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Artículos de revistas sobre el tema "Mitosis"

Autor: Grafiati

Publicado: 4 de junio de 2021

Última modificación: 25 de julio de 2025

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1

Stratton, Miranda, and Tim Stearns. "Mitosis sans Mitosis: The Mitotic Oscillator in Differentiation." Developmental Cell 43, no. 4 (2017): 385–86. http://dx.doi.org/10.1016/j.devcel.2017.11.001.

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2

Kantsavaya, I., and O. Alekseenko. "Effect of Beta-lactam Antibiotics on Microscopic Parameters in the Allium-test." Bulletin of Science and Practice 5, no. 10 (2019): 25–31. http://dx.doi.org/10.33619/2414-2948/47/03.

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The work examines the effect of beta-lactam antibiotics (cefotaxime, ampicillin, augmentin) on the pathology of mitosis in the Allium–test. Research methods: Allium–test, cytogenetic analysis, statistical analysis. It was established that the use of individual tested beta-lactam antibiotics increases the percentage of pathological mitoses in the cell by 1.8–3.3 times compared with the value in the control. With the combined use of cefotaxime and Augmentin, synergism appeared, as a result, the value of mitosis pathology turned out to be at the level of the number in the control; minimally repre

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3

Kantsavaya, I., and O. Alekseenko. "Effect of Beta-lactam Antibiotics on Microscopic Parameters in the Allium-test." Bulletin of Science and Practice 5, no. 10 (2019): 25–31. https://doi.org/10.33619/2414-2948/47/03.

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The work examines the effect of beta-lactam antibiotics (cefotaxime, ampicillin, augmentin) on the pathology of mitosis in the Allium-test. Research methods: Allium-test, cytogenetic analysis, statistical analysis. It was established that the use of individually tested beta-lactam antibiotics increases the percentage of pathological mitoses in the cell by 1.8–3.3.3 times compared with the value in the control. With the combined use of cefotaxime and augmentin, synergism appeared, as a result, the value of mitosis pathology turned out to be at the level of the number in the control;

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4

Solnica-Krezel, L., T. G. Burland, and W. F. Dove. "Variable pathways for developmental changes of mitosis and cytokinesis in Physarum polycephalum." Journal of Cell Biology 113, no. 3 (1991): 591–604. http://dx.doi.org/10.1083/jcb.113.3.591.

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The development of a uninucleate ameba into a multinucleate, syncytial plasmodium in myxomycetes involves a change from the open, astral mitosis of the ameba to the intranuclear, anastral mitosis of the plasmodium, and the omission of cytokinesis from the cell cycle. We describe immunofluorescence microscopic studies of the amebal-plasmodial transition (APT) in Physarum polycephalum. We demonstrate that the reorganization of mitotic spindles commences in uninucleate cells after commitment to plasmodium formation, is completed by the binucleate stage, and occurs via different routes in individu

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5

Douglas, Pauline, Ruiqiong Ye, Nicholas Morrice, Sébastien Britton, Laura Trinkle-Mulcahy, and Susan P. Lees-Miller. "Phosphorylation of SAF-A/hnRNP-U Serine 59 by Polo-Like Kinase 1 Is Required for Mitosis." Molecular and Cellular Biology 35, no. 15 (2015): 2699–713. http://dx.doi.org/10.1128/mcb.01312-14.

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Scaffold attachment factor A (SAF-A), also called heterogenous nuclear ribonuclear protein U (hnRNP-U), is phosphorylated on serine 59 by the DNA-dependent protein kinase (DNA-PK) in response to DNA damage. Since SAF-A, DNA-PK catalytic subunit (DNA-PKcs), and protein phosphatase 6 (PP6), which interacts with DNA-PKcs, have all been shown to have roles in mitosis, we asked whether DNA-PKcs phosphorylates SAF-A in mitosis. We show that SAF-A is phosphorylated on serine 59 in mitosis, that phosphorylation requires polo-like kinase 1 (PLK1) rather than DNA-PKcs, that SAF-A interacts with PLK1 in

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6

Nasuda, Shuhei, Bernd Friebe, and Bikram S. Gill. "Gametocidal Genes Induce Chromosome Breakage in the Interphase Prior to the First Mitotic Cell Division of the Male Gametophyte in Wheat." Genetics 149, no. 2 (1998): 1115–24. http://dx.doi.org/10.1093/genetics/149.2.1115.

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Abstract Male gametogenesis was cytologically analyzed in wheat lines homozygous or hemizygous for gametocidal (Gc) factors with different modes of action. The first and second meiotic divisions in all lines were cytologically normal. The postmeiotic mitoses were normal in the homozygous lines; however, chromosome fragments and bridges were observed in the mitoses of the hemizygous lines. The morphology of the chromosome fragments suggests that the Gc genes induce chromosome breaks in the G1 phase prior to DNA synthesis of the first postmeiotic mitosis. The age of an anther was correlated with

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7

Brinkley, William (B R. ). "Romancing mitosis and the mitotic apparatus." Molecular Biology of the Cell 25, no. 21 (2014): 3270–72. http://dx.doi.org/10.1091/mbc.e14-06-1123.

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One of the earliest lessons students learn in biology is the process of mitosis and how cells divide to produce daughter cells. Although first described more than a century ago by early investigators such as E. B. Wilson, many aspects of mitosis and cell division remain the subject of considerable research today. My personal investigations and research contributions to the study of mitosis were made possible by recent developments in the field when I began my career, including access to novel mammalian cell culture models and electron and fluorescence microscopy. Building upon those innovation

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8

Sturm, Bart, David Creytens, Jan Smits, et al. "Computer-Aided Assessment of Melanocytic Lesions by Means of a Mitosis Algorithm." Diagnostics 12, no. 2 (2022): 436. http://dx.doi.org/10.3390/diagnostics12020436.

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An increasing number of pathology laboratories are now fully digitised, using whole slide imaging (WSI) for routine diagnostics. WSI paves the road to use artificial intelligence (AI) that will play an increasing role in computer-aided diagnosis (CAD). In melanocytic skin lesions, the presence of a dermal mitosis may be an important clue for an intermediate or a malignant lesion and may indicate worse prognosis. In this study a mitosis algorithm primarily developed for breast carcinoma is applied to melanocytic skin lesions. This study aimed to assess whether the algorithm could be used in dia

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9

Cancel, Limary M., and John M. Tarbell. "The role of mitosis in LDL transport through cultured endothelial cell monolayers." American Journal of Physiology-Heart and Circulatory Physiology 300, no. 3 (2011): H769—H776. http://dx.doi.org/10.1152/ajpheart.00445.2010.

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We ( 7 ) have previously shown that leaky junctions associated with dying or dividing cells are the dominant pathway for LDL transport under convective conditions, accounting for >90% of the transport. We ( 8 ) have also recently shown that the permeability of bovine aortic endothelial cell monolayers is highly correlated with their rate of apoptosis and that inhibiting apoptosis lowers the permeability of the monolayers to LDL. To explore the role of mitosis in the leaky junction pathway, the microtubule-stabilizing agent paclitaxel was used to alter the rate of mitosis, and LDL flux and w

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10

Helfer, Hanspeter, and Amy S. Gladfelter. "AgSwe1p Regulates Mitosis in Response to Morphogenesis and Nutrients in Multinucleated Ashbya gossypii Cells." Molecular Biology of the Cell 17, no. 10 (2006): 4494–512. http://dx.doi.org/10.1091/mbc.e06-03-0215.

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Nuclei in the filamentous, multinucleated fungus Ashbya gossypii divide asynchronously. We have investigated what internal and external signals spatially direct mitosis within these hyphal cells. Mitoses are most common near cortical septin rings found at growing tips and branchpoints. In septin mutants, mitoses are no longer concentrated at branchpoints, suggesting that the septin rings function to locally promote mitosis near new branches. Similarly, cells lacking AgSwe1p kinase (a Wee1 homologue), AgHsl1p (a Nim1-related kinase), and AgMih1p phosphatase (the Cdc25 homologue that likely coun

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11

Wang, Richard R. C., Xiaomei Li, and N. Jerry Chatterton. "Cytological evidence for assortment mitosis leading to loss of heterozygosity in rice." Genome 49, no. 5 (2006): 556–57. http://dx.doi.org/10.1139/g06-015.

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In the root meristem cells of the rice line AMR, which causes loss of heterozygosity in its hybrids, both normal and assortment mitoses were observed. During normal mitosis, chromosomes did not form homologous pairs at metaphase; all chromosomes lined up at the equatorial plate and 2 chromatids of each chromosome disjoined at the centromere and moved toward opposite poles. During assortment mitosis, varying numbers of paired homologues were observed at mitotic metaphase. Two groups of 12 chromosomes separated and moved towards the opposite poles of daughter cells with few chromosomes having th

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12

Su, Tin Tin, and Patrick H. O'Farrell. "Chromosome Association of Minichromosome Maintenance Proteins in Drosophila Mitotic Cycles." Journal of Cell Biology 139, no. 1 (1997): 13–21. http://dx.doi.org/10.1083/jcb.139.1.13.

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Minichromosome maintenance (MCM) proteins are essential DNA replication factors conserved among eukaryotes. MCMs cycle between chromatin bound and dissociated states during each cell cycle. Their absence on chromatin is thought to contribute to the inability of a G2 nucleus to replicate DNA. Passage through mitosis restores the ability of MCMs to bind chromatin and the ability to replicate DNA. In Drosophila early embryonic cell cycles, which lack a G1 phase, MCMs reassociate with condensed chromosomes toward the end of mitosis. To explore the coupling between mitosis and MCM–chromatin interac

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13

Steinbeck, RG. "Pathologic mitoses and pathology of mitosis in tumorigenesis." European Journal of Histochemistry 45, no. 4 (2009): 311. http://dx.doi.org/10.4081/1640.

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14

Murray, Andrew W. "Never-in-mitosis in mitosis." Nature 353, no. 6346 (1991): 701–2. http://dx.doi.org/10.1038/353701a0.

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15

Su, Tin Tin, Devin H. Parry, Bryon Donahoe, Cheng-Ting Chien, Patrick H. O’Farrell, and Amanda Purdy. "Cell cycle roles for two 14-3-3 proteins during Drosophila development." Journal of Cell Science 114, no. 19 (2001): 3445–54. http://dx.doi.org/10.1242/jcs.114.19.3445.

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Drosophila 14-3-3ε and 14-3-3ζ proteins have been shown to function in RAS/MAP kinase pathways that influence the differentiation of the adult eye and the embryo. Because 14-3-3 proteins have a conserved involvement in cell cycle checkpoints in other systems, we asked (1) whether Drosophila 14-3-3 proteins also function in cell cycle regulation, and (2) whether cell proliferation during Drosophila development has different requirements for the two 14-3-3 proteins. We find that antibody staining for 14-3-3 family members is cytoplasmic in interphase and perichromosomal in mitosis. Using mutants

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16

Bernard, Pascal, Kevin Hardwick, and Jean-Paul Javerzat. "Fission Yeast Bub1 Is a Mitotic Centromere Protein Essential for the Spindle Checkpoint and the Preservation of Correct Ploidy through Mitosis." Journal of Cell Biology 143, no. 7 (1998): 1775–87. http://dx.doi.org/10.1083/jcb.143.7.1775.

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The spindle checkpoint ensures proper chromosome segregation by delaying anaphase until all chromosomes are correctly attached to the mitotic spindle. We investigated the role of the fission yeast bub1 gene in spindle checkpoint function and in unperturbed mitoses. We find that bub1+ is essential for the fission yeast spindle checkpoint response to spindle damage and to defects in centromere function. Activation of the checkpoint results in the recruitment of Bub1 to centromeres and a delay in the completion of mitosis. We show that Bub1 also has a crucial role in normal, unperturbed mitoses.

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17

Schatten, Heide, Christopher N. Hueser, and Amitabha Chakrabarti. "Centrosome Structure And Function Is Altered By Experimental Manipulations With Formamide: Implications For Abnormal Cell Divisions During Cancer." Microscopy and Microanalysis 5, S2 (1999): 1286–87. http://dx.doi.org/10.1017/s1431927600019759.

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The formation of abnormal mitosis associated with cancer has been intriguing for many decades. While microtubules had been the focus of previous studies, recent research has focused on centrosomes, microtubule organizing centers which organize the mitotic apparatus during cell division. During normal mitosis centrosomes form two poles but in cancer, centrosomes can form three, four, or more poles, and organize tripolar, quadripolar, and multipolar mitoses, respectively. This has severe consequences for genomic stability because chromosomes are separated unequally to three, four, or more poles.

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18

Meitinger, Franz, Hazrat Belal, Robert L. Davis, et al. "Control of cell proliferation by memories of mitosis." Science 383, no. 6690 (2024): 1441–48. http://dx.doi.org/10.1126/science.add9528.

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Mitotic duration is tightly constrained, and extended mitosis is characteristic of problematic cells prone to chromosome missegregation and genomic instability. We show here that mitotic extension leads to the formation of p53-binding protein 1 (53BP1)–ubiquitin-specific protease 28 (USP28)–p53 protein complexes that are transmitted to, and stably retained by, daughter cells. Complexes assembled through a Polo-like kinase 1–dependent mechanism during extended mitosis and elicited a p53 response in G 1 that prevented the proliferation of the progeny of cells that experienced an approximately th

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19

Kurihara, Daisuke. "Mitotic kinases regulate chromosome dynamics during mitosis." PLANT MORPHOLOGY 23, no. 1 (2011): 81–89. http://dx.doi.org/10.5685/plmorphol.23.81.

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20

Staiber, Wolfgang. "Chromosome elimination in germ line – soma differentiation of Acricotopus lucidus (Diptera, Chironomidae)." Genome 49, no. 3 (2006): 269–74. http://dx.doi.org/10.1139/g05-103.

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During germ line – soma differentiation in early syncytial embryonic development of the chironomid Acricotopus lucidus, a complement of supernumerary chromosomes, the so-called germ line limited chromosomes (Ks), is excluded from the future somatic nuclei in the course of elimination mitoses. The Ks lag behind in the equatorial plane, while the somatic chromosomes (Ss) segregate equally. After elimination mitoses, the Ks are only present in the pole cells, the primary germ cells. In the divisions before their elimination, the Ks frequently showed delayed separation of sister chromatids with hi

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21

Su, Tin Tin, and Patrick H. O'Farrell. "Chromosome Association of Minichromosome Maintenance Proteins in Drosophila Endoreplication Cycles." Journal of Cell Biology 140, no. 3 (1998): 451–60. http://dx.doi.org/10.1083/jcb.140.3.451.

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Minichromosome maintenance (MCM) proteins are essential eukaryotic DNA replication factors. The binding of MCMs to chromatin oscillates in conjunction with progress through the mitotic cell cycle. This oscillation is thought to play an important role in coupling DNA replication to mitosis and limiting chromosome duplication to once per cell cycle. The coupling of DNA replication to mitosis is absent in Drosophila endoreplication cycles (endocycles), during which discrete rounds of chromosome duplication occur without intervening mitoses. We examined the behavior of MCM proteins in endoreplicat

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22

Gupta, Mita, Deborah Trott, and Andrew C. G. Porter. "Rescue of a Human Cell Line from Endogenous Cdk1 Depletion by Cdk1 Lacking Inhibitory Phosphorylation Sites." Journal of Biological Chemistry 282, no. 7 (2006): 4301–9. http://dx.doi.org/10.1074/jbc.m607910200.

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Cells that transiently overexpress cyclin-dependent kinase 1 lacking inhibitory phosphorylation sites (Cdk1-AF) undergo premature and catastrophic mitosis, reflecting the key role for Cdk1 in promoting a timely transit from G2 into mitosis. Conversely, cells depleted of Cdk1 undergo repeated S phases without intervening mitoses (endoreduplication), reflecting a role for Cdk1 in preventing premature S phases. It is not known how Cdk1 prevents entry into S phase at times in G2 when it does not promote mitosis. Also uncertain is the extent of redundancy between inhibitory phosphorylation and othe

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23

McIntosh, J. Richard. "Mitosis." Cold Spring Harbor Perspectives in Biology 8, no. 9 (2016): a023218. http://dx.doi.org/10.1101/cshperspect.a023218.

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24

McIntosh and M. Koonce. "Mitosis." Science 246, no. 4930 (1989): 622–28. http://dx.doi.org/10.1126/science.2683078.

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25

Barral, Yves. "Mitosis." Developmental Cell 6, no. 5 (2004): 608–10. http://dx.doi.org/10.1016/s1534-5807(04)00138-8.

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26

Maslak, P. "Mitosis." ASH Image Bank 2005, no. 0301 (2005): 101301. http://dx.doi.org/10.1182/ashimagebank-2005-101301.

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27

Earnshaw, William C., and Ann F. Pluta. "Mitosis." BioEssays 16, no. 9 (1994): 639–43. http://dx.doi.org/10.1002/bies.950160908.

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28

Hand, A. R., and B. Ho. "Mitosis and Hypertrophy of Intercalated Duct Cells and Endothelial Cells in the Isoproterenol-treated Rat Parotid Gland." Journal of Dental Research 64, no. 8 (1985): 1031–38. http://dx.doi.org/10.1177/00220345850640080201.

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Chronic treatment of rats with isoproterenol (IPR) induces mitosis and differentiation of intercalated duct cells, and mitosis and hypertrophy of endothelial cells of the microvasculature in the parotid gland. Mitoses occurred in duct cells situated at or near the acinar-intercalated duct junction, and were most numerous after six IPR injections. These cells increased in size, contained numerous electronlucent granules, and were incorporated into the hypertrophic acini. The granules contained branching filamentous or finely reticulated material, and did not react with histochemical stains for

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29

Ma, Li, Xiangshan Zhao, and Xueliang Zhu. "Mitosin/CENP-F in mitosis, transcriptional control, and differentiation." Journal of Biomedical Science 13, no. 2 (2006): 205–13. http://dx.doi.org/10.1007/s11373-005-9057-3.

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30

Gómez-Conde, Eduardo, Raul Mena-López, Pablo Hernndez-Jaúregui, Mónica González-Camacho, and Rossana Arroyo. "Trichomonas vaginalis: Chromatin and Mitotic Spindle during Mitosis." Experimental Parasitology 96, no. 3 (2000): 130–38. http://dx.doi.org/10.1006/expr.2000.4551.

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31

Quintanar-Stephano, A., and C. Valverde-R. "Mitogenic effects of thyroxine and TRH on thyrotrophs and somatotrophs of the anterior pituitary gland in thyroidectomized rats." Journal of Endocrinology 154, no. 1 (1997): 149–53. http://dx.doi.org/10.1677/joe.0.1540149.

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Abstract The question of whether thyroxine (T4) and TRH have a mitogenic effect on pituitary thyrotrophs and somatotrophs in thyroidectomized rats was investigated. Mitoses were counted in hematoxylin–eosin-stained or periodic acid–Schiff–hematoxylin-stained pituitary slides or immunostained for TSH or GH using male rats thyroidectomized for 5 months. Ten days before they were killed groups of rats were injected with different doses of T4 (0·5, 3 or 10 μg i.m. every second day for 10 days), TRH alone (100 ng s.c. three times a day for 10 days), or T4 plus TRH (same doses as above). Mitoses (st

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32

Gillett, Emily S., Christopher W. Espelin, and Peter K. Sorger. "Spindle checkpoint proteins and chromosome–microtubule attachment in budding yeast." Journal of Cell Biology 164, no. 4 (2004): 535–46. http://dx.doi.org/10.1083/jcb.200308100.

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Accurate chromosome segregation depends on precise regulation of mitosis by the spindle checkpoint. This checkpoint monitors the status of kinetochore–microtubule attachment and delays the metaphase to anaphase transition until all kinetochores have formed stable bipolar connections to the mitotic spindle. Components of the spindle checkpoint include the mitotic arrest defective (MAD) genes MAD1–3, and the budding uninhibited by benzimidazole (BUB) genes BUB1 and BUB3. In animal cells, all known spindle checkpoint proteins are recruited to kinetochores during normal mitoses. In contrast, we sh

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33

R Shihabuddin, Abdul, and Sabeena Beevi K. "Efficient mitosis detection: leveraging pre-trained faster R-CNN and cell-level classification." Biomedical Physics & Engineering Express 10, no. 2 (2024): 025031. http://dx.doi.org/10.1088/2057-1976/ad262f.

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Abstract The assessment of mitotic activity is an integral part of the comprehensive evaluation of breast cancer pathology. Understanding the level of tumor dissemination is essential for assessing the severity of the malignancy and guiding appropriate treatment strategies. A pathologist must manually perform the intricate and time-consuming task of counting mitoses by examining biopsy slices stained with Hematoxylin and Eosin (H&E) under a microscope. Mitotic cells can be challenging to distinguish in H&E-stained sections due to limited available datasets and similarities among mitoti

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34

Trinkaus, J. P. "The midblastula transition, the YSL transition and the onset of gastrulation in Fundulus." Development 116, Supplement (1992): 75–80. http://dx.doi.org/10.1242/dev.116.supplement.75.

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The first signs of cell motility appear in Fundulus toward the end of cleavage, after cleavages 11 and 12. When blastomeres cease cleaving, their surfaces undulate and form blebs. At first, these blebbing cells remain in place. Gradually thereafter they begin movement, with blebs and fllolamellipodia serving as organs of locomotion. Non-motile cleaving blastomeres have thus differentiated into motile blastula cells. This transformation corresponds to the midblastula transition of amphibian embryos. Gastrulation in Fundulus begins with vegetalward contraction of the external yolk syncytial laye

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35

Kuraś, M., and A. Malinowska. "Influence of 1-p-D-arabinofuranosylcytosine on mitotic activity of apical meristem of onion (Allium cepa L.) roots." Acta Societatis Botanicorum Poloniae 47, no. 1–2 (2015): 173–87. http://dx.doi.org/10.5586/asbp.1978.015.

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The influence of increasing cytosine arabinoside (ara-C) concentration (50, 100, 300 and 500 μg/ml) on the mitotic activity of the apical meristem of onion adventitious roots was investigated during 24-h incubation in ara-C and postincubation in water. Incubation in ara-C inhibits reversibly mitosis, the degree of inhibition being dependent on the concentration used. 50 μg/ml ara-C causes only a slight and transitional mitotic depression, while 100—500 μg/ml reduces the per cent of mitoses in various degrees after 12-h incubation whereas after 24 h all concentrations (100—500) reduce mitosis t

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36

Chow, Zi Long, Aye Aye Thike, Hui Hua Li, Nur Diyana Md Nasir, Joe Poh Sheng Yeong, and Puay Hoon Tan. "Counting Mitoses With Digital Pathology in Breast Phyllodes Tumors." Archives of Pathology & Laboratory Medicine 144, no. 11 (2020): 1397–400. http://dx.doi.org/10.5858/arpa.2019-0435-oa.

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Context.— Mitotic count is an important histologic criterion for grading and prognostication in phyllodes tumors (PTs). Counting mitoses is a routine practice for pathologists evaluating neoplasms, but different microscopes, variable field selection, and areas have led to possible misclassification. Objective.— To determine whether 10 high-power fields (HPFs) or whole slide mitotic counts correlated better with PT clinicopathologic parameters using digital pathology (DP). We also aimed to find out whether this study might serve as a basis for an artificial intelligence (AI) protocol to count m

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37

Di Bari, Maria, Vanessa Tombolillo, Francesco Alessandrini, et al. "M2 Muscarinic Receptor Activation Impairs Mitotic Progression and Bipolar Mitotic Spindle Formation in Human Glioblastoma Cell Lines." Cells 10, no. 7 (2021): 1727. http://dx.doi.org/10.3390/cells10071727.

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Background: Glioblastoma multiforme (GBM) is characterized by several genetic abnormalities, leading to cell cycle deregulation and abnormal mitosis caused by a defective checkpoint. We previously demonstrated that arecaidine propargyl ester (APE), an orthosteric agonist of M2 muscarinic acetylcholine receptors (mAChRs), arrests the cell cycle of glioblastoma (GB) cells, reducing their survival. The aim of this work was to better characterize the molecular mechanisms responsible for this cell cycle arrest. Methods: The arrest of cell proliferation was evaluated by flow cytometry analysis. Usin

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38

Lorson, Monique A., H. Robert Horvitz, and Sander van den Heuvel. "LIN-5 Is a Novel Component of the Spindle Apparatus Required for Chromosome Segregation and Cleavage Plane Specification in Caenorhabditis elegans." Journal of Cell Biology 148, no. 1 (2000): 73–86. http://dx.doi.org/10.1083/jcb.148.1.73.

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Successful divisions of eukaryotic cells require accurate and coordinated cycles of DNA replication, spindle formation, chromosome segregation, and cytoplasmic cleavage. The Caenorhabditis elegans gene lin-5 is essential for multiple aspects of cell division. Cells in lin-5 null mutants enter mitosis at the normal time and form bipolar spindles, but fail chromosome alignment at the metaphase plate, sister chromatid separation, and cytokinesis. Despite these defects, cells exit from mitosis without delay and progress through subsequent rounds of DNA replication, centrosome duplication, and abor

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39

David, Rachel. "Dissecting mitosis." Nature Reviews Molecular Cell Biology 11, no. 5 (2010): 310. http://dx.doi.org/10.1038/nrm2892.

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40

Quiñones, Carlos García, Carlos Madriles, Jesús Sánchez, Pedro Marcuello, Antonio González, and Dean M. Tullsen. "Mitosis compiler." ACM SIGPLAN Notices 40, no. 6 (2005): 269–79. http://dx.doi.org/10.1145/1064978.1065043.

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41

Møller-Jensen, Jakob, Jonas Borch, Mette Dam, Rasmus B. Jensen, Peter Roepstorff, and Kenn Gerdes. "Bacterial Mitosis." Molecular Cell 12, no. 6 (2003): 1477–87. http://dx.doi.org/10.1016/s1097-2765(03)00451-9.

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42

Donhuijsen, K. "Mitosis counts:." Human Pathology 17, no. 11 (1986): 1122–25. http://dx.doi.org/10.1016/s0046-8177(86)80417-8.

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Stuedell, David. "Biomorphic mitosis." Computers & Graphics 15, no. 3 (1991): 455. http://dx.doi.org/10.1016/0097-8493(91)90017-c.

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Clarke, Duncan J. "Mitosis: Introduction." Cell Cycle 1, no. 5 (2002): 298–99. http://dx.doi.org/10.4161/cc.1.5.141.

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Kiritchenko, Valentina. "Geometric mitosis." Mathematical Research Letters 23, no. 4 (2016): 1071–97. http://dx.doi.org/10.4310/mrl.2016.v23.n4.a5.

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Husseman, J., D. Nochlin, and I. Vicent. "ATTEMPTED MITOSIS." Journal of Neuropathology and Experimental Neurology 58, no. 5 (1999): 530. http://dx.doi.org/10.1097/00005072-199905000-00095.

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Crncec, Adrijana, and Helfrid Hochegger. "Triggering mitosis." FEBS Letters 593, no. 20 (2019): 2868–88. http://dx.doi.org/10.1002/1873-3468.13635.

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Pines, Jonathon, and Conly L. Rieder. "Re-staging mitosis: a contemporary view of mitotic progression." Nature Cell Biology 3, no. 1 (2001): E3—E6. http://dx.doi.org/10.1038/35050676.

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Huang, Xin, and Jianlong Wang. "Mitotic Bookmarking: Maintaining the Stem Cell Identity during Mitosis." Cell Stem Cell 20, no. 6 (2017): 741–42. http://dx.doi.org/10.1016/j.stem.2017.05.002.

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