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Literatura académica sobre el tema "Complex syndrome"

Autor: Grafiati

Publicado: 4 de junio de 2021

Última modificación: 1 de febrero de 2022

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Artículos de revistas sobre el tema "Complex syndrome"

Zargar, Sumera. "Blepharophimosis Syndrome with Retinitis Pigmentosa (RP) A Rare Syndrome Complex". Journal of Medical Science And clinical Research 05, n.º 01 (13 de enero de 2017): 15559–61. http://dx.doi.org/10.18535/jmscr/v5i1.59.

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Colby, Candice C. y John M. Del Gaudio. "Stylohyoid Complex Syndrome". Archives of Otolaryngology–Head & Neck Surgery 137, n.º 3 (21 de marzo de 2011): 248. http://dx.doi.org/10.1001/archoto.2011.25.

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Shams, Soheila, Majid Kyavar, Parham Sadeghipour, Hamideh Khesali, Kambiz Mozaffari, Nejat Mahdieh y Mohammad Esmail Zanganehfar. "Carney Complex syndrome". Cardiovascular Pathology 49 (noviembre de 2020): 107231. http://dx.doi.org/10.1016/j.carpath.2020.107231.

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Yılmaz, İnsu, Leylagül Kaynar, Nuri Tutar, Çiğdem Pala, Özlem Canöz, Afra Yıldırım, Hakan Büyükoğlan y İnci Gülmez. "Response of Complex Undefined Hypereosinophilic Syndrome to Treatment with Imatinib". Turkish Thoracic Journal 17, n.º 3 (10 de septiembre de 2016): 118–21. http://dx.doi.org/10.5578/ttj.30508.

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Hernandez, W., A. Raja y C. Capuano. "Complex regional pain syndrome". Journal of the American Podiatric Medical Association 89, n.º 10 (1 de octubre de 1999): 534–39. http://dx.doi.org/10.7547/87507315-89-10-534.

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Complex regional pain syndrome is a chronic pain syndrome that is often instigated by postoperative or post-traumatic events. The disease process can progress through three stages, the first of which tends to respond best to treatment. A review of the literature is presented, followed by a report of a patient who developed symptoms of complex regional pain syndrome following a water-skiing accident.

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Palman, A. D. "Complex sleep apnea syndrome". Zhurnal nevrologii i psikhiatrii im. S.S. Korsakova 117, n.º 4. Vyp. 2 (2017): 60. http://dx.doi.org/10.17116/jnevro20171174260-66.

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Hayek, Salim M. y Nagy A. Mekhail. "Complex Regional Pain Syndrome". Physician and Sportsmedicine 32, n.º 5 (mayo de 2004): 18–25. http://dx.doi.org/10.3810/psm.2004.05.254.

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Sharma, Anuja. "Complex sleep apnea syndrome". Indian Journal of Sleep Medicine 5, n.º 3 (2010): 81–83. http://dx.doi.org/10.5005/ijsm-5-3-81.

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Rho, Richard H., Randall P. Brewer, Tim J. Lamer y Peter R. Wilson. "Complex Regional Pain Syndrome". Mayo Clinic Proceedings 77, n.º 2 (febrero de 2002): 174–80. http://dx.doi.org/10.4065/77.2.174.

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Cooney, William P. "Complex Regional Pain Syndrome". Mayo Clinic Proceedings 77, n.º 7 (julio de 2002): 733–34. http://dx.doi.org/10.4065/77.7.733-a.

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Tesis sobre el tema "Complex syndrome"

Osborne, Scott. "Immunopathology of complex regional pain syndrome". Thesis, University of Liverpool, 2013. http://livrepository.liverpool.ac.uk/14485/.

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Both increased mast cells numbers and raised immune mediator concentrations indicate immune activation in the affected skin of patients with early CRPS, but little is known about regional immune cell involvement in late stage CRPS. The aim of the current study was to determine skin immune cell populations in longstanding CRPS. Using 6mm skin punch biopsies from CRPS-affected and non-affected tissues, and a combination of chemical and immunofluorescence staining, we examined the density and function of key cell populations including mast cells, epidermal Langerhans cells (LCs), and tissue resident T-cells. We found no significant differences in either overall immune cell infiltrates, or mast cell density between CRPS-affected and non-affected sub-epidermal tissue sections, contrasting recent findings in early CRPS by other groups. However, CD1a+ LC densities in the epidermal layer were significantly decreased in affected compared to non-affected CRPS limbs (p = <0.01). T-cell clones isolated from CRPS- affected sub-epidermal tissues displayed a trend towards increased IL-13 production in ELIspot assays when compared to T-cells isolated from non-affected areas, suggesting a Th2 bias. Immune cell abnormalities are maintained in late stage CRPS disease as manifest by changes in epidermal LC density and tissue resident T-cell phenotype.

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Kohr, Danielle [Verfasser]. "Autoimmunity in complex regional pain syndrome / Danielle Kohr". Gießen : Universitätsbibliothek, 2011. http://d-nb.info/1063177618/34.

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Lewis, Jenny. "Body perception disturbance in complex regional pain syndrome". Thesis, University of Southampton, 2008. https://eprints.soton.ac.uk/65409/.

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Complex regional pain syndrome (CRPS) is a painful, debilitating condition that is poorly understood. The syndrome is characterised by pain, motor disturbances and abnormalities in trophic, sudomotor, vascular temperature and sensation. The underlying mechanisms are unknown. Clinical observations have identified a novel phenomenon whereby patients pay little attention to, and fail to care for, their painful affected limb. The literature describes this phenomenon in terms of neglect-like symptoms similar to neurological neglect as described in stroke literature. However, this does not seem to fully fit with or explain the nature of clinical observations. Therefore the aim of the qualitative first study was to more fully describe the phenomenon through an investigation of the patient experience and words used to describe those experiences. Six themes emerged from the data and were as follows: hostile feelings; spectrum of disassociation; disparity between what is apparent and what is felt; distorted mental image of affected parts; awareness of limb position and conscious attention. From these findings a theory emerged which serves to further our understanding of body perception disturbance in CRPS. Based on these findings, the second study aimed to quantify a feature of body perception disturbance by measuring limb position accuracy of those with CRPS compared to Healthy Controls (HC) and those with Rheumatological Pain (RP). The CRPS group were significantly less accurate in positioning of both the affected and unaffected upper limbs (median=9°, Interquartile rang e (IQR), 5.7°-13.3°) compared to both HC (6.5°, IQR, 4°-10.7°) and RP groups (7.7°, IQR, 5 °-11.7°). In the CRPS group position accuracy of the affected limb significantly improved with vision (8.3° in view, 10.7° not in view). Pain intensity was significantly greater in the CRPS (6.5, IQR, 5.4-7.7) than the RP group (4.6, IQR, 3.6-5.7). Based on the findings of this research programme, a definition of body perception disturbance in CRPS is presented. Furthermore, a disrupted body schema model is proposed as an explanation of the central mechanisms responsible for body perception disturbance in CRPS.

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Smedley, Richard. "Online social support for complex regional pain syndrome". Thesis, University of Nottingham, 2016. http://eprints.nottingham.ac.uk/32694/.

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Individuals living with Complex Regional Pain Syndrome (CRPS) often experience difficulties taking part in social and recreational activities, which can leave them with a greatly reduced social network and limited opportunities for obtaining social support. Online support communities may provide individuals with an alternative way of obtaining social support, but few studies have examined these communities in the context of CRPS. Furthermore, most online support community research has focussed on established communities, and little is known about how new communities become established. This thesis examines a bespoke CRPS online support community with two broad aims: to examine the development of online support processes in relation to the launch of a new online support community, and to investigate the provision of social support for CRPS within an online support community. The dataset comprised 221 messages posted by 23 participants. Study 1 used the full dataset to examine engagement with the online support community, focussing on the number of individuals who used the forum (membership growth), how they used it (header analysis) and how they introduced themselves (introductory messages). Study 2 used the full dataset to investigate how support processes became established, the support content of messages, and how this contributed to the CRPS ‘four pillars of intervention’. Study 3 used four longitudinal case studies from the dataset to conduct a linguistic analysis of messages, focussing on support providing behaviour and the number of replies received. The results indicate that support processes start almost immediately when a new online support community is launched, and membership growth is closely linked to promotional strategies. Online support may play an important role in CRPS self-management by contributing to the ‘four pillars of intervention’, and there is a possibility that diffusion of responsibility may occur in forums. The longitudinal case study approach may produce important new insights and suggests that the use of health words is unrelated to the number of replies received, the use of self and other-oriented messages may be linked to health status and support providing activities, and that the ratio of positive-to-negative words could potentially be used to identify individuals who might benefit from additional support.

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Forouzanfar, Tymour. "Complex Regional Pain Syndrome Type I measurements and treatment /". [Maastricht : Maastricht : Universiteit Maastricht] ; University Library, Maastricht University [Host], 2004. http://arno.unimaas.nl/show.cgi?fid=7558.

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Niehof, Sjoerd Petrus. "Video thermography: complex regional pain syndrome in the picture". [S.l.] : Rotterdam : [The Author] ; Erasmus University [Host], 2007. http://hdl.handle.net/1765/10704.

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Maga, Tara Kristen. "Unraveling the complex genetics of atypical hemolytic uremic syndrome". Diss., University of Iowa, 2012. https://ir.uiowa.edu/etd/2935.

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Atypical hemolytic uremic syndrome (aHUS) is characterized by acute renal failure, thrombocytopenia, and microangiopathic hemolytic anemia. aHUS is far less common and more severe than typical HUS, which is caused by E. coli infection and manifests as diarrheal illness. The pathogenesis of the disease is linked to dysregulation of the alternative pathway of the complement cascade. Mutations in the complement regulators factor H (CFH), membrane cofactor protein (MCP), factor B (CFB), and factor I (CFI) have been implicated in aHUS. These loss or gain of function mutations lead to uncontrolled complement activity and immune-mediated host cell damage. Establishing a genetic etiology is important as it helps to direct treatment during the acute phase of disease and when transplantation is considered. It has been shown in previous studies that the age of onset as well the severity of the disease is correlated with the type of mutation a patient is found to carry. In forty percent of aHUS patients a mutation in CFH, MCP, CFB, CFI, C3 or THBD is not detected. These data strongly suggest that other genetic factors are involved in the pathogenesis of aHUS and that comprehensive mutation detection in aHUS patients is essential to provide diagnostic and prognostic information, and improve their clinical care. My thesis work has aimed to identify the other genetic contributors to this disease. To achieve this goal we began by screening the largest American cohort of aHUS patients for mutations in CFH, MCP, CFB, CFI, C3, THBD as well as CFHR5. This study identified over thirty novel mutations and suggests a more comprehensive genetic screening method that would better serve patients. To complement these studies multiplex ligation-dependent probe amplification was used to detect genetic rearrangements within the factor H related genes. A number of unique fusion proteins were seen in aHUS patients, all of which are predicted to affect the function of CFH. To discover mutations in novel genes that are causally related to aHUS, we have optimized a platform called CASCADE (Capture and Sequencing of Complement-Associated Disease Exons), which is based on targeted-genome capture and next-generation sequencing. This study revealed an unexpected role for ADAMTS13 and other genes in the coagulation pathway as modifiers of aHUS. Using functional assays we show two of the ADAMTS13 variants alter the behavior of this protein. This work has changed how we view this disease by identifying several novel candidate genes, for which we hope future analysis will lead to a better understanding of their role in aHUS. Using this knowledge we can provide better and more personalized treatments for patients.

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Graaff, Esther de. "The fragile X syndrome complex behavior of a simple repeat /". [S.l.] : Rotterdam : [The Author] ; Erasmus University [Host], 1996. http://hdl.handle.net/1765/13736.

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Mitrani, Leila Mical. "Certain syndrome or complex conundrum? : the pollination of Erica lanuginosa". Bachelor's thesis, University of Cape Town, 2010. http://hdl.handle.net/11427/26380.

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The flower of Erica lanuginosa has a tightly closed corolla, held in place by hinged sepals. with a dull reddish-pink colour which makes make it hard to determine a likely pollinator. Rodent trapping and pollen analysis of faecal matter showed it unlikely to be pollinated by a rodent. Flowers excluded from external pollination showed no seed set, hence it is not considered to be self-pollinated. Nectar analysis are inconclusive as an indicator of pollination syndrome. Entomophily by a robust insect with a medium length proboscis is considered unlikely due to phenology and morphology of the flower. Omothiphily is a possibility as stem thickness correlates with previous studies investigating the correlation between stem thickness and pollination syndromes. The pollination syndrome of Erica lanuginosa remains indeterminate by I hypothesize that, due to phenology, thick supportive, stem and large quantities of nectar and close-formed flower, which needs to be manoeuvred open, its pollinator is likely a short-billed generalist-feeding bird restricted by food choice during the winter months.

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Bu, Fengxiao. "Exploring the genetics of a complex disease - atypical hemolytic uremic syndrome". Diss., University of Iowa, 2016. https://ir.uiowa.edu/etd/3055.

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Atypical hemolytic uremic syndrome (aHUS) is a rare renal disorder characterized by thrombotic microangiopathy, thrombocytopenia, and acute kidney injury. Its pathogenesis has been attributed to a ‘triggering' event that leads to dysregulation of the complement cascade at the level of the endothelial cell surface. Consistent with this understanding of the disease, mutations in complement genes have been definitively implicated in aHUS. However, the existence of other genetic contributors is supported by two observations. First, in ~50% of cases, disease-causing variants are not identified in complement genes, and second, disease penetrance is typically incomplete and highly variable. To test this hypothesis, we identified pathways established to have crosstalk with the complement cascade, focusing initially on the coagulation pathway. Using targeted genomic enrichment and massively parallel sequencing we screened 36 European-American patients with sporadic aHUS patients for genetic variants in 85 complement and coagulation genes, identifying deleterious rare variants in several coagulation genes. The most frequently mutated coagulation gene in our study cohort was PLG, which encodes a zymogen of plasmin and plays key role in fibrinolysis. These results implicate the coagulation pathway in the pathogenesis of aHUS. Based on this outcome, we developed a clinical genetic testing panel to screen disease-related genes in a group of ultra-rare complement-mediated diseases that includes, in addition to aHUS, thrombotic thrombocytopenic purpura (TTP), C3 glomerulonephritis (C3GN) and dense deposit disease (DDD) patients. Data from 193 patients validate the usage of this panel in clinical practice and also provide confirmatory insight into the pathogeneses of these diseases. Specifically, we found that in aHUS and TTP patients, variants were frequently identified in complement regulator genes, while in C3GN and DDD patients, variants were additionally found in C3 convertase genes. To understand variability in disease penetrance, we completed targeted genetic screening in two aHUS families grossly discordant for disease penetrance, identifying in one family a co-segregating Factor X-deficiency variant (F10 p.Glu142Lys) that abrogated the effect of the complement mutation. Functional studies of the F10 p.Glu142Lys variant show that it decreases Factor X activity predicting to a hypo-coagulable state and further illustrating the importance of complement-coagulation crosstalk in exacerbating, but also mitigating the aHUS phenotype. In our final studies, we have sought to complete a comprehensive analysis for other potentially related pathways by using bioinformatics to identify candidate pathways coupled with whole exome sequencing. Preliminary data from 43 aHUS patients and 300 controls suggest that pathways for dermatan and heparan sulfate synthesis, which are relevant to the formation of the extra-cellular matrix and cell surface adhesion, may be implicated in the aHUS.

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Libros sobre el tema "Complex syndrome"

Lawson, Erin F. y Joel P. Castellanos, eds. Complex Regional Pain Syndrome. Cham: Springer International Publishing, 2021. http://dx.doi.org/10.1007/978-3-030-75373-3.

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Juris, Elena. Positive options for complex regional pain syndrome (CRPS): Self-help and treatment. Nashville, Tennessee: Hunter House, an imprint of Turner Publishing Company, 2014.

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Treacy, Valerie J. Gaining control of PMS: A simple approach to a complex problem. Fort Wayne, IN: V.J. Treacy, 1994.

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A cop doc's guide to public-safety complex trauma syndrome: Using five police personality styles. Amityville, N.Y: Baywood Pub. Co., 2009.

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McCleery, Robert E. Charles Harwood Complex, Saint Croix, United States, Virgin Islands. [Atlanta, Ga.?]: Dept. of Health and Human Services, Centers for Disease Control and Prevention, National Institute for Occupational Safety and Health, 2001.

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Parenting your complex child : become a powerful advocate for the autistic, Down syndrome, PDD, bipolar, or other special-needs child. New York: AMACOM, 2006.

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A holistic protocol for the immune system: HIV/ARC/AIDS, candidiasis, chronic fatigue syndrome, herpes, and other oppotunistic infections. 6^a ed. Joshua Tree, CA: Tree of Life Publications, 1995.

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C, McArthur Justin, ed. AIDS and neurology. Edinburgh: Churchill Livingstone, 1995.

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Brazier, Alex. A Double deficiency?: A report on the Social Security Act 1986 and people with acquired immune deficiency syndrome (AIDS), AIDS related complex (ARC) and HIV infection. London: Terrence Higgins Trust, 1989.

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Hickey, MichaelS. Handbook of enteral, parenteral, and ARC/AIDS nutritional therapy. St. Louis: Mosby Year Book, 1992.

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Capítulos de libros sobre el tema "Complex syndrome"

Wilson, W. A. "Family Studies and the Major Histocompatibility Complex". En Hughes Syndrome, 348–57. London: Springer London, 2000. http://dx.doi.org/10.1007/978-1-4471-3666-8_35.

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Rastogi, Rahul. "Postoperative Complex Regional Pain Syndrome". En Thoracic Outlet Syndrome, 647–53. London: Springer London, 2013. http://dx.doi.org/10.1007/978-1-4471-4366-6_92.

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Chada, Aditya, Faisal Zahiruddin y Nancy Collop. "Obesity Hypoventilation Syndrome". En Complex Sleep Breathing Disorders, 127–36. Cham: Springer International Publishing, 2021. http://dx.doi.org/10.1007/978-3-030-57942-5_11.

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Backonja, Miroslav y Victor Wang. "Peripheral Injury and CRPS". En Complex Regional Pain Syndrome, 23–32. Cham: Springer International Publishing, 2021. http://dx.doi.org/10.1007/978-3-030-75373-3_2.

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Richardson, Patricia A., Heather Poupore-King, Anya Griffin, Corinne Cooley y Rashmi P. Bhandari. "Behavioral Health Interventions for CRPS". En Complex Regional Pain Syndrome, 79–105. Cham: Springer International Publishing, 2021. http://dx.doi.org/10.1007/978-3-030-75373-3_6.

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Kitzman, Jamie y Anna Woodbury. "Adjuvant Treatments for CRPS". En Complex Regional Pain Syndrome, 149–77. Cham: Springer International Publishing, 2021. http://dx.doi.org/10.1007/978-3-030-75373-3_8.

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Goh, En Lin, Swathikan Chidambaram y Daqing Ma. "Treatment Algorithm for Complex Regional Pain Syndrome". En Complex Regional Pain Syndrome, 229–49. Cham: Springer International Publishing, 2021. http://dx.doi.org/10.1007/978-3-030-75373-3_12.

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Hudson, James, Eric Lake, Erin Spruit, Michael Terrell, Kevin Cooper, Colleen McFawn y Nicholas Gut. "Comprehensive Rehabilitation of Patients with Complex Regional Pain Syndrome". En Complex Regional Pain Syndrome, 107–48. Cham: Springer International Publishing, 2021. http://dx.doi.org/10.1007/978-3-030-75373-3_7.

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Charipova, Karina, Kyle Gress, Ivan Urits, Elyse M. Cornett, Omar Viswanath y Alan David Kaye. "Chronic Regional Pain Syndrome in the Geriatric Patient". En Complex Regional Pain Syndrome, 311–21. Cham: Springer International Publishing, 2021. http://dx.doi.org/10.1007/978-3-030-75373-3_15.

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Haight, Elena S., Nolan A. Huck, Claire E. Jordan y Vivianne L. Tawfik. "Complex Regional Pain Syndrome: An Introduction". En Complex Regional Pain Syndrome, 3–21. Cham: Springer International Publishing, 2021. http://dx.doi.org/10.1007/978-3-030-75373-3_1.

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Actas de conferencias sobre el tema "Complex syndrome"

Kim, Jae Hun. "Thermography for Complex Regional Pain Syndrome". En Quantitative InfraRed Thermography Asia 2017. QIRT Council, 2017. http://dx.doi.org/10.21611/qirt.2017.013.

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Hirata, Koichi, Keisuke Suzuki, Tomoyuki Miyamoto y Yuichi Inoue. "Restless legs syndrome (RLS)". En 2011 IEEE/ICME International Conference on Complex Medical Engineering - CME 2011. IEEE, 2011. http://dx.doi.org/10.1109/iccme.2011.5876801.

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Govor, Isabela, Gerry Mackin y Dragos Valceanu. "P574 Complex regional pain syndrome and HPV vaccine: a case report of complex regional pain syndrome and literature review". En Faculty of Paediatrics of the Royal College of Physicians of Ireland, 9th Europaediatrics Congress, 13–15 June, Dublin, Ireland 2019. BMJ Publishing Group Ltd and Royal College of Paediatrics and Child Health, 2019. http://dx.doi.org/10.1136/archdischild-2019-epa.908.

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Shrivastav, M. y S. Musley. "Spinal cord stimulation for complex regional pain syndrome". En 2009 Annual International Conference of the IEEE Engineering in Medicine and Biology Society. IEEE, 2009. http://dx.doi.org/10.1109/iembs.2009.5334418.

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Toepfer, V., G. Weinreich y H. Teschler. "Prevalence and Treatment of Complex Sleep Apnea Syndrome." En American Thoracic Society 2009 International Conference, May 15-20, 2009 • San Diego, California. American Thoracic Society, 2009. http://dx.doi.org/10.1164/ajrccm-conference.2009.179.1_meetingabstracts.a3615.

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Sghir, M., S. Salah, A. Haj Salah, W. Kessomtini y Z. Ben Salah. "AB1076 Complex regional pain syndrome type 1: which treatment?" En Annual European Congress of Rheumatology, EULAR 2018, Amsterdam, 13–16 June 2018. BMJ Publishing Group Ltd and European League Against Rheumatism, 2018. http://dx.doi.org/10.1136/annrheumdis-2018-eular.7477.

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Okabe, Shiho, Satoshi Kasagi, Maki Higuchi, Fusae Kawana, Takatoshi Kasai y Koji Narui. "Characteristics And Clinical Course Of Complex Sleep Apnea Syndrome". En American Thoracic Society 2010 International Conference, May 14-19, 2010 • New Orleans. American Thoracic Society, 2010. http://dx.doi.org/10.1164/ajrccm-conference.2010.181.1_meetingabstracts.a6532.

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Vasconcelos Pereira, A., R. Rodrigues Oliveira, R. Almeida, J. Durán y A. Reis. "78 Complex regional pain syndrome following post-herpetic neuralgia". En ESRA 2021 Virtual Congress, 8–9–10 September 2021. BMJ Publishing Group Ltd, 2021. http://dx.doi.org/10.1136/rapm-2021-esra.78.

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Gan, Jo Han, Anita Sri, Juan Kaski, Lucy Hepburn y Luke Starling. "90 Anaesthetic and peri-operative complications in paediatric patients with brugada syndrome". En GOSH Conference 2019, Care of the Complex Child. BMJ Publishing Group Ltd and Royal College of Paediatrics and Child Health, 2019. http://dx.doi.org/10.1136/archdischild-2019-gosh.90.

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Agten, C. A., A. Kobe, I. Barnaure, J. Galley, C. W. A. Pfirrmann y F. Brunner. "Role of MR Imaging in Complex Regional Pain Syndrome Revisited". En 26th Annual Scientific Meeting of the European Society of Musculoskeletal Radiology (ESSR). Georg Thieme Verlag KG, 2019. http://dx.doi.org/10.1055/s-0039-1692568.

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Informes sobre el tema "Complex syndrome"

Aleksandrov, A. V., N. V. Nikitina y N. V. Aleksandrova. USING ULTRASOUND CRITERIA FOR COMPLEX EVALUATION OF PAIN SYNDROME IN THE KNEE JOINTS IN PATIENTS WITH RHEUMATOID ARTHRITIS AND OSTEOARTHRITIS. Планета, 2018. http://dx.doi.org/10.18411/978-5-907109-24-7-2018-xxxv-10-13.

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Simberkoff, M. J. y J. Hamilton. Longterm Follow-Up of Patients in CSP No. 298 'Treatment of Patients with Acquired Deficiency Syndrome (AIDS) and AIDS Related Complex. Fort Belvoir, VA: Defense Technical Information Center, abril de 1993. http://dx.doi.org/10.21236/ada266282.

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