Thematische Bibliographien / Vitamin therapy. 0

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Auswahl der wissenschaftlichen Literatur zum Thema „Vitamin therapy. 0“

Autor: Grafiati
Veröffentlicht am 4. Juni 2021
Zuletzt aktualisiert am 29. Juli 2025

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Zeitschriftenartikel zum Thema "Vitamin therapy. 0"

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Chang, Huan-Cheng, Chien-Lung Chen, Te-Li Chiu, Shu-I. Chen, Amy Ming-Fang Yen, and Tony Hsiu-Hsi Chen. "Effectiveness of recombinant human erythropoietin, vitamin D3and iron therapy on long-term survival of patients with end-stage renal disease receiving haemodialysis: analysis of 702 patients after 10-year follow-up." Public Health Nutrition 12, no. 12 (2009): 2410–15. http://dx.doi.org/10.1017/s1368980009005308.

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AbstractObjectiveFew studies have been conducted to investigate the influence of recombinant human erythropoietin (rhEPO) on the long-term prognosis of end-stage renal disease (ESRD).DesignA retrospective cohort study.SettingThe largest regional hospital renowned for haemodialysis in northern Taiwan.SubjectsA total of 702 ESRD patients undergoing haemodialysis between 1993 and 2002 were evaluated.ResultsThe rate of overall use of rhEPO, vitamin D3or Fe therapy was 62 %. The 10-year survival rate in patients with rhEPO supplementation was statistically more favourable than that in patients with
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2

GARG, RAVENDRA, NIRPENDRA SINGH, and ANURADHA DUBE. "Intake of nutrient supplements affects multiplication ofLeishmania donovaniin hamsters." Parasitology 129, no. 6 (2004): 685–91. http://dx.doi.org/10.1017/s0031182004006055.

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The role of the essential nutrients, vitamins A,B (complex), C and E and iron, as prophylactic as well as supportive therapy in experimental visceral leishmaniasis (VL), was studied in hamsters. Prophylactic administration of vitamin C (50, 100 and 250 mg/kg) from day15 to day 0 (15 doses) significantly reduced the intake ofLeishmania donovaniin hamsters but had no therapeutic effect. In contrast, vitamins A,B complex and E and iron, whether used prophylactically or therapeutically, promoted parasite multiplication. The efficacy of sodium stibogluconate, a reference antileishmanial drug, was a
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Bayewitz, Ashrei, Barbara Scorziello, Clarice Maala, Emily Giannattasio, and Henny Heisler Billett. "Effects of Preoperative Vitamin K On the INR in Bridging Therapy." Blood 120, no. 21 (2012): 3407. http://dx.doi.org/10.1182/blood.v120.21.3407.3407.

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Abstract Abstract 3407 Patients who require interruption of their warfarin therapy before surgery need a safe and effective bridging protocol. Objective: We investigated whether routine incorporation of oral Vitamin K three days before surgery would improve perioperative management. Patients/Methods: Patients on chronic warfarin therapy requiring perioperative bridging therapy were the participants in this observational study. Results from two bridging protocols, 30 cases each, with and without oral Vitamin K, were analyzed. Day of Surgery was considered Day 0. The first cohort (control arm) o
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Okolonkwo, Benjamin Nnamdi, Maureen Ibitoroko George-Opuda, Doris Nnenna Ajibo, Clement Ugochukwu Nyenke, and Kingsley Kalawari Odiabara. "Study on Antidotal Therapy of Vitamins in Paraquat Induced Dyslipidemia in Rats." Journal of Applied Health Sciences and Medicine 3, no. 7 (2023): 11–18. http://dx.doi.org/10.58614/jahsm372.

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Paraquat (PQ) is a toxic chemical substance used in farming for weed management. It is an herbicide with harmful effect when exposed to animals. This study aimed at assessing the effect of vitamin E and C on fasting lipid profile in paraquat induced toxicity in rats. The experimental study consisted of 180 rats divided equally into 4 groups (A, B, C and D) with each group having 3 subgroups of 15 rats (0, VE and VEC). A group served as the control group while B, C, D were test groups. B0, C0 and D0 were treated with 0.02g, 0.04g and 0.06g of paraquat respectively twice in two weeks for 3 month
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White, K. L. M., D. M. Chalmers, I. G. Martin, et al. "Dietary antioxidants and DNA damage in patients on long-term acid-suppression therapy: a randomized controlled study." British Journal of Nutrition 88, no. 3 (2002): 265–71. http://dx.doi.org/10.1079/bjn2002619.

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Free radicals and reactive species producedin vivocan trigger cell damage and DNA modifications resulting in carcinogenesis. Dietary antioxidants trap these species limiting their damage. The present study evaluated the role of vitamins C and E in the prevention of potentially premalignant modifications to DNA in the human stomach by supplementing patients who, because of hypochlorhydria and possible depletion of gastric antioxidants, could be at increased risk of gastric cancer. Patients undergoing surveillance for Barrett's oesophagus (n100), on long-term proton pump inhibitors were randomiz
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Wei, Shi, Hiroyuki Tanaka, Toshihide Kubo, Taeko Ono, Susumu Kanzaki, and Yoshiki Seino. "Growth hormone increases serum 1,25-dihydroxyvitamin D levels and decreases 24,25-dihydroxyvitamin D levels in children with growth hormone deficiency." European Journal of Endocrinology 136, no. 1 (1997): 45–51. http://dx.doi.org/10.1530/eje.0.1360045.

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Abstract The influence of growth hormone (GH) on calcium–phosphorus metabolism and modulation of vitamin D metabolism has been demonstrated, but the mechanism remains unclear. We investigated the effect of a 6-month course of GH therapy on vitamin D and mineral metabolism in twelve GH-deficient children. Before GH therapy, levels of vitamin D metabolites and other biochemistry data were within normal ranges. All patients responded to GH therapy with increased growth velocity. 1,25-Dihydroxyvitamin D levels increased after 1 month of treatment and remained at these higher levels, with a signifi
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Guzman-Prado, Yuli, Ondrej Samson, Jonathan P. Segal, Jimmy K. Limdi, and Bu’Hussain Hayee. "Vitamin D Therapy in Adults With Inflammatory Bowel Disease: A Systematic Review and Meta-Analysis." Inflammatory Bowel Diseases 26, no. 12 (2020): 1819–30. http://dx.doi.org/10.1093/ibd/izaa087.

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Abstract Background Vitamin D deficiency has been implicated in the pathogenesis of inflammatory bowel disease. Emerging literature suggests that optimization of vitamin D levels may be associated with improvements in disease activity and quality of life. We conducted a meta-analysis exploring the effect of vitamin D on serum 25-hydroxyvitamin D (s-25[OH]D) levels, clinical improvement, and biomarkers. Methods MEDLINE, EMBASE, the Cochrane Library, and sources for grey literature were searched from inception until September 2019. The primary outcome was s-25(OH)D mean differences. Heterogeneit
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Casper, Howard H., Arnold D. Alstad, David B. Tacke, Ladon J. Johnson, and W. Eugene Lloyd. "Evaluation of Vitamin K3 Feed Additive for Prevention of Sweet Clover Disease." Journal of Veterinary Diagnostic Investigation 1, no. 2 (1989): 116–19. http://dx.doi.org/10.1177/104063878900100204.

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Sweet clover poisoning in cattle is caused by an anticoagulant (dicumarol) that is formed in moldy sweet clover hay. Previous experiments with vitamin K3 and vitamin K3 in therapy trials indicated that vitamin K3 was effective in reducing prothrombin times but vitamin K3 was not. As a possible alternative in the use of toxic sweet clover hays, vitamin K3 was evaluated to see if it would prevent hemorrhagic crises when fed to cattle consuming toxic sweet clover hay. Vitamin K3 levels of 0, 0.45, 4.5, 11, and 45 mg/kg body weight/day were fed to 173-235-kg steers consuming toxic (40–50 ppm dicum
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Ayres, Samuel. "Yellow nail syndrome controlled by vitamin E therapy." Journal of the American Academy of Dermatology 15, no. 4 (1986): 714–15. http://dx.doi.org/10.1016/s0190-9622(86)80105-0.

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Day, Andrew S., Daniel A. Lemberg, Rachel Messenger, and Helen J. Woodhead. "High Dose Vitamin D Therapy in Paediatric IBD." Gastroenterology 140, no. 5 (2011): S—512. http://dx.doi.org/10.1016/s0016-5085(11)62124-0.

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Bücher zum Thema "Vitamin therapy. 0"

1

Bricklin, Mark. Dictionnaire des remèdes naturels. Québec/Amérique, 1985.

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2

Reichrath, Jörg, Wolfgang Tilgen, and Michael Friedrich, eds. Vitamin D Analogs in Cancer Prevention and Therapy. Springer Berlin Heidelberg, 2003. http://dx.doi.org/10.1007/978-3-642-55580-0.

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Pauling, Linus. Das Vitamin-Programm: Topfit bis ins hohe Alter. Goldmann, 1992.

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Pauling, Linus. Linus Pauling's Vitamin-Programm: Plädoyer für ein gesundes Leben. Bertelsmann, 1990.

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Pauling, Linus. Como viver mais e melhor: O que os médicos não dizem sobre sua saúde. Editora Best Seller, 1988.

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Pauling, Linus. Abusez des vitamines! Tchou, 1988.

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Pauling, Linus. Pōringu hakushi no kaiteki chōjugaku. Heibonsha, 1987.

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Pauling, Linus. How to live longer and feel better. Avon Books, 1987.

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Pauling, Linus. How to live longer and feel better. W.H. Freeman, 1986.

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Pauling, Linus. How to Live Longer and Feel Better. Avon Books, 1996.

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Buchteile zum Thema "Vitamin therapy. 0"

1

Helfrich, Yolanda R., Ginette A. Okoye, Dana L. Sachs, and Sewon Kang. "Topical Vitamin D3." In Comprehensive Dermatologic Drug Therapy. Elsevier, 2021. http://dx.doi.org/10.1016/b978-0-323-61211-1.00049-8.

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Bellorin-Font, Ezequiel, Thanh-Mai Vo, and Kevin J. Martin. "Use of Vitamin D Sterols and Calcimimetics in Patients With End-Stage Renal Disease." In Handbook of Dialysis Therapy. Elsevier, 2023. http://dx.doi.org/10.1016/b978-0-323-79135-9.00041-0.

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Labyed, Marwah, Najwa Hassou, Mohammed El Mzibri, and Moulay Mustapha Ennaji. "Vitamin D new therapy for breast cancer prevention." In Oncogenic Viruses. Elsevier, 2023. http://dx.doi.org/10.1016/b978-0-12-824156-1.00010-8.

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Offenberger, Holly M., and Ronald Ross Watson. "Vitamin A Supplementation Therapy for Patients with Cystic Fibrosis." In Diet and Exercise in Cystic Fibrosis. Elsevier, 2015. http://dx.doi.org/10.1016/b978-0-12-800051-9.00019-5.

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Vo, Thanh-Mai, and Kevin J. Martin. "Use of Vitamin D Sterols and Calcimimetics in Patients With End-Stage Renal Disease." In Handbook of Dialysis Therapy. Elsevier, 2017. http://dx.doi.org/10.1016/b978-0-323-39154-2.00061-8.

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Niino, M., and Y. Miyazaki. "Role of Vitamin D in Multiple Sclerosis Pathogenesis and Therapy." In Nutrition and Lifestyle in Neurological Autoimmune Diseases. Elsevier, 2017. http://dx.doi.org/10.1016/b978-0-12-805298-3.00009-8.

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Sherbet, G. V. "Vitamin D3 in Cell Proliferation, Apoptosis and Differentiation." In Growth Factors and Their Receptors in Cell Differentiation, Cancer and Cancer Therapy. Elsevier, 2011. http://dx.doi.org/10.1016/b978-0-12-387819-9.00018-9.

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Azu, Onyemaechi O., and Edwin C. S. Naidu. "Vitamin E and Testicular Damage Protection in Highly Active Antiretroviral Therapy." In HIV/AIDS. Elsevier, 2018. http://dx.doi.org/10.1016/b978-0-12-809853-0.00019-5.

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Ramzan, Khalida, Aqib Ramzan, Abid Dar, Bisma Mohi ud din dar, and Ifra Nisar. "Vitamin D3 therapy and clinical outcome in recurrent pregnancy loss patients." In Reproductive Immunogenetics: A Molecular and Clinical Overview. Elsevier, 2025. https://doi.org/10.1016/b978-0-443-13657-3.00006-4.

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Becker, Richard C., and Frederick A. Spencer. "Anticoagulants." In Fibrinolytic and Antithrombotic Therapy. Oxford University Press, 2006. http://dx.doi.org/10.1093/oso/9780195155648.003.0040.

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Anticoagulant therapy, particularly when used in combination with fibrinolytics and less often platelet antagonists, can cause life-threatening hemorrhage. This supports the importance of coagulation proteases in several phases of thrombus development; paradoxically, several anticoagulants can also cause microvascular and macrovascular thrombotic disorders. Hemorrhage is the most common adverse effect associated with warfarin administration. To predict the risk of bleeding, Beyth and colleagues (1998) developed a 5-point scoring system, with 1 point given for each of the following: . . . • Age
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Konferenzberichte zum Thema "Vitamin therapy. 0"

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Lorenz, R., M. Holweger, GE Vogel, and M. Classen. "SUBSTITUTION THERAPY WITH ANTITHROMBIN III: CLINICAL EXPERIENCE WITH DECOMPENSATED LIVER CIRRHOSIS AND HEMORRHAGIC COMPLICATIONS." In XIth International Congress on Thrombosis and Haemostasis. Schattauer GmbH, 1987. http://dx.doi.org/10.1055/s-0038-1643069.

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In patients with decompensated liver cirrhosis, the coagulation disorder requires therapy only when bleeding complications occur. As therapy involves the risk of triggering a consumption coagulopathy, the discussion here examines the beneficial effect of boosting the inhibitor Potential by supplementing the administration of fresh plasma with substitution of AT III concentrate. To investigate this question, we conducted a retrospective study on the clinical course in 19 patients with decompensated liver cirrhosis, hepatic encephalopathy and bleeding complications.The following treatment scheme
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