Literatura académica sobre el tema "Lymphangion"

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Artículos de revistas sobre el tema "Lymphangion"

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Venugopal, Arun M., Randolph H. Stewart, Glen A. Laine, Ranjeet M. Dongaonkar y Christopher M. Quick. "Lymphangion coordination minimally affects mean flow in lymphatic vessels". American Journal of Physiology-Heart and Circulatory Physiology 293, n.º 2 (agosto de 2007): H1183—H1189. http://dx.doi.org/10.1152/ajpheart.01340.2006.

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The lymphatic system returns interstitial fluid to the central venous circulation, in part, by the cyclical contraction of a series of “lymphangion pumps” in a lymphatic vessel. The dynamics of individual lymphangions have been well characterized in vitro; their frequencies and strengths of contraction are sensitive to both preload and afterload. However, lymphangion interaction within a lymphatic vessel has been poorly characterized because it is difficult to experimentally alter properties of individual lymphangions and because the afterload of one lymphangion is coupled to the preload of another. To determine the effects of lymphangion interaction on lymph flow, we adapted an existing mathematical model of a lymphangion (characterizing lymphangion contractility, lymph viscosity, and inertia) to create a new lymphatic vessel model consisting of several lymphangions in series. The lymphatic vessel model was validated with focused experiments on bovine mesenteric lymphatic vessels in vitro. The model was then used to predict changes in lymph flow with different time delays between onset of contraction of adjacent lymphangions (coordinated case) and with different relative lymphangion contraction frequencies (noncoordinated case). Coordination of contraction had little impact on mean flow. Furthermore, orthograde and retrograde propagations of contractile waves had similar effects on flow. Model results explain why neither retrograde propagation of contractile waves nor the lack of electrical continuity between lymphangions adversely impacts flow. Because lymphangion coordination minimally affects mean flow in lymphatic vessels, lymphangions have flexibility to independently adapt to local conditions.
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Quick, Christopher M., Arun M. Venugopal, Ranjeet M. Dongaonkar, Glen A. Laine y Randolph H. Stewart. "First-order approximation for the pressure-flow relationship of spontaneously contracting lymphangions". American Journal of Physiology-Heart and Circulatory Physiology 294, n.º 5 (mayo de 2008): H2144—H2149. http://dx.doi.org/10.1152/ajpheart.00781.2007.

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To return lymph to the great veins of the neck, it must be actively pumped against a pressure gradient. Mean lymph flow in a portion of a lymphatic network has been characterized by an empirical relationship (Pin − Pout = −Pp + RLQL), where Pin − Pout is the axial pressure gradient and QL is mean lymph flow. RL and Pp are empirical parameters characterizing the effective lymphatic resistance and pump pressure, respectively. The relation of these global empirical parameters to the properties of lymphangions, the segments of a lymphatic vessel bounded by valves, has been problematic. Lymphangions have a structure like blood vessels but cyclically contract like cardiac ventricles; they are characterized by a contraction frequency ( f) and the slopes of the end-diastolic pressure-volume relationship [minimum value of resulting elastance ( Emin)] and end-systolic pressure-volume relationship [maximum value of resulting elastance ( Emax)]. Poiseuille's law provides a first-order approximation relating the pressure-flow relationship to the fundamental properties of a blood vessel. No analogous formula exists for a pumping lymphangion. We therefore derived an algebraic formula predicting lymphangion flow from fundamental physical principles and known lymphangion properties. Quantitative analysis revealed that lymph inertia and resistance to lymph flow are negligible and that lymphangions act like a series of interconnected ventricles. For a single lymphangion, Pp = Pin ( Emax − Emin)/ Emin and RL = Emax/ f. The formula was tested against a validated, realistic mathematical model of a lymphangion and found to be accurate. Predicted flows were within the range of flows measured in vitro. The present work therefore provides a general solution that makes it possible to relate fundamental lymphangion properties to lymphatic system function.
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Bertram, Christopher D., Charlie Macaskill, Michael J. Davis y James E. Moore. "Consequences of intravascular lymphatic valve properties: a study of contraction timing in a multi-lymphangion model". American Journal of Physiology-Heart and Circulatory Physiology 310, n.º 7 (1 de abril de 2016): H847—H860. http://dx.doi.org/10.1152/ajpheart.00669.2015.

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The observed properties of valves in collecting lymphatic vessels include transmural pressure-dependent bias to the open state and hysteresis. The bias may reduce resistance to flow when the vessel is functioning as a conduit. However, lymphatic pumping implies a streamwise increase in mean pressure across each valve, suggesting that the bias is then potentially unhelpful. Lymph pumping by a model of several collecting lymphatic vessel segments (lymphangions) in series, which incorporated these properties, was investigated under conditions of adverse pressure difference while varying the refractory period between active muscular contractions and the inter-lymphangion contraction delay. It was found that many combinations of the timing parameters and the adverse pressure difference led to one or more intermediate valves remaining open instead of switching between open and closed states during repetitive contraction cycles. Cyclic valve switching was reliably indicated if the mean pressure in a lymphangion over a cycle was higher than that in the lymphangion upstream, but either lack of or very brief valve closure could cause mean pressure to be lower downstream. Widely separated combinations of refractory period and delay time were found to produce the greatest flow-rate for a given pressure difference. The efficiency of pumping was always maximized by a long refractory period and lymphangion contraction starting when the contraction of the lymphangion immediately upstream was peaking. By means of an ex vivo experiment, it was verified that intermediate valves in a chain of pumping lymphangions can remain open, while the lymphangions on either side of the open valve continue to execute contractions.
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Venugopal, Arun M., Christopher M. Quick, Glen A. Laine y Randolph H. Stewart. "Optimal postnodal lymphatic network structure that maximizes active propulsion of lymph". American Journal of Physiology-Heart and Circulatory Physiology 296, n.º 2 (febrero de 2009): H303—H309. http://dx.doi.org/10.1152/ajpheart.00360.2008.

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The lymphatic system acts to return lower-pressured interstitial fluid to the higher-pressured veins by a complex network of vessels spanning more than three orders of magnitude in size. Lymphatic vessels consist of lymphangions, segments of vessels between two unidirectional valves, which contain smooth muscle that cyclically pumps lymph against a pressure gradient. Whereas the principles governing the optimal structure of arterial networks have been identified by variations of Murray's law, the principles governing the optimal structure of the lymphatic system have yet to be elucidated, although lymph flow can be identified as a critical parameter. The reason for this deficiency can be identified. Until recently, there has been no algebraic formula, such as Poiseuille's law, that relates lymphangion structure to its function. We therefore employed a recently developed mathematical model, based on the time-varying elastance model conventionally used to describe ventricular function, that was validated by data collected from postnodal bovine mesenteric lymphangions. From this lymphangion model, we developed a model to determine the structure of a lymphatic network that optimizes lymph flow. The model predicted that there is a lymphangion length that optimizes lymph flow and that symmetrical networks optimize lymph flow when the lymphangions downstream of a bifurcation are 1.26 times the length of the lymphangions immediately upstream. Measured lymphangion lengths (1.14 ± 0.5 cm, n = 74) were consistent with the range of predicted optimal lengths (0.1–2.1 cm). This modeling approach was possible, because it allowed a structural parameter, such as length, to be treated as a variable.
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Venugopal, Arun M., Randolph H. Stewart, Glen A. Laine y Christopher M. Quick. "Nonlinear lymphangion pressure-volume relationship minimizes edema". American Journal of Physiology-Heart and Circulatory Physiology 299, n.º 3 (septiembre de 2010): H876—H882. http://dx.doi.org/10.1152/ajpheart.00239.2009.

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Lymphangions, the segments of lymphatic vessel between two valves, contract cyclically and actively pump, analogous to cardiac ventricles. Besides having a discernable systole and diastole, lymphangions have a relatively linear end-systolic pressure-volume relationship (with slope Emax) and a nonlinear end-diastolic pressure-volume relationship (with slope Emin). To counter increased microvascular filtration (causing increased lymphatic inlet pressure), lymphangions must respond to modest increases in transmural pressure by increasing pumping. To counter venous hypertension (causing increased lymphatic inlet and outlet pressures), lymphangions must respond to potentially large increases in transmural pressure by maintaining lymph flow. We therefore hypothesized that the nonlinear lymphangion pressure-volume relationship allows transition from a transmural pressure-dependent stroke volume to a transmural pressure-independent stroke volume as transmural pressure increases. To test this hypothesis, we applied a mathematical model based on the time-varying elastance concept typically applied to ventricles (the ratio of pressure to volume cycles periodically from a minimum, Emin, to a maximum, Emax). This model predicted that lymphangions increase stroke volume and stroke work with transmural pressure if Emin < Emax at low transmural pressures, but maintain stroke volume and stroke work if Emin= Emax at higher transmural pressures. Furthermore, at higher transmural pressures, stroke work is evenly distributed among a chain of lymphangions. Model predictions were tested by comparison to previously reported data. Model predictions were consistent with reported lymphangion properties and pressure-flow relationships of entire lymphatic systems. The nonlinear lymphangion pressure-volume relationship therefore minimizes edema resulting from both increased microvascular filtration and venous hypertension.
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Razavi, Mohammad S., Tyler S. Nelson, Zhanna Nepiyushchikh, Rudolph L. Gleason y J. Brandon Dixon. "The relationship between lymphangion chain length and maximum pressure generation established through in vivo imaging and computational modeling". American Journal of Physiology-Heart and Circulatory Physiology 313, n.º 6 (1 de diciembre de 2017): H1249—H1260. http://dx.doi.org/10.1152/ajpheart.00003.2017.

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The intrinsic contraction of collecting lymphatic vessels serves as a pumping system to propel lymph against hydrostatic pressure gradients as it returns interstitial fluid to the venous circulation. In the present study, we proposed and validated that the maximum opposing outflow pressure along a chain of lymphangions at which flow can be achieved increases with the length of chain. Using minimally invasive near-infrared imaging to measure the effective pumping pressure at various locations in the rat tail, we demonstrated increases in pumping pressure along the length of the tail. Computational simulations based on a microstructurally motivated model of a chain of lymphangions informed from biaxial testing of isolated vessels was used to provide insights into the pumping mechanisms responsible for the pressure increases observed in vivo. These models suggest that the number of lymphangions in the chain and smooth muscle cell force generation play a significant role in determining the maximum outflow pressure, whereas the frequency of contraction has no effect. In vivo administration of nitric oxide attenuated lymphatic contraction, subsequently lowering the effective pumping pressure. Computational simulations suggest that the reduction in contractile strength of smooth muscle cells in the presence of nitric oxide can account for the reductions in outflow pressure observed along the lymphangion chain in vivo. Thus, combining modeling with multiple measurements of lymphatic pumping pressure provides a method for approximating intrinsic lymphatic muscle activity noninvasively in vivo while also providing insights into factors that determine the extent that a lymphangion chain can transport fluid against an adverse pressure gradient. NEW & NOTEWORTHY Here, we report the first minimally invasive in vivo measurements of the relationship between lymphangion chain length and lymphatic pumping pressure. We also provide the first in vivo validation of lumped parameter models of lymphangion chains previously developed through data obtained from isolated vessel testing.
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Mozokhina, Anastasia y Rostislav Savinkov. "Mathematical Modelling of the Structure and Function of the Lymphatic System". Mathematics 8, n.º 9 (1 de septiembre de 2020): 1467. http://dx.doi.org/10.3390/math8091467.

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This paper presents current knowledge about the structure and function of the lymphatic system. Mathematical models of lymph flow in the single lymphangion, the series of lymphangions, the lymph nodes, and the whole lymphatic system are considered. The main results and further perspectives are discussed.
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Bubnova, N. A., R. P. Borisova y N. A. Kubyshkina. "Theory of active lymph transport: morphofunctional foundations and clinical aspects". Regional blood circulation and microcirculation 19, n.º 3 (7 de octubre de 2020): 80–89. http://dx.doi.org/10.24884/1682-6655-2020-19-3-80-89.

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Its given significance of lymphangion as a structural-functional unit in the new theory of structure and functions of the lymphatic system. The construction of lymphangion in pathogenesis of lymph edema is represented. Treatment and prophylaxis must be directed at all parts of the lymphatic system.
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Dongaonkar, R. M., T. L. Nguyen, C. M. Quick, C. L. Heaps, J. Hardy, G. A. Laine, E. Wilson y R. H. Stewart. "Mesenteric lymphatic vessels adapt to mesenteric venous hypertension by becoming weaker pumps". American Journal of Physiology-Regulatory, Integrative and Comparative Physiology 308, n.º 5 (1 de marzo de 2015): R391—R399. http://dx.doi.org/10.1152/ajpregu.00196.2014.

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Lymphangions, the segments of lymphatic vessels between two adjacent lymphatic valves, actively pump lymph. Acute changes in transmural pressure and lymph flow have profound effects on lymphatic pump function in vitro. Chronic changes in pressure and flow in vivo have also been reported to lead to significant changes in lymphangion function. Because changes in pressure and flow are both cause and effect of adaptive processes, characterizing adaptation requires a more fundamental analysis of lymphatic muscle properties. Therefore, the purpose of the present work was to use an intact lymphangion isovolumetric preparation to evaluate changes in mesenteric lymphatic muscle mechanical properties and the intracellular Ca2+ in response to sustained mesenteric venous hypertension. Bovine mesenteric veins were surgically occluded to create mesenteric venous hypertension. Postnodal mesenteric lymphatic vessels from mesenteric venous hypertension (MVH; n = 6) and sham surgery (Sham; n = 6) animals were isolated and evaluated 3 days after the surgery. Spontaneously contracting MVH vessels generated end-systolic active tension and end-diastolic active tension lower than the Sham vessels. Furthermore, steady-state active tension and intracellular Ca2+ concentration levels in response to KCl stimulation were also significantly lower in MVH vessels compared with those of the Sham vessels. There was no significant difference in passive tension in lymphatic vessels from the two groups. Taken together, these results suggest that following 3 days of mesenteric venous hypertension, postnodal mesenteric lymphatic vessels adapt to become weaker pumps with decreased cytosolic Ca2+ concentration.
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Davis, Michael J., Joshua P. Scallan, John H. Wolpers, Mariappan Muthuchamy, Anatoliy A. Gashev y David C. Zawieja. "Intrinsic increase in lymphangion muscle contractility in response to elevated afterload". American Journal of Physiology-Heart and Circulatory Physiology 303, n.º 7 (1 de octubre de 2012): H795—H808. http://dx.doi.org/10.1152/ajpheart.01097.2011.

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Collecting lymphatic vessels share functional and biochemical characteristics with cardiac muscle; thus, we hypothesized that the lymphatic vessel pump would exhibit behavior analogous to homeometric regulation of the cardiac pump in its adaptation to elevated afterload, i.e., an increase in contractility. Single lymphangions containing two valves were isolated from the rat mesenteric microcirculation, cannulated, and pressurized for in vitro study. Pressures at either end of the lymphangion [input pressure (Pin), preload; output pressure (Pout), afterload] were set by a servo controller. Intralymphangion pressure (PL) was measured using a servo-null micropipette while internal diameter and valve positions were monitored using video methods. The responses to step- and ramp-wise increases in Pout (at low, constant Pin) were determined. PL and diameter data recorded during single contraction cycles were used to generate pressure-volume (P-V) relationships for the subsequent analysis of lymphangion pump behavior. Ramp-wise Pout elevation led to progressive vessel constriction, a rise in end-systolic diameter, and an increase in contraction frequency. Step-wise Pout elevation produced initial vessel distention followed by time-dependent declines in end-systolic and end-diastolic diameters. Significantly, a 30% leftward shift in the end-systolic P-V relationship accompanied an 84% increase in dP/d t after a step increase in Pout, consistent with an increase in contractility. Calculations of stroke work from the P-V loop area revealed that robust pumps produced net positive work to expel fluid throughout the entire afterload range, whereas weaker pumps exhibited progressively more negative work as gradual afterload elevation led to pump failure. We conclude that lymphatic muscle adapts to output pressure elevation with an intrinsic increase in contractility and that this compensatory mechanism facilitates the maintenance of lymph pump output in the face of edemagenic and/or gravitational loads.
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Tesis sobre el tema "Lymphangion"

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Madabushi, Venugopal Arun. "A computational approach to study the effect of multiple lymphangion coordination on lymph flow". Thesis, Texas A&M University, 2004. http://hdl.handle.net/1969.1/2670.

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The lymphatic system acts to return fluid from the interstitial space back into the blood circulation. In normal conditions, lymphangions, the segment of lymphatic vessel in between valves, cyclically contract and can pump lymph from low pressure tissues to the higher-pressure veins of the neck. With edema, however, this pressure gradient can reverse, and the role of contraction is less clear. Like ventricles, lymphangions are sensitive to both preload and afterload. Unlike ventricles, lymphangions are arranged in series, so that the outlet pressure of one lymphangion becomes the inlet pressure of another. Anything that alters the relative timing and frequency of adjacent lymphangions alters both preload and afterload of each lymphangion and thus mean lymph flow. To explore the effect of timing and frequency of contraction on lymph flow, we developed a computational model of a lymphatic vessel with lymphangions described by the classic description of time-varying elastance. When pumping up a pressure gradient, as in normal conditions, or when pumping down a pressure gradient, as in some cases of edema, we found that flow was optimized when the lymphangions in the vessel were pumping with a very short time delay between their cycles, and the flow was reduced when the time delay between the contractions was reduced to zero. However, a difference in frequency between adjacent lymphangions alters instantaneous flow but does not affect mean flow. These results suggest an important role for the timing of the contraction in optimizing lymph flow. However, a difference in frequencies between adjacent lymphangions has little effect on altering lymph flow, suggesting that tight control of lymphangion coordination may not be critical for lymphatic function.
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Rouard, Christophe. "Lymphangiome kystique de la rate : à propos d'un cas et revue de la littérature". Montpellier 1, 1995. http://www.theses.fr/1995MON11171.

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Rajagopalan, Shruti. "Device for automating in vitro characterization of lymphatic vessel function". Texas A&M University, 2004. http://hdl.handle.net/1969.1/1323.

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The lymphatic system consists of a network of vessels which work to return the interstitial fluid back to the blood circulation. Individual units called lymphangions, segments of lymphatic vessels between two valves, pump cyclically to propel lymph. Lymphangions are similar to the heart in that they are sensitive to both preload and afterload. To describe the heart independent of preload and afterload, investigators developed the concept of time-varying elastance. We evaluated the applicability of this concept to lymphangions by analyzing preliminary data obtained from the bovine mesenteric vessels. We found that there were some limitations to the applicability of this concept to lymphangions, as there was a high degree of variability with respect to contraction strength and frequency of individual time-varying elastance curves. To better characterize lymphangion mechanics, we built a device which would enable real-time isobaric, isometric and isotonic experiments in vitro. We performed all three experiments on lymphatic vessel segments and obtained input and output pressures, output flow, instantaneous radii and wall tension. The characterization of the lymphangion using these parameters can be the first step to simulate the behavior of a lymphatic vesssel and later the behavior of an entire lymphatic system.
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SENNEVILLE, ISABELLE. "Le lymphangiome vulvaire : a propos d'une observation". Lille 2, 1988. http://www.theses.fr/1988LIL2M162.

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TAHON, OLIVIER. "Lymphangiome du foie : a propos de deux cas et revue de la litterature". Amiens, 1988. http://www.theses.fr/1988AMIEM020.

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Saldar, Khan Isabelle. "Lymphangiome kystique du petit epiploon de l'adulte". Bordeaux 2, 1990. http://www.theses.fr/1990BOR25001.

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BUDIN, HERVE. "Etude anatomo-clinique des lymphangiomes kystiques du mediastin : a propos de deux observations personnelles ; revue de la litterature". Lyon 1, 1990. http://www.theses.fr/1990LYO1M048.

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Weiland, Nadine. "Lymphangiome kystique du pancreas : a propos d'un cas ; revue de la litterature". Nancy 1, 1992. http://www.theses.fr/1992NAN11180.

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LETURQUE, JACQUES. "Le lymphangiome kystique peritoneal de l'adulte : a propos d'un cas". Amiens, 1992. http://www.theses.fr/1992AMIEM008.

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Hajimirsadeghi, Elham. "Le lymphangiome kystique du médiastin à extension cervicale : à propos d'un cas". Montpellier 1, 1995. http://www.theses.fr/1995MON11162.

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Libros sobre el tema "Lymphangion"

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Cohen, Seymour R. Lymphangiomas of the larynx in infants and children: A survey of pediatric lymphangioma. St. Louis, MO: Annals Pub. Co., 1986.

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Sri-Ram, Kesavan, Anthony Mcgrath, Eric Yeung, Ben Spiegelberg, Nick Kalson, Barry Rose, Rob Pollock y John Skinner. Benign tumours of soft tissues. Oxford University Press, 2011. http://dx.doi.org/10.1093/med/9780199550647.003.002003.

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♦ Ganglion cyst♦ Intramuscular myxoma♦ Myositis ossificans♦ Nodular fasciitis♦ Haemangioma♦ Lipoma♦ Cavernous lymphangioma♦ Glomus tumour♦ Neurofibroma♦ Desmoid tumour♦ Elastofibroma♦ Schwannoma♦ Synovial chondromatosis.
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Foo, Eric y Bonnie N. Joe. Mass in Male (Gynecomastia, Cancer). Editado por Christoph I. Lee, Constance D. Lehman y Lawrence W. Bassett. Oxford University Press, 2018. http://dx.doi.org/10.1093/med/9780190270261.003.0028.

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Gynecomastia is a benign condition manifesting as enlarged breasts in men and boys. This increased breast tissue is caused by excess fibroglandular deposits and is caused by hormonal imbalances, commonly due to estrogen excess or from various drugs, such as spironolactone, ketoconazole, cimetidine, ranitidine, and specific HIV therapies. Patients generally present with bilateral subareolar enlarged breasts (however, unilateral gynecomastia also occurs), which commonly exhibit tenderness on palpation, swelling, palpable lumps, or nipple discharge. This chapter reviews the important imaging protocols, pitfalls, differential diagnoses, radiology–pathology correlation, and management recommendations for gynecomastia and cancer of the male breast. Topics discussed include gynecomastia, breast cancer, pseudogynecomastia, hypogonadism, and lymphangioma.
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Capítulos de libros sobre el tema "Lymphangion"

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Waldschmidt, J., U. Waldschmidt, S. Grasshoff y D. Cholewa. "Die Lymphangiome des Pharynx / Lymphangioma of the Pharynx". En Deutsche Gesellschaft für Chirurgie, 883. Berlin, Heidelberg: Springer Berlin Heidelberg, 2001. http://dx.doi.org/10.1007/978-3-642-56458-1_344.

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Strauss, Alexander. "Lymphangiom". En Ultraschallpraxis, 133–34. Berlin, Heidelberg: Springer Berlin Heidelberg, 2004. http://dx.doi.org/10.1007/978-3-662-10678-5_33.

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Mocellin, Simone. "Lymphangioma". En Soft Tissue Tumors, 527–28. Cham: Springer International Publishing, 2020. http://dx.doi.org/10.1007/978-3-030-58710-9_164.

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Franchi, Alessandro. "Lymphangioma". En Encyclopedia of Pathology, 181–82. Cham: Springer International Publishing, 2020. http://dx.doi.org/10.1007/978-3-030-41894-6_5005.

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Mazabraud, André. "Lymphangioma". En Pathology of bone tumours, 299–307. Berlin, Heidelberg: Springer Berlin Heidelberg, 1998. http://dx.doi.org/10.1007/978-3-642-95839-7_26.

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Campanacci, Mario, Franco Bertoni y Patrizia Bacchini. "Lymphangioma". En Bone and Soft Tissue Tumors, 595. Berlin, Heidelberg: Springer Berlin Heidelberg, 1990. http://dx.doi.org/10.1007/978-3-662-29279-2_36.

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Franchi, Alessandro. "Lymphangioma". En Encyclopedia of Pathology, 1–2. Cham: Springer International Publishing, 2019. http://dx.doi.org/10.1007/978-3-319-28845-1_5005-1.

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Savas, Jessica Alexis y Gloria F. Graham. "Lymphangioma Circumscriptum". En Dermatological Cryosurgery and Cryotherapy, 517–20. London: Springer London, 2016. http://dx.doi.org/10.1007/978-1-4471-6765-5_95.

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Chikkamuniyappa, Shylashree, Josephine Heim-Hall, Jaishree Jagirdar y Armando E. Fraire. "Cystic Lymphangioma". En Atlas of Neoplastic Pulmonary Disease, 67–68. Boston, MA: Springer US, 2009. http://dx.doi.org/10.1007/978-0-387-89839-1_16.

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Weissferdt, Annikka y Cesar A. Moran. "Mediastinal Lymphangioma". En Encyclopedia of Pathology, 243–47. Cham: Springer International Publishing, 2018. http://dx.doi.org/10.1007/978-3-319-66796-6_30.

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Actas de conferencias sobre el tema "Lymphangion"

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Bertram, Christopher D., Charles Macaskill y James E. Moore. "Effects of Lymphangion Subdivision in a Numerical Model of a Lymphatic Vessel". En ASME 2011 Summer Bioengineering Conference. American Society of Mechanical Engineers, 2011. http://dx.doi.org/10.1115/sbc2011-53231.

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We have recently reported development of a lumped-parameter model for several lymphangions in series [1]. The model provides for both active smooth muscle contraction (intrinsic pumping) and passive compression of the lymphatic by external tissues (extrinsic pumping). The valves which define the lymphangions vary their resistance sigmoidally, having a high (low) resistance for an adverse (favorable) pressure difference. With no refractory period between sinusoidal active tension episodes, maximum pumping efficiency was reached when each lymphangion contracted 135° after that immediately upstream; simultaneous contraction (corresponding to the situation of extrinsic pumping) was especially inefficient.
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Merkulova, Ksenia y Dmitry E. Postnov. "Mathematical model of lymphangion contractility". En Saratov Fall Meeting 2019: Computations and Data Analysis: from Nanoscale Tools to Brain Functions, editado por Dmitry E. Postnov. SPIE, 2020. http://dx.doi.org/10.1117/12.2565761.

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Stiukhina, Elena S., Nikolay I. Lvov, Evgeniya A. Kozlova, Grigory E. Brill y Dmitry E. Postnov. "Laser-stimulated lymphangion activity in rat mesentery". En Saratov Fall Meeting 2018: Computations and Data Analysis: from Nanoscale Tools to Brain Functions, editado por Dmitry E. Postnov. SPIE, 2019. http://dx.doi.org/10.1117/12.2523313.

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Dixon, J. Brandon, Ryan Akin, Mike Weiler y Timothy Kassis. "Non-Invasive Assessment of Lymphatic Pumping Pressure in a Rat Tail Model Utilizing Near-Infrared Imaging". En ASME 2013 Summer Bioengineering Conference. American Society of Mechanical Engineers, 2013. http://dx.doi.org/10.1115/sbc2013-14416.

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The lymphatic vasculature consists of a network of vessels that promote unidirectional transport of fluid, proteins, and cells from the interstitium back into the blood, providing functions essential for maintaining fluid balance, immune cell trafficking, and lipid absorption from the intestine. The lymphatics generate flow through both extrinsic pumping mechanisms, such as contraction of surrounding skeletal muscle, and through the intrinsic contractility of each lymphatic vessel unit known as a lymphangion. Specialized lymphatic muscle, working in coordination with uni-directional valves separating each lymphangion, serves to contract up to 80% of the vessel diameter and drive flow from the interstitium back to the venous circulation.
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5

Moore, James E. "Pumping Characteristics of the Lymphatic System". En ASME 2010 First Global Congress on NanoEngineering for Medicine and Biology. ASMEDC, 2010. http://dx.doi.org/10.1115/nemb2010-13034.

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The lymphatic system is crucial for maintaining fluid and protein balance, and for immune function. It also plays an important role in the spread of cancer. Lymphedema can form when parts of the system are not functioning, such as in patients who have undergone lymph node resection as part of cancer therapy. Lymphedema is a painful, debilitating condition for which there is no cure. This research is aimed at constructing multi-scale models of lymphatic pumping function, incorporating information from the cellular to the whole organ level. Lymphatic endothelial and smooth muscle cell mechanotransduction events are incorporated into a single lymphangion pumping unit. The performance of this pumping unit is shown to exhibit behaviors observed in experiments with rat mesentery.
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6

Taumberger, N., P. Gasparello, P. Greimel, M. Barth, M. Ragossnig, R. Hochstätter, AM Schütz, U. Lang y P. Klaritsch. "Zwei Fälle mit kongenitalem Lymphangiom". En 29. Deutscher Kongress für Perinatale Medizin. Deutsche Gesellschaft für Perinatale Medizin (DGPM) – „Hinterm Horizont geht's weiter, zusammen sind wir stark“. Georg Thieme Verlag KG, 2019. http://dx.doi.org/10.1055/s-0039-3401201.

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Singla, A., D. Theegarten, C. Aigner y K. Darwiche. "Zystisches Lymphangiom des Mediastinums: Eine tickende Zeitbombe". En 60. Kongress der Deutschen Gesellschaft für Pneumologie und Beatmungsmedizin e. V. Georg Thieme Verlag KG, 2019. http://dx.doi.org/10.1055/s-0039-1678288.

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Kasper, Victor, Alex Roman, Pedro Ruschel, Larissa Bianchini, Miguel Neto, Bárbara Battistel y Daniela Schwingel. "Orbital lymphangioma: case report and management paradigms". En XXXII Congresso Brasileiro de Neurocirurgia. Thieme Revinter Publicações Ltda, 2018. http://dx.doi.org/10.1055/s-0038-1673227.

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Le, Q.-K., J. Ruff, S. Huss, R. Lellé, C. De Santis, F. Metke, K. Zdanyte y R. Witteler. "Lymphangioma circumscriptum – Eine wichtige Differentialdiagnose zur Vulvaneoplasie". En Kongressabstracts zur Tagung 2020 der Deutschen Gesellschaft für Gynäkologie und Geburtshilfe (DGGG). © 2020. Thieme. All rights reserved., 2020. http://dx.doi.org/10.1055/s-0040-1718289.

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Weisgerber, C., B. Aktas y N. Dornhöfer. "Selten und doch belastend – Lymphangioma circumscriptum simplex der Vulva". En Kongressabstracts zur 13. Jahrestagung der Mitteldeutschen Gesellschaft für Frauenheilkunde und Geburtshilfe e.V. (MGFG). Georg Thieme Verlag KG, 2019. http://dx.doi.org/10.1055/s-0039-1692104.

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Informes sobre el tema "Lymphangion"

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Rua Carneiro, Carlos Santos. Infected Abdominal Cystic Lymphangioma: A Rare Cause of Inflammatory Acute Abdom. Science Repository OÜ, febrero de 2019. http://dx.doi.org/10.31487/j.scr.2019.01.008.

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