Thematische Bibliographien / VECTOR DE VIRUS

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Auswahl der wissenschaftlichen Literatur zum Thema „VECTOR DE VIRUS“

Autor: Grafiati

Veröffentlicht am 17. September 2021

Zuletzt aktualisiert am 14. Februar 2022

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Zeitschriftenartikel zum Thema "VECTOR DE VIRUS"

Deyle, David R., Yi Li, Erik M. Olson und David W. Russell. „Nonintegrating Foamy Virus Vectors“. Journal of Virology 84, Nr. 18 (30.06.2010): 9341–49. http://dx.doi.org/10.1128/jvi.00394-10.

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ABSTRACT Foamy viruses (FVs), or spumaviruses, are integrating retroviruses that have been developed as vectors. Here we generated nonintegrating foamy virus (NIFV) vectors by introducing point mutations into the highly conserved DD35E catalytic core motif of the foamy virus integrase sequence. NIFV vectors produced high-titer stocks, transduced dividing cells, and did not integrate. Cells infected with NIFV vectors contained episomal vector genomes that consisted of linear, 1-long-terminal-repeat (1-LTR), and 2-LTR circular DNAs. These episomes expressed transgenes, were stable, and became progressively diluted in the dividing cell population. 1-LTR circles but not 2-LTR circles were found in all vector stocks prior to infection. Residual integration of NIFV vectors occurred at a frequency 4 logs lower than that of integrase-proficient FV vectors. Cre recombinase expressed from a NIFV vector mediated excision of both an integrated, floxed FV vector and a gene-targeted neo expression cassette, demonstrating the utility of these episomal vectors. The broad host range and large packaging capacity of NIFV vectors should make them useful for a variety of applications requiring transient gene expression.

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Choi, Charles Q. „Vector without Virus“. Scientific American 292, Nr. 3 (März 2005): 30. http://dx.doi.org/10.1038/scientificamerican0305-30c.

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Bukovsky, Anatoly A., Jin-Ping Song und Luigi Naldini. „Interaction of Human Immunodeficiency Virus-Derived Vectors with Wild-Type Virus in Transduced Cells“. Journal of Virology 73, Nr. 8 (01.08.1999): 7087–92. http://dx.doi.org/10.1128/jvi.73.8.7087-7092.1999.

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ABSTRACT The interaction of human immunodeficiency virus (HIV)-derived vectors with wild-type virus was analyzed in transduced cells. Vector transcripts upregulated by infection had no measurable effect on HIV type 1 (HIV-1) expression but competed efficiently for encapsidation, inhibiting the infectivity and spread of HIV-1 in culture and leading to mobilization and recombination of the vector. These effects were abrogated with a self-inactivating vector.

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Russell, RA, G. Vassaux, P. Martin-Duque und MO McClure. „Transient foamy virus vector production by adenovirus vectors“. Gene Therapy 11, Nr. 3 (22.01.2004): 310–16. http://dx.doi.org/10.1038/sj.gt.3302177.

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Hofmann, Wolfgang, David Schubert, Jason LaBonte, Linda Munson, Susan Gibson, Jonathan Scammell, Paul Ferrigno und Joseph Sodroski. „Species-Specific, Postentry Barriers to Primate Immunodeficiency Virus Infection“. Journal of Virology 73, Nr. 12 (01.12.1999): 10020–28. http://dx.doi.org/10.1128/jvi.73.12.10020-10028.1999.

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ABSTRACT By using replication-defective vectors derived from human immunodeficiency virus type 1 (HIV-1), simian immunodeficiency virus (SIVmac), and murine leukemia virus (MuLV), all of which were pseudotyped with the vesicular stomatitis virus (VSV) G glycoprotein, the efficiency of postentry, early infection events was examined in target cells of several mammalian species. Titers of HIV-1 vectors were significantly lower than those of SIVmacand MuLV vectors in most cell lines and primary cells from Old World monkeys. By contrast, most New World monkey cells exhibited much lower titers for the SIVmac vector compared with those of the HIV-1 vector. Prosimian cells were resistant to both HIV-1 and SIVmac vectors, although the MuLV vector was able to infect these cells. Cells from other mammalian species were roughly equivalent in susceptibility to the three vectors, with the exception of rabbit cells, which were specifically resistant to the HIV-1 vector. The level of HIV-1 vector expression was very low in transduced cells of rodent, rabbit, cow, and pig origin. Early postentry restriction of primate immunodeficiency virus infection exhibits patterns largely coincident with species borders and applies to diverse cell types within an individual host, suggesting the involvement of species-specific, widely expressed cellular factors.

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Su, RuiJun, Rency L. Rosales, Martin Lochelt und Neil C. Josephson. „Transduction of Primate Cells with Feline Foamy Virus Envelope Pseudotyped Prototype Foamy Virus Vectors.“ Blood 104, Nr. 11 (16.11.2004): 5276. http://dx.doi.org/10.1182/blood.v104.11.5276.5276.

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Abstract Because of their genetic and biological similarity to humans, non-human primates are the best pre-clinical models for testing the efficacy and safety of gene therapy systems. However, the presence of endogenous simian foamy virus infection in nearly all non-human primates kept in captivity complicates foamy virus (FV) vector stem cell transduction studies in these animals. A major concern is that repopulating cells exposed to FV vector stocks will elicit an immune response in non-human primate hosts. Though human serum does not inactivate prototype foamy virus (PFV) vectors, a one hour incubation of PFV vector stock in the presence of serum samples from Papio Cynophalus (baboon), Macaca Mulatta (rhesus macaque), or Macaca Fasicularis (long-tailed macaque) results in a 75–100% drop in titer. To overcome this serum mediated inactivation we sought to pseudotype PFV vectors in the feline foamy virus (FFV) envelope. The wild-type envelope from the FUV strain of FFV does not pseudotype our PFV vectors. Therefore we generated chimeras with regions of both the FFV and PFV envelope. By substituting portions of the FFV envelope leader peptide sequence and membrane spanning domain with corresponding PFV envelope regions we generated chimeric envelopes capable of high titer (105–106 FFU/ml) PFV vector production. Serum samples from Macaca Mulatta produced less inactivation of the FFV pseudotyped than the PFV pseudotyped vectors. Furthermore, both the PFV and FFV pseudotyped vectors demonstrated efficient transduction of baboon mesenchymal stem cells (27–43%) and baboon embryonic stem cells (37–40%). However, the FFV pseudotyped vectors transduced both human and baboon CD34+ cells less efficiently than the PFV pseudotyped vectors. We plan to test PFV vectors pseudotyped by other FV envelopes for inactivation by primate serum, and for their ability to transduce primate hematopoietic cells.

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Young, Won-Bin, Gary L. Lindberg und Charles J. Link. „DNA Methylation of Helper Virus Increases Genetic Instability of Retroviral Vector Producer Cells“. Journal of Virology 74, Nr. 7 (01.04.2000): 3177–87. http://dx.doi.org/10.1128/jvi.74.7.3177-3187.2000.

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ABSTRACT Retroviral vector producer cells (VPC) have been considered genetically stable. A clonal cell population exhibiting a uniform vector integration pattern is used for sustained vector production. Here, we observed that the vector copy number is increased and varied in a population of established LTKOSN.2 VPC. Among five subclones of LTKOSN.2 VPC, the vector copy number ranged from 1 to approximately 29 copies per cell. A vector superinfection experiment and Northern blot analysis demonstrated that suppression of helper virus gene expression decreased Env-receptor interference and allowed increased superinfection. The titer production was tightly associated with helper virus gene expression and varied between 0 and 2.2 × 105 CFU/ml in these subclones. In one analyzed subclone, the number of integrated vectors increased from one copy per cell to nine copies per cell during a 31-day period. Vector titer was reduced from 1.5 × 105 CFU to an undetectable level. To understand the mechanism involved, helper virus and vectors were examined for DNA methylation status by methylation-sensitive restriction enzyme digestion. We demonstrated that DNA methylation of helper virus 5′ long terminal repeat occurred in approximately 2% of the VPC population per day and correlated closely with inactivation of helper virus gene expression. In contrast, retroviral vectors did not exhibit significant methylation and maintained consistent transcription activity. Treatment with 5-azacytidine, a methylation inhibitor, partially reversed the helper virus DNA methylation and restored a portion of vector production. The preference for methylation of helper virus sequences over vector sequences may have important implications for host-virus interaction. Designing a helper virus to overcome cellular DNA methylation may therefore improve vector production. The maintenance of increased viral envelope-receptor interference might also prevent replication-competent retrovirus formation.

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Hariharan, Mangala J., David A. Driver, Kay Townsend, Duane Brumm, John M. Polo, Barbara A. Belli, Donald J. Catton et al. „DNA Immunization against Herpes Simplex Virus: Enhanced Efficacy Using a Sindbis Virus-Based Vector“. Journal of Virology 72, Nr. 2 (01.02.1998): 950–58. http://dx.doi.org/10.1128/jvi.72.2.950-958.1998.

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ABSTRACT Previously we reported the development of a plasmid DNA expression vector system derived from Sindbis virus (T. W. Dubensky, Jr., et al., J. Virol. 70:508–519, 1996). In vitro, such vectors exhibit high-level heterologous gene expression via self-amplifying cytoplasmic RNA replication. In the present study, we demonstrated the in vivo efficacy of the Sindbis virus-based pSIN vectors as DNA vaccines. A single intramuscular immunization of BALB/c mice with pSIN vectors expressing the glycoprotein B of herpes simplex virus type 1 induced a broad spectrum of immune responses, including virus-specific antibodies, cytotoxic T cells, and protection from lethal virus challenge in two different murine models. In addition, dosing studies demonstrated that the pSIN vectors were superior to a conventional plasmid DNA vector in the induction of all immune parameters tested. In general, 100- to 1,000-fold-lower doses of pSIN were needed to induce the same level of responsiveness as that achieved with the conventional plasmid DNA vector. In some instances, significant immune responses were induced with a single dose of pSIN as low as 10 ng/mouse. These results indicate the potential usefulness of alphavirus-based vectors for DNA immunization in general and more specifically as a herpes simplex virus vaccine.

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Tsai, Chi-Wei, Adib Rowhani, Deborah A. Golino, Kent M. Daane und Rodrigo P. P. Almeida. „Mealybug Transmission of Grapevine Leafroll Viruses: An Analysis of Virus–Vector Specificity“. Phytopathology® 100, Nr. 8 (August 2010): 830–34. http://dx.doi.org/10.1094/phyto-100-8-0830.

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To understand ecological factors mediating the spread of insect-borne plant pathogens, vector species for these pathogens need to be identified. Grapevine leafroll disease is caused by a complex of phylogenetically related closteroviruses, some of which are transmitted by insect vectors; however, the specificities of these complex virus–vector interactions are poorly understood thus far. Through biological assays and phylogenetic analyses, we studied the role of vector-pathogen specificity in the transmission of several grapevine leafroll-associated viruses (GLRaVs) by their mealybug vectors. Using plants with multiple virus infections, several virus species were screened for vector transmission by the mealybug species Planococcus ficus and Pseudococcus longispinus. We report that two GLRaVs (-4 and -9), for which no vector transmission evidence was available, are mealybug-borne. The analyses performed indicated no evidence of mealybug–GLRaV specificity; for example, different vector species transmitted GLRaV-3 and one vector species, Planococcus ficus, transmitted five GLRaVs. Based on available data, there is no compelling evidence of vector–virus specificity in the mealybug transmission of GLRaVs. However, more studies aimed at increasing the number of mealybug species tested as vectors of different GLRaVs are necessary. This is especially important given the increasing importance of grapevine leafroll disease spread by mealybugs in vineyards worldwide.

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Gildow, F. E., D. A. Shah, W. M. Sackett, T. Butzler, B. A. Nault und S. J. Fleischer. „Transmission Efficiency of Cucumber mosaic virus by Aphids Associated with Virus Epidemics in Snap Bean“. Phytopathology® 98, Nr. 11 (November 2008): 1233–41. http://dx.doi.org/10.1094/phyto-98-11-1233.

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Cucumber mosaic virus (CMV) is a major component of the virus complex that has become more pronounced in snap bean in the midwestern and northeastern United States since 2001. Multiple-vector-transfer tests were done to estimate the CMV transmission efficiencies (p) of the main aphid species identified in commercial snap bean fields in New York and Pennsylvania. The four most efficient vectors (p > 0.05) were Aphis gossypii, A. glycines, Acyrthosiphon pisum, and Therioaphis trifolii, which were all significant species in the migratory aphid populations in snap bean. Moderately efficient vectors (0.01 < p < 0.04) were A. spiraecola, A. craccivora, Macrosiphum euphorbiae, and Rhopalosiphum maidis. Poor vectors (p < 0.01) included A. fabae, Nearctaphis bakeri, and Myzus persicae. Only one species, Sitobion avenae, failed to transmit CMV in replicated tests. Estimates of p were consistent between different clones of the same aphid species and among three different field isolates of CMV tested. Single-vector-transfer test results for a subset of the species supported those obtained via the multiple-vector-transfer approach. Our results are consistent with the notion that A. glycines is a major vector of recent CMV epidemics in snap bean, but that species is only one of several that are involved.

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Dissertationen zum Thema "VECTOR DE VIRUS"

Mills, Mary Katherine. „Vector-pathogen interactions within the vector, Culicoides sonorensis“. Diss., Kansas State University, 2017. http://hdl.handle.net/2097/38154.

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Doctor of Philosophy
Division of Biology
Kristin Michel
The biting midge, Culicoides sonorensis, vectors orbiviruses of economic importance, such as epizootic hemorrhagic disease virus (EHDV). Due to the limitations in available molecular tools, critical Culicoides-orbivirus interactions underlying vector competence remain unclear. To provide a foundation for the study of midge-EHDV interactions, RNA interference (RNAi) was developed as a reverse genetic tool, and EHDV-2 infection dynamics were determined within C. sonorensis. To develop RNAi, exogenous double-stranded RNA (dsRNA) was injected into C. sonorensis adults specific to the C. sonorensis inhibitor of apoptosis protein 1 (CsIAP1) ortholog (dsCsIAP1). A significant decrease in CsIAP1 transcripts was observed in whole midges, with highest reduction in the midgut. In addition, dsCsIAP1-injected midges had increased mortality, a loss of midgut tissue integrity, and increased caspase activity. The longevity and midgut phenotypes were partially reversed by the co-injection of dsRNA specific to the C. sonorensis initiator caspase Dronc ortholog and CsIAP1. These results demonstrated that RNAi can be achieved in the midge midgut through injection of target dsRNAs into the hemolymph. Furthermore, the time course of EHDV-2 infection within C. sonorensis was characterized. EHDV-2 infection was observed in the midgut and secondary tissues, including the salivary glands, by 5 days post-feeding (dpf). These data are consistent with dissemination of EHDV-2 to secondary susceptible tissues throughout the midge via the hemolymph and indicate that virus transmission by C. sonorensis may occur as early as 5 dpf. This work provides a foundation for the future study of Culicoides-orbivirus interactions, including the antiviral role of RNAi at the midgut barrier.

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Gaafar, Yahya Zakaria Abdou [Verfasser]. „Plant virus identification and virus-vector-host interactions / Yahya Zakaria Abdou Gaafar“. Göttingen : Niedersächsische Staats- und Universitätsbibliothek Göttingen, 2019. http://d-nb.info/1220909262/34.

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Everett, Anthany Laurence. „Virus vector gene inserts are stabilized in the presence of satellite panicum mosaic virus coat protein“. [College Station, Tex. : Texas A&M University, 2008. http://hdl.handle.net/1969.1/ETD-TAMU-3043.

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Russell, Rebecca Alice. „Prototype foamy virus gene expression and hybrid vector development“. Thesis, Imperial College London, 2003. http://ethos.bl.uk/OrderDetails.do?uin=uk.bl.ethos.408262.

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Draper, Simon J. „Development of virus vector-based blood-stage Malaria Vaccines“. Thesis, University of Oxford, 2006. http://ethos.bl.uk/OrderDetails.do?uin=uk.bl.ethos.509922.

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McAleer, Barry E. „Expression of mumps virus proteins in eukaryote vector systems“. Thesis, Queen's University Belfast, 1997. http://ethos.bl.uk/OrderDetails.do?uin=uk.bl.ethos.263462.

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Sweeney, Nathan Paul. „Foamy virus vector integration and application in gene therapy“. Thesis, Imperial College London, 2015. http://hdl.handle.net/10044/1/50704.

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Foamy viruses (FVs) are unique ancient retroviruses that infect all non-human primates, but do not cause disease. We aimed to understand the FV pre-integration complex by isolating it from infected cells and characterising its protein constituents. Using a PCR to quantify integration in infected cells, we determined that integration occurs from 10 hours post-transduction. In synchronised cells, the peak of integration correlated well with cells passing through mitosis. However, we were unable to detect in vitro strand-transfer activity to indicate that active pre-integration complexes had been isolated. We conclude that FV pre-integration complexes are likely to be inactive in the conditions tested. A further aim was to optimise FV vectors for use in mesenchymal stem cells and test this vector in mouse models of sphingolipidoses, namely metachromatic leukodystrophy and Krabbe disease. We permitted transduction of cells at a high multiplicity of infection by exchanging the envelope from the prototype FV to that of the macaque. We tested various FV vectors in mesenchymal stem cells and determined that the non-toxic macaque envelope increased transduction efficiency from under 65% to over 95% in a single round of transduction. We achieved high and sustained transgene expression using the phosphoglycerate kinase promoter. Transduced MSCs delivered to the brains of the mouse model for metachromatic leukodystrophy caused only a modest improvement in sulphatide storage, the primary biochemical output for efficacy, although results are inconclusive. In the mouse model for Krabbe disease, transduced MSCs delivered to the brain or the peritoneum had no effect on disease progression. In conclusion, FV vectors are suited to gene therapy of MSCs since they offer the highest transduction efficiency from a single round of transduction, while MSC based gene therapy strategies for Krabbe disease or metachromatic leukodystrophy are unlikely to offer clinical benefit.

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Hu, Nai-Chung. „The development of penguinpox virus (PEPV) as a vaccine vector : transfer vector construction and rescue of virus growth in rabbit kidney cells (RK-13) by vaccinia virus K1“. Master's thesis, University of Cape Town, 2010. http://hdl.handle.net/11427/10687.

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Includes bibliographical references (leaves 131-137).
Of the many vaccine trials which have taken place, the most promising results have been obtained from the recent phase 3 clinical trial which tested the ability of a dual protein and Canarypox virus recombinant to protect humans against HIV-1 infections. Because poxviruses are being developed as vaccine vectors against a number of diseases, it is important to continue the search for novel poxvirus vectors, in particular, those that do not cross-neutralize one another. This thesis describes the preliminary work performed on the development of Penguinpox virus (PEPV) as a vaccine vector.

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Pizzato, Massimo. „Retroviral vectors for gene therapy : characterisation of vector particle-cell interaction and development of novel packaging cell lines“. Thesis, Institute of Cancer Research (University Of London), 2000. http://ethos.bl.uk/OrderDetails.do?uin=uk.bl.ethos.313365.

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Twiddy, Sally Susanna. „The molecular epidemiology and evolution of dengue virus“. Thesis, University of Oxford, 2002. http://ethos.bl.uk/OrderDetails.do?uin=uk.bl.ethos.269490.

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Bücher zum Thema "VECTOR DE VIRUS"

Nagai, Yoshiyuki, Hrsg. Sendai Virus Vector. Tokyo: Springer Japan, 2013. http://dx.doi.org/10.1007/978-4-431-54556-9.

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Mukhopadhyay, S. Plant virus, vector epidemiology and management. Enfield, NH: Science Publishers, 2010.

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Mukhopadhyay, S. Plant virus, vector epidemiology and management. Enfield, NH: Science Publishers, 2010.

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Mukhopadhyay, S. Plant virus, vector epidemiology and management. Enfield, NH: Science Publishers, 2010.

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Nebbache, Salim. The virus-vector relationship of maize streak virus with Cicadulina leafhoppers. Norwich: University ofEast Anglia, 1988.

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Decraemer, W. The Family Trichodoridae: Stubby Root and Virus Vector Nematodes. Dordrecht: Springer Netherlands, 1995. http://dx.doi.org/10.1007/978-94-015-8482-1.

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Decraemer, W. The family Trichodoridae: Stubby root and virus vector nematodes. Dordrecht: Kluwer Academic Publishers, 1995.

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International Symposium on Viruses with Fungal Vectors (1987 St. Andrews University). Viruses with fungal vectors. Wellesbourne, Warwick: Association of Applied Biologists, 1988.

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Ghose, Abhijit. The canarypox virus ALVAC as a vector in cancer gene therapy. Ottawa: National Library of Canada, 1999.

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Viral vectors for gene therapy: Methods and protocols. New York: Humana Press, 2011.

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Buchteile zum Thema "VECTOR DE VIRUS"

Díaz-Menéndez, Marta, und Clara Crespillo-Andújar. „The Vector“. In Zika Virus Infection, 21–30. Cham: Springer International Publishing, 2017. http://dx.doi.org/10.1007/978-3-319-59406-4_4.

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Conzelmann, Karl-Klaus. „Reverse Genetics of Mononegavirales: The Rabies Virus Paradigm“. In Sendai Virus Vector, 1–20. Tokyo: Springer Japan, 2013. http://dx.doi.org/10.1007/978-4-431-54556-9_1.

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Nagai, Yoshiyuki, und Atsushi Kato. „Sendai Virus Biology and Engineering Leading up to the Development of a Novel Class of Expression Vector“. In Sendai Virus Vector, 21–68. Tokyo: Springer Japan, 2013. http://dx.doi.org/10.1007/978-4-431-54556-9_2.

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Iida, Akihiro, und Makoto Inoue. „Concept and Technology Underlying Sendai Virus (SeV) Vector Development“. In Sendai Virus Vector, 69–89. Tokyo: Springer Japan, 2013. http://dx.doi.org/10.1007/978-4-431-54556-9_3.

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Wiegand, Marian, und Wolfgang J. Neubert. „Genome Replication-Incompetent Sendai Virus Vaccine Vector Against Respiratory Viral Infections That Is Capable of Eliciting a Broad Spectrum of Specific Immune Response“. In Sendai Virus Vector, 91–126. Tokyo: Springer Japan, 2013. http://dx.doi.org/10.1007/978-4-431-54556-9_4.

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Seki, Sayuri, und Tetsuro Matano. „Development of Vaccines Using SeV Vectors Against AIDS and Other Infectious Diseases“. In Sendai Virus Vector, 127–49. Tokyo: Springer Japan, 2013. http://dx.doi.org/10.1007/978-4-431-54556-9_5.

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Yonemitsu, Yoshikazu, Yasuji Ueda und Mamoru Hasegawa. „BioKnife, a Modified Sendai Virus, to Resect Malignant Tumors“. In Sendai Virus Vector, 151–69. Tokyo: Springer Japan, 2013. http://dx.doi.org/10.1007/978-4-431-54556-9_6.

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Fusaki, Noemi, und Hiroshi Ban. „Induction of Human Pluripotent Stem Cells by the Sendai Virus Vector: Establishment of a Highly Efficient and Footprint-Free System“. In Sendai Virus Vector, 171–83. Tokyo: Springer Japan, 2013. http://dx.doi.org/10.1007/978-4-431-54556-9_7.

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Yonemitsu, Yoshikazu, Takuya Matsumoto und Yoshihiko Maehara. „Gene Therapy for Peripheral Arterial Disease Using Sendai Virus Vector: From Preclinical Studies to the Phase I/IIa Clinical Trial“. In Sendai Virus Vector, 185–99. Tokyo: Springer Japan, 2013. http://dx.doi.org/10.1007/978-4-431-54556-9_8.

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Butter, N. S. „Vector-Virus Management“. In Insect Vectors and Plant Pathogens, 397–428. Boca Raton, FL : CRC Press, Taylor & Francis Group, [2018]: CRC Press, 2018. http://dx.doi.org/10.1201/9780429503641-15.

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Konferenzberichte zum Thema "VECTOR DE VIRUS"

Chisholm, Paul Joseph. „Competition with non-vectors mediates virus-vector interactions“. In 2016 International Congress of Entomology. Entomological Society of America, 2016. http://dx.doi.org/10.1603/ice.2016.115741.

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Redinbaugh, Margaret (Peg). „Vector-virus interactions in maize agroecosystems in East Africa“. In 2016 International Congress of Entomology. Entomological Society of America, 2016. http://dx.doi.org/10.1603/ice.2016.94561.

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Ayres, Constância. „Tracking the incrimination ofAedes aegyptias a Zika virus vector“. In 2016 International Congress of Entomology. Entomological Society of America, 2016. http://dx.doi.org/10.1603/ice.2016.109197.

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Cristea, Paul Dan. „Phase and Vector Analysis of H5N1 Avian Influenza Virus“. In 2006 8th Seminar on Neural Network Applications in Electrical Engineering. IEEE, 2006. http://dx.doi.org/10.1109/neurel.2006.341191.

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Raafat, Nermin, Chantal Mengus, Michael Heberer, Giulio C. Spagnoli und Paul Zajac. „Abstract 1500: Modulation of recombinant vaccinia virus vector immunogenicity“. In Proceedings: AACR 101st Annual Meeting 2010‐‐ Apr 17‐21, 2010; Washington, DC. American Association for Cancer Research, 2010. http://dx.doi.org/10.1158/1538-7445.am10-1500.

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Morales-Hojas, Ramiro. „Genomics of bluetongue virus vector competence inCulicoides sonorensis(Diptera: Ceratopogonidae)“. In 2016 International Congress of Entomology. Entomological Society of America, 2016. http://dx.doi.org/10.1603/ice.2016.109119.

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Gadhave, Kiran Ramesh. „Potyvirus transmission and vector-virus interactions in cucurbit production systems“. In 2016 International Congress of Entomology. Entomological Society of America, 2016. http://dx.doi.org/10.1603/ice.2016.112843.

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Muik, Alexander, Janine Kimpel, Reinhard Tober, Carles Urbiola und Dorothee von Laer. „Abstract B37: VSV-GP: A vaccine vector and oncolytic virus“. In Abstracts: AACR Special Conference: Tumor Immunology and Immunotherapy: A New Chapter; December 1-4, 2014; Orlando, FL. American Association for Cancer Research, 2015. http://dx.doi.org/10.1158/2326-6074.tumimm14-b37.

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Sanders, Christopher. „Culicoidesand reassortant bluetongue viruses: A study of virus/vector/host interactions“. In 2016 International Congress of Entomology. Entomological Society of America, 2016. http://dx.doi.org/10.1603/ice.2016.106585.

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„Analysis of the Genomes of Chikungunya Virus and Dengue Virus Using Decision Tree, Apriori Algorithm, and Support Vector Machine“. In 2016 the 6th International Workshop on Computer Science and Engineering. WCSE, 2016. http://dx.doi.org/10.18178/wcse.2016.06.049.

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Berichte der Organisationen zum Thema "VECTOR DE VIRUS"

Durden, Lance A., Thomas M. Logan, Mark L. Wilson und Kenneth J. Linthicum. Experimental Vector Incompetence of a Soft Tick, Ornithodoros sonrai (Acari: Argasidae), for Crimean-Congo Hemorrhagic Fever Virus. Fort Belvoir, VA: Defense Technical Information Center, Januar 1993. http://dx.doi.org/10.21236/ada265568.

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Linthicum, K. J., C. L. Bailey, C. J. Tucker, K. D. Mitchell und T. M. Logan. Application of Polar-Orbiting, Meteorological Satellite Data to Detect Flooding of Rift Valley Fever Virus Vector Mosquito Habitats in Kenya. Fort Belvoir, VA: Defense Technical Information Center, Januar 1990. http://dx.doi.org/10.21236/ada233281.

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Hall, Simon J. Construction of a Vesicular Stomatitis Virus Expressing Both a Fusogenic Glycoprotein and IL-12: A Novel Vector for Prostate Cancer Therapy. Fort Belvoir, VA: Defense Technical Information Center, Januar 2006. http://dx.doi.org/10.21236/ada462813.

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Dropulic, Lesia. Development of Targeted Sindbis Virus Vectors for Potential Application to Breast Cancer Therapy. Fort Belvoir, VA: Defense Technical Information Center, September 2001. http://dx.doi.org/10.21236/ada404597.

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Dropulic, Lesia K. Development of Targeted Sindbis Virus Vectors for Potential Application to Breast Cancer Therapy. Fort Belvoir, VA: Defense Technical Information Center, September 2002. http://dx.doi.org/10.21236/ada411347.

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Dropulie, Lesia K. Development of targeted Sindbis Virus Vectors for Potential Application to Breast Cancer Therapy. Fort Belvoir, VA: Defense Technical Information Center, Oktober 2000. http://dx.doi.org/10.21236/ada392586.

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