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Tesis sobre el tema "Glycogénose de type III"

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Publicado: 14 de diciembre de 2024
Última modificación: 31 de julio de 2025

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1

Vidal, Patrice. "Développement d'un traitement de thérapie génique pour la glycogénose de type III." Electronic Thesis or Diss., Sorbonne université, 2018. http://www.theses.fr/2018SORUS571.

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La glycogénose de type III (GSDIII) est une maladie génétique récessive due à des mutations affectant l’activité de l'enzyme de débranchement du glycogène (GDE). Les symptômes sont une hépatomégalie et une hypoglycémie chez l’enfant puis une faiblesse musculaire dégénérative chez l’adulte. Aucun traitement curatif n'existe pour GSDIII. Nous avons dans un premier temps développé un modèle de souris GSDIII viable possédant phénotype proche de la maladie de l’homme. La thérapie génique permet le traitement des maladies métaboliques et neuromusculaires. En thérapie génique in vivo, les vecteurs dé
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2

Rossiaud, Lucille. "Modélisation et compréhension de la glycogénose de type III grâce à l'utilisation de cellules souches pluripotentes induites humaines." Electronic Thesis or Diss., université Paris-Saclay, 2024. https://www.biblio.univ-evry.fr/theses/2024/interne/2024UPASL091.pdf.

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La glycogénose de type III (GSDIII) est une maladie génétique rare due à un déficit en enzyme débranchante du glycogène (GDE), provoquant une accumulation de glycogène dans le foie, le cœur et les muscles squelettiques. Alors que les atteintes hépatiques dominent durant l'enfance, les atteintes musculaires progressent et deviennent prédominantes à l'âge adulte. L'absence de modèles humains freine la compréhension de cette pathologie et la mise au point de traitements.Dans ce contexte, mon premier objectif était de créer des modèles pathologiques humains in vitro à partir de cellules souches pl
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3

Douillard-Guilloux, Gaëlle. "Nouvelles approches thérapeutiques dans la glycogénose de type 2." Paris 7, 2008. http://www.theses.fr/2008PA077119.

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La glycogénose de type II est une maladie liée au déficit en alpha-glucosidase acide. Elle est caractérisée par une accumulation intra-lysosomale de glycogène dans le muscle squelettique, Notre travail a été de développer de nouvelles approches thérapeutiques pour cette pathologie. Nous avons tout d'abord tenté de moduler la synthèse du glycogène (thérapie par inhibtion de substrat) en inhibant les enzymes clés de sa biosynthèse : la glycogénine (GYG) et la glycogène synthase (GYS), à l'aide de shRNA. Un vecteur viral AAV contenant le shARN-GYS2 a été construit et injecté par voie intramuscula
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4

Hordeaux, Juliette. "Thérapie génique des manifestations neurologiques de la maladie de Pompe (glycogénose de type II)." Nantes, 2014. http://www.theses.fr/2014NANT2098.

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La maladie de Pompe ou glycogénose de type II est une maladie de surcharge Iysosomiale causée par la mutation de l'enzyme alpha-glucosidase acide (GAA) à l'origine d'une accumulation de glycogène dans le coeur, les muscles et le système nerveux central (SNC). La forme infantile classique est caractérisée par une hypotonie et une cardiomyopathie fatale. La correction du coeur par enzymothérapie a permis d'allonger l'espérance de vie des patients révélant une nouvelle histoire naturelle. Le phénotype neurologique émergent et la correction partielle des muscles pourraient être liés à la surcharge
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5

Clar, Julie. "Nouvelles stratégies d’étude et de prévention des complications hépatorénales de la glycogénose de type Ia." Thesis, Lyon 1, 2014. http://www.theses.fr/2014LYO10163.

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La glycogénose de type Ia (GSDIa) est une maladie métabolique rare causée par un déficit en glucose-6- phosphatase (G6Pase), menant à l'absence de production endogène de glucose. Cette pathologie est caractérisée par des hypoglycémies sévères, une hépatomégalie et une stéatose hépatique ainsi qu'une néphromégalie. En absence de traitement curatif, la prise en charge de cette maladie repose actuellement sur des mesures diététiques très strictes. Cependant, des complications apparaissent avec l'âge comme le développement de tumeurs hépatiques et la progression de la néphropathie vers l'insuffisa
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6

Nicolino, Marc. "Glycogénose Type II (Maladie de Pompe) : approche d'une thérapie génique et caractérisation des anomalies moléculaires." Paris 5, 1999. http://www.theses.fr/1999PA05CD17.

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7

Mutel, Élodie. "Caractérisation d'un nouveau modèle murin de glycogénose de type 1a : du métabolisme glucidique à la thérapie génique." Phd thesis, Université Claude Bernard - Lyon I, 2011. http://tel.archives-ouvertes.fr/tel-00858006.

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La glycogénose de type 1a (GSD1a) est une maladie métabolique rare liée à une absence d'activité glucose‐6 phosphatase (G6Pase). La G6Pase est une enzyme clé de la production endogène de glucose (PEG) catalysant l'hydrolyse du G6P en glucose avant sa libération dans le sang. Cette fonction est restreinte au foie, aux reins et à l'intestin. La GSD1a est caractérisée par des hypoglycémies chroniques, une hépatomégalie associée à une stéatose hépatique et une néphromégalie. A plus longterme, la plupart des patients développent des adénomes. Un modèle murin de GSD 1a existe mais les souris ne surv
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8

Mutel, Élodie. "Caractérisation d’un nouveau modèle murin de glycogénose de type 1a : du métabolisme glucidique à la thérapie génique." Thesis, Lyon 1, 2011. http://www.theses.fr/2011LYO10005/document.

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La glycogénose de type 1a (GSD1a) est une maladie métabolique rare liée à une absence d’activité glucose‐6 phosphatase (G6Pase). La G6Pase est une enzyme clé de la production endogène de glucose (PEG) catalysant l’hydrolyse du G6P en glucose avant sa libération dans le sang. Cette fonction est restreinte au foie, aux reins et à l’intestin. La GSD1a est caractérisée par des hypoglycémies chroniques, une hépatomégalie associée à une stéatose hépatique et une néphromégalie. A plus longterme, la plupart des patients développent des adénomes. Un modèle murin de GSD 1a existe mais les souris ne surv
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9

Monteillet, Laure. "La maladie chronique rénale de la glycogénose de type I, des mécanismes moléculaires aux nouvelles stratégies thérapeutiques." Thesis, Lyon, 2019. http://www.theses.fr/2019LYSE1140.

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La glycogénose de type Ia (GSDIa) est une maladie métabolique rare causée par une déficience en glucose-6-phosphatase (G6Pase), due à des mutations de la sous-unité catalytique (G6PC). Cette enzyme confère au foie, aux reins et à l’intestin la capacité de produire du glucose. Les patients atteints de GSDIa sont donc incapables de produire du glucose et souffrent d’hypoglycémies sévères lors de jeûnes courts. De plus, la déficience en G6Pase provoque une accumulation de glucose-6 phosphate dans le foie et les reins, conduisant à l’accumulation de glycogène et de lipides. A long terme, la plupar
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10

Shelly, Claire. "Type III subfactors and planar algebras." Thesis, Cardiff University, 2012. http://orca.cf.ac.uk/44565/.

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In this thesis we investigate the theory of planar algebras for type III subfactors. We show directly how to associate a planar algebra to a type III subfactor using endomorphisms and intertwiners. We begin by describing how to define a type III version of the Temperley-Lieb planar algebra before giving a general definition of a type III planar algebra. We define a presenting map using endomorphisms and intertwiners and prove that this defines a type III subfactor planar algebra. We show that the definition of a type III subfactor planar algebra may be extended by removing the sphericality con
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11

Bagaud, Catherine. "Aspect physiopathologique et clinique de la glycogénose 1 bis et intérêt du facteur de croissance dans son traitement à propos d'un cas." Bordeaux 2, 1994. http://www.theses.fr/1994BOR2M206.

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12

Pichon, Julien. "Impact de la surexpression de FoxO3a sur la physiopathologie musculaire de la maladie de Pompe (Glycogénose de type II)." Thesis, Nantes, 2020. http://archive.bu.univ-nantes.fr/pollux/show.action?id=36cea62a-e454-42c5-a21c-1fd5b915de61.

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La maladie de Pompe est une maladie de surcharge lysosomiale due à une mutation de l'alphaglucosidase acide (GAA) conduisant notamment à une atteinte musculaire sévère. Aucun traitement ne permet de corriger efficacement et durablement le muscle squelettique. Une meilleure compréhension des mécanismes pathogéniques impliqués permettrait de proposer des stratégies thérapeutiques plus adaptées. Les travaux présentés ici ont pour objectifs i) de caractériser la physiopathologie musculaire de la maladie de Pompe grâce au modèle murin Gaa"' et ii) d'évaluer l'effet de la surexpression de Fox03a sur
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13

Louw, Cassandra Alexandrovna. "Characterisation of Trypanosomal Type III and Type IV Hsp40 proteins." Thesis, Rhodes University, 2009. http://hdl.handle.net/10962/d1003985.

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The heat shock protein-70 (Hsp70) family of molecular chaperones are ubiquitous highly conserved proteins that are critical for the viability of cellular homeostasis. The ATPase activity of Hsp70 proteins is critical to their function as the affinity of a given Hsp70 for non-native substrate is modulated by ATP binding and hydrolysis. When bound to ATP, Hsp70s possess a low affinity for a given substrate protein, while the hydrolysis of ATP to ADP causes a conformational change that results in a high affinity for substrate proteins. The basal ATPase activity of Hsp70s is too low to facilitate
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14

Liljeholm, Maria. "Congenital Dyserythropoietic Anemia type III (CDA III) : diagnostics, genetics and morbidity." Doctoral thesis, Umeå universitet, Institutionen för strålningsvetenskaper, 2016. http://urn.kb.se/resolve?urn=urn:nbn:se:umu:diva-117454.

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The Congenital Dyserythropoietic Anemias (CDA) are rare hereditary hemolytic disorders with large bi- to multi-nucleated erythroblasts in the bone marrow. Hemolysis is negative in a direct antiglobulin test (DAT). Based on morphology and clinical picture, three major forms of CDAs, type I, II, and III have been defined. CDA III, dominantly inherited, constitutes the rarest type with a majority of cases belonging to a family in Västerbotten, Sweden. The genetic background of CDA I and CDA II has been linked to mutations in CDAN1 and SEC23B respectively. The mutation of CDA III has been linked t
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15

Bode, M. (Michaela). "Characterization of type I and type III collagens in human tissues." Doctoral thesis, Oulun yliopisto, 2000. http://urn.fi/urn:isbn:9514255534.

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Abstract Fibrillar type I and III collagens are the major constituents of the extracellular matrix, providing the tissue with tensile strength and influencing cell attachment and migration. The amount of type III collagen and the extent of its processing and cross-link maturation were studied in human atherosclerotic plaques, abdominal aortic aneurysms, colon and ovarian cancer, and finally, colon diverticulosis, using a novel radioimmunoassay for the cross-linked aminoterminal telopeptide of type III collagen. In addition, immunoassays for different structural domains of type I and type III c
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16

Peplinski, Adam. "Numerical simulations of type III planetary migration." Doctoral thesis, Stockholm University, Department of Astronomy, 2008. http://urn.kb.se/resolve?urn=urn:nbn:se:su:diva-7461.

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<p>Planets are believed to form in primordial gas-dust discs surrounding newborn stars. An important breakthrough in our understanding of planetary formation was the discovery of extra-solar planets around sun-like stars, especially the frequent occurrence of giant planets on close orbits (hot Jupiters). The mechanisms involved in the formation of these objects remain uncertain, however the difficulties associated with their formation at their observed orbital radius has awoken an interest in theories for the migration of protoplanetary cores due to gravitational interaction with the disc. The
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17

Singh, Mona P. "Type III secretion in enteropathogenic Escherichia coli." Thesis, Imperial College London, 2008. http://ethos.bl.uk/OrderDetails.do?uin=uk.bl.ethos.486563.

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Type III secretion systems (T3SS) are complex multi-component structures that are utilised in two biological contexts: translocation ofbacterial effector proteins into eukaryotic cells and flagellar protein export. The two types ofT3SS share a conserved core set ofhomologous proteins which are integrated into a larger apparatus that includes a hol1'ow filamentous surface extension, which comprises the flagellar hook and filament, or the needle and the translocation apparatus in the case of flagellar or non-flagellar systems, respectively. Enteropathogenic Escherichia coli (EPEC) and enterohaem
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18

Baker, M. G. "Investigations on the type-III rearrangement problem." Thesis, University of Cambridge, 1985. http://ethos.bl.uk/OrderDetails.do?uin=uk.bl.ethos.372877.

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19

Martin-Touaux, Elsa. "Caractérisation physiopathologique de la glycogénose de type II et développement de différentes approches thérapeutiques sur un modèle murin de la maladie." Paris 7, 2003. http://www.theses.fr/2003PA077235.

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20

Ulander, Anna Karin. "KIF23 expression in congenital dyserythropoietic anemia type III." Thesis, Umeå universitet, Biomedicinsk laboratorievetenskap, 2012. http://urn.kb.se/resolve?urn=urn:nbn:se:umu:diva-57964.

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21

Billings, K. S. "Studies on core-swapped fibronectin type III domains." Thesis, University of Cambridge, 2007. http://ethos.bl.uk/OrderDetails.do?uin=uk.bl.ethos.596637.

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Two homologous fibronectin type III (fnIII) domains, FNfn10 (the tenth fibronectin type III domain of human fibronectin) and TNfn3 (the third fibronectin type III domain of human tenascin), have, essentially, the same backbone structure although they share only ~24% of sequence identity. FNoTNc is a core swapped protein, containing the “outside” (surface and loops) of FNfn10 and the hydrophobic core of TNfn3. Despite the extent of redesign, FNoTNc retains the structure of the parent proteins allowing us to gain insights into which components of each parent protein are responsible for different
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22

Li, Zhiya. "New Bismuth(III)-catalyzed Sakurai type Allylation Reactions." Thesis, Université Laval, 2010. http://www.theses.ulaval.ca/2010/27512/27512.pdf.

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23

Pallett, Mitchell. "Characterisation of the type III secretion effector NleF." Thesis, Imperial College London, 2015. http://hdl.handle.net/10044/1/28244.

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The human-specific enteropathogenic Escherichia coli and the mouse-restricted Citrobacter rodentium colonise the host intestinal mucosa via attaching and effacing lesions, inducing severe diarrhoeal disease and transmissible murine colonic hyperplasia, respectively. C. rodentium is widely used as an in vivo model for EPEC infection, sharing a highly homologous arsenal of virulence factors and a similar infection strategy with EPEC. EPEC and C. rodentium pathogenesis relies on the locus of enterocyte effacement-encoded type III secretion system and delivery of effectors into the host cytosol to
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24

Benrabah, Sabria. "Passivation des matériaux III-N de type GaN." Thesis, Lyon, 2021. http://www.theses.fr/2021LYSE1310.

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Pour répondre aux demandes de développement de nouveaux produits dans les domaines des convertisseurs électroniques de puissance pour les voitures électriques, des panneaux solaires, des éoliennes et des nouvelles technologies d'éclairage à base de LED ou de composants RF, la recherche s'est concentrée sur les matériaux à large bande interdite directe, dont le nitrure de gallium (GaN). Le GaN a suscité un grand intérêt en raison de ses propriétés exceptionnelles pour les dispositifs électroniques de puissance de la prochaine génération. Avec une vitesse de saturation élevée et une tension de f
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25

SPOSITO, BENEDETTA. "Type III Interferons: Running Interference with Mucosal Repair." Doctoral thesis, Università degli Studi di Milano-Bicocca, 2023. https://hdl.handle.net/10281/402377.

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Gli interferoni (IFN) sono mediatori e regolatori fondamentali della risposta immunitaria dell'ospite a virus e ad altri agenti microbici. Gli IFN di tipo I e di tipo III (o IFN-λ) sono tra le prime citochine ad essere indotte in seguito a infezioni virali. Il legame tra gli IFN e i rispettivi recettori attiva vie di trasduzione del segnale simili tra loro che inducono l'espressione di geni stimolati dagli IFN (ISG) con funzioni antivirali. La caratteristica principale che rende ciascuna di queste famiglie di IFN unica e non ridondante è l'esistenza di recettori distinti che fanno sì che gli I
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26

Gjorgjieva, Monika. "Identification des mécanismes moléculaires impliqués dans le développement des pathologies hépatiques et rénales dans des modèles murins de glycogénose de type 1a." Thesis, Lyon, 2018. http://www.theses.fr/2018LYSE1007/document.

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La glycogénose de type I (GSDI) est une maladie génétique rare, due à une déficience en glucose-6 phosphatase (G6Pase), enzyme clé de la production endogène de glucose. En plus des hypoglycémies sévères, la perte de l'activité G6Pase conduit à l'accumulation de glycogène, mais aussi de lipides dans le foie et les reins. A long-terme, la plupart des patients développent des tumeurs hépatiques et une maladie rénale chronique (MRC).Le but de cette thèse a été de caractériser les mécanismes moléculaires impliqués dans la carcinogenèse hépatique et la MRC grâce à des modèles murins viables et uniqu
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27

Gjorgjieva, Monika. "Identification des mécanismes moléculaires impliqués dans le développement des pathologies hépatiques et rénales dans des modèles murins de glycogénose de type 1a." Electronic Thesis or Diss., Lyon, 2018. http://www.theses.fr/2018LYSE1007.

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La glycogénose de type I (GSDI) est une maladie génétique rare, due à une déficience en glucose-6 phosphatase (G6Pase), enzyme clé de la production endogène de glucose. En plus des hypoglycémies sévères, la perte de l'activité G6Pase conduit à l'accumulation de glycogène, mais aussi de lipides dans le foie et les reins. A long-terme, la plupart des patients développent des tumeurs hépatiques et une maladie rénale chronique (MRC).Le but de cette thèse a été de caractériser les mécanismes moléculaires impliqués dans la carcinogenèse hépatique et la MRC grâce à des modèles murins viables et uniqu
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28

Costa, Tiago R. D. "YopD translocator function in Yersinia pseudotuberculosis type III secretion." Doctoral thesis, Umeå universitet, Institutionen för molekylärbiologi (Teknisk-naturvetenskaplig fakultet), 2012. http://urn.kb.se/resolve?urn=urn:nbn:se:umu:diva-61544.

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Type III secretion systems (T3SS) are a common feature of Gram-negative bacteria, allowing them to inject anti-host effectors into the interior of infected eukaryotic cells. By this mechanism, these virulence factors help the bacteria to modulate eukaryotic cell function in its favor and subvert host innate immunity. This promotes a less hostile environment in which infecting bacteria can colonize and cause disease. In pathogenic Yersinia, a crucial protein in this process is YopD. YopD is a T3S substrate that, together with YopB, forms a translocon pore in the host cell membrane through which
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29

Xiao, Yanmei. "Regulation of type III secretion system in Pseudomonas syringae." Diss., Manhattan, Kan. : Kansas State University, 2005. http://hdl.handle.net/2097/130.

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30

Main, Alison. "Structural and functional studies of fibronectin type III molecules." Thesis, University of Oxford, 1993. http://ethos.bl.uk/OrderDetails.do?uin=uk.bl.ethos.359449.

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31

Xu, Xuefang. "Regulation of type III secretion in enterohaemorrhagic Escherichia coli." Thesis, University of Edinburgh, 2011. http://hdl.handle.net/1842/5941.

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Enterohaemorrhagic Escherichia coli (EHEC) strains are associated with gastrointestinal and severe systemic disease in humans. EHEC O157:H7 is the most common serotype causing human infections in North America and the UK. Human infections mainly originate from cattle, through either direct contact with infected animals or indirectly through contamination of food or water with animal faeces. From the sequencing of EHEC O157 strains, it is clear that the genomes contain multiple prophages, many of them cryptic, which define this E. coli pathotype. These regions include the locus of enterocyte ef
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32

Röhrich-Dönitz, Anelia Dorothea. "Regulation of type III secretion hierarchy in Shigella flexneri." Thesis, University of Bristol, 2013. http://ethos.bl.uk/OrderDetails.do?uin=uk.bl.ethos.633196.

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Type III secretion systems (T3SS) are protein injection devices used by Gram-negative bacteria to manipulate eukaryotic cells. In Shigella, the T3SS is assembled when the environmental conditions are appropriate for invasion. However, secretion is only activated when physical contact of the injection needle with the host cell generates an activation signal. The signal is transmitted to the cytoplasm where it triggers secretion. First, translocators are secreted which form a pore in the host cell membrane. Second, effector proteins are translocated into the host cell. The activation process is
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33

Gunasena, Deepthi Kaushalya. "Type III secretion systems in bacterial pathogens of fish." Thesis, University of Reading, 2005. http://ethos.bl.uk/OrderDetails.do?uin=uk.bl.ethos.413881.

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34

Thomas, Joanne. "Functional analysis of flagellum-specific type III export chaperones." Thesis, University of Cambridge, 2004. http://ethos.bl.uk/OrderDetails.do?uin=uk.bl.ethos.615655.

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35

Ahmed, Essa Ismaeil. "Type III Deep Eutectic Solvents (DESS) as base lubricants." Thesis, University of Leicester, 2015. http://hdl.handle.net/2381/33465.

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Mineral oils are the most commonly used fraction of formulated lubricating oils except for some specialised applications where synthetic oils are employed. An alternative approach has been suggested using ionic liquids (ILs) due to their high viscosity index and high thermal stability. The aim of this study is to examine the use a different type of ionic liquids in the form of Deep Eutectic Solvents (DESs) as base lubricants as these have significantly improved environmental credentials. The first stage of the study involved the investigation of thermo-physical properties such as heat capacity
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36

Wang, Zhibo. "Functional Characterization of Four Xanthomonas euvesicatoria Type III Effectors." Diss., Virginia Tech, 2020. http://hdl.handle.net/10919/104984.

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Pepper and tomato, as two common, popular, and important vegetables grown worldwide, provide human beings with high quality fruit of flavor and aroma, and a high concentration of vitamins and antioxidants. Pepper and tomato production is frequently affected by various pathogens, including nematodes, fungi, and bacteria. Among those phytopathogens, Xanthomonas euvesicatoria (Xe) causes a severe bacterial spot (BS) disease on pepper and tomato. The BS disease could cause a loss of approximately 10% of the total crop yield in the world. Breeding tomato and pepper cultivars with improved BS diseas
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37

Bronstein, Philip Alan. "Identification and characterization of a type III chaperone, InvB /." Thesis, Connect to this title online; UW restricted, 2001. http://hdl.handle.net/1773/11524.

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38

Steed, Robert John. "Saturation of Intersubband Transitions in p-type and N-type III-V Qua'ntum Wells." Thesis, Imperial College London, 2007. http://ethos.bl.uk/OrderDetails.do?uin=uk.bl.ethos.487524.

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Intersubband transitions (ISBTs) in quantum wells have had extensive prior research but remain interesting. This thesis continues the use of saturation experiments for the characterisation of ISBT relaxation times ie. the time taken for carriers to scatter between a quantum well's subbands. Saturation experiments were carried out using an optical parametric generator as a tunable source of mid-infrared laser light (6.4 --t 8.6 pm) and intensities of -200MWcm-2 were reached. For this thesis, two p-doped samples were saturated, each sample containing multiple GaAs/AIGaAs quantum wells. Both samp
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39

Lin, Herbert Y. (Herbert Yih-Fuu). "Expression cloning and characterization of the type II and type III TGF-β receptors". Thesis, Massachusetts Institute of Technology, 1993. http://hdl.handle.net/1721.1/12507.

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40

Mandaliev, Peter Nikolov. "Mechanisms of Nd(III) and Eu(III) uptake by cementitious materials /." [S.l.] : [s.n.], 2008. http://e-collection.ethbib.ethz.ch/show?type=diss&nr=18095.

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41

Podrebarac, James. "Development of Recombinant Human Collagen Type I and Type III Injectable Hydrogels for Cardiac Therapy." Thesis, Université d'Ottawa / University of Ottawa, 2017. http://hdl.handle.net/10393/36038.

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Functional biomaterials are being developed as scaffolds to support endogenous cells and to promote the regeneration of ischemic tissue. The aim for this study was to develop a new translational platform for injectable hydrogels using recombinant human collagen (rHC) of two types: type I (TI) and type III (TIII). The collagen solutions were characterized to ensure batch-to-batch consistency and protein integrity. The hydrogel preparation protocol was extensively monitored to ensure ease of use and high-quality production. Post-gelation, rHC TIII have a higher viscosity compared to rHC TI, yet
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42

Dendy, Shauneen Marguerite. "Cholesterol synthesis in type III hyperlipoproteinemic and non-hyperlipidemic individuals." Thesis, University of British Columbia, 1990. http://hdl.handle.net/2429/28979.

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The purpose of this study was to investigate whether increased endogenous cholesterol synthesis contributes to the elevated plasma cholesterol levels observed in type III hyperlipoproteinemia (type III HLP). Eight apolipoprotein (apo) E2 subjects with type III HLP and 8 apo E2 non-hyperlipidemic control subjects (controls) were given a priming bolus dose of deuterium oxide (D₂O) (0.7 g D₂O/kg body H2O). Daily M1 (central) pool free cholesterol fractional synthetic rate (FSR) was calculated as the incorporation rate of deuterium from body water into plasma free cholesterol. Blood samples were c
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43

Rodgers, Loren E. "Recognition of effectors by the bacterial type III secretion system." Diss., Connect to a 24 p. preview or request complete full text in PDF format. Access restricted to UC campuses, 2008. http://wwwlib.umi.com/cr/ucsd/fullcit?p3307124.

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Thesis (Ph. D.)--University of California, San Diego, 2008.<br>Title from first page of PDF file (viewed July 2, 2008). Available via ProQuest Digital Dissertations. Vita. Includes bibliographical references.
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Robin, Ekman. "The GHP formalism, with applications to Petrov type III spacetimes." Thesis, Umeå universitet, Institutionen för fysik, 2014. http://urn.kb.se/resolve?urn=urn:nbn:se:umu:diva-88352.

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We give a review of the construction and application of spinor fields in general relativity and an account of the spinor-based Geroch-Held-Penrose (GHP) formalism. Specifically, we discuss using the GHP formalism to integrate Einstein's equations as suggested by Held  and developed by Edgar and Ludwig and discuss the similaritites with the Cartan-Karlhede classification of spacetimes. We use this integration method to find a one-parameter subclass and a degenerate case, for which the Cartan-Karlhede algorithm terminates at second order, of the Petrov type III, vacuum Robinson-Trautman metrics.
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45

Mitsopoulos, Konstantinos. "The assembly of type III membrane proteins in Escherichia coli." Thesis, University of Sussex, 2000. http://ethos.bl.uk/OrderDetails.do?uin=uk.bl.ethos.310262.

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46

Elliott, Jayne Louise. "Properties and interactions of type III intermediate filaments with CRYAB." Thesis, Durham University, 2013. http://etheses.dur.ac.uk/7017/.

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Glial fibrillary acidic protein is a type three intermediate filament found within astrocytes in the central nervous system and mutations have been found as the cause of Alexander disease, resulting in protein aggregates of Rosenthal fibers with αBcrystallin. Here two glial fibrillary acidic protein mutants of R79C and R239H, located in the LNDR and rod 2A domain respectively, were assembled in vitro and their morphology, assembly competence and interactions with αB-crystallin were assessed. Both mutants were unable to form usual filament lengths but instead were similar to unit length filamen
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47

Zhang, Weiqiang. "Synthesis of novel chiral pyrrolidine-type (salen)Mn(III) complexes." Thesis, Swansea University, 2006. https://cronfa.swan.ac.uk/Record/cronfa42403.

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The thesis reports the total syntheses of new chiral pyrrolidine-type salen ligands 5.4 and their corresponding Mn(III) complexes 5.5. The salen ligands were synthesized by condensation of tras-(3R,4R)-diaminopyrrolidine (3.12) or trans-(3R,4R)-1-benzyl-3,4-diaminopyrrolidine (3.10) with two equivalents of (R)-3-formyl-2-hydroxy-2'-phenyl-1,1'-binaphthalene [(R)-4.9]. The salen ligands were transformed to their corresponding Mn(III) complexes following a general procedure. The catalytic performances of the synthesized (salen)Mn(III) complexes in asymmetric epoxidation of 1,2-dihydronaphthalene
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Wu, Shuchi. "Structural and functional characterization of a Xanthomonas Type III effector." Diss., Virginia Tech, 2015. http://hdl.handle.net/10919/73219.

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Rice bacterial leaf streak disease caused by Xanthomonas oryzae pv. oryzicola (Xoc) is one of the most important rice bacterial diseases. Xanthomonas type III effector gene avrRxo1 is conserved in diverse Xoc strains and its homologues have been identified from several other gram-negative bacteria species such as Burkholderia and Acidovorax. In this research, we studied the protein structure of AvrRxo1 and illustrated its virulence mechanism.We determined the three-dimensional structure of the complex of AvrRxo1 and its cognate chaperone Arc1 (AvrRxo1 required chaperone 1). The AvrRxo1: Arc1
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Legrand, Isabelle. "Glycogenose de type iii : a propos de deux nouveaux cas." Université Louis Pasteur (Strasbourg) (1971-2008), 1986. http://www.theses.fr/1986STR1M169.

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Shan, Libo. "Characterization of type III effectors of Pseudomonas syringae pv. tomato /." Search for this dissertation online, 2003. http://wwwlib.umi.com/cr/ksu/main.

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