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Literatura académica sobre el tema "EDIL3"

Autor: Grafiati

Publicado: 4 de junio de 2021

Última modificación: 4 de febrero de 2022

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Artículos de revistas sobre el tema "EDIL3"

1

Oplawski, Marcin, Konrad Dziobek, Nikola Zmarzły, Beniamin Grabarek, Barbara Tomala, Ewa Leśniak, Iwona Adwent, Piotr Januszyk, Dariusz Dąbruś y Dariusz Boroń. "Evaluation of Changes in the Expression Pattern of EDIL3 in Different Grades of Endometrial Cancer". Current Pharmaceutical Biotechnology 20, n.º 6 (9 de julio de 2019): 483–88. http://dx.doi.org/10.2174/1389201020666190408112822.

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Background:EDIL3 is an extracellular matrix protein that plays a key role in angiogenesis. Changes in the pattern of its expression also affect cellular processes and the tumor microenvironment. Elevated level of EDIL3 is considered an unfavorable prognostic marker of survival.Objective:The aim of this study was to evaluate the changes in EDIL3 expression in endometrial cancer at various degrees of its differentiation (G1-G3) and to discuss its potential role as a molecular diagnostic marker and therapeutic target.Methods:The study group consisted of 45 patients with endometrial cancer: G1, 17; G2, 15; G3, 13. The control group (C) included 15 patients without neoplastic changes. The expression of EDIL3 was assessed using immunohistochemistry. Statistical analysis was performed using the Statistica 12 PL software (p<0.05).Results:Analysis of EDIL3 expression showed that the average optical density of the reaction product in G1 reached 130% of the control, while the values in G2 and G3 were 153% and 158%, respectively. Regardless of the endometrial cancer grade, an increase in EDIL3 level was observed compared to the control.Conclusion:In our study, we demonstrated overexpression of EDIL3 protein in endometrial cancer. Differences in expression between degrees of tumor differentiation suggest the potential of using changes in EDIL3 level as a new complementary diagnostic marker and target for anti-angiogenic therapy.

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2

Niu, Xuping, Qixin Han, Yanmin Liu, Juan Li, Ruixia Hou, Junqin Li y Kaiming Zhang. "Psoriasis-associated angiogenesis is mediated by EDIL3". Microvascular Research 132 (noviembre de 2020): 104056. http://dx.doi.org/10.1016/j.mvr.2020.104056.

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Heng, Lisong, Zhen Jia, Jian Sun, Yitong Zhao, Kun Zhang, Yangjun Zhu y Shemin Lu. "Integrated Analysis of Competing Endogenous RNAs Network Reveals Potential Signatures in Osteosarcoma Development". Technology in Cancer Research & Treatment 19 (1 de enero de 2020): 153303382095702. http://dx.doi.org/10.1177/1533033820957025.

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The purpose of this work was to extract key players such as mRNAs and long non-coding RNA (lncRNAs) in the etiopathogenesis of osteosarcoma (OS). The sequencing analyses (mRNAs and lncRNAs) of OS were conducted followed by differentially expressed mRNAs and lncRNAs (DEmRNAs and DElncRNAs) identification between U-2OS cells with has-miR-590-5p overexpression and negative control cells. Following this, the co-expression and functional enrichment analyses of DEmRNAs and DElncRNAs were carried out. Also, the miRNAs-DElncRNAs-DEmRNAs regulatory network was constructed with DElncRNAs-miRNAs and DElncRNAs-DEmRNAs pairs after the target gene analysis of miRNA. In addition, the ceRNA-has-miR-590-5p was further extracted based on the has-miR-590-5p-DElncRNAs and DElncRNAs-DEmRNAs interactions. Finally, the results of the bioinformatics analysis was verified by reverse-transcription polymerase chain reaction (RT-PCR). Totally, 980 DEmRNAs (539 up-regulated DEmRNAs and 441 down-regulated DEmRNAs) and 682 DElncRNAs (352 up-regulated DElncRNAs and 330 down-regulated DElncRNAs) were extracted between cells with hsa-miR-590-5p overexpression and normal cells. The functional analyses suggested that up-regulated genes were significantly enriched in several GO terms such as signal transduction and cytokine-cytokine receptor interaction pathway while down-regulated genes ( SCUBE3, HIST1H4E and EDIL3) were associated with calcium ion binding, cell surface function and nucleosome assembly. Additionally, the miRNAs-DEmRNAs-DEmRNAs network represented 220 pairs among 41 miRNAs, 38 DElncRNAs and 61 DEmRNAs. Furthermore, the ceRNA-hsa-miR-590-5p network consisted of 70 interaction pairs including hsa-miR-590-5p- SCUBE3-CTB-113D17.1, hsa-miR-590-5p- EDIL3-CTB-113D17.1 and hsa-miR-590-5p- HIST1H4E-CTB-113D17.1) among hsa-miR-590-5p, 30 DEmRNAs and 4 down-regulated DElncRNAs. Meanwhile, the RT-PCR results incidated that compared with the blank (KB) and negative control (NC) group, the mRNA expression of SCUBE3, HIST1H4E, and EDIL3 were significantly descreased in mimics group (P value <0.05). The lncRNA CTB-113D17.1 might implicate with OS development probably via serving as a hsa-miR-590-5p sponge to regulate gene targets ( SCUBE3, EDIL3 and HIST1H4E), which will facilitate the deep understandings of OS progression.

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Lee, Jeong-Eun, Pyong-Gon Moon, Young-Eun Cho, Young-Bum Kim, In-San Kim, Hoyong Park y Moon-Chang Baek. "Identification of EDIL3 on extracellular vesicles involved in breast cancer cell invasion". Journal of Proteomics 131 (enero de 2016): 17–28. http://dx.doi.org/10.1016/j.jprot.2015.10.005.

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5

Sun, Jian-Cong. "High expression level of EDIL3 in HCC predicts poor prognosis of HCC patients". World Journal of Gastroenterology 16, n.º 36 (2010): 4611. http://dx.doi.org/10.3748/wjg.v16.i36.4611.

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6

Feng, Ming-Xuan, Ming-Ze Ma, Ying Fu, Jun Li, Tao Wang, Feng Xue, Jian-Jun Zhang et al. "Elevated autocrine EDIL3 protects hepatocellular carcinoma from anoikis through RGD-mediated integrin activation". Molecular Cancer 13, n.º 1 (2014): 226. http://dx.doi.org/10.1186/1476-4598-13-226.

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7

Shen, Wei, Shanbang Zhu, Haifeng Qin, Ming Zhong, Jinhui Wu, Rui Zhang y Hongyuan Song. "EDIL3 knockdown inhibits retinal angiogenesis through the induction of cell cycle arrest in vitro". Molecular Medicine Reports 16, n.º 4 (31 de julio de 2017): 4054–60. http://dx.doi.org/10.3892/mmr.2017.7122.

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8

Jiang, Shu-Heng, Yang Wang, Jian-Yu Yang, Jun Li, Ming-Xuan Feng, Ya-Hui Wang, Xiao-Mei Yang et al. "Overexpressed EDIL3 predicts poor prognosis and promotes anchorage-independent tumor growth in human pancreatic cancer". Oncotarget 7, n.º 4 (28 de diciembre de 2015): 4226–40. http://dx.doi.org/10.18632/oncotarget.6772.

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9

Oh, Sin-Hye, Jung-Woo Kim, Yuri Kim, Mi Nam Lee, Min-Suk Kook, Eun Young Choi, Suhn-Young Im y Jeong-Tae Koh. "The extracellular matrix protein Edil3 stimulates osteoblast differentiation through the integrin α5β1/ERK/Runx2 pathway". PLOS ONE 12, n.º 11 (28 de noviembre de 2017): e0188749. http://dx.doi.org/10.1371/journal.pone.0188749.

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10

Xia, Hongping, Jianxiang Chen, Ming Shi, Hengjun Gao, Karthik Sekar, Veerabrahma Pratap Seshachalam, London Lucien P. J. Ooi y Kam M. Hui. "EDIL3 is a novel regulator of epithelial-mesenchymal transition controlling early recurrence of hepatocellular carcinoma". Journal of Hepatology 63, n.º 4 (octubre de 2015): 863–73. http://dx.doi.org/10.1016/j.jhep.2015.05.005.

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Tesis sobre el tema "EDIL3"

1

Sutanto, Ricardo [Verfasser], Claudia [Akademischer Betreuer] Günther y Elisabeth [Gutachter] Naschberger. "Deregulated Expression of SPARCL1, EDIL3 and HEY2 in Primary Colorectal Carcinoma and Corresponding Liver Metastasis / Ricardo Sutanto ; Gutachter: Elisabeth Naschberger ; Betreuer: Claudia Günther". Erlangen : Friedrich-Alexander-Universität Erlangen-Nürnberg (FAU), 2021. http://d-nb.info/1227040865/34.

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2

Araújo, Carlos Jorge Trindade. "Molecular characterization of a de novo t (11;18) translocation associated with Peter´s anomaly". Master's thesis, Faculdade de Ciências e Tecnologia, 2013. http://hdl.handle.net/10362/11893.

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Dissertação para obtenção do grau de Mestre em Genética Molecular e Biomedicina
Peter’s anomaly (PA) is a congenital defect of the anterior chamber of the eye. The aim of this study is molecular characterization of a de novo balanced chromosome translocation [t(11;18)(q23.3;q11.2)] identified in a proband with syndromic form of Peter´s anomaly (ectopia lentis and mild CNS abnormalities). Chromosome breakpoints were identified at nucleotide resolution. The 11q23.3 breakpoint is at position 120,097,868 (genome assembly GRCh37/hg19) within intron 3 of out of first protein homolog gene (OAF) while, 18q11.2 breakpoint in the intergenic region between CTAGE1 and RBBP8 genes, at position 20,220,714. Although OAF is disrupted, its expression level is unchanged in proband´s lymphoblastoid cell line (LCL). Cell adhesion protein Nectin 1 or PVRL1, located 500 kb upstream from 11q23.3 breakpoint, reveal 3.5 fold increase in proband’s LCL. Expression levels of additional genes from breakpoint regions are not significantly different. Furthermore, RT-qPCR confirmed that expression level CYP1B1 from chromosome 2p22.2, and EDIL3 from chromosome 5q14 are significantly changed in proband´s LCL, 16.5 fold decrease and 126 fold increase, respectively. Alterations in CYP1B1 have been reported as PA causing mutations. Therefore, mutation screening of CYP1B1 was performed and no pathogenic mutation was identified in the proband, although, a disease-causing 13 bp, homozygous deletion was identified in a Portuguese patient with PA. In conclusion, we hypothesize that PVRL1 is a candidate gene for at least some of the observed clinical features, which is elicited in mouse models by association of its paralog PVRL3 to lens and other ocular defects involving the ciliary body. Furthermore, the involvement of the POU family domain containing transcription factor (POU2F3) from 11q23.3 and CYP1B1 or EDIL3, localized outside of the breakpoint regions, in the underlying molecular pathogenesis of syndromic PA remains to be determined.

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Stapane, Lilian. "Biominéralisation de la coquille d oeuf de poule : mise en évidence d'un transport vésiculaire du minéral impliquant les protéines EGF-like repeats and discoidin-like domains 3 (EDIL3) et Milk fat globule EGF-factor 8 (MFGE8)". Thesis, Tours, 2019. http://www.theses.fr/2019TOUR4026.

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L’œuf de poule est une chambre aseptique et autosuffisante qui permet le développement harmonieux du poussin en 21 jours. La coquille constitue la première barrière physique de l’œuf et protège ce dernier contre les agressions physiques et microbiennes. Lors de la minéralisation de la coquille dans le fluide utérin, la matrice organique secrétée par l’utérus de la poule joue un rôle prépondérant. Pour décrypter le rôle de deux protéines majeures de la matrice organique (EGF-like repeats and discoidin-like domains 3 : EDIL3 et Milk fat globule EGF-factor 8 : MFGE8), différentes approches in silico et expérimentales ont été réalisées. Ainsi, nous avons retracé l’histoire évolutive de ces deux protéines, qui indique une divergence d’EDIL3 et MFGE8, il y a environ 480 millions d’années. Les résultats obtenus nous ont également permis de mettre en évidence un transport vésiculaire du minéral (carbonate de calcium amorphe), impliqué dans ce phénomène de biominéralisation. EDIL3 et MFGE8 joueraient un rôle prépondérant dans ce mécanisme en participant à l’adressage sur le site de minéralisation. Ce travail a été complété par une analyse des autres acteurs moléculaires du transport vésiculaire. Ces acteurs participent au fonctionnement vésiculaire, à l’apport du calcium et des bicarbonates vésiculaires, ainsi qu’à la stabilisation du carbonate de calcium amorphe. Pour finir, nous proposons un modèle global et cohérent du transport des précurseurs minéraux nécessaires à la minéralisation de la coquille. Ce modèle impliquerait trois voies (transcellulaire, vésiculaire et paracellulaire) dont les rôles respectifs devront être explorés
The chicken egg is an aseptic and a self-sufficient chamber allowing the harmonious development of the chicken embryo in 21 days. The eggshell is the first physical barrier of the egg, which protects the egg from mechanical and microbial stresses. During eggshell biomineralization in the uterine fluid, the organic matrix, which is secreted by the uterus, ensures a crucial role in this process. To decipher the role of two major proteins of the organic matrix (EGF-like repeats and discoidin-like domains 3 : EDIL3 et Milk fat globule EGF-factor 8 : MFGE8), in silico and experimental approaches were performed. We firstly investigated the common evolution history of EDIL3 and MFGE8. We have estimated a divergent point between both paralogs 480 million years ago. The results also allowed us to highlight a vesicular transport of mineral (amorphous calcium carbonate) involved in eggshell biomineralization. EDIL3 and MFGE8 proteins would guide the vesicles to the mineralizing site and consequently ensure a critical role in this transport. This work also described additional molecular actors involved in vesicles formation, in calcium and bicarbonates supply and to the stabilization of amorphous calcium carbonate. We therefore proposed a global and coherent model of mineral transport during eggshell calcification. In this model, three alternative pathways could be involved (transcellular, vesicular and paracellular). Further investigations will be necessary to determine the respective roles of these pathways

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4

Pondini, Ellero. "Sistema decisionale per la scelta delle macchine dei cantieri civili ed edili". Bachelor's thesis, Alma Mater Studiorum - Università di Bologna, 2009. http://amslaurea.unibo.it/288/.

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5

Galmiche, Marie. "Epidémiologie des troubles du comportement alimentaire et analyses biocliniques des patients de la cohorte EDILS". Thesis, Normandie, 2019. http://www.theses.fr/2019NORMR066/document.

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Les troubles du comportement alimentaire (TCA) touchent une part importante de la population et constitue un réel problème de santé publique, il est indispensable d’approfondir nos connaissances sur la physiopathologie des TCA afin d’identifier des perspectives thérapeutiques. L’écriture d’une revue systématique a permis d’évaluer la prévalence des TCA selon le sexe, l’origine géographique ou l’âge de la population étudiée et confirme son augmentation au cours des 15 dernières années. L’analyse des données épidémiologiques des patients avec un TCA de la cohorte Eating Disorder and Longitudinal Survey (EDILS) a souligné le stress et le régime alimentaire comme facteurs de survenue et des similarités entre les comorbidités des 3 catégories larges de TCA (Restrictive, boulimique et compulsive). Ces comorbidités étaient fortement associées entre elles et, selon un cercle vicieux pourrait contribuer au maintien de la physiopathologie des TCA. La littérature suggère l’implication d’une dérégulation peptidergique de la prise alimentaire au cours des TCA qui pourrait inclure un mécanisme immunitaire. Les analyses des échantillons plasmatiques de la cohorte ont permis de mieux définir les profils biologiques de peptides et immunoglobulines associés aux 3 catégories larges de TCA et d’évaluer leur utilité pour le phénotypage des patients. La collecte de selles dans la cohorte offre également la possibilité de préciser les profils de microbiote au cours des différents types de TCA. Cette thèse a donc permis de mieux définir les caractéristiques cliniques des TCA et les perturbations de l’axe microbiote-intestin-cerveau associées, en particulier les dérèglements biologiques
Eating disorders (ED) affect a large population and are a serious public health issue. It is thus essential to deepen our knowledge of ED pathophysiology in order to open new therapeutic perspectives. The prevalence of ED according to sex, geographical origin or age of the study population and its increase over the last 15 years has been highlighted in a systematic review. The analysis of patients with ED from the Eating Disorder and Longitudinal Survey (EDILS) cohort underlined stress and / or diet as triggering factors and noted similarities between the comorbidities of the 3 broad categories of ED (Restrictive, bulimic and compulsive). These comorbidities were strongly associated with each other, and contributed in a vicious circle to the perpetuation of ED. The literature suggests during ED a dysregulation of neuropeptide signaling of food intake that may include some immunological mechanisms. Analyzes of plasma samples from the EDILS cohort allowed a better definition of the peptides and immunoglobulins profiles associated with the 3 broad categories of ED and to evaluate their utility for phenotyping patients. The collection of feces in the cohort offers the possibility of refining the specificity of the microbiota according to different type of ED. This thesis allows to better define the clinical characteristics of 3 broad categories of ED and disturbances of the microbiota-intestine-brain axis, particularly the associated biological dysregulations of neuropeptide signaling

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6

Pelser, Stephan. "Funktionelle Analyse der EID1 LIKE Gene EDL1, EDL2 und EDL3 von Arabidopsis thaliana". [S.l. : s.n.], 2006.

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Acciarino, Andrea. "L'ottimizzazione del layout del cantiere edile attraverso l'utilizzo di algoritmi genetici multi-obiettivo: caso di studio". Master's thesis, Alma Mater Studiorum - Università di Bologna, 2019.

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Un buon layout di cantiere è fondamentale per garantire il corretto svolgimento delle lavorazioni e le condizioni di sicurezza al suo interno. Il progetto del cantiere ha un impatto significativo sui costi e sulla durata della costruzione. Il progetto del layout di cantiere comprende la ricerca della collocazione efficace ed efficiente delle facility temporanee e permanenti all’interno del sito dove sorgerà la costruzione. Per questo motivo, il progetto del layout di cantiere può essere considerato come un problema di ottimizzazione dove obiettivi spesso conflittuali devono essere raggiunti. Negli ultimi anni, il problema è stato risolto con varie tecniche di ottimizzazione metaeuristiche basate sulla minimizzazione di un criterio legato ai costi di costruzione e, talvolta, dei rischi per la sicurezza. L’obiettivo di questa tesi è quello di formulare un modello di ottimizzazione multi-obiettivo del layout di cantiere in grado di minimizzare il costo totale di interazione tra le facility e i rischi in termini di sicurezza. Il modello è stato sviluppato con l’utilizzo di algoritmi genetici, implementati in un software di modellazione parametrica, in grado di trovare l’insieme degli ottimi di Pareto. In conclusione, il modello sviluppato è stato utilizzato per la risoluzione di un caso studio reale per analizzarne le prestazioni.

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8

Servadei, Martina. "Metodi di Project Management e Lean Construction per la programmazione dei lavori nel cantiere edile: caso di studio". Master's thesis, Alma Mater Studiorum - Università di Bologna, 2019.

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Il lavoro di tesi svolto consiste nel confronto tra diverse metodologie di programmazione dei lavori della fase impiantistica di un cantiere sito a San Giovanni in Persiceto, composto da tre unità abitative disposte ognuna su tre livelli. Le tre differenti programmazioni sono state confrontate secondo tre parametri fondamentali quali tempo totale di cantiere, valore di denaro prodotto per settimana e indice di tempo di fermo delle squadre di lavoro. Il punto di partenza è l’analisi della programmazione effettuata con un approccio di Project Management tradizionale attraverso l’utilizzo del software Microsoft Project. Successivamente l’analisi si è spostata allo studio e applicazione della Teoria dei Vincoli di E. Goldratt, che, attraverso l’eliminazione di alcuni errori tipici che vengono effettuati sia in fase di progettazione che di esecuzione mira ad una riduzione delle tempistiche e quindi dei costi complessivi. Infine, le varie lavorazioni di cantiere sono state schedulate secondo metodi e concetti di Lean Construction con l’utilizzo del software Vico Control.

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Tortorella, Giulia. "La strategia BIM a supporto del progetto della sicurezza nel cantiere edile: il caso dell'ospedale di primo soccorso a Lampedusa". Master's thesis, Alma Mater Studiorum - Università di Bologna, 2014. http://amslaurea.unibo.it/7546/.

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Throughout this research, the whole life cycle of a building will be analyzed, with a special focus on the most common issues that affect the construction sector nowadays, such as safety. In fact, the goal is to enhance the management of the entire construction process in order to reduce the risk of accidents. The contemporary trend is that of researching new tools capable of reducing, or even eliminating, the most common mistakes that usually lead to safety risks. That is one of the main reasons why new technologies and tools have been introduced in the field. The one we will focus on is the so-called BIM: Building Information Modeling. With the term BIM we refer to wider and more complex analysis tool than a simple 3D modeling software. Through BIM technologies we are able to generate a multi-dimension 3D model which contains all the information about the project. This innovative approach aims at a better understanding and control of the project by taking into consideration the entire life cycle and resulting in a faster and more sustainable way of management. Furthermore, BIM software allows for the sharing of all the information among the different aspects of the project and among the different participants involved thus improving the cooperation and communication. In addition, BIM software utilizes smart tools that simulate and visualize the process in advance, thus preventing issues that might not have been taking into consideration during the design process. This leads to higher chances of avoiding risks, delays and cost increases. Using a hospital case study, we will apply this approach for the completion of a safety plan, with a special focus onto the construction phase.

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Chetvertkova, Vera [Verfasser], Edil [Akademischer Betreuer] Mustafin, Ulrich [Akademischer Betreuer] Ratzinger y Oliver [Akademischer Betreuer] Kester. "Verification of Monte Carlo transport codes by activation experiments / Vera Chetvertkova. Gutachter: Ulrich Ratzinger ; Oliver Kester. Betreuer: Edil Mustafin". Frankfurt am Main : Univ.-Bibliothek Frankfurt am Main, 2013. http://d-nb.info/104409401X/34.

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Libros sobre el tema "EDIL3"

1

Caniato, Giovanni. Le arti edili a Venezia. Roma: Edilstampa, 1990.

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2

Bellardi, Lauralba. Istituzioni bilaterali e contrattazione collettiva: Il settore edile (1945-1988). Milano, Italy: F. Angeli, 1989.

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Parenti, Marco. Ël mal dla pera: La parlata piemontese nel cantiere edile. Dronero (Cuneo): L'arciere, 2001.

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Giuseppe, Saitta. Fare i mattoni senza la paglia: La cooperativa edile "La Sicilia" nei miei ricordi. Palermo: Dharba, 1991.

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Lella, Raffaele di. Contratto collettivo dell'edilizia: Commento al C.C.N.L. delle imprese industriali ed artigiane edili. Napoli: Casa editrice Jovene, 1989.

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Gallo, Stefano. Costruire insieme: La bilateralità nelle costruzioni : storia dell'Ente livornese cassa edile, 1962-2012. Pisa: ETS, 2012.

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Menzani, Tito. La cooperazione edile nel Lughese e nel Faentino dalla Liberazione alla RES Coop. Ravenna: Longo, 2003.

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Luoghi per lavorare, pregare, morire: Edifici e maestranze edili negli interessi delle élites micenee. Firenze: Firenze University Press, 2013.

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Carotti, Attilio. Prontuario di ingegneria edile e strutturale: Tecnologie innovative e applicazioni avanzate in ingegneria civile. Torino: UTET, 2000.

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Pennesi, Paolo. Sicurezza e appalti nei cantieri edili: Nuovi obblighi penali in vigore dal 18 aprile 2000. Torino: G. Giappichelli, 2000.

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Capítulos de libros sobre el tema "EDIL3"

1

"Introduzione". En Edilo, ›Epigrammi‹, 1–55. De Gruyter, 2020. http://dx.doi.org/10.1515/9783110630008-001.

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"Testo e commento". En Edilo, ›Epigrammi‹, 61–188. De Gruyter, 2020. http://dx.doi.org/10.1515/9783110630008-002.

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"Bibliografia". En Edilo, ›Epigrammi‹, 191–232. De Gruyter, 2020. http://dx.doi.org/10.1515/9783110630008-003.

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"Index verborum". En Edilo, ›Epigrammi‹, 233–38. De Gruyter, 2020. http://dx.doi.org/10.1515/9783110630008-004.

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"Criteri della presente edizione". En Edilo, ›Epigrammi‹, 56–57. De Gruyter, 2020. http://dx.doi.org/10.1515/9783110630008-005.

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"Conspectus siglorum". En Edilo, ›Epigrammi‹, 58. De Gruyter, 2020. http://dx.doi.org/10.1515/9783110630008-006.

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"Testimonia". En Edilo, ›Epigrammi‹, 59–60. De Gruyter, 2020. http://dx.doi.org/10.1515/9783110630008-007.

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"Auctarium lectionum. Appendix coniecturarum". En Edilo, ›Epigrammi‹, 189–90. De Gruyter, 2020. http://dx.doi.org/10.1515/9783110630008-008.

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"Index locorum". En Edilo, ›Epigrammi‹, 239–41. De Gruyter, 2020. http://dx.doi.org/10.1515/9783110630008-009.

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"Index nominum et rerum notabilium". En Edilo, ›Epigrammi‹, 242–49. De Gruyter, 2020. http://dx.doi.org/10.1515/9783110630008-010.

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Actas de conferencias sobre el tema "EDIL3"

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Beckham, Carla, Christopher Silvers, Jayme Olsen, Peng-nien Yin, Chia-Hao Wu, Huei-ju Ting, Fred Hagen, Emelian Scosyrev y Edward Messing. "Abstract B05: Urothelial carcinoma exosomes contain Del1/EDIL-3 and facilitate bladder cancer progression". En Abstracts: Third AACR International Conference on Frontiers in Basic Cancer Research - September 18-22, 2013; National Harbor, MD. American Association for Cancer Research, 2013. http://dx.doi.org/10.1158/1538-7445.fbcr13-b05.

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