxycholesterol, an inhibitor of cholesterol synthesis, to

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15

Hauser, Jennifer E. M. S. "Genetic Epidemiology of Radiation Sensitivity and Basal Cell Carcinoma in Childhood Cancer Survivors." University of Cincinnati / OhioLINK, 2015. http://rave.ohiolink.edu/etdc/view?acc_num=ucin1447689192.

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16

Lymperatou, Dionysia. "Exploring the impact of anti-oestrogen resistance on the capacity of breast cancer cells to modulate bone cell function and their sensitivity to bisphosphonates." Thesis, Cardiff University, 2017. http://orca.cf.ac.uk/109360/.

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Acquired resistance to endocrine therapy is a major limiting factor for their clinical effectiveness, resulting in disease relapse and an associated poor prognosis. Acquired resistance is also associated with the development of an invasive and migratory phenotype in vitro that may promote metastatic spread in vivo of which bone is the most frequent site. However, it is not currently known whether endocrine resistance affects the ability of breast cancer cells to modulate bone cell function important in establishing bone metastases or whether a resistant phenotype alters sensitivity to agents c

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Huichalaf, Carbonell Mariela [Verfasser], and Stefan [Akademischer Betreuer] Wölfl. "Metabolic Profiling of Cancer Cells and Correlations between Metabolism, Gene Expression and Drug Sensitivity / Mariela Huichalaf Carbonell ; Betreuer: Stefan Wölfl." Heidelberg : Universitätsbibliothek Heidelberg, 2018. http://d-nb.info/1177383950/34.

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18

Guo, Yang. "Overexpression of Heat-Shock Protein 27 (HSP27) increases gemcitabine sensitivity in pancreatic cancer cells through s-phase arrest and apoptosis." Diss., Ludwig-Maximilians-Universität München, 2014. http://nbn-resolving.de/urn:nbn:de:bvb:19-176979.

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19

Valiathan, Chandni Rajan. "Identifying a transcriptional signature for cell sensitivity to the cancer chemotherapy agent, BCNU." Thesis, Massachusetts Institute of Technology, 2011. http://hdl.handle.net/1721.1/65773.

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Thesis (Ph. D.)--Massachusetts Institute of Technology, Computational and Systems Biology Program, 2011.<br>Cataloged from PDF version of thesis.<br>Includes bibliographical references.<br>Many organisms have evolved DNA damage response mechanisms to deal with the constant damage to DNA caused by endogenous and exogenous agents. These mechanisms activate cell cycle checkpoints to allow time for DNA repair or, in the case of severely damaged DNA, initiate cell death mechanisms to maintain genomic integrity. The cell's response to DNA damaging agents includes wide spread changes in the transcrip

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20

Oatman, Nicole. "Mechanisms regulating cancer cell sensitivity and acquired resistance to Stearoyl-CoA Desaturase inhibition." University of Cincinnati / OhioLINK, 2019. http://rave.ohiolink.edu/etdc/view?acc_num=ucin1573572568302598.

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21

Verardi, Laura <1988&gt. "The Function of Cyclin D1 in DNA Repair: A New Possible Approach to Increase Sensitivity of Ovarian Cancer Cells to Irradiation." Doctoral thesis, Alma Mater Studiorum - Università di Bologna, 2017. http://amsdottorato.unibo.it/8070/1/Verardi_Laura_tesi.pdf.

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Cyclin D1 has been recently investigated as an important protein in homologous recombination-directed repair (HDR) mechanism. Cyclin D1 is recruited to damaged foci by BRCA2 and it enhances RAD51 binding to BRCA2. Furthermore, cyclin D1 is essential for RAD51 gene up-regulation in case of double-stranded break (DSB). Meanwhile molecule 3l, an E-3-(2-chloro-3-indolylmethylene)1,3-dihydroindol-2one, has been showed to reduce cyclin D1 level and, for this reason, 3l treatment could be used to study cyclin D1 role in DNA damage repair. Scope of this thesis is to find a new possible combined appro

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Kozlak, Maria. "AsiDNA, a Unique DNA Repair Inhibitor, Triggers Sensitization and Bioenergetic Adaptation in Cancer Cells." Thesis, Université Paris-Saclay (ComUE), 2019. http://www.theses.fr/2019SACLS101.

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Le but d’un traitement anticancéreux est d’être spécifique et efficacité dans la durée vis-à-vis des cellules tumorales. De nombreux agents chimiothérapeutiques ont rencontré des obstacles quant à leur utilisation en raison de leur toxicité pour les cellules saines ou de la résistance développée par les cellules cancéreuses. Cela souligne la nécessité de développer des médicaments alternatifs. Notre laboratoire a développé une classe d'inhibiteurs de réparation de l'ADN, Dbait, qui agissent en détournant et en hyperactivant les protéines de la réparation de l’ADN, telles que la protéine PARP e

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Law, Edward William Preston. "Determining mechanisms of sensitivity and resistance to HSP90 inhibition in non-small cell lung cancer." Thesis, University of Leicester, 2016. http://hdl.handle.net/2381/38121.

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BACKGROUND: HSP90 is a molecular chaperone which supports the maturation of numerous client proteins, many of which are involved in oncogenic signaling pathways such as PI3K/AKT, MAPK and JAK/STAT. HSP90 is an appealing target for small molecule inhibitors due to its ability to simultaneously downregulate multiple oncogenic conduits. Results in the clinic have been mixed and biomarkers of response to HSP90 inhibitors for patient stratification remain obscure. RESULTS: 1. EML4-ALK is a HSP90 client which degrades in response to HSP90 inhibition. Variants of EML4-ALK exist with disparate structu

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Siu, Wing-fai, and 邵穎暉. "Melatonin and prostate cancer cell proliferation: interplay with castration, epidermal growth factor and androgen sensitivity." Thesis, The University of Hong Kong (Pokfulam, Hong Kong), 2001. http://hub.hku.hk/bib/B23829850.

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25

Siu, Wing-fai. "Melatonin and prostate cancer cell proliferation : interplay with castration, epidermal growth factor and androgen sensitivity /." Hong Kong : University of Hong Kong, 2001. http://sunzi.lib.hku.hk/hkuto/record.jsp?B23829850.

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26

Panzilius, Elena [Verfasser], and Magdalena [Akademischer Betreuer] Götz. "Dissection and identification of cellular contexts that determine sensitivity to ferroptosis in human mammary epithelial cells and breast cancer / Elena Panzilius ; Betreuer: Magdalena Götz." München : Universitätsbibliothek der Ludwig-Maximilians-Universität, 2020. http://d-nb.info/121603916X/34.

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27

Guo, Yang [Verfasser], and Max [Akademischer Betreuer] Schnurr. "Overexpression of Heat-Shock Protein 27 (HSP27) increases gemcitabine sensitivity in pancreatic cancer cells through s-phase arrest and apoptosis / Yang Guo. Betreuer: Max Schnurr." München : Universitätsbibliothek der Ludwig-Maximilians-Universität, 2014. http://d-nb.info/1062877586/34.

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28

Powell, Chase David. "Role of Heat Shock Transcription Factor 1 in Ovarian Cancer Epithelial-Mesenchymal Transition and Drug Sensitivity." Scholar Commons, 2017. http://scholarcommons.usf.edu/etd/7079.

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The heat shock response (HSR) is a robust cellular reaction to mitigate protein damage from heat and other challenges to the proteome. This protective molecular program in humans is controlled by heat shock transcription factor 1 (HSF1). Activation of HSF1 leads to the induction of an array of cytoprotective genes, many of which code for chaperones. These chaperones, known as heat shock proteins (HSPs), are responsible for maintaining the functional integrity of the proteome. HSPs achieve this by promoting proper folding and assembly of nascent proteins, refolding denatured

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Hultman, Bo. "Clinical and Experimental Studies in Peritoneal Metastases from Gastric Cancer." Doctoral thesis, Uppsala universitet, Kolorektalkirurgi, 2013. http://urn.kb.se/resolve?urn=urn:nbn:se:uu:diva-197776.

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Gastric cancer (GC) is one of leading causes of death in the world, and peritoneal metastases (PM) are a major site of recurrence. PM from GC implies a poor prognosis, with median overall survival (mOS) approximately 3 months and no survival at five years. The aims of this thesis were to explore the incidence and evaluate prognostic factors for mOS of PM from GC in a defined population; to investigate the outcome of a new multimodal treatment; to analyse the treatment costs, and to investigate differences in drug sensitivity between individual patient samples and between various tumours. The i

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Dabare, Abeysinghe Arachchige Nandike Prashanth M. "Development of new monoclonal antibodies and colorimetric assays for improved detection of testicular germ cell tumours (TGCTs) and determining the relevance of over-expressed P53 in TGCT sensitivity to treatment." Thesis, Queen Mary, University of London, 1999. http://ethos.bl.uk/OrderDetails.do?uin=uk.bl.ethos.312172.

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31

Terry, Samantha Y. A. "A role for topoisomerase II alpha in chromosome damage in human cell lines." Thesis, University of St Andrews, 2010. http://hdl.handle.net/10023/873.

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Human response to ionising radiation (IR) shows a wide variation. This is most clearly seen in the radiation-response of cells as measured by frequencies of chromosomal aberrations. Different frequencies of IR-induced aberrations can be conveniently observed in phytohaemagglutin-stimulated peripheral blood T-lymphocytes from both normal individuals and sporadic cancer cases, in either metaphase chromosomes or as micronuclei in the following cell cycle. Metaphase cells show frequent chromatid breaks, defined as chromatid discontinuities or terminal deletions, if irradiated in the G 2 -phase of

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Fournel, Ludovic. "Influence Du Cisplatine sur l'expression du Check-Point Immunitaire PD-1/PD-L1 Dans Le Cancer Broncho-Pulmonaire Non A Petites Cellules Cisplatin Increases PD-L1 Expression and Optimizes Immune Check-Point Blockade in Non-Small Cell Lung Cancer Modulation of Lung Cancer Cell Plasticity and Heterogeneity with the Restoration of Cisplatin Sensitivity by Neurotensin Antibody." Thesis, université Paris-Saclay, 2020. http://www.theses.fr/2020UPASS077.

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Malgré les nombreux progrés réalisés ces dernières années dans la prise en charge thérapeutique du cancer broncho-pulmonaire, cette pathologie reste la première cause de décès lié au cancer dans le monde. L’enjeu majeur pour cette maladie est donc de développer de nouveaux traitements et d’optimiser l’utilisation des drogues existantes, en particulier les sels de platine. Les protocoles combinant des inhibiteurs de points de contrôle immunitaire avec des sels de platine est actuellement en plein essor malgré un certain manque en études précliniques. Dans ce travail, j’ai cherché à évaluer l'im

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Engel, Roxane. "The nuclear export of DNA topoisomerase iialpha in hematological myeloma cell lines as a function of drug sensitivity : clinical implications and a theoretical approach for overcoming the observed drug resistance /." [Tampa, Fla.] : University of South Florida, 2005. http://purl.fcla.edu/fcla/etd/SFE0001358.

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34

PUGNALONI, SOFIA. "Taste sensitivity in cancer patient." Doctoral thesis, Università Politecnica delle Marche, 2020. http://hdl.handle.net/11566/274088.

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Le modificazioni della sensibilità gustativa dovute alla chemioterapia possono contribuire all'elevata prevalenza della malnutrizione nei pazienti oncologici. La diagnosi di disgeusia è importante per la prognosi dei pazienti. L'obiettivo di questa tesi è quello di contribuire a prevenire e/o ridurre le alterazioni del gusto e la malnutrizione aprendo prospettive future che considerano la valutazione precoce delle alterazioni del gusto per una migliore gestione dell’alimentazione del paziente. Nello studio I, 35 pazienti oncologici sottoposti a chemioterapia sono stati confrontati con contro

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Langlands, Fiona Elizabeth. "Sensitivity to radiotherapy in breast cancer." Thesis, University of Leeds, 2010. http://ethos.bl.uk/OrderDetails.do?uin=uk.bl.ethos.582111.

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Radiotherapy (RT) is a component of treatment for breast cancer in more than 50% of cases. Patients are selected for RT on the basis of limited clinical and histopathological factors with no reference to the molecular phenotype of tumours. Breast cancer is a highly heterogeneous disease and some tumours are refractory to RT treatment. Selection of patients for RT using predictive markers may improve RT efficacy and cancer outcomes. The aims of the work described in this thesis were to identify and test potential predictive markers for RT in cancer treatment. I examined whether the classic hist

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Chang, Sue-Ling. "Breast cancer subtypes and screening mammography sensitivity." Thesis, Université Laval, 2014. http://www.theses.ulaval.ca/2014/30680/30680.pdf.

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Les cancers du sein peuvent être classifiés selon le statut de récepteur d’estrogène (RE), de récepteur de progestérone (RP), de récepteur HER2, ou selon quatre sous-types (Luminal A, Luminal B, HER2-enrichi, Triple-négatif) ayant des propriétés biologiques et cliniques différentes. La sensibilité du dépistage par mammographie pourrait varier selon ces types de cancers mais ceci n’est pas encore clair. L’agressivité de la tumeur, mesurée par le grade histologique pourrait expliquer cette association. Les types de cancers d’intervalle ont été comparés à ceux de cancers détectés par dépistage pa

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Uzzell, Valerie Joy. "Sensitivity and noise in primate retinal ganglion cells /." Diss., Connect to a 24 p. preview or request complete full text in PDF format. Access restricted to UC campuses, 2005. http://wwwlib.umi.com/cr/ucsd/fullcit?p3190165.

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Lincoln, Maximilian Christian. "Multidrug resistance and collateral sensitivity of tumour cells." Thesis, National Library of Canada = Bibliothèque nationale du Canada, 1997. http://www.collectionscanada.ca/obj/s4/f2/dsk2/tape16/PQDD_0018/MQ37141.pdf.

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Coulson-Gilmer, Camilla Lucette. "Cancer stem cells and chemoresistance in ovarian cancer." Thesis, University of Leeds, 2017. http://etheses.whiterose.ac.uk/18470/.

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The high mortality rate associated with epithelial ovarian cancer (EOC) is due to its insidious onset, leading to late diagnosis as well as eventual development of chemoresistance in the majority of patients. Cancer stem-like cells (CSCs) are thought to contribute to development of multi-drug resistant (MDR) tumours partly through their high level of ABC-transporter expression, which enables them to survive chemotherapy. ABC-transporter (MRP1, MRP2, BCRP, Pgp) and putative CSC-marker (ALDH1A1, CD44) expression was therefore evaluated by immunohistochemistry in a paraffin-embedded cohort of 57

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Drayton, Ross. "Drug sensitivity and apoptosis in tamoxifen resistant breast cancer." Thesis, Cardiff University, 2007. http://orca.cf.ac.uk/55694/.

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Tamoxifen has long been used in the treatment of hormone responsive breast cancer. However, tumours frequently develop resistance within 2-5 years of treatment, characterised by the return of tumour growth. The epidermal growth factor receptor EGFR is an important contributing factor in allowing formerly tamoxifen sensitive tumours to grow in the presence of tamoxifen. High levels of EGFR in many tumours correlate with a poor prognosis and an increased resistance to cytotoxic drugs. It was the initial aim of this study to ascertain whether the increased EGFR signalling associated with tamoxife

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Múnera, Jorge O. "Ets2 regulates colonic stem cells and sensitivity to tumorigenesis." Diss., [La Jolla] : University of California, San Diego, 2010. http://wwwlib.umi.com/cr/ucsd/fullcit?p3397225.

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Thesis (Ph. D.)--University of California, San Diego, 2010.<br>Title from first page of PDF file (viewed March 30, 2010). Available via ProQuest Digital Dissertations. Vita. Includes bibliographical references (p. 101-108).

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Sherwood, Benedict T. "Radiosensitivity in bladder cancer cells." Thesis, University of Leicester, 2005. http://hdl.handle.net/2381/29874.

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Potentially curative treatment options for patients with organ-confined transitional cell carcinoma (TCC) of the bladder (T1-4a/N0/M0) are radical cystectomy or radiotherapy (RT)-based 'bladder-preserving' regimens. A substantial number of patients who receive RT fail to respond (approximately 50%). Consequently, a greater understanding of the mechanisms of radioresistence is required, together with predictive information regarding the response of tumours to RT. Hypoxia and intrinsic cellular Radiosensitivity (IRS) are examined here, a factors that may influence the outcome of RT.;An immunohis

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Chan, Ching Wan. "Apoptosis in breast cancer cells." Thesis, University of Bristol, 2004. http://hdl.handle.net/1983/8971525c-0de9-4e21-9677-ab73d61ae65c.

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Dai, Prè Elena <1990&gt. "ELECTRICAL CHARACTERIZATION OF CANCER CELLS." Master's Degree Thesis, Università Ca' Foscari Venezia, 2015. http://hdl.handle.net/10579/6321.

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In this work, the impedance of different cancer cell lines was measured using a lab-on-chip device developed by imec (interuniversity electronics center) in Leuven, Belgium. The motivation of this thesis is the detection of circulating tumour cells (CTCs) from cancer metastatic patients. Currently CTC detection is challenging because of the low abundance and phenotype similarity with white blood cells. Imec is developing a proprietary transistor-embedded device for massively parallel on-chip single cell characterization in the European project MIRACLE (Magnetic Isolation and moleculaR Analysi

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Grero, Dhanya. "Cytotoxicity of Vγ9Vδ2 T cells towards Colon Cancer Cells". Thesis, Uppsala universitet, Institutionen för biologisk grundutbildning, 2014. http://urn.kb.se/resolve?urn=urn:nbn:se:uu:diva-230976.

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Immunotherapies for cancer are widely studied at present. We are currently studying a specific form of “Vγ9Vδ2 T cells” found in the peripheral blood of healthy donors that can be used for the killing of HT-29 colon cancer cells. In order to determine the cytotoxicity of effectors, Vγ9Vδ2 T cells towards target cells, HT-29, it is important to first evaluate the absolute number of Vγ9Vδ2 T cells in a mixed cell population, and next to determine the phenotypic characterization, their activity and cytotoxicity in the presence of target cells. A flow cytometry and bead based assay was developed t

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Fung, Kwong-lam, and 馮廣林. "Chemoresistance induced by mesenchymal stromal cells on cancer cells." Thesis, The University of Hong Kong (Pokfulam, Hong Kong), 2013. http://hdl.handle.net/10722/205639.

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Human mesenchymal stromal cells (hMSCs) are part of bone marrow micro-environment that supports hematopoiesis. However, hMSCs also enhance tumor progression and survival when they become part of the cancer micro-environment. I aimed to investigate the interaction between hMSCs and cancer cells during chemotherapy. Firstly, I studied the interaction between hMSCs and T-lineage acute lymphoblastic leukemia (T-ALL) cells under pegylated arginase I (BCT-100) treatment. Three T-ALL cell lines were sensitive to BCT-100 but not hMSCs. Conversely, hMSCs could partly protect all T-ALL cell lines fro

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Briggs, Clare. "An investigation into ultraviolet radiation sensitivity and human skin cancer." Thesis, Lancaster University, 2002. http://ethos.bl.uk/OrderDetails.do?uin=uk.bl.ethos.403927.

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Yang, Chun-ting, and 楊俊廷. "The role of stroma cells in radio-sensitivity of cervical cancer HeLa cells." Thesis, 2011. http://ndltd.ncl.edu.tw/handle/75069684690592311309.

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碩士<br>慈濟大學<br>分子生物暨人類遺傳學系碩士班<br>99<br>Radiotherapy is one of effective treatments for patients with cervical cancers, especially for those with direct invasion or distal metastasis. However, outcome of radiotherapy is not consistent. In this study, role of stromal cells in radio-sensitivity of cervical cancer HeLa cells cultured in condition media (CMs) derived form coculture of HeLa cells and stromal cells were investigated. To prepare conditioned media from stromal cells or mixcultures (Stromal cells : HeLa cells = 1:1), stromal cells prepared from stromal cells closed to the cervical tumor

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HUANG, JING-MIN, and 黃經民. "Characteristics of semipermeable microcapsule culture for cancer cells and amticancer sensitivity assay." Thesis, 1988. http://ndltd.ncl.edu.tw/handle/85040222747335977359.

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Fernandes, Andreia Silva. "Cancer cells: a crosstalk between general metabolism and sensitivity to apoptotic signals." Master's thesis, 2017. http://hdl.handle.net/10348/8800.

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The transformation of a normal cell into a cancer cell is a pathological process involving several stages, which modifies cellular metabolism and supports: rapid generation of ATP, high rate of macromolecules biosynthesis and maintenance of the appropriate cellular redox state. Mitochondria play a central role in the eukaryotic cell because of their relevance in biosynthetic and bioenergetic metabolisms. Additionally, mitochondria are also responsible for the production and/or control of various types of signals (including molecules), involved in cellular signalling processes that modulate var

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