Bibliografías temáticas / Cannabinoid CB1 receptor

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Literatura académica sobre el tema "Cannabinoid CB1 receptor"

Autor: Grafiati
Publicado: 11 de marzo de 2023
Última modificación: 31 de julio de 2025

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Artículos de revistas sobre el tema "Cannabinoid CB1 receptor"

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Soraya, hiva, Ruohollah Seddigh, Fatemeh Hadi, and Mohammad Faramarzi. "Chemical cannabis; The New Trend of addiction in Iran." Iranian Journal of Psychiatry and Clinical Psychology 28, no. 1 (2022): 10. http://dx.doi.org/10.32598/ijpcp.28.1.4010.1.

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Synthetic cannabinoids (SC) are a heterogeneous group of substances with a high affinity for cannabinoid receptors. Unlike Δ9-tetrahydrocannabinol (THC), synthetic cannabinoids are incredibly potent, highly productive, have more affinity for the Cannabinoid receptor type 1 (CB1), and Cannabinoid receptor type 2 (CB2), and are designed to accelerate the effects of tetrahydrocannabinol. Also, there is experimental evidence that SCs acts on non-cannabinoid receptors, such as the 5-HT2B receptor or dopaminergic receptors. (1, 2).
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Walsh, Kenneth B., and Andrea E. Holmes. "Pharmacology of Minor Cannabinoids at the Cannabinoid CB1 Receptor: Isomer- and Ligand-Dependent Antagonism by Tetrahydrocannabivarin." Receptors 1, no. 1 (2022): 3–12. http://dx.doi.org/10.3390/receptors1010002.

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(1) Background: In addition to the major phytocannabinoids, trans-Δ9-tetrahydrocannabinol (Δ9-THC) and cannabidiol (CBD), the cannabis plant (Cannabis sativa L.) synthesizes over 120 additional cannabinoids that are known as minor cannabinoids. These minor cannabinoids have been proposed to act as agonists and antagonists at numerous targets including cannabinoid type 1 (CB1) and type 2 (CB2) receptors, transient receptor potential (TRP) channels and others. The goal of the present study was to determine the agonist effects of the minor cannabinoids: cannabinol (CBN), cannabigerol (CBG), canna
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Li, Yong, and Jimok Kim. "CB2 Cannabinoid Receptor Knockout in Mice Impairs Contextual Long-Term Memory and Enhances Spatial Working Memory." Neural Plasticity 2016 (2016): 1–14. http://dx.doi.org/10.1155/2016/9817089.

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Neurocognitive effects of cannabinoids have been extensively studied with a focus on CB1 cannabinoid receptors because CB1 receptors have been considered the major cannabinoid receptor in the nervous system. However, recent discoveries of CB2 cannabinoid receptors in the brain demand accurate determination of whether and how CB2 receptors are involved in the cognitive effects of cannabinoids. CB2 cannabinoid receptors are primarily involved in immune functions, but also implicated in psychiatric disorders such as schizophrenia and depression. Here, we examined the effects of CB2 receptor knock
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SMALL-HOWARD, Andrea L., Lori M. N. SHIMODA, Chaker N. ADRA, and Helen TURNER. "Anti-inflammatory potential of CB1-mediated cAMP elevation in mast cells." Biochemical Journal 388, no. 2 (2005): 465–73. http://dx.doi.org/10.1042/bj20041682.

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Cannabinoids are broadly immunosuppressive, and anti-inflammatory properties have been reported for certain marijuana constituents and endogenously produced cannabinoids. The CB2 cannabinoid receptor is an established constituent of immune system cells, and we have recently established that the CB1 cannabinoid receptor is expressed in mast cells. In the present study, we sought to define a role for CB1 in mast cells and to identify the signalling pathways that may mediate the suppressive effects of CB1 ligation on mast cell activation. Our results show that CB1 and CB2 mediate diametrically op
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Dobovišek, Luka, Fran Krstanović, Simona Borštnar, and Nataša Debeljak. "Cannabinoids and Hormone Receptor-Positive Breast Cancer Treatment." Cancers 12, no. 3 (2020): 525. http://dx.doi.org/10.3390/cancers12030525.

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Breast cancer (BC) is the most common cancer in women worldwide. Approximately 70–80% of BCs express estrogen receptors (ER), which predict the response to endocrine therapy (ET), and are therefore hormone receptor-positive (HR+). Endogenous cannabinoids together with cannabinoid receptor 1 and 2 (CB1, CB2) constitute the basis of the endocannabinoid system. Interactions of cannabinoids with hypothalamic–pituitary–gonadal axis hormones are well documented, and two studies found a positive correlation between peak plasma endogenous cannabinoid anandamide with peak plasma 17β-estradiol, luteiniz
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GÓMEZ DEL PULGAR, Teresa, Guillermo VELASCO, and Manuel GUZMÁN. "The CB1 cannabinoid receptor is coupled to the activation of protein kinase B/Akt." Biochemical Journal 347, no. 2 (2000): 369–73. http://dx.doi.org/10.1042/bj3470369.

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Cannabinoids exert most of their effects in the central nervous system through the CB1 cannabinoid receptor. This G-protein-coupled receptor has been shown to be functionally coupled to inhibition of adenylate cyclase, modulation of ion channels and activation of extracellular-signal-regulated kinase. Using Chinese hamster ovary cells stably transfected with the CB1 receptor cDNA we show here that ∆9-tetrahydrocannabinol (THC), the major active component of marijuana, induces the activation of protein kinase B/Akt (PKB). This effect of THC was also exerted by the endogenous cannabinoid anandam
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Bow, Eric W., and John M. Rimoldi. "The Structure–Function Relationships of Classical Cannabinoids: CB1/CB2 Modulation." Perspectives in Medicinal Chemistry 8 (January 2016): PMC.S32171. http://dx.doi.org/10.4137/pmc.s32171.

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The cannabinoids are members of a deceptively simple class of terpenophenolic secondary metabolites isolated from Cannabis sativa highlighted by (-)-Δ 9 -tetrahydrocannabinol (THC), eliciting distinct pharmacological effects mediated largely by cannabinoid receptor (CB1 or CB2) signaling. Since the initial discovery of THC and related cannabinoids, synthetic and semisynthetic classical cannabinoid analogs have been evaluated to help define receptor binding modes and structure–CB1/CB2 functional activity relationships. This perspective will examine the classical cannabinoids, with particular em
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Zibolka, Juliane, Anja Wolf, Lisa Rieger, et al. "Influence of Cannabinoid Receptor Deficiency on Parameters Involved in Blood Glucose Regulation in Mice." International Journal of Molecular Sciences 21, no. 9 (2020): 3168. http://dx.doi.org/10.3390/ijms21093168.

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Cannabinoids are known to influence hormone secretion of pancreatic islets via G protein-coupled cannabinoid receptor type 1 and 2 (CB1 and CB2). The present study was designed to further investigate the impact of cannabinoid receptors on the parameters involved in insulin secretion and blood glucose recognition. To this end, CB1 and CB2 receptor knockout mice (10–12 week old, both sexes) were characterised at basal state and compared to wild-type mice. The elimination of cannabinoid receptor signalling resulted in alterations of blood glucose concentrations, body weights and insulin levels. C
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Černe, Katarina. "Toxicological properties of Δ9-tetrahydrocannabinol and cannabidiol". Archives of Industrial Hygiene and Toxicology 71, № 1 (2020): 1–11. http://dx.doi.org/10.2478/aiht-2020-71-3301.

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AbstractCannabis sativa L. contains more than 100 phytocannabinoids that can interact with cannabinoid receptors CB1 and CB2. None of the cannabinoid receptor ligands is entirely CB1- or CB2-specific. The effects of cannabinoids therefore differ not just because of different potency at cannabinoid receptors but also because they can interact with other non-CB1 and non-CB2 targets, such as TRPV1, GPR55, and GPR119. The most studied phytocannabinoid is Δ9-tetrahydrocannabinol (THC). THC is a partial agonist at both cannabinoid receptors, but its psychotomimetic effect is produced primarily via a
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Barth, Francis, and Murielle Rinaldi-Carmona. "The Development of Cannabinoid Antagonists." Current Medicinal Chemistry 6, no. 8 (1999): 745–55. http://dx.doi.org/10.2174/0929867306666220401143808.

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The discovery of two distinct cannabinoid receptors (CB1 and C B 2 ) in the early 1990's has revived the research on cannabinoid antagonists. While the search for antagonists based on the structure of agonists (classical cannabinoids or aminoalkylindoles) appeared rather disappointing, the first potent cannabinoid antagonists were developed in a new chemical series: the diarylpyrazoles. Since its discovery in 1994, the selective CB1 antagonist SR 141716 has become a major pharmacological tool to elucidate the physiological role of the CB 1 cannabinoid receptor and its endogenous ligand. The se
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Tesis sobre el tema "Cannabinoid CB1 receptor"

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Smith, Tricia. "Effects of Cannabinoid Receptor Interacting Protein (CRIP1a) on Cannabinoid Receptor (CB1) Function." VCU Scholars Compass, 2009. http://scholarscompass.vcu.edu/etd/1977.

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EFFECTS OF CANNABINOID RECEPTOR INTERACTING PROTEIN (CRIP1a) ON CANNABINOID (CB1) RECEPTOR FUNCTION. By Tricia Hardt Smith, B.S., M.S. A dissertation submitted in partial fulfillment of the requirements for the degree of Doctor of Philosophy at Virginia Commonwealth University Virginia Commonwealth University, 2009. Major Director: Dana E. Selley, Ph.D., Department of Pharmacology and Toxicology This dissertation examines modulation of cannabinoid CB1 receptor function by Cannabinoid Receptor Interacting Protein (CRIP1a), a novel protein that binds the C-terminus of CB1 receptors. In Human
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Daigle, Tanya L. "Molecular mechanisms of CB1 cannabinoid receptor signaling and internalization /." Thesis, Connect to this title online; UW restricted, 2007. http://hdl.handle.net/1773/10527.

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Slaughter, Kimari. "Synthesis and Development of Potential CB1 Receptor Neutral Antagonists." ScholarWorks@UNO, 2012. http://scholarworks.uno.edu/td/1483.

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Cannabis and its derivatives have been used for both medicinal and recreational purposes. The study of this plant led to the discovery of over 60 cannabinoids, found exclusively in cannabis, that contribute to the behavioral effects of cannabis use, the most common is delta-9-tetrahydrocannabinol. Cannabinoid receptors function to increase activity in the mesolimbic dopamine reward system. Dopamine is a neurotransmitter that plays a major role in addition and its regulation plays a crucial role in mental and physical well-being. There is evidence that CB1 receptors are important to the reinfor
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Horswill, James G. "Pharmacological characterisation of a novel cannabinoid CB1 receptor allosteric modulator." Thesis, University of Reading, 2011. http://ethos.bl.uk/OrderDetails.do?uin=uk.bl.ethos.541953.

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Grim, Travis. "Synthetic cannabinoids versus delta-9-tetrahydrocannabinol: abuse-related consequences of enhanced efficacy at the cannabinoid 1 receptor." VCU Scholars Compass, 2015. http://scholarscompass.vcu.edu/etd/4039.

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In the past ten years, synthetic cannabinoids (SC) have emerged as drugs of abuse. Unlike D9-tetrahydrocannabinol (THC), many SCs are associated with serious health complications and death. One way in which THC and SCs differ lies with their enhanced potency and efficacy at the CB1 receptor. No current methods exist to measure efficacy at the CB1 receptor in vivo, and the abuse-related properties of SC cannabinoids are not well explored. Here, we utilized CB1 wild type (WT), heterozygous (HET), and knockout (KO) mice. By employing CB1 ligands which differ in efficacy we have developed a method
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Wing, Victoria Caroline. "The role of the cannabinoid CB1 receptor subtype in nicotine dependence." Thesis, University of Newcastle Upon Tyne, 2009. http://ethos.bl.uk/OrderDetails.do?uin=uk.bl.ethos.500924.

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Tobacco smoking, considered an addiction to nicotine, is a worldwide health problem but limited effective pharmacotherapies are available. Nicotine acts on nicotinic acetylcholine receptors in the brain thus interacting with a range of neurotransmitter systems of which the mesocorticolimbic dopamine system is considered crucial for drug dependence. The endocannabinoid system has also been implicated in nicotine dependence and the cannabinoid CB1 receptor antagonist rimonabant has shown efficacy as a smoking cessation aid. This thesis aimed to further examine the role of CBl receptors in the mo
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Marcu, Jahan Phillip. "Novel Insights into CB1 Receptor Signaling and the Anabolic Role of Cannabinoid Receptors in Bone." Diss., Temple University Libraries, 2013. http://cdm16002.contentdm.oclc.org/cdm/ref/collection/p245801coll10/id/233543.

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Cell Biology<br>Ph.D.<br>Activation of the CB1 receptor is modulated by aspartate residue D2.63176 in transmembrane helix (TMH) II. Interestingly, D2.63 does not affect the affinity for ligand binding at the CB1 receptor. Studies in class A GPCRs have suggested an ionic interaction between residues of TMHII and VII. In this report, modeling studies identified residue K373, in the extracellular (EC)-3 loop, in charged interactions with D2.63. We investigated this possibility by performing reciprocal mutations and biochemical studies. D2.63176A, K373A, D2.63176A-K373A, and the reciprocal mutant
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Feliszek, Monika [Verfasser]. "Age-dependent cannabinoid CB1 receptor plasticity and search for histamine H4 receptors in the brain / Monika Feliszek." Bonn : Universitäts- und Landesbibliothek Bonn, 2016. http://d-nb.info/1119888875/34.

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Jacob, Wolfgang. "Role of the Cannabinoid Receptor Type 1 (CB1) in Synaptic Plasticity, Memory and Emotionality." Diss., lmu, 2007. http://nbn-resolving.de/urn:nbn:de:bvb:19-72307.

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Marsicano, Giovanni. "Physiological role of the cannabinoid receptor 1 (CB1) in the murine central nervous system." Thesis, Open University, 2001. http://oro.open.ac.uk/58198/.

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The cannabinoid system is involved in many functions of mammalian brain, such as learning and memory, pain perception and 'locomotion. The "brain type" cannabinoid receptor CB 1 is one of the key elements of the cannabinoid system. In this Thesis, some aspects of the neurobiology of mouse CB 1 are described. CB 1 mRNA distribution was analysed by single and double in situ hybridization (ISH), revealing the expression of the receptor in specific neuronal subpopulations. This expression pattern suggests many putative functional cross-talks between the cannabinoid system and other signalling mole
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Libros sobre el tema "Cannabinoid CB1 receptor"

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Desroches, Julie. Peripheral analgesia involves cannabinoid receptors. Edited by Paul Farquhar-Smith, Pierre Beaulieu, and Sian Jagger. Oxford University Press, 2018. http://dx.doi.org/10.1093/med/9780198834359.003.0034.

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This landmark paper by Agarwal and colleagues was published in 2007, when the exact contribution of the activation of the cannabinoid type 1 receptor (CB1) receptors expressed on the peripheral terminals of nociceptors in pain modulation was still uncertain. At that time, while it was clearly demonstrated that the central nervous system (CNS) was involved in the antinociceptive effects induced by the activation of the CB1 receptor, many strains of mice in which the gene encoding the CB1 receptor was deleted by conditional mutagenesis were used to study the specific role of these receptors in p
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Abood, Mary E., and Thomas Gamage. The cloning and characterization of the cannabinoid type 1 receptor. Edited by Paul Farquhar-Smith, Pierre Beaulieu, and Sian Jagger. Oxford University Press, 2018. http://dx.doi.org/10.1093/med/9780198834359.003.0025.

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The cloning and characterization of the first cannabinoid receptor (now known as the cannabinoid type 1 (CB1) receptor) by Matsuda et al. in the landmark paper discussed in this chapter was a seminal discovery in 1990. While the analgesic properties of marijuana had been known for thousands of years, the mechanisms through which marijuana produces analgesia were not understood. The identification and functional characterization of the CB1 receptor led to the discovery of an endogenous cannabinoid system (the endocannabinoid system), which has now been shown to be important not only for acute a
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Abood, Mary E., Nephi Stella, and Roger G. Sorensen. EndoCANNABINOIDS: Actions at Non-CB1/CB2 Cannabinoid Receptors. Springer New York, 2014.

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Abood, Mary E., Nephi Stella, and Roger G. Sorensen. EndoCANNABINOIDS: Actions at Non-CB1/CB2 Cannabinoid Receptors. Springer London, Limited, 2012.

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Abood, Mary E., Roger G. Sorensen, and Nephi Stella. endoCANNABINOIDS: Actions at Non-CB1/CB2 Cannabinoid Receptors. Springer, 2012.

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Iversen, Leslie. The Pharmacology of Delta-9-Tetrahydrocannabinol (THC). Oxford University Press, 2018. http://dx.doi.org/10.1093/oso/9780190846848.003.0002.

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The 19th century was a great era for plant chemistry. Many complex drug molecules, known as alkaloids, were isolated and identified from plants. This chapter discusses the history of the discovery of delta-9-tetrahyrocannabinol (THC) as the psychoactive substance in cannabis products and also the discovery of the cannabinoid receptors CB-1 and CB-2 in the body and brain. The mechanism of action of cannabinoids on such receptors to inhibit neurotransmitter release or other actions is also discussed. In addition, various methods for the ingestion of cannabis, such as smoking and vaping, are revi
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Rodriguez, Emma. Cannabinoid CB1: A Study of Morphological Levels and Distribution of MRNA and CB1 Receptors. Independently Published, 2019.

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Iversen, Leslie. Peripheral and Central Effects of THC. Oxford University Press, 2018. http://dx.doi.org/10.1093/oso/9780190846848.003.0003.

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Although there is currently only limited knowledge of how activation of the CB-1 receptor in brain leads to the many actions of THC, some general features of cannabinoid control mechanisms are emerging. This chapter discusses THC in relation to the inhibition of neurotransmitter release, cardiovascular effects, effects on motility and pain, and behavioral models including the “Billy Martin tetrad.” It also discusses human laboratory models, including studies on learning and memory. A key effect of cannabis is intoxication, and the subjective effects of cannabis are reported in detail. Finally,
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Capítulos de libros sobre el tema "Cannabinoid CB1 receptor"

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Rech, Glenn R., and Samer N. Narouze. "Cannabinoid Receptor 1 (CB1)." In Cannabinoids and Pain. Springer International Publishing, 2021. http://dx.doi.org/10.1007/978-3-030-69186-8_7.

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Zhang, Yanan, Herbert H. Seltzman, Marcus Brackeen, and Brian F. Thomas. "Structure–Activity Relationships and Conformational Freedom of CB1 Receptor Antagonists and Inverse Agonists." In The Cannabinoid Receptors. Humana Press, 2009. http://dx.doi.org/10.1007/978-1-59745-503-9_4.

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Katona, István. "Endocannabinoid Receptors: CNS Localization of the CB1 Cannabinoid Receptor." In Behavioral Neurobiology of the Endocannabinoid System. Springer Berlin Heidelberg, 2009. http://dx.doi.org/10.1007/978-3-540-88955-7_3.

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Salamone, John D., Kelly Sink, Kristen N. Segovia, et al. "Cannabinoid Cb1 Receptor Antagonists/Inverse Agonists and Food-Seeking Behavior." In Handbook of Behavior, Food and Nutrition. Springer New York, 2011. http://dx.doi.org/10.1007/978-0-387-92271-3_29.

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Iliopoulos-Tsoutsouvas, Christos, Markos-Orestis Georgiadis, Lipin Ji, Spyros P. Nikas, and Alexandros Makriyannis. "Chapter 3. Natural Compounds and Synthetic Drugs to Target Type-1 Cannabinoid (CB1) Receptor." In Drug Discovery. Royal Society of Chemistry, 2020. http://dx.doi.org/10.1039/9781839160752-00048.

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Pertwee, Roger G. "CB1 and CB2 Receptor Pharmacology." In Cannabinoids and the Brain. Springer US, 2008. http://dx.doi.org/10.1007/978-0-387-74349-3_7.

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Pertwee, Roger G., and Adèle Thomas. "Therapeutic Applications for Agents that Act at CB1 and CB2 Receptors." In The Cannabinoid Receptors. Humana Press, 2009. http://dx.doi.org/10.1007/978-1-59745-503-9_13.

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Antinori, Silvia, and Liana Fattore. "How CB1 Receptor Activity and Distribution Contribute to Make the Male and Female Brain Different Toward Cannabinoid-Induced Effects." In Endocannabinoids and Lipid Mediators in Brain Functions. Springer International Publishing, 2017. http://dx.doi.org/10.1007/978-3-319-57371-7_2.

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Wiley, Jenny L., and Billy R. Martin. "Preclinical Pharmacological and Brain Bioassay Systems for CB1 Cannabinoid Receptors." In The Cannabinoid Receptors. Humana Press, 2009. http://dx.doi.org/10.1007/978-1-59745-503-9_12.

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Thomas, Brian F., Yanan Zhang, Marcus Brackeen, Kevin M. Page, S. Wayne Mascarella, and Herbert H. Seltzman. "Conformational Characteristics of the Interaction of SR141716A with the CB1 Cannabinoid Receptor as Determined Through the Use of Conformationally Constrained Analogs." In Drug Addiction. Springer New York, 2008. http://dx.doi.org/10.1007/978-0-387-76678-2_41.

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Actas de conferencias sobre el tema "Cannabinoid CB1 receptor"

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Goncalves Romeu, Paula, Enrique De Madaria, Karina Cárdenas-Jaen, et al. "Cannabinoid CB1 receptor and its endogenous lipid mediator 2-AG alleviates severity in a pancreatitis-associated lung injury mouse model." In ERS Congress 2024 abstracts. European Respiratory Society, 2024. http://dx.doi.org/10.1183/13993003.congress-2024.pa1719.

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Espinosa-Bustos, C., C. F. Lagos, J. Romero-Parra, et al. "Synthesis and Docking of new (2-(2,5-dimethoxyphenyl)-1Hbenzo[ d]imidazol-1-yl)(aryl)methanone derivatives designed as novel cannabinoid CB1 receptor antagonists." In 14th Brazilian Meeting on Organic Synthesis. Editora Edgard Blücher, 2013. http://dx.doi.org/10.5151/chempro-14bmos-r0169-1.

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Gabrielli, Ângelo, Camila Sousa Bragunce Alves, Bruna Oliveira Bicalho, and Débora Pimenta Alves. "Benefits and Challenges of Cannabis Use in the Treatment of Refractory Epilepsy." In XIII Congresso Paulista de Neurologia. Zeppelini Editorial e Comunicação, 2021. http://dx.doi.org/10.5327/1516-3180.239.

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Introduction: Refractory epilepsy (RE) is a disease that causes continuous and debilitating seizures. Due to the ineffectiveness of antiepileptic therapies, there is a growing interest in drugs made with cannabidiol (CBD), a substance extracted from Cannabis. Objective: To point out benefits and challenges of the use of CBD in the treatment of RE. Methods: Literature review performed at PubMed, with the descriptors Epilepsy, Drug Therapy and Cannabis. Results: It is suggested that CBD is mediated by cannabinoid receptors coupled to protein G, by blockade of NMDA receptors, by GABAergic modulat
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Elói, Daniel Vinicius, Daniel Lopes Marques de Araújo, Gabriela Fonseca Marçal, Luana Soares Vargas, Matheus Garcia Ribeiro, and Nicollas Nunes Rabelo. "Canabinoids as a therapeutic alternative in refractory epilepsy." In XIII Congresso Paulista de Neurologia. Zeppelini Editorial e Comunicação, 2021. http://dx.doi.org/10.5327/1516-3180.554.

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Introduction: Epilepsy is characterized by abnormal electrical discharges in the brain that generate neuronal hyperexcitability and hypersynchrony. In the last years, pharmacological strategies have been efficient in the control of epileptic seizures of approximately 80% of patients, however, there are still refractory cases. Objective: To elucidate new forms of epilepsy treatment with cannabinoids. Design: Systematic Review performed at Centro Universitário Atenas – Paracatu – Minas Gerais. Methods: Literature review performed in the SciELO and PubMed databases, with the following terms: epil
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Sabater García, Luz María, Verónica Alarcón Ortiz, Alejandra Carrillo Llamas, Pablo García Verdú, and María Ángeles de Haro Rivas. "Síndrome hiperemético cannabinoide: A propósito de un caso clínico." In 22° Congreso de la Sociedad Española de Patología Dual (SEPD) 2020. SEPD, 2020. http://dx.doi.org/10.17579/sepd2020p085.

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- Objetivo: Actualizar la evidencia científica disponible en relación con el Síndrome hiperemético producido por el consumo crónico de cannabis a propósito de un caso clínico. - Material y métodos: Se ha llevado a cabo una revisión sistemática entre las principales fuentes de literatura científica y se han empleado los programas Selene y Ágora Plus para recopilar los diferentes datos médicos del caso (Palabras clave: “hyperemesis”, “Cannabis”, “THC”, “cannabinoid”). - Resultados: * Exposición de caso clínico (tabla en póster). * Etiología: Se piensa que el uso prolongado de cannabis conduciría
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"PV-006 - REVISIÓN SISTEMÁTICA PSICOSIS DUAL." In 24 CONGRESO DE LA SOCIEDAD ESPAÑOLA DE PATOLOGÍA DUAL. SEPD, 2022. http://dx.doi.org/10.17579/abstractbooksepd2022.pv006.

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Resumen: El sistema dopaminérgico participa en la recompensa cerebral en el trastorno por uso de sustancias (TUS). En la transición a la dependencia la prominencia del incentivo y los cambios neuroadaptivos en distintos sistemas de neurotransmisión son importantes. La excitabilidad cerebral y la hiperreactividad al estrés promueven un estado emocional negativo e hipodopaminergia prefrontal. La psicosis dual está presente en el 30-50% de trastornos psicóticos. La teoría de la vulnerabilidad compartida se propone como hipótesis explicativa. Objetivos: revisión sistemática de la neurobiología de
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"PV-125 - HIPEREMESIS CANNABINOIDE. DUCHAS CALIENTES COMPULSIVAS EN PACIENTE CONSUMIDOR CRÓNICO DE CANNABIS. A PROPÓSITO DE UN CASO." In 24 CONGRESO DE LA SOCIEDAD ESPAÑOLA DE PATOLOGÍA DUAL. SEPD, 2022. http://dx.doi.org/10.17579/abstractbooksepd2022.pv125.

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Objetivos: Se establecen como objetivos presentar la evolución de un paciente varón de 28 años con diagnóstico de Esquizofrenia y trastorno por consumo de cannabis que ingresa en la Unidad de Hospitalización Breve de Psiquiatría (UHB). Asimismo, se establece el objetivo de revisar la bibliografía disponible relacionada con la mejoría sintomática de la ansiedad a través de un número elevado de duchas calientes en pacientes consumidores de cannabis. Material y métodos: Se desarrolla el caso clínico de un varón de 28 años que ingresa en la UHB de Psiquiatría por episodio psicótico en el contexto
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Abdalla, J., J. K. Park, N. J. Coffey, et al. "Deletion of Cannabinoid 1 Receptor (CB1R) in Myeloid Cells Prevents Lung Inflammation and Neutrophil Infiltration in Bleomycin-Induced Pulmonary Fibrosis in Mice." In American Thoracic Society 2020 International Conference, May 15-20, 2020 - Philadelphia, PA. American Thoracic Society, 2020. http://dx.doi.org/10.1164/ajrccm-conference.2020.201.1_meetingabstracts.a2387.

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Zawatsky, C., J. K. Park, N. J. Coffey, et al. "Activation of Cannabinoid 1 Receptor (CB1R) in Myeloid Cells Induces Lymphocyte Infiltration in Lung Via Regulating CXCL13 in Bleomycin-Induced Pulmonary Fibrosis." In American Thoracic Society 2020 International Conference, May 15-20, 2020 - Philadelphia, PA. American Thoracic Society, 2020. http://dx.doi.org/10.1164/ajrccm-conference.2020.201.1_meetingabstracts.a2391.

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Cinar, R., M. R. Iyer, J. K. Park, et al. "MRI-1867, Dual Target Cannabinoid Receptor 1 (CB1R) and Inducible Nitric Oxide Synthase (iNOS) Inhibitor, for Effective Anti-Fibrotic Therapy for Hermansky-Pudlak Syndrome Pulmonary Fibrosis in Pale Ear Mic." In American Thoracic Society 2020 International Conference, May 15-20, 2020 - Philadelphia, PA. American Thoracic Society, 2020. http://dx.doi.org/10.1164/ajrccm-conference.2020.201.1_meetingabstracts.a1198.

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Informes sobre el tema "Cannabinoid CB1 receptor"

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Yerofeyeva, Anna-Maria V., Sergey V. Pinchuk, Svetlana N. Rjabceva, and Alla Yu Molchanova. Neuropathic pain correction method using mesenchymal stem cells and cannabinoid receptor CB2 stimulation. Peeref, 2023. http://dx.doi.org/10.54985/peeref.2303p4142545.

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Lazarov, Nikolai E., Dimitrinka Y. Atanasova, Angel D. Dandov, and Nikolay D. Dimitrov. Anandamide-induced Expression of CB1 Cannabinoid Receptors in the Rat Mesencephalic Trigeminal Nucleus after Short-term Thermal Stress. "Prof. Marin Drinov" Publishing House of Bulgarian Academy of Sciences, 2018. http://dx.doi.org/10.7546/crabs.2018.09.16.

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