Bibliografías temáticas / Leukotrienes

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Literatura académica sobre el tema "Leukotrienes"

Autor: Grafiati
Publicado: 4 de junio de 2021
Última modificación: 25 de julio de 2025

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Artículos de revistas sobre el tema "Leukotrienes"

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Uotila, P., K. Punnonen, R. Tammivaara, and CT Jansén. "Detection of leukotrienes in the serum of asthmatic and psoriatic patients." Acta Dermato-Venereologica 66, no. 5 (1986): 381–85. http://dx.doi.org/10.2340/0001555566381385.

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Purified serum samples from asthmatic and psoriatic patients and healthy controls were analysed by high-pressure liquid chromatography (HPLC) and the amounts of leukotrienes were measured from the corresponding HPLC fractions by specific radioimmunoassays. In the serum of healthy controls the amounts of leukotrienes B4, C4 and D4 were very small or negligible. Rather great amount of leukotriene B4 was, however, detected in the serum of many asthmatic and psoriatic patients. The amount of leukotriene B4 was in the serum of asthmatic patients 120 +/- 20 pmol/ml (n = 11, mean +/- SEM) and in that
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Paterson, N. A. "Influence of hypoxia on histamine and leukotriene release from dispersed porcine lung cells." Journal of Applied Physiology 61, no. 5 (1986): 1790–95. http://dx.doi.org/10.1152/jappl.1986.61.5.1790.

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The ability of hypoxia (PO2 57 Torr) and anoxia (PO2 0 Torr) to induce the release of histamine or sulfidopeptide leukotrienes from dispersed porcine parenchymal lung cells was examined. Spontaneous release of histamine (9.2 +/- 1.3%) was not significantly increased during hypoxia or anoxia, and spontaneous leukotriene release was not detected under any conditions. The release of leukotriene induced by A23187 (78 +/- 11 pmol leukotriene D4 equivalent/10(7) parenchymal lung cells) was unchanged during hypoxia and was significantly reduced (55.4 +/- 7.7% control leukotriene release) during anoxi
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Petersen, Bodil, K. Frank Austen, Kenneth D. Bloch, et al. "Cysteinyl Leukotrienes Impair Hypoxic Pulmonary Vasoconstriction in Endotoxemic Mice." Anesthesiology 115, no. 4 (2011): 804–11. http://dx.doi.org/10.1097/aln.0b013e31822e94bd.

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Background Sepsis impairs hypoxic pulmonary vasoconstriction (HPV) in patients and animal models, contributing to systemic hypoxemia. Concentrations of cysteinyl leukotrienes are increased in the bronchoalveolar lavage fluid of patients with sepsis, but the contribution of cysteinyl leukotrienes to the impairment of HPV is unknown. Methods Wild-type mice, mice deficient in leukotriene C(4) synthase, the enzyme responsible for cysteinyl leukotriene synthesis, and mice deficient in cysteinyl leukotriene receptor 1 were studied 18 h after challenge with either saline or endotoxin. HPV was measure
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Jackson, W. F. "Regional differences in mechanism of action of oxygen on hamster arterioles." American Journal of Physiology-Heart and Circulatory Physiology 265, no. 2 (1993): H599—H603. http://dx.doi.org/10.1152/ajpheart.1993.265.2.h599.

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Leukotrienes have been implicated in the arteriolar constriction induced by elevated PO2 in the hamster cheek pouch. The role of leukotrienes in arteriolar O2 reactivity in other tissues has not been studied. To test the hypothesis that leukotrienes mediate O2 reactivity in all tissues, the effects of a leukotriene receptor antagonist, SKF-102922 (10 microM), a 5-lipoxygenase inhibitor, SC-43251 (30 microM), and a 5-lipoxygenase-activating protein antagonist, MK-886 (10 microM), on arteriolar O2 reactivity in hamster cheek pouch were compared with their effects on cremasteric arteriolar O2 rea
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Denzlinger, C., A. Kapp, M. Grimberg, HH Gerhartz, and W. Wilmanns. "Enhanced endogenous leukotriene biosynthesis in patients treated with granulocyte-macrophage colony-stimulating factor." Blood 76, no. 9 (1990): 1765–70. http://dx.doi.org/10.1182/blood.v76.9.1765.1765.

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Abstract The hematopoietic cytokine granulocyte-macrophage colony-stimulating factor (GM-CSF) is being used in clinical trials for its potential in the treatment of hematopoietic insufficiency due to various causes. Involvement of leukotrienes in the effects of GM-CSF is suggested by analytical and pharmacologic evidence obtained in vitro. However, until now no data in support of a role of leukotrienes in GM-CSF action in vivo have been presented. In the present investigation this question was approached by measurement of endogenous cysteinyl leukotriene formation in patients treated with the
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Denzlinger, C., A. Kapp, M. Grimberg, HH Gerhartz, and W. Wilmanns. "Enhanced endogenous leukotriene biosynthesis in patients treated with granulocyte-macrophage colony-stimulating factor." Blood 76, no. 9 (1990): 1765–70. http://dx.doi.org/10.1182/blood.v76.9.1765.bloodjournal7691765.

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The hematopoietic cytokine granulocyte-macrophage colony-stimulating factor (GM-CSF) is being used in clinical trials for its potential in the treatment of hematopoietic insufficiency due to various causes. Involvement of leukotrienes in the effects of GM-CSF is suggested by analytical and pharmacologic evidence obtained in vitro. However, until now no data in support of a role of leukotrienes in GM-CSF action in vivo have been presented. In the present investigation this question was approached by measurement of endogenous cysteinyl leukotriene formation in patients treated with the cytokine
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Jones, T. R., Y. Guindon, R. Young та ін. "L-648,051, sodium 4-[3-(4-acetyl-3-hydroxy-2-propylphenoxy)-propylsulfonyl]-γ-oxo-benzenebutanoate: a leukotriene D4 receptor antagonist". Canadian Journal of Physiology and Pharmacology 64, № 12 (1986): 1535–42. http://dx.doi.org/10.1139/y86-258.

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L-648,051, sodium 4-[3-(4-acetyl-3-hydroxy-2-propylphenoxy)propylsulfonyl]-γ-oxo-benzenebutanoate is a selective and competitive inhibitor of [3H]leukotriene D4 (KB value of 4.0 μM) and to a lesser extent [3H]leukotriene C4 (Ki value of 36.7 μM) binding in guinea pig lung homogenates. Functionally, it selectively antagonized contractions of guinea pig trachea induced by leukotrienes C4, D4, E4, and F4 in concentrations that did not antagonize contractions induced by acetylcholine, histamine, serotonin, prostaglandin F2α, or U-44069 (endoperoxide analogue). Schild plot analysis indicated that L
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Mancuso, Peter, Theodore J. Standiford, Teresa Marshall, and Marc Peters-Golden. "5-Lipoxygenase Reaction Products Modulate Alveolar Macrophage Phagocytosis of Klebsiella pneumoniae." Infection and Immunity 66, no. 11 (1998): 5140–46. http://dx.doi.org/10.1128/iai.66.11.5140-5146.1998.

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ABSTRACT The leukotrienes are potent lipid mediators of inflammation formed by the 5-lipoxygenase-catalyzed oxidation of arachidonic acid. Although the effects of leukotrienes on neutrophil chemotaxis and activation have been established, their role in modulating innate host defense mechanisms is poorly understood. In a previous study (M. Bailie, T. Standiford, L. Laichalk, M. Coffey, R. Strieter, and M. Peters-Golden, J. Immunol. 157:5221–5224, 1996), we used 5-lipoxygenase knockout mice to establish a critical role for endogenous leukotrienes in pulmonary clearance and alveolar macrophage ph
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Meshram, Deepak, Khushbo Bhardwaj, Charulata Rathod, Gail B. Mahady, and Kapil K. Soni. "The Role of Leukotrienes Inhibitors in the Management of Chronic Inflammatory Diseases." Recent Patents on Inflammation & Allergy Drug Discovery 14, no. 1 (2020): 15–31. http://dx.doi.org/10.2174/1872213x14666200130095040.

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Background: Leukotrienes are powerful mediators of inflammation and interact with specific receptors in target cell membrane to initiate an inflammatory response. Thus, Leukotrienes (LTs) are considered to be potent mediators of inflammatory diseases including allergic rhinitis, inflammatory bowel disease and asthma. Leukotriene B4 and the series of cysteinyl leukotrienes (C4, D4, and E4) are metabolites of arachidonic acid metabolism that cause inflammation. The cysteinyl LTs are known to increase vascular permeability, bronco-constriction and mucus secretion. Objectives: To review the publis
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Steinhilber, D. "5-Lipoxygenase: A Target for Antiinflammatory Drugs Revisited." Current Medicinal Chemistry 6, no. 1 (1999): 71–85. http://dx.doi.org/10.2174/0929867306666220207211259.

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Abstract: Arachidonate 5-lipoxygenase is the key enzyme in leukotriene biosynthesis and catalyzes the initial steps in the conversion of arachidonic acid to biologically active leukotrienes. Leukotrienes are considered as potent potent mediators of inflammatory and allergic reactions which are locally released by leukocytes and other 5-LO expressing cells and exert their effects via binding to specific membrane receptors and, as suggested recently, the nuclear receptor PPARa. Because of the proinflammatory profile of leukotrienes it was assumed that leukotriene biosynthesis inhibitors and leuk
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Tesis sobre el tema "Leukotrienes"

1

Qiu, Hong. "Leukotrienes and leukotriene receptors : potential roles in cardiovascular diseases /." Stockholm, 2007. http://diss.kib.ki.se/2007/978-91-7357-056-5/.

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Crawley, John Edward. "Leukotrienes." Thesis, University of Nottingham, 1991. http://ethos.bl.uk/OrderDetails.do?uin=uk.bl.ethos.334790.

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Gyllfors, Pär. "Clinical studies of asthma phenotypes focusing on the role of the leukotrienes /." Stockholm, 2006. http://diss.kib.ki.se/2006/91-7357-001-X/.

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Bäck, Magnus. "Studies of receptors and modulatory mechanisms in functional responses to cysteinyl-leukotrienes in smooth muscle /." Stockholm, 2001. http://diss.kib.ki.se/2001/91-628-4846-1/.

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Manro, A. "Synthesis of compounds related to leukotrienes." Thesis, University of Salford, 1987. http://ethos.bl.uk/OrderDetails.do?uin=uk.bl.ethos.381849.

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McColl, Shaun Reuss. "Factors affecting the regulation of leukotriene production by neutrophils." Title page, contents and summary only, 1987. http://web4.library.adelaide.edu.au/theses/09PH/09phm1288.pdf.

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Caroppo, V. "EFFECTS OF CYSTEINYL LEUKOTRIENES ON PLATELET ACTIVATION." Doctoral thesis, Università degli Studi di Milano, 2016. http://hdl.handle.net/2434/363389.

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Platelet are key players in haemostasis and represent a pivotal link between inflammation, immunity and atherogenesis. Cysteinyl leukotrienes (CysLTs) such as LTC4, LTD4, and LTE4 are potent lipid inflammatory mediators which interact with G protein-coupled receptors, CysLT1R, CysLT2R . However, LTE4, the most stable cystenyl leukotriene, is not putative substrate for these two receptor. Recently have been hypothesized that a third cystenyl leukotreine receptor exists, and a computer modeling suggests P2Y12, receptor for ADP present on platelet surface, as the putative receptor. But GPR99 is
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Butler, Paul Ian. "The synthesis and evaluation of potential inhibitors of 5-lipoxygenase." Thesis, University of Reading, 1993. http://ethos.bl.uk/OrderDetails.do?uin=uk.bl.ethos.357783.

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Sousa, Paula T. de. "The synthesis of novel thiophene analogues of leukotrienes." Thesis, University of East Anglia, 1992. http://ethos.bl.uk/OrderDetails.do?uin=uk.bl.ethos.317613.

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Galton, S. A. "Production of leukotrienes by the porcine pulmonary artery." Thesis, University College London (University of London), 1987. http://ethos.bl.uk/OrderDetails.do?uin=uk.bl.ethos.376096.

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Libros sobre el tema "Leukotrienes"

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Roberto, Levi, Krell Robert D, New York Academy of Sciences., and Conference on the Biology of the Leukotrienes (1987 : Philadelphia, Pa.), eds. Biology of the leukotrienes. New York Academy of Sciences, 1988.

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Rodger, Ian, Jack Botting, and Sven-Erik Dahlén, eds. Leukotrienes. Springer Netherlands, 1998. http://dx.doi.org/10.1007/978-94-011-4880-1.

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E, Pike John, and Morton Douglas R, eds. Chemistry of the prostaglandins and leukotrienes. Raven Press, 1985.

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Uriel, Zor, Naor Zvi, Kohen Fortüne, and International Conference on: Leukotrienes and Prostanoids in Health and Disease (1985 : Tel Aviv, Israel and Rehovot, Israel), eds. Leukotrienes and prostanoids in health and disease. Raven Press, 1986.

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Martyn, Bailey J., and International Spring Symposium on Health Sciences (4th : 1984 : Washington, D.C.), eds. Prostaglandins, leukotrienes, and lipoxins: Biochemistry, mechanism of action, and clinical applications. Plenum Press, 1985.

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Bailey, J. Martyn, ed. Prostaglandins, Leukotrienes, and Lipoxins. Springer US, 1985. http://dx.doi.org/10.1007/978-1-4684-4946-4.

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Folco, Giancarlo, Bengt Samuelsson, and Robert C. Murphy, eds. Novel Inhibitors of Leukotrienes. Birkhäuser Basel, 1999. http://dx.doi.org/10.1007/978-3-0348-8703-8.

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Giancarlo, Folco, Murphy Robert C, and Samuelsson Bengt, eds. Novel inhibitors of leukotrienes. Birkhäuser Verlag, 1999.

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Giancarlo, Folco, Samuelsson Bengt, and Murphy Robert C, eds. Novel inhibitors of leukotrienes. Birkhäuser Verlag, 1999.

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Bailey, J. Martyn, ed. Prostaglandins, Leukotrienes, Lipoxins, and PAF. Springer US, 1991. http://dx.doi.org/10.1007/978-1-4899-0727-1.

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Capítulos de libros sobre el tema "Leukotrienes"

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Mayatepek, Ertan. "Leukotrienes." In Physician's Guide to the Diagnosis, Treatment, and Follow-Up of Inherited Metabolic Diseases. Springer Berlin Heidelberg, 2014. http://dx.doi.org/10.1007/978-3-642-40337-8_38.

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Hay, Douglas W. P., and David Raeburn. "Leukotrienes." In Airways Smooth Muscle: Neurotransmitters, Amines, Lipid Mediators and Signal Transduction. Birkhäuser Basel, 1995. http://dx.doi.org/10.1007/978-3-0348-7504-2_6.

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Okunishi, Katsuhide, and Marc Peters-Golden. "Leukotrienes." In Inflammation and Allergy Drug Design. Wiley-Blackwell, 2011. http://dx.doi.org/10.1002/9781444346688.ch23.

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Sjölander, Anita. "Leukotrienes." In Encyclopedia of Cancer. Springer Berlin Heidelberg, 2015. http://dx.doi.org/10.1007/978-3-642-27841-9_3334-2.

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Bäck, Magnus. "Leukotrienes." In Compendium of Inflammatory Diseases. Springer Basel, 2016. http://dx.doi.org/10.1007/978-3-7643-8550-7_105.

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Bach, Michael K. "Leukotrienes." In The Reticuloendothelial System. Springer US, 1986. http://dx.doi.org/10.1007/978-1-4684-5158-0_11.

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Sjölander, Anita. "Leukotrienes." In Encyclopedia of Cancer. Springer Berlin Heidelberg, 2017. http://dx.doi.org/10.1007/978-3-662-46875-3_3334.

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Sjölander, Anita. "Leukotrienes." In Encyclopedia of Cancer. Springer Berlin Heidelberg, 2011. http://dx.doi.org/10.1007/978-3-642-16483-5_3334.

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Gooch, Jan W. "Leukotrienes." In Encyclopedic Dictionary of Polymers. Springer New York, 2011. http://dx.doi.org/10.1007/978-1-4419-6247-8_14113.

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Nakamura, Motonao, and Takehiko Yokomizo. "Leukotrienes." In Encyclopedia of Molecular Pharmacology. Springer International Publishing, 2020. http://dx.doi.org/10.1007/978-3-030-21573-6_87-1.

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Actas de conferencias sobre el tema "Leukotrienes"

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Eap, R., E. Jacques, S. Plante, and J. Chakir. "Cysteinyl Leukotrienes Increase Collagen Production in Bronchial Fibroblasts of Asthmatic Subjects Via Cysteinyl Leukotriene Receptor 2." In American Thoracic Society 2009 International Conference, May 15-20, 2009 • San Diego, California. American Thoracic Society, 2009. http://dx.doi.org/10.1164/ajrccm-conference.2009.179.1_meetingabstracts.a5060.

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Jaksch-Bogensperger, H., A. Koller, Th Fischer, et al. "The role of Cysteinyl-Leukotrienes in preeclamptic patients." In Kongressabstracts zur Gemeinsamen Jahrestagung der Österreichischen Gesellschaft für Gynäkologie und Geburtshilfe (OEGGG) und der Bayerischen Gesellschaft für Geburtshilfe und Frauenheilkunde e.V. (BGGF). Georg Thieme Verlag KG, 2021. http://dx.doi.org/10.1055/s-0041-1730483.

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Ptaszynski, S., V. Arsenault, A. Al-Rabea, and C. Bergeron. "Leukotrienes and Airway Epithelial-Mesenchymal Transition – Implications in Bronchiolitis Obliterans." In American Thoracic Society 2009 International Conference, May 15-20, 2009 • San Diego, California. American Thoracic Society, 2009. http://dx.doi.org/10.1164/ajrccm-conference.2009.179.1_meetingabstracts.a5634.

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Kaditis, A., K. Chaidas, E. Alexopoulos, et al. "Urine Concentrations of Cysteinyl Leukotrienes in Children with Obstructive Sleep-Disordered Breathing." In American Thoracic Society 2009 International Conference, May 15-20, 2009 • San Diego, California. American Thoracic Society, 2009. http://dx.doi.org/10.1164/ajrccm-conference.2009.179.1_meetingabstracts.a6342.

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Matsuyama, T., K. Machida, H. Matsuyama, Y. Dotake, K. Takagi, and H. Inoue. "The Effect of Long-acting Muscarinic Antagonist on Airway Inflammation Through Leukotrienes." In American Thoracic Society 2023 International Conference, May 19-24, 2023 - Washington, DC. American Thoracic Society, 2023. http://dx.doi.org/10.1164/ajrccm-conference.2023.207.1_meetingabstracts.a1300.

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Parker, D., D. A. Flores, J. K. Bentley, and M. B. Hershenson. "Cysteinyl Leukotrienes Are Required for Rhinovirus-induced Mucous Metaplasia in Immature Mice." In American Thoracic Society 2024 International Conference, May 17-22, 2024 - San Diego, CA. American Thoracic Society, 2024. http://dx.doi.org/10.1164/ajrccm-conference.2024.209.1_meetingabstracts.a1383.

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Crimi, C., S. Sichili, A. Salamone, et al. "Exhaled Breath Condensate (EBC) Cysteinyl Leukotrienes (cys-LTs) in Gastroesophageal Reflux Disease-Related Asthma." In American Thoracic Society 2009 International Conference, May 15-20, 2009 • San Diego, California. American Thoracic Society, 2009. http://dx.doi.org/10.1164/ajrccm-conference.2009.179.1_meetingabstracts.a5703.

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Krohne, Sheryl G. "Use of the KOWA FC-1000 to Measure Aqueous Humor Protein and Cells in the Dog." In Noninvasive Assessment of the Visual System. Optica Publishing Group, 1991. http://dx.doi.org/10.1364/navs.1991.md3.

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Breakdown of the blood-aqueous barrier (BAB) resulting in protein and cells in the aqueous humor of humans and many animals has been documented.1 This breakdown occurs during uveitis, and following surgery and paracentesis. BAB breakdown occurs following these initiating insults because endothelial cells contract in the ciliary body, causing protein leakage from the vascular system which results Jn dysjunction of the epithelial layers, further vascular damage and protein leakage.2,3 These changes are mediated by prostaglandins, leukotrienes, free radicals, neurostimulators (such as in the anti
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Altraja, S., J. Jaama, and A. Altraja. "Cysteinyl Leukotrienes Cause Down-Regulation of Glucocorticoid Receptor in Human Bronchial Epithelial Cells Via CysLT1Receptor." In American Thoracic Society 2009 International Conference, May 15-20, 2009 • San Diego, California. American Thoracic Society, 2009. http://dx.doi.org/10.1164/ajrccm-conference.2009.179.1_meetingabstracts.a1403.

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Bikov, András, Orsolya Gall, Zoltan Sutto, et al. "Bronchoalveolar lavage fluid leukotrienes in the pathomechanism of acute inflammatory events in lung transplant recipients." In ERS International Congress 2016 abstracts. European Respiratory Society, 2016. http://dx.doi.org/10.1183/13993003.congress-2016.pa4148.

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Informes sobre el tema "Leukotrienes"

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Muza, S. R., D. Kaminsky, C. S. Fulco, L. E. Banderet, and A. Cymerman. Cysteinyl Leukotriene Blockade Does Not Prevent Acute Mountain Sickness. Defense Technical Information Center, 2004. http://dx.doi.org/10.21236/ada423394.

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Goldman, Gideon, Richard Welbourn, C. R. aleri, David Shepro, and Herbert B. Hechtman. Leukotriene B4 and Thromboxane A2 are Essential Cofactors in CD 18 Dependent Neutrophil Diapedesis. Defense Technical Information Center, 1990. http://dx.doi.org/10.21236/ada360180.

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