Bibliografías temáticas / Atherosclerosis

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Literatura académica sobre el tema "Atherosclerosis"

Autor: Grafiati
Publicado: 4 de junio de 2021
Última modificación: 25 de julio de 2025

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Artículos de revistas sobre el tema "Atherosclerosis"

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Napieralski, Rudolf, Ernst Wagner, Harry Gebhard, et al. "Alternation of histone and DNA methylation in human atherosclerotic carotid plaques." Thrombosis and Haemostasis 114, no. 08 (2015): 390–402. http://dx.doi.org/10.1160/th14-10-0852.

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SummaryLittle is known about epigenetics and its possible role in atherosclerosis. We here analysed histone and DNA methylation and the expression of corresponding methyltransferases in early and advanced human atherosclerotic carotid lesions in comparison to healthy carotid arteries. Western Blotting was performed on carotid plaques from our biobank with early (n=60) or advanced (n=60) stages of atherosclerosis and healthy carotid arteries (n=12) to analyse di-methylation patterns of histone H3 at positions K4, K9 and K27. In atherosclerotic lesions, di-methylation of H3K4 was unaltered and t
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Li, Junxi, Xinying Fu, Renyi Yang, and Wei Zhang. "Atherosclerosis Vascular Endothelial Secretion Dysfunction and Smooth Muscle Cell Proliferation." Journal of Healthcare Engineering 2022 (March 9, 2022): 1–13. http://dx.doi.org/10.1155/2022/9271879.

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Atherosclerosis is a chronic inflammatory disease of the arterial wall and the main cause of cardiovascular disease and cerebrovascular disease. In recent years, the mortality rate of atherosclerotic diseases has become higher and higher. This article aims to study the dysregulation of atherosclerotic vascular endothelial secretion and smooth muscle cell proliferation, and put forward and practice the pathological research of atherosclerotic disease. This article describes in detail atherosclerosis, endothelial dysfunction, and smooth muscle cell proliferation, and studies the causes of athero
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Mohmand-Borkowski, Adam, Dareus O. Conover, and Tomasz Rozmyslowicz. "Fibroblast Activation Protein Compared with Other Markers of Activated Smooth Muscle Cells, Extracellular Matrix Turnover and Inflammation in a Mouse Model of Atherosclerosis." Metabolites 15, no. 4 (2025): 243. https://doi.org/10.3390/metabo15040243.

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Background: Fibroblast activation protein (FAP) is a cell surface glycoprotein expressed by myofibroblastic cells in areas of active tissue remodeling, such as wound healing, fibrosis, and certain chronic inflammatory lesions. As FAP is uniquely present in chronic inflammatory lesions and has an important role in extracellular matrix (ECM) turnover, it appears to have all the characteristics necessary for involvement in atherosclerosis and atherosclerotic plaque rupture and has become a potential target in the treatment of myocardial infarction. Methods: To further understand the role of FAP,
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Moatassem, Salah Amer*1 Randa Abdel WahabReda Mabrouk2 Omar Hussein Omar3 Tarekkhairy Abdel Dayem4 Samia Ahmed Abdul-Rahman5& Basma Kamel Abdel Aziz6. "FRAILTY AND SUBCLINICAL ATHEROSCLEROSIS." Indian Journal of Medical Research and Pharmaceutical Sciences 4, no. 3 (2017): 1–9. https://doi.org/10.5281/zenodo.343751.

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<strong>Background:</strong> Frailty as well as atherosclerotic diseases are commonly prevalent among elderly subjects and usually share both complex pathway and risk factors. Hence it was important to study if there is an association between frailty and risk factors of atherosclerosisat its early stages (subclinical atherosclerosis) using the Ankle Brachial Index (ABI), carotid intima media thickness (CIMT) and Abdominal Aortic Diameter (AAD) as markers of subclinical atherosclerosis. <strong>Material and Methods:</strong> A cross-sectional study conducted on 90 elderly participants (60-year-
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Šačić, Dalila, Sofija Glumac, and Sanja Radojević-Škodrić. "The significance of inflammation in pathogenesis, diagnosis and treatment of atherosclerosis." Medicinski podmladak 76, no. 1 (2025): 42–47. https://doi.org/10.5937/mp76-48564.

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Atherosclerosis is a chronic disease whose progression starts from birth. The basic substrate of an atherosclerotic lesion is the deposition of lipids in the intima of the blood vessel, which results in gradual narrowing and impaired blood flow through the tissues. The clinical manifestations of the atherosclerotic process are numerous and they depend on the degree of narrowing and the speed of the stenosis. The most common clinical manifestations are the result of rupture of the atherosclerotic plaque, which activates the process of thrombogenesis and leads to the development of blockage of b
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Xia, Mingcan, Kangkang Yang, Nadia Guerra, et al. "NKG2D/ligands-mediated immune activation promotes atherosclerosis (142.7)." Journal of Immunology 184, no. 1_Supplement (2010): 142.7. http://dx.doi.org/10.4049/jimmunol.184.supp.142.7.

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Abstract Atherosclerosis, a chronic vascular disease, is characterized by deposition of fatty substances and accumulation of immune cells in susceptible regions of arteries. Increasing evidence show that abnormal metabolic conditions, such as dyslipdemia and diabetes, induce immune cell activation and inflammation, which play crucial roles in progression of atherosclerosis. However, molecular linkers of metabolic disorder, immune activation and atherosclerosis are not well understood. Here we report that ligands for NKG2D, an immune-activating receptor, were upregulated in multiple tissues and
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Konstantinova, E. V., A. A. Bogdanova, A. A. Sagatelyan, A. I. Kovaikin, E. S. Pershina, and M. Yu Gilyarov. "Features of atherosclerosis in carotid and coronary arteries." Meditsinskiy sovet = Medical Council, no. 14 (October 18, 2021): 44–53. http://dx.doi.org/10.21518/2079-701x-2021-14-44-53.

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Atherothrombosis is a leading cause of myocardial infarction and ischemic atherothrombotic stroke. It represents a stage of atherosclerosis which is a pathologic process throughout the circulatory system. However, atherosclerosis has specific development characteristics in different vascular beds. Multiple factors contribute to atherosclerosis formation and progression such as genetic factors, vessel hemodynamics, and vessel anatomy. A better understanding of differences in vessels would improve prevention and treatment of atherosclerosis and its complication. In this article we review feature
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Juricic, Stefan, Jovana Klac, Sinisa Stojkovic, et al. "Molecular and Pathophysiological Mechanisms Leading to Ischemic Heart Disease in Patients with Diabetes Mellitus." International Journal of Molecular Sciences 26, no. 9 (2025): 3924. https://doi.org/10.3390/ijms26093924.

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Coronary atherosclerosis in patients with diabetes mellitus is the most significant pathophysiological mechanism responsible for ischemic heart disease. Atherosclerosis in diabetes is premature, more diffuse, and more progressive, and it affects more coronary blood vessels compared to non-diabetics. Atherosclerosis begins with endothelial dysfunction, continues with the formation of fatty streaks in the intima of coronary arteries, and ends with the appearance of an atherosclerotic plaque that expands centrifugally and remodels the coronary artery. If the atherosclerotic plaque is injured, a t
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Pakzad, Bahram, Elham Rajae, Saeid Shahrabi, et al. "T-Cell Molecular Modulation Responses in Atherosclerosis Anergy." Laboratory Medicine 51, no. 6 (2020): 557–65. http://dx.doi.org/10.1093/labmed/lmaa003.

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Abstract Atherosclerosis continues to be a major cause of death in patients with cardiovascular diseases. The cooperative role of immunity has been recently considered in atherosclerotic plaque inflammation, especially adaptive immune response by T cells. In this review, we examine the possible role of T cells in atherosclerosis-mediated inflammation and conceivable therapeutic strategies that can ameliorate complications of atherosclerosis. The cytokines secreted by T-lymphocyte subsets, different pathophysiological profiles of microRNAs (miRs), and the growth factor/receptor axis have divers
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Li, Yang, Yi Feng, Genshan Ma, Chengxing Shen, and Naifeng Liu. "Coronary tortuosity is negatively correlated with coronary atherosclerosis." Journal of International Medical Research 46, no. 12 (2018): 5205–9. http://dx.doi.org/10.1177/0300060518804723.

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Objective The impact of coronary tortuosity on coronary atherosclerosis remains unclear. This study was performed to determine to the relationship between coronary tortuosity and the presence of coronary atherosclerosis. Methods Tortuosity and the presence of coronary atherosclerosis in the main coronary arteries were evaluated. The coronary artery was divided into non-tortuous and tortuous segments. The incidence of coronary atherosclerosis between the two segments was compared. Results The prevalence of coronary atherosclerotic stenosis was significantly lower in the tortuous than non-tortuo
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Tesis sobre el tema "Atherosclerosis"

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Calvert, Patrick Andrew. "Virtual-histology intravascular ultrasound in vulnerable atherosclerosis." Thesis, University of Cambridge, 2011. http://ethos.bl.uk/OrderDetails.do?uin=uk.bl.ethos.609857.

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McCord, Barbara Norton. "Fatigue of atherosclerotic plaque." Diss., Georgia Institute of Technology, 1992. http://hdl.handle.net/1853/15890.

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Dennis, Maxine Elizabeth. "Oestrogen and atherosclerosis." University of Western Australia. School of Pathology and Laboratory Medicine, 2009. http://theses.library.uwa.edu.au/adt-WU2009.0134.

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[Truncated abstract] Our understanding of the actions of oestrogen on the vasculature has recently been questioned following the results of large clinical trials revealing a negative effect of hormone replacement therapy (HRT) on cardiovascular disease (CVD) risk amongst postmenopausal women. It is important to determine how a hormone with numerous positive effects on intermediate pathways of atherosclerosis fails to offer cardioprotection. Further investigation into the actions of oestrogen in the vasculature may add to our current understanding of the pathogenesis of atherosclerosis and oest
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Jatta, Ken. "Inflammation in Atherosclerosis." Doctoral thesis, Örebro : Universitetsbiblioteket, 2006. http://urn.kb.se/resolve?urn=urn:nbn:se:oru:diva-478.

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Shakya, Arvind. "Mechanism of matrix metalloproteinase-14 (mmp-14) regulation during atherosclerosis." Diss., Columbia, Mo. : University of Missouri-Columbia, 2006. http://hdl.handle.net/10355/4436.

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Thesis (Ph. D.)--University of Missouri-Columbia, 2006.<br>"December 2006" The entire dissertation/thesis text is included in the research.pdf file; the official abstract appears in the short.pdf file (which also appears in the research.pdf); a non-technical general description, or public abstract, appears in the public.pdf file. Vita. Includes bibliographical references.
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Crisby, Milita. "Cell death in atherosclerosis /." Stockholm, 1998. http://diss.kib.ki.se/1998/91-628-3191-7/.

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Ahmed, Ejaz. "Immune mechanisms in atherosclerosis /." Stockholm, 2001. http://diss.kib.ki.se/2001/91-628-4612-4/.

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Arno, Gavin. "Chlamydia Pheunomiae and atherosclerosis." Thesis, St George's, University of London, 2007. http://ethos.bl.uk/OrderDetails.do?uin=uk.bl.ethos.498342.

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Kharbanda, Rajesh Kumar. "Endothelial dysfunction in atherosclerosis." Thesis, University College London (University of London), 2003. http://ethos.bl.uk/OrderDetails.do?uin=uk.bl.ethos.409090.

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Alissa, Eman Mokbel. "Micronutrient status and atherosclerosis." Thesis, University of Surrey, 2005. http://ethos.bl.uk/OrderDetails.do?uin=uk.bl.ethos.419967.

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Libros sobre el tema "Atherosclerosis"

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Drew, Angela F. Atherosclerosis. Humana Press, 2000. http://dx.doi.org/10.1385/159259073x.

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Ramji, Dipak, ed. Atherosclerosis. Springer US, 2022. http://dx.doi.org/10.1007/978-1-0716-1924-7.

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Gotlieb, Avrum I., B. Lowell Langille, and Sergey Fedoroff, eds. Atherosclerosis. Springer US, 1991. http://dx.doi.org/10.1007/978-1-4615-3754-0.

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Wang, Hong, and Cam Patterson, eds. Atherosclerosis. John Wiley & Sons, Inc, 2015. http://dx.doi.org/10.1002/9781118828533.

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T, Lee K., Albany Medical College. Dept. of Pathology., and Saratoga Springs Conference on Atherosclerosis (1984), eds. Atherosclerosis. New York Academy of Sciences, 1985.

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Watanabe, Teruo, and Jianglin Fan. Atherosclerosis. Springer Nature Singapore, 2025. https://doi.org/10.1007/978-981-96-3675-4.

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Saratoga International Conference on Atherosclerosis (4th 1996 Hawaii). Atherosclerosis IV: Recent advances in atheroslcerosis research : the fourth Saratoga International Conference on Atherosclerosis. The New York Academy of Sciences, 1997.

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George, Sarah J., and Johnson Jason. Atherosclerosis: Molecular and cellular mechanisms. Wiley-VCH, 2010.

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Kim, Jong S., Louis R. Caplan, and K. S. Lawrence Wong, eds. Intracranial Atherosclerosis. Wiley-Blackwell, 2008. http://dx.doi.org/10.1002/9781444300673.

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Naghavi, Morteza, ed. Asymptomatic Atherosclerosis. Humana Press, 2011. http://dx.doi.org/10.1007/978-1-60327-179-0.

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Capítulos de libros sobre el tema "Atherosclerosis"

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Carlson, Lars A. "Atherosclerosis and clinical atherosclerosis." In Comprehensive lipid testing and management. Springer Healthcare Ltd., 2011. http://dx.doi.org/10.1007/978-1-908517-33-3_1.

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Hristov, Michael, and Christian Weber. "Atherosclerosis." In Arterial Disorders. Springer International Publishing, 2015. http://dx.doi.org/10.1007/978-3-319-14556-3_3.

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Roth, Elliot J. "Atherosclerosis." In Encyclopedia of Clinical Neuropsychology. Springer International Publishing, 2018. http://dx.doi.org/10.1007/978-3-319-57111-9_2159.

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Paeng, Jin-Chul. "Atherosclerosis." In Clinical PET and PET/CT. Springer New York, 2012. http://dx.doi.org/10.1007/978-1-4419-0802-5_21.

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Roth, Elliot J. "Atherosclerosis." In Encyclopedia of Clinical Neuropsychology. Springer New York, 2011. http://dx.doi.org/10.1007/978-0-387-79948-3_2159.

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Carter, Jennifer. "Atherosclerosis." In Encyclopedia of Behavioral Medicine. Springer International Publishing, 2020. http://dx.doi.org/10.1007/978-3-030-39903-0_1213.

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Abrams, David B., J. Rick Turner, Linda C. Baumann, et al. "Atherosclerosis." In Encyclopedia of Behavioral Medicine. Springer New York, 2013. http://dx.doi.org/10.1007/978-1-4419-1005-9_1213.

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Vuong, Phat N., and Colin Berry. "Atherosclerosis." In The Pathology of Vessels. Springer Paris, 2002. http://dx.doi.org/10.1007/978-2-8178-0786-7_4.

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Roth, Elliot J. "Atherosclerosis." In Encyclopedia of Clinical Neuropsychology. Springer International Publishing, 2018. http://dx.doi.org/10.1007/978-3-319-56782-2_2159-2.

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Kaltenbach, Martin, and Ronald E. Vlietstra. "Atherosclerosis." In Concise Cardiology. Steinkopff, 1991. http://dx.doi.org/10.1007/978-3-642-85407-1_4.

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Actas de conferencias sobre el tema "Atherosclerosis"

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Brown, Thomas. "Atherosclerosis." In ACM SIGGRAPH 2009 Computer Animation Fesitval. ACM Press, 2009. http://dx.doi.org/10.1145/1596685.1596697.

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Cruz, Mariana Portella Lopes, João Gustavo dos Anjos Morais Oliveira, Gabriela Sarno Brandão, et al. "Influence of atherosclerosis on intracranial aneurysm rupture: a literature review." In XIII Congresso Paulista de Neurologia. Zeppelini Editorial e Comunicação, 2021. http://dx.doi.org/10.5327/1516-3180.391.

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Background: Aneurysms have their wall more susceptible to rupture. It is postulated whether the presence of atherosclerotic plaque can influence this, protecting or not. Objective: This review aims to investigate atherosclerosis (AT) as a potential risk factor for rupture of intracranial aneurysms. Design and Setting: this is a literature review, produced in Bahiana School of Medicine and Public Health. Methods: The evaluated studies were obtained in published between 2011 and 2021, using the MeSH terms with following search: ((“atherosclerosis” OR “atherogenesis” OR “atheroscleroses” OR “Vasc
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Van Doormaal, Mark A., and C. Ross Ethier. "Design Optimization of an In Vitro Shear Stress and Mass Transfer Modifier for Endothelial Cell Culture." In ASME 2009 Summer Bioengineering Conference. American Society of Mechanical Engineers, 2009. http://dx.doi.org/10.1115/sbc2009-205439.

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Atherosclerosis, a disease of the large and medium arteries, is the leading cause of death in Western countries. Fluid dynamic and mass transfer phenomena are thought to play a role in the initiation and progression of atherosclerosis [1–3]. Atherosclerotic lesions are known to localize to areas such as bifurcations and curved arterial segments where shear stress and mass transfer patterns differ from those in straight arteries. However, the precise roles of wall shear stress and mass transfer in the localization of atherosclerotic lesions are not known, and this remains an active area of rese
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Teng, Zhongzhao, Allen H. Hoffman, Jie Zheng, Pamela K. Woodard, and Dalin Tang. "Ultimate Strength of the Adventitia and Media of Human Atherosclerotic Carotid Arteries in the Axial and Circumferential Directions." In ASME 2009 Summer Bioengineering Conference. American Society of Mechanical Engineers, 2009. http://dx.doi.org/10.1115/sbc2009-204715.

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The abrupt closure of an artery by an occlusive thrombus is the main cause of myocardial infarcts and other thrombotic sequelae of atherosclerosis. This thrombosis is often associated with rupture of an atherosclerotic plaque [1,2]. Histology has shown that most rupture sites are also sites of increased mechanical stress [2]. It has been widely accepted that atherosclerosis leads to locally increased stresses in the region of lesions. However, validation of this hypothesis has been impeded by a lack of experimental data on the material strength of atherosclerotic tissues. Knowledge of mechanic
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Campbell, Ian C., Daiana Weiss, John N. Oshinski, and W. Robert Taylor. "Histological Determination of Murine Plaque Mechanics and Implications for Plaque Rupture." In ASME 2010 Summer Bioengineering Conference. American Society of Mechanical Engineers, 2010. http://dx.doi.org/10.1115/sbc2010-19295.

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Among the most common models in atherosclerosis research is the atherosclerosis-prone mouse. Genetically manipulated mouse strains such as the ApoE−/− mouse will reliably form plaques under certain conditions, and these lesions have been noted to exhibit morphological and biochemical similarities with human atherosclerotic plaques. Unlike plaques in humans, however, murine plaques are not observed to rupture and form occlusive thrombi [1]. As atherosclerosis and its complications are the leading cause of death in the modern world, a comprehensive understanding of the mechanisms of plaque disru
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Szekanecz, Z. "SP0131 Autoimmune atherosclerosis." In Annual European Congress of Rheumatology, 14–17 June, 2017. BMJ Publishing Group Ltd and European League Against Rheumatism, 2017. http://dx.doi.org/10.1136/annrheumdis-2017-eular.7194.

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Van der Heiden, K., H. C. Groen, P. C. Evans, et al. "Non-Invasive Molecular Imaging of Shear Stress-Induced Endothelial Activation and Atherosclerotic Plaque Vulnerability." In ASME 2012 Summer Bioengineering Conference. American Society of Mechanical Engineers, 2012. http://dx.doi.org/10.1115/sbc2012-80515.

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Atherosclerosis is a lipid- and inflammation driven disease of the larger arteries and is found at specific locations in the arterial tree, i.e. at branches and bends where endothelial cells are exposed to low and low, oscillatory shear stress. Shear stress, the frictional force acting on the endothelial cells as a result of the blood flow, affects endothelial physiology. It determines the location of atherosclerotic lesion development as low and low, oscillatory shear stress induce pro-inflammatory transcription factors but reduce expression and/or activity of anti-inflammatory transcription
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Liu, Biyue, and Dalin Tang. "Computer Simulations of the Blood Flows and the Growth of Stenosis in Arteries With Bends and Bifurcations." In ASME 2009 Summer Bioengineering Conference. American Society of Mechanical Engineers, 2009. http://dx.doi.org/10.1115/sbc2009-203654.

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Heart attack and stroke are the chief and the 3rd largest causes of death in the United States, respectively. A heart attack occurs when the blood supply to an area of heart muscle is blocked, usually by a clot in a coronary artery; a stroke occurs when the blood supply to a region of the brain is lost. The most frequent cause of loss of blood supply to brain tissue or to heart muscle is atherosclerosis, which involves complex interactions between the artery wall and the blood flow. Caro et al first suggested that the distribution of fatty streaking in human aorta may be coincident with region
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Hoffman, Allen H., Zhongzhao Teng, Calvin Mui, Jie Zheng, Pamela K. Woodard, and Dalin Tang. "Stiffness Comparisons Between Adventitia, Media and Full Thickness Specimens From Human Atherosclerotic Carotid Arteries." In ASME 2009 Summer Bioengineering Conference. American Society of Mechanical Engineers, 2009. http://dx.doi.org/10.1115/sbc2009-206401.

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Arteries display highly nonlinear, anisotropic material behavior and can be considered to be a layered composite of fiber oriented materials composed of three layers: intima, media and adventitia. The intima does not affect the material properties of the artery. Thus, the mechanical properties of an artery result from the combined interaction of the media and adventitia with each layer displaying different material properties. It has been widely accepted that atherosclerosis changes the material properties of the arterial wall. However, little experimental data exists relating the properties o
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van der Giessen, Alina G., Jolanda J. Wentzel, Frans N. van de Vosse, Antonius F. van der Steen, Pim J. de Feyter, and Frank J. Gijsen. "Plaque and Shear Stress Distribution in Human Coronary Bifurcations: a Multi-Slice Computed Tomography Study." In ASME 2007 Summer Bioengineering Conference. American Society of Mechanical Engineers, 2007. http://dx.doi.org/10.1115/sbc2007-176309.

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It is generally accepted that early atherosclerosis develops in low shear-stress (SS) regions such as the outer wall of arterial bifurcations and the inner bend of curved vessels (1). However, in clinical practice, it is common to observe atherosclerotic plaques at the flow-divider, or carina, of coronary bifurcations (2). Plaques at the carina are more frequently found in symptomatic patients, and may represent a more advanced stage of atherosclerosis. The carina is located in a region which is exposed to high SS. We hypothesize that if plaques are located in atheroprotective high SS regions,
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Informes sobre el tema "Atherosclerosis"

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Ugusman, Azizah, Nur Syahidah Nor Hisam, Nur Syakirah Othman, et al. PHARMACOLOGICAL INTERVENTIONS FOR INTRAPLAQUE NEOVASCULARIZATION IN ATHEROSCLEROSIS. INPLASY - International Platform of Registered Systematic Review and Meta-analysis Protocols, 2024. http://dx.doi.org/10.37766/inplasy2024.3.0005.

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Atherosclerosis Biomarkers by Computed Tomography Angiography (CTA). Chair Andrew Buckler, Luca Saba, and Uwe Joseph Schoepf. Radiological Society of North America (RSNA) / Quantitative Imaging Biomarkers Alliance (QIBA), 2023. http://dx.doi.org/10.1148/qiba/20230328.

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The clinical application of Computed Tomography Angiography (CTA) is widely available as a technique to optimize the therapeutic approach to treating vascular disease. Evaluation of atherosclerotic arterial plaque characteristics is currently based on qualitative biomarkers. However, the reproducibility of such findings has historically been limited even among experts (1). Quantitative imaging biomarkers have been shown to have additive value above traditional qualitative imaging metrics and clinical risk scores regarding patient outcomes (2). However, many definitions and cut-offs are present
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Saba, Luca, and Uwe Joseph Schoepf. Atherosclerosis Biomarkers by Computed Tomography Angiography (CTA) - Maintenance version June 2024. Chair Andrew Buckler. Radiological Society of North America (RSNA) / Quantitative Imaging Biomarkers Alliance (QIBA), 2024. http://dx.doi.org/10.1148/qiba/202406.

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The clinical application of Computed Tomography Angiography (CTA) is widely available as a technique to optimize the therapeutic approach to treating vascular disease. Evaluation of atherosclerotic arterial plaque characteristics is currently based on qualitative biomarkers. However, the reproducibility of such findings has historically been limited even among experts. Quantitative imaging biomarkers have been shown to have additive value above traditional qualitative imaging metrics and clinical risk scores regarding patient outcomes. However, many definitions and cut-offs are present in the
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Wang, Jun, Congcong Wang, Hongjuan Fu, Zezhong Liu, Yimin Zhang, and Tong Zhang. TNF alpha antagonists improve oxidative stress and atherosclerosis induced by rheumatoid arthritis. INPLASY - International Platform of Registered Systematic Review and Meta-analysis Protocols, 2023. http://dx.doi.org/10.37766/inplasy2023.1.0033.

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song, jiayuan. Impact of Alcohol Consumption on atherosclerosis: A Systematic Review and Meta-analysis. INPLASY - International Platform of Registered Systematic Review and Meta-analysis Protocols, 2025. https://doi.org/10.37766/inplasy2025.1.0031.

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wang, chen, peizheng zhang, and yuting yang. Effect of traditional Chinese exercise on abnormal lipid metabolism in patients with atherosclerosis. INPLASY - International Platform of Registered Systematic Review and Meta-analysis Protocols, 2021. http://dx.doi.org/10.37766/inplasy2021.9.0093.

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Men, Jie, Wenjuan Wang, Jian Zhao, Jie Wen, Qingqing Hao, and Shufeng Li. Effectiveness of exercise in atherosclerosis in ischemic strokes: a systematic review and meta-analysis. INPLASY - International Platform of Registered Systematic Review and Meta-analysis Protocols, 2022. http://dx.doi.org/10.37766/inplasy2022.9.0078.

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Zulkefli, Siti Bazilah, Fathimah Mohamad, Nurul Alimah Abdul Nasir, Ahmad Nazrun Shuid, and Nasibah Azme. Therapeutic Strategies for Post-Menopausal Atherosclerosis: A Systematic Review of Drug Interventions in Animal Models. INPLASY - International Platform of Registered Systematic Review and Meta-analysis Protocols, 2025. https://doi.org/10.37766/inplasy2025.6.0033.

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Aminuddin, Amilia, Ubashini Vijakumaran, Nazirah Samah, et al. Unveiling TIMPs: A Systematic Review of Their Role as Biomarkers in Atherosclerosis and Coronary Artery Disease. INPLASY - International Platform of Registered Systematic Review and Meta-analysis Protocols, 2024. http://dx.doi.org/10.37766/inplasy2024.7.0070.

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Ni, Zhichao, Qinwen Xiao, Zhongyuan Huang, Zihao Xia, and Dezhong Peng. Efficacy and safety of Acupuncture for carotid atherosclerosis: A protocol for systematic review and network meta-analysis. INPLASY - International Platform of Registered Systematic Review and Meta-analysis Protocols, 2023. http://dx.doi.org/10.37766/inplasy2023.2.0005.

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